Relevant bibliographies by topics / Saphenous Vein Graft Disease

Academic literature on the topic 'Saphenous Vein Graft Disease'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Saphenous Vein Graft Disease"

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Rao, Sarita, K. Roshan Rao, and Achukatla Kumar. "Saphenous Vein Graft Disease Interventions." Indian Journal of Cardiovascular Disease in Women - WINCARS 06, no. 03 (July 2021): 199–208. http://dx.doi.org/10.1055/s-0041-1736323.

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AbstractIn the current era, coronary artery bypass grafting (CABG) is being increasingly performed using total arterial revascularization or a hybrid procedure of stenting of non-LAD disease and minimal access left internal mammary artery (LIMA) to LAD grafts, in order to minimize the need for vein grafts. Still, we encounter saphenous vein graft (SVG) disease, and it might require PCI, which often presents with unique challenges. The current favored strategy is to attempt PCI of the native coronary, if feasible, especially in long degenerated SVG disease, as it has shown better short- and long-term outcome. PCI is preferred over repeat CABG for early recurrent symptoms after CABG in patent LIMA graft and amenable anatomy patients. Balloon predilatation is not recommended unless delivery of an EPD or stent is not possible. Distal protection should be considered the standard of care for percutaneous coronary intervention (PCI) in most patients with older vein grafts, as periprocedural myocardial infarction and no reflow are the Achilles heel of SVG PCI. Intragraft vasodilators should be used liberally, even before balloon angioplasty/stenting. Avoid postdilatation, and usage of undersized but a longer stent length to reduce plaque extrusion through stent struts is preferred. Consider thrombectomy in lesions with a heavy thrombus burden. Keep activated clotting time on the higher side than in conventional PCI. Prolonged dual antiplatelet therapy (DAPT) based on the DAPT score is recommended. With all the precautions and care, we still need a fair wind in our favor to sail through the vein grafts disease.
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Kim, Francis Y., Gregary Marhefka, Nicholas J. Ruggiero, Suzanne Adams, and David J. Whellan. "Saphenous Vein Graft Disease." Cardiology in Review 21, no. 2 (2013): 101–9. http://dx.doi.org/10.1097/crd.0b013e3182736190.

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Gedela, Maheedhar, Shenjing Li, Udit Bhatnagar, Adam Stys, and Tomasz Stys. "Orbital Atherectomy and Heavily Calcified Saphenous Vein Graft Intervention." Texas Heart Institute Journal 47, no. 1 (February 1, 2020): 41–43. http://dx.doi.org/10.14503/thij-18-6640.

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Percutaneous coronary intervention in the diseased saphenous vein graft differs significantly from that in the diseased native coronary artery. After being exposed to arterial pressures over time, vein grafts have substantially different plaque characteristics, with more inflammatory cells, more diffuse disease, and less calcification. Severe calcification of saphenous vein grafts, although uncommon, poses a high risk of stent underexpansion. Orbital atherectomy for treatment of de novo calcified coronary lesions has been associated with better outcomes at 5-year follow-up. However, there are no published data on the use of orbital atherectomy to treat severely calcified saphenous vein graft lesions. We present the case of a 77-year-old woman with non-ST-segment-elevation myocardial infarction who underwent successful orbital atherectomy to prepare a severely calcified saphenous vein graft lesion for stent implantation.
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Motwani, Joseph G., and Eric J. Topol. "Aortocoronary Saphenous Vein Graft Disease." Circulation 97, no. 9 (March 10, 1998): 916–31. http://dx.doi.org/10.1161/01.cir.97.9.916.

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Demircelik, Bora, Muzaffer Cakmak, Yunus Nazli, Ozgul M. Gurel, Nermin Akkaya, Mustafa Cetin, Zehra Cetin, Yusuf Selcoki, Alparslan Kurtul, and Beyhan Eryonucu. "Adropin: A New Marker for Predicting Late Saphenous Vein Graft Disease after Coronary Artery Bypass Grafting." Clinical & Investigative Medicine 37, no. 5 (October 4, 2014): 338. http://dx.doi.org/10.25011/cim.v37i5.22014.

