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Journal articles on the topic 'Serratia marcescens'

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Published: 4 June 2021
Last updated: 18 May 2024

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1

HEJAZI, A., and F. R. FALKINER. "Serratia marcescens." Journal of Medical Microbiology 46, no. 11 (November 1, 1997): 903–12. http://dx.doi.org/10.1099/00222615-46-11-903.

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2

Yannelli, Barbara, Paul E. Schoch, and Burke A. Cunha. "Serratia marcescens." Clinical Microbiology Newsletter 9, no. 20 (October 1987): 157–60. http://dx.doi.org/10.1016/0196-4399(87)90078-x.

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3

Ikpa, Amadi, and Ogbonda KH. "Enhancing Contact Tracing for Serratia marcescens Biofilm on High-Usage Body Towels in Rivers State Bathrooms." Journal of Healthcare and Biomedical Science 1, no. 2 (August 7, 2023): 1–10. http://dx.doi.org/10.31098/jhbs.v1i2.1384.

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The placement of body towels before and after use in bathrooms where Serratia marcescens proliferates calls for concern, as Serratia marcescens, an airborne opportunistic pathogen has been reported keratinolytic. Serratia marcescens, a bacterium commonly noticed with a pink or red slimy appearance in toilet sinks, bowls, and tiles presents an aesthetically, unappealing and disgusting appearance in the toilet surroundings. The study aimed to trace the presence of Serratia marcescens on frequently used body towels hung in the bathroom doors. Swabs from forty (40) differently used towels were collected from twenty (20) volunteered homes and analyzed using standard microbiological procedures. Microbiological procedures involved inoculating the swab sample on a prepared peptone broth and plating on MacConkey agar media, followed by identification and streak of the recovered isolate onto Congo red agar media for biofilm formation. Results showed the recovery of Serratia marcescens isolates. The three homes showed a Serratia marcescens count of 3 X 10, 1 X 102, and 7 X 10 CFU per swab for house units C, I, and P respectively. Serratia marcescens could form a biofilm, a basic feature that allowed it to strive on a body towel. The results derived strongly identified the presence of Serratia marcescens biofilm on body towels hung in the bathrooms. This could have health implications for towel users due to the bacteria's keratolytic properties. Hence, the need for constant surveillance to support effective measures of hygiene, aimed at preventing the spread of Serratia marcescens is recommended.
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4

CN, Amadi-Ikpa, and Ogbonda KH. "Enhancing Contact Tracing for Serratia marcescens Biofilm on High-Usage Body Towels in Rivers State Bathrooms." Journal of Healthcare and Biomedical Science 1, no. 2 (June 29, 2023): 1–10. http://dx.doi.org/10.31098/jhbs.v2i1.1608.

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The placement of body towels before and after use in bathrooms where Serratia marcescens proliferates calls for concern, as Serratia marcescens, an airborne opportunistic pathogen has been reported keratinolytic. Serratia marcescens, a bacterium commonly noticed with a pink or red slimy appearance in toilet sinks, bowls, and tiles presents an aesthetically, unappealing and disgusting appearance in the toilet surroundings. The study aimed to trace the presence of Serratia marcescens on frequently used body towels hung in the bathroom doors. Swabs from forty (40) differently used towels were collected from twenty (20) volunteered homes and analyzed using standard microbiological procedures. Microbiological procedures involved inoculating the swab sample on a prepared peptone broth and plating on MacConkey agar media, followed by identification and streak of the recovered isolate onto Congo red agar media for biofilm formation. Results showed the recovery of Serratia marcescens isolates. The three homes showed a Serratia marcescens count of 3 X 10, 1 X 102, and 7 X 10 CFU per swab for house units C, I, and P respectively. Serratia marcescens could form a biofilm, a basic feature that allowed it to strive on a body towel. The results derived strongly identified the presence of Serratia marcescens biofilm on body towels hung in the bathrooms. This could have health implications for towel users due to the bacteria's keratolytic properties. Hence, the need for constant surveillance to support effective measures of hygiene, aimed at preventing the spread of Serratia marcescens is recommended.
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5

Nazzaro, Gianluca. "Etymologia: Serratia marcescens." Emerging Infectious Diseases 25, no. 11 (November 2019): 2012. http://dx.doi.org/10.3201/eid2511.et2511.

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6

Theccanat, G., L. Hirschfield, and H. Isenberg. "Serratia marcescens meningitis." Journal of Clinical Microbiology 29, no. 4 (1991): 822–23. http://dx.doi.org/10.1128/jcm.29.4.822-823.1991.

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7

Yanagi, T., K. Katagiri, T. Sonada, and S. Takayasu. "Serratia marcescens granuloma." British Journal of Dermatology 136, no. 2 (February 1997): 289–90. http://dx.doi.org/10.1111/j.1365-2133.1997.tb14919.x.

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8

REISSELL, PENTTI, and ELLI JANSSON. "Serratia Marcescens Septicemia." Acta Medica Scandinavica 176, no. 2 (April 24, 2009): 253–56. http://dx.doi.org/10.1111/j.0954-6820.1964.tb00932.x.

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9

Acar, Jacques F. "Serratia marcescens Infections." Infection Control 7, no. 5 (May 1986): 273–80. http://dx.doi.org/10.1017/s0195941700064201.

