Relevant bibliographies by topics / SGSY

Academic literature on the topic 'SGSY'

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Published: 18 May 2024

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Journal articles on the topic "SGSY"

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Dr. P. RAMALAKSHMI, Dr P. RAMALAKSHMI. "Women Empowerment Through SGSY Program in Virudhunagar." Indian Journal of Applied Research 4, no. 4 (October 1, 2011): 53–56. http://dx.doi.org/10.15373/2249555x/apr2014/213.

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Chatterjee, Shankar. "ECONOMIC IMPROVEMENT OF RURAL BPL HOUSEHOLDS THROUGH SHGS: A STUDY AT AURANGABAD DISTRICT OF MAHARASHTRA." International Journal of Research -GRANTHAALAYAH 7, no. 3 (March 31, 2019): 328–34. http://dx.doi.org/10.29121/granthaalayah.v7.i3.2019.978.

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This study was carried out among the members of self-help groups (SHGs) at Aurangabad District of Maharashtra in the Month of March 2019. Altogether, two SHGs located in two different areas along with the few members of SHGs were contacted to get an idea about the functioning of self-help group (SHG) in the district as well as earning (income) of members after joining in the SHG. The SHGs were part of earlier Swarnajayanthi Gram Swarojgar Yojana (SGSY), a self-employment scheme sponsored under Ministry of Rural Development, Government of India. In view of this few lines about SGSY and its progress are presented here for the benefit of readers. So, the study is based on both primary and secondary data.
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Ray, Sthitapragyan. "Alleviating Poverty through Micro-finance: SGSY Experience in Orissa." Sociological Bulletin 57, no. 2 (May 2008): 211–40. http://dx.doi.org/10.1177/0038022920080204.

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SINGH, RAHUL KUMAR, R. K. DOHAREY, N. K. TIWARI, and CHANDAN KUMAR SINGH. "Constraints in efficient functioning of dairy enterprise under SGSY." VETERINARY SCIENCE RESEARCH JOURNAL 7, no. 1 (April 15, 2016): 39–41. http://dx.doi.org/10.15740/has/vsrj/7.1/39-41.

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Sivasankar, P. R., M. Madhusudan Varma, and K. Ekambaram. "Evaluation of Swamajayanthi Gram Swarozgar Yojana (SGSY): A Case Study." SEDME (Small Enterprises Development, Management & Extension Journal): A worldwide window on MSME Studies 33, no. 2 (June 2006): 29–36. http://dx.doi.org/10.1177/0970846420060203.

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Tadalapur, Dr Veershetty C. "Role of NGOs in Social Mobilization in the context of SGSY." Indian Journal of Applied Research 1, no. 9 (October 1, 2011): 209–11. http://dx.doi.org/10.15373/2249555x/jun2012/74.

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Pankaja, HK, SV Suresha, and NS Shivalinge Gowda. "Impact of swarna jayanthi gram swarozgar yojana (SGSY) on women empowerment." International Journal of Chemical Studies 8, no. 1 (January 1, 2020): 1358–62. http://dx.doi.org/10.22271/chemi.2020.v8.i1s.8442.

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Narasaiah, P. V., Sudarsana Murthy, and P. Suresh. "Development of Women Entrepreneurship through Swamajayanthi Gram Swarozgar Yojana (SGSY) - A Review." SEDME (Small Enterprises Development, Management & Extension Journal): A worldwide window on MSME Studies 33, no. 2 (June 2006): 37–42. http://dx.doi.org/10.1177/0970846420060204.

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P, Dona. "Performance of Activity Group under the Swarnjayanti Gram Swarozgar Yojana (SGSY) in Kerala." International Journal of Pure & Applied Bioscience 6, no. 6 (December 31, 2018): 54–59. http://dx.doi.org/10.18782/2320-7051.7067.

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Bori, Bhumika. "Impact of Swarnajayanti Gram Swarozgar Yojana (Sgsy) On Poverty Alleviation in Golaghat District, Assam." IOSR Journal of Humanities and Social Science 19, no. 9 (2014): 53–56. http://dx.doi.org/10.9790/0837-19955356.

