Relevant bibliographies by topics / Schistosoma japonicum

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Academic literature on the topic 'Schistosoma japonicum'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 February 2023

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Journal articles on the topic "Schistosoma japonicum"

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GOBERT, G. N., D. J. STENZEL, M. K. JONES, D. E. ALLEN, and D. P. McMANUS. "Schistosoma japonicum: immunolocalization of paramyosin during development." Parasitology 114, no. 1 (January 1997): 45–52. http://dx.doi.org/10.1017/s0031182096008001.

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This paper describes the localization of paramyosin immunoreactivity in Schistosoma japonicum and represents the first comparative immunolocalization study among schistosome adult, cercariae and lung schistosomula by electron microscopy. A polyclonal antibody was utilized to immunolabel paramyosin or paramyosin-like proteins. Paramyosin was localized within the muscle layer of all 3 developmental stages. Furthermore, paramyosin was localized within granules of the post-acetabular glands of cercariae, and within the tegument matrix and surface of lung schistosomules. Adults and cercariae did not display any detectable paramyosin on the surface or within the tegument. The possible functions of paramyosin within S. japonicum and the relevance of these findings in relation to the reported protective properties of paramyosin as an anti-schistosome vaccine target molecule are discussed.
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Fernández-Soto, Pedro, Catalina Avendaño, Anna Sala-Vizcaíno, Beatriz Crego-Vicente, Begoña Febrer-Sendra, Juan García-Bernalt Diego, Ana Oleaga, et al. "Molecular Markers for Detecting Schistosoma Species by Loop-Mediated Isothermal Amplification." Disease Markers 2020 (July 24, 2020): 1–11. http://dx.doi.org/10.1155/2020/8042705.

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Schistosomiasis is considered a neglected parasitic disease. Around 280,000 people die from it annually, and more than 779 million people are at risk of getting infected. The schistosome species which infect human beings are Schistosoma mansoni, Schistosoma haematobium, Schistosoma intercalatum, Schistosoma japonicum, Schistosoma guineensis, and Schistosoma mekongi. This disease is also of veterinary significance; the most important species being Schistosoma bovis since it causes the disease in around 160 million livestock in Africa and Asia. This work was aimed at designing and developing a genus-specific loop-mediated isothermal amplification (LAMP) method for detecting the most important schistosome species affecting humans and for the species-specific detection of S. bovis. Bioinformatics tools were used for primer design, and the LAMP method was standardised for detecting the ITS-1 region from S. intercalatum, S. haematobium, S. mansoni, S. japonicum, and S. bovis DNA (generic test) and the NADH 1 gene for specifically detecting S. bovis (at different DNA concentrations). Detection limits achieved were 1 pg DNA for S. mansoni, 0.1 pg for S. haematobium, 1 pg for S. intercalatum, and 10 pg for S. bovis. No amplification for S. japonicum DNA was obtained. The LAMP designed for the amplification of S. bovis NADH-1 worked specifically for this species, and no other DNA from other schistosome species included in the study was amplified. Two highly sensitive LAMP methods for detecting different Schistosoma species important for human and veterinary health were standardised. These methods could be very useful for the diagnosis and surveillance of schistosome infections.
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PICA-MATTOCCIA, L., A. RUPPEL, C. M. XIA, and D. CIOLI. "Praziquantel and the benzodiazepine Ro 11-3128 do not compete for the same binding sites in schistosomes." Parasitology 135, no. 1 (September 4, 2007): 47–54. http://dx.doi.org/10.1017/s0031182007003514.

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SUMMARYThe benzodiazepine Ro 11-3128 (methyl-clonazepam) presents several similarities with praziquantel with regard to its anti-schistosomal mode of action, since both drugs cause spastic paralysis, calcium influx and tegumental disruption in the parasites. In order to know whether the two compounds share the same binding sites in the schistosomes, we performed in vivo and in vitro competition experiments. We took advantage of the fact that Ro 11-3128 is active against immature Schistosoma mansoni (whereas praziquantel is inactive), and praziquantel is active against S. japonicum (which is insensitive to Ro 11-3128). An excess of praziquantel did not inhibit the activity of Ro 11-3128 against immature S. mansoni and an excess of Ro 11-3128 did not inhibit the activity of praziquantel against S. japonicum, suggesting that the schistosome binding sites of the two drugs are different. On the other hand, cytochalasin D, an agent known to perturb – among other things – calcium channel function, was capable of inhibiting the schistosomicidal activity of both praziquantel and Ro 11-3128, thus adding another element of similarity between the two anti-schistosomal agents. A similar, albeit partial, inhibition of the schistosomicidal activity of the two drugs was exerted by some of the classical calcium channel blockers. Taken together, these results suggest that praziquantel and Ro 11-3128, although binding to different schistosome receptor sites, may use the same basic anti-schistosomal effector mechanisms.
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Du, Xiaofeng, Malcolm Jones, Sujeevi Nawaratna, Shiwanthi Ranasinghe, Chunrong Xiong, Pengfei Cai, Donald McManus, and Hong You. "Gene Expression in Developmental Stages of Schistosoma japonicum Provides Further Insight into the Importance of the Schistosome Insulin-Like Peptide." International Journal of Molecular Sciences 20, no. 7 (March 28, 2019): 1565. http://dx.doi.org/10.3390/ijms20071565.

