Academic literature on the topic 'Sensitisation'

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Journal articles on the topic "Sensitisation"

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Mikubayeva, E. V., N. S. Kobotayeva, and E. E. Sirotkina. "Combined Sensitisation of Benzaldehyde Diphenylhydrazones: Effect of Hydrazone Structure on Sensitization Efficiency." Eurasian Chemico-Technological Journal 6, no. 2 (July 12, 2017): 133. http://dx.doi.org/10.18321/ectj602.

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Injection, spectral and combined (injection-spectral) sensitisations of several benzaldehyde diphenylhydrazones have been studied using the layers of amorphous selenium and complex compounds based on<br />pyril dyes to broaden photosensitivity spectrum of electrophotographic carrier and to study sensitisation mechanism. Two photogeneration mechanisms for charge carriers have been shown to exist at a combined<br />sensitisation, i.e. generation in the injection layer followed by the injection into a transport layer and generation in a transport layer on a dye. The excited dye molecules have been established to create hole strapping sites in a transport layer: it is exhibited in a different nature of photodischarge curves in selenium- and dye-absorbing regions at the negative surface potential. The effect of the substituent in a benzaldehyde fragment on the efficiency of injection, spectral and combined sensitisations of benzaldehyde diphenylhydrazones<br />has been studied. The ionization potentials of hydrazones were determined by the two following methods: according to the charge transfer band of hydrazone-chloranil charge transfer complexes and<br />by means of quantum-chemical calculations of hydrazone molecules to interpret the results obtained. In the course of quantum-chemical calculations the conjugation character was also determined in the molecules of benzaldehyde diphenylhydrazones.
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Warner, J. A., A. C. Jones, E. A. Miles, and J. O. Warner. "Prenatal sensitisation." Pediatric Allergy and Immunology 7, S9 (December 1996): 98–101. http://dx.doi.org/10.1111/j.1399-3038.1996.tb00406.x.

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Galand, E., J. Niézette, and J. Vanderschueren. "Lyoluminescence Sensitisation." Radiation Protection Dosimetry 47, no. 1-4 (May 1, 1993): 603–6. http://dx.doi.org/10.1093/oxfordjournals.rpd.a081817.

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Galand, E., J. Niézette, and J. Vanderschueren. "Lyoluminescence Sensitisation." Radiation Protection Dosimetry 47, no. 1-4 (May 1, 1993): 603–6. http://dx.doi.org/10.1093/rpd/47.1-4.603.

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Safford, B., N. Gilmour, and N. Aptula. "Refinement of the dermal sensitisation threshold for skin sensitisation." Toxicology Letters 196 (July 2010): S103—S104. http://dx.doi.org/10.1016/j.toxlet.2010.03.370.

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Landa, L., K. Šlais, and A. Šulcová. "Impact of Cannabinoid Receptor Ligands on Sensitisation to Methamphetamine Effects on Rat Locomotor Behaviour." Acta Veterinaria Brno 77, no. 2 (2008): 183–91. http://dx.doi.org/10.2754/avb200877020183.

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The repeated administration of various drugs of abuse may lead to a gradually increased behavioural response to these substances, particularly an increase in locomotion and stereotypies may occur. This phenomenon is well known and described as behavioural sensitisation. An increased response to the drug tested, elicited by previous repeated administration of another drug is recognised as cross-sensitisation. Based on our earlier experiences with studies on mice, which confirmed sensitisation to methamphetamine and described cross-sensitisation to methamphetamine after pre-treatment with cannabinoid CB1 receptor agonist, we focused the present study on the use of another typical laboratory animal - the rat. A biological validity of the sensitisation phenomenon was expected to be enhanced if the results of both mouse and rat studies were conformable. Similar investigation in rats brought very similar results to those described earlier in mice. However, at least some interspecies differences were noted in the rat susceptibility to the development of sensitisation to methamphetamine effects. Comparing to mice, it was more demanding to titrate a dose of methamphetamine producing behavioural sensitisation. Furthermore, we were not able to provoke cross-sensitisation by repeated administration of cannabinoid CB1 receptor agonist methanandamide and similarly, we did not demonstrate the suppression of cross-sensitisation in rats that were repeatedly given combined pre-treatment with cannabinoid CB1 receptor antagonist AM 251 and methamphetamine. Finally, unlike mice, an alternative behavioural change was registered after repeated methamphetamine treatment instead: the occurrence of stereotypic behaviour (nose rubbing).
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Elholm, Grethe, Vivi Schlünssen, Gert Doekes, Ioannis Basinas, Anneli Clea Skjelmose Bolund, Charlotte Hjort, Pernille Milvang Grønager, Øyvind Omland, and Torben Sigsgaard. "High exposure to endotoxin in farming is associated with less new-onset pollen sensitisation." Occupational and Environmental Medicine 75, no. 2 (August 23, 2017): 139–47. http://dx.doi.org/10.1136/oemed-2017-104384.