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Dissertations / Theses on the topic "SGSY"

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Roy, Mahendra. "Panchayats, participatory rural development and livelihood strategies: a block level study of self help groups under SGSY in the coochbehar district of West Bengal." Thesis, University of North Bengal, 2013. http://hdl.handle.net/123456789/1552.

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Norman, Zandra. "SGSN-MME Test Node Pool - Resources utilization for SGSN test nodes." Thesis, KTH, Skolan för informations- och kommunikationsteknik (ICT), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-143343.

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The SGSN-MME node, which is important in wireless networks, handles many users and therefore the uptime requirements for it are very high. The goal at Ericsson is to reach 99.9999% uptime for their SGSN-MME nodes and to reach this a lot of testing is required. Therefore the test process during the SGSN-MME development is both resource expensive and time consuming. To optimize both resource utilization and test runtimes a common test node pool solution for their different test tools has been proposed. During this thesis a first exploratory investigation about how to optimize such a solution was made. During the investigation different aspects were evaluated and a first input about how an optimal solution can be implemented is proposed. By having a scheduling layer in the common node pool, which determines how many nodes each regression job will get, depending on current load, the number of test cases in the job and the current node utilization optimized solutions can be found. Future work in the area is still needed, but the exploratory research made during this thesis will give a good base to continue from.
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Muthuswamy, Papanash Om Prakash. "SGSN integration and implementation." Thesis, Uppsala universitet, Institutionen för informationsteknologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-174826.

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The goal of this Thesis project is to implement a prototype  for Serving GPRS Support Node (SGSN) which is the central part of the General packet radio service (GPRS). SGSN is responsible for the transfer of data packets between the mobile stations covered in its geographical service area. The SGSN should be integrated into existing Mobile Arts products, open source projects and nanoBTS radio hardware to construct a minimal GPRS network that support sending and receiving Short Message Service (SMS). The prototype  is tested with radio hardware and mobile phones.
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Novoa, Carolina. "RecQ-like helicase SGS1 counteracts DNA : RNA hybrid induced genome instability." Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/60964.

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Dividing cells are constantly under threat from both endogenous and exogenous DNA damaging stresses that can lead to mutations and structural variations in DNA. One contributor to genome instability is three-stranded DNA:RNA hybrid structures called R-loops. Though R-loops are known to induce DNA damage and DNA replication stress, it is unclear whether they are recognized and processed by an established DNA repair pathway prior to inducing DNA breaks. Canonically, DNA repair proteins work downstream of R-loop-induced DNA damage to stimulate repair and suppress genome instability. Recently, the possibility that some DNA repair pathways actively destabilize R-loops, thus preventing unscheduled DNA damage has emerged. Here we identify the helicase SGS1 as a suppressor of R-loop stability. Our data reveals that SGS1 depleted cells accumulate R-loops. In addition, we define a role for transcription in genome instability of cells lacking SGS1, which is consistent with an R-loop based mechanism. Hyper-recombination in SGS1 mutants is dependent on transcript length, transcription rate, and active DNA replication. Also, rDNA instability in sgs1Δ can be suppressed by ectopic expression of RNaseH1, a protein that degrades DNA:RNA hybrids. Interestingly, R-loops are known to form at rDNA loci. We favour a model in which SGS1 contributes to the stabilization of stalled replication forks associated with transcription complexes, and unresolved DNA:RNA hybrids. Finally, we showed that knockdown of the human Sgs1 orthologue BLM in HCT116 cells also led to the accumulation of more R-loops than control HCT116 cells. In summary, our data supports the idea that some DNA repair proteins involved in replication fork stabilization might also prevent and process R-loops.
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Giangrande, Angela. "Etude in vivo et in vitro de la regulation de sgs3." Strasbourg 1, 1988. http://www.theses.fr/1988STR13069.

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Giangrande, Angela. "Etude in vivo et in vitro de la régulation de Sgs3." Grenoble 2 : ANRT, 1988. http://catalogue.bnf.fr/ark:/12148/cb376139014.