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Purpose: Saphenous vein graft disease (SVGD), defined as an occlusion of 50% or more of the SVG excluding distal anastomotic occlusion, is an important predictor of morbidity after coronary artery bypass grafting (CABG). Late graft occlusion is a serious complication that often limits the use of the saphenous vein as a coronary bypass graft. Late graft occlusion is particularly common in old, degenerated venous grafts with advanced atherosclerotic plaques. Adropin has been implicated in the homeostatic control of metabolism. The purpose of this study was to investigate whether serum adropin levels are associated with late SVGD following CABG. Methods: Thirty-eight patients with SVGD involving at least one graft (occluded group; 14 females, 24 males) and 42 patients with a patent saphenous vein graft (patent group; 15 females, 27 males) were enrolled in this study. Venous blood samples were taken from all of the participants to measure plasma adropin levels using an enzyme-linked immunsorbent assay kit. Results: The mean adropin level was significantly lower in the occluded group than in the patent group (3.2 ± 0.71 vs. 4.9 ± 1.51 ng/mL, p < 0.001). Multivariate regression analysis showed that the adropin level was the independent predictor of late saphenous vein graft occlusion. Conclusions: Adropin levels are lower in patients with late saphenous vein graft occlusion and these reduced adropin levels, together with other factors, may lead to saphenous vein graft occlusion. Larger and prospective studies are needed to determine if adropin plays a role in the pathogenesis of SVGD.
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Tepelenis, Kostas, Georgios Papathanakos, Alexandra Barbouti, Georgios Paraskevas, Aikaterini Kitsouli, Maria Alexandra Kefala, Nikolaos Tepelenis, Panagiotis Kanavaros, and Panagiotis Kitsoulis. "Phlebosclerosis in lower extremities veins – a systematic review." Vasa 49, no. 5 (August 2020): 349–58. http://dx.doi.org/10.1024/0301-1526/a000868.

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Summary. Phlebosclerosis is a venous wall degenerative disease which has gained little popularity in the literature due to its uncertain clinical significance. The objective of this review is to evaluate the epidemiology, etiology and clinical significance of phlebosclerosis in lower extremities veins, particularly the effect of preexisting phlebosclerosis of the great saphenous vein on vein graft patency. Medline was searched from inception until November 1, 2019. Reference lists of included studies were scanned. Only articles published after 1949 were included. Two reviewers independently screened titles/abstracts and full-text papers for any study design in relation to phlebosclerosis in lower extremities veins and abstracted data. A total of 16 Cohort studies and one case-control study (3708 participants, mean age 61.8 years, 59.3 % men, and 40.7 % women) were included after screening 317 titles and abstracts, and 80 full-text articles. The incidence of phlebosclerosis ranged from 1.5–9.7 % depending on the radiological features. On the contrary, the incidence of the phlebosclerotic great saphenous vein prior to its use as a vein graft was 26.9–91 % on histological examination. The small saphenous vein was the most common location of phlebosclerosis followed by the great saphenous vein. There is a link between phlebosclerosis and age, venous insufficiency and haemodialysis. As for the vein graft patency seven studies demonstrated a correlation between preexisting phlebosclerosis and vein graft stenosis, whereas three studies failed to prove any association. In conclusion, the radiological incidence of phlebosclerosis depended on the ultrasound findings. Its presence in the great saphenous vein prior to its use as a vein graft is established on histological examination. The small saphenous vein is mainly affected. Risk factors included age, haemodialysis, and venous insufficiency. Preexisting wall thickness of the great saphenous vein graft seemed to affect negatively its patency in bypass surgery.
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Lau, G. T., H. C. Lowe, and L. Kritharides. "Cardiac Saphenous Vein Bypass Graft Disease." Seminars in Vascular Medicine 4, no. 2 (May 2004): 153–59. http://dx.doi.org/10.1055/s-2004-835373.

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Aydın, Cihan, and Mustafa Abanoz. "The roles of CHA2DS2-VASc score and blood inflammatory parameters in predicting the patency of saphenous vein grafts in patients with coronary artery bypass graft surgery." Medical Science and Discovery 8, no. 10 (October 18, 2021): 601–7. http://dx.doi.org/10.36472/msd.v8i10.617.

