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Relevant bibliographies by topics / Serratia marcescens

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Academic literature on the topic 'Serratia marcescens'

Author: Grafiati

Published: 4 June 2021

Last updated: 18 May 2024

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Journal articles on the topic "Serratia marcescens"

1

HEJAZI, A., and F. R. FALKINER. "Serratia marcescens." Journal of Medical Microbiology 46, no. 11 (November 1, 1997): 903–12. http://dx.doi.org/10.1099/00222615-46-11-903.

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Yannelli, Barbara, Paul E. Schoch, and Burke A. Cunha. "Serratia marcescens." Clinical Microbiology Newsletter 9, no. 20 (October 1987): 157–60. http://dx.doi.org/10.1016/0196-4399(87)90078-x.

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Ikpa, Amadi, and Ogbonda KH. "Enhancing Contact Tracing for Serratia marcescens Biofilm on High-Usage Body Towels in Rivers State Bathrooms." Journal of Healthcare and Biomedical Science 1, no. 2 (August 7, 2023): 1–10. http://dx.doi.org/10.31098/jhbs.v1i2.1384.

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The placement of body towels before and after use in bathrooms where Serratia marcescens proliferates calls for concern, as Serratia marcescens, an airborne opportunistic pathogen has been reported keratinolytic. Serratia marcescens, a bacterium commonly noticed with a pink or red slimy appearance in toilet sinks, bowls, and tiles presents an aesthetically, unappealing and disgusting appearance in the toilet surroundings. The study aimed to trace the presence of Serratia marcescens on frequently used body towels hung in the bathroom doors. Swabs from forty (40) differently used towels were collected from twenty (20) volunteered homes and analyzed using standard microbiological procedures. Microbiological procedures involved inoculating the swab sample on a prepared peptone broth and plating on MacConkey agar media, followed by identification and streak of the recovered isolate onto Congo red agar media for biofilm formation. Results showed the recovery of Serratia marcescens isolates. The three homes showed a Serratia marcescens count of 3 X 10, 1 X 102, and 7 X 10 CFU per swab for house units C, I, and P respectively. Serratia marcescens could form a biofilm, a basic feature that allowed it to strive on a body towel. The results derived strongly identified the presence of Serratia marcescens biofilm on body towels hung in the bathrooms. This could have health implications for towel users due to the bacteria's keratolytic properties. Hence, the need for constant surveillance to support effective measures of hygiene, aimed at preventing the spread of Serratia marcescens is recommended.

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CN, Amadi-Ikpa, and Ogbonda KH. "Enhancing Contact Tracing for Serratia marcescens Biofilm on High-Usage Body Towels in Rivers State Bathrooms." Journal of Healthcare and Biomedical Science 1, no. 2 (June 29, 2023): 1–10. http://dx.doi.org/10.31098/jhbs.v2i1.1608.

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The placement of body towels before and after use in bathrooms where Serratia marcescens proliferates calls for concern, as Serratia marcescens, an airborne opportunistic pathogen has been reported keratinolytic. Serratia marcescens, a bacterium commonly noticed with a pink or red slimy appearance in toilet sinks, bowls, and tiles presents an aesthetically, unappealing and disgusting appearance in the toilet surroundings. The study aimed to trace the presence of Serratia marcescens on frequently used body towels hung in the bathroom doors. Swabs from forty (40) differently used towels were collected from twenty (20) volunteered homes and analyzed using standard microbiological procedures. Microbiological procedures involved inoculating the swab sample on a prepared peptone broth and plating on MacConkey agar media, followed by identification and streak of the recovered isolate onto Congo red agar media for biofilm formation. Results showed the recovery of Serratia marcescens isolates. The three homes showed a Serratia marcescens count of 3 X 10, 1 X 102, and 7 X 10 CFU per swab for house units C, I, and P respectively. Serratia marcescens could form a biofilm, a basic feature that allowed it to strive on a body towel. The results derived strongly identified the presence of Serratia marcescens biofilm on body towels hung in the bathrooms. This could have health implications for towel users due to the bacteria's keratolytic properties. Hence, the need for constant surveillance to support effective measures of hygiene, aimed at preventing the spread of Serratia marcescens is recommended.