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Pešková, Zuzana. "Využití internetu v marketingových aktivitách mezinárodní firmy SGS." Master's thesis, Vysoká škola ekonomická v Praze, 2008. http://www.nusl.cz/ntk/nusl-3233.

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Diplomová práce analyzuje možnosti využití internetu v marketingu se zaměřením na webové stránky a jejich roli v rámci marketingové strategie; elektronickou poštu a její dominantní postavení v rámci komunikace, extranet a jeho využití v rámci komunikace se zákazníky a intranet pro interní komunikaci. Část práce je také věnována reklamě na internetu. Konkrétně je práce věnována využití těchto nástrojů ve společnosti SGS.
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Campos-Doerfler, Lillian. "The Role of Sgs1 and Exo1 in the Maintenance of Genome Stability." Thesis, University of South Florida, 2018. http://pqdtopen.proquest.com/#viewpdf?dispub=10642568.

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Genome instability is a hallmark of human cancers. Patients with Bloom’s syndrome, a rare chromosome breakage syndrome caused by inactivation of the RecQ helicase BLM, result in phenotypes associated with accelerated aging and develop cancer at a very young age. Patients with Bloom’s syndrome exhibit hyper-recombination, but the role of BLM and increased genomic instability is not fully characterized. Sgs1, the only member of the RecQ family of DNA helicases in Saccharomyces cerevisiae, is known to act both in early and late stages of homology-dependent repair of DNA damage. Exo1, a 5'–3' exonuclease, first discovered to play a role in mismatch repair has been shown to participate in parallel to Sgs1 in processing the ends of DNA double-strand breaks, an early step of homology-mediated repair. Here we have characterized the genetic interaction of SGS1 and EXO1 with other repair factors in homology-mediated repair as well as DNA damage checkpoints, and characterize the role of post-translational modifications, and protein-protein interactions in regulating their function in response to DNA damage. In S. cerevisiae cells lacking Sgs1, spontaneous translocations arise by homologous recombination in small regions of homology between three non-allelic, but related sequences in the genes CAN1, LYP1, and ALP1. We have found that these translocation events are inhibited if cells lack Mec1/ATR kinase while Tel1/ATM acts as a suppressor, and that they are dependent on Rad59, a protein known to function as one of two sub-pathways of Rad52 homology-directed repair.

Through a candidate screen of other DNA metabolic factors, we identified Exo1 as a strong suppressor of chromosomal rearrangements in the sgs1Δ mutant. The Exo1 enzymatic domain is located in the N-terminus while the C-terminus harbors mismatch repair protein binding sites as well as phosphorylation sites known to modulate its enzymatic function at uncapped telomeres. We have determined that the C-terminus is dispensable for Exo1’s roles in resistance to DNA-damaging agents and suppressing mutations and chromosomal rearrangements. Exo1 has been identified as a component of the error-free DNA damage tolerance pathway of template switching. Exo1 promotes template switching by extending the single strand gap behind stalled replication forks. Here, we show that the dysregulation of the phosphorylation of the C-terminus of Exo1 is detrimental in cells under replication stress whereas loss of Exo1 suppresses under the same conditions, suggesting that Exo1 function is tightly regulated by both phosphorylation and dephosphorylation and is important in properly modulating the DNA damage response at stalled forks.

It has previously been shown that the strand exchange factor Rad51 binds to the C-terminus of Sgs1 although the significance of this physical interaction has yet to be determined. To elucidate the function of the physical interaction of Sgs1 and Rad51, we have generated a separation of function allele of SGS1 with a single amino acid change (sgs1-FD) that ablates the physical interaction with Rad51. Alone, the loss of the interaction of Sgs1 and Rad51 in our sgs1-FD mutant did not cause any of the defects in response to DNA damaging agents or genome rearrangements that are observed in the sgs1 deletion mutant. However, when we assessed the sgs1-FD mutant in combination with the loss of Sae2, Mre11, Exo1, Srs2, Rrm3, and Pol32 we observed genetic interactions that distinguish the sgs1-FD mutant from the sgs1 deletion mutant. Negative and positive genetic interactions with SAE2, MRE11, EXO1, SRS2, RRM3, and POL32 suggest the role of the physical interaction of Sgs1 and Rad51 is in promoting homology-mediated repair possibly by competing with single-strand binding protein RPA for single-stranded DNA to promote Rad51 filament formation.

