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Journal articles on the topic 'Selectively bred mice'

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Author: Grafiati

Published: 10 December 2022

Last updated: 30 January 2023

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1

Smolen, Andrew, Toni N. Smolen, and Jennifer L. van de Kamp. "Sensitivity of inbred and selectively bred mice to ethanol." Alcohol 4, no. 1 (January 1987): 57–62. http://dx.doi.org/10.1016/0741-8329(87)90061-9.

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2

Ewalds-Kvist, S. Béatrice M., Ritva-Kajsa Selander, and N. Kenneth Sandnabba. "Sex-Related Coping Responses in Mice Selectively Bred for Aggression." Perceptual and Motor Skills 84, no. 3 (June 1997): 911–14. http://dx.doi.org/10.2466/pms.1997.84.3.911.

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Sex differences in strategies of coping with novel situations were studied in three strains of mice with regard to metabolism and open-field and maze activity as well as learning-induced adjustment. The 140 mice were selectively bred for high (Turku Aggressive [TA]) and low (Turku Nonaggressive [TNA]) levels of aggressiveness and originated from a Swiss albino stock normally distributed [N] for aggressiveness. The results indicated that TNA sex differences are more similar to those of the control N mice as compared to those of TA mice. In maze learning, however, the sex differences of TA mice are more in agreement with those of the N strain. Recordings of metabolism and open-field as well as maze activity were correlates of both gender and strain. Sex differences in learning-induced open-field coping behavior were unrelated to strain.

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3

Kvist, Béatrice. "Learning in Mice Selectively Bred for High and Low Aggressiveness." Psychological Reports 64, no. 1 (February 1989): 127–30. http://dx.doi.org/10.2466/pr0.1989.64.1.127.

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Male mice selectively bred for aggressiveness (TA) and nonaggressiveness (TNA) were compared on maze performance and passive avoidance behavior The two lines performed the learning tasks in separate ways but the significant differences in performance were probably due to factors other than the brightness of one particular line.

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4

Buckman, Jennifer F., and Charles K. Meshul. "Immunocytochemical analysis of glutamate and GABA in selectively bred mice." Brain Research 760, no. 1-2 (June 1997): 193–203. http://dx.doi.org/10.1016/s0006-8993(97)00281-3.

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5

Nehrenberg, Derrick L., Ramona M. Rodriguiz, Michel Cyr, Xiaodong Zhang, Jean M. Lauder, Jean-Louis Gariépy, and William C. Wetsel. "An anxiety-like phenotype in mice selectively bred for aggression." Behavioural Brain Research 201, no. 1 (July 2009): 179–91. http://dx.doi.org/10.1016/j.bbr.2009.02.010.

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6

Meek, T. H., B. P. Lonquich, R. M. Hannon, and T. Garland. "Endurance capacity of mice selectively bred for high voluntary wheel running." Journal of Experimental Biology 212, no. 18 (August 28, 2009): 2908–17. http://dx.doi.org/10.1242/jeb.028886.

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7

Crabbe, John C., Lauren C. Kruse, Alexandre M. Colville, Andy J. Cameron, Stephanie E. Spence, Jason P. Schlumbohm, Lawrence C. Huang, and Pamela Metten. "Ethanol Sensitivity in High Drinking in the Dark Selectively Bred Mice." Alcoholism: Clinical and Experimental Research 36, no. 7 (February 8, 2012): 1162–70. http://dx.doi.org/10.1111/j.1530-0277.2012.01735.x.

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8

Mathes, Wendy Foulds, Derrick L. Nehrenberg, Ryan Gordon, Kunjie Hua, Theodore Garland, and Daniel Pomp. "Dopaminergic dysregulation in mice selectively bred for excessive exercise or obesity." Behavioural Brain Research 210, no. 2 (July 2010): 155–63. http://dx.doi.org/10.1016/j.bbr.2010.02.016.

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9

Kosobud, Ann E., Stephen J. Cross, and John C. Crabbe. "Neural sensitivity to pentylenetetrazol convulsions in inbred and selectively bred mice." Brain Research 592, no. 1-2 (October 1992): 122–28. http://dx.doi.org/10.1016/0006-8993(92)91666-3.

