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Academic literature on the topic 'Scavenger molecules of oxygen reactive species'

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Published: 10 March 2023

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Journal articles on the topic "Scavenger molecules of oxygen reactive species"

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Misra, Hari S., Nivedita P. Khairnar, Atanu Barik, K. Indira Priyadarsini, Hari Mohan, and Shree K. Apte. "Pyrroloquinoline-quinone: a reactive oxygen species scavenger in bacteria." FEBS Letters 578, no. 1-2 (November 4, 2004): 26–30. http://dx.doi.org/10.1016/j.febslet.2004.10.061.

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Festjens, N., M. Kalai, J. Smet, A. Meeus, R. Van Coster, X. Saelens, and P. Vandenabeele. "Butylated hydroxyanisole is more than a reactive oxygen species scavenger." Cell Death & Differentiation 13, no. 1 (September 2, 2005): 166–69. http://dx.doi.org/10.1038/sj.cdd.4401746.

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Mahajan, Nitin, Heidi Y. Shi, Thomas J. Lukas, and Ming Zhang. "Tumor-suppressive Maspin Functions as a Reactive Oxygen Species Scavenger." Journal of Biological Chemistry 288, no. 16 (March 7, 2013): 11611–20. http://dx.doi.org/10.1074/jbc.m112.410852.

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Mori, Hiroko, Toshiyuki Arai, Hisanari Ishii, Nobuyuki Endo, Toshinori Suzuki, and Kazuhiko Fukudal. "Pterin-6-aldehyde, Xanthine Oxidase Inhibitor and Superoxide Scavenger, Directly React with Peroxynitrite." Pteridines 10, no. 1 (February 1999): 32–34. http://dx.doi.org/10.1515/pteridines.1999.10.1.32.

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The effect of pterin-6-aldehyde (P6A), xanthine oxidase inhibitor and superoxide scavenger, on the production of nitrotyrosine as a footprint of tyrosine nitration by peroxynitrite, was compared with that of uric acid, a peroxynitrite scavenger. The amounts of tyrosine, P6A and nitrotyrosine were quantified using reversed-phase high-performance liquid chromato-graphy (RP-HPLC). P6A suppressed nitrotyrosine formation less effectively than uric acid, that is, 0.25 mM P6A reduced nitrotyrosine formation to 67.9± 10.8%, while 0.025 mM uric acid reduced it to 34.2± 1.6%. In living systems, peroxynitrite is generated by the reaction of super-oxide with nitric oxide and has a variety of toxic effects. Our results show that P6A is not necessarily a strong scavenger of peroxynitrite. However, since P6A is a potent scavenger of superoxide, P6A is thought to totally suppress peroxynitrite generation. Compounds that scavenge both superoxide and peroxynitrite may be useful in tissue damage in which reactive oxygen species are involved.
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Singh, Neha, Satish C. Bhatla, and Vadim Demidchik. "Plants and human beings engage similar molecular crosstalk with nitric oxide under stress conditions." Functional Plant Biology 46, no. 8 (2019): 695. http://dx.doi.org/10.1071/fp19018.

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Human beings and plants experience a variety of stress conditions and adapt themselves through novel molecular crosstalk in their cellular constituents. Nitric oxide (NO), haemoglobin and melatonin interact with each other not only in blood stream of human beings, but also in the cells and metabolically active conducting strands of plants. Specialised sites of biosynthesis and differential intracellular spatial distribution of these molecules have been clearly demonstrated by the authors in plant systems. This has led to an understanding of the role of these molecules under salt stress conditions experienced by plants: NO is a modulator of enzyme activity through S-nitrosylation and tyrosine nitration, haemoglobin (phytoglobin) is an NO scavenger, and melatonin is a reactive oxygen species (ROS) scavenger involved in key crosstalk in both plants and humans facing stress. Our recent work on heme oxygenase (HO) activity modulation by stress in plants, and its interaction with NO, further demonstrates common features of molecular crosstalk in protecting plants and human beings from stress.
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Voronkova, Y. S., O. S. Voronkova, V. A. Gorban, and K. K. Holoborodko. "Oxidative stress, reactive oxygen species, antioxidants: a review." Ecology and Noospherology 29, no. 1 (May 9, 2018): 52–55. http://dx.doi.org/10.15421/031809.