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ObjectivesLittle is known about risk factors for new onset and loss of atopic sensitisation in adulthood. The aim is to examine the longitudinal effect of quantitatively assessed endotoxin exposures on changes in specific allergen sensitisation in young adults.MethodsThe cohort consisted of 1113 young Danish farmers and rural controls, with a mean age of 19 years at baseline. Sensitisation to birch pollen, grass pollen, cat dander and house dust mite was measured by specific IgE levels in serum samples from baseline and at 15 years’ follow-up. Changes in sensitisation were analysed in relation to cumulative endotoxin exposure during follow-up, considering early life farm exposure.ResultsEndotoxin exposure during follow-up was significantly associated with less new onset of specifically grass and birch pollen sensitisation. For the highest versus lowest quartile of cumulative endotoxin exposure, the OR for new-onset IgE sensitisation was 0.35 (0.13–0.91) for birch and 0.14 (0.05–0.50) for grass. On the other hand, loss of pollen sensitisation showed a positive, although mostly non-significant, association with increased levels of endotoxin exposure. Endotoxin exposure was not associated with significant changes in cat dander and house dust mite sensitisation.ConclusionsHigh exposure to endotoxin during young adulthood appears to protect against new onset of pollen sensitisation, independent of childhood farm exposure.
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Ribeiro, J. C., B. Sousa-Pinto, J. Fonseca, S. Caldas Fonseca, and L. M. Cunha. "Edible insects and food safety: allergy." Journal of Insects as Food and Feed 7, no. 5 (August 13, 2021): 833–47. http://dx.doi.org/10.3920/jiff2020.0065.

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Edible insects are a unique food source, requiring extensive allergenic risk assessment before its safe introduction in the food market. In a recent systematic review, crustacean allergic subjects were identified as a risk group due to cross-reactivity mainly mediated by tropomyosin and arginine kinase. Immunologic co-sensitisation to house dust mites (HDM) was also demonstrated, but its clinical significance and molecular mechanisms were unclear. Furthermore, case reports of food allergy to insects were also analysed but lack of contextual information hindered the analysis. The main goal of this review is to provide an update of new information regarding food allergy caused by insects, covering relevant topics considering the guidelines for allergic risk assessment in novel foods. Newly published studies have further confirmed the role of tropomyosin as a cross-reactive allergen between edible insects and crustaceans, although there are some questions regarding the immunoglobulin E (IgE)-reactivity of this allergen in mealworm species. Furthermore, only specific treatments (enzymatic hydrolysis combined with thermal treatments) were able to eliminate IgE-reactivity of edible insects. Primary sensitisation (e.g. to Tenebrio molitor) has also been shown to be an important pathway for the development of food allergies, with responsible allergens being dependent on the route of sensitisation. However, more studies are necessary to better understand the potential of primary sensitisation causing cross-reactivity with other insect species, crustaceans or HDM. The clinical significance and molecular mechanisms involved in cross-reactivity between edible insects and HDM are still unclear, and a major focus should be given to better understand which allergens cause co-sensitisations between HDM and edible insects and what is the risk of HDM-only allergic subjects consuming edible insects. Contextual information about the reported cases of allergic reactions to insects have further demonstrated that insect-rearing workers and subjects with allergic diseases (in particular, food allergy to crustaceans) are the major risk groups.
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Čelakovská, Jarmila, Květuše Ettlerová, Karel Ettler, and Josef Bukač. "Egg Allergy in Adolescent and Adult Patient Suffering from Atopic Dermatitis – Association with Concomitant Allergic Diseases." Acta Medica (Hradec Kralove, Czech Republic) 58, no. 1 (2015): 9–14. http://dx.doi.org/10.14712/18059694.2015.85.