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Objective: Coronary artery bypass graft (CABG) surgery is a common treatment method in which saphenous vein grafts (SVG) and arterial grafts are used together in severe coronary artery disease. The CHA2DS2-VASc score is used to predict thromboembolic events in nonvalvular atrial fibrillation as well as to predict prognosis in cardiovascular events. In this study, we planned to research the relation between CHA2DS2-VASc score and postoperative SVG patency rates in patients undergoing CABG. Materials and Methods: One hundred seventeen patients with angina after CABG surgery who underwent coronary angiography were analyzed retrospectively. Stenosis of 50% or more in at least one saphenous vein graft was accepted as saphenous vein graft disease (SVGD). We compared these patients in two groups concerning the presence of 50% or more stenosis in the SVG. These two groups were; Group 1 (n = 66); with saphenous vein graft disease, Group 2 (n = 51) without saphenous vein graft disease, respectively. Results: A total of 117 patients participating in the study. Sixty-six patients in group 1 had SVGD (Mean age: 68,13±8,22, 60,6% male). Fifty-one patients in group 2 did not have SVGD (Mean age: 66,92±9,44, 72,5% male ). The mean CHA2DS2-VASc score was significantly higher in group 1 compared to group 2. [5 (2-7) vs. 2 (1-7), respectively, P<0,001]. As a result of multivariate analysis, CHA2DS2-VASc score (OR: 5,263, CI 95%: 2,176- 12,728, P<0.001) and SII (OR: 1,236, CI 95%: 1,120-2,955, P=0.007) were determined as independent predictors for predicting SVGD Conclusion: In the light of the results we have found, the CHA2DS2-VASc score and SII, which are easy to calculate in daily practice, can help us in predicting SVGD.
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Alexander, Jason, Charles Gutierrez, and Steven Katz. "Non-Greater Saphenous Vein Grafting for Infrageniculate Bypass." American Surgeon 68, no. 7 (July 2002): 611–14. http://dx.doi.org/10.1177/000313480206800711.

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Infrainguinal bypass grafting with greater saphenous vein has proven to be a highly effective procedure with primary 5-year patency and limb salvage rates exceeding 80 per cent. However, because of prior usage or intrinsic venous disease the greater saphenous vein is often not available as a conduit. Numerous studies have shown that patency rates for prosthetic bypass grafting to the infrageniculate vessels are clearly inferior to that reported for greater saphenous vein bypass. In this report we summarize our experience with the use of alternate autogenous vein grafting to the infrageniculate vessels. The records of all patients undergoing autogenous bypass grafting to the infrageniculate vessels using a conduit other than the greater saphenous vein between 1992 and 1999 were reviewed. Graft survival curves were plotted using the Kaplan-Meier method and results are reported using the Society for Vascular Surgery/International Society for Cardiovascular Surgery guidelines. Forty-eight patients underwent a total of 51 infrageniculate bypass procedures using non-greater saphenous autogenous conduits. Thirty-nine patients had reconstructions performed with single segments of arm vein, two had their operations performed with lesser saphenous vein, and ten had grafts created with two segments of non-greater saphenous autogenous vein. Twenty-one grafts were performed to the infrageniculate popliteal artery and 30 were performed to the tibial vessels. Primary and primary assisted patency rates at 30 months were 49 and 75 per cent. Limb salvage was 87 per cent. Infrainguinal bypass grafting using non-greater saphenous autogenous conduits can yield quite satisfactory intermediate limb salvage and patency rates. However, close graft surveillance and prompt intervention are required to avoid graft failure.
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Timaran, Carlos H., Scott L. Stevens, Michael B. Freeman, and Mitchell H. Goldman. "Infrainguinal Bypass Grafting Using Lyophilized Saphenous Vein Allografts for Limb Salvage." Cardiovascular Surgery 10, no. 4 (August 2002): 315–19. http://dx.doi.org/10.1177/096721090201000405.

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Critical ischemia in patients with extensive femoropopliteal occlusive disease often ends in amputation in the absence of a suitable autologous vein for reconstruction. Cryopreserved vascular allografts have been used as an alternative conduit with poor results. Antigenicity and rejection are assumed to account for graft failure. Lyophilized vessels have demonstrated patency and structural integrity in the vascular system in our previous experimental studies. We report four patients that underwent femorodistal bypass grafting with lyophilized saphenous veins who lacked usable autologous vein for arterial reconstruction. Early graft thrombosis occurred in three patients who required major amputations. Duplex scans for graft surveillance did not reveal previous significant abnormalities. These cases demonstrate that the clinical use of lyophilized venous allografts for infrainguinal arterial reconstructions failed to yield satisfactory patency and limb salvage. Lyophilized veins therefore are not useful alternative conduits in patients with critical ischemia and no suitable autologous vein grafts.
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Dissertations / Theses on the topic "Saphenous Vein Graft Disease"

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Li, Jun. "Pre-existing intimal hyperplasia and overexpression of TGF-ß1 in saphenous vein grafts before myocardial revascularization in humans: implications for aortocoronary saphenous vein graft disease." Ulm : Univ. Ulm , Med. Fak, 2001. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-8610.

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Ohnaka, Motoaki. "Effect of microRNA-145 to prevent vein graft disease in rabbits by regulation of smooth muscle cell phenotype." Kyoto University, 2014. http://hdl.handle.net/2433/192144.

