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Journal articles on the topic 'SENSORIMOTORIA'

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Published: 9 March 2023

Last updated: 10 March 2023

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1

Chiera, Marco. "Cura manuale integrata nella malattia di Parkinson." PNEI REVIEW, no. 2 (November 2022): 45–56. http://dx.doi.org/10.3280/pnei2022-002005.

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La malattia di Parkinson è sempre stata considerata come squisitamente neurologica e caratterizzata da neurodegenerazione per l'accumulo della proteina a-sinucleina nella substantia nigra. Tuttavia, diversi studi mostrano come lo stato di salute dell'intero organismo possa influenzare il processo di accumulo dell'a-sinucleina tramite processi bottom-up, fra cui la neuroinfiammazione. Inoltre, che il corpo sia così centrale nel curare persone con Parkinson è mostrato anche dalle ricerche sull'interocezione, ovvero quel processo tramite cui l'organismo percepisce cosa sta accadendo al suo in- terno al fine di meglio rispondere alle sfide ambientali. In caso di Parkinson, questo processo risulta alterato con conseguenze negativa sulla sensomotricità. A tal proposito, la letteratura scientifica mostra molteplici vie per agire sui processi di regolazione biologica in caso di malattia di Parkinson, e fra queste un ruolo importante lo giocano l'educazione sensorimotoria e le terapie manuali, le quali hanno la possibilità di agire sulle vie interocettive e sull'equilibrare i livelli di infiammazione sistemica, in particolare intestinale.

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2

Zech, A., and M. Hübscher. "Sensomotorisches Training zur Prävention von Sprunggelenksverletzungen." Deutsche Zeitschrift für Sportmedizin 2012, no. 01 (January 1, 2012): 5–8. http://dx.doi.org/10.5960/dzsm.2011.060.

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Гершкович, В. А., and Д. К. Урих. "Эффект «проигрыша» при выполнении простой сенсомоторной задачи в ситуации соревнования." Психология. Журнал Высшей школы экономики 14, no. 1 (March 30, 2017): 178–88. http://dx.doi.org/10.17323/1813-8918-2017-1-178-188.

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4

Choi, Julia T., Eileen P. G. Vining, Susumu Mori, and Amy J. Bastian. "Sensorimotor function and sensorimotor tracts after hemispherectomy." Neuropsychologia 48, no. 5 (April 2010): 1192–99. http://dx.doi.org/10.1016/j.neuropsychologia.2009.12.013.

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5

Greier, K., and L. Ressle. "Sensomotorische Feedbackleistung bei adipösen und normalgewichtigen 11- 15-jährigen Schülerinnen und Schülern." Deutsche Zeitschrift für Sportmedizin 2012, no. 02 (February 1, 2012): 36–40. http://dx.doi.org/10.5960/dzsm.2011.063.

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6

Della-Maggiore, Valeria, Sofia M. Landi, and Jorge I. Villalta. "Sensorimotor Adaptation." Neuroscientist 21, no. 2 (August 13, 2014): 109–25. http://dx.doi.org/10.1177/1073858414545228.

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7

Clark, Andy, and Josefa Toribio. "Sensorimotor chauvinism?" Behavioral and Brain Sciences 24, no. 5 (October 2001): 979–80. http://dx.doi.org/10.1017/s0140525x01290116.

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While applauding the bulk of the account on offer, we question one apparent implication, namely, that every difference in sensorimotor contingencies corresponds to a difference in conscious visual experience.

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8

KURITA, Yuichi, Yuki KOIKE, Takayuki TANAKA, and Toshio TSUJI. "SEnS: Sensorimotor Enhancing Suit that Improves Sensorimotor Performance." Proceedings of JSME annual Conference on Robotics and Mechatronics (Robomec) 2016 (2016): 1A1–02b4. http://dx.doi.org/10.1299/jsmermd.2016.1a1-02b4.

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9

Ibrahim, Abeer R., Azza M. Atya, and Al Shimaa R. Azab. "INFLUENCE OF SENSORIMOTOR TRAINING ON BALANCE AND PAIN PARAMETERS IN CHILDREN WITH HEMOPHILIA." International Journal of Physiotherapy and Research 5, no. 2 (April 11, 2017): 1912–19. http://dx.doi.org/10.16965/ijpr.2017.101.

