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Books on the topic 'Sensory and motor disturbances'

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Published: 4 June 2021
Last updated: 20 February 2023

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1

R, Benecke, Conrad B, and Marsden C. David, eds. Motor disturbances 1. London: Academic, 1987.

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2

Sensory-motor integration activities. Tucson, Ariz: Therapy Skill Builders, 1989.

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3

Drost, Ulrich C. Sensory-motor coupling in musicians. Göttingen: Cuvillier Verlag, 2005.

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4

Sensory motor issues in autism. San Antonio, Tex: Therapy Skill Builders, 1998.

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5

Hong, Chia Swee, and Heidi Rumford. Sensory Motor Activities for Early Development. Second edition. | Abingdon, Oxon ; New York : Routledge, 2020.: Routledge, 2020. http://dx.doi.org/10.4324/9780429299735.

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6

Struppler, Albrecht, and Adolf Weindl, eds. Clinical Aspects of Sensory Motor Integration. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71540-2.

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7

Grossberg, Stephen. Neural dynamics of adaptive sensory-motor control. New York: Pergamon Press, 1989.

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8

Hilbig, Reinhard, Albert Gollhofer, Otmar Bock, and Dietrich Manzey. Sensory Motor and Behavioral Research in Space. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-68201-3.

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9

Darcy, Myers, ed. Sensory integration: Practical strategies and sensory motor activities for use in the classroom. Grand Rapids, Mich: LDA, 2002.

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10

Hodapp, Robert M. Development and disabilities: Intellectual, sensory, and motor impairments. Cambridge: Cambridge University Press, 1998.

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11

Jones, Edward G., and Alan Peters, eds. Sensory-Motor Areas and Aspects of Cortical Connectivity. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2149-1.

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12

Cunic, Danny. Discrimination of motor and sensory processing in human EEG. Ottawa: National Library of Canada, 2000.

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13

Chapin, John K., Ph. D. and Moxon Karen A, eds. Neural prostheses for restoration of sensory and motor function. Boca Raton: CRC Press, 2001.

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14

Kratz, Laura E. Movement and fundamental motor skills for sensory deprived children. Springfield, Ill., U.S.A: Thomas, 1987.

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15

Bessière, Pierre, Christian Laugier, and Roland Siegwart, eds. Probabilistic Reasoning and Decision Making in Sensory-Motor Systems. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-79007-5.

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16

Pierre, Bessière, Laugier Christian, and Siegwart Roland, eds. Probabilistic reasoning and decision making in sensory-motor systems. Berlin: Springer, 2008.

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17

Murray, E. Lee, and Veda V. Vedanarayanan. Motor and Sensory Disturbance. Edited by Karl E. Misulis and E. Lee Murray. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190259419.003.0004.

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Localization and characterization of motor and sensory deficits is often the first step in establishing a differential diagnosis of a neurologic presentation. This chapter discusses motor and sensory functions and deficits individually and collectively. The emphasis is on disorders commonly encountered in hospital practice.
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18

Valls-Solé, Josep. Reflex studies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0010.

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Reflex studies are an important part of clinical neurophysiology assessment in health and disease. They are essential to get information on conduction in proximal segments of peripheral nerves, spinal and supraspinal integration of sensory inputs on the motor pathway, and excitability of motor structures. They do not require special equipment, except for a sweep-triggering hammer that is essential, for instance, to elicit monosynaptic reflexes, such as the jaw jerk. For consensual reflexes, it is also recommended to use two recording channels, which facilitate recognition of potential disturbances in the afferent or efferent path of the reflex. What follows is a review of some of the most relevant reflexes that can be studied for neurophysiology assessment in clinical practice.
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19

Aminoff, Michael J. Clinical Observations on the Nervous System. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190614966.003.0009.

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With his remarkable knowledge of anatomy and his analytical mind, Bell developed into an outstanding clinical neurologist even before the specialty had been invented. Thus it was that in his later years, when finally he returned to the University of Edinburgh as professor of surgery, referrals and requests for consultation were often for him to provide a neurological opinion rather than to perform surgical operations. His clinical observations regarding motor or sensory disturbances involving the face are of particular interest given his interests in the facial expression of emotions and the innervation of the face. Bell described or elaborated on several clinical disorders, although not always recognizing them as distinct entities—Bell’s palsy and spasms; the numb chin; motor neuron diseases; muscular dystrophy; myotonia; various movement disorders (torticollis and writer’s cramp); atlantoaxial dislocation; trigeminal neuralgia; and referred pain.
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20

Payne, Russell A., and Kimberly S. Harbaugh. Median Neuropathy—Pronator Teres Syndrome and Anterior Interosseous Neuropathy. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0003.