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We showed previously that the Schistosoma japonicum insulin-like peptide (SjILP) binds the worm insulin receptors, thereby, activating the parasite’s insulin pathway and emphasizing its important role in regulating uptake of glucose, a nutrient essential for parasite survival. Here we show that SjILP is differentially expressed in the schistosome life cycle and is especially highly transcribed in eggs, miracidia, and adult female worms. RNA inference was employed to knockdown SjILP in adults in vitro, with suppression confirmed by significantly reduced protein production, declined adenosine diphosphate levels, and reduction in glucose consumption. Immunolocalization showed that SjILP is located to lateral gland cells of mature intra-ovular miracidia in the schistosome egg, and is distributed on the ciliated epithelium and internal cell masses of newly transformed miracidia. In schistosomula, SjILP is present on the tegument in two antero-lateral points, indicating highly polarized expression during cercarial transformation. Analysis of serum from S. japonicum-infected mice by ELISA using a recombinant form of SjILP as an antigen revealed IgG immunoreactivity to this molecule at 7 weeks post-infection indicating it is likely secreted from mature eggs into the host circulation. These findings provide further insights on ILP function in schistosomes and its essential roles in parasite survival and growth in different development stages.
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HOLA-JAMRISKA, L., J. P. DALTON, J. AASKOV, and P. J. BRINDLEY. "Dipeptidyl peptidase I and III activities of adult schistosomes." Parasitology 118, no. 3 (March 1999): 275–82. http://dx.doi.org/10.1017/s0031182098003746.

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Soluble extracts of adult Schistosoma japonicum and S. mansoni were examined for the presence of proteolytic activities ascribable to dipeptidyl peptidases (DPPs) at a range of pH from 4 to 11 using synthetic peptidyl substrates diagnostic of DPPs I, II, III and IV. Activity capable of cleaving the DPP I-specific substrates H-Gly-Arg-NHMec and H-Gly-Phe-NHMec which exhibited a pH optimum of 5·5 was observed in extracts of schistosomes. Female schistosomes exhibited greater DPP I activity than male schistosomes, while female S. japonicum showed substantially more activity than female S. mansoni. The specific activities against H-Gly-Arg-NHMec were 21·5 and 1·9 nmoles NHMec/min/mg protein for female and male S. japonicum and 8·5 and 1·9 nmoles NHMec/min/mg for female and male S. mansoni. The biochemical properties of schistosome DPP I were similar to mammalian DPP I (=cathepsin C) in that schistosome DPP I was only slowly inhibited by the cysteine protease inhibitor trans-epoxysuccinyl-1-leucylamido (4-guanidino)- butane, partly inhibited by the blocked diazomethyl ketones Z-Phe-Ala-CHN2 and Z-Phe-Phe-CHN2, but enhanced by halide ions. At pH 8·5, activity against the DPP III-specific substrate H-Arg-Arg-NHMec was evident in schistosome extracts, and this activity appeared to be due to a zinc metallo-exopeptidase because it was inhibited by 1,10-phenathroline and by EDTA. DPP II or DPP IV activity was not detected in the schistosome extracts.
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Wilson, R. Alan. "Schistosomiasis then and now: what has changed in the last 100 years?" Parasitology 147, no. 5 (January 22, 2020): 507–15. http://dx.doi.org/10.1017/s0031182020000049.

