Academic literature on the topic 'Schizophrenia – Genetic aspects'
Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles
Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Schizophrenia – Genetic aspects.'
Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.
You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.
Journal articles on the topic "Schizophrenia – Genetic aspects"
Morozova, A. Yu, E. A. Zubkov, Ya A. Zorkina, A. M. Reznik, G. P. Kostyuk, and V. P. Chekhonin. "Genetic aspects of schizophrenia." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 117, no. 6 (2017): 126. http://dx.doi.org/10.17116/jnevro201711761126-132.
Full textKarayiorgou, Maria. "Genetic aspects of schizophrenia." Clinical Neuroscience Research 1, no. 1-2 (January 2001): 158–63. http://dx.doi.org/10.1016/s1566-2772(00)00015-3.
Full textBöök, Jan A. "Genetic aspects of schizophrenia." Clinical Genetics 19, no. 5 (April 23, 2008): 357. http://dx.doi.org/10.1111/j.1399-0004.1981.tb00726.x.
Full textBellivier, F. "Schizophrenia, antipsychotics and diabetes: Genetic aspects." European Psychiatry 20, S4 (December 2005): S335—S339. http://dx.doi.org/10.1016/s0924-9338(05)80187-7.
Full textMuir, Walter J. "Genetic aspects of the biology of schizophrenia." Current Opinion in Psychiatry 5, no. 1 (February 1992): 2–5. http://dx.doi.org/10.1097/00001504-199202000-00002.
Full textTrifu, Simona Corina, Anca Vlăduţi, and Antonia Ioana Trifu. "Genetic aspects in schizophrenia. Receptoral theories. Metabolic theories." Romanian Journal of Morphology and Embryology 61, no. 1 (2020): 25–32. http://dx.doi.org/10.47162/rjme.61.1.03.
Full textKambarova, D. K., and A. G. Golubev. "Biochemical and genetic aspects of pathogenesis of schizophrenia." Journal of Evolutionary Biochemistry and Physiology 47, no. 5 (October 2011): 407–19. http://dx.doi.org/10.1134/s0022093011050021.
Full textGalderisi, S., and M. Maj. "Deficit schizophrenia: An overview of clinical, biological and treatment aspects." European Psychiatry 24, no. 8 (December 2009): 493–500. http://dx.doi.org/10.1016/j.eurpsy.2009.03.001.
Full textPopov, Nikolay T., Vili K. Stoyanova, Nadezhda P. Madzhirova, and Tihomir I. Vachev. "Epigenetic aspects in schizophrenia etiology and pathogenesis." Folia Medica 54, no. 2 (October 1, 2012): 12–16. http://dx.doi.org/10.2478/v10153-011-0082-x.
Full textSyvälahti, E. K. G. "Biological Factors in Schizophrenia Structural and Functional Aspects." British Journal of Psychiatry 164, S23 (April 1994): 9–14. http://dx.doi.org/10.1192/s0007125000292672.
Full textDissertations / Theses on the topic "Schizophrenia – Genetic aspects"
Zhang, Xiaowei, and 张晓薇. "Twin studies on childhood externalizing behavior and schizophrenia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208003.
Full textWycoff, Jeffrey M. L. "Neuropsychological patterns in RISC identified schizotypic subjects." Virtual Press, 1993. http://liblink.bsu.edu/uhtbin/catkey/864928.
Full textDepartment of Psychological Science
Wong, Nai-kei, and 黃乃淇. "Expression of myelin-related genes in an immune-precipitated mouse model of schizophrenia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B4378026X.
Full textWong, Hoi-man Emily, and 黃凱敏. "Genome-wide association analyses on complex diseases: from single-nucleotide polymorphism to copy numbervariation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hub.hku.hk/bib/B50534099.
Full textpublished_or_final_version
Psychiatry
Doctoral
Doctor of Philosophy
Truter, Erika. "Genetic association analysis of polymorphisms in four cytochrome P450 genes, the MDR1 gene and treatment-outcome in Xhosa schizophrenia patients." Thesis, Link to online version, 2007. http://hdl.handle.net/10019/350.
Full textWright, Galen Egan Buckley. "Molecular genetic analysis of two genes, CYP2D6 and COMT, in the schizophrenia-susceptibility locus on chromosome 22q in the Xhosa population." Thesis, Stellenbosch : Stellenbosch University, 2012. http://hdl.handle.net/10019.1/20366.
Full textWolff, Anne-Lise. "A kinematic investigation of oculomotor and skeletomotor performance in schizotypy /." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85104.
Full textThis thesis investigates oculomotor and skeletomotor function in clinically unaffected individuals who are at elevated risk for schizophrenia based on their scores on either a positive-symptom schizotypy questionnaire (Perceptual Aberration Scale) (n = 21) or a negative-symptom schizotypy questionnaire (Physical Anhedonia) (n = 20), and in Controls (n = 29).