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10

Millichap, J. Gordon. "Supplementary Sensorimotor Seizures." Pediatric Neurology Briefs 9, no. 10 (October 1, 1995): 79. http://dx.doi.org/10.15844/pedneurbriefs-9-10-12.

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11

Millichap, J. Gordon. "Supplementary Sensorimotor Seizures." Pediatric Neurology Briefs 9, no. 5 (May 1, 1995): 34. http://dx.doi.org/10.15844/pedneurbriefs-9-5-2.

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12

Overduin, Simon A., and Philip Servos. "Symmetric Sensorimotor Somatotopy." PLoS ONE 3, no. 1 (January 30, 2008): e1505. http://dx.doi.org/10.1371/journal.pone.0001505.

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13

Li, Yuebing. "Axonal Sensorimotor Polyneuropathies." CONTINUUM: Lifelong Learning in Neurology 23, no. 5 (October 2017): 1378–93. http://dx.doi.org/10.1212/con.0000000000000514.

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14

Barker, R. "Supplementary Sensorimotor Area." Journal of Neurology, Neurosurgery & Psychiatry 61, no. 1 (July 1, 1996): 124. http://dx.doi.org/10.1136/jnnp.61.1.124.

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15

Crochet, Sylvain, and Carl C. H. Petersen. "Cortical Sensorimotor Reverberations." Neuron 86, no. 5 (June 2015): 1116–18. http://dx.doi.org/10.1016/j.neuron.2015.05.030.

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16

NEEMAN, RENATE L. "ORTHOKINETIC SENSORIMOTOR TREATMENT." Australian Occupational Therapy Journal 20, no. 3 (August 27, 2010): 122–25. http://dx.doi.org/10.1111/j.1440-1630.1973.tb00643.x.

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17

Rocco, Mary M., and Joshua C. Brumberg. "The sensorimotor slice." Journal of Neuroscience Methods 162, no. 1-2 (May 2007): 139–47. http://dx.doi.org/10.1016/j.jneumeth.2007.01.002.

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18

Su, David K., and Jeffrey G. Ojemann. "Electrocorticographic sensorimotor mapping." Clinical Neurophysiology 124, no. 6 (June 2013): 1044–48. http://dx.doi.org/10.1016/j.clinph.2013.02.114.

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19

Zieliński, Cezary. "Sensorimotor Robot Control." IFAC Proceedings Volumes 28, no. 10 (July 1995): 745–49. http://dx.doi.org/10.1016/s1474-6670(17)51609-2.

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20

Roberts, Tom. "Understanding ‘sensorimotor understanding’." Phenomenology and the Cognitive Sciences 9, no. 1 (March 11, 2009): 101–11. http://dx.doi.org/10.1007/s11097-009-9125-7.

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21

Rescorla, Michael. "Bayesian Sensorimotor Psychology." Mind & Language 31, no. 1 (February 2016): 3–36. http://dx.doi.org/10.1111/mila.12093.

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22

Niedermeyer, E. "Supplementary sensorimotor area." Electroencephalography and Clinical Neurophysiology 99, no. 3 (September 1996): 293. http://dx.doi.org/10.1016/0013-4694(96)85831-7.

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23

DiZio, Paul, and James R. Lackner. "Sensorimotor aspects of high-speed artificial gravity: III. Sensorimotor adaptation." Journal of Vestibular Research 12, no. 5-6 (August 1, 2003): 291–99. http://dx.doi.org/10.3233/ves-2003-125-609.

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As a countermeasure to the debilitating physiological effects of weightlessness, astronauts could live continuously in an artificial gravity environment created by slow rotation of an entire spacecraft or be exposed to brief daily "doses" in a short radius centrifuge housed within a non-rotating spacecraft. A potential drawback to both approaches is that head movements made during rotation may be disorienting and nauseogenic. These side effects are more severe at higher rotation rates, especially upon first exposure. Head movements during rotation generate aberrant vestibular stimulation and Coriolis force perturbations of the head-neck motor system. This article reviews our progress toward distinguishing vestibular and motor factors in side effects of rotation, and presents new data concerning the rates of rotation up to which adaptation is possible. We have studied subjects pointing to targets during constant velocity rotation, because these movements generate Coriolis motor perturbations of the arm but do not involve unusual vestibular stimulation. Initially, reaching paths and endpoints are deviated in the direction of the transient lateral Coriolis forces generated. With practice, subjects soon move in straighter paths and land on target once more. If sight of the arm is permitted, adaptation is more rapid than in darkness. Initial arm movement trajectory and endpoint deviations are proportional to Coriolis force magnitude over a range of rotation speeds from 5 to 20 rpm, and there is rapid, complete motor adaptation at all speeds. These new results indicate that motor adaptation to high rotation rates is possible. Coriolis force perturbations of head movements also occur in a rotating environment but adaptation gradually develops over the course of many head movements.

