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Relevant bibliographies by topics / Sertoli cell

Academic literature on the topic 'Sertoli cell'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 March 2023

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Journal articles on the topic "Sertoli cell"

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Iliadou, Paschalia, Christos Tsametis, Athina Kaprara, Ioannis Papadimas, and Dimitrios Goulis. "The Sertoli cell: Novel clinical potentiality." HORMONES 14, no. 4 (October 15, 2015): 504–14. http://dx.doi.org/10.14310/horm.2002.1648.

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de, Kretser DM. "Germ cell-Sertoli cell interactions." Reproduction, Fertility and Development 2, no. 3 (1990): 225. http://dx.doi.org/10.1071/rd9900225.

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The interactions between the Sertoli cells and germ cells are progressively becoming an important part of testicular physiology. This paper explores the cytological basis for these interactions, detailing the cyclic changes in the Sertoli cells in concert with the stages of the seminiferous cycle and the nature of the blood-testis barrier. These cytological changes are correlated with a number of variations in the function of Sertoli cells. The mechanisms by which germ cells and Sertoli cells interact are explored and can be divided into those using cell-to-cell contact and others utilizing paracrine factors.

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NISTAL, M., F. JIMENEZ, and R. PANIAGUA. "Sertoli cell types in the Sertoli-cell-only syndrome: relationships between Sertoli cell morphology and aetiology." Histopathology 16, no. 2 (April 3, 2007): 173–80. http://dx.doi.org/10.1111/j.1365-2559.1990.tb01086.x.

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Scialli, Anthony R. "The sertoli cell." Reproductive Toxicology 9, no. 2 (March 1995): 211–13. http://dx.doi.org/10.1016/s0890-6238(99)80005-7.

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Tsai, Robert Y. L. "SERTOLI CELL BIOLOGY." In Vitro Cellular & Developmental Biology - Animal 41, no. 5 (2005): 177. http://dx.doi.org/10.1290/br030501.1.

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Hadley, M. A., S. W. Byers, C. A. Suárez-Quian, H. K. Kleinman, and M. Dym. "Extracellular matrix regulates Sertoli cell differentiation, testicular cord formation, and germ cell development in vitro." Journal of Cell Biology 101, no. 4 (October 1, 1985): 1511–22. http://dx.doi.org/10.1083/jcb.101.4.1511.

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Sertoli cell preparations isolated from 10-day-old rats were cultured on three different substrates: plastic, a matrix deposited by co-culture of Sertoli and peritubular myoid cells, and a reconstituted basement membrane gel from the EHS tumor. When grown on plastic, Sertoli cells formed a squamous monolayer that did not retain contaminating germ cells. Grown on the matrix deposited by Sertoli-myoid cell co-cultures, Sertoli cells were more cuboidal and supported some germ cells but did not allow them to differentiate. After 3 wk however, the Sertoli cells flattened to resemble those grown on plastic. In contrast, the Sertoli cells grown on top of the reconstituted basement membrane formed polarized monolayers virtually identical to Sertoli cells in vivo. They were columnar with an elaborate cytoskeleton. In addition, they had characteristic basally located tight junctions and maintained germ cells for at least 5 wk in the basal aspect of the monolayer. However, germ cells did not differentiate. Total protein, androgen binding protein, transferrin, and type I collagen secretion were markedly greater when Sertoli cells were grown on the extracellular matrices than when they were grown on plastic. When Sertoli cells were cultured within rather than on top of reconstituted basement membrane gels they reorganized into cords. After one week, tight junctional complexes formed between adjacent Sertoli cells, functionally compartmentalizing the cords into central (adluminal) and peripheral (basal) compartments. Germ cells within the cords continued to differentiate. Thus, Sertoli cells cultured on top of extracellular matrix components assume a phenotype and morphology more characteristic of the in vivo, differentiated cells. Growing Sertoli cells within reconstituted basement membrane gels induces a morphogenesis of the cells into cords, which closely resemble the organ from which the cells were dissociated and which provide an environment permissive for germ cell differentiation.

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GROOTEGOED, J. ANTON, PIET J. DEN BOER, and PETRA MACKENBACH. "Sertoli Cell-Germ Cell Communication." Annals of the New York Academy of Sciences 564, no. 1 Regulation of (July 1989): 232–42. http://dx.doi.org/10.1111/j.1749-6632.1989.tb25900.x.

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Benahmed, M., J. Reventos, E. Tabone, and J. M. Saez. "Cultured Sertoli cell-mediated FSH stimulatory effect on Leydig cell steroidogenesis." American Journal of Physiology-Endocrinology and Metabolism 248, no. 2 (February 1, 1985): E176—E181. http://dx.doi.org/10.1152/ajpendo.1985.248.2.e176.