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The final publication is available at http://dx.doi.org/10.1016/j.jtcvs.2013.11.054. Motoaki Ohnaka, Akira Marui, Kenichi Yamahara, Kenji Minakata, Kazuhiro Yamazaki, Motoyuki Kumagai, Hidetoshi Masumoto, Shiro Tanaka, Tadashi Ikeda, Ryuzo Sakata, Effect of microRNA-145 to prevent vein graft disease in rabbits by regulation of smooth muscle cell phenotype, The Journal of Thoracic and Cardiovascular Surgery, Volume 148, Issue 2, August 2014, Pages 676-682.e2, ISSN 0022-5223.
Kyoto University (京都大学)
0048
新制・課程博士
博士(医学)
甲第18544号
医博第3937号
新制||医||1006(附属図書館)
31444
京都大学大学院医学研究科医学専攻
(主査)教授 木村 剛, 教授 野田 亮, 教授 瀬原 淳子
学位規則第4条第1項該当
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PRANDI, FRANCESCA. "Identification of early pathophysiological events underlying venous coronary bypass stenosis by a mechano-biology approach." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2014. http://hdl.handle.net/10281/50493.

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Saphenous vein graft disease represents an unresolved problem in coronary artery bypass grafting. After surgery, progressive modification in the vein wall occurs, leading to occlusion of the graft lumen. This process, called intima hyperplasia, involves the participation of vein-resident cells as well as the recruitment of vein-extrinsic cells. Arterial wall strain has recently emerged as one of the factor that can contribute to the pathogenesis of the vein graft disease. Therefore, in collaboration with Department of Bioengineering, Politecnico di Milano we developed a culture system for the ex vivo pressure stimulation of vein segments. This new ex vivo vein culture system is able to reproduce the wall strain typical of the arterial circulation. The ex vivo vein culture system (ECVS) adopted in this project has been validated and proved as a valuable, reliable, easy handling and versatile tool for studying arterial pressure events triggered in VGD. The biological data achieved confirm an important contribution of the arterial-like wall strain in SV structural and biochemical changes, activation of vessel resident cells and in the expression of molecular signals involved in the pathogenesis of IH. Using this system, we found that either venous- or arterial-specific pressure regimens induced vein pro-pathologic commitment involving upregulation of Matrix Metallo-Proteases 2/9, and induction of microRNAs-21/146a/221. By contrast, arterial-like pressure caused a significant morphological rearrangement of the vein, a suppression of Tissue Inhibitor of Metallo Protease-1, an enhanced expression of TGF-β1 and BMP-2 mRNAs and, finally, the upregulation of microRNAs-138/200b/200c. In coronary-pressure stimulated vessels, the density of the adventitial vasa vasorum was significantly increased. This was accompanied by an increased presence of cells co-expressing NG2, CD44 and SM22α markers in the adventitia, identifying them as multipotent mesenchymal cells/smooth muscle cells progenitors with a pericyte origin. An increase in Histone H3 Lysine 4 methylation and histone H4 Lysines 9/16 acetylation levels was finally found in adventitial cells and vasa vasorum. The present findings suggest a mechanistic role of the arterial-like pulsatile pressure in reinforcement of SV-resident cells pro-pathologic commitment in vein bypass failure, by activation of mechanical-dependent transcriptional circuitries and of pericyte-derived cells located in the vessel adventitia. The ultimate goal of this project is to find a treatment that can prevent, avoid or reduce the incidence of the vein graft disease in patients subjected to bypass surgery with saphenous vein. This treatment could include one or more targets identified in this work focusing on the early stage of the pathological adaptation of the SV to the new hemodynamic environment.
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Cornelissen, Jacqueline. "Saphenous vein bypass graft occlusion : signalling pathways and apoptosis." Thesis, University of the West of England, Bristol, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431153.

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Crook, Martin. "Mechanisms of monocyte adhesion to human saphenous vein." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324233.

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WADA, KENTARO, TOMOYUKI NODA, KENICHI HATTORI, HIDEKI MAKI, AKIRA KITO, and HIROFUMI OYAMA. "AIR VENT OF VEIN GRAFT IN EXTRACRANIAL-INTRACRANIAL BYPASS SURGERY." Nagoya University School of Medicine, 2012. http://hdl.handle.net/2237/16744.

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Gopakumar, Bhaskaran Nair. "Molecular strategies to inhibit vein graft disease." Thesis, University of Leeds, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426787.

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Dreifaldt, Mats. "Conduits in coronary artery bypass grafting surgery : Saphenous vein, radial and internal thoracic arteries." Doctoral thesis, Örebro universitet, Institutionen för hälsovetenskap och medicin, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-33265.