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24

Harel, Hani, Idit Lavi, Raviv Allon, Dafna Michael, and Ronit Wollstein. "Postoperative Treatment of Distal Radius Fractures Using Sensorimotor Rehabilitation." Journal of Wrist Surgery 08, no. 01 (September 27, 2018): 002–9. http://dx.doi.org/10.1055/s-0038-1672151.

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Background Sensorimotor and specifically proprioception sense has been used in rehabilitation to treat neurological and joint injuries. These feedback loops are not well understood or implemented in wrist treatment. We observed a temporary sensorimotor loss, following distal radius fractures (DRF) that should be addressed postsurgery. Purpose The purpose of this prospective therapeutic study was to compare the outcomes of patients following surgery for DRF treated using a sensorimotor treatment protocol with those patients treated according to the postoperative standard of care. Patients and Methods Patients following surgery for DRF sent for hand therapy were eligible for the study. Both the evaluation and treatment protocols included a comprehensive sensorimotor panel. Patients were randomized into standard and standard plus sensorimotor postoperative therapy and were evaluated a few days following surgery, at 6 weeks, and 3 months postsurgery. Results Sixty patients following surgery were randomized into the two treatment regimens. The initial evaluation was similar for both groups and both demonstrated significant sensorimotor deficits, following surgery for DRF. There was documented sensorimotor and functional improvement in both groups with treatment. The clinical results were better in the group treated with the sensorimotor-proprioception protocol mostly in the wrist; however, not all of the differences were significant. Conclusion Patients after surgery for DRF demonstrate significant sensorimotor deficits which may improve faster when utilizing a comprehensive sensorimotor treatment protocol. However, we did not demonstrate efficacy of the protocol in treating proprioceptive deficits. Further study should include refinement of functional outcome evaluation, studying of the treatment protocol, and establishment of sensorimotor therapeutic guidelines for other conditions. Level of Evidence This is a level II, therapeutic study.

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Salomon, Roy, Pierre Progin, Alessandra Griffa, Giulio Rognini, Kim Q. Do, Philippe Conus, Silvia Marchesotti, et al. "Sensorimotor Induction of Auditory Misattribution in Early Psychosis." Schizophrenia Bulletin 46, no. 4 (February 11, 2020): 947–54. http://dx.doi.org/10.1093/schbul/sbz136.

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Abstract Dysfunction of sensorimotor predictive processing is thought to underlie abnormalities in self-monitoring producing passivity symptoms in psychosis. Experimentally induced sensorimotor conflict can produce a failure in bodily self-monitoring (presence hallucination [PH]), yet it is unclear how this is related to auditory self-monitoring and psychosis symptoms. Here we show that the induction of sensorimotor conflict in early psychosis patients induces PH and impacts auditory-verbal self-monitoring. Participants manipulated a haptic robotic system inducing a bodily sensorimotor conflict. In experiment 1, the PH was measured. In experiment 2, an auditory-verbal self-monitoring task was performed during the conflict. Fifty-one participants (31 early psychosis patients, 20 matched controls) participated in the experiments. The PH was present in all participants. Psychosis patients with passivity experiences (PE+) had reduced accuracy in auditory-verbal self-other discrimination during sensorimotor stimulation, but only when sensorimotor stimulation involved a spatiotemporal conflict (F(2, 44) = 6.68, P = .002). These results show a strong link between robotically controlled alterations in sensorimotor processing and auditory misattribution in psychosis and provide evidence for the role of sensorimotor processes in altered self-monitoring in psychosis.

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Clavel Vázquez, María Jimena. "A match made in heaven: predictive approaches to (an unorthodox) sensorimotor enactivism." Phenomenology and the Cognitive Sciences 19, no. 4 (December 20, 2019): 653–84. http://dx.doi.org/10.1007/s11097-019-09647-0.