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To determine the precise role of Sertoli cells in the stimulating effects of follicle stimulating hormone (FSH) on Leydig cell activity, porcine purified Leydig and Sertoli cells were cultured separately or together in a chemically defined medium in the absence or presence of porcine, FSH 50 ng/ml. Leydig cell activity was evaluated using two parameters: human chorionic gonadotropin (hCG) binding sites; and hCG-stimulated cAMP production and testosterone secretion. First, it was found that FSH increases Leydig cell activity in crude Leydig cell preparations (40–60% of Leydig cells), whereas it exerts no effect on purified Leydig cells (greater than 90% of Leydig cells). Second, FSH stimulates the activity of Leydig cells cocultured with Sertoli cells, whereas it remains without effect on purified Leydig cells cultured alone. This stimulating effect of FSH on Leydig cell activity is dependent on the Sertoli cell number in the coculture. These data 1) show that the stimulating effect of FSH on Leydig cell function is mediated by Sertoli cells and 2) support the concept of local control of Leydig cell function originating from Sertoli cells.

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Halder, Ashutosh, and Manish Jain. "Sertoli cell only syndrome: Status of sertoli cell maturation and function." Indian Journal of Endocrinology and Metabolism 16, no. 8 (2012): 512. http://dx.doi.org/10.4103/2230-8210.104154.

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Fatima, Arooj, Sajid Mushtaq, and Asif Loya. "Expression of NKX 3.1 in Sertoli Cell Tumors." Pakistan Armed Forces Medical Journal 72, no. 6 (December 29, 2022): 1965–68. http://dx.doi.org/10.51253/pafmj.v72i6.6430.

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Objective: To evaluate the NKX3.1 expression by immunohistochemistry in normal testicular parenchyma and in Sertoli cell tumours and Sertoli Leydig cell tumours of the testes and ovary. Study Design: Retrospective longitudinal study. Place and Duration of Study: Shaukat Khanum Memorial Cancer Hospital and Research Centre, Lahore Pakistan, from 2010-2021. Methodology: We used immunohistochemistry to evaluate the positivity and loss of nuclear expression of NKX3.1 in the Sertoli cell tumour (11 cases), Sertoli Leydig cell tumour (31 cases) and in normal testicular parenchyma (7 cases). Results: In our study, there were 49 cases. All the cases of benign testicular parenchyma expressed positivity with nuclear staining of NKX 3.1 in Sertoli cells. Two out of 11 Sertoli cell tumours expressed positivity with nuclear positivity of NKX 3.1 in Sertoli cell component (18.18%) and 9 of the cases showed loss of staining of NKX 3.1 (81.8%). All Sertoli Leydig cell tumours showed loss of staining of NKX 3.1. Conclusion: Nuclear expression of NKX 3.1 is seen in Sertoli cells of normal testicular parenchyma. This staining is lost in Sertoli cell tumours and Sertoli Leydig cell tumours.

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Dissertations / Theses on the topic "Sertoli cell"

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Wang, Qiufan Claire, and 王秋帆. "Mechanisms of junctional restructuring at the sertoli-sertoli and sertoli-germ cell interfaces during spermatogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40887686.

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Wang, Qiufan Claire. "Mechanisms of junctional restructuring at the sertoli-sertoli and sertoli-germ cell interfaces during spermatogenesis." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40887686.

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Maguire, Sharon Marie. "Germ cell modulation of Sertoli cell function." Thesis, University of Edinburgh, 1994. http://hdl.handle.net/1842/20662.

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The aim of this work was to assess the influence of germ cells on the expression of selected Sertoli cell mRNAs. To this end, adult rats were treated with 650mg/kg methoxyacetic acid (MAA) to induce the specific depletion of >80% of pachytene and later spermatocytes from most tubules, and expression of selected Sertoli cell mRNAs was then assessed at various times after treatment when particular germ cell types were depleted selectively (see Bartlett et al. 1988; Allenby et al., 1991). Studies on the expression of cyclic protein 2(CP-2) mRNA supported the hypothesis that germ cells can influence the cyclic function of Sertoli cells. Expression of CP-2 mRNA was shown by Northern blot analysis to decrease significantly 21 days after MAA treatment. In situ hybridisation showed that CP-2 mRNA expression was decreased or absent from tubules at stages at which CP-2 mRNA is normally expressed (stages IV-VII) when elongate spermatids were depleted selectively from these tubules. This decrease was reflected in loss of CP-2 protein production. These observations lead us to hypothesise that elongate spermatids positively modulate CP-2 expression in the Sertoli cell, with this modulation occurring at the level of transcription. In conclusion, this study demonstrated that germ cells do appear to influence Sertoli cell gene expression. This can occur at the level of transcription as was demonstrated by the effect of elongate spermatids on CP-2 mRNA expression, or may occur post-transcriptionally, as would appear to be the case with ABP.