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10

Steward, Michael W., Carolynne Stanley, and Maria D. Furlong. "Antibody affinity maturation in selectively bred high and low-affinity mice." European Journal of Immunology 16, no. 1 (1986): 59–63. http://dx.doi.org/10.1002/eji.1830160112.

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11

Jensen, Bryan E., Kayla G. Townsley, Kolter B. Grigsby, Pamela Metten, Meher Chand, Miracle Uzoekwe, Alex Tran, et al. "Ethanol-Related Behaviors in Mouse Lines Selectively Bred for Drinking to Intoxication." Brain Sciences 11, no. 2 (February 4, 2021): 189. http://dx.doi.org/10.3390/brainsci11020189.

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Alcohol use disorder (AUD) is a devastating psychiatric disorder that has significant wide-reaching effects on individuals and society. Selectively bred mouse lines are an effective means of exploring the genetic and neuronal mechanisms underlying AUD and such studies are translationally important for identifying treatment options. Here, we report on behavioral characterization of two replicate lines of mice that drink to intoxication, the High Drinking in the Dark (HDID)-1 and -2 mice, which have been selectively bred (20+ generations) for the primary phenotype of reaching high blood alcohol levels (BALs) during the drinking in the dark (DID) task, a binge-like drinking assay. Along with their genetically heterogenous progenitor line, Hs/Npt, we tested these mice on: DID and drinking in the light (DIL); temporal drinking patterns; ethanol sensitivity, through loss of righting reflex (LORR); and operant self-administration, including fixed ratio (FR1), fixed ratio 3:1 (FR3), extinction/reinstatement, and progressive ratio (PR). All mice consumed more ethanol during the dark than the light and both HDID lines consumed more ethanol than Hs/Npt during DIL and DID. In the dark, we found that the HDID lines achieved high blood alcohol levels early into a drinking session, suggesting that they exhibit front loading like drinking behavior in the absence of the chronicity usually required for such behavior. Surprisingly, HDID-1 (female and male) and HDID-2 (male) mice were more sensitive to the intoxicating effects of ethanol during the dark (as determined by LORR), while Hs/Npt (female and male) and HDID-2 (female) mice appeared less sensitive. We observed lower HDID-1 ethanol intake compared to either HDID-2 or Hs/Npt during operant ethanol self-administration. There were no genotype differences for either progressive ratio responding, or cue-induced ethanol reinstatement, though the latter is complicated by a lack of extinguished responding behavior. Taken together, these findings suggest that genes affecting one AUD-related behavior do not necessarily affect other AUD-related behaviors. Moreover, these findings highlight that alcohol-related behaviors can also differ between lines selectively bred for the same phenotype, and even between sexes within those same line.

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12

Dlugosz, E. M., M. A. Chappell, D. G. McGillivray, D. A. Syme, and T. Garland. "Locomotor trade-offs in mice selectively bred for high voluntary wheel running." Journal of Experimental Biology 212, no. 16 (July 31, 2009): 2612–18. http://dx.doi.org/10.1242/jeb.029058.

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13

McCRAE, ANNE, LEONARD FIRESTONE, EDWARD J. GALLAHER, and PETER WINTER. "Halothane Requirement in Mice Selectively Bred for Sensitivity or Resistance to Diazepam." Annals of the New York Academy of Sciences 625, no. 1 Molecular and (June 1991): 555–57. http://dx.doi.org/10.1111/j.1749-6632.1991.tb33889.x.

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14

Kippin, Tod E. "Adaptations Underlying the Development of Excessive Alcohol Intake in Selectively Bred Mice." Alcoholism: Clinical and Experimental Research 38, no. 1 (December 10, 2013): 36–39. http://dx.doi.org/10.1111/acer.12327.

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15

KVIST, S. BEATRICE M., and RITVA-KAJSA SELANDER. "Mice selectively bred for an open field activity increase after maze learning." Scandinavian Journal of Psychology 31, no. 2 (June 1990): 128–38. http://dx.doi.org/10.1111/j.1467-9450.1990.tb00824.x.