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Background: A few reports demonstrate the occurrence of egg allergy in adolescent and adult patients suffering from atopic dermatitis and the association of this allergy to other food and aeroallergens. Aims and Objectives: The aim of this study is to evaluate the occurrence of egg allergy in patients suffering from atopic dermatitis at the age 14 years and older and to evaluate the relationship between egg allergy or egg sensitisation and the sensitisation to dust, mites, feather, and animal dander. Materials and Methods: Complete dermatological and allergological examination was performed. These parameters were examined: food allergy and food sensitisation to egg white and yolk, to mites, animal dander (mixture), feather and dust. The statistical evaluation of the relations among egg allergy, egg sensitisation and sensitisation to mites, animal dander (mixture), feather and dust was performed. Two hundred and eighty eight patients were included in the study (90 men, 198 women, with the average age 25.2). Results and Conclusion: Egg allergy was recorded in 5% and egg sensitisation in 20% of patients; sensitisation to dust is recorded more often in patients with positive results in sIgE for egg white and/or yolk.
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Dooms-Goossens, An. "Sensitisation to Corticosteroids." Drug Safety 13, no. 2 (August 1995): 123–29. http://dx.doi.org/10.2165/00002018-199513020-00006.

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Dissertations / Theses on the topic "Sensitisation"

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Oostingh, Gertie Janneke. "Human sensitisation to porcine transplantation antigens." Thesis, Open University, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.272953.

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Patel, Manisha. "Singlet oxygen sensitisation in supercritical fluids." Thesis, Loughborough University, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.418373.

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Green, Brett James. "Detection and diagnosis of fungal allergic sensitisation." University of Sydney, 2005. http://hdl.handle.net/2123/978.