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Nazzaro, Gianluca. "Etymologia: Serratia marcescens." Emerging Infectious Diseases 25, no. 11 (November 2019): 2012. http://dx.doi.org/10.3201/eid2511.et2511.

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6

Theccanat, G., L. Hirschfield, and H. Isenberg. "Serratia marcescens meningitis." Journal of Clinical Microbiology 29, no. 4 (1991): 822–23. http://dx.doi.org/10.1128/jcm.29.4.822-823.1991.

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7

Yanagi, T., K. Katagiri, T. Sonada, and S. Takayasu. "Serratia marcescens granuloma." British Journal of Dermatology 136, no. 2 (February 1997): 289–90. http://dx.doi.org/10.1111/j.1365-2133.1997.tb14919.x.

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REISSELL, PENTTI, and ELLI JANSSON. "Serratia Marcescens Septicemia." Acta Medica Scandinavica 176, no. 2 (April 24, 2009): 253–56. http://dx.doi.org/10.1111/j.0954-6820.1964.tb00932.x.

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Acar, Jacques F. "Serratia marcescens Infections." Infection Control 7, no. 5 (May 1986): 273–80. http://dx.doi.org/10.1017/s0195941700064201.

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The recognition of serratia as an opportunistic human pathogen can be dated from 1959, when the microorganism entered the family of Enterobacteriaceae, with features recognizable in the clinical laboratory and related to the Klebsiella/Enterobacter group. Since then, physicians have been challenged to establish the significance of isolation of serratia from a clinical specimen.

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Mahrooqi, Muna AL. "Epidemiology of Serratia Marcescens in the Neonatal ICU of A Tertiary Hospital in Oman over a 10 Years Period." Journal of Infectious Diseases & Travel Medicine 6, no. 2 (2022): 1–8. http://dx.doi.org/10.23880/jidtm-16000163.

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Background: Serratia marcescens has been described as a significant nosocomial organism. Several S. marcescens outbreaks in Neonatal Intensive Care Units (NICUs) were described as causing fatal meningitis, sepsis or pneumonitis in premature or low birth weight neonates with a mortality rate of 44%. The primary objective of this study is to describe the outcome (mortality and length of hospital stay) of S. marcescen s infection in NICU at a tertiary care hospital over ten years (2009 -2018). Secondary objectives are to describe the incidence of S. marcescens infection/colonization in NICU, study the risk factors associated with S. marcescens infection/colonization, and the microbiology of this organism. Method: A retrospective, unmatched case-control study was conducted between January 2009 to December 2018. Data were analyzed using IBM SPSS Statistics 28.0. A multivariate binary logistic regression analysis was performed to determine the independent predictors of Serratia marcescens and mortality among Serratia marcescens infected patients. The Odds Ratio (OR) was reported with its 95% CI. A P-value less than 0.05 was considered statistically significant. Result: A total of 93 cases had a positive culture of S. marcescens in neonates hospitalized in the NICU during the study period and 201 controls were included. 50.5% (n=47) of cases were male and 49.5% (n=46) were females. The clinical features of infection by S. marcescens range from asymptomatic colonization (16.1%) to potentially fatal sepsis (38.7%) and meningitis (1.1%). 13 cases (17.3 %) had colonization before infection. Mortality rate among infected neonates was 17%. Multivariate analysis showed that female gender (OR= 1.969, 95% CI= 1.020-3.801, P= 0.044), premature birth ((OR= 2.670, 95% CI= 1.156-6.167, P= 0.021). C-section (OR= 3.238, 95% CI= 1.591-6.591, P= 0.001), type of feeding and surgery (OR= 3.719, 95% CI= 1.546-8.946, P= 0.003) were independent predictors for acquiring S. marcescens . Female gender was an independent factor for mortality from Serratia infection (OR= 6.741, 95% CI= 1.307-34.767, P= 0.023). Conclusion: S. marcescens is an important pathogen that has a propensity to cause difficult-to-control outbreaks in NICUs. Healthcare workers' awareness of this organism and enhancement of infection prevention and control measures is a vital requirement to prevent HAIs among susceptible neonates.