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Oxidative stress is a disturbance of the balance between the production of reactive oxygen species (ROS) and antioxidants. Oxidative stress is caused by the presence of any of a number of reactive oxygen species, which the cell is unable to counterbalance. The result is damage to one or more biomolecules including DNA, RNA, proteins and lipids. Oxidative stress has been implicated in the natural aging process as well as a variety of disease states, such as neoplastic, metabolic, neurological etc., accompanied by different complications. Risk factors of generation of oxidative stress are oxidizing species, induced by pathologies include alcohol consumption, cigarette smoking, diet, gender, geographic location specifically at high altitude and occupation. ROS are composed of superoxide, hydroxyl, peroxyl, hydroperoxyl and alkoxyl radicals, hydrogen peroxide and singlet oxygen and ozone. These compounds produced endogenous in reaction of autooxidation in respiratory chaine of bioobjects. Among exogenous sources of ROS can be listed exposure of pollutants, toxins, heavy metals, drugs with different chemical origin and effects, radiation, electromagnetic fields, alcohol, cigarette smoke, stresses, allergies, dietary factors, temperature and microscopic form of life, such as bacteria, yeasts, viruses etc. The oxidative stress in biological systems is often characterized by increase in the formation of radicals; decrease in small-molecular-weight and lipid soluble antioxidants; disturbance in cellular redox balance; oxidative damage to cellular components (biomacromolecules). The presence of oxidative stress may be tested in one of three ways: direct measurement of the ROS; measurement of the resulting damage to biomolecules; and detection of antioxidant levels. Directly measuring ROS might seem the preferred method, but many reactive oxygen species are extremely unstable and difficult to measure directly. Many markers of damage are extremely stable and therefore provide a more reliable method to measure oxidative stress. Another approach is to measure the levels of antioxidant enzymes and other redox molecules which serve to counterbalance ROS generated in the cell. At the same time, it must emphasize that oxidative stress not only has a cytotoxic effect, but also plays an important role in the modulation of messengers that regulate essential cell membrane functions, which are vital for survival. For prevention of oxidative stress cells produce or uptake antioxidants – substance significantly delays or prevents oxidation of that substrate. Antioxidants may be enzymatic and non-enzymatic in nature in which enzymatic system directly or indirectly help in defence against the ROS. Antioxidants are involved in the prevention of oxidants and ROS formation; exhibits scavenger of ROS; and repairs the oxidized molecules through sources like dietary or consecutive antioxidants. Among non-enzymatic antioxidants distinguished glutathione, α-tocopherol, ascorbic acid, beta-carotene, and uric acid; these are mostly considered to be chain-breaking antioxidants in that they interrupt the auto-catalytic spread of radical reactions. Among enzymatic antioxidants most known superoxide dismutase, catalase, glutathione-SH peroxidase. The significant correlation found between ROS, parameters of oxidative stress and pathology indicate that there is a need in finding of measures of prevention of endo- and exogenous factors provoke their generation. There is a need to continue to explore the relationship between free radicals, pathological processes and the complications of them, and to elucidate the mechanisms by which increased oxidative stress accelerates the development of complications, in an effort to expand treatment options. Improvement of complications control seems to be a beneficial factor to decrease oxidative stress. For a better investigation of oxidative stress, it would be wise to supplement the clinical research by determination of special products typical for oxidative stress that let to understand mechanism of some pathological processes more clearly.
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Liu, Fu-Chao, Hsin-I. Tsai, and Huang-Ping Yu. "Organ-Protective Effects of Red Wine Extract, Resveratrol, in Oxidative Stress-Mediated Reperfusion Injury." Oxidative Medicine and Cellular Longevity 2015 (2015): 1–15. http://dx.doi.org/10.1155/2015/568634.

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Resveratrol, a polyphenol extracted from red wine, possesses potential antioxidative and anti-inflammatory effects, including the reduction of free radicals and proinflammatory mediators overproduction, the alteration of the expression of adhesion molecules, and the inhibition of neutrophil function. A growing body of evidence indicates that resveratrol plays an important role in reducing organ damage following ischemia- and hemorrhage-induced reperfusion injury. Such protective phenomenon is reported to be implicated in decreasing the formation and reaction of reactive oxygen species and pro-nflammatory cytokines, as well as the mediation of a variety of intracellular signaling pathways, including the nitric oxide synthase, nicotinamide adenine dinucleotide phosphate oxidase, deacetylase sirtuin 1, mitogen-activated protein kinase, peroxisome proliferator-activated receptor-gamma coactivator 1 alpha, hemeoxygenase-1, and estrogen receptor-related pathways. Reperfusion injury is a complex pathophysiological process that involves multiple factors and pathways. The resveratrol is an effective reactive oxygen species scavenger that exhibits an antioxidative property. In this review, the organ-protective effects of resveratrol in oxidative stress-related reperfusion injury will be discussed.
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Rodrigues, Eliseu, Lilian R. B. Mariutti, Adélia F. Faria, and Adriana Z. Mercadante. "Microcapsules containing antioxidant molecules as scavengers of reactive oxygen and nitrogen species." Food Chemistry 134, no. 2 (September 2012): 704–11. http://dx.doi.org/10.1016/j.foodchem.2012.02.163.

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Kovary, Karla, Tatiana S. Louvain, Maria C. Costa e. Silva, Franco Albano, Barbara B. M. Pires, Gustavo A. T. Laranja, Celso L. S. Lage, and Israel Felzenszwalb. "Biochemical behaviour of norbixin duringin vitroDNA damage induced by reactive oxygen species." British Journal of Nutrition 85, no. 4 (April 2001): 431–40. http://dx.doi.org/10.1079/bjn2000287.

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Naturally occurring antioxidants such as carotenoids are extensively studied for their potential in reducing the risk for cancer and other chronic diseases. In the present study, the radical-scavenger activity of the food additive norbixin, a water-soluble carotenoid extracted fromBixa orellanaseeds and commercialized as annatto, was evaluated under conditions of DNA damage induced by reactive oxygen species, particularly by hydroxyl radicals. The cell-free scavenger activity of norbixin was evaluated using plasmid DNA as target molecule and Sn2+or Fe2+as oxidant. The addition of H2O2enhanced DNA breakage induced by metal ions, particularly Fe2+. Under these conditions, norbixin started to protect plasmid DNA against single- and double-strand breakage at a metal:norbixin ratio of 1:1 (Sn2+) and 1:10 (Fe2+). However, at lower ratios to Sn2+, norbixin enhanced Sn2+-induced DNA breakage (P<0.05). The ability of norbixin to protect genomic DNA against oxidative damage was assessed in murine fibroblasts submitted to H2O2-induced oxidative stress and the results were evaluated by the comet assay. Under low serum conditions (2 % fetal bovine serum (FBS)), a protective effect of norbixin against H2O2-induced DNA breakage was inversely related to its concentration, a protection ranging from 41 % (10 μM) TO 21 % (50 μm). At higher concentrations of norbixin, however, oxidative DNA breakage was still enhanced, even in the presence of a high serum concentration (10 % FBS). Under normal conditions, norbixinper sehas no detectable genotoxic or cytotoxic effects on murine fibroblasts. The antimutagenic potential of norbixin against oxidative mutagens was also evaluated by theSalmonella typhimuriumassay, with a maximum inhibition of 87 % against the mutagenicity induced by H2O2. Although plasmid DNA and Ames data indicated that norbixin can protect DNA against oxidative damage, it seems to be a risky guardian of genomic DNA as it can also increase the extent of oxidative damage.
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Kim, Hyoung Jin, Sun Young Koo, Bong-Hyun Ahn, Oeuk Park, Doo Hoe Park, Dong Ook Seo, Jong Heon Won, et al. "NecroX as a novel class of mitochondrial reactive oxygen species and ONOO− scavenger." Archives of Pharmacal Research 33, no. 11 (November 2010): 1813–23. http://dx.doi.org/10.1007/s12272-010-1114-4.