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A novel technique for saphenous vein (SV) graft harvesting, the No-touch technique (NT), has been developed at the Dept. of Cardiovascular surgery, Örebro University hospital. With NT the SV is harvested with a pedicle of surrounding tissue. This avoids graft spasm and eliminates the need for distension. The surrounding tissue acts as a structural support and is a rich source of vaso-dilating agents. A randomized controlled trial (RCT) has shown a significantly higher patency rate for NT SV grafts compared to SV grafts harvested with conventional technique (CT). This thesis evaluates some of the properties of the surrounding tissue and compares patency rates between NT SV and radial artery (RA) grafts and patency rates for internal thoracic artery (ITA) grafts harvested with and without surrounding tissue. Paper I investigated vasa vasorum (VV) in SV grafts and showed that the NT preserves an intact VV whereas CT does not. This could be one of the mechanisms underlying the improved patency for NT SV grafts. Paper II evaluated VV and associated nitric oxide (NO) in SV and arterial grafts. SV grafts showed a higher number and larger VV, which correlated with NO production, compared to arterial grafts. NT SV grafts showed higher activity for e-NOS compared to CT SV grafts. Paper III is a RCT comparing patency rates between NT SV and RA grafts, three years after surgery, showing a significantly higher patency rate for NT SV grafts. Paper IV is a RCT comparing patency rates for ITA graft harvested with and without surrounding tissue and did not show any difference between graft preparations. In conclusion, the NT for SV graft harvesting preserves an intact vasa vasorum and associated NO production. NT SV grafts show a higher patency rate than RA grafts. Harvesting of ITA with or without surrounding tissue does not affect patency rate.
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Paul, Timir, Samit Bhatheja, Hemang Panchal, Shimin Zheng, Subhash Banerjee, Sunil V. Raso, Luis Guzman, et al. "Outcomes of Saphenous Vein Graft Intervention With and Without Embolic Protection Device: A Comprehensive Review and Meta-Analysis." Digital Commons @ East Tennessee State University, 2017. https://dc.etsu.edu/etsu-works/2771.

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Background: Current guidelines give a class I recommendation to use of embolic protection devices (EPD) for saphenous vein graft (SVG) intervention; however, studies have shown conflicting results. The objective of this meta-analysis is to compare all-cause mortality, major adverse cardiovascular events, myocardial infarction (MI), or target vessel revascularization in SVG intervention with and without EPD. Methods and Results: Literature was searched through October 2016. Eight studies (n=52 893) comparing SVG intervention performed with EPD (n=11 506) and without EPD (n=41 387) were included. There was no significant difference in all-cause mortality (odds ratio [OR], 0.79; confidence interval [CI], 0.55–1.12; P=0.19), major adverse cardiovascular events (OR, 0.73, CI, 0.51–1.05; P=0.09), target vessel revascularization (OR, 1.0; CI, 0.95–1.05; P=0.94), periprocedural MI (OR, 1.12; CI, 0.65–1.90, P=0.69), and late MI (OR, 0.80; CI, 0.52–1.23; P=0.30) between the 2 groups. Sensitivity analysis excluding CathPCI Registry study showed no difference in periprocedural MI, late MI, and target vessel revascularization; however, it favored EPD use in all-cause mortality and major adverse cardiovascular events. Further sensitivity analysis including only observational studies revealed no difference in all-cause mortality, major adverse cardiovascular events, target vessel revascularization, and late MI. Additional analysis after excluding CathPCI Registry study revealed no difference in outcomes. Conclusions: This study including 52 893 patients suggests no apparent benefit in routine use of EPD during SVG intervention in the contemporary real-world practice. Further randomized clinical trials are needed in current era to evaluate long-term outcomes in routine use of EPD, and meanwhile, current guideline recommendations on EPD use should be revisited.
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Kauhanen, Petteri. "Vascular regulation of hemostasis and fibrinolysis : with special focus on vein graft disease." Helsinki : University of Helsinki, 2001. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/kauhanen/.

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Books on the topic "Saphenous Vein Graft Disease"

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R, Bates Eric, and Holmes David R. 1945-, eds. Saphenous vein bypass graft disease. New York: M. Dekker, 1998.

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Zamboni, Paolo, Erika Mendoza, and Sergio Gianesini, eds. Saphenous Vein-Sparing Strategies in Chronic Venous Disease. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-70638-2.

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Spontaneous and induced intima formation in blood vessels. Austin: R.G. Landes, 1995.