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Doctor of Philosophy(PhD),
Airborne fungi are ubiquitous in the environment and human exposure is inevitable. Such fungi differ greatly in their taxonomic, physical, ecological and pathogenic characteristics. Currently, 69 000 species have been taxonomically classified and more than 80 of these are recognised to be aeroallergen sources. Many strategies have evolved to sample, identify and interpret fungal exposure to these species, however no strategy serves all purposes as exposure is a complex and dynamic process confounded by spatial, temporal and geographic variations in airborne counts, in addition to the inadequacies of the immunodiagnostic techniques available. To date, the interpretation of personal exposure and sensitisation to fungal allergens has been restricted to a few select species and the contribution of other genera, airborne hyphae and fragmented conidia to allergic disease are all poorly understood. The aim of the thesis was to utilize the Halogen Immunoassay (HIA) to diagnose fungal allergic sensitisation, to investigate the distribution and factors influencing allergens of fungi in the air and to understand what is actually inhaled in exposure settings. The novelty of the HIA derives from its unique ability to provide allergen sources that are actively secreted by the collected fungal spores and hyphae, which are bound to protein binding membranes (PBM) and then immunoprobed. In Chapter 2, the HIA was compared to the commercial in vitro Pharmacia UniCap assay (CAP) and the in vivo skin prick test (SPT), using 30 sera from subjects SPT positive to Aspergillus fumigatus and/or Alternaria alternata and 30 who were SPT negative to these fungi but sensitised to non-fungal allergens. Sera were analysed by CAP and the HIA against A. alternata, A. fumigatus, Cladosporium herbarum and Epicoccum purpurascens and compared statistically. Between 3% and 7% of SPT negative sera were identified to have specific IgE towards A. fumigatus and A. iv alternata, respectively. For the SPT positive sera, significant associations were found between the HIA and CAP scores for all fungal species tested (P<0.0001). Correlations between the HIA and SPT however, were weakly correlated for A. alternata (rs = 0.44, P<0.05) but not for A. fumigatus. In Chapter 3, personal exposure to indoor fungal aerosols was examined using the HIA to identify the fungal components that people were allergic to. Personal air sampling pumps (PASs) collected airborne fungal propagules onto PBMs for 2.5 hours indoors (n=21). Collected fungi were incubated overnight in a humid chamber to promote the germination of conidia. The membranes were then immunostained with pooled human Alternaria species-positive sera. All air samples contained fungal hyphae that expressed soluble allergens and were significantly higher in concentration than counts of conidia of individual well-characterised allergenic genera. Approximately 25% of all hyphae expressed detectable allergen compared to non-stained hyphae (P<0.05) and the resultant localisation of immunostaining was heterogeneous among hyphae. Fungal conidia of ten genera that were previously uncharacterised as allergen sources accounted for 8% of the total conidia that demonstrated IgE binding. In Chapter 4, the number and identity of fungi inhaled by 34 adults in an outdoor community setting was measured over 2 hour periods by people wearing Intra-nasal air samplers (INASs) and compared to fungal counts made with a Burkard spore trap and filter air samplers worn on the lapel. Using INAS, the most prevalent fungi inhaled belonged to soil borne spores of Alternaria, Arthrinium, Bipolaris, Cladosporium, Curvularia, Epicoccum, Exserohilum, Fusarium, Pithomyces, Spegazzinia, Tetraploa and Xylariaceae species, in addition to hyphal fragments. These results showed that inhaled exposure in most people varied in a 2-fold range with 10-fold outliers. In addition, the INAS and personal air filters agreed more with each other than with Burkard spore trap counts. The analysis was further confounded by different sampling efficiencies, locations of devices and ability to visualise and count fungal propagules. In Chapter 5, a double immunostaining technique based on the HIA was developed and applied to the conidia, hyphae and fungal fragments of A. alternata, A. fumigatus and Penicillium chrysogenum to discriminate between sources of allergens, v using IgE and to identify the fungi, using a fungal-specific antibody. The localisation of immunostaining was heterogeneous between both conidia and the state of germination with greater concentrations of double immunostaining detected following germination for each fungal species (P<0.0001). Fragmented A. alternata hyphae and morphologically indiscernible fragments could be identified for the first time using this technique. In Chapter 6, the factors affecting the release of allergen from the spores of eleven different species were studied. For nine of eleven species, between 5.7% and 92% of spores released allergen before germination. Ungerminated spores of P. chrysogenum and Trichoderma viride did not release detectable allergen. After germination, all spores that germinated eluted allergen from their hyphae. Upon germination there was a significant increase in the percentage of spores eluting detectable allergen (P<0.0001) and the localisation of allergen along the hyphae varied between species. Increased elution of allergen post germination might be a common feature of many species of allergenic fungi following inhalation. Additionally, Chapter 6 explored the extent to which inhaled spores or hyphae germinate after deposition in the nasal cavity and thus cause exposure to allergens. Twenty subjects had their noses lavaged at three separate intervals, (1) at the beginning of the experiment, (2) after one hour indoors and (3) after one hour outdoors. The recovery of spores and hyphal fragments from the nasal cavity varied between individuals and was significantly greater after outdoor exposures. Germinated fungal spores were recovered often in high concentrations for Aspergillus-Penicillium species, however the proportion between ungerminated and germinated spores were much lower for other genera recovered. Conclusions: Our analysis of cultured and wild-type fungi presents a new paradigm of natural fungal exposure, which in addition to commonly recognized species, implicates airborne hyphae, fragmented conidia and the conidia of a much more diverse range of genera as airborne allergens. Exposure is heterogeneous between individuals in the same geographic locality and the spectrum of fungal genera inhaled differs with the method of analysis. Many of the spores inhaled are likely to be allergenic, however upon germination there is an increased elution of allergen and this might be a common vi feature of many fungal species following inhalation. This project also provides novel techniques to diagnose fungal allergy by immunostaining wild-type fungi to which a patient is exposed with the patient’s own serum. Such an immunoassay combines environmental with serological monitoring on a patient specific basis and potentially avoids many problems associated with extract variability, based on the performance of current diagnostic techniques for fungal allergy.
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Wildman, Scott Shaw. "Purinergic signalling : sensitisation of recombinant P2X receptors." Thesis, University College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325339.