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The recognition of serratia as an opportunistic human pathogen can be dated from 1959, when the microorganism entered the family of Enterobacteriaceae, with features recognizable in the clinical laboratory and related to the Klebsiella/Enterobacter group. Since then, physicians have been challenged to establish the significance of isolation of serratia from a clinical specimen.
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10

Mahrooqi, Muna AL. "Epidemiology of Serratia Marcescens in the Neonatal ICU of A Tertiary Hospital in Oman over a 10 Years Period." Journal of Infectious Diseases & Travel Medicine 6, no. 2 (2022): 1–8. http://dx.doi.org/10.23880/jidtm-16000163.

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Background: Serratia marcescens has been described as a significant nosocomial organism. Several S. marcescens outbreaks in Neonatal Intensive Care Units (NICUs) were described as causing fatal meningitis, sepsis or pneumonitis in premature or low birth weight neonates with a mortality rate of 44%. The primary objective of this study is to describe the outcome (mortality and length of hospital stay) of S. marcescen s infection in NICU at a tertiary care hospital over ten years (2009 -2018). Secondary objectives are to describe the incidence of S. marcescens infection/colonization in NICU, study the risk factors associated with S. marcescens infection/colonization, and the microbiology of this organism. Method: A retrospective, unmatched case-control study was conducted between January 2009 to December 2018. Data were analyzed using IBM SPSS Statistics 28.0. A multivariate binary logistic regression analysis was performed to determine the independent predictors of Serratia marcescens and mortality among Serratia marcescens infected patients. The Odds Ratio (OR) was reported with its 95% CI. A P-value less than 0.05 was considered statistically significant. Result: A total of 93 cases had a positive culture of S. marcescens in neonates hospitalized in the NICU during the study period and 201 controls were included. 50.5% (n=47) of cases were male and 49.5% (n=46) were females. The clinical features of infection by S. marcescens range from asymptomatic colonization (16.1%) to potentially fatal sepsis (38.7%) and meningitis (1.1%). 13 cases (17.3 %) had colonization before infection. Mortality rate among infected neonates was 17%. Multivariate analysis showed that female gender (OR= 1.969, 95% CI= 1.020-3.801, P= 0.044), premature birth ((OR= 2.670, 95% CI= 1.156-6.167, P= 0.021). C-section (OR= 3.238, 95% CI= 1.591-6.591, P= 0.001), type of feeding and surgery (OR= 3.719, 95% CI= 1.546-8.946, P= 0.003) were independent predictors for acquiring S. marcescens . Female gender was an independent factor for mortality from Serratia infection (OR= 6.741, 95% CI= 1.307-34.767, P= 0.023). Conclusion: S. marcescens is an important pathogen that has a propensity to cause difficult-to-control outbreaks in NICUs. Healthcare workers' awareness of this organism and enhancement of infection prevention and control measures is a vital requirement to prevent HAIs among susceptible neonates.
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11

Wu, Zhiwei, Qing Ye, Xiaoyan Wang, Xianzeng Zhang, and Shusen Xie. "Rapid identification of Klebsiella pneumoniae and Serratia marcescens by surface-enhanced Raman spectroscopy." BIO Web of Conferences 59 (2023): 01019. http://dx.doi.org/10.1051/bioconf/20235901019.

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Two types of pathogenic bacteria, Klebsiella pneumoniae and Serratia marcescens, had been reported as important causes of hospital-acquired infection. Rapid and accurate identification of Klebsiella pneumoniae and Serratia marcescens is vitally important for the selection of appropriate treatment modalities. In this article, the feasibility of using surface-enhanced Raman Spectroscopy (SERS) to identify Klebsiella pneumoniae and Serratia marcescens was explored. Spectrum samples were obtained from Klebsiella pneumoniae infections (n=1000) and Serratia marcescens infections (n=1000). The differences between the spectra of two types of pathogenic bacteria were also analyzed. Moreover, Principal Component Analysis- Linear Discriminant Analysis (PCA-LDA) algorithm was used to discriminate the spectra of pathogenic bacteria.
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12

Harris, Abigail K. P., Neil R. Williamson, Holly Slater, Anthony Cox, Sophia Abbasi, Ian Foulds, Henrik T. Simonsen, Finian J. Leeper, and George P. C. Salmond. "The Serratia gene cluster encoding biosynthesis of the red antibiotic, prodigiosin, shows species- and strain-dependent genome context variation." Microbiology 150, no. 11 (November 1, 2004): 3547–60. http://dx.doi.org/10.1099/mic.0.27222-0.