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AbstractOnly with the completion of the life cycles of Fasciola hepatica in 1883 and 30 years later those of Schistosoma japonicum (1913), Schistosoma haematobium and Schistosoma mansoni (1915) did research on schistosomiasis really get underway. One of the first papers by Cawston in 1918, describing attempts to establish the means of transmission of S. haematobium in Natal, South Africa, forms the historical perspective against which to judge where we are now. Molecular biology techniques have produced a much better definition of the complexity of the schistosome species and their snail hosts, but also revealed the extent of hybridization between human and animal schistosomes that may impact on parasite adaptability. While diagnostics have greatly improved, the ability to detect single worm pair infections routinely, still falls short of its goal. The introduction of praziquantel ~1982 has revolutionized the treatment of infected individuals and led directly to the mass drug administration programmes. In turn, the severe pathological consequences of high worm burdens have been minimized, and for S. haematobium infections the incidence of associated squamous cell carcinoma has been reduced. In comparison, the development of effective vaccines has yet to come to fruition. The elimination of schistosomiasis japonica from Japan shows what is possible, using multiple lines of approach, but the clear and present danger is that the whole edifice of schistosome control is balanced on the monotherapy of praziquantel, and the development of drug resistance could topple that.
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YOU, HONG, and DONALD P. MCMANUS. "Vaccines and diagnostics for zoonotic schistosomiasis japonica." Parasitology 142, no. 2 (October 31, 2014): 271–89. http://dx.doi.org/10.1017/s0031182014001310.

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SUMMARYSchistosomiasis is one of the most prevalent, insidious and serious of the tropical parasitic diseases. Although the effective anthelmintic drug, praziquantel, is widely available and cheap, it does not protect against re-infection, drug-resistant schistosome may evolve and mass drug administration programmes based around praziquantel are probably unsustainable long term. Whereas protective anti-schistosome vaccines are not yet available, the zoonotic nature of Schistosoma japonicum provides a novel approach for developing a transmission-blocking veterinary vaccine in domestic animals, especially bovines, which are major reservoir hosts, being responsible for up to 90% of environmental egg contamination in China and the Philippines. However, a greater knowledge of schistosome immunology is required to understand the processes associated with anti-schistosome protective immunity and to reinforce the rationale for vaccine development against schistosomiasis japonica. Importantly as well, improved diagnostic tests, with high specificity and sensitivity, which are simple, rapid and able to diagnose light S. japonicum infections, are required to determine the extent of transmission interruption and the complete elimination of schistosomiasis following control efforts. This article discusses aspects of the host immune response in schistosomiasis, the current status of vaccine development against S. japonicum and reviews approaches for diagnosing and detecting schistosome infections in mammalian hosts.
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Quezada, Landys A. Lopez, and James H. McKerrow. "Schistosome serine protease inhibitors: parasite defense or homeostasis?" Anais da Academia Brasileira de Ciências 83, no. 2 (June 2011): 663–72. http://dx.doi.org/10.1590/s0001-37652011000200025.

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Serpins are a structurally conserved family of macromolecular inhibitors found in numerous biological systems. The completion and annotation of the genomes of Schistosoma mansoni and Schistosoma japonicum has enabled the identification by phylogenetic analysis of two major serpin clades. S. mansoni shows a greater multiplicity of serpin genes, perhaps reflecting adaptation to infection of a human host. Putative targets of schistosome serpins can be predicted from the sequence of the reactive center loop (RCL). Schistosome serpins may play important roles in both post-translational regulation of schistosome-derived proteases, as well as parasite defense mechanisms against the action of host proteases.
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IMASE, A., T. KUMAGAI, H. OHMAE, Y. IRIE, and Y. IWAMURA. "Localization of mouse type 2 Alu sequence in schistosomes." Parasitology 119, no. 3 (September 1999): 315–21. http://dx.doi.org/10.1017/s0031182099004667.

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Localization of the type 2 Alu sequence (B2), a highly repetitive DNA sequence in the mouse genome, was examined by in situ polymerase chain reaction (in situ PCR) in schistosomes. The signals to the B2 sequence were detected in the cytoplasm of the tegumental membrane and in the nuclei of the mesenchymal, testicular, ovarian and vitelline cells of 8- week Schistosoma japonicum. In contrast, it was difficult to detect any signals of this sequence in 8-week S. mansoni, whereas in 24-week male S. mansoni the signals were observed in the cytoplasm of the tegumental tubercles and in the nuclei of the mesenchymal and testicular cells. On the other hand, in 24-week female S. mansoni the signals were found in the nuclei of the mesenchymal, ovarian and vitelline cells but not found in the tegument. On the contrary, no hybridization band of the B2 sequence was detected in the amplified DNA of 3-week schistosomula of either species. These observations proved that the host DNA sequences existed in restricted schistosome cells and were accumulated in the schistosome body during their development.
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Greer, George J., C. K. Ow-Yang, and Hoi-Sen Yong. "Schistosoma malayensis n. sp.: A Schistosoma japonicum-Complex Schistosome from Peninsular Malaysia." Journal of Parasitology 74, no. 3 (June 1988): 471. http://dx.doi.org/10.2307/3282058.