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Dissertations / Theses on the topic "Scavenger molecules of oxygen reactive species"

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Fukuhara, Ryoji. "Molecular cloning, gene expression, and evolution of enzymes that scavenge reactive oxygen species in primates." 京都大学 (Kyoto University), 2005. http://hdl.handle.net/2433/145142.

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Phillips, Darren C. "Ceramide and reactive oxygen species (ROS) as signal transduction molecules in inflammation." Thesis, Aston University, 2003. http://publications.aston.ac.uk/12363/.

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The application of synthetic ceramides to U937 monocytes for short (2 hours) or long (16 hours) treatment periods reduced the membrane expression of proteins associated with cell-cell interaction. Furthermore, ceramide treated U937 monocytes demonstrated reduced adhesion to 5 or 24 hour LPS activated human umbilical vein endothelial cells (HUVEC) but not resting HUVEC. Consequently it is hypothesised that the targeted treatment of monocytes from patients with cardiovascular diseases with short chain synthetic ceramide may reduce disease progression. Herein, the anti-inflammatory and immunosuppressant drug, methotrexate, is described to require ROS production for the induction of cytostasis or cytotoxicity in U937 monocytes and Jurkat T-cells respectively. Further, ROS are critical for methotrexate to abrogate monocyte interaction with activated HUVEC in vitro. The histological feature of RA of enhanced infiltration, survivability and hyporesponsiveness of T-cells within the diseased synovium has been suggested to arise from aberrant signalling. No difference in the concentrations of endogenous T-cell ceramide, the related lipid diacylglycerol (DAG) and cytosolic peroxide ex vivo was observed. TCR activation following PHA exposure in vitro for 72 hours did not induced maintained perturbations in DAG or ceramide in T-cells from RA patients or healthy individuals. However, T-cells from RA patients failed to upregulate cytosolic peroxide in response to PHA, unlike those from normals, despite expressing identical levels of the activation marker CD25. This inability to upregulate cytosolic peroxide may contribute to the T-cell pathology associated with RA by affecting the signalling capacity of redox sensitive biomolecules. These data highlight the importance of two distinctive cellular pools of ROS in mediating complex biological events associated with inflammatory disease and suggest that modulation of cellular ceramides represents a novel therapeutic strategy to minimise monocyte recruitment.
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Premnauth, Gurdat. "Design, Synthesis and Biological Evaluation of New Molecules to Selectively Target Specific Cancers." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1613744938434214.

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Wilke, Julian [Verfasser], Herbert [Akademischer Betreuer] Waldmann, and Markus [Gutachter] Kaiser. "Identification and characterization of small molecules inducing cellular reactive oxygen species accumulation / Julian Wilke ; Gutachter: Markus Kaiser ; Betreuer: Herbert Waldmann." Dortmund : Universitätsbibliothek Dortmund, 2020. http://d-nb.info/1225937809/34.

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Kujbida, Paula da Silva. "Efeitos das microcistinas sobre funções de neutrófilos." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/9/9141/tde-05062017-151650/.