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The prodigiosin biosynthesis gene cluster (pig cluster) from two strains of Serratia (S. marcescens ATCC 274 and Serratia sp. ATCC 39006) has been cloned, sequenced and expressed in heterologous hosts. Sequence analysis of the respective pig clusters revealed 14 ORFs in S. marcescens ATCC 274 and 15 ORFs in Serratia sp. ATCC 39006. In each Serratia species, predicted gene products showed similarity to polyketide synthases (PKSs), non-ribosomal peptide synthases (NRPSs) and the Red proteins of Streptomyces coelicolor A3(2). Comparisons between the two Serratia pig clusters and the red cluster from Str. coelicolor A3(2) revealed some important differences. A modified scheme for the biosynthesis of prodigiosin, based on the pathway recently suggested for the synthesis of undecylprodigiosin, is proposed. The distribution of the pig cluster within several Serratia sp. isolates is demonstrated and the presence of cryptic clusters in some strains shown. The pig cluster of Serratia marcescens ATCC 274 is flanked by cueR and copA homologues and this configuration is demonstrated in several S. marcescens strains, whilst these genes are contiguous in strains lacking the pig cluster.
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13

Indahwardani, Herliyana, Eny Widajati, and Giyanto. "Aplikasi Bakteri dalam Perlakuan Seed Coating untuk Mempertahankan Viabilitas dari Benih Cabai (Capsicum annuum L.) yang Sehat." Buletin Agrohorti 5, no. 1 (January 24, 2017): 9–16. http://dx.doi.org/10.29244/agrob.v5i1.15883.

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Penelitian ini bertujuan mengetahui pengaruh perlakuan pelapisan benih menggunakan bakteri Bacillus subtilis, Pseudomonas keompok. fluorescens dan Serratia marcescens terhadap viabilitas benih cabai (Capsicum annuum L.) selama di penyimpanan. Penelitian ini mengunakan rancangan petak tersarang dengan tiga ulangan. Petak utama adalah periode simpan (0, 3, 6, 9, 12, 15, 18, 21 dan 24 minggu) dan anak petak adalah perlakuan coating dengan bakteri tertentu (Bacillus subtilis, Pseduomonas kelompok fluorescens, Serratia marcescen dan kontrol). Benih cabai IPB C5 masih memiliki viabilitas yang cukup baik hingga akhir penyimpanan, ditunjukkan dengan nilai daya berkecambah sebesar 77.33%. Perlakuan kontrol dan coating menggunakan bakteri menunjukkan nilai daya berkecambah, indeks vigor, kecepatan tumbuh dan bobot kering kecambah normal yang tidak berbeda nyata. Ketiga bakteri yang digunakan sebagai pelapis masih dapat hidup sampai periode simpan 24 minggu dengan populasi 5.89 x 104 cfu g-1 untuk Pseudomonas kelompok fluorescens, 4.79 x 104 cfu g-1 untuk Bacillus subtilis dan 1.70 x 104 cfu g-1 untuk Serratia marcescens.
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14

de Courten, Christian, Patricio Sancho, and David BenEzra. "Metastatic Serratia marcescens Endophthalmitis." Journal of Pediatric Ophthalmology & Strabismus 25, no. 1 (January 1988): 45–47. http://dx.doi.org/10.3928/0191-3913-19880101-12.

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15

Ewete, Temitope, and Adegboyega Alabi. "Serratia marcescens Lacrimal Canaliculitis." Ophthalmology Research: An International Journal 6, no. 2 (January 10, 2016): 1–4. http://dx.doi.org/10.9734/or/2016/28205.

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16

Malvone, Leonard. "Ciprofloxacin-Resistant Serratia marcescens." Infection Control and Hospital Epidemiology 12, no. 6 (June 1991): 342. http://dx.doi.org/10.2307/30145207.

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17

Shah, Sonya B., Alok S. Bansal, Michael P. Rabinowitz, Carl Park, Edward H. Bedrossian, and Ralph C. Eagle. "ENDOGENOUS SERRATIA MARCESCENS ENDOPHTHALMITIS." Retinal Cases & Brief Reports 8, no. 1 (2014): 7–9. http://dx.doi.org/10.1097/icb.0b013e318298bf6a.

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18

Schechter, Marcos C., Jennifer O. Spicer, Sol del Mar Aldrete, and Colleen S. Kraft. "Serratia marcescens Infectious Endocarditis." Infectious Diseases in Clinical Practice 26, no. 4 (July 2018): 216–19. http://dx.doi.org/10.1097/ipc.0000000000000614.

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19

Malvone, Leonard. "Ciprofloxacin-Resistant Serratia marcescens." Infection Control & Hospital Epidemiology 12, no. 6 (June 1991): 342. http://dx.doi.org/10.1086/646352.

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20

Ostrowsky, B. E. "Bacériémies à Serratia marcescens." Revue Française des Laboratoires 2002, no. 344 (June 2002): 12–13. http://dx.doi.org/10.1016/s0338-9898(02)80008-7.

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21

Guo, Hsiao-Pei, and Tsung-Jen Wang. "Fulminant Serratia marcescens Panophthalmitis." American Journal of the Medical Sciences 353, no. 4 (April 2017): e7. http://dx.doi.org/10.1016/j.amjms.2016.09.005.

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22

Hogman, CF, H. Fritz, and L. Sandberg. "Posttransfusion Serratia marcescens septicemia." Transfusion 33, no. 3 (March 1993): 189–91. http://dx.doi.org/10.1046/j.1537-2995.1993.33393174441.x.

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23

Verschraegen, G., D. Voet, G. Claeys, M. Delanghe, H. Van Pelt, and M. Dierendonck. "Pseudobacteriuria with Serratia marcescens." Journal of Hospital Infection 12, no. 3 (October 1988): 238–40. http://dx.doi.org/10.1016/0195-6701(88)90015-1.