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Petersen, Cecilia. "Paracrine regulation of Sertoli cell proliferation /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-443-7/.

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Samy, Eileen Teresa. "Regulation of testin and prostaglandin D2 synthetase expression in sertoli cells: a molecular and cell biologystudy and its implication in sertoli-germ cell interactions." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B3122331X.

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Pearce, Kristen (Kristen Joanne) 1974. "Regulation of adhesion between round spermatids and Sertoli cells in the testis." Monash University, Dept. of Obstetrics and Gynaecology, 2003. http://arrow.monash.edu.au/hdl/1959.1/6606.

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Wong, Ching-hang. "Cell-cell interactions and cell junction dynamics in the mammalian testis." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31993084.

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McCabe, Mark James, and markmccabe02@hotmail com. "Hormonal regulation of the testicular Sertoli cell tight junction." RMIT University. Applied Sciences, 2008. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20081212.100348.

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The Sertoli cell tight junction (TJ) of the seminiferous epithelium is important for the developmental process of spermatogenesis as it separates germ cells in the seminiferous tubules from the general circulation in the testicular interstitium. Absence of the TJ leads to spermatogenic arrest and infertility. TJs form at puberty as circulating gonadotrophins luteinising hormone/testosterone and follicle stimulating hormone increase. Several studies have demonstrated hormonal regulation of the two major TJ proteins, claudin-11 and occludin, and also of TJ function in vitro and in vivo. Men with low levels of circulating gonadotrophins exhibit an immature and dysfunctional TJ phenotype, which is reversed upon the exogenous application of gonadotrophins. This thesis hypothesises that claudin-11 and occludin are the major contributors to TJ function, and that gonadotrophins regulate TJ function and structure via these two proteins in several species including humans. This PhD was divided into four separate studies to address these hypotheses. The first study selectively silenced the genetic expression of claudin-11 and occludin with small interfering RNA (siRNA) in cultured immature rat Sertoli cells to determine their contribution to Sertoli cell TJ function in vitro. siRNA treatment against either protein significantly (p less than 0.01) reduced TJ function by ~50% as assessed by transepithelial electrical resistance. Immunocytochemistry displayed marked reductions in the localisation of these proteins to the TJ after siRNA treatment. It was concluded that both proteins significantly contributed to TJ function in vitro. The second and third studies then aimed to study hormonal regulation of the TJ in vivo. Weekly injections of the gonadotrophin releasing hormone antagonist acyline were used to suppress circulating gonadotrophins and spermatogenesis in adult rats. Acyline treatment disrupted i) the localisation of occludin to the TJ and ii) TJ function as shown by permeability to a biotin tracer, which was impermeable to TJs in controls. Short-term hormone replacement partially restored the effects of gonadotrophin suppression. It was concluded that gonadotrophins regulate the maintenance of the TJ in rats in vivo. The third study used the hypogonadal (hpg) mouse, which is a naturally occurring model of gonadotrophin deficiency with inactive spermatogenesis. Claudin-11 in hpg mice was not localised at the TJs, and these were dysfunctional as shown by permeability to biotin. Following hormone treatment, TJs were structurally and functionally competent, demonstrating that gonadotrophins also regulate the formation of TJs in vivo. The fourth study subsequently analysed TJs in gonadotrophin suppressed men, and it was found that claudin-11 staining was reduced from continuous bands in control men, to punctate staining in gonadotrophin-suppressed men, demonstrating that gonadotrophins also regulate the localisation of claudin-11 to the TJ in men in vivo. In summary, it is concluded that the Sertoli cell TJ is hormonally regulated, and that the major contributors to TJ function in vivo and in vitro are claudin-11 and occludin. It is hypothesised that the reduction of claudin-11 localisation to the TJ in men may also result in a loss of human Sertoli cell TJ function, suggesting that the TJ may be a potential target of hormonal contraception in men.

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Gregory, Christopher Wayne. "Biochemical characterization of two hormonally-regulated Sertoli cell proteins /." The Ohio State University, 1993. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487847761308818.

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Wong, Ching-hang, and 黃政珩. "Cell-cell interactions and cell junction dynamics in the mammalian testis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31993084.