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Microcistinas (MCs) são heptapeptídeos cíclicos produzidos por cianobactérias e possuem potente hepatotoxicidade e atividade promotora de tumor. Em intoxicações agudas induzidas por MCs ocorre infiltração leucocitária no foco inflamatório. Embora os mecanismos de hepatotoxicidade não são claros, o recrutamento de neutrófilos no fígado pode contribuir ao dano tecidual e desenvolvimento tumoral causados por xenobióticos. O objetivo dessa tese foi investigar os efeitos de três estruturalmente distintas MCs (MC-LA, MC-YR e MC-LR) nas seguintes funções de neutrófilos: síntese e expressão de moléculas de adesão, rolamento, adesão, migração e liberação de citocinas e de ROS. Nos ensaios de migração em bolsa de ar, as três MCs induziram similarmente a migração de leucócitos in vivo em tecido subcutâneo de ratos e diferencialmente a secreção de citocinas pró-inflamatórias (CINC, IL-1β, TNF-α, VEGF-α e MIP) no exsudato. Concentrações elevadas de CINC-2αβ foram encontradas nos exsudatos inflamatórios de animais após injeção de MC-LA, MC-LR ou MC-YR. MIP-2 elevou-se apenas em exsudatos de animais expostos a MC-LR. Não foram observadas alterações em secreção de IL-1β, TNF-α e VEGF-α. Estudos de microscopia intravital mostraram que apenas a aplicação tópica de MC-LR reforçou o rolamento e a adesão de leucócitos no endotélio de vênulas mesentéricas pós-capilares. Esses últimos resultados podem ser dependentes de aumento e expressão da síntese da L-selectina e β2-integrina nos neutrófilos, tal como avaliado por FACS e PCR-RT, respectivamente. Adicionalmente, em ensaios em câmara de Boyden in vitro, as três MCs promoveram locomoção direta de neutrófilos e aumentaram a migração dessas células em resposta ao fMLP, além do aumento de cálcio intracelular observado por microscopia confocal. Os efeitos das MC-LA, MC-YR e MC-LR em neutrófilos humanos e de ratos tiveram o mesmo padrão de resposta. A análise de viabilidade celular, fragmentação de DNA, despolarização de membrana mitocondrial e liberação de ROS intracelulares foram avaliadas pela técnica de FACS. A concentração de ROS extracelular foi medida por quimiluminescência amplificada por lucigenina. A produção de citocinas em células tratadas foram determinadas por ELISA. Observamos aumento da liberação de IL-8, CINC-2α β e da concentração de ROS extracelular por neutrófilos humanos e de ratos pela exposição dessas células com as MCs. Os resultados aqui obtidos mostram o efeito pró-inflamatório direto das MC-LA, MC-YR e MC-LR. A possibilidade de os neutrófilos migrarem e responderem localmente a MCs contribui para a toxicidade destas toxinas.
Microcystins (MCs) are a family of heptapeptide toxins produced by some genera of Cyanobacteria. MCs have potent hepatotoxicity and tumor-promoting activity. Leukocyte infiltration in the liver was observed in MC-induced acute intoxication. Although the mechanisms of hepatotoxicity induced by MCs are still unclear, neutrophil infiltration in the liver may play an important role in triggering toxic injury and tumor development. The purpose of this thesis was to investigate the effects of three structurally distinct MCs (MC-LA, MC-YR and MC-LR) in the neutrophil functions: synthesis and expression of adhesion molecules, rolling, adhesion, migration and release of cytokines and ROS. In migration assays of the air pouch, the three MCs similarly induced the migration of leukocytes in vivo in subcutaneous tissue of rats and differentially the secretion of pro-inflammatory cytokines (CINC-2αβ, IL-1-β, TNF--α, VEGF- α and MIP-2) in exudates. Elevated concentrations of CINC-2αβ were found in the inflamed exudates from animals injected with MC-LA, MC-LR or MC-YR, although MIP-2 was only detected in the exudates from animals injected with MC-LR. There were no changes in the secretion of IL-1-β, TNF-α and VEGF--α. Intravital microscopic studies showed that topical application of MC-LR enhanced the numbers of rolling and adhered leukocytes in the endothelium of postcapillary mesenteric venules. The latter effects may be dependent upon induction of the synthesis and expression of L-selectin and -α2-integrin in neutrophils, as assessed by flow cytometry and RT-PCR, respectively. Conversely, the three toxins promoted direct locomotion of neutrophils and enhanced their migration in response to fMLP, as measured by Boyden chamber assays, and increased intracellular calcium, a messenger in the chemotaxic process. The effects of MC-LA, MC-YR and MC-LR in human neutrophils and mice had the same pattern of response. The analyses of cell viability, DNA fragmentation, mitochondrial membrane depolarization of and release of intracellular ROS were evaluated by the technique of FACS. Extracellular ROS content was measured by lucigenin-amplified chemiluminescence, and cytokines were determined by ELISA. We found that these MCs increased interleukin-8 (IL-8), cytokine-induced neutrophil chemoattractant-2αβ (CINC-2αβ) and extracellular ROS levels in human and rat neutrophils. In conclusion, our results showed that MCs act on specific pathways of neutrophil recruitment, indicating their potential effect on neutrophils activation. This process can significantly contribute to the pathogenesis of hepatic damage due to generation of ROS by neutrophils as well as act on hepatocytes under such conditions and potentially increase injury processes induced by MCs.
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Tikhomirova, Anastasiia. "Studies of Photoinduced DNA Damage by Phenanthrene Dihydrodioxin and Light-driven Electron Delocalization in Pyridinium Molecules." Bowling Green State University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1561918589357022.

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Perez-Rodriguez, Jacqueline. "ROLE OF OXIDATIVE STRESS AND T CELL HOMING IN THE DEVELOPMENT OF MURINE SYNGENEIC GRAFT-VERSUS-HOST DISEASE." UKnowledge, 2009. http://uknowledge.uky.edu/gradschool_diss/804.

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Syngeneic graft-versus-host disease (SGVHD) is induced by reconstituting lethally irradiated mice with syngeneic BM cells followed by a 21 day treatment with the immunosuppressive agent cyclosporine A (CsA). Clinical symptoms of the disease appear 2-3 weeks following cessation of CsA therapy and disease-associated inflammation occurs primarily in the colon and liver. The development of SGVHD is a complex process resulting from the cooperative interaction of multiple effector cell populations including NK cells, T cells and macrophages. TH1 cytokines (IL-12, TNF-α, IFN- γ), produced by these effector cells, serve as inflammatory mediators contributing to the pathogenesis of SGVHD. The SGVHD conditioning agents, irradiation and CsA, are both required for the development of disease and contribute to the production of oxidative stress. Time course studies revealed increased reactive oxygen and nitrogen species (ROS/RNS), as well as, increased colon mRNA levels for TNF-α and iNOS in CsA-treated versus control BMT animals. Since ROS/RNS are known to mediate CsA toxicity, studies were undertaken to determine the effect of oxidative stress on the induction of SGVHD. In vivo treatment with the antioxidant MnTBAP caused a reduction in colon mRNA levels for iNOS and TNF-α, as well as delayed disease development, suggesting a role for oxidative stress in the development of SGVHD. In addition, CD4+ T cells have been shown to play an important role in the inflammatory response observed in the gut of SGVHD mice. Time course studies revealed significant increases in the migration of CD4+ T cells as early as day 14 post- BMT into the colon of CsA mice as well as significant elevated mRNA levels of cell adhesion molecules. Homing studies revealed that a labeled CD4+ T cell line, generated from SGVHD mice, migrated in larger numbers into the gut of CsA-treated mice compared to control animals. This study demonstrated that CD4+ T cells responsible for the pathogenesis observed in murine SGVHD are present early after BMT in colons of CsA-treated mice, suggesting that during the 21 days of immunosuppression therapy functional mechanisms are in place that result in increased homing of effector cells to colons of CsA-treated mice.
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Pollum, Marvin. "Applying Fundamental Photochemistry to Drive Drug Development: The Photo-Dynamics and Reactions of Sulfur-Substituted Nucleic Acids." Case Western Reserve University School of Graduate Studies / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=case1481287737895585.

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BOCCALINI, GIULIA. "CELLULAR MODELS OF HYPOXIA-REOXYGENATION FOR THE STUDY OF NEW MOLECULES WITH THERAPEUTIC POTENTIAL IN ISCHEMIC HEART DISEASE." Doctoral thesis, 2015. http://hdl.handle.net/2158/1045351.