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24

Cunha, Burke A., Inge Gurevich, and Patricia Tafuro. "Nosocomial Serratia marcescens peritonitis." Clinical Microbiology Newsletter 9, no. 7 (April 1987): 55–56. http://dx.doi.org/10.1016/0196-4399(87)90015-8.

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25

Kim, Ki Seok, Koon Sig Park, Si Myung Byun, Jae Gu Pan, and Yong Chul Shin. "Overproduction of Serratia marcescens metalloprotease (SMP) from the recombinant Serratia marcescens strains." Biotechnology Letters 17, no. 5 (May 1995): 497–502. http://dx.doi.org/10.1007/bf00132017.

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26

Amini, Kumarss, and Maryam Akbari. "Cloning and Expression of Serratia Marcescens Prodigiosin Gene in Ecoli XL1blue." Jundishapur Journal of Medical Sciences 20, no. 2 (June 1, 2021): 102–11. http://dx.doi.org/10.32598/jsmj.20.2.1.

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Background and Objectives: Serratia is a gram-negative bacterium. The pigmentation property of Serratia Marcescens is used as a marker of dust particles in the environment and in the hospital. Today biopigments are also widely used in the manufacture and production of pharmaceutical products. Prodigiosin is a promising drug due to its reported properties of antifungal immunosuppressive and anti-proliferative activities. In the present study, cloning of pig gene- isolated from Serratia Marcescens in Ecoli XL1blue was performed. Subjects and Methods 60 Samples were taken from clinical sources of patients hospitalized with urinary tract infections in Saveh Hospitals. Serratia Marcescens were identified and isolated by different tests. The pig gene was cloned by T-A cloning using PTG-19 vector into the Escherichia coli XL1blue as host. Expression of cloned gene in recombinant colonies was evaluated by Real time PCR. The phylogenetic tree was plotted using clustalX and Mega5 software Results Screening of samples identified 12 isolates of Serratia Marcescens from then 4 isolates had pig gene. Expression of Pig gene in Escherichia coli XL1blue was confirmed by Real-Tima PCR. As a result of phylogenetic studies, some close relatives of serratia have been identified as candidates for further studies Conclusion Serratia Marcescens can be considered as a rich source of pigments with many applications and can be used as indigenous strains to produce Prodigiosin.
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27

Pant, Narayan Dutt, and Manisha Sharma. "Involvement of Serratia Marcescens along with Citrobacter freundii in causing Septic Arthritis." International Journal of Medicine and Biomedical Sciences 1, no. 2 (February 29, 2016): 17–21. http://dx.doi.org/10.55530/ijmbiosnepal.v1i2.12.

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Involvement of Serratia marcescens and Citrobacter freundii in causing septic arthritis is extremely rare. Here, we report a case of septic arthritis of knee joint following a recent arthroplasty, due to dual infection by Serratia marcescens and Citrobacter freundii in a diabetic patient. The patient had recent history of undergoing knee arthroplasty for functional and structural restoration of the knee joint injured due to saw injury. Serratia marcescens and Citrobacter freundii are known as common hospital acquired pathogens but septic arthritis due to these organisms is very rare even in health care settings.
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28

Harimawan, Ardiyan, Hary Devianto, Ignatius Chandra Kurniawan, and Josephine Christine Utomo. "INFLUENCE OF INITIAL pH SOLUTION ON BIOFILM FORMATION AND CORROSION OF CARBON STEEL BY Serratia marcescens." Reaktor 17, no. 2 (June 13, 2017): 89. http://dx.doi.org/10.14710/reaktor.17.2.89-95.

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The growth of Serratia marcescens depends on its metabolism, which is influenced by environmental factors, such as pH and temperature. The metabolic activity of Serratia marcescens may influence the corrosion of carbon steel by forming a biofilm on the metal surface. This research is focused on determining the effect of pH on carbon steel corrosion caused by Serratia marcescens. The medium used as immersion solution was a mixture of synthetic seawater and Luria-Bertani medium with a volume ratio of 4:1. The carbon steel coupons with a size of 1 cm x 1 cm were immersed in the solution with initial pH of 5, 7, and 9. The analyses of biofilm were conducted by total plate count (TPC), scanning electron microscopy (SEM) and Fourier transform infrared spectroscopy (FTIR). Biofilm was detected evenly on the metal surface and decreased with an increase in incubation pH. The biofilm consists of some functional groups, such as alcohol, alkane, amine, nitro, sulphate, carboxylic acid, and polysulfide. The analyses of the corrosion were conducted by gravimetric and X-ray diffraction (XRD). The pHs of 5 and 9 were found to give an increase in the corrosion rate. The average corrosion rate at pH variations of 5, 7, and 9 were 2.5309 g/m2.day; 2.2844 g/m2.day; and 2.9756 g/m2.day, respectively. Nevertheless, the corrosion products were not detected by XRD analysis. Keywords: biocorrosion; carbon steel; pH; seawater; Serratia marcescens Abstrak PENGARUH pH AWAL LARUTAN PADA PEMBENTUKAN BIOFILM DAN KOROSI BAJA KARBON OLEH Serratia marcescens. Laju pertumbuhan Serratia marcescens bergantung pada aktivitas metabolise mikroba, yang akan sangat dipengaruhi oleh faktor lingkungan, seperti pH dan temperatur. Aktivitas metabolisme Serratia marcescens dapat memengaruhi korosi pada baja karbon dengan membentuk lapisan biofilm pada permukaan logam. Penelitian ini bertujuan untuk menentukan efek pH pada korosi baja karbon yang disebabkan oleh Serratia marcescens. Media yang digunakan sebagai larutan perendam adalah campuran air laut sintetis dan media Luria-Bertani dengan perbandingan volume sebesar 4:1. Kupon baja karbon dengan ukuran 1 cm x 1 cm direndam dalam larutan dengan pH awal 5, 7, dan 9. Analisis lapisan biofilm dilakukan dengan total plate count (TPC), scanning electron microscopy (SEM) dan Fourier transform infrared spectroscopy (FTIR). Lapisan biofilm tumbuh secara merata pada permukaan logam dan berkurang seiring dengan peningkatan pH inkubasi. Lapisan biofilm mengandung berbagai gugus fungsional, seperti alkohol, alkana, amin, nitro, sulfat, asam karboksilat, dan polisulfida. Analisa korosi dilakukan dengan gravimetri dan X-ray diffraction (XRD). Penggunaan pH 5 dan 9 memberikan peningkatan terhadap laju korosi. Laju korosi rata-rata pada pH 5, 7, dan 9 ditentukan sebesar 2,5309 g/m2.day; 2,2844 g/m2.day; and 2,9756 g/m2.day. Namun, produk korosi tidak terdeksi oleh analisis XRD. Kata kunci: biokorosi; baja karbon; pH; air laut; Serratia marcescens
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29