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Nilsson, Caroline. "Cytokine profiles, infections and IgE sensitisation in childhood /." Stockholm : Department of Clinical Science and Education, Södersjukhuset : Sachs' Children's Hospital : Karolinska institutet, 2006. http://diss.kib.ki.se/2006/91-7140-720-0/.

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Davies, Catherine L. "Intermolecular sensitisation of Lanthanide Luminescence in Amphiphilic Systems." Thesis, University of York, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520025.

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De, Silva Mahappuque Anushika Sumali. "Ca²⁺ sensitisation and desensitisation mechanisms in pulmonary artery." Thesis, King's College London (University of London), 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499978.

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Modulation of Ca²⁺sensitivity of the smooth muscle contractile apparatus plays a fundamental role in the regulation of force. Increased CA²⁺ sensitivity may contribute towards various disease conditions such as pulmonary hypertension. Ca²⁺ sensitivity is regulated by the balance between myosin light chain kinase (MLCK)-dependent phosphorylation and myosin phosphatase (MP)-dependent dephosphorylation of myosin light chain (MLC-20). Several protein kinases have been implicated in modulating this process. In particular RhoA and its effector Rho kinase (Rhok) inhibits MP activity via the MYPTl subunit; whereas protein kinase C (PKC) activates MP inhibitory protein CPI-17. Additionally the mitogen activated protein kinases (MAPK) and heat shock protein 27 (HSP27) may effectively modulate Ca²⁺ sensitivity via the cytoskeleton.
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Wright, Phillip James. "Sensitisation of lanthanide luminescence by rhenium tetrazolato complexes." Thesis, Curtin University, 2016. http://hdl.handle.net/20.500.11937/1135.

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Luminescent inorganic compounds have received a great deal of attention due to the ability to modulate their properties and obtain highly emissive compounds for applications in light emitting devices and cellular labelling. This study targets the formation of emissive, multinuclear, tetrazolato complexes of Re(I), and utilises these complexes as sensitisers for lanthanide luminescence. The results increase the understanding of energy transfer mechanisms in d-f metal complexes, and highlight an interesting area for potential investigation.
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Boziaris, Ioannis S. "Bacterial injury and sensitisation of gram-negatives to nisin." Thesis, University of Surrey, 2000. http://epubs.surrey.ac.uk/842954/.

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Nisin is a bacteriocin produced by Lactococcus lactis subsp. lactis, which is active against Gram-positive organisms including bacterial spores. It is not generally active against Gram-negative bacteria, yeasts and fungi. Gram negatives show nisin-sensitivity when their outer membrane permeability is altered by various means, such as treatments with chelators, e.g. EDTA, osmotic shock, heating, freezing, freeze-drying, high- pressure etc. Application of chelators and nisin is effective against Gram-negatives when exogenous nisin is added. Nisin produced in situ and chelators are not an effective combination, since nisin production follows the pH drop caused by sugar fermentation, and this interferes with the sequestering ability of the chelators. Presence of nisin during thermal inactivation of Gram-negatives though is effective. Bacteria become structurally injured during heating showing sensitivity to agents like SDS and deoxycholate and extended detection times by impedimetry. These injured bacteria are inactivated by nisin, with a concomitant reduction of the measured D-values. Low pH and the presence of small amount of chelators enhance the injury and inactivation and reduce D-values further. Gram-negative bacteria injured by chilling and freezing are also sensitive to nisin. The effectiveness of nisin is reduced in a food environment mostly of nisin binding to fat, and food particles. D-values were decreased less or not at all in egg white and liquid whole egg, respectively, and rapid chilling of bacteria attached to chicken skin in presence of nisin did not give the effect seen in laboratory media. Nisin is active against heat-, chill-, and freezing-stressed Gram-negatives only if it is present during the treatments. When the stress factor is removed, the bacteria recover their nisin resistance, implying transient susceptibility to nisin, but not to smaller molecules. This is probably due to rapid reorganisation and restoration of OM permeability damage, rather than biochemical repair. The LPS chain length influences the sensitisation of Gram-negatives to nisin, only in the case of freezing, where the strain with the shorter LPS chain was more sensitive than the wild type. Heat-, and freezing-stressed bacteria lost lipopolysaccharides and increased their cell surface hydrophobicity. This was not seen with chill-stressed bacteria, which were sensitive to nisin though. This indicates that release of LPS is not a prerequisite for nisin sensitivity in Gram-negatives.
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Hamlin, Adam Scott. "Functional Neuroanatomy of Morphine-Induced Abstinence, Tolerance, and Sensitisation." University of Sydney, 2006. http://hdl.handle.net/2123/1164.