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16

Kest, Benjamin, Gabrielle L. McLemore, Bogdan Sadowski, Jeffrey S. Mogil, John K. Belknap, and Charles E. Inturrisi. "Acute morphine dependence in mice selectively-bred for high and low analgesia." Neuroscience Letters 256, no. 2 (November 1998): 120–22. http://dx.doi.org/10.1016/s0304-3940(98)00772-1.

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17

Sadowski, Bogdan, and Marek Konarzewski. "Analgesia in Selectively Bred Mice Exposed to Cold in Helium/Oxygen Atmosphere." Physiology & Behavior 66, no. 1 (March 1999): 145–51. http://dx.doi.org/10.1016/s0031-9384(98)00282-0.

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18

Dudek, Bruce C., Duckhyun Kim Yi, David M. Gilliam, and Keith T. Irtenkauf. "Comparisons of subcolonies of selectively bred Long-Sleep and Short-Sleep mice." Behavior Genetics 23, no. 3 (May 1993): 245–50. http://dx.doi.org/10.1007/bf01082462.

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19

Sandnabba, N. Kenneth. "Predatory aggression in male mice selectively bred for isolation-induced intermale aggression." Behavior Genetics 25, no. 4 (July 1995): 361–66. http://dx.doi.org/10.1007/bf02197286.

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20

Eastwood, Emily C., and Tamara J. Phillips. "Opioid sensitivity in mice selectively bred to consume or not consume methamphetamine." Addiction Biology 19, no. 3 (November 12, 2012): 370–79. http://dx.doi.org/10.1111/adb.12003.

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21

Allan, Andrea M., and Robert L. Isaacson. "Ethanol-induced grooming in mice selectively bred for differential sensitivity to ethanol." Behavioral and Neural Biology 44, no. 3 (November 1985): 386–92. http://dx.doi.org/10.1016/s0163-1047(85)90712-5.

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22

Hood, Kathryn E., and Karen S. Quigley. "Exploratory behavior in mice selectively bred for developmental differences in aggressive behavior." Developmental Psychobiology 50, no. 1 (2007): 32–47. http://dx.doi.org/10.1002/dev.20264.

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23

Van Dijk, G., I. Jonas, C. J. Nyakas, T. Garland, L. M. Vaanholt, and G. H. Visser. "Regulation of energy balance in mice selectively bred for high voluntary activity." Appetite 51, no. 2 (September 2008): 407. http://dx.doi.org/10.1016/j.appet.2008.04.249.

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24

Eisenmann, Joey C. "Postnatal Development of Metabolic Traits in Mice Selectively Bred for High Running Capacity." Medicine & Science in Sports & Exercise 41 (May 2009): 52. http://dx.doi.org/10.1249/01.mss.0000353092.89812.59.

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25

Zgombick, John M., and Gene Erwin. "Central Mechanisms of Ethanol-Induced Adrenocortical Response in Selectively Bred Lines of Mice." Neuroendocrinology 46, no. 4 (1987): 324–32. http://dx.doi.org/10.1159/000124840.

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26

Boehm, Stephen L., John C. Crabbe, and Tamara J. Phillips. "Sensitivity to Ethanol-Induced Motor Incoordination in FAST and SLOW Selectively Bred Mice." Pharmacology Biochemistry and Behavior 66, no. 2 (June 2000): 241–47. http://dx.doi.org/10.1016/s0091-3057(00)00264-1.

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27

Marley, R. J., D. M. Arros, K. K. Henricks, M. E. Marley, and L. L. Miner. "Sensitivity to cocaine and amphetamine among mice selectively bred for differential cocaine sensitivity." Psychopharmacology 140, no. 1 (November 9, 1998): 42–51. http://dx.doi.org/10.1007/s002130050737.

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28

Risinger, Fred O., Dorcas H. Malott, Liesl K. Prather, Douglas R. Niehus, and Christopher L. Cunningham. "Motivational properties of ethanol in mice selectively bred for ethanol-induced locomotor differences." Psychopharmacology 116, no. 2 (October 1994): 207–16. http://dx.doi.org/10.1007/bf02245064.

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29

Weerts, Elise M., Lawrence G. Miller, Kathryn E. Hood, and Klaus A. Miczek. "Increased GABAA-dependent chloride uptake in mice selectively bred for low aggressive behavior." Psychopharmacology 108, no. 1-2 (July 1992): 196–204. http://dx.doi.org/10.1007/bf02245307.