Together, these studies characterize additional roles for domains of Sgs1 and Exo1 that are not entirely understood as well as their roles in combination with DNA damage checkpoints, and repair pathways that are necessary for maintaining genome stability.

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Campos-Doerfler, Lillian. "The Role of Sgs1 and Exo1 in the Maintenance of Genome Stability." Scholar Commons, 2017. http://scholarcommons.usf.edu/etd/7006.

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Genome instability is a hallmark of human cancers. Patients with Bloom’s syndrome, a rare chromosome breakage syndrome caused by inactivation of the RecQ helicase BLM, result in phenotypes associated with accelerated aging and develop cancer at a very young age. Patients with Bloom’s syndrome exhibit hyper-recombination, but the role of BLM and increased genomic instability is not fully characterized. Sgs1, the only member of the RecQ family of DNA helicases in Saccharomyces cerevisiae, is known to act both in early and late stages of homology-dependent repair of DNA damage. Exo1, a 5′–3′ exonuclease, first discovered to play a role in mismatch repair has been shown to participate in parallel to Sgs1 in processing the ends of DNA double-strand breaks, an early step of homology-mediated repair. Here we have characterized the genetic interaction of SGS1 and EXO1 with other repair factors in homology-mediated repair as well as DNA damage checkpoints, and characterize the role of post-translational modifications, and protein-protein interactions in regulating their function in response to DNA damage. In S. cerevisiae cells lacking Sgs1, spontaneous translocations arise by homologous recombination in small regions of homology between three non-allelic, but related sequences in the genes CAN1, LYP1, and ALP1. We have found that these translocation events are inhibited if cells lack Mec1/ATR kinase while Tel1/ATM acts as a suppressor, and that they are dependent on Rad59, a protein known to function as one of two sub-pathways of Rad52 homology-directed repair. Through a candidate screen of other DNA metabolic factors, we identified Exo1 as a strong suppressor of chromosomal rearrangements in the sgs1∆ mutant. The Exo1 enzymatic domain is located in the N-terminus while the C-terminus harbors mismatch repair protein binding sites as well as phosphorylation sites known to modulate its enzymatic function at uncapped telomeres. We have determined that the C-terminus is dispensable for Exo1’s roles in resistance to DNA-damaging agents and suppressing mutations and chromosomal rearrangements. Exo1 has been identified as a component of the error-free DNA damage tolerance pathway of template switching. Exo1 promotes template switching by extending the single strand gap behind stalled replication forks. Here, we show that the dysregulation of the phosphorylation of the C-terminus of Exo1 is detrimental in cells under replication stress whereas loss of Exo1 suppresses under the same conditions, suggesting that Exo1 function is tightly regulated by both phosphorylation and dephosphorylation and is important in properly modulating the DNA damage response at stalled forks. It has previously been shown that the strand exchange factor Rad51 binds to the C-terminus of Sgs1 although the significance of this physical interaction has yet to be determined. To elucidate the function of the physical interaction of Sgs1 and Rad51, we have generated a separation of function allele of SGS1 with a single amino acid change (sgs1-FD) that ablates the physical interaction with Rad51. Alone, the loss of the interaction of Sgs1 and Rad51 in our sgs1-FD mutant did not cause any of the defects in response to DNA damaging agents or genome rearrangements that are observed in the sgs1 deletion mutant. However, when we assessed the sgs1-FD mutant in combination with the loss of Sae2, Mre11, Exo1, Srs2, Rrm3, and Pol32 we observed genetic interactions that distinguish the sgs1-FD mutant from the sgs1∆mutant. Negative and positive genetic interactions with SAE2, MRE11, EXO1, SRS2, RRM3, and POL32 suggest the role of the physical interaction of Sgs1 and Rad51 is in promoting homology-mediated repair possibly by competing with single-strand binding protein RPA for single-stranded DNA to promote Rad51 filament formation. Together, these studies characterize additional roles for domains of Sgs1 and Exo1 that are not entirely understood as well as their roles in combination with DNA damage checkpoints, and repair pathways that are necessary for maintaining genome stability.
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Chaix, Alexandre. "Genetic analysis and meiotic role of the Saccharomyces cerevisiae RecQ helicase SGS1." Thesis, University of Leicester, 2007. http://hdl.handle.net/2381/30371.