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Dissertations / Theses on the topic "Schistosoma japonicum"

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Xia, Mingyi. "Contribution à l'étude du développement et de la varabilité génétique de Schistosoma japonicum." Perpignan, 1990. http://www.theses.fr/1990PERP0089.

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Les recherches portent sur le developpement larvaire intramolluscal, la variabilite genetique et certaines relations de parente de schistosoma japonicum, agent de la bilharziose arterioveineuse humaine en chine continentale. Ces recherches demontrent que s. Japonicum presente, par rapport aux autres schistosomes de l'homme, de nombreux caracteres originaux concernant le lieu de penetration des miracidiums, la localisation des sporocystes primaires, la morphologie des sporocystes primaires et secondaires, la dynamique des populations larvaires, la strategie de colonisation de l'espace-mollusque, la chronobiologie des emissions cercariennes, la sensibilite des stades larvaires intramolluscaux au praziquantel. Le processus de replication des sporocystes secondaires est decrit pour la premiere fois dans le cycle de s. Japonicum; il permet un clonage indefini du parasite par transplantations micro-chirurgicales. Sur la base d'une comparaison entre s. Japonicum originaire des provinces de anhui et sichuan, il est montre qu'il existe un polymorphisme de compatibilite. Le rejet des parasites allopatriques par les mollusques peut se produire a des stades tardifs de la parasitose et provoquer des phenomenes de self-cure. Le polymorphisme de compatibilite est susceptible de renforcer les effets de l'isolement geographique, donc la divergence genetique entre populations; un aspect inattendu de cette divergence concerne les modalites de nage des miracidiums. Des essais de croisement entre s. Japonicum et s. Mansoni montrent que, malgre l'eloignement phyletique des especes, l'interaction entre males et femelles reste possible dans plusieurs processus de la sexualite, parmi lesquels la formation des couples; ces essais de croisement induisent une reproduction parthenogenetique
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Shrivastava, Jaya. "Molecular epidemiology of Schistosoma japonicum." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.414227.

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Laha, Thewarach. "Retrotransposable elements in the genome of Schistosoma japonicum /." [St. Lucia, Qld.], 2002. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe16631.pdf.

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Hurst, Maria. "Schistosomiasis japonica in the pig : aspects of pathology and pathogenesis /." Uppsala : Swedish Univ. of Agricultural Sciences (Sveriges lantbruksuniv.), 2000. http://epsilon.slu.se/avh/2000/91-576-5944-3.pdf.

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Moertel, Luke Paul Frank, and mobileluke@hotmail com /. Luke Moertel@qimr edu au. "Microarray Analysis of the Schistosoma japonicum Transcriptome." Central Queensland University. Chemical and Biomedical Sciences, 2007. http://library-resources.cqu.edu.au./thesis/adt-QCQU/public/adt-QCQU20070705.120939.

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Schistosomiasis, a disease of humans caused by helminth parasites of the genus Schistosoma, kills 200 to 500 thousand people annually, endangering over 600 million people world-wide with 200 million people infected in 2003 [1, 2]. Three species of schistosome are primarily responsible for human infections, namely, Schistosoma haematobium, endemic to Africa, India, and the Middle East, S. mansoni, endemic to Africa / South America, and S. japonicum endemic to China and the Philippines [3]. The major pathological effects of schistosomiasis result from the deposition of parasite ova in human tissues and the subsequent intense granulomatous response induced by these eggs. There is a high priority to provide an effective sub-unit vaccine against these schistosome flukes, using proteins encoded by cDNAs expressed by the parasites at critical phases of their development. One technique that may expedite this gene identification is the use of microarrays for expression analysis. A 22,575 feature custom oligonucleotide DNA microarray designed from public domain databases of schistosome ESTs (Expressed Sequence Tags) was used to explore differential gene expression between the Philippine (SJP) and Chinese (SJC) strains of S. japonicum, and between males and females. It was found that 593, 664 and 426 probes were differentially expressed between the two geographical strains when mix sexed adults, male worms and female worms were compared respectively. Additionally, the study revealed that 1,163 male- and 1,016 female-associated probes were differentially expressed in SJP whereas 1,047 male- and 897 female-associated probes were differentially expressed in SJC [4]. Further to this, a detailed real time PCR expression study was used to explore the differential expression of eight genes of interest throughout the SJC life cycle, which showed that several of the genes were down-regulated in different life cycle stages. The study has greatly expanded previously published data of strain and gender-associated differential expression in S. japonicum. Further, the new data will provide a stepping stone for understanding the complexities of the biology, sexual differentiation, maturation, and development of human schistosomes, signaling new approaches for identifying novel intervention and diagnostic targets against schistosomiasis [4].
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Lu, Dabing. "Transmission dynamics of Schistosoma japonicum within China." Thesis, Imperial College London, 2009. http://hdl.handle.net/10044/1/5301.