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Pre-clinical studies of new molecules which can be developed as possible therapeutic effectors: in particular, the study of the protective effects of the hormone relaxin, as well as scavenger molecules of oxygen reactive species on cardiac hypoxia/reoxygenation.
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Vaňková, Kateřina. "Biologické účinky jedlých řas." Doctoral thesis, 2018. http://www.nusl.cz/ntk/nusl-388763.

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Nutritional factors with antioxidant properties, such as those contained in edible algae or green plants, might be implicated in protection against cancer development. Chlorophyll and other tetrapyrrolic compounds, structurally related to heme and antioxidant bile pigment bilirubin, belong to important candidate molecules, which might be responsible for these effects. Based on our studies demonstrating antiproliferative effects of S. platensis edible alga extract on experimental model of human pancreatic adenocarcinoma we investigated in detail the effect of chlorophyll occurring abundantly in this alga. Since only scarce data exist on the antiproliferative effects of chlorophylls, the aim of our study was to assess these effects. The study was performed on experimental models of human pancreatic and prostate cancer. The inhibitory effects of chlorophylls (chlorophyll a, chlorophyll b, chlorophyllin and pheophytin a) on cell proliferation and cell viability were investigated in in vitro studies. Chlorophylls reduced the mRNA expression as well as activity of hemeoxygenase in tested pancreatic cancer cells. Simultaneously, chlorophylls played an important role in redox environment of studied cancer cell lines including modulation of mitochondrial membrane potential, reactive oxygen species (ROS)...
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Books on the topic "Scavenger molecules of oxygen reactive species"

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Lo, Yvonne Yim Chung. Reactive oxygen species as signaling molecules regulating chondrocyte gene expression of fox, jun and collagenase. Ottawa: National Library of Canada, 1995.

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Book chapters on the topic "Scavenger molecules of oxygen reactive species"

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Martemyanov, Kirill A., Pooja Parameswaran, Irene Aligianis, Mark Handley, Marga Gual-Soler, Tomohiko Taguchi, Jennifer L. Stow, et al. "ROS (Reactive Oxygen Species)." In Encyclopedia of Signaling Molecules, 1691. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101203.

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Rovira, Ilsa I., and Toren Finkel. "Reactive Oxygen Species as Signaling Molecules." In Oxidative Stress in Aging, 293–307. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-420-9_16.

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Desikan, Radhika, John Hancock, and Steven Neill. "Reactive Oxygen Species as Signalling Molecules." In Antioxidants and Reactive Oxygen Species in Plants, 169–96. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470988565.ch7.

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Yadav, Divyansh, and Seema Nara. "Nanozymes for Neurodegenerative Diseases." In Proceedings of the Conference BioSangam 2022: Emerging Trends in Biotechnology (BIOSANGAM 2022), 77–95. Dordrecht: Atlantis Press International BV, 2022. http://dx.doi.org/10.2991/978-94-6463-020-6_9.

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AbstractNeurodegenerative diseases are incurable diseases that get worse as time passes. These diseases are very heterogeneous in nature but have common characteristics like abnormal deposition of protein, glycation, inflammation in particular areas of the brain, and progressive neuronal loss due to oxidative stress. Among these, oxidative stress alone causes a high level of degeneration of neurons. To reduce oxidative stress, natural antioxidants are used but they have some drawbacks like instability, high cost and low reusability. To overcome this, nanozymes are introduced and we have emphasized on major nanozymes whose antioxidant capability has been proven which are gold nanozymes, fullerene, nanoceria, and quantum dots. Gold nanoparticles and their conjugates with other molecules can mimic the enzymatic activity of superoxide dismutase and catalase which decrease the amount of hydrogen peroxide and superoxide radicals in cells. Gold Nanozyme treatment reduces the oxidative stress, nitrite, and sulfhydryl levels in the brain and also rectifies the superoxide dismutase, glutathione, and catalase activity levels. Fullerenols has shown superoxide dismutase activity which was 268 times more effective than mannitol and 37 times more effective than Vitamin E for lipid radicals. Nanoceria has the ability to mimic Superoxide Dismutase as well as catalase activity, can also detoxify peroxynitrite. Quantum dots (QDs) like Graphene Oxide QDs can scavenge the reactive oxygen species and also show indirect activity which alleviates the pathogenesis of the disease. Thus, a nanozyme can be used as an efficient nanomedicine if it is tailored to possess high catalytic activity while eliminating all complications.
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del Río, Luis A., Luisa M. Sandalio, Francisco J. Corpas, María C. Romero-Puertas, and José M. Palma. "Peroxisomes as a Cellular Source of ROS Signal Molecules." In Reactive Oxygen Species in Plant Signaling, 95–111. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-00390-5_6.

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Goswami, Shyamal K. "Aging, Free Radicals, and Reactive Oxygen Species: An Evolving Concept." In Models, Molecules and Mechanisms in Biogerontology, 199–212. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-32-9005-1_11.

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Zaid, Abbu, and Shabir H. Wani. "Reactive Oxygen Species Generation, Scavenging and Signaling in Plant Defense Responses." In Bioactive Molecules in Plant Defense, 111–32. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-27165-7_7.

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Das, Suman, and Dhermendra K. Tiwari. "Reactive Oxygen Species Producing Photoactivatable Molecules and Their Biological Applications." In Free Radical Biology and Environmental Toxicity, 21–41. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-83446-3_2.

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Sandalio, Luisa M., María Rodríguez-Serrano, María C. Romero-Puertas, and Luis A. del Río. "Role of Peroxisomes as a Source of Reactive Oxygen Species (ROS) Signaling Molecules." In Peroxisomes and their Key Role in Cellular Signaling and Metabolism, 231–55. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6889-5_13.

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Indo, Hiroko P., Clare L. Hawkins, Ikuo Nakanishi, Ken-ichiro Matsumoto, Hirofumi Matsui, Shigeaki Suenaga, Michael J. Davies, Daret K. St Clair, Toshihiko Ozawa, and Hideyuki J. Majima. "Role of Mitochondrial Reactive Oxygen Species in the Activation of Cellular Signals, Molecules, and Function." In Handbook of Experimental Pharmacology, 439–56. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/164_2016_117.