de Ondarza, José. "Ozone Sensitivity and Catalase Activity in Pigmented and Non-Pigmented Strains of Serratia Marcescens." Open Microbiology Journal 11, no. 1 (March 31, 2017): 12–22. http://dx.doi.org/10.2174/1874285801711010012.

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Background:Ozone exposure rapidly leads to bacterial death, making ozone an effective disinfectant in food industry and health care arena. However, microbial defenses may moderate this effect and play a role in the effective use of oxidizing agents for disinfection.Serratia marcescensis an opportunistic pathogen, expressing genes differentially during infection of a human host. A better understanding of regulatory systems that control expression ofSerratia’s virulence genes and defenses is therefore valuable.Objective:Here, we investigated the role of pigmentation and catalase inSerratia marcescenson survival to ozone exposure.Method:Pigmented and non-pigmented strains ofSerratia marcescenswere cultured to exponential or stationary phase and exposed to 5 ppm of gaseous ozone for 2.5 – 10 minutes. Survival was calculated via plate counts. Catalase activity was measured photometrically and tolerance to hydrogen peroxide was assayed by disk-diffusion.Results:Exposure ofS. marcescensto 5 ppm gaseous ozone kills > 90% of cells within 10 minutes in a time and concentration-dependent manner. Although pigmentedSerratia(grown at 28°C) survived ozonation better than unpigmentedSerratia(grown at 35°C), non-pigmented mutant strains ofSerratiahad similar ozone survival rates, catalase activity and H2O2tolerance as wild type strains. Rather, ozone survival and catalase activity were elevated in 6 hour cultures compared to 48 hour cultures.Conclusion:Our studies did not bear out a role for prodigiosin in ozone survival. Rather, induction of oxidative stress responses during exponential growth increased both catalase activity and ozone survival in both pigmented and unpigmentedS. marcescens.
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30

Tahiri, F., A. Lalaoui, D. Kaouani, F. Bennaoui, N. El-Idrissi Slitine, N. Soraa, and F. M. R. Maouainine. "Serratia marcescens Meningitis: Neonatal Case." Asian Journal of Pediatric Research 12, no. 3 (May 5, 2023): 28–33. http://dx.doi.org/10.9734/ajpr/2023/v12i3243.

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Serratia marcescens belongs to the Enterobacteriaceae family, commonly found in water, soil, animals, insects, plants. Although Serratia marcescens has low virulence, it causes nosocomial infections and outbreaks in extremely immunocompromised or critically ill patients, particularly in intensive care units, particularly neonatal units. This pathogen causes a wide range of clinical symptoms in newborns, including keratitis, conjunctivitis, urinary tract infections, pneumonia, surgical site infections, sepsis, bloodstream infection, and meningitis. The bloodstream is the most commonly infected location, followed by the respiratory tract and the gastrointestinal tract. Serratia marcescens strains implicated in epidemic events have often proven to be multiresistant. Indeed, this species has an inherent resistance to multiple antibiotic families. Often, the particular source of infection cannot be determined. However, infected hands of healthcare professionals are thought to be a key vector for its spread. Infection of the central nervous system by this bacterium in the neonatal period leads to serious neurological sequelae with high mortality interest of early and adequate management. Through our work we report the clinical presentation, the positive diagnosis as well as the therapeutic management and the evolution of a newborn having presented a meningitis with Serratia marcescens complicated by ventriculitis.
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31

Atmomarsono, M., Nurbaya, Nurhidayah, and E. Susianingsih. "Use of Serratia marcescens MY1112 as Probiotic Bacteria for Tiger Shrimp Culture in the Acid-sulfate Soil Ponds." IOP Conference Series: Earth and Environmental Science 1328, no. 1 (April 1, 2024): 012004. http://dx.doi.org/10.1088/1755-1315/1328/1/012004.