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Dissertations / Theses on the topic "Serratia marcescens"

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Oxley, David. "Surface polysaccharides of Serratia marcescens." Thesis, University of Hull, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252968.

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Brigden, C. J. "Surface carbohydrates of Serratia marcescens." Thesis, University of Hull, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375628.

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Jessop, Helen L. "The immunochemistry of serratia marcescens." Thesis, Aston University, 1986. http://publications.aston.ac.uk/12463/.

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Yan, Qiang. "Metabolic Engineering of Serratia marcescens." VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5348.

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The potential value of the chitin biomass (e.g. food waste) is recently considered being ignored by landfill. Chitin can be a potential cheap carbon source for converting into value-added chemicals by microorganisms. Serratia marcescens is a chitinolytic bacterium that harbors endogenous chitinase systems. With goals of characterzing S. marcescens chitinolytic capabilities and applying S. marcescens to chemical production from chitin, my dissertation main content includes five chapters: 1) Chapter 1 highlights background information of chitin source, S. marcescens and potential metabolic engineering targets using chitin as a substrate; 2) Chapter 2 demonstrates that ChiR is a key regulator in regulating 9 chitinase-related genes in S. marcescens Db11 and manipulation of chiR can be a useful and efficient genetic target to enhance chitin utilization; 3) Chapter 3 reports the production of N-acetylneuraminic acid (Neu5Ac) from chitin by a bottom-up approach of engineering the nonconventional chitinolytic bacterium, Serratia marcescens, including native constitutive promoter characterization and transcriptional and translational pathway balancing; 4) Chapter 4 describes improvement of S. marcescens chitinolytic capability by an adaptive evolution approach; 5) Chapter 5 elucidates S. marcescens intracellular metabolite profile using a constraint-based genome-scale metabolic model (iSR929) based on genomic annotation of S. marcescens Db11. Overall, the dissertation work is the first report of demonstrating the concept of chitin-based CBP using S. marcescens and the computational model and genetic molecular tools developed in this dissertation are valuable but not limited to design-build-test of S. marcescens for contributing to the field of biological science and metabolic engineering applications.

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Silva, Cristina Ferraz. "Produção biotecnologica de surfactante por Serratia marcescens." [s.n.], 2002. http://repositorio.unicamp.br/jspui/handle/REPOSIP/256676.

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Orientador: Glaucia Maria Pastore
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Engenharia de Alimentos
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Resumo: Biosurfactantes são compostos produzidos por microrganismos os quais possuem em sua molécula uma porção hidrofilica (solúvel em água) e uma porção hidrofóbica (insolúvel em água). Essas moléculas são capazes de reduzir a tensão superficial e interfacial em ambas soluções aquosas e misturas de hidrocarbonetos, as quais fazem desses compostos potenciais candidatos para aumentar a recuperação de óleo e processos de deemulsificação. No presente trabalho foi estudada a produção de biosurfactante por uma linhagem de bactéria. O microrganismo considerado foi pré-selecionado como produtor de biosurfactante em trabalhos anteriores e identificado nesta dissertação como Serratía marcescens. As melhores condições para produção do biosurfactante em agitador rotativo foram determinadas através do processo de otimização utilizando Planejamento Experimental. Além disso, foram estudadas algumas propriedades do biosurfactante, como por exemplo, capacidade emulsificante e estabilidade em diferentes pHs e temperaturas. Finalmente, testou-se a aplicação do biosurfactante produzido avaliando-se o efeito da sua adição na atividade de lipase de Rhízopus sp. quando comparado ao efeito produzido por surfactantes químicos.
Abstract: Biosurfactants are compounds produced by microrganisms those molecules include a hydrophilic portion (water soluble) and a hydrophobic portion (water insoluble). These molecules are capable of reducing surface and interfacial tensions in both aqueous solutions and hydrocarbon mixtures, which makes them potential candidates for enhancing oil recovery and deemulsification processes. Biosurfactant production by a bacterial strain was studied in this work. The microrganism considered was isolated in previous studies and identified in this work as Serratia marcescens. The best conditions for biosurfactant production in shake flasks were determined through an optimization process using an Experimental Design. Móreover, some properties of the biosurfactant were studied, for example, its emulsifying capacity and stability at different pH values and temperatures. Finally, the application of the biosurfactant produced was tested evaluating the effect of its addition on the activity of Rhizopus sp. Lipase, as compared to the effect produced by chemical surfactants.
Mestrado
Mestre em Ciência de Alimentos

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6

Perrakis, Anastassis. "Structural studies of chitinase A from Serratia marcescens." Thesis, University of York, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307167.