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Schistosoma japonicum, a multi-host parasite, remains highly endemic in China and has recently re-emerged in previously controlled regions. One reason may be, given the current human- and bovine-based control policy, due to a serious lack of knowledge of the potential role for other species of mammals, including dogs, cats and small rodents, in the transmission. This thesis aims to contribute to our understanding of transmission dynamics of S. japonicum by investigating the implications of different reservoirs in the transmission across two contrasting geographical regions/settings: the marshland with the disease persistence versus the hilly region where the disease was once controlled. Longitudinal characterization of S. japonicum infection at both definitive host and intermediate host levels was performed throughout 2006-2007, with the highest prevalence and infection intensity observed in rodents in the hilly region and in the agriculturally important domestic animals (cattle and goats) in the marshland. Three chronobiological trials of cercarial emergence were performed to identify any host (with nocturnal vs diurnal activity)-associated biological traits of the parasite. A late afternoon shedding pattern was observed in the hilly region, compatible with a nocturnal rodent reservoir, and a morning-afternoon dual shedding pattern within marshland areas, consistent with a diurnal bovine major reservoir. Characterization of the parasite population genetic diversity, using microsatellite markers, at both larval stages, also indicated cattle to be the main definitive host reservoir species in the marshland, which was further confirmed by sibling relationship analyses. In the hilly regions, however, epidemiological, biological and molecular data indicated that, in addition to the role of rodents as the main reservoirs to maintain the disease, dogs, with their higher mobility, may also play a significant role in S. japonicum transmission in these areas. The implications of these results, in terms of parasite strain sub-structuring and targeted disease control, were discussed.
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Gobert, Geoffrey Norman. "Ultrastructural and cytochemical characterization of the Schistosoma japonicum tegument." Thesis, Queensland University of Technology, 1997.

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Jamriska, Lubomira. "Characterisation of dipeptidyl peptidase I of the Asian blood fluke, Schistosoma japonicum." Thesis, Queensland University of Technology, 2000.

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Rudge, James W. "Schistosoma japonicum as a zoonosis : Population genetics and transmission dynamics." Thesis, Imperial College London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.508493.

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Zhang, Yaobi. "Schistosoma japonicum : studies on defined antigen vaccines and irradiated vaccines." Thesis, London School of Hygiene and Tropical Medicine (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285204.

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Books on the topic "Schistosoma japonicum"

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G, Chen M., Mott Kenneth E, and WHO Parasitic Diseases Programme. Schistosomiasis Unit., eds. Progress in assessment of morbidity due to schistosomiasis: Reviews of recent literature : Schistosoma haematobium, Schistosoma intercalatum, Schistosoma japonicum, Schistosoma mansoni. London: Bureau of Hygiene and Tropical Diseases, 1989.

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Chikugogawa Ryūiki Miyairigai Bokumetsu Taisaku Renraku Kyōgikai. Jimukyoku. Chikugogawa ryūiki ni okeru Nihon jūketsu kyūchūbyō to miyairigai. Kurume-shi: Chikugogawa Ryūiki Miyairigai Bokumetsu Taisaku Renraku Kyōgikai, 2000.

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Berger, Stephen, and Inc Gideon Informatics. Schistosoma Japonicum: Global Status. Gideon Informatics, Incorporated, 2022.

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Berger, Stephen, and Inc Gideon Informatics. Schistosoma Japonicum: Global Status. Gideon Informatics, Incorporated, 2019.

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Berger, Stephen, and Inc Gideon Informatics. Schistosoma Japonicum: Global Status. Gideon Informatics, Incorporated, 2021.

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Carabin, Hélène, Maria V. Johansen, Jennifer F. Friedman, Stephen T. McGarvey, Henry Madsen, Zhou Xiao-Nong, and Steven Riley. Zoonotic schistosomosis (schistosomiasis). Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0062.