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AbstractIt has been pointed out that Sensorimotor Enactivism, a theory that claims that perception is enacted and brought about by movement, says very little about the neural mechanisms that enable perception. For the proponents of the predictive approach to Sensorimotor Enactivism, this is a challenge that can be met by introducing predictive processing into the picture. However, the compatibility between these theories is not straightforward. Firstly, because they seem to differ in their stand towards representations: while Sensorimotor Enactivism is said to belong to the non-representational wing of cognitive science, predictive processing has a representational profile. And secondly, because they exhibit different explanatory strategies: while Sensorimotor Enactivism prioritizes the interactions of the embodied agent, predictive processing has internalist commitments. The aim of this paper is to address these concerns and show that a predictive approach to Sensorimotor Enactivism is viable. More specifically, I focus on the Free-Energy approach, a theory that falls within the ballpark of predictive processing. In this paper I argue for the following claims. I argue that (a) both Sensorimotor Enactivism and the Free-Energy approach may be understood for some systems in representational terms. The non-representational reading of Sensorimotor Enactivism is not mandatory and neither is the representational reading of the Free-Energy approach. (b) Sensorimotor Enactivism is, in this respect, compatible with both representational and non-representational interpretations of the FEA. So, the position towards representations of these frameworks should not stand in the way of a predictive approach to Sensorimotor Enactivism. I also show that (c) the Free-Energy approach allows for an account that prioritizes the interaction of the embodied agent with the environment. This is the explanatory strategy followed by Sensorimotor Enactivism. To justify this strategy and following other proponents of Sensorimotor Enactivism, I argue that by referring to the interactions of the embodied agent a better account of the phenomena in question is provided. On this basis, I claim that (d) Sensorimotor Enactivism and the Free-Energy approach are compatible in what concerns their explanatory strategy as well. Thus, making the case for the viability of the predictive approach to Sensorimotor Enactivism.

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27

Avivi-Arber, Limor, Ruth Martin, Jye-Chang Lee, and Barry J. Sessle. "Face sensorimotor cortex and its neuroplasticity related to orofacial sensorimotor functions." Archives of Oral Biology 56, no. 12 (December 2011): 1440–65. http://dx.doi.org/10.1016/j.archoralbio.2011.04.005.

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28

Petruševičienė, Daiva, Raimondas Savickas, and Aleksandras Kriščiūnas. "Evaluation of sensorimotor reactions during early rehabilitation for patients after cerebral stroke." Medicina 43, no. 12 (December 11, 2007): 942. http://dx.doi.org/10.3390/medicina43120122.

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Early inpatient rehabilitation is extremely important in functional improvement of patients suffering from cerebral stroke. From our point of view, in rehabilitation of patients after cerebral stroke, the estimation of sensorimotor reactions that enables the evaluation of sensorimotor functional changes is highly relevant. The article describes the comparison of sensorimotor reactions in two subgroups – stroke patients and healthy individuals – by applying Sensoneck system. The evaluation was performed before early stage of rehabilitation and thereafter (following early rehabilitation). In order to estimate the correlation between changes in functional independence and sensorimotor reactions, Functional Independence Measure was used. The study revealed that stroke patients had sensorimotor dysfunctions. During early rehabilitation, the quality of motion performance improved slightly, and sensorimotor reactions improved statistically significantly (P<0.05). The relationship between Functional Independence Measure and Sensoneck scores was not significant (P>0.05).

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Pickavance, John P., Oscar T. Giles, J. Ryan Morehead, Faisal Mushtaq, Richard M. Wilkie, and Mark Mon-Williams. "Sensorimotor ability and inhibitory control independently predict attainment in mathematics in children and adolescents." Journal of Neurophysiology 127, no. 4 (April 1, 2022): 1026–39. http://dx.doi.org/10.1152/jn.00365.2021.

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Previous research downplays the role of sensorimotor skills in the development of higher-order cognitive domains such as mathematics: using inadequate sensorimotor measures, differences in “executive function” account for any shared variance. Utilizing a high-resolution, kinematic measure of a sensorimotor skill previously linked to mathematics attainment, we show that inhibitory control alone cannot account for this relationship. The practical implication is that the development of children’s sensorimotor skills must be considered in their intellectual development.