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7

Hamilton, Jaeger. "Secretion of the chitinolytic machinery in Serratia marcescens." Thesis, University of Dundee, 2013. https://discovery.dundee.ac.uk/en/studentTheses/7f7a0d4f-1ac2-4ca1-81fc-459d5ec87712.

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There are six known secretion systems in Gram negative bacteria, referred to as Type 1 to Type 6 respectively, which are dedicated to moving substrate across the outer membrane. Secretion systems are broadly separated into those that move their substrate across the cell envelope in a single translocation event (one-step systems), and those that are dependent on the Sec or Tat machineries for export to the periplasm (two-step systems). Serratia marcescens is an important opportunistic human pathogen and has gathered a lot of interest due to its repertoire of secreted proteins. These include the haem-scavenging protein HasA, which is secreted by a Type 1 secretion system, and the cytotoxic haemolysin ShlA, which is secreted as part of a two-partner Type 5 secretion system. Serratia marcescens also encodes a Type 6 secretion system, which is known to translocate at least six effector molecules directly into other bacterial target cells. Serratia marcescens is a model organism in terms of its ability to degrade the quite intractable polymer chitin, for which it produces three chitinase enzymes ChiA, ChiB, ChiC and a chitin-binding protein Cbp21, which hydrolyse the ß-1,4 link in the chitin chain and promote binding of chitinase to the chitin substrate respectively. These chitinolytic enzymes are utilised by S. marcescens for both basic physiology and also in pathogenesis. In this work, genetic, biochemical and proteomic approaches identified, for the first time, genes that are essential for the secretion of all three chitinases as well as Cbp21. A genetic screen identified genes encoding a holin-like membrane protein (ChiW) and a putative L-alanyl-D-glutamate endopeptidase (ChiX). Subsequent quantitative proteomics experiments and biochemical analyses established that ChiW and ChiX were required for secretion of the entire chitinolytic machinery. Chitinase secretion was observed to be blocked at a late stage in the mutant strains as normally secreted enzymes were found to accumulate in the periplasm, thus implicating ChiW and ChiX in a novel outer membrane protein translocation process. It is proposed that the bacterial genome-encoded holin-like protein and endopeptidase identified represent a putative secretion system utilised by Gram-negative bacteria. In addition to this, genes encoding the chitinolytic machinery and the putative secretion apparatus were shown to be bimodally regulated and co-ordinately expressed.

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Moya, Torres Aniel. "The role of Serratia marcescens OmpF and OmpC porins in antibiotic resistance and virulence." Microbiology, 2014. http://hdl.handle.net/1993/30388.

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Serratia marcescens is a microorganism that constitutes one of the primary causes of nosocomial outbreaks in hospitals. One characteristic of S. marcescens clinical isolates is the high resistance to antimicrobials used in the clinic. Recent reports have attributed antibiotic resistance to altered porin expression. In this study, S. marcescens Db11 isogenic porin mutants were generated using the generalized transducing phage IF3 to move marked target-genes between isogenic strain backgrounds, prior to removal of the antibiotic resistance cassette by Flp-FRT strategy. Mutants for three classical porins were obtained and the effect of ompF and ompC deletion on antimicrobial resistance was evaluated by MIC. The use of this method avoided the incorporation of additional resistance markers and is an alternative strategy to create clean unmarked Serratia mutant strains. The lack of OmpF, but not OmpC, significantly increased MIC values to the β-lactam drugs such as ampicillin and cefoxitin as well as to nitrofurantoin. Genetic deletion of both ompF and ompC did not compromise the integrity of the bacterial cell envelope in optimal growth conditions, suggesting that other outer-membrane porins may function in a compensatory role to facilitate nutrient uptake and cell envelope integrity. S. marcescens is a pathogen of C. elegans and can be used to study host response to bacterial infections. The host model Caenorhabditis elegans was used in this study to investigate if porin deficits affected bacterial virulence. When porin mutants were evaluated in the C. elegans host model, the virulence of the single porin mutant strains increased in comparison to the wild-type. This study demonstrated that mutations of ompF and ompC did not attenuate S. marcescens virulence, but rather demonstrated a hypervirulent phenotype when they were assessed in C. elegans. The absence of OmpF and OmpC porins in S. marcescens appeared to increase the bacterial invasion of C. elegans nematode tissue. Further studies are required to fully investigate the hypervirulent phenotype of these mutant strains. This study reveals that decrease of outer membrane permeability due to porin mutation alters antimicrobial resistance and does not generate virulence attenuation in S. marcescens Db11.
May 2015