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30

Mogil, Jeffrey S., Przemyslaw Marek, Pamela Flodman, Anne M. Spence, Wendy F. Sternberg, Benjamin Kest, Bogdan Sadowski, and John C. Liebeskind. "One or two genetic loci mediate high opiate analgesia in selectively bred mice." Pain 60, no. 2 (February 1995): 125–35. http://dx.doi.org/10.1016/0304-3959(94)00098-y.

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31

Erwin, V. Gene, Andrew D. Campbell, Reneé Myers, and Daniel E. Womer. "Cross-tolerance between ethanol and neurotensin in mice selectively bred for ethanol sensitivity." Pharmacology Biochemistry and Behavior 51, no. 4 (August 1995): 891–99. http://dx.doi.org/10.1016/0091-3057(95)00070-d.

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32

Kirchhoff, Aaron M., Eric L. Barker, and Julia A. Chester. "Endocannabinoids and Fear-Related Behavior in Mice Selectively Bred for High or Low Alcohol Preference." Brain Sciences 9, no. 10 (September 26, 2019): 254. http://dx.doi.org/10.3390/brainsci9100254.

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Alcohol use disorders (AUDs) have a high incidence of co-morbidity with stress-related psychopathologies, such as post-traumatic stress disorder (PTSD). Genetic and pharmacological studies support a prominent role for the endocannabinoid system (ECS) in modulating stress-related behaviors relevant to AUDs and PTSD. Mouse lines selectively bred for high (HAP) and low (LAP) alcohol preference show reproducible differences in fear-potentiated startle (FPS), a model for PTSD-related behavior. The first experiment in this study assessed levels of the endocannabinoids, anandamide (AEA) and sn-2 arachidonylglycerol (2-AG), in the prefrontal cortex (PFC), amygdala (AMG), and hippocampus (HIP) of male and female HAP1 and LAP1 mice following the expression of FPS to determine whether ECS responses to conditioned-fear stress (FPS) were correlated with genetic propensity toward high or low alcohol preference. The second experiment examined effects of a cannabinoid receptor type 1 agonist (CP55940) and antagonist (rimonabant) on the expression of FPS in HAP1 and LAP1 male and female mice. The estrous cycle of females was monitored throughout the experiments to determine if the expression of FPS differed by stage of the cycle. FPS was greater in male and female HAP1 than LAP1 mice, as previously reported. In both experiments, LAP1 females in diestrus displayed greater FPS than LAP1 females in metestrus and estrus. In the AMG and HIP, AEA levels were greater in male fear-conditioned HAP1 mice than LAP1 mice. There were no line or sex differences in effects of CP55940 or rimonabant on the expression of FPS. However, surprisingly, evidence for anxiogenic effects of prior treatment with CP55940 were seen in all mice during the third drug-free FPS test. These findings suggest that genetic differences in ECS function in response to fear-conditioning stress may underlie differences in FPS expression in HAP1 and LAP1 selected lines.

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33

McCrae, Anne F., Edward J. Gallaher, Peter M. Winter, and Leonard L. Firestone. "Volatile Anesthetic Requirements Differ in Mice Selectively Bred for Sensitivity or Resistance to Diazepam." Anesthesia & Analgesia 76, no. 6 (June 1993): 1313–17. http://dx.doi.org/10.1213/00000539-199306000-00022.

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34

McCrae, Anne F., Edward J. Gallaher, Peter M. Winter, and Leonard L. Firestone. "Volatile Anesthetic Requirements Differ in Mice Selectively Bred for Sensitivity or Resistance to Diazepam." Anesthesia & Analgesia 76, no. 6 (June 1993): 1313–17. http://dx.doi.org/10.1213/00000539-199376060-00022.

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35

Girard, Isabelle, Enrico L. Rezende, and Theodore Garland Jr. "Leptin Levels and Body Composition of Mice Selectively Bred for High Voluntary Locomotor Activity." Physiological and Biochemical Zoology 80, no. 6 (November 2007): 568–79. http://dx.doi.org/10.1086/521086.