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Asiatic schistosomiosis is a very old disease with Schistosoma japonicum eggs found in human remains > 2000 years old from Hunan and Hubei provinces in China (Mao and Shao 1982). The original description of Asiatic schistosomiosis was made by Fujii in 1847 (Sasa 1972). The life cycle was fi rst described by Kawanashi (1904) who noted trematode-like eggs in cat faeces. The same year, Katsurada recovered adult worms from a cat from Katayama, Japan (Okabe 1964). Fujinami and Nakamura (1909) first reported skin infection with S. japonicum cercariae of different mammals, and Miyairi and Suzuki (1914) discovered that Oncomelania hupensis served as intermediate host where miracidia developed into sporocysts and further into cercariae (Jordan 2000). The snail hosts of S. japonicum were discovered in China by Faust and Meleney (1923), The Philippines by Tubangui (1932) and in Indonesia by Carvey et al. (1973). In addition to the skin as the principal route of infection, Suda (1924) described oral infection and several authors described the intrauterine route of infection. (Okabe 1964; Sasa 1972).Following the understanding of the lifecyle, control measures including wearing closely woven clothing, composting of faeces with urine for at least 14 days, replacing cattle with horses, killing of rodents especially rats, killing of snails by lime, copper sulphate or salt water, were proven to have some efficacy. In Japan, an effective integrated control programme started after Second World War with the last human case being reported in 1978 (Jordan 2000 ). The National Schistosomiosis Control Programme in China started in 1955 and at that time more than 10 million people were infected with S. japonicum (Wu 2002). Emetine and antimony potassium tartrate were among the first drugs with proven efficacy against schistosomiosis in humans. Later antimony and finally praziquantel and artemether have been introduced as highly effective drugs with only minor adverse effects (Wu 2002).
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Book chapters on the topic "Schistosoma japonicum"

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Ringelmann, R., and Beate Heym. "Schistosoma japonicum." In Parasiten des Menschen, 227–28. Heidelberg: Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_83.

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Wu, Zhongdao. "Schistosoma japonicum." In Encyclopedia of Parasitology, 2443–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_2824.

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Wu, Zhongdao. "Schistosoma japonicum." In Encyclopedia of Parasitology, 1–7. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_2824-2.

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Despommier, Dickson D., Robert W. Gwadz, and Peter J. Hotez. "Schistosomes: Schistosoma mansoni (Sambon 1907), Schistosoma japonicum (Katsurada 1904), Schistosoma haematobium (Bilharz 1852)." In Parasitic Diseases, 108–21. New York, NY: Springer New York, 1995. http://dx.doi.org/10.1007/978-1-4612-2476-1_18.

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Xu, Jia-Xi, and Meng-Shen Cai. "Synthesis of antigenic peptides for 26 kDa glutathione S-transferase of Schistosoma japonicum." In Peptides, 256–57. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-010-9069-8_70.

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Seto, Edmund Y. W., and Elizabeth J. Carlton. "Disease Transmission Models for Public Health Decision-Making: Designing Intervention Strategies for Schistosoma japonicum." In Advances in Experimental Medicine and Biology, 172–83. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6064-1_12.

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Barban do Patrocinio, Andressa. "Schistosomiasis: Discovery of New Molecules for Disease Treatment and Vaccine Development." In Infectious Diseases. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104738.

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The parasite blood flukes belonging to the genus Schistosoma cause schistosomiasis. Among the Schistosoma species that infect humans, three stand out: Schistosoma japonicum (S. japonicum), which occurs in Asia, mainly in China and the Philippines; Schistosoma haematobium (S. haematobium), which occurs in Africa; and Schistosoma mansoni (S. mansoni), which occurs in Africa and South America and the center of Venezuela (Brazil). Research has shown that these species comprise strains that are resistant to Praziquantel (PZQ), the only drug of choice to fight the disease. Moreover, patients can be reinfected even after being treated with PZQ , and this drug does not act against young forms of the parasite. Therefore, several research groups have focused their studies on new molecules for disease treatment and vaccine development. This chapter will focus on (i) parasite resistance to PZQ , (ii) molecules that are currently being developed and tested as possible drugs against schistosomiasis, and (iii) candidates for vaccine development with a primary focus on clinical trials.
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Lee Willingham Iii, Arve, Remigio Olveda, Gemiliano Aligui, h√©l√®ne Carabin, Jonathan Kurtis, and Stephen Mcgarvey. "Interdisciplinary research on Schistosoma japonicum." In The Changing Face of Disease, 114–29. CRC Press, 2004. http://dx.doi.org/10.1201/9780203300183.ch7.