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Pronator teres syndrome results from median nerve compression or irritation at the elbow region. Patients typically note volar forearm pain and median sensory disturbance that includes the palm. Electrodiagnostic studies are helpful in excluding carpal tunnel syndrome and cervical radiculopathy, and findings may be normal in pronator syndrome. A lack of sensory findings and motor loss in flexion of the distal phalanx of the radial three digits suggests anterior interosseous nerve palsy, typically due to neuralgic amyotrophy. When conservative treatment fails, surgical release of all potential points of compression is successful in alleviating symptoms in the majority of patients with pronator syndrome.
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21

Monico, Caro. Complex Regional Pain Syndrome for Ambulatory Surgery. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0055.

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Complex regional pain syndrome (CRPS) is a disease of the nervous system characterized by pain localized in an extremity. This pain is typically out of proportion to the inciting event and is accompanied by sensory disturbances, as well as motor, vasomotor, and sudomotor signs and symptoms. CRPS is a challenging clinical presentation and diagnosis. The etiology of this previously rare condition in children, is typically post-traumatic. It’s management requires a biopsychosocial approach. The principal modality that will improve pain and function in children with CRPS is physical therapy together with an interdisciplinary approach to management. The key to successful treatment involves early appropriate intervention, education for the child and family, and excellent communication between team members. This chapter uses a case study of a 12-year-old girl with CRPS to illustrate these concepts.
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22

Motor Disturbances II Apl. Elsevier, 1990. http://dx.doi.org/10.1016/c2012-0-01438-x.

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23

Peñagarikano, Olga, and Daniel H. Geschwind. CNTNAP2 and Autism Spectrum Disorders. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199744312.003.0016.

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Although autism was described in the early 1940s as a disorder of affective contact (Kanner, 1943), it was not classified as a neurodevelopmental disorder with a biological basis until the early 1980s, when studies reported its high heritability (Folstein & Rutter, 1977; Ritvo et al., 1985) and co-occurrence with chromosomal abnormalities (Gillberg & Wahlstrom, 1985; Wahlström et al., 1986). Today, autism is considered a heterogeneous neurodevelopmental syndrome and therefore termed autism spectrum disorder (ASD), characterized by variable deficits in social behavior and language, restrictive interests, and repetitive behaviors. Autism spectrum disorder has an estimated prevalence of 1:150–1:200 (Centers for Disease Control and Prevention, 2007), being one of the most common childhood disorders. In addition to the core domains necessary for diagnosis, a number of other behavioral abnormalities are frequently associated with ASD, including epilepsy, sensory abnormalities, hyperactivity, motor abnormalities, sleep disturbances, and gastrointestinal symptoms (Geschwind, 2009).
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24

Fink, Ba. Sensory Motor Integration Activities. Therapy Skill Builders, 1989.

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25

*. Binder/Sensory Motor Integration. Elsevier, 1995.

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26

Brealey, David, and Nicholas Hirsch. Diagnosis, assessment, and management of Guillain–Barré syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0246.

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The Guillain–Barré Syndrome describes a spectrum of acute inflammatory polyneuropathies and is the commonest cause of acute flaccid paralysis within the western world. The pathophysiology is complex and poorly understood, but appears to be an immune-mediated destruction of either the myelin sheath and/or the axons, predominantly of motor nerves. The clinical presentation is classically a rapid, ascending, flaccid paralysis, with minimal sensory deficit. This may ascend to involve respiratory or bulbar muscle function. These patients need careful monitoring and, if deteriorating, should be electively intubated and ventilated. Autonomic instability and sensory disturbance, including pain, is common. Treatment of the underlying condition relies upon immunomodulation with either intravenous immunoglobulin or plasma exchange. Supportive care is aimed at maintaining a safe airway, ventilatory support, and managing the complications of autonomic dysfunction and prolonged immobility. Mortality rates range up to 20%, but are significantly better in specialist neuromedical units. Survivors are often left with significant disability.
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27

Bates, David. Cauda equina lesions, radiculopathies, and sphincter disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0678.

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Pathological processes involving the spinal roots and cauda equina present with symptoms of lower motor neurone and first order sensory neurone damage. Pain is a common, though not inevitable, symptom. Pathological processes may be acute, as with a prolapsed intervertebral disc or chronic and extend over many years, as with spondylotic bony changes or structural diseases such as spondylolisthesis. The cauda equina carries innervation to the bladder, rectum, corpus cavernosum, and seminal vesicles and damage commonly presents with sphincter disturbance and impotence. In general the nerve roots throughout the spine and cauda equina are more resistant to injury and pathological processes than the spinal cord; rapid diagnosis and surgical intervention where indicated, may improve outcome considerably.
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28

Erfle, Stacie. Skill Builder ABC: Fine Motor, Sensory, Gross Motor. Skill Builder Books, 2019.

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29

Bates, David. Spinal cord disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0650.