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Conference papers on the topic "Scavenger molecules of oxygen reactive species"

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YOSHIKI, Y., Y. AKIYAMA, K. ABE, and K. OKUBO. "THE QUANTIFICATION OF REACTIVE OXYGEN SPECIES AND THEIR SCAVENGER FROM PHOTON EMISSION." In Proceedings of the 11th International Symposium. WORLD SCIENTIFIC, 2001. http://dx.doi.org/10.1142/9789812811158_0091.

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Shafiee, Ashkan, Elham Ghadiri, Muhamad Mat Salleh, Muhammad Yahaya, and Anthony Atala. "Inkjet Printing Of A Reactive Oxygen Species Scavenger For Flexible Bioelectronics Applications In Neural Resilience." In 2018 International Flexible Electronics Technology Conference (IFETC). IEEE, 2018. http://dx.doi.org/10.1109/ifetc.2018.8583903.

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Yuan, Long, Rosalin Mishra, Hima Patel, Samar Alanazi, and Joan Garrett. "Abstract 4900: Reactive oxygen species scavenger extends the efficacy of BRAF inhibitors in BRAF-mutant melanoma." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-4900.

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Tekutskaya, E., I. Raybova, and Lyubov Ramazanovna Gusaruk. "THE DEGREE OF OXIDATIVE DAMAGE TO DNA IN VITRO AS A MOLECULAR PREDICTOR OF DISORDERS CAUSED BY EPIGENETIC AND EXOGENOUS FACTORS." In NEW TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2021. http://dx.doi.org/10.47501/978-5-6044060-1-4.49.

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In this work, we studied the mechanisms of oxidative damage to DNA molecules isolated from whole blood of healthy donors and patients with epigenetic disease (epidermolysis bullosa) when exposed to an alternating magnetic field of low frequency in vitro, associated with the formation of reactive oxygen species.
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Schaffer, W. M., and T. V. Bronnikova. "Modeling Peroxidase-Oxidase Interactions." In ASME 2011 Dynamic Systems and Control Conference and Bath/ASME Symposium on Fluid Power and Motion Control. ASMEDC, 2011. http://dx.doi.org/10.1115/dscc2011-5946.

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Reactive oxygen species (ROS) and peroxidase-oxidase (PO) reactions are Janus-faced contributors to cellular metabolism. At low concentrations, reactive oxygen species serve as signaling molecules; at high concentrations, as destroyers of proteins, lipids and DNA. Correspondingly, PO reactions are both sources and consumers of ROS. In the present paper, we study a well-tested model of the PO reaction based on horseradish peroxidase chemistry. Our principal predictions are these: 1. Under hypoxia, the PO reaction can emit pulses of hydrogen peroxide at apparently arbitrarily long intervals. 2. For a wide range of input rates, continuing infusions of ROS are transduced into bounded dynamics. 3. The response to ROS input is hysteretic. 4. With sufficient input, regulatory capacity is exceeded and hydrogen peroxide, but not superoxide, accumulates. These results are discussed with regard to the episodic nature of neurodevelopmental and neurodegenerative diseases that have been linked to oxidative stress and to downstream interactions that may result in positive feedback and pathology of increasing severity.
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Görög, P., J. D. Pearson, and V. V. Kakkar. "GENERATION OF REACTIVE OXYGEN METABOLITES BY PHAGOCYTOSING ENDOTHELIAL CELLS: REGULATORY ROLE OF THE GLYCOCALYX." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642862.

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We have studied the ability of particular stimuli to induce the release of reactive oxygen metabolites from sub-cultured monolayers of human umbilical vein endothelial cells. Superoxide production was measured as the superoxide dismutase-inhibitable reduction of exogenous cytochrome C, and H2O2 production was measured by the horseradish peroxidase catalysed oxidation of scopoletin. Basal release of either metabolite from undisturbed monolayers was low (80pmol O2 and 65pmol H2O2 in 3 h from dishes of 3×108 cells). Addition of 1μM diameter polystyrene microspheres (4.5×108 particles/dish) which were phagocytosed by the cells progressively over 3h, caused a dramatic increase in release of both metabolites (2nmol O2 and 490pmol H2O2 at 3h). Release of O2 was linear, whereas release of H2O2 was more rapid in the first hour. Addition of formaldehyde-fixed human platelets (2.5×l06/dish) significantly enhanced O2 production (490pmol at 2h) and raised, thought not significantly, H2O2 release (130pmol at 2h); addition of a stabilized lipid emulsion (lipofundin-S ; particle diameter 1μm, 3.6×108 particles/dish) caused a significant rise in O2 production (1.3nmol at 2h) but measurement of its effect on H2O2 release was prevented by interference in the assay. Similar rises in H2O2 and O2 production were induced by treatment with 10™7M phorbol myristate acetate. Pretreatment of endothelial cells with 12.5mU /ml neuraminidase or 0.5U/ml heparinase for 60 min to alter their glycocalyx composition substantially enhanced the effect of microspheres on H2O2 and O2 generation, particularly at early times (lh). We conclude that the interactions of particulates, including platelets and lipids, with endothelial cells can lead to the generation of significant pericellular levels of reactive oxygen species. These metabolites may oxidise a wide variety of nearby molecules, leading to cell damage and altered uptake characteristics for lipoproteins containing peroxidised lipids. These effects are exacerbated when endothelial cell glycocalyx composition is disrupted.
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Durand, Erwann, Nastassia Kaugarenia, Nathalie Barouh, Pierre Villeneuve, and Romain Kapel. "Antioxidant chelating peptides production from Rapeseed meal proteins proteolysis." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/whcd7145.