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Escobar, Marcelo Martins. "Atividade citotoxica do sobrenadante de cultura de Serratia marcescens fitopatogenica." [s.n.], 2002. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317315.

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Orientadores : Tomomasa Yano, Gleize Villela Carbonell
Dissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia
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Mestrado

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Berlanga, Herranz Mercedes. "Mecanismos de resistencia a las quinolonas en Serratia marcescens." Doctoral thesis, Universitat de Barcelona, 1999. http://hdl.handle.net/10803/672847.

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Esta tesis doctoral forma parte de un conjunto de trabajos desarrollados por el grupo de investigación del Dr. M. Viñas del Departamento de Microbiología y Parasitología Sanitarias de Ia Division de Ciencias de Ia Salud de la Universidad de Barcelona. La mayoría de los trabajos publicados hacen referenda a especies como Escherichia coli, Pseudomonas aeruginosa o Staphylococcus aureus, pero en Serratia marcescens los mecanismos de resistencia han sido poco estudiados. Sin embargo S. marcescens ha producido durante los ultimos afws un inusual elevado número de episodios infecciosos en los centros hospitalarios españoles, además estudios epidemiológicos han constatado un elevado porcentaje de aislados resistentes a las fluoroquinolonas en Serratia y Enterobacter, que es superior a cualquier otro género de las enterobacteriáceas. El objetivo principal de esta tesis ha sido Ia de aportar más conocimientos en el estudio de los mecanismos de resistencia a l as quinolonas en S. marcescens. Los objetivos concretes de la presente tesis doctoral son: 1. La obtención y caracterización de mutantes espontáneos resistentes at ciprofloxacino obtenidos in vitro. 2. Investigar la importancia de la membrana externa (papel del lipopolisacarido y porinas) y las características fisicoquímicas de las quinolonas (hidrofobicidad y grado de protonacion en funcion del pH externo) en la acumulación de estos antibióticos por Ia bacteria S. marcescens. 3. Comprobar si existen variaciones en la acumulación del ciprofloxacino,debido a Ia competencia por la vía de entrada, incubando este antibiótico simultanéamente con otros agentes antibacterianos que pasan a través de las porinas. 4. La busqueda y caracterización de bombas de reflujo en Serratia marcescens, ya que una baja permeabilidad no podría explicar por sí sola niveles significativos de resistencia clínica, de tal manera que sería/n necesario/s otro/s mecanismo/s que actuaran de forma sinérgica. Se cree que este segundo factor podría ser el reflujo o extrusión de substancias. 5. Estudiar el mecanismo de acción antibacteriano del ciprofloxacino y ácido nalídxico en S. marcescens. 6. Estudiar las variaciones de la susceptibilidad y acumulación de quinolonas en presencia de analgésicos (ácido salicílico y paracetamol). 7. El estudio comparativo de Ia acumulación y susceptibilidad de los derivados del ciprofloxacino en S. marcescens. 8. Comparar Ia acumulación y susceptibilidad del ciprofloxacino en S. marcescens, Escherichia coli, Pseudomonas aeru9inosa y Staphylococcus aureus. 9. Comprobar si existe alguna mutación en la región QRDR (quinolone resistance determining region) comprendida entre los residuos ala-67 y gln -106 de la subunidad A de la DNA girasa, ya que algunas mutaciones puntuales en GyrA de la DNA-girasa causan elevada resistencia.