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Wagle Shukla, Aparna, Jill L. Ostrem, David E. Vaillancourt, Robert Chen, Kelly D. Foote, and Michael S. Okun. "Physiological effects of subthalamic nucleus deep brain stimulation surgery in cervical dystonia." Journal of Neurology, Neurosurgery & Psychiatry 89, no. 12 (January 11, 2018): 1296–300. http://dx.doi.org/10.1136/jnnp-2017-317098.

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BackgroundSubthalamic nucleus deep brain stimulation (STN DBS) surgery is clinically effective for treatment of cervical dystonia; however, the underlying physiology has not been examined. We used transcranial magnetic stimulation (TMS) to examine the effects of STN DBS on sensorimotor integration, sensorimotor plasticity and motor cortex excitability, which are identified as the key pathophysiological features underlying dystonia.MethodsTMS paradigms of short latency afferent inhibition (SAI) and long latency afferent inhibition (LAI) were used to examine the sensorimotor integration. Sensorimotor plasticity was measured with paired associative stimulation paradigm, and motor cortex excitability was examined with short interval intracortical inhibition and intracortical facilitation. DBS was turned off and on to record these measures.ResultsSTN DBS modulated SAI and LAI, which correlated well with the acute clinical improvement. While there were no changes seen in the motor cortex excitability, DBS was found to normalise the sensorimotor plasticity; however, there was no clinical correlation.ConclusionModulation of sensorimotor integration is a key contributor to clinical improvement with acute stimulation of STN. Since the motor cortex excitability did not change and the change in sensorimotor plasticity did not correlate with clinical improvement, STN DBS demonstrates restricted effects on the underlying physiology.Clinical trial registrationNCT01671527.

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Roussel, Nathalie Anne, Margot De Kooning, Jo Nijs, Patrick Cras, Kristien Wouters, and Liesbeth Daenen. "The Role of Sensorimotor Incongruence in Pain in Professional Dancers." Motor Control 19, no. 4 (October 2015): 271–88. http://dx.doi.org/10.1123/ijsnem.2013-0074.

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This study evaluated whether dancers with pain experience more sensory changes during an experimentally induced sensorimotor incongruent task and explored the relationship between sensorimotor incongruence and self-reported measures (e.g., Short Form 36-questionnaire (SF-36), psychosocial variables and physical activity). Forty-four dancers were subjected to a bimanual coordination test simulating sensorimotor incongruence (i.e., performing congruent and incongruent arm movements while viewing a whiteboard or mirror) and completed standardized questionnaires. Significantly more dancers experienced sensory changes during the performance of incongruent movements while viewing a mirror (p < .01), but the intensity of the reported sensations was very low. No differences were observed between dancers with and without baseline pain, but significant negative associations were found between sensorimotor incongruence and subscores of the SF-36. Sensorimotor incongruence can provoke small sensory changes in dancers but appears unrelated to baseline pain symptoms. Sensorimotor incongruence appears to be related to quality of life.

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Wollstein, Ronit, Dafna Michael, and Hani Harel. "A Protocol for Evaluation and Rehabilitation of Distal Radius Fractures Using Sensorimotor Input: A Case Series." Journal of Hand Surgery (Asian-Pacific Volume) 22, no. 02 (May 15, 2017): 150–55. http://dx.doi.org/10.1142/s0218810417500174.

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Background: Proprioception and sensorimotor input are used to treat neurological and joint injuries. Following distal radius fractures (DRF) there is a temporary loss of proprioception that should be addressed. We created a protocol for evaluation, and a treatment plan following wrist surgery that is based on proprioceptive and sensorimotor input. We describe a series of patients undergoing surgery for DRF that were evaluated and treated with these protocols. Methods: Both evaluation and treatment protocols included comprehensive sensorimotor procedures performed with eyes open and closed. These included Semmes- Weinstein, static and moving 2-point discrimination, vibration, temperature testing, Moberg pick-up- test, stereognosis and proprioception. Results: A series of twelve patients was evaluated and treated with the protocol following surgical treatment for DRF. Patients demonstrated significant sensorimotor deficits, which improved utilizing the comprehensive sensorimotor treatment protocol. Conclusions: Further study is necessary to validate the results of this pilot series. Use of proprioception and sensorimotor input may improve outcomes of rehabilitation following DRF.