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Non-traumatic spinal cord disease may be caused by compression due to tumour, infection or haematoma, inflammation, infection or post-infection, metabolic disturbances, infarction, and degeneration. The diagnosis is often made easier by the clinical assessment: the patient’s age, the speed of onset of the disease, severity of the deficits, the pattern of motor and sensory involvement, and presence of pain and sphincter symptoms are all important in making an assessment of the site and likely nature of the spinal disease.Investigations are obligatory to confirm a diagnosis and to direct therapy. MRI is the most useful investigation. It has largely replaced myelography which should now only be considered in patients with indwelling cardiac pacing wires. Additional investigations including examination of the cerebrospinal fluid, evoked potentials, and specific blood tests may be required and the value of plain X-rays, CT scan, and, in some instances, angiography should not be overlooked.The remainder of this chapter will consider specific disorders, identifying pathology, clinical presentation, investigation, and management. Acute and chronic conditions are considered separately and those affecting the cauda equina, spinal root, and sphincters are considered in Chapter 29.
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30

Camilleri, Michael. Mayo Clinic Illustrated Textbook of Neurogastroenterology. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197512104.001.0001.

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Mayo Clinic Illustrated Textbook of Neurogastroenterology reflects experience covering the spectrum and maturation of the field of gastrointestinal motility— from studies of exotic patterns of myoelectric recordings in research laboratories to a clinical discipline with major advances in the clinical management of thousands of patients seen by gastroenterologists, primary care physicians, surgeons, or neurologists. Development of the specialty has been characterized by advances in imaging, developments of methods to measure diverse motor and sensory functions from the stomach to the anorectum, introduction of genetic testing, and a plethora of novel pharmacologic and interventional therapies that have revolutionized its practice. The book covers the spectrum of neurogastroenterologic disorders: from those that are associated with genetic and molecular disorders, through disturbances of the extrinsic neural control or the enteric neuromuscular apparatus, to dysfunction associated with disorders of the gut-brain axis; it reflects a desire to pass on clinical and mechanistic insights and advances in therapies that are relevant to a diverse spectrum of clinicians or clinicians in training who care for the estimated 40% of patients presenting to gastroenterologists with symptoms suggestive of disorders of stomach, intestinal, colonic, or anorectal function.
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31

Sensory Circuits A Sensory Motor Skills Programme For Children. LDA, 2009.

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32

J, Glencross Denis, Piek Jan P, and Motor Control & Human Skill Research Workshop (2nd : 1993 : Mandurah, W.A.), eds. Motor control and sensory motor integration: Issues and directions. Amsterdam: Elsevier, 1995.

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33

Glencross, D. J., and J. P. Piek. Motor Control and Sensory-Motor Integration: Issues and Directions. Elsevier Science & Technology Books, 1995.

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34

The Sensory Connection. Sensory Resources, 2005.

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35

Clinical Aspects of Sensory Motor Integration. Springer, 2011.

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36

HONG. Sensory Motor Activities F/early Developmt. Not Avail, 1996.

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37

A, Berthoz, Graf, Werner, 1948 Oct. 10-, and Vidal Pierre Paul, eds. The Head-neck sensory motor system. New York: Oxford University Press, 1992.

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38

Struppler, Albrecht, and Adolf Weindl. Clinical Aspects of Sensory Motor Integration. Springer, 2011.

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39

*. Video W/Label-Sensory Motor Integration. Elsevier, 1995.

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40

Struppler, Albrecht, and Adolf Weindl. Clinical Aspects of Sensory Motor Integration. Springer London, Limited, 2013.

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41

Gopnik, Myrna, and Eric Keller. Motor and Sensory Processes of Language. Taylor & Francis Group, 2013.

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42

Gopnik, Myrna, and Eric Keller. Motor and Sensory Processes of Language. Taylor & Francis Group, 2013.

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43

Berthoz, Alain, Werner Graf, and P. P. Vidal, eds. The Head-Neck Sensory Motor System. Oxford University Press, 1992. http://dx.doi.org/10.1093/acprof:oso/9780195068207.001.0001.

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44

Hong, Chia Swee, Helen Gabriel, and Cathy St John. Sensory Motor Activities for Early Development. Routledge, 2018. http://dx.doi.org/10.4324/9781315171180.

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45

Eric, Keller, and Gopnik Myrna, eds. Motor and sensory processes of language. Hillsdale, N.J: L. Erlbaum Associates, 1987.

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46

1919-, Struppler A., and Weindl A, eds. Clinical aspects of sensory motor integration. Berlin: Springer-Verlag, 1987.

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47

Sensory Motor Activities for Early Development. Speechmark Publishing Ltd, 1997.

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48

Shergold, Carol. Sensory-motor coordination: Adapting to disruptions. 2001.

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49

Seaman, Janet A. Sensory Motor Experiences for the Home. Amer Alliance for Health Physical, 1995.

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50

Keller, Eric. Motor and Sensory Processes of Language. Psychology Press, 2013. http://dx.doi.org/10.4324/9780203767702.

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