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Doctor of Philosophy (PhD)
The investigation into the relationship between neural plasticity in the rat forebrain associated with opiate-induced behaviours yielded two major results. The major finding of the functional neuroanatomy of acute morphine dependence was that doses of naloxone that induced hyperalgesia following a brief exposure to morphine, in previously drug-naïve rats, caused a specific induction of the inducible transcription factor (itf) proteins c-Fos and zif268 in the extended amygdala. Moreover, doses of naloxone that caused a simple reversal in morphine analgesia failed to induce itf proteins in these same brain regions. This increase in itf proteins was specific to regions of the extended amygdala that receive and process nociceptive information relayed via the spino-parabrachio-amygdaloid pathway and was not observed in other regions that are involved in supraspinal pain modulation such as the rostral ventromedial medulla and the periaqueductal gray. We also found that acute morphine increased c-Fos protein in the basolateral amygdala and the major output nucleus of the central amygdala the medial subdivision. Acute morphine also up-regulated c-Fos protein in striatal, midbrain, and hypothalamic nuclei. A unique finding of the current study was that prolonged exposure to morphine was required to induce c-Fos in these brain regions, as the subsequent administration of naloxone 30-minutes after morphine either reversed or blocked this induction. These results indicate the potential role of the amygdala in analgesia following systemic morphine and in pain facilitation during acute morphine abstinence. Investigation into the neurons and circuitry that undergo long-term neuroplasticity in response to repeated morphine exposure revealed that network-level changes in the distribution of Fos protein in the nucleus accumbens and striatum predicted both tolerance to catalepsy and psychomotor sensitisation. Drug-naïve rats became profoundly cataleptic following morphine, an effect that rats with a drug-history became tolerant. Rats with a history of morphine exposure showed an increase in stereotyped behaviours compared to drug-naïve rats. The major finding of this study was that a shift in the induction of c-Fos protein from a matrix predominance in drug-naïve rats toward a patch predominance in drug-sensitised rats in the accumbens core predicted both tolerance to catalepsy and sensitisation of oral stereotyped behaviours. Acute injection of morphine in a drug-naïve rat induced catalepsy and increased the number of c-Fos-positive neurons in matrix striatopallidal projection neurons of the rostral accumbens core. An increase in activity of striatopallidal projection neurons, which give rise to the indirect pathway, could potentially increase inhibitory drive to the pedunculopontine nucleus (PPN). The PPN, long known as a site of termination for basal ganglia output, is thought to direct the outflow of incentive-motivational and sensorimotor information from the nucleus accumbens to pons, medullary, and spinal cord nuclei translating the incentive impact of the stimuli into appropriate motor, autonomic and emotive responses (Winn et al., 1997). Inhibition of this nucleus would cause the animal to be unable to initiate a movement and in effect lock up, which is precisely what cataleptic postures look like. In contrast c-Fos-positive neurons were decreased in the rostral matrix and increased in patch striatonigral projection neurons along the rostro-caudal extent of the accumbens core when morphine was administered to drug-sensitised rats. Striatonigral neurons located in the patch give rise to the direct pathway innervating the dopaminergic neurons in both substantia pars compacta and the dopamine rich islands in the substantia nigra pars reticulata (Berendse et al., 1992; Gerfen, 1992; Furuta et al., 2002). Activity of this pathway is thought to be involved in the initiation of movement (Gerfen, 1992; Gerfen and Wilson, 1996), however, when this pathway is overstimulated as is the case when morphine is injected in drug-sensitised rats this could potentially cause increased activity of PPN neurons leading to repetitive psychomotor behaviours or stereotypy. This data adds to the growing body of evidence that suggests that long-term neuroadaptations induced by drugs of abuse including morphine that lead to behavioural sensitisation involves the circuitry that includes the nucleus accumbens.
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Books on the topic "Sensitisation"

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Hope, Jayne Catherine. Cytokine regulation of primary contact sensitisation. Manchester: University ofManchester, 1994.