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Balanev, D. Yu, P. R. Tyutyunnikov, and D. A. Kokh. "Human Sensorimotor Activity as a Factor of Cognitive Resource Development." Bulletin of Kemerovo State University 24, no. 6 (December 29, 2022): 752–59. http://dx.doi.org/10.21603/2078-8975-2022-24-6-752-759.

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The cognitive resource phenomenon, its factors, and performance are a relevant topic of Russian psychology. Numerous publications feature the psychological content of the cognitive resource concept and various phenomena as forms of its manifestation. However, domestic psychology sees no cognitive resource potential in sensorimotor activity, nor does it see sensorimotor activity as a factor that facilitates human cognitive resources. The article considers sensorimotor activity as a cognitive resource and describes a transspective analysis of various approaches to the phenomenon of cognitive resource. The authors defined the latter as a complex multi-level construct. Various cognitive resource models proved that sensorimotor activity is a manifestation of the cognitive resource and its integral part. However, the transspective analysis requires further research on sensorimotor activity in the cognitive resource structure.

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Bologna, Matteo, and Giulia Paparella. "Neurodegeneration and Sensorimotor Function." Brain Sciences 10, no. 11 (November 1, 2020): 808. http://dx.doi.org/10.3390/brainsci10110808.

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Sensorimotor integration is an essential function for both motor control and learning. Over recent decades, a growing body of evidence has emerged in support of the role of altered sensorimotor integration in the pathophysiology of various neurological conditions and movement disorders, particularly bradykinesia, tremor, and dystonia. However, the various causes and mechanisms underlying altered sensorimotor integration in movement disorders are still not entirely understood. The lack of complete insight into the pathophysiological role of altered sensorimotor integration in movement disorders is certainly due to the heterogeneity of movement disorders as well as to the variable occurrence of neurodegenerative phenomena, even in idiopathic movement disorders, which contribute to pathophysiology in a complex and often not easily interpretable way. Clarifying the possible relationship between neurodegenerative phenomena and sensorimotor deficits in movement disorders and other neurological conditions may guide the development of a more detailed disease prognosis and lead, perhaps, to the implementation of novel and individualized therapeutic interventions.

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Sedlić, Marija, Gordana Sičaja, Krešimir Luetić, and Hrvoje Budinčević. "Cryoglobulinemia and sensorimotor polyneuropathy." Infektološki glasnik 40, no. 1 (September 17, 2020): 35–37. http://dx.doi.org/10.37797/ig.40.1.5.

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Secondary cryoglobulinemia is the most common extrahepatic manifestation of hepatitis C. There are different genotypes of hepatitis C, and the ones that are most frequently associated with cryoglobulinemia are genotypes 1b and 2a. Cryoglobulinemia affects various organs and can cause vasculitis, arthralgia, skin changes, glomerulonephritis and neurological manifestations, including peripheral neuropathy. The aim of this report was to emphasize the importance of early diagnosis of hepatitis C infection in patients who are presenting with sensomotor polyneuropathy.

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Desrochers, Brunfeldt, Sidiropoulos, and Kagerer. "Sensorimotor Control in Dystonia." Brain Sciences 9, no. 4 (April 11, 2019): 79. http://dx.doi.org/10.3390/brainsci9040079.

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This is an overview of the sensorimotor impairments in dystonia, a syndrome characterized by sustained or intermittent aberrant movement patterns leading to abnormal movements and/or postures with or without a tremulous component. Dystonia can affect the entire body or specific body regions and results from a plethora of etiologies, including subtle changes in gray and white matter in several brain regions. Research over the last 25 years addressing topics of sensorimotor control has shown functional sensorimotor impairments related to sensorimotor integration, timing, oculomotor and head control, as well as upper and lower limb control. In the context of efforts to update the classification of dystonia, sensorimotor research is highly relevant for a better understanding of the underlying pathology, and potential mechanisms contributing to global and regional dysfunction within the central nervous system. This overview of relevant research regarding sensorimotor control in humans with idiopathic dystonia attempts to frame the dysfunction with respect to what is known regarding motor control in patients and healthy individuals. We also highlight promising avenues for the future study of neuromotor control that may help to further elucidate dystonia etiology, pathology, and functional characteristics.

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37

Worobey, John. "Temperament and sensorimotor intelligence†." Early Child Development and Care 28, no. 1 (January 1987): 1–11. http://dx.doi.org/10.1080/0300443870280101.