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The oxidative chemical degradation produced by reactive species (free radicals, oxygen, etc.) is responsible for the deterioration of most of the formulated products. One of the main properties of an antioxidant lies in its capacity to limit the chemical propagation of oxidation by reducing free radicals. Another strategy to prevent oxidation is binding transition metals, since they are ubiquitous and deeply involved in the initiation and propagation of lipids oxidation. Naturally occurring phospholipids, polyphenols, proteins, or peptides that can bind metal ions could be more valued than synthetic molecules, for human wellbeing, but also to align with consumer preferences. Yet, EDTA salts and sodium citrate remain the most common metal chelators in foods. In this study, we went to investigate a strategy to develop naturally produced antioxidants peptides from edible plant biomass, such as rapeseed. Several enzymatic hydrolyses of total rapeseed protein isolate with various proteases have been performed, and the produced peptides were screened for their antioxidant capacity. Peptides generated with Prolyve® allowed for particularly high Fe2+ chelation capacity (EC50 = 247 ± 27 µg). Accordingly, the enzymatic processing step with Prolyve® was modeled and optimized to minimize reaction costs and maximize peptide recovery. Then, lipid oxidation was studied in the presence or in the absence of chelating peptides, in micellar, bulk, and oil-in-water emulsion systems, and compared with EDTA salts and sodium citrate. Results clearly emphasized a very interesting potential from the peptides sample to prevent lipid oxidation by chelation of transition metals in emulsified models.This result is particularly important to develop the potential of applications of rapeseed meal in various food formulations. In addition, this study emphasized an approach aiming at developing food chelator peptides from plant proteins, having multifunctional properties, and through sustainable processing.
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Reports on the topic "Scavenger molecules of oxygen reactive species"

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Dickman, Martin B., and Oded Yarden. Characterization of the chorismate mutase effector (SsCm1) from Sclerotinia sclerotiorum. United States Department of Agriculture, January 2015. http://dx.doi.org/10.32747/2015.7600027.bard.

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Sclerotinia sclerotiorum is a filamentous fungus (mold) that causes plant disease. It has an extremely wide range of hosts (>400 species) and causes considerable damage (annual multimillion dollar losses) in economically important crops. It has proven difficult to control (culturally or chemically) and host resistance to this fungus has generally been inadequate. It is believed that this fungus occurs in almost every country. Virulence of this aggressive pathogen is bolstered by a wide array of plant cell wall degrading enzymes and various compounds (secondary metabolites) produced by the fungus. It is well established that plant pathogenic fungi secrete proteins and small molecules that interact with host cells and play a critical role in disease development. Such secreted proteins have been collectively designated as “effectors”. Plant resistance against some pathogens can be mediated by recognition of such effectors. Alternatively, effectors can interfere with plant defense. Some such effectors are recognized by the host plant and can culminate in a programmed cell death (PCD) resistant response. During the course of this study, we analyzed an effector in Sclerotiniasclerotiorum. This specific effector, SsCM1 is the protein chorismatemutase, which is an enzyme involved in a pathway which is important in the production of important amino acids, such a Tryptophan. We have characterized the Sclerotiniaeffector, SsCM1, and have shown that inactivation of Sscm1 does not affect fungal vegetative growth, development or production of oxalic acid (one of this fungus’ secondary metabolites associated with disease) production. However, yhis does result in reduced fungal virulence. We show that, unexpectedly, the SsCM1 protein translocates to the host chloroplast, and demonstrated that this process is required for full fungal virulence. We have also determined that the fungal SsCM1 protein can interact with similar proteins produced by the host. In addition, we have shown that the fungal SsCM1 is able to suppress at least some of the effects imposed by reactive oxygen species which are produced as a defense mechanism by the host. Last, but not least, the results of our studies have provided evidence contradicting the current dogma on at least some of the mechanist aspects of how this pathogen infects the host. Contrary to previousons, indicating that this pathogen kills its host by use of metabolites and enzymes that degrade the host tissue (a process called necrotrophy), we now know that at least in the early phases of infection, the fungus interacts with live host tissue (a phenomenon known as biotrophy). Taken together, the results of our studies provide novel insights concerning the mechanistic aspects of Sclerotinia-host interactions. We hope this information will be used to interfere with the disease cycle in a manner that will protect plants from this devastating fungus.
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Shenker, Moshe, Paul R. Bloom, Abraham Shaviv, Adina Paytan, Barbara J. Cade-Menun, Yona Chen, and Jorge Tarchitzky. Fate of Phosphorus Originated from Treated Wastewater and Biosolids in Soils: Speciation, Transport, and Accumulation. United States Department of Agriculture, June 2011. http://dx.doi.org/10.32747/2011.7697103.bard.