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SGS1, a Saccharomyces cerevisiae 3 '-5' DNA helicase, is a homologue of the Escherichia coli RecQ gene. It is essential for genomic stability of both during mitosis and meiosis. The purpose of this thesis is to provide a better understanding of the role of this helicase during meiotic recombination. In meiosis, SGS1 mutant cells display a decrease in sporulation efficiency and spore viability. In addition, the unusual spore viability pattern observed in SGS1 mutants cannot be explained solely by meiosis I or meiosis II missegregations. These problems could be partially explained by defects in mitotic chromosome segregation or problems with meiotic S-phase. Cytological experiments demonstrating an increase in synapsis initiation complexes and axial associations in sgs1Delta could be explained by an early function of Sgs1p in meiosis, such as the unwinding of inappropriate strand invasion events. Consistent with this, we observe increased gene conversion, increased homeologous recombination and increased interaction between sister choromatids.;Recent observations have suggested that, Sgs1p and Top3p in S. cerevisiae, and the human orthologue protein BLM, in conjunction with the Top3alpha protein, can dissolve double Holliday junctions. Physical analyses of double-strand break repair in meiosis, combined the genetic analysis of this work, indicate a late function of the Sgs1 protein in the dissolution of double Holliday junctions. We have shown an unusual class of tetrads in which non-sister spores and recombinant spores are dead. We interpret this as a consequence of the failure to untangle intertwined chromatids. This defect in SGS1 mutant strains could be explained by either the presence of pre-meiotic S-phase catenates, a defect in crossover resolution and/or a defect in the dissolution of closely spaced double Holliday junctions.
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Books on the topic "SGSY"

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Swarnjayanti Gram Swarozgar Yojana (India). Swarnjayanti Gram Swarozgar Yojana (SGSY): Guidelines. New Delhi: Ministry of Rural Development, Govt. of India, 2003.

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G, Rajanikanth, and National Institute of Rural Development (India), eds. Impact of SGSY and IAY programmes on minorities: An evaluation study. Hyderabad: National Institute of Rural Development, Ministry of Rural Development, Govt. of India, 2011.

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Sarkar, Soumitra. Microfinance: Concepts, systems, perceptions, and impact : a review of SGSY operations in India. New Delhi: Readworthy Publications, 2011.

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National Institute of Rural Development (India), ed. Institutional credit for rural livelihoods: A study of SGSY in the regions of high poverty. Hyderabad: National Institute of Rural Development, Ministry of Rural Development, Govt. of India, 2009.

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author, Nath Pradip Kumar, and Centre for Planning, Monitoring and Evaluation (India), eds. Impact of IAY and SGSY/NRLM on minorities: An evaluation study under prime minister's new 15-point programme. Hyderabad: Centre for Planning, Monitoring, and Evaluation, National Institute of Rural Development and Panchayati Raj, 2015.

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Establishment, Building Research, ed. Hawksley SGS houses: Technical information. [Watford]: Building Research Establishment, 1986.

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Skovron, Phil. SGS--Society of General Safety: Independent petroleum analysis lab assistant. Bellingham, Wash: Huxley College of Environmental Studies, Western Washington University, 2001.

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Greenstreet, I. Strategic marketing strategy of a smart card technology for SGS-Thompson. Oxford: Oxford Brookes University, 1996.

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Sim, Alex G. Flight characteristics of a modified Schweizer SGS 1-36 sailplane at low and very high angles of attack. Edwards, Calif: Ames Research Center, 1990.