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Books on the topic "Serratia marcescens"

1

Jessop, Helen Lavinia. The immunochemistry of serratia marcescens. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1986.

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Kamarudin, Md Isa. Reservoir and fomites of Serratia marcescens intramammary infection. 1994.

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Poerio, Joseph. Studies on Pigment Fractions Produced by Serratia Marcescens Bizio. Creative Media Partners, LLC, 2021.

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Asokan, Neethu, Jiji K. M, and Hemalatha N. Production of Prodigiosin – Like Pigment From Serratia Marcescens: Optimized Media and Oil Refinery Effluent Formulated Media. LAP LAMBERT Academic Publishing, 2019.

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Book chapters on the topic "Serratia marcescens"

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Schomburg, Dietmar, and Margit Salzmann. "Serratia marcescens nuclease." In Enzyme Handbook 3, 937–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76463-9_196.

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Quirós, C., L. A. García, and M. Díaz. "Modeling Protease Production by Immobilised Serratia marcescens." In Advances in Bioprocess Engineering, 227–32. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-017-0641-4_32.

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Györffy, B., and I. Kállay. "Development of Resistance to Streptomycin in Serratia marcescens." In Novartis Foundation Symposia, 233–40. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470719053.ch13.

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Ghigo, J. M., S. Létoffé, P. Delepelaire, and C. Wandersman. "Protease secretion by Erwinia chrysanthemi and Serratia marcescens." In Biological Membranes: Structure, Biogenesis and Dynamics, 215–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78846-8_21.

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Fuchs, R. L., S. A. McPherson, and D. J. Drahos. "Cloning and Expression of a Serratia Marcescens Gene Encoding Chitinase." In Plant Pathogenic Bacteria, 478. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3555-6_101.

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Shibatani, Takeji, Hiroaki Matsumae, and Tetsuya Tosa. "Asymmetric Hydrolysis of Phenylglycidate Ester by Esterase from Serratia marcescens." In Biochemical Engineering for 2001, 92–95. Tokyo: Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-68180-9_22.

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Komatsubara, Saburo, Naoki Sakurai, Yuji Imai, Makoto Masuda, and Tetsuya Tosa. "Molecular Breeding of a Biotin-Hyperproducing Strain of Serratia marcescens." In Biochemical Engineering for 2001, 170–72. Tokyo: Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-68180-9_43.

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Ahmad, Wan Azlina, Wan Yunus Wan Ahmad, Zainul Akmar Zakaria, and Nur Zulaikha Yusof. "Optimization of Pigment Production: Case of Chromobacterium violaceum and Serratia marcescens." In SpringerBriefs in Molecular Science, 45–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-24520-6_3.

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Ferraz, Cristina, Álvaro A. De Araújo, and Gláucia M. Pastore. "The Influence of Vegetable Oils on Biosurfactant Production by Serratia marcescens." In Biotechnology for Fuels and Chemicals, 841–47. Totowa, NJ: Humana Press, 2002. http://dx.doi.org/10.1007/978-1-4612-0119-9_68.

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Braun, Volkmar, and Ralf Hertle. "Structure, Activity, Activation, and Secretion of the Serratia marcescens Hemolysin/Cytolysin." In Microbial Fundamentals of Biotechnology, 222–37. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2005. http://dx.doi.org/10.1002/3527602720.ch13.

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Conference papers on the topic "Serratia marcescens"

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Erat, Tuğba, Aysun Yahši, Tuğ;çe Tural Kara, Halil Özdemir, Ebru Azapağasi, Oktay Perk, Tanil Kendirli, et al. "P258 Bloodstream infections caused by serratia marcescens in paediatric patients." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.346.

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Çakmak, Ayşe İdil, Meryem Çetin, and Özgen Köseoğlu Eser. "A Case of Bilateral Congenital Dacryocystocele Infected with Serratia marcescens." In 4th International Symposium on Innovative Approaches in Health and Sports Sciences. SETSCI, 2019. http://dx.doi.org/10.36287/setsci.4.9.064.