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Beneficial use of reclaimed wastewater (RW) and biosolids (BS) in soils is accompanied by large input of sewage-originated P. Prolonged application may result in P accumulation up to levelsBeneficial use of reclaimed wastewater (RW) and biosolids (BS) in soils is accompanied by large input of sewage-originated P. Prolonged application may result in P accumulation up to levels that impair plant nutrition, increase P loss, and promote eutrophication in downstream waters. This study aims to shed light on the RW- and BS-P forms in soils and to follow the processes that determine P reactivity, solubility, availability, and loss in RW and BS treated soils. The Technion group used sequential P extraction combined with measuring stable oxygen isotopic composition in phosphate (δ18OP) and with 31P-NMR studies to probe P speciation and transformations in soils irrigated with RW or fresh water (FW). The application of the δ18OP method to probe inorganic P (Pi) speciation and transformations in soils was developed through collaboration between the Technion and the UCSC groups. The method was used to trace Pi in water-, NaHCO3-, NaOH-, and HCl- P fractions in a calcareous clay soil (Acre, Israel) irrigated with RW or FW. The δ18OP signature changes during a month of incubation indicated biogeochemical processes. The water soluble Pi (WSPi) was affected by enzymatic activity yielding isotopic equilibrium with the water molecules in the soil solution. Further it interacted rapidly with the NaHCO3-Pi. The more stable Pi pools also exhibited isotopic alterations in the first two weeks after P application, likely related to microbial activity. Isotopic depletion which could result from organic P (PO) mineralization was followed by enrichment which may result from biologic discrimination in the uptake. Similar transformations were observed in both soils although transformations related to biological activity were more pronounced in the soil treated with RW. Specific P compounds were identified by the Technion group, using solution-state 31P-NMR in wastewater and in soil P extracts from Acre soils irrigated by RW and FW. Few identified PO compounds (e.g., D-glucose-6-phosphate) indicated coupled transformations of P and C in the wastewater. The RW soil retained higher P content, mainly in the labile fractions, but lower labile PO, than the FW soil; this and the fact that P species in the various soil extracts of the RW soil appear independent of P species in the RW are attributed to enhanced biological activity and P recycling in the RW soil. Consistent with that, both soils retained very similar P species in the soil pools. The HUJ group tested P stabilization to maximize the environmental safe application rates and the agronomic beneficial use of BS. Sequential P extraction indicated that the most reactive BS-P forms: WSP, membrane-P, and NaHCO3-P, were effectively stabilized by ferrous sulfate (FeSul), calcium oxide (CaO), or aluminum sulfate (alum). After applying the stabilized BS, or fresh BS (FBS), FBS compost (BSC), or P fertilizer (KH2PO4) to an alluvial soil, P availability was probed during 100 days of incubation. A plant-based bioassay indicated that P availability followed the order KH2PO4 >> alum-BS > BSC ≥ FBS > CaO-BS >> FeSul-BS. The WSPi concentration in soil increased following FBS or BSC application, and P mineralization further increased it during incubation. In contrast, the chemically stabilized BS reduced WSPi concentrations relative to the untreated soil. It was concluded that the chemically stabilized BS effectively controlled WSPi in the soil while still supplying P to support plant growth. Using the sequential extraction procedure the persistence of P availability in BS treated soils was shown to be of a long-term nature. 15 years after the last BS application to MN soils that were annually amended for 20 years by heavy rates of BS, about 25% of the added BS-P was found in the labile fractions. The UMN group further probed soil-P speciation in these soils by bulk and micro X-ray absorption near edge structure (XANES). This newly developed method was shown to be a powerful tool for P speciation in soils. In a control soil (no BS added), 54% of the total P was PO and it was mostly identified as phytic acid; 15% was identified as brushite and 26% as strengite. A corn crop BS amended soil included mostly P-Fe-peat complex, variscite and Al-P-peat complex but no Ca-P while in a BS-grass soil octacalcium phosphate was identified and o-phosphorylethanolamine or phytic acid was shown to dominate the PO fraction that impair plant nutrition, increase P loss, and promote eutrophication in downstream waters. This study aims to shed light on the RW- and BS-P forms in soils and to follow the processes that determine P reactivity, solubility, availability, and loss in RW and BS treated soils. The Technion group used sequential P extraction combined with measuring stable oxygen isotopic composition in phosphate (δ18OP) and with 31P-NMR studies to probe P speciation and transformations in soils irrigated with RW or fresh water (FW). The application of the δ18OP method to probe inorganic P (Pi) speciation and transformations in soils was developed through collaboration between the Technion and the UCSC groups. The method was used to trace Pi in water-, NaHCO3-, NaOH-, and HCl- P fractions in a calcareous clay soil (Acre, Israel) irrigated with RW or FW. The δ18OP signature changes during a month of incubation indicated biogeochemical processes. The water soluble Pi (WSPi) was affected by enzymatic activity yielding isotopic equilibrium with the water molecules in the soil solution. Further it interacted rapidly with the NaHCO3-Pi. The more stable Pi pools also exhibited isotopic alterations in the first two weeks after P application, likely related to microbial activity. Isotopic depletion which could result from organic P (PO) mineralization was followed by enrichment which may result from biologic discrimination in the uptake. Similar transformations were observed in both soils although transformations related to biological activity were more pronounced in the soil treated with RW. Specific P compounds were identified by the Technion group, using solution-state 31P-NMR in wastewater and in soil P extracts from Acre soils irrigated by RW and FW. Few identified PO compounds (e.g., D-glucose-6-phosphate) indicated coupled transformations of P and C in the wastewater. The RW soil retained higher P content, mainly in the labile fractions, but lower labile PO, than the FW soil; this and the fact that P species in the various soil extracts of the RW soil appear independent of P species in the RW are attributed to enhanced biological activity and P recycling in the RW soil. Consistent with that, both soils retained very similar P species in the soil pools. The HUJ group tested P stabilization to maximize the environmental safe application rates and the agronomic beneficial use of BS. Sequential P extraction indicated that the most reactive BS-P forms: WSP, membrane-P, and NaHCO3-P, were effectively stabilized by ferrous sulfate (FeSul), calcium oxide (CaO), or aluminum sulfate (alum). After applying the stabilized BS, or fresh BS (FBS), FBS compost (BSC), or P fertilizer (KH2PO4) to an alluvial soil, P availability was probed during 100 days of incubation. A plant-based bioassay indicated that P availability followed the order KH2PO4 >> alum-BS > BSC ≥ FBS > CaO-BS >> FeSul-BS. The WSPi concentration in soil increased following FBS or BSC application, and P mineralization further increased it during incubation. In contrast, the chemically stabilized BS reduced WSPi concentrations relative to the untreated soil. It was concluded that the chemically stabilized BS effectively controlled WSPi in the soil while still supplying P to support plant growth. Using the sequential extraction procedure the persistence of P availability in BS treated soils was shown to be of a long-term nature. 15 years after the last BS application to MN soils that were annually amended for 20 years by heavy rates of BS, about 25% of the added BS-P was found in the labile fractions. The UMN group further probed soil-P speciation in these soils by bulk and micro X-ray absorption near edge structure (XANES). This newly developed method was shown to be a powerful tool for P speciation in soils. In a control soil (no BS added), 54% of the total P was PO and it was mostly identified as phytic acid; 15% was identified as brushite and 26% as strengite. A corn crop BS amended soil included mostly P-Fe-peat complex, variscite and Al-P-peat complex but no Ca-P while in a BS-grass soil octacalcium phosphate was identified and o-phosphorylethanolamine or phytic acid was shown to dominate the PO fraction.
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