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36

Quinlan, J. J., P. M. Winter, E. J. Gallaher, and L. L. Firestone. "HALOTHANE ENHANCES GABA-GATED CHLORIDE FLUX IN MICE SELECTIVELY BRED FOR SENSITIVITY TO DIAZEPAM." Anesthesiology 75, no. 3 (September 1, 1991): A582. http://dx.doi.org/10.1097/00000542-199109001-00581.

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37

Meek, T. H., J. C. Eisenmann, and T. Garland. "Western diet increases wheel running in mice selectively bred for high voluntary wheel running." International Journal of Obesity 34, no. 6 (February 16, 2010): 960–69. http://dx.doi.org/10.1038/ijo.2010.25.

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38

Crabbe, John C., Alexandre M. Colville, Lauren C. Kruse, Andy J. Cameron, Stephanie E. Spence, Jason P. Schlumbohm, Lawrence C. Huang, and Pamela Metten. "Ethanol Tolerance and Withdrawal Severity in High Drinking in the Dark Selectively Bred Mice." Alcoholism: Clinical and Experimental Research 36, no. 7 (February 6, 2012): 1152–61. http://dx.doi.org/10.1111/j.1530-0277.2011.01715.x.

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39

LEPPANEN, PIA K., and S. BEATRICE M. EWALDS-KVIST. "Crossfostering in mice selectively bred for high and low levels of open-field thigmotaxis." Scandinavian Journal of Psychology 46, no. 1 (February 2005): 21–29. http://dx.doi.org/10.1111/j.1467-9450.2005.00431.x.

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40

Su, Zhong, and Colin Dobson. "H-2 genes and resistance to infection with Heligmosomoides polygyrus in selectively bred mice." International Journal for Parasitology 27, no. 5 (May 1997): 595–600. http://dx.doi.org/10.1016/s0020-7519(97)00029-5.

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41

Wone, Bernard W. M., Won C. Yim, Heidi Schutz, Thomas H. Meek, and Theodore Garland. "Mitochondrial haplotypes are not associated with mice selectively bred for high voluntary wheel running." Mitochondrion 46 (May 2019): 134–39. http://dx.doi.org/10.1016/j.mito.2018.04.002.

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42

Hannon, Robert M., Thomas H. Meek, Wendy Acosta, Robert C. Maciel, Heidi Schutz, and Theodore Garland. "Sex-Specific Heterosis in Line Crosses of Mice Selectively Bred for High Locomotor Activity." Behavior Genetics 41, no. 4 (December 24, 2010): 615–24. http://dx.doi.org/10.1007/s10519-010-9432-3.

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43

Can, Adem, Nicholas J. Grahame, and Todd D. Gould. "Affect-Related Behaviors in Mice Selectively Bred for High and Low Voluntary Alcohol Consumption." Behavior Genetics 42, no. 2 (October 12, 2011): 313–22. http://dx.doi.org/10.1007/s10519-011-9505-y.

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44

Atkins, Alison, Melinda Helms, Laurie O'Toole, and John Belknap. "Stereotypic behaviors in mice selectively bred for high and low methamphetamine-induced stereotypic chewing." Psychopharmacology 157, no. 1 (August 1, 2001): 96–104. http://dx.doi.org/10.1007/s002130100774.

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45

Lufty, Kabirullah, Bogdan Sadowski, Ik-Sung Kwon, and Eckard Weber. "Morphine analgesia and tolerance in mice selectively bred for divergent swim stress-induced analgesia." European Journal of Pharmacology 265, no. 3 (November 1994): 171–74. http://dx.doi.org/10.1016/0014-2999(94)90428-6.

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46

Kamens, Helen M., Sue Burkhart-Kasch, Carrie S. McKinnon, Na Li, Cheryl Reed, and Tamara J. Phillips. "Ethanol-related traits in mice selectively bred for differential sensitivity to methamphetamine-induced activation." Behavioral Neuroscience 120, no. 6 (2006): 1356–66. http://dx.doi.org/10.1037/0735-7044.120.6.1356.

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47

Marek, Przemyslaw, Jeffrey S. Mogil, John K. Belknap, Bogdan Sadowski, and John C. Liebeskind. "Levorphanol and swim stress-induced analgesia in selectively bred mice: evidence for genetic commonalities." Brain Research 608, no. 2 (April 1993): 353–57. http://dx.doi.org/10.1016/0006-8993(93)91479-c.