In Manuscript 1, we review the evidence suggesting that skeletomotor deficits are present in neuroleptic-naive schizophrenia patients and high-risk populations. The review supports the notion of skeletomotor dysfunction in these groups and underscores the lack of studies using instrumentation to characterize the deficits. In Manuscript 2, we compare the oculomotor performance of positive-symptom and negative-symptom schizotypes to that of controls. Results suggest that smooth pursuit deficits identify high-risk individuals with either positive or negative symptomatology, while antisaccade deficits identify primarily individuals with positive symptoms. In Manuscript 3, we use high-speed instrumentation and kinematic measures to evaluate skeletomotor function, and to assess the relationship between oculomotor and skeletomotor deficits in positive and negative-symptom schizotypes. This study revealed differential patterns of skeletomotor deficits in positive- and negative-symptom schizotypy, with both patterns suggestive of frontal-striatal dysfunction. In general, oculomotor and skeletomotor deficits were not associated.
Together these results support the notion of motor deficits across domains in risk for schizophrenia. In addition, they highlight the importance of distinguishing between positive and negative symptomatology when investigating the pathophysiology of risk for schizophrenia.
Morgan, Vera Anne. "Intellectual disability co-occurring with schizophrenia and other psychiatric illness : epidemiology, risk factors and outcome." University of Western Australia. School of Psychiatry and Clinical Neurosciences, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0209.
Full textHitzeroth, Angelika. "Association of genetic variants and the susceptibility to abnormal involuntary movements and tardive dyskinesia (TD) in Xhosa schizophrenia patients." Thesis, Stellenbosch : University of Stellenbosch, 2007. http://hdl.handle.net/10019.1/2147.
Full textNo obvious explanations exist for the development of abnormal involuntary movements (AIM), but several hypotheses have been proposed for tardive dyskinesia (TD) development. Since TD seems to have a genetic basis, several genetic variants have been investigated in TD development in various populations. Few studies have focused on African populations. This study focused on genetic variants (previously investigated in other populations) and the development and severity of AIM and TD in a Xhosa schizophrenia population. Genotype and allele frequencies determined were compared to those described in the literature for other populations. Following a report of an association between Ala-9Val and schizophrenia in a Turkish population, this study subsequently investigated this association in the Xhosa population. MnSOD Ala-9Val was genotyped using HEX-SSCP analysis and the DRD3 Ser9Gly variant was genotyped using restriction enzyme digestion by MscI. Genotyping was followed by statistical comparisons of the various groups, as well as association analyses between the variant and schizophrenia (only for MnSOD), AIM, or TD development and severity. The groups included a Xhosa schizophrenia group, a subgroup of the Xhosa schizophrenia group that had AIM (AIM+) and did not have AIM (AIM-), a subgroup of the AIM+ group that had TD (TD+), and a healthy Xhosa control group. A possible interaction between Ala-9Val and Ser9Gly in the development of AIM and TD was also investigated. Lastly, it was attempted to genotype CYP2D6*4, CYP2D6*10 and CYP2D6*17 using various PCR methods followed by restriction enzyme analysis. MnSOD Ala-9Val genotype and allele frequencies were similar to those of the Turkish population, but differed to those of the Asian populations. No association between Ala-9Val and the development and severity of schizophrenia was found. However, a relationship between genotype and AIM or TD development was observed, as well as an association between TD severity and Ala- 9Val genotype. DRD3 Ser9Gly genotype and allele frequencies were similar to those of the African American population, but differed from other populations. No significant association between Ser9Gly and the development and severity of AIM or TD was detected, nor was an interactive effect between Ala-9Val and Ser9Gly in AIM or TD development observed. The genotyping of CYP2D6 proved difficult and these variants could therefore not be analysed. The CYP2D6*4 genotype and allele frequencies that could be determined from some samples, were similar to the frequencies described previously for African populations. While we did not find an association between Ser9Gly in TD or AIM development and severity, nor an interaction between Ala-9Val and Ser9Gly, we did observe a relationship between Ala-9Val and AIM or TD development and TD severity. The effect of this variant is probably small and other variants, specifically those in genes involved in free radical removal should be investigated in combination with Ala-9Val. With regard to CYP2D6 it is suggested that high-throughput genotyping methods (e.g. microarray technology) should be used in the future. This will enable simultaneous genotyping of several variants and can be used in various populations. This study is the first of its kind by focusing on the unique South African Xhosa population and TD or AIM development.
Koen, Liezl. "Chromosomal aberrations in the Xhosa schizophrenia population." Thesis, Stellenbosch : Stellenbosch University, 2008. http://hdl.handle.net/10019.1/1189.