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Han'guk chungso kiŏp ŭi kung, chŭk, t'ong chŏllyak: 9988 kangso kiŏp [SGs] 36-kye = 36 paths to global small giants. Sŏul-si: Kŭllobŏl Kangso Kiŏp Yŏn'guso, 2011.

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Book chapters on the topic "SGSY"

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Schade, Konrad. "Algorithmen für das SGSM." In Stochastische Optimierung, 95–106. Wiesbaden: Vieweg+Teubner Verlag, 2012. http://dx.doi.org/10.1007/978-3-8348-8345-2_6.

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Monnier, Philippe. "Congenital Subglottic Stenosis (C-SGS)." In Pediatric Airway Surgery, 119–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-13535-4_8.

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Fuchs, L. "An Exact SGS-Model for Les." In Advances in Turbulence VI, 23–26. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0297-8_6.

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Olsson, M., and L. Fuchs. "Significant Terms in Dynamic SGS-Modeling." In Direct and Large-Eddy Simulation I, 73–83. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1000-6_7.

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Eberle, Thomas S., and Niklaus Reichle. "Die Schweizerische Gesellschaft für Soziologie (SGS)." In Handbuch Geschichte der deutschsprachigen Soziologie, 1–23. Wiesbaden: Springer Fachmedien Wiesbaden, 2016. http://dx.doi.org/10.1007/978-3-658-07998-7_61-1.

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Amstutz, Max D. "Corporate Governance konkret — SGS als Beispiel." In Herausforderungen an das Management, 115–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55563-3_8.

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Eberle, Thomas S., and Niklaus Reichle. "Die Schweizerische Gesellschaft für Soziologie (SGS)." In Handbuch Geschichte der deutschsprachigen Soziologie, 895–917. Wiesbaden: Springer Fachmedien Wiesbaden, 2017. http://dx.doi.org/10.1007/978-3-658-07614-6_61.

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Garnier, E., N. Adams, and P. Sagaut. "Relation Between SGS Model and Numerical Discretization." In Scientific Computation, 119–53. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2819-8_6.

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Jang, Min, Choong Kun Lee, and Hyoung Taek Kim. "Development of a Commercial SGSN System for IMT2000 UMTS." In Mobile Communications, 433–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/3-540-36555-9_45.

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Singh, Lal, and Ram Bahadur Patel. "User Authentication in VANET Using SGSK (Self Generated Session Key)." In Advances in Intelligent Systems and Computing, 196–203. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39875-0_21.

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Conference papers on the topic "SGSY"

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Lee, Jong, Yong Liu, and Liang Yu. "SGST." In the 2nd ACM SIGSPATIAL International Workshop. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/2064959.2064964.

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Wang, Nian, Zhe Zhang, Tingting Li, Jing Xiao, and Li Cui. "SGSF." In IPSN '19: The 18th International Conference on Information Processing in Sensor Networks. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3302506.3310392.

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"SGSC 2011 Organizing Committee." In 2011 IEEE 9th International Symposium on Parallel and Distributed Processing with Applications Workshops (ISPAW). IEEE, 2011. http://dx.doi.org/10.1109/ispaw.2011.91.

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"Message from the SGSC 2011 Organizers." In 2011 IEEE 9th International Symposium on Parallel and Distributed Processing with Applications Workshops (ISPAW). IEEE, 2011. http://dx.doi.org/10.1109/ispaw.2011.83.

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Leibee, Jack. "SGSS: Modernizing NASA's TDRS Ground System." In SpaceOps 2012. Reston, Virigina: American Institute of Aeronautics and Astronautics, 2012. http://dx.doi.org/10.2514/6.2012-1240286.

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Canto, Leonardo Dos Santos. "ANP Resolution 43: Operational Safety Management System - SGSO." In SPE International Conference on Health, Safety and Environment in Oil and Gas Exploration and Production. Society of Petroleum Engineers, 2010. http://dx.doi.org/10.2118/126895-ms.