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Gender, Sensitisation Workshop (2nd 2002 Kampala Uganda). Workshop proceedings: 2nd Gender Sensitisation Workshop. Arusha, Tanzania: ANNEA, 2001.

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Shaw, Linda Mary. Mechanisms of agonsit-induced calcium sensitisation in small arteries. Manchester: University of Manchester, 1996.

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Yuan, Yudie. Crystallographic effects in sensitisation of a aluminium-magnesium alloy. Birmingham: University of Birmingham, 2000.

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Harassment, Jawaharlal Nehru University Gender Sensitisation Committee against Sexual. Gender Sensitisation Committee against Sexual Harassment (GSCASH): Rules and procedures. New Delhi: Jawaharlal Nehru University, 1997.

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Agency, Medical Devices. Latex sensitisation in the health care setting: (use of latex gloves). London: Medical Devices Agency, 1996.

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CENWOR (Organization : Sri Lanka) and Sri Lanka. Natural Resources, Energy & Science Authority, eds. Gender sensitisation for scientists: Report of a workshop held on Friday, 2nd August 2002. [Colombo]: Centre for Women's Research in collaboration with the Ministry of Women's Affairs, 2000.

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Gender Sensitisation Workshop (1993 Nyeri, Kenya). Gender, development, and education: Proceedings of the Gender Sensitisation Workshop, Nyeri, January 28th-31th [sic], 1993. [Nairobi: Dept. of Development Studies, Kenyatta University, 1993.

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Gender, Responsive Planning and Programming Sensitisation Workshop (2nd 1991 Masinga Tourist Lodge Kenya). Gender Responsive Planning and Programming Sensitisation Workshop: Masinga Tourist Lodge, 10-15 February 1991 : workshop report. [Nairobi]: African Women Development and Communication Network (FEMNET), 1991.

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Gender Sensitisation Workshop for SPC Staff (1993 Nouméa, New Caledonia). Gender Sensitisation Workshop for SPC Staff (SPC headquarters, Noumea, New Caledonia, 8-10 December 1993): Report. Noumea, New Caledonia: South Pacific Commission, 1994.

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Book chapters on the topic "Sensitisation"

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Gibson, J. R. "Drug Sensitisation." In Pharmacology of the Skin II, 209–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74054-1_17.

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Carnés, Jerónimo, Carlos H. de Larramendi, María Angeles López-Matas, Angel Ferrer, and Julio Huertas. "Allergenic Sensitisation Mediated by Wolfberry." In Lycium Barbarum and Human Health, 179–98. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9658-3_12.

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Lepoittevin, J. P., and V. Mutterer. "Molecular Aspects of Fragrance Sensitisation." In Fragrances, 49–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80340-6_7.

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Bylin, G. "Healths problems related to respiratory sensitisation." In Use of Mechanistic Information in Risk Assessment, 305–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78640-2_33.

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Prys-Picard, Curig, and Robert Niven. "Severe Asthma with Fungal Sensitisation (SAFS)." In Aspergillosis: From Diagnosis to Prevention, 761–75. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2408-4_45.

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Schmelz, Martin. "Modulation of Pruritus: Peripheral and Central Sensitisation." In Pruritus, 27–31. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84882-322-8_5.

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Novianto, Rony, Benjamin Johnston, and Mary-Anne Williams. "Habituation and Sensitisation Learning in ASMO Cognitive Architecture." In Social Robotics, 249–59. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-02675-6_25.

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Chester, John, and Sarah Brown. "‘Chemo-Radio-Sensitisation’ in Head and Neck Cancer." In A Practical Guide to Designing Phase II Trials in Oncology, 141–50. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118763612.ch8.

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Vesterberg, Olof. "Indicators for Sensitisation to Ni, Co, and Cr, a Eu-Project." In Trace Elements in Man and Animals 10, 688–89. New York, NY: Springer US, 2002. http://dx.doi.org/10.1007/0-306-47466-2_220.

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Smith, Camilla K., and Sharon A. M. Hotchkiss. "Protein-hapten Binding and Immunorecognition Events During the Sensitisation and Elicitation Stages of ACD." In Allergic Contact Dermatitis, 207–21. London: CRC Press, 2023. http://dx.doi.org/10.1201/9780429121463-6.