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48

Gilliam, David M., Lori E. Kotch, Bruce C. Dudek, and Edward P. Riley. "Ethanol Teratogenesis in Selectively Bred Long-Sleep and Short-Sleep Mice: A Comparison to Inbred C57BL/6J Mice." Alcoholism: Clinical and Experimental Research 13, no. 5 (October 1989): 667–72. http://dx.doi.org/10.1111/j.1530-0277.1989.tb00402.x.

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49

Lazartigues, Eric, Shannon M. Dunlay, Puspha Sinnayah, Curt D. Sigmund, and Robin L. Davisson. "Brain-Selective Expression of Exogenous Angiotensin (AT1) Receptors Causes Enhanced Cardiovascular Sensitivity." Hypertension 36, suppl_1 (October 2000): 681. http://dx.doi.org/10.1161/hyp.36.suppl_1.681-b.

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19 To examine the functional consequences of overexpression of AT1 receptors selectively in brain of both normal mice and in mice otherwise lacking endogenous AT1 receptors, we generated a novel transgenic model with AT1 receptors targeted selectively to neurons in the CNS. A fusion transgene consisting of 2.8 Kb of rat neuron-specific enolase(NSE) 5’ flanking region, and a cDNA encoding the full open reading frame of the AT1A receptor was constructed and transgenic mice (NSE-AT1) were generated. Of the six transgenic founder lines identified, two exhibited overexpression of the transgene selectively in brain. Using both immunohistochemistry and a high affinity fluorescently labeled angiotensin II (AngII) probe, we observed high levels of transgenic AT1A receptors in brain of both lines. Distribution of the receptors was particularly prominent in regions involved in cardiovascular regulation, including brainstem and lamina terminalis nuclei. Focusing on NSE-AT1 line 6085/2, direct recording of baseline blood pressure in conscious freely moving mice revealed no difference between transgenic and wildtype controls. However, intracerebroventricular injection of AngII (50, 100, 200 ng, 200 nL) in conscious NSE-AT1 mice (n=5) caused an increased pressor response (Δ17±2, 22±4, 33±5) and a profoundly enhanced bradycardia (Δ-48±6, -92±27, -114±17) compared to wildtypes (n=8) (BP: Δ7±2, 17±2, 21±1; HR: Δ-15±12, -23±7, -34±10). Our data show that brain-selective overexpression of exogenous AT1 receptors results in enhanced cardiovascular sensitivity to central AngII. This model, along with a model harboring this transgene but lacking AT1 receptors elsewhere (NSE-AT1 mice bred with AT1 knockouts), provide a new approach and important tools for identifying the cardiovascular regulatory roles of brain AT1 receptors relative to those in peripheral tissues.

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50

Hitzemann, Iancu, Reed, Baba, Lockwood, and Phillips. "Regional Analysis of the Brain Transcriptome in Mice Bred for High and Low Methamphetamine Consumption." Brain Sciences 9, no. 7 (June 30, 2019): 155. http://dx.doi.org/10.3390/brainsci9070155.

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Transcriptome profiling can broadly characterize drug effects and risk for addiction in the absence of drug exposure. Modern large-scale molecular methods, including RNA-sequencing (RNA-Seq), have been extensively applied to alcohol-related disease traits, but rarely to risk for methamphetamine (MA) addiction. We used RNA-Seq data from selectively bred mice with high or low risk for voluntary MA intake to construct coexpression and cosplicing networks for differential risk. Three brain reward circuitry regions were explored, the nucleus accumbens (NAc), prefrontal cortex (PFC), and ventral midbrain (VMB). With respect to differential gene expression and wiring, the VMB was more strongly affected than either the PFC or NAc. Coexpression network connectivity was higher in the low MA drinking line than in the high MA drinking line in the VMB, oppositely affected in the NAc, and little impacted in the PFC. Gene modules protected from the effects of selection may help to eliminate certain mechanisms from significant involvement in risk for MA intake. One such module was enriched in genes with dopamine-associated annotations. Overall, the data suggest that mitochondrial function and glutamate-mediated synaptic plasticity have key roles in the outcomes of selective breeding for high versus low levels of MA intake.

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