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Kyambikwa Bisangamo, Célestin. "Epidemiology and Control of Schistosomiasis." In Infectious Diseases. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.105170.

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Human schistosomiasis is caused by the genus Schistosoma. Its prevalence and morbidity are highest among schoolchildren, adolescents, and young adults. It is prevalent in poor communities without access to safe drinking water and adequate sanitation. The agents of etiology of these diseases are Schistosoma mansoni, Schistosoma haematobium, Schistosoma guineensis, Schistosoma intercalatum, Schistosoma japonicum, and Schistosoma mekongi. Symptoms include anemia, stunting, fever, cough, abdominal pain, diarrhea, hepatosplenomegaly, genital lesions, and eosinophilia. Freshwater mollusks are suitable intermediate hosts, and the definitive hosts are the parasitized men. The transmission gap of disease is bridged when people come into contact with unwholesome water sources infested. People are infected through their usual agricultural, domestic, professional, or recreational activities, which expose them to contaminated water. Various animals, such as cattle, dogs, cats, rodents, pigs, horses, and goats, serve as reservoirs. Treatment of at-risk people on a wide scale, access to good water, improved sanitation, hygiene education, and snail control are all used to combat schistosomiasis. The WHO’s schistosomiasis control strategy focuses on reducing disease by regularly administering praziquantel to affected populations on a large scale. It entails the regular treatment of all at-risk populations. Disease transmission should be halted in specific countries where transmission is low.
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HSü, H. F., and S. Y. Li HSü. "IMMUNIZATION AGAISNT SCHISTOSOMA JAPONICUM IN RHESUS MONKEYS." In Proceedings of the First International Congress of Parasitology, 719. Elsevier, 1999. http://dx.doi.org/10.1016/b978-0-08-011427-9.50081-0.

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Conference papers on the topic "Schistosoma japonicum"

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Kassa, B., M. H. Lee, R. Kumar, C. Mickael, L. Sanders, R. M. Tuder, M. Mentink-Kane, and B. B. Graham. "Experimental Schistosoma Japonicum-Induced Pulmonary Hypertension." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a4441.

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Kassa, B., R. Kumar, C. Mickael, L. Sanders, D. Koyanagi, R. M. Tuder, and B. B. Graham. "Schistosoma Japonicum Can Induce Pulmonary Hypertension in Mice." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5044.

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Fang, GuiJie, and XianFeng Qiao. "Molecular Cloning and Analysis of Glutathione S-Transferase Gene of Schistosoma japonicum." In 2010 2nd International Conference on Information Engineering and Computer Science (ICIECS). IEEE, 2010. http://dx.doi.org/10.1109/iciecs.2010.5678431.

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Zhao, Mingzhe, Ningzhong Liu, and Qiangyi Li. "Blurred video detection algorithm based on support vector machine of schistosoma japonicum miracidium." In 2016 International Conference on Advanced Mechatronic Systems (ICAMechS). IEEE, 2016. http://dx.doi.org/10.1109/icamechs.2016.7813468.

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Kassa, B., R. Kumar, C. Mickael, L. Sanders, D. Koyanagi, R. M. Tuder, and B. B. Graham. "Differential Effects of Schistosoma Japonicum Versus Mansoni on Th2 Inflammation in the Lungs to Cause Pulmonary Hypertension." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7859.

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Samarang, Made Agus Nurjana, Malonda Maksud, Sri Murtini, and Fadjar Satrija. "Profile of Antigen Excretory-Secretory Schistosoma Japonicum in the Development of Elisa Method to Detect Schistosomiasis in Indonesia." In 2nd International Conference on Veterinary, Animal, and Environmental Sciences (ICVAES 2020). Paris, France: Atlantis Press, 2021. http://dx.doi.org/10.2991/absr.k.210420.005.

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Lazzarotto, Eduardo Luís, Felipe Rhuan Gobi, Kawan Gabriel da Silva, and Fernanda Mauer D'Agostini. "A INFLUÊNCIA DAS PARASITOSES INTESTINAIS SOBRE A SUPRESSÃO DA RESPOSTA PRÓ-INFLAMATÓRIA EM PACIENTES COM ARTRITE REUMATOIDE." In I Congresso Brasileiro de Parasitologia Humana On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/701.