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Kim, Dayeon, and Munchurl Kim. "SGSR: A Saliency-Guided Image Super-Resolution Network." In 2023 IEEE International Conference on Image Processing (ICIP). IEEE, 2023. http://dx.doi.org/10.1109/icip49359.2023.10222146.

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Pagni, Andrea, Rinaldo Poluzzi, and GianGuido Rizzotto. "Fuzzy logic program at SGS-Thomson." In Optical Tools for Manufacturing and Advanced Automation, edited by Bruno Bosacchi and James C. Bezdek. SPIE, 1993. http://dx.doi.org/10.1117/12.165016.

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Schüssler, Fabian. "Science with the Southern Gamma-ray Survey Observatory (SGSO)." In 36th International Cosmic Ray Conference. Trieste, Italy: Sissa Medialab, 2019. http://dx.doi.org/10.22323/1.358.0786.

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Macias, Mauricio, Cristian Barria, Alejandra Acuna, and Claudio Cubillos. "SGSI support throught malware's classification using a pattern analysis." In 2016 IEEE International Conference on Automatica (ICA-ACCA). IEEE, 2016. http://dx.doi.org/10.1109/ica-acca.2016.7778516.

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Reports on the topic "SGSY"

1

Bell, Thomas E. Project Boeing SGS (Final Report). Office of Scientific and Technical Information (OSTI), December 2014. http://dx.doi.org/10.2172/1177423.

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2

Gafni, Yedidya, and Vitaly Citovsky. Inactivation of SGS3 as Molecular Basis for RNA Silencing Suppression by TYLCV V2. United States Department of Agriculture, November 2013. http://dx.doi.org/10.32747/2013.7593402.bard.

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The Israeli isolate of Tomato yellow leaf curl geminivirus(TYLCV-Is) is a major tomato pathogen, causing extensive crop losses in Israel and in the south-eastern U.S. Yet, little is known about the molecular mechanisms of its interaction with tomato cells. One of the most interesting aspects of such interaction is how the invading virus counteracts the RNA silencing response of the plant. In the former BARD project, we have shown that TYLCV-Is V2 protein is an RNA silencing suppressor, and that this suppression is carried out via the interaction of V2 with the SGS3 component of the plant RNA silencing machinery. This reported project was meant to use our data as a foundation to elucidate the molecular mechanism by which V2 affects the SGS3 activity. While this research is likely to have an important impact on our understanding of basic biology of virus-plant interactions and suppression of plant immunity, it also will have practical implications, helping to conceive novel strategies for crop resistance to TYLCV-Is. Our preliminary data in regard to V2 activities and our present knowledge of the SGS3 function suggest likely mechanisms for the inhibitory effect of V2 on SGS3. We have shown that V2 possess structural and functional hallmarks of an F-box protein, suggesting that it may target SGS3 for proteasomal degradation. SGS3 contains an RNA-binding domain and likely functions to protect the cleavage produces of the primary transcript for subsequent conversion to double-stranded forms; thus, V2 may simply block the RNA binding activity of SGS3. V2 may also employ a combination of these mechanisms. These and other possibilities were tested in this reported project.
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3

Gafni, Yedidya, Moshe Lapidot, and Vitaly Citovsky. Dual role of the TYLCV protein V2 in suppressing the host plant defense. United States Department of Agriculture, January 2013. http://dx.doi.org/10.32747/2013.7597935.bard.