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Ceceña-Seldner, Felipe A. Aortocoronary saphenous vein bypass graft disease. Physicians & Scientists Pub. Co, 2000.

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Mendoza, Erika, Paolo Zamboni, and Sergio Gianesini. Saphenous Vein-Sparing Strategies in Chronic Venous Disease. Springer, 2018.

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Saphenous Vein-Sparing Strategies in Chronic Venous Disease. Springer, 2018.

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Saphenous Vein Graft Lesions and Thrombectomy for Acute Myocardial Infarction, An Issue of Interventional Cardiology Clinics. Elsevier, 2013.

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Kockx, Mark M. Spontaneous and Induced Intima Formation in Blood Vessels. Springer London, Limited, 2013.

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Gu, Wenduo, Yao Xie, and Qingbo Xu. Animal models to study pathophysiology of the vasculature. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0005.

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Animal models are designed to be preliminary tools for a better understanding of the pathogenesis, improvement in diagnosis, prevention, and therapy of vascular diseases in humans. Animal models are easily manageable, as compounding effects of dietary and environmental factors can be controlled experimentally. Blood vessel samples can be taken for detailed experimental and biomolecular examination. A thorough understanding of the animal models used is necessary and complete analysis must be validated so that the data can be extrapolated to humans. There are several species that are used for studying vascular pathophysiology, including mice, rats, rabbits, and pigs. Attracted by the well-defined genetic systems, a number of investigators have begun to use the mouse as an experimental system for arteriosclerosis research. Because vascular disorder is a complicated disease, which includes spontaneous (native) atherosclerosis, transplant arteriosclerosis, vein graft atherosclerosis, and angioplasty-induced restenosis, several models for studying all types of vascular disease have recently been established. Using these animal models, much knowledge concerning the pathogenesis of the disease and therapeutic intervention has been gained. This chapter will not attempt to cover all aspects of animal models, but will rather focus on the major progress in understanding the pathophysiology of the vasculature, the (dis)advantages of a variety of models, and how specific models can be appropriately chosen for different purposes of study.
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Book chapters on the topic "Saphenous Vein Graft Disease"

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Friedewald, Vincent E. "Aortocoronary Saphenous Vein Graft Aneurysm." In Clinical Guide to Cardiovascular Disease, 185–90. London: Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-7293-2_12.

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Grondin, C. M., and J. C. Thornton. "The Natural History of Saphenous Vein Grafts." In Coronary Artery Graft Disease, 3–15. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78637-2_1.

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Angelini, G. D., and A. C. Newby. "Smooth Muscle Cell Proliferation Responses in Organ Cultures of Human Saphenous Vein." In Coronary Artery Graft Disease, 212–27. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78637-2_13.

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Karasu, C., K. Ayrancioglu, H. Soncul, G. Ozansoy, and V. Melih Altan. "Diabetes Induced Vascular Changes in Human Internal Mammary Artery and Saphenous Vein Grafts." In Angiogenesis in Health and Disease, 383. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3358-0_48.

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Cardon, J. M., A. Joyeux, D. Noblet, M. M. Faye, and P. Bousquet. "Homolateral Long Saphenous Vein as a Valvulated Graft for Reflux Treatment of Deep Venous Postthrombotic Disease." In Modern Vascular Surgery, 515–22. New York, NY: Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4612-2632-1_54.

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Sarwar, Bilal, and Sorin J. Brener. "Saphenous Vein Graft Interventions." In PanVascular Medicine, 2271–80. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-37078-6_76.

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Sarwar, Bilal, and Sorin J. Brener. "Saphenous Vein Graft Interventions." In PanVascular Medicine, 1–12. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-37393-0_76-1.

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Komiyama, Nobuyuki. "Angioscopy of Saphenous Vein Graft." In Coronary Angioscopy, 117–29. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55546-9_11.

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Stauber, Bradley D., Reginald I. Low, Jeffrey A. Southard, and Gagan D. Singh. "Complex Case: Saphenous Vein Graft." In Clinical Cases in Interventional Cardiology, 73–79. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-60490-9_12.

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Cantrell, Matthew, Edward Fry, and James Hermiller. "Percutaneous Saphenous Vein Graft and Arterial Graft Intervention." In Catheter-Based Cardiovascular Interventions, 575–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-27676-7_32.

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Conference papers on the topic "Saphenous Vein Graft Disease"

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MacLennan, M. J., B. J. Leavitt, J. D. Schmoker, and N. C. Chesler. "Pressure Increases Inert Particle Uptake in Human Saphenous Vein." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2234.