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Seah, Siew-Wei, Sheila Nathan, and Kiew-Lian Wan. "Toxicity evaluation of prodigiosin from Serratia marcescens in a Caenorhabditis elegans model." In THE 2016 UKM FST POSTGRADUATE COLLOQUIUM: Proceedings of the Universiti Kebangsaan Malaysia, Faculty of Science and Technology 2016 Postgraduate Colloquium. Author(s), 2016. http://dx.doi.org/10.1063/1.4966725.

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Iqbal, Faiq, Gires Usup, and Asmat Ahmad. "Anti-biofilm activity of Pseudoalteromonas flavipulchra SktPp1 against Serratia marcescens SMJ-11." In THE 2015 UKM FST POSTGRADUATE COLLOQUIUM: Proceedings of the Universiti Kebangsaan Malaysia, Faculty of Science and Technology 2015 Postgraduate Colloquium. AIP Publishing LLC, 2015. http://dx.doi.org/10.1063/1.4931228.

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Shah, K., M. Subramanian, S. Rashid, and V. Cueto. "A Rare Case of Serratia Marcescens Infective Endocarditis Initially Presenting as Pneumonia." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3968.

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Kalam, S., A. Al-Fahad, H. Simmons, V. Bradshaw, A. Ghareeb, K. Lang Ping Nam, and G. Antunes. "P245 Serratia marcescens (SM): a significant pathogen in the adult bronchiectasis microbiome?" In British Thoracic Society Winter Meeting 2019, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 4 to 6 December 2019, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2019. http://dx.doi.org/10.1136/thorax-2019-btsabstracts2019.388.

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Steager, Edward, Jigarkumar Patel, Chang-Beom Kim, Socheth Bith, Chandan Naik, Lindsay Reber, and Min Jun Kim. "Self-Powered Bacterial Transportation Systems in Low Reynolds Number Fluidic Environments." In ASME 2007 International Mechanical Engineering Congress and Exposition. ASMEDC, 2007. http://dx.doi.org/10.1115/imece2007-41198.

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Abstract:

We have studied self-coordinated bacterial transportation systems using SU-8 microbarges in low Reynolds number fluidic environments. Flagellated Serratia marcescens bacteria were attached to microbarges using a blotting technique that creates a bacterial monolayer carpet. These bacterial carpets naturally self-coordinate to propel the microbarges in a manner that have been controlled by phototactic means. We additionally demonstrate phototactic control of these barges. Tracking algorithms were designed to study the motion of the microbarges.

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Lester, Adrienne C., Audrey A. Trotman, and Ramble O. Ankumah. "A Intermediate Products During the Aerobic Degradation of Sweetpotato Plants by Serratia Marcescens." In International Conference On Environmental Systems. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2003. http://dx.doi.org/10.4271/2003-01-2684.

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Vinogradov, Evgeny, Buko Lindner, Guntram Seltmann, Joanna Radziejewska-Lebrecht, and Otto Holst. "THE STRUCTURES OF THE CORE-LIPID A REGIONS OF LIPOPOLYSACCHARIDES FROM SERRATIA MARCESCENS." In XXIst International Carbohydrate Symposium 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.692.

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Andreia dos Santos, Renata, Lucas Albuquerque Rosendo, Dayana Montero Rodríguez, Galba Maria de Campos Takaki, and Marcos Antônio Barbosa de Lima. "PRODUÇÃO SUSTENTÁVEL DE PRODIGIOSINA E BIOSSURFACTANTE POR Serratia marcescens UCP 1549 EM ESTADO SÓLIDO." In XVII Encontro Nacional de Estudantes de Engenharia Ambiental e V Fórum Latino-Americano de Engenharia e Sustentabilidade. Recife, Brasil: Even3, 2019. http://dx.doi.org/10.29327/15304.17-160771.

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Reports on the topic "Serratia marcescens"

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Iglesias, Gabrielle. Serratia Marcescens Mutations in the Elevated Ultraviolet Radiation Conditions of the Stratosphere. Ames (Iowa): Iowa State University. Library. Digital Press, January 2011. http://dx.doi.org/10.31274/ahac.8129.

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