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Abstract Probiotic bacteria have been commonly used for disease prevention in aquaculture systems, but sometimes these probiotic bacteria do not work in certain ponds. This experiment aimed to find out if the probiotic bacteria of Serratia marcescens MY1112 could work properly for tiger shrimp culture in acid-sulfate soil ponds. Eight 0.5-ha ponds located in Samataring village of Sinjai regency were used. Two treatments of bacteria probiotic combinations were applied here, namely A) Brevibacillus laterosporus BT951, Serratia marcescens MY1112, and Bacillus licheniformis BM58; and B) Use of Bacillus subtilis BM12 to replace S. marcescens MY1112 in the bacteria combination. The shrimp production and their survival rate in treatment A were significantly better than those of treatment B. However, probiotic bacteria of Serratia marcescens MY1112 could work better in the acid-sulfate soil pond if combined with the dolomite application weekly.
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32

Pavlović, H., J. Hardi, V. Slačanac, M. Halt, and D. Kocevski. "Inhibitory effect of goat and cow milk fermented by Bifidobacterium longum on Serratia marcescens and Campylobacter jejuni." Czech Journal of Food Sciences 24, No. 4 (November 12, 2011): 164–71. http://dx.doi.org/10.17221/3312-cjfs.

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This study was performed to determine the influence of fermented goat and cow milk produced by the use of Bifidobacterium longum Bb-46 on pathogenic Serratia marcescens and Campylobacter jejuni strains. The correlation between the inhibitory effect and some fermentation parameters (the number of viable probiotic cells and pH of fermented milk) was also determined. Bifidobacterium longum counts and pH values were also measured in milk samples during fermentation. The results showed that the inhibitory effect of Bifidobacterium longum Bb-46 fermented goat milk on Serratia marcescens increased with the fermentation time. The highest inhibitory effect of fermented cow milk occurred in the middle course of fermentation. Statistically significant correlation between the inhibition degree of Serratia marcescens growth and pH values of fermented goat milk was noted as opposed to the correlation between the inhibition degree of Serratia marcescens growth and pH values of fermented cow milk which was not statistically significant. All samples of goat and cow fermented milk exhibited inhibitory effects on the growth of Campylobacter jejuni.  
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33

Bhadra, Bhaskar, Pradosh Roy, and Ranadhir Chakraborty. "Serratia ureilytica sp. nov., a novel urea-utilizing species." International Journal of Systematic and Evolutionary Microbiology 55, no. 5 (September 1, 2005): 2155–58. http://dx.doi.org/10.1099/ijs.0.63674-0.

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A Gram-negative, rod-shaped, urea-dissolving and non-spore-forming bacterium, designated strain NiVa 51T, was isolated from water of the River Torsa in Hasimara, Jalpaiguri district, West Bengal, India. On the basis of 16S rRNA gene sequence similarity, strain NiVa 51T was shown to belong to the γ-Proteobacteria and to be related to Serratia marcescens subsp. sakuensis (98·35 %) and S. marcescens subsp. marcescens (98·30 %); however, strain NiVa 51T exhibited only 43·7 % similarity to S. marcescens by DNA–DNA hybridization. The G+C content of the genomic DNA of the isolate was 60 mol%. Both biochemical characteristics and fatty acid analysis data supported the affiliation of strain NiVa 51T to the genus Serratia. Furthermore, strain NiVa 51T was found to utilize urea as nitrogen source. The results of DNA–DNA hybridization as well as physiological and biochemical tests allowed genotypic and phenotypic differentiation of strain NiVa 51T from recognized Serratia species. Strain NiVa 51T therefore represents a novel species, for which the name Serratia ureilytica sp. nov. is proposed, with type strain NiVa 51T (=LMG 22860T=CCUG 50595T).
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34

Cohen, Steven M., Harry W. Flynn, and Darlene Miller. "Endophthalmitis Caused by Serratia marcescens." Ophthalmic Surgery, Lasers and Imaging Retina 28, no. 3 (March 1997): 195–200. http://dx.doi.org/10.3928/1542-8877-19970301-04.

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35

Rastogi, V., P. Purohit, BP Peters, and PS Nirwan. "PULMONARY INFECTION WITH SERRATIA MARCESCENS." Indian Journal of Medical Microbiology 20, no. 3 (July 2002): 167–68. http://dx.doi.org/10.1016/s0255-0857(21)03254-0.

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36

Nazzaro, Gianluca, and Stefano Veraldi. "Serratia marcescens: an Italian story." International Journal of Dermatology 56, no. 7 (April 23, 2017): 795–96. http://dx.doi.org/10.1111/ijd.13632.

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37

Sridhar, Jayanth, Ajay E. Kuriyan, Harry W. Flynn, William E. Smiddy, Vincent D. Venincasa, and Darlene Miller. "ENDOPHTHALMITIS CAUSED BY SERRATIA MARCESCENS." Retina 35, no. 6 (June 2015): 1095–100. http://dx.doi.org/10.1097/iae.0000000000000509.

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38

Peeters, A., B. Vandercam, C. J. M. Sindic, P. Hantson, and P. Mahieu. "Community-acquired Serratia marcescens meningitis." Journal of Infection 35, no. 3 (November 1997): 303–4. http://dx.doi.org/10.1016/s0163-4453(97)93384-3.