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Conference papers on the topic "Sensitisation"

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Snovskaya, MA, LS Namazova-Baranova, OV Kozhevnicova, IL Mitushin, AS Batyrova, and AA Marushina. "P27 Age dependant features of children sensitisation to vegetables." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.115.

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Feary, Johanna, Jennifer Canizales, Susie Schofield, Bernadette Fitzgerald, Meinir Jones, and Paul Cullinan. "Sensitisation to mouse allergens in contemporary laboratory animal practice." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4779.

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Steen Pettersen, P., T. Neogi, K. Magnusson, H. B. Hammer, T. Uhlig, T. K. Kvien, and I. K. Haugen. "SAT0574 Sensitisation and pain severity in patients with hand osteoarthritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.3511.

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Ivan, F. X., P. Y. Tiew, K. X. Thng, T. K. Jaggi, E. P. P. Hwee, M. S. Koh, O. T. How, J. A. Abisheganaden, and S. H. Chotirmall. "The Metagenomic Resistome in Severe Asthma with Fungal Sensitisation (SAFS)." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3927.

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Virji, Mohammed Abbas, Christine Schuler, Marcia Stanton, Michael Kent, and Alexandr Stefaniak. "O47-5 Association of metrics of peak exposure with beryllium sensitisation." In Occupational Health: Think Globally, Act Locally, EPICOH 2016, September 4–7, 2016, Barcelona, Spain. BMJ Publishing Group Ltd, 2016. http://dx.doi.org/10.1136/oemed-2016-103951.242.

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Holden, Karl, David Lo, and Erol Gaillard. "Is fungal sensitisation associated with exacerbation-prone or severe preschool wheezing?" In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa953.

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Wark, Peter, Sikander Hussaini, Carl Holdar, Heather Powell, Christopher Oldmeadow, and Peter Gibson. "Omalizumab is an effective intervention in severe asthma with aspergillus sensitisation." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.2263.

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Kunal, Shekhar, Bala Krishnan Menon, and Ashok Shah. "Serum Vitamin D levels in patients with allergic rhinitis and fungal sensitisation." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa1160.

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Hodgekiss, Claire, Hasan Arshad, Graham Roberts, John Holloway, Elinor Jenkins, and Ramesh Kurukulaaratchy. "Atopic Sensitisation of the Isle of Wight Birth Cohort at 26 Years." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4049.

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Feary, Johanna, Bernadette Fitzgerald, Susie Schofield, Meinir Jones, and Paul Cullinan. "Sensitisation to mouse allergens in contemporary laboratory animal workers: The SPIRAL study." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa392.

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Reports on the topic "Sensitisation"

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Chopra, Deepta, Kas Sempere, and Meenakshi Krishnan. Assessing Unpaid Care Work: A Participatory Toolkit. Institute of Development Studies, March 2021. http://dx.doi.org/10.19088/ids.2021.016.

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Abstract:
This is a participatory toolkit for understanding unpaid care work and its distribution within local communities and families. Together, these tools provide a way of ascertaining and capturing research participants’ understanding of women’s unpaid care work – giving special attention to the lived experiences of carrying out unpaid care work and receiving care. Please note that these tools were developed and used in a pre-Covid-19 era and that they are designed to be implemented through face-to-face interactions rather than online means. We developed the first iteration of these tools in our ‘Balancing Care Work and Paid Work’ project as part of the Growth of Economic Opportunities for Women (GrOW) programme. The mixed-methods project sought to collect data across four countries – India, Nepal, Tanzania, and Rwanda – with data collected in four sites in each country (16 sites in total). The participatory tools were developed with two main intentions: (1) as a data collection tool to gain a broader understanding of the social norms and perspectives of the wider community in each of the 16 sites; and (2) to be implemented with our local partners as a sensitisation tool for the community regarding women’s unpaid care work burdens. While it is not essential to apply these tools in the order that they are presented, or even all of them, we would suggest that this toolkit be used in its entirety, to gather in-depth knowledge of social norms around the distribution of unpaid care, and the impacts that these have on care providers’ lives and livelihoods.
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