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Books on the topic "Sertoli cell"

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Dee, Russell Lonnie, and Griswold Michael D, eds. The Sertoli cell. Clearwater, FL: Cache River Press, 1993.

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D, Griswold Michael, ed. The Sertoli cell. Clearwater, FL: Cache River Press, 1993.

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K, Skinner Michael, and Griswold Michael D, eds. Sertoli cell biology. Amsterdam: Elsevier Academic Press, 2005.

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Oliveira, Pedro F., and Marco G. Alves. Sertoli Cell Metabolism and Spermatogenesis. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-19791-3.

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Beck, F. F., B. Christ, F. Clascá, D. E. Haines, H. W. Korf, W. Kummer, E. Marani, et al., eds. Regulation of Sertoli Cell and Germ Cell Differentation. Berlin/Heidelberg: Springer-Verlag, 2005. http://dx.doi.org/10.1007/3-540-29446-5.

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Alves, Marco G., and Pedro F. Oliveira, eds. Sertoli Cells. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7698-0.

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Sertoli Cell Biology. Elsevier, 2005. http://dx.doi.org/10.1016/b978-0-12-647751-1.x5000-4.

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Sertoli Cell Biology. Elsevier, 2015. http://dx.doi.org/10.1016/c2013-0-01369-2.

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Griswold, Michael D. Sertoli Cell Biology. Elsevier Science & Technology Books, 2014.

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Griswold, Michael D. Sertoli Cell Biology. Elsevier Science & Technology Books, 2014.

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Book chapters on the topic "Sertoli cell"

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Colecchia, Maurizio, and Alessia Bertolotti. "Sertoli Cell Tumor." In Encyclopedia of Pathology, 1–3. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-28845-1_3755-1.

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Colecchia, Maurizio, and Alessia Bertolotti. "Sertoli Cell Tumor." In Encyclopedia of Pathology, 378–80. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-41894-6_3755.

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Oliveira, Pedro F., and Marco G. Alves. "Sertoli Cell and Germ Cell Differentiation." In Sertoli Cell Metabolism and Spermatogenesis, 25–39. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-19791-3_4.

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Vogl, A. Wayne, Kuljeet S. Vaid, and Julian A. Guttman. "The Sertoli Cell Cytoskeleton." In Advances in Experimental Medicine and Biology, 186–211. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-09597-4_11.

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Colecchia, Maurizio, and Alessia Bertolotti. "Sertoli Cell Tumor, Endocrine." In Endocrine Pathology, 731–33. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-62345-6_5340.

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Colecchia, Maurizio, and Alessia Bertolotti. "Sertoli Cell Tumor, Endocrine." In Encyclopedia of Pathology, 1–3. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-28845-1_5340-1.

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, Alexander K. C. Leung, Thomas Kolter, Ute Schepers, Konrad Sandhoff, et al. "Sertoli Cell Only Syndrome." In Encyclopedia of Molecular Mechanisms of Disease, 1921–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1611.

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Oliveira, Pedro F., and Marco G. Alves. "Modulation of Sertoli Cell Metabolism." In Sertoli Cell Metabolism and Spermatogenesis, 57–71. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-19791-3_6.

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Jarak, Ivana, Pedro F. Oliveira, Gustavo Rindone, Rui A. Carvalho, María N. Galardo, María F. Riera, Silvina B. Meroni, and Marco G. Alves. "Assessing Sertoli Cell Metabolic Activity." In Methods in Molecular Biology, 157–71. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7698-0_12.

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Gupta, G. S. "Paracrine Role of Sertoli Cell." In Proteomics of Spermatogenesis, 21–46. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/0-387-27655-6_2.

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Conference papers on the topic "Sertoli cell"

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Jethwani, Umesh, and Divya Jethwani. "Sertoli cell tumor of ovary: A rare case report." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685324.