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39

CAMPBELL, JUDITH R., THOMAS DIACOVO, and CAROL J. BAKER. "Serratia marcescens meningitis in neonates." Pediatric Infectious Disease Journal 11, no. 10 (October 1992): 881–86. http://dx.doi.org/10.1097/00006454-199210000-00015.

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40

Taylor, Stephanie Parks, and Brice Taylor. "Pseudohemoptysis Due to Serratia marcescens." Journal of General Internal Medicine 29, no. 6 (November 2, 2013): 962–63. http://dx.doi.org/10.1007/s11606-013-2649-0.

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41

Newsom, S. W. B. "Serratia marcescens: A colourful microbe." British Journal of Infection Control 9, no. 1 (January 2008): 25–27. http://dx.doi.org/10.1177/1469044607085004.

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42

Ruiz-Sánchez, A., R. Cruz-Camarillo, R. Salcedo-Hernández, and J. E. Barboza-Corona. "Chitinases from Serratia marcescens Nima." Biotechnology Letters 27, no. 9 (May 2005): 649–53. http://dx.doi.org/10.1007/s10529-005-3661-1.

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43

Mardanova, A. M., L. M. Bogomol’naya, Yu D. Romanova, and M. R. Sharipova. "Efflux systems in Serratia marcescens." Microbiology 82, no. 6 (November 2013): 668–79. http://dx.doi.org/10.1134/s0026261714010093.

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44

Curós, Núria, Meritxell Sallés, Elisabet García-Casares, and Sonia Molinos. "Bursitis séptica por Serratia marcescens." Medicina Clínica 127, no. 1 (June 2006): 37. http://dx.doi.org/10.1157/13089871.

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45

Braun, Volkmar, Hannelore G�nther, Burkard Neu�, and Christiane Tautz. "Hemolytic activity of Serratia marcescens." Archives of Microbiology 141, no. 4 (May 1985): 371–76. http://dx.doi.org/10.1007/bf00428852.

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46

Damayanti, Berliana, Sumardi Sumardi, Achmad Arifiyanto, Kusuma Handayani, M. Kanedi, Meishy Handerlin Putri, and Cindy Lukyta Ratih Riyanto. "Pengaruh Media Pertumbuhan dan pH Terhadap Aktivitas Biosurfaktan dari Bakteri Serratia marcescens strain MBC 1 pada Minyak Jelantah." IJCA (Indonesian Journal of Chemical Analysis) 5, no. 1 (March 1, 2022): 01–08. http://dx.doi.org/10.20885/ijca.vol5.iss1.art1.

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Campuran minyak dan bahan kimia pada surfaktan yang dibuang langsung ke lingkungan akan mengakibatkan penurunan kesuburan tanah sertamenghambat proses degradasi oleh mikroorganisme. Oleh karena itu dibutuhkan senyawa alami yang mampu mampu meningkatkan kelarutan minyak jelantah dalam air seperti biosurfaktan. Salah satu bakteri penghasil biosurfaktan yaitu Serratia marcescens. Penelitian ini bertujuan untuk mengetahui aktivitas biosurfaktan bakteri dari Serratia marcescens strain MBC 1 yang ditumbuhkan di media fermentasi tryptone water, limbah cair jagung dan limbah cair singkong dengan pH 6,7 dan 8. Uji yang dilakukan diantaranya uji emulsifikasi, oil displacement dan drop collapse. Hasil penelitian menunjukan biosurfaktan Serratia marcescens strain MBC 1 mampu meningkatkan kelarutan minyak jelantah dalam air. Hasil produksi pada media limbah jagung dengan pH 7 menunjukan aktivitas emulsifikasi paling optimum yaitu sebesar 49,26%.
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47

Hutsul, Joanne, Elizabeth Worobec, Tom R. Parr Jr., and Gerald W. Becker. "Comparative analyses of Serratia spp. outer membrane porin proteins." Canadian Journal of Microbiology 39, no. 4 (April 1, 1993): 442–47. http://dx.doi.org/10.1139/m93-064.

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Eight Serratia strains and several members of the Enterobacteriaceae family were used in immunoblot and Southern DNA hybridization experiments and probed with antibody and DNA probes specific for the 41-kDa Serratia marcescens porin, to determine the extent of homology between Gram-negative porins. Immunoblot analyses performed using porin-specific rabbit sera and cell envelope preparations from these strains revealed that all strains produced at least one cross-reactive protein in the 41-kDa molecular weight range. Chromosomal DNA from each of the same strains was used in Southern analyses, probed with a 20-base-length oligonucleotide probe deduced from the N-terminal amino acid sequence of the 41-kDa Serratia marcescens porin. The probe hybridized to DNA from all of the Serratia species and six of the nine other enteric bacteria. Putative porin proteins from all the Serratia species were subjected to N-terminal amino acid sequencing and porin functional analysis using the black lipid bilayer method. All amino acid sequences were identical, with one exception in which an asparagine was substituted for an aspartic acid in Serratia rubidaea. All porins had very similar porin function (single channel conductance ranging between 1.72 and 2.00 nS). The results from this study revealed that a strong conservation exists among the Serratia porins and those produced by other enteric bacteria.Key words: porins, Serratia marcescens, homology studies.
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48

Wilson, D. J., J. H. Kirk, R. D. Walker, and Q. W. Bosworth. "Serratia marcescens mastitis in a dairy herd." Journal of the American Veterinary Medical Association 196, no. 7 (April 1, 1990): 1102–5. http://dx.doi.org/10.2460/javma.1990.196.07.1102.