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Worobey, John. "Temperament and sensorimotor intelligence." Early Child Development and Care 27, no. 1 (1987): 1–11. http://dx.doi.org/10.1080/0300443870280101a.

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39

Uematsu, Sumio, Ronald P. Lesser, and Barry Gordon. "Localization of Sensorimotor Cortex." Neurosurgery 30, no. 6 (June 1, 1992): 904–13. http://dx.doi.org/10.1097/00006123-199206000-00015.

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40

Lust, Carol, and Martha C. Powell. "Sensorimotor Half-Day Camp." Occupational Therapy In Health Care 8, no. 4 (January 1992): 79–92. http://dx.doi.org/10.1080/j003v08n04_06.

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Buch, Vivek, John Frederick Burke, Ashwin G. Ramayya, Cameron Brandon, Eric Hudgins, Andrew Richardson, and Timothy H. Lucas. "210 Human Sensorimotor Electrocorticography." Neurosurgery 63 (August 2016): 182. http://dx.doi.org/10.1227/01.neu.0000489779.53428.e3.

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42

Wolpert, Daniel M. "Computations in Sensorimotor Learning." Cold Spring Harbor Symposia on Quantitative Biology 79 (2014): 93–98. http://dx.doi.org/10.1101/sqb.2014.79.024919.

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43

Sridhar, Kasi S., Andrea B. Cohen, Jocelyn H. Bruce-Gregorios, and D. Ram Ayyar. "Sensorimotor Myeloradiculoneuropathy in Thymoma." American Journal of Clinical Oncology 14, no. 6 (December 1991): 487–91. http://dx.doi.org/10.1097/00000421-199112000-00006.

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Wolpert, Daniel M., Jörn Diedrichsen, and J. Randall Flanagan. "Principles of sensorimotor learning." Nature Reviews Neuroscience 12, no. 12 (October 27, 2011): 739–51. http://dx.doi.org/10.1038/nrn3112.

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Guyton, David L. "Ocular Torsion: Sensorimotor Principles." American Orthoptic Journal 37, no. 1 (January 1987): 13–21. http://dx.doi.org/10.1080/0065955x.1987.11981728.

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46

Lust, Carol, and Martha Powell. "Sensorimotor Half-Day Camp." Occupational Therapy In Health Care 8, no. 4 (March 24, 1993): 79–92. http://dx.doi.org/10.1300/j003v08n04_06.

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Mima, T., T. Nagamine, N. Nishitani, N. Mikuni, A. Ikeda, H. Fukuyama, T. Takigawa, J. Kimura, and H. Shibasaki. "Cortical myoclonus: Sensorimotor hyperexcitability." Neurology 50, no. 4 (April 1, 1998): 933–42. http://dx.doi.org/10.1212/wnl.50.4.933.

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Buch, Ethan R., Sook-Lei Liew, and Leonardo G. Cohen. "Plasticity of Sensorimotor Networks." Neuroscientist 23, no. 2 (July 8, 2016): 185–96. http://dx.doi.org/10.1177/1073858416638641.

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Abstract:

Redundancy is an important feature of the motor system, as abundant degrees of freedom are prominent at every level of organization across the central and peripheral nervous systems, and musculoskeletal system. This basic feature results in a system that is both flexible and robust, and which can be sustainably adapted through plasticity mechanisms in response to intrinsic organismal changes and dynamic environments. While much early work of motor system organization has focused on synaptic-based plasticity processes that are driven via experience, recent investigations of neuron–glia interactions, epigenetic mechanisms and large-scale network dynamics have revealed a plethora of plasticity mechanisms that support motor system organization across multiple, overlapping spatial and temporal scales. Furthermore, an important role of these mechanisms is the regulation of intrinsic variability. Here, we review several of these mechanisms and discuss their potential role in neurorehabilitation.

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Fanselow, Erika E., and Barry W. Connors. "Navigating a Sensorimotor Loop." Neuron 45, no. 3 (February 2005): 329–30. http://dx.doi.org/10.1016/j.neuron.2005.01.022.

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Gambi, Chiara, and Martin J. Pickering. "Sensorimotor communication and language." Physics of Life Reviews 28 (March 2019): 34–35. http://dx.doi.org/10.1016/j.plrev.2019.01.015.

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