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TYLCV-Is is a major tomato pathogen, causing extensive crop losses in Israel and the U.S. We have identified a TYLCV-Is protein, V2, which acts as a suppressor of RNA silencing. Intriguingly, the counter-defense function of V2 may not be limited to silencing suppression. Our recent data suggest that V2 interacts with the tomato CYP1 protease. CYP1 belongs to the family of papain-like cysteine proteases which participate in programmed cell death (PCD) involved in plant defense against pathogens. Based on these data we proposed a model for dual action of V2 in suppressing the host antiviral defense: V2 targets SGS3 for degradation and V2 inhibits CYP1 activity. To study this we proposed to tackle three specific objectives. I. Characterize the role of V2 in SGS3 proteasomal degradation ubiquitination, II. Study the effects of V2 on CYP1 maturation, enzymatic activity, and accumulation and, III. Analyze the effects of the CYP1-V2 interaction on TYLCV-Is infection. Here we describe results from our study that support our hypothesis: the involvement of the host's innate immune system—in this case, PCD—in plant defense against TYLCV-Is. Also, we use TYLCV-Is to discover the molecular pathway(s) by which this plant virus counters this defense. Towards the end of our study we discovered an interesting involvement of the C2 protein encoded by TYLCV-Is in inducing Hypersensitive Response in N. benthamianaplants which is not the case when the whole viral genome is introduced. This might lead to a better understanding of the multiple processes involved in the way TYLCV is overcoming the defense mechanisms of the host plant cell. In a parallel research supporting the main goal described, we also investigated Agrobacteriumtumefaciens-encoded F-box protein VirF. It has been proposed that VirF targets a host protein for the UPS-mediated degradation, very much the way TYLCV V2 does. In our study, we identified one such interactor, an Arabidopsistrihelix-domain transcription factor VFP3, and further show that its very close homolog VFP5 also interacted with VirF. Interestingly, interactions of VirF with either VFP3 or VFP5 did not activate the host UPS, suggesting that VirF might play other UPS-independent roles in bacterial infection. Another target for VirF is VFP4, a transcription factor that both VirF and its plant functional homolog VBF target to degradation by UPS. Using RNA-seqtranscriptome analysis we showed that VFP4 regulates numerous plant genes involved in disease response, including responses to viral and bacterial infections. Detailed analyses of some of these genes indicated their involvement in plant protection against Agrobacterium infection. Thus, Agrobacterium may facilitate its infection by utilizing the host cell UPS to destabilize transcriptional regulators of the host disease response machinery that limits the infection.
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4

Citovsky, Vitaly, and Yedidya Gafni. Suppression of RNA Silencing by TYLCV During Viral Infection. United States Department of Agriculture, December 2009. http://dx.doi.org/10.32747/2009.7592126.bard.

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The Israeli isolate of Tomato yellow leaf curl geminivirus (TYLCV-Is) is a major tomato pathogen, causing extensive (up to 100%) crop losses in Israel and in the south-eastern U.S. (e.g., Georgia, Florida). Surprisingly, however, little is known about the molecular mechanisms of TYLCV-Is interactions with tomato cells. In the current BARD project, we have identified a TYLCV-Is protein, V2, which acts as a suppressor of RNA silencing, and showed that V2 interacts with the tomato (L. esculentum) member of the SGS3 (LeSGS3) protein family known to be involved in RNA silencing. This proposal will use our data as a foundation to study one of the most intriguing, yet poorly understood, aspects of TYLCV-Is interactions with its host plants – possible involvement of the host innate immune system, i.e., RNA silencing, in plant defense against TYLCV-Is and the molecular pathway(s) by which TYLCV-Is may counter this defense. Our project sought two objectives: I. Study of the roles of RNA silencing and its suppression by V2 in TYLCV-Is infection of tomato plants. II. Study of the mechanism by which V2 suppresses RNA silencing. Our research towards these goals has produced the following main achievements: • Identification and characterization of TYLCV V2 protein as a suppressor of RNA silencing. (#1 in the list of publications). • Characterization of the V2 protein as a cytoplasmic protein interacting with the plant protein SlSGS3 and localized mainly in specific, not yet identified, bodies. (#2 in the list of publications). • Development of new tools to study subcellular localization of interacting proteins (#3 in the list of publications). • Characterization of TYLCV V2 as a F-BOX protein and its possible role in target protein(s) degradation. • Characterization of TYLCV V2 interaction with a tomato cystein protease that acts as an anti-viral agent. These research findings provided significant insights into (I) the suppression of RNA silencing executed by the TYLCV V2 protein and (II) characterization some parts of the mechanism(s) involved in this suppression. The obtained knowledge will help to develop specific strategies to attenuate TYLCV infection, for example, by blocking the activity of the viral suppressor of gene silencing thus enabling the host cell silencing machinery combat the virus.
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