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Abstract Cardiovascular disease is one of the leading causes of death in the United States, and coronary artery bypass graft surgery (CABG) is one of the mainstays of treatment for this disease [1]. Since artificial vascular grafts suitable for coronary bypass are not yet available, autologous internal mammary artery (IMA) and saphenous vein are used to bypass diseased tissue [1]. While IMA grafts have high long-term patency rates, saphenous vein grafted into the arterial position tends to stenose and eventually thrombose.
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MacLennan, M. J., B. J. Leavitt, J. D. Schmoker, and N. C. Chesler. "Pressure Increases Inert Particle Uptake in Human Saphenous Vein." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2572.

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Abstract Cardiovascular disease is one of the leading causes of death in the United States, and coronary artery bypass graft surgery (CABG) is one of the mainstays of treatment for this disease (Niklason et al., 1999). Since artificial vascular grafts suitable for coronary bypass are not yet available, the autologous internal mammary artery (IMA) and saphenous vein are used to bypass diseased tissue (Niklason et al., 1999). While IMA grafts have high long-term patency rates, a saphenous vein grafted into the arterial position tends to stenose and eventually thrombose.
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Ladak, Shameem, Liam McQueen, Lathishia JoelDavid, Gavin Murphy, and Mustafa Zakkar. "BS36 Acute arterial haemodynamics activation of endothelial to mesenchymal transition in long saphenous veins. Impact on vein graft disease." In British Cardiovascular Society Annual Conference, ‘100 years of Cardiology’, 6–8 June 2022. BMJ Publishing Group Ltd and British Cardiovascular Society, 2022. http://dx.doi.org/10.1136/heartjnl-2022-bcs.216.

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Ladak, Shameem, Liam McQueen, Lathishia JoelDavid, Gavin Murphy, and Mustafa Zakkar. "BS36 Acute arterial haemodynamics activation of endothelial to mesenchymal transition in long saphenous veins. Impact on vein graft disease." In British Cardiovascular Society Annual Conference, ‘100 years of Cardiology’, 6–8 June 2022. BMJ Publishing Group Ltd and British Cardiovascular Society, 2022. http://dx.doi.org/10.1136/heartjnl-2022-bcs.216.

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Pedroso, Pedro D., Andreas S. Anayiotos, Brad L. Hershey, Evangelos Eleftheriou, and William L. Holman. "Preliminary Near Wall Hemodynamic Evaluation of a Coronary Artery Bypass Graft Model With a Flow Streamlining Implant." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32566.

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Coronary artery disease (CAD) is the leading cause of death in the world today. According to the American Heart Association 529,659 people in 1999 died as a result of CAD [1]. Starting in the 1960’s, surgeons have used Coronary Artery Bypass Graft (CABG) techniques in order to reestablish blood flow to the heart. Today, the procedure remains the same, using autologous grafts, such as the mammary artery and the saphenous vein. An unresolved problem, is that a significant number of CABGs reocclude months to years postoperatively. In the case of Saphenous Vein Grafts (SVGs) typically 50% of these bypasses are totally occluded months to years after the procedure, the remaining half being more than 50% occluded [2]. The re-occlusion of CABGs is due to a process labeled intimal hyperplasia (IH). Investigators have shown that IH, believed by some to be a remodeling process, occurs at branch sites, regions of curvature, and anastomotic junctions [3,4]. At these sites there are low residence times, slow secondary structures, disturbed flow, and areas of recirculation, therefore the onset of IH is believed to be hemodynamically linked. Most recently, floor IH has been attributed to four variables: time averaged wall shear stress (WSS), oscillating shear index (OSI), spatial wall shear stress gradients (WSSG), and temporal WSSG [5]. Adverse values of these parameters, in the case of SVGs, are believed to be caused by impedance mismatch at the anastomosis site. Over time this characteristic causes a bulge at the sinus. Such a morphology additionally contributes to disturbed flows which tend to propagate down the CABG and are believed to play a major role in the development of IH and the eventual failure of the graft.
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Anayiotos, Andreas S., Pedro D. Pedroso, Ramakrishna Venugopalan, Evangelos Eleftheriou, and Maria A. Advincula. "Flow Evaluation of a Compliant Coronary Artery Anastomosis Model." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/bed-23123.