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Summary Serratia marcescens caused clinical mastitis in 5 cows and nonclinical mastitis in 21 cows of a 190-cow herd. Repeated bacteriologic culture of specimens from the cows, postmilking teat dip, environment, and equipment was performed. Serratia marcescens was not isolated from the dip, environment, or equipment. Progress of the infection in cows was monitored for 10 months. Some cows remained infected with S marcescens for at least 10 months. Economic loss estimates were based on Dairy Herd Improvement Association linear score reports. The average nonclinical loss was about $22/cow.
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49

Muchtar, Sarah Desmia, Eny Widajati, and Giyanto. "Pelapisan Benih Menggunakan Bakteri Probiotik untuk Mempertahankan Viabilitas Benih Jagung Manis (Zea mays saccharata Sturt.) selama Penyimpanan." Buletin Agrohorti 1, no. 4 (January 10, 2014): 26. http://dx.doi.org/10.29244/agrob.1.4.26-33.

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<p><em>Penelitian ini bertujuan untuk mengetahui pengaruh perlakuan pelapisan benih menggunakan bakteri Bacillus subtilis, Serratia marcescens, dan Pseudomonas kelompok fluorescens terhadap viabilitas dan daya simpan benih jagung manis (Zea mays saccharataSturt.). Penelitian disusun dengan rancangan petak tersarang (Nested Design) dengan petak utama adalah periode simpan minggu ke 0, 3, 6, 9, 12, 15, 18, 21, dan 24 dan anak petak adalah perlakuan coating dengan bakteri Bacillus subtilis, Serratia marcescens, Pseudomonas kelompok fluorescens, dan kontrol. Hasil penelitian menunjukkan adanya interaksi yang sangat nyata antara perlakuan coating benih dan periode simpan terhadap daya berkecambah, berat kering kecambah normal, kadar air, dan populasi bakteri. Perlakuan coating benih dan periode simpan menunjukkan interaksi yang nyata terhadap indeks vigor benih. Tolok ukur kecepatan tumbuh benihhanya dipengaruhi oleh faktor tunggal periode simpan. Benih yang dilapisi bakteri menghasilkan nilai berat kering kecambah normal yang nyata lebih baik daripada benih tanpa coating. Benih tanpa coating memiliki nilai daya berkecambah sebesar 56.7% pada periode simpan 24 minggu. Benih yang dilapisi bakteri Bacillus subtilis dapat mempertahankan daya berkecambah hingga 64.0% sampai periode simpan 24 minggu, sedangkan Serratia marcescens mampu mempertahankan daya berkecambah hingga 60.0% . Berdasarkan tolok ukur daya berkecambah, pelapisan benih menggunakan bakteri Bacillus subtilis dan Serratia marcescens merupakan perlakuan pelapisan benih yang potensial untuk lebih dikembangkan. Bakteri Bacillussubtilis lebih mampu bertahan hidup selama penyimpanan dibanding dengan bakteri Serratia marcescens maupun Pseudomonas fluorescens. Populasi bakteri Bacillus subtilis sampai dengan periode simpan 24 minggu adalah 14.2 × 10<sup>4</sup> cfu g<sup>-1</sup>.</em></p>
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50

Coulthurst, Sarah J., C. Léopold Kurz, and George P. C. Salmond. "luxS mutants of Serratia defective in autoinducer-2-dependent ‘quorum sensing’ show strain-dependent impacts on virulence and production of carbapenem and prodigiosin." Microbiology 150, no. 6 (June 1, 2004): 1901–10. http://dx.doi.org/10.1099/mic.0.26946-0.

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The enzyme LuxS is responsible for the production of autoinducer-2 (AI-2), a molecule that has been implicated in quorum sensing in many bacterial species. This study investigated whether there is a luxS-dependent signalling system in the Gram-negative bacteria Serratia spp. Serratia marcescens is a broad-host-range pathogen and an important cause of nosocomial infections. Production of AI-2 activity was detected in S. marcescens ATCC 274 and Serratia ATCC 39006 and their luxS genes were sequenced. luxS mutants were constructed in these strains and were analysed to determine which phenotypes are regulated by luxS and therefore, potentially, by AI-2. The phenotypes of the luxS mutants included decreased carbapenem antibiotic production in Serratia ATCC 39006 and decreased prodigiosin and secreted haemolysin production in S. marcescens ATCC 274. The luxS mutant of S. marcescens ATCC 274 was also found to exhibit modestly reduced virulence in a Caenorhabditis elegans model. Finally, it was shown that the culture supernatant of a wild-type strain contains a signal, presumably AI-2, capable of complementing the prodigiosin defect of the luxS mutant of another strain, even when substantially diluted. It is concluded that luxS modulates virulence and antibiotic production in Serratia, in a strain-dependent manner, and that, for at least one phenotype, this regulation is via extracellular signalling.
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