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Introduction: Sertoli-Leydig cell tumor (SLCT) is a rare ovarian tumor, Constitute less than 0.5% of ovarian tumors. Most tumors are unilateral, confined to the ovaries. They are seen during the second and third decades of life. They are characterized by the presence of testicular structures that produce androgens. Patients have symptoms of virilization (depending on the quantity of androgen). Case Report: A 42-year-old woman presented Amenorrhea for 14 months. Change in her voice for 1 year and Excessive hair growth on her face, chest, and limbs for the last 2 months. She complained of vague abdominal discomfort. No history of anorexia, weight loss, increased libido. Her medical and family history was unremarkable. On examination - Hirsutism and clitoromegaly. Lump of size 10x8 cm palpable in left iliac fossa. Vaginal examination revealed a firm and mobile cystic mass in the right adnexa. An ultrasound examination of the pelvis showed a 17x 13x 9-cm heterogeneous solid cystic mass replacing the left ovary. The right ovary and the uterus were normal. CECT Scan Abdomen-Large heterogenous encapsulated solid soft tissue mass lesions containing areas of calcification arising from left ovary of size 17x13x10.6cm causing displacement of urinary bladder and surrounding bowel loops. Serum testosterone level -2 ng/mL (normal, 0.2–1.2 ng/mL); (DHEAS), CA 125, and alpha fetoprotein (AFP) -normal. On Laparotmy-Large mass of size 17 X 13 cm arising from left adnexa. Uterus and right ovary grossly normal. Total Abdominal hysterectomy, B/L Salpingo-opherectomy and infracolic omentectomy was done. Peritoneal washing were sent for cytologic examination for malignant cells. No liver metastasis. The post operative period was uneventful. Histopathology revealed- confirmed it be Sertoli Leydig cell tumor. 3month follow up – resolution of her virilization symptoms. No increase of her hirsutism. Repeat testosterone levels - within normal range. Conclusion: Only few cases of SLCT have been reported till date Prognosis depends on extent of disease, stage of disease, tumour differentiation, grade. The treatment should be individualized according to the location, state of spread and the patient’s condition.

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Stanton, C. A., C. F. Risquez Cordovez, and S. A. Woods. "Sertoli-Leydig Cell Tumor Presenting as Pseudo-Meigs' Syndrome." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3178.

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Karacaoğlu, Elif. "T-2 Toxin Disrupts Sertoli Cell Barrier via Changes in Tight and Adherent Junctional Proteins in SerW3 Cells." In 15th International Congress of Histochemistry and Cytochemistry. Istanbul: LookUs Scientific, 2017. http://dx.doi.org/10.5505/2017ichc.op-45.

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Arda, Oktay. "Light and electron microscopic examination of endoplasmic reticulum stress-induced changes on Sertoli cell cytoplasm." In European Microscopy Congress 2020. Royal Microscopical Society, 2021. http://dx.doi.org/10.22443/rms.emc2020.991.

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Resende, G., B. Ruiz, V. Sanz, YL Viana, and E. Azevedo. "364 A case of sertoli-leydig cell tumor of ovary in young woman: a rare disease." In IGCS Annual 2019 Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-igcs.364.

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Karnezis, Anthony N., Yemin Wang, Jamie Magrill, Jacqueline Keul, Stefan Kommoss, Basile Tessier-Cloutier, Lily Proctor, et al. "Abstract B46: DICER1 and FOXL2 mutations correlate with clinicopathologic features of ovarian Sertoli-Leydig cell tumors." In Abstracts: AACR Special Conference: Addressing Critical Questions in Ovarian Cancer Research and Treatment; October 1-4, 2017; Pittsburgh, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.ovca17-b46.

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Wang, Yemin, Jiamin Chen, Winnie Yang, Janine Senz, Michael S. Anglesio, Blake Gilks, Gregg B. Morin, and David G. Huntsman. "Abstract POSTER-BIOL-1349: The oncogenic role of DICER1 RNase IIIb domain mutations in ovarian sertoli-leydig cell tumors." In Abstracts: 10th Biennial Ovarian Cancer Research Symposium; September 8-9, 2014; Seattle, WA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.ovcasymp14-poster-biol-1349.

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Lang, Lang, Ting Zhang, Yu-Bin Ji, and Miao Yu. "Influence of Butyl Benzyl Phthalate and Its Metabolites on Sertoli Cells of Testicle." In 2015 International Conference on Medicine and Biopharmaceutical. WORLD SCIENTIFIC, 2016. http://dx.doi.org/10.1142/9789814719810_0118.

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Korolev, Y. N., L. A. Nikulina, and L. V. Mihaylik. "Ultrastructural analysis of Sertoli cells under the action of low-intensity electromagnetic radiation radiation (experimental study)." In Arbat readings. Знание-М, 2020. http://dx.doi.org/10.38006/907345-01-0.2020.40.44.

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Yuniarifa, Conita, Joko Wahyu Wibowo, and Taufiqurrachman Nasihun. "The effect of vitamin C, vitamin E, glutation and zink combination on the number of sertoli and leydig cells: Pre-clinical test in male wistar rats (Rattus norvegicus) exposed to cigarette smoke." In INTERNATIONAL CONFERENCE ON BIOINFORMATICS AND NANO-MEDICINE FROM NATURAL RESOURCES FOR BIOMEDICAL RESEARCH: 3rd Annual Scientific Meeting for Biomedical Sciences. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5109980.

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