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Abstract Coronary artery bypass grafting (CABG) is a common surgical procedure for coronary artery diseases or severe stenosis of the coronary arteries. It makes use of a peripheral vein (usually the saphenous vein of the leg) to form a conduit between the aorta and the coronary artery distal to the obstructive lesion. It is rapidly becoming the treatment of choice in cases where the vessel is more than 70% occluded or when angioplasty is not possible. However, a significant number of grafts subsequently fail due to acute thrombosis in the early post-operative period or to restenosis within months or years. Intimal thickening in the CABG anastomosis has been implicated as the major cause of restenosis and long-term graft failure. Several studies point to the interplay between non-uniform hemodynamics (including disturbed flows and recirculation zones), wall shear stress, and long particle residence time as possible etiologies. An important feature of the anastomosis geometry, is a bulge that forms at the veno-arterial junction. This sinus forms as a result of the stretching of the thin venous wall when the graft is exposed to aortic pressure conditions. The resultant sinus, and the impedance mismatch of the vein-artery connection, contribute to a complicated region of highly disturbed flow at the divider and may have a primary role in restenosis and final failure of the graft.
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Madhavan, Krishna, Walter Bonani, Craig Lanning, and Wei Tan. "Development and Biomechanical Characterization of a Novel Bilayer Vascular Graft." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19613.

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Vascular grafts are currently used to treat cardiovascular diseases such as arthrosclerosis by bypass surgery and as vascular access in hemodialysis [1]. There are a number of types of grafts including autologous vessels (such saphenous vein), synthetic grafts (such as expanded polytetrafluoroethylene) and tissue engineered blood vessels. Currently synthetic grafts are most commonly used as blood vessel replacements and there are a number of problems associated with them. One main impediment is that these grafts are not suitable for small-diameter (less than 6mm) vessel replacement [1, 2], due to high occlusion rates. The major concern over the other alternatives such as autologous vessels and tissue engineered products is their availability. Thus, new approaches to constructing biomimetic small-diameter blood vessel equivalents, that are immediately available, may address the unmet demand in this area. Therefore, we have designed a novel bilayer vascular construct which is made up of a nanofibrous intimal-equivalent as thromboresistant vessel lumen and a mimetic extracellular matrix (ECM) as medial-equivalent for smooth muscle cells (SMC) from native artery to invade and remodel the ECM.
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Pullens, Rolf A. A., Maria Stekelenburg, Carlijn V. C. Bouten, Frank P. T. Baaijens, and Mark J. Post. "3D Coculture of Human Endothelial Cells and Myofibroblasts for Vascular Tissue Engineering." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176099.

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Cardiovascular disease is still the number one cause of death in the industrialized world. Diseased small diameter blood vessels are frequently replaced by native grafts. However, these vessels have a limited life time [1], for example the patency at 10 year after coronary artery bypass grafting of saphenous vein grafts is 57% [2]. Tissue engineering (TE) of small diameter blood vessels seems a promising approach to overcome these shortcomings or address the increasing need for substitutes during follow up surgery. Mechanical conditioning of myofibroblast (MFs) seeded constructs appears to be beneficial for functional tissue properties, such as cell proliferation, ECM production and mechanical strength [3,4]. Without a functional endothelial cell (ECs) layer however, patency may be compromised by thrombogenecity. Construction of an EC layer might on the other hand affect the tissue composition during culture, as was shown for bovine ECs, which influenced proliferation and ECM production of smooth muscle cells [5].
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Shahriman, A. B., M. N. Rahman Y., Siti Khadijah Za'Ba, Khairunizam Wan, Abdul Halim Ismail, A. H. Khairul, and SA Roohi. "Hemodynamic study on upper extremity: Simulation on straight reverse saphenous vein graft." In 2012 International Conference on Biomedical Engineering (ICoBE). IEEE, 2012. http://dx.doi.org/10.1109/icobe.2012.6179006.

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Franz, Thomas, Helena van der Merwe, Peter Zilla, Deon Bezuidenhout, and B. Daya Reddy. "Tubular Nitinol Knit Meshes for External Reinforcement of Saphenous Vein Grafts: A Numerical Design Study." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-68624.

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The difference in mechanical properties between grafts and host arteries is a complicating factor for vascular bypass surgery and can cause patho-physiological problems after implantation [1–7]. Diffuse and focal intimal hyperplasia, one of the key factors of vein graft failure, has been attributed to over-distension and diametric irregularities of the veins when exposed to the arterial circulation [8]. The external reinforcement of saphenous vein grafts with open-mesh knitted Nitinol structures is suggested to prevent over-distension, smooth the luminal diameter, and address the mismatch in mechanical properties of vein graft and host vessel. The objectives of this work were: 1) development of Finite Element (FE) models of knitted Nitinol structures to assess mechanical behaviour and structural properties, e.g. vascular compliance, and 2) proof of feasibility of the FE method developed for structural design optimisation of the Nitinol mesh.
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