Full textBACKGROUND: Schizophrenia is a heterogeneous illness resulting from complex gene-environment interplay. The majority of molecular genetic work done has involved Caucasian populations, with studies in these and Asian populations showing 2-32% of sufferers to have chromosomal aberrations. So far the discovery of a specific susceptibility mechanism or gene still eludes us, but the use of endophenotypes is advocated as a useful tool in this search. No cytogenetic studies of this nature have been reported in any African schizophrenia population. AIM: The aim of the study was to combine genotypic and phenotypic data, collected in a homogenous population in a structured manner, with the hope of characterising an endophenotype that could be used for more accurate identification of individuals with possible chromosomal abnormalities. METHODOLOGY: A structured clinical interview was conducted on 112 Xhosa schizophrenia patients. (Diagnostic Interview for Genetic Studies, including Schedules for the Assessment of Negative and Positive Symptoms.) Blood samples (karyotyping and/or FISH analysis) as well as urine samples (drug screening) were obtained and nine head and facial measurements were performed. Descriptive statistics were compiled with reference to demographic, clinical and morphological variables. Comparisons between mean differences for these variables were made.
Books on the topic "Schizophrenia – Genetic aspects"
Genetic models of schizophrenia. Amsterdam, Netherlands: Elsevier, 2009.
Find full textSearching for the causes of schizophrenia. Oxford: Oxford University Press, 1994.
Find full textCortical deficits in schizophrenia: From genes to function. New York: Springer, 2008.
Find full textTorrey, E. Fuller. Schizophrenia and manic-depressive disorder: The biological roots of mental illness as revealed by the landmark study of identical twins. New York: Basic Books, 1994.
Find full textDahlem Workshop on Biological Perspectives of Schizophrenia (1986 Berlin, Germany). Biological perspectives of schizophrenia: Report of the Dahlem Workshop on Biological Perspectives of Schizophrenia, Berlin 1986, October 26-31. Chichester: Wiley, 1987.
Find full textBoklage, Charles E. How new humans are made: Cells and embryos, twins and chimeras, left and right, mind/self\soul, sex, and schizophrenia. Singapore: World Scientific, 2010.
Find full textHunter, Beatrice Trum, and Beatrice Trum Hunter. Gluten intolerance: The widespread genetic defect that can cause arthritis, enteritis, schizophrenia and other health problems. New Canaan, CT: Keats, 1987.
Find full textBoklage, Charles E. How new humans are made: Cells and embryos, twins and chimeras, left and right, mind/self/soul sex, and schizophrenia. New Jersey: World Scientific, 2009.
Find full textBoklage, Charles E. How new humans are made: Cells and embryos, twins and chimeras, left and right, mind/self/soul, sex, and schizophrenia. New Jersey: World Scientific, 2010.
Find full textLang, Mary V. Trends In Schizophrenia Research. Nova Biomedical Books, 2004.
Find full textBook chapters on the topic "Schizophrenia – Genetic aspects"
Scourfield, Jane, and Peter McGuffin. "Genetic aspects." In Schizophrenia in Children and Adolescents, 119–34. Cambridge University Press, 2000. http://dx.doi.org/10.1017/cbo9780511526800.007.
Full textKinney, D. K. "Schizophrenia and Bipolar Disorder: Genetic Aspects." In International Encyclopedia of the Social & Behavioral Sciences, 13537–43. Elsevier, 2001. http://dx.doi.org/10.1016/b0-08-043076-7/03366-0.
Full textKwon, Ester J., Takahiro Soda, and Li-Huei Tsai. "Neurodevelopment and Schizophrenia." In Neurobiology of Mental Illness, edited by Pamela Sklar, 327–37. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0025.
Full textBrüne, Martin. "Schizophrenia spectrum and other psychotic disorders." In Textbook of Evolutionary Psychiatry and Psychosomatic Medicine, 159–76. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780198717942.003.0008.
Full textAndreasen, Nancy C. "The core dimensions of schizophrenia." In New Oxford Textbook of Psychiatry, edited by John R. Geddes, Nancy C. Andreasen, and Guy M. Goodwin, 565–73. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198713005.003.0057.
Full textJablensky, Assen, Hannah E. Jongsma, James B. Kirkbride, and Peter B. Jones. "Epidemiology of schizophrenia and related disorders." In Oxford Textbook of Neurologic and Neuropsychiatric Epidemiology, edited by Carol Brayne, Valery L. Feigin, Lenore J. Launer, and Giancarlo Logroscino, 277–92. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198749493.003.0028.
Full textLarge, Charles H. "Genetic association of voltage-gated ion channels with psychotic disorders." In Psychotic Disorders, edited by Michael A. P. Bloomfield and Oliver D. Howes, 335–40. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190653279.003.0037.
Full textTsuang, Ming T., William S. Stone, and Stephen V. Faraone. "Schizoaffective and schizotypal disorders." In New Oxford Textbook of Psychiatry, 595–602. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199696758.003.0080.
Full textGhaemi, S. Nassir. "Fundamentals of Neurobiology." In Clinical Psychopharmacology, edited by S. Nassir Ghaemi, 17–28. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199995486.003.0002.
Full text