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Introdução: A artrite reumatoide (AR) é uma doença autoimune de caráter inflamatório e crônico caracterizada por uma sinovite crônica e simétrica, que leva a erosão do osso e cartilagem. Objetivo: Relacionar a resposta inflamatória de AR e a influência das parasitoses intestinais. Metodologia: Revisão bibliográfica de artigos disponibilizados nas plataformas Scielo, Pubmed e Scientific Research Publising, nas línguas portuguesa e inglesa, datados entre 2008 a 2019, com os seguintes descritores: artrite reumatoide parasitoses intestinais, enteroparasitoses pacientes com AR, influência das parasitoses intestinais em pacientes com AR. Resultados: A membrana sinovial é considerada o tecido no qual se inicia e se mantém o processo inflamatório na AR. Atualmente, a causa de AR é a desregulação da produção de interleucina (IL)‐ 17, responsável pela ativação das células T e B, bem como dos macrófagos, que liberam citocinas como IL‐ 1 , IL‐ 6 e fator de necrose tumoral (TNF‐ α). Estudos indicam que helmintos podem modular a resposta imune proporcionando um ambiente anti-inflamatório que ajude sua sobrevivência no hospedeiro, anulando respostas imunes pró-inflamatórias, suprimem respostas dos linfócitos Th-1 e Th-17, além de estimular o desenvolvimento de respostas dos linfócitos Th-2, importantes nas doenças reumáticas autoimunes, como AR, pois a infecção de helmintos contribui para o alívio dos sintomas. Estudos indicam que Schistosoma mansoni, Schistosoma japonicum, Ascaris suum, Heligmosomoides polygyrus bakeri e Hymenolepsis diminuta são parasitas que possuem efeito protetor no processo inflamatório são. Um estudo, o qual fez uso de administração de um extrato de Ascaris suum na artrite experimental induzida por colágeno e por zimosan em ratos e camundongos, assinalou melhoria clínica e estrutural nas duas tipologias, reduzindo a liberação de mediadores inflamatórios como o NO, IL-1 β e IL-10. O efeito benéfico ocorreu a nível funcional e estrutural, abrangendo hiperalgesia articular, redução de danos da cartilagem articular, prevenção da perda de glicosaminoglicanos e revogação da sinovite crônica com redução de macrófagos e linfócitos. Conclusão: A produção de um ambiente imunologicamente controlado como resultado da infecção por helmintos pode diminuir a severidade de uma doença reumática simultânea. Helmintos influenciam beneficamente na resposta inflamatória, podendo auxiliar no tratamento de doenças autoimunes como a AR.
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Widayati, Anis Nur, Muhamad Faozan, Junus Widjaja, Ahmad Erlan, and Malonda Maksud. "IMPLEMENTATION OF THE SCHISTOSOMIASIS CONTROL TEAM (PEDA’ TEAM) IN BADA HIGHLAND, CENTRAL SULAWESI PROVINCE, INDONESIA." In International Conference on Public Health. The International Institute of Knowledge Management, 2021. http://dx.doi.org/10.17501/24246735.2020.6101.

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Background. Schistosomiasis has been a community health problem in endemic areas. In Indonesia, it is caused by trematode Schistosoma japonicum, with snail Oncomelania hupensis lindoensis as its intermediate host. This disease is only found in three endemic areas in Central Sulawesi Province: Napu and Bada highlands, Poso Regency, and Lindu highland in Sigi Regency. Various schistosomiasis control efforts were accomplished for more than the last twenty years. However, the schistosomiasis prevalence in Indonesia still fluctuates. The study aimed to determine the effectiveness of the Peda’ team in the research area. Method. One strategy of schistosomiasis control by community empowerment, namely the Bada Model, was implemented in Bada highland, Poso Regency, Central Sulawesi Province, from May to November 2019. One of the model’s elements is the formation of the schistosomiasis control village team (namely Peda’ Team), which was trained about schistosomiasis control. After the training, they were assigned several duties, including schistosomiasis socialization to the village community, stool sample collection, snail habitat control, snail and rats survey, and assisting the mass drug administration. The evaluation was conducted in stool sample coverage and the snail habitat area. Results. The results showed an increase in stool sample collection coverage in the community, becoming more than 80% in almost all endemic villages. We found the decrease from 26 become three focus areas because of the active effort to control the schistosomiasis intermediate snail focus area. Based on the results can be concluded that the Peda team formation can support schistosomiasis control more useful in the research area Keywords: Schistosomiasis, Bada Model, Community Empowerment
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