Academic literature on the topic 'Sanger'

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Journal articles on the topic "Sanger"

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Lagnado, John. "Fred Sanger." Biochemist 27, no. 6 (December 1, 2005): 37–39. http://dx.doi.org/10.1042/bio02706037.

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Yasunari, Kristie. "Margaret Sanger." Peace Review 12, no. 4 (December 2000): 619–26. http://dx.doi.org/10.1080/10402650020014735.

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Tippett, P. "Ruth Sanger." Vox Sanguinis 81, no. 4 (November 2001): 211–12. http://dx.doi.org/10.1046/j.1423-0410.2001.00121.x.

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Watts, Geoff. "Frederick Sanger." Lancet 382, no. 9908 (December 2013): 1872. http://dx.doi.org/10.1016/s0140-6736(13)62614-8.

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Estrada-Rivadeneyra, Diego. "Sanger sequencing." FEBS Journal 284, no. 24 (November 24, 2017): 4174. http://dx.doi.org/10.1111/febs.14319.

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Mollison, Patrick L. "Ruth Sanger." Transfusion 42, no. 1 (January 2002): 125–26. http://dx.doi.org/10.1046/j.1537-2995.2002.00054.x.

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Tack, Jan. "Response to Sanger." American Journal of Gastroenterology 109, no. 3 (March 2014): 445–46. http://dx.doi.org/10.1038/ajg.2014.17.

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Davies, Martin J. "Celera vs Sanger." Trends in Biotechnology 19, no. 3 (March 2001): 88. http://dx.doi.org/10.1016/s0167-7799(01)01588-8.

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Dodson, Guy. "Fred Sanger: sequencing pioneer." Biochemist 27, no. 6 (December 1, 2005): 31–35. http://dx.doi.org/10.1042/bio02706031.

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Fred Sanger is a biochemist who practised chemistry and whose discoveries have been fundamental to the development of modern biological science. His insights into sequencing methods and the central roles played by proteins and nucleic acids in the molecular processes of biology led him to dedicate a lifetime to their investigation. Together with his shrewd chemical judgement, his experimental skill and his tenacity, this made him one of the most productive and remarkable biochemists of our time.
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Brenner, S. "Frederick Sanger (1918-2013)." Science 343, no. 6168 (January 16, 2014): 262. http://dx.doi.org/10.1126/science.1249912.

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Dissertations / Theses on the topic "Sanger"

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Eberts, Carolyn. "The Sanger Brand: The Relationship of Margaret Sanger and the Pre-War Japanese Birth Control Movement." Bowling Green, Ohio : Bowling Green State University, 2010. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=bgsu1268832853.

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Collazo, Verena. "Reverse Sanger-Sequenzierung mittels MALDI-TOF-Massenspektrometrie." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=964226871.

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Thornley, David John. "Analysis of trace data from fluorescence based Sanger sequencing." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286265.

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Takara, Alexandre Hideaki. "Padronização da genotipagem da variante G202A da G6PD A- análise comparativa da relação custo-benefício entre TETRA-ARMS e sequenciamento Sanger /." Botucatu, 2018. http://hdl.handle.net/11449/165615.

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Orientador: Paulo Eduardo Martins Ribolla
Resumo: A deficiência da enzima Glicose-6-Fosfato Desidrogenase (G6PD) é uma anormalidade genética de alta prevalência populacional que resulta em uma menor reatividade do sistema de óxido-redução eritrocitário, geralmente sem repercussões clínicas; estima-se que mais de 300 milhões de pessoas são portadoras dessa alteração. A enzima é expressa em todos os tecidos e catalisa a primeira etapa da Via das Pentoses. Nas hemácias, essa via é de fundamental importância na manutenção do equilíbrio de seu estado redox e a deficiência dessa enzima pode favorecer eventos hemolíticos agudos e crônicos; e em recém nascidos, pode contribuir para o agravamento da icterícia neonatal. O diagnóstico da deficiência baseia-se na atividade enzimática, identificada através de testes quantitativos e qualitativos. Os testes qualitativos limitam-se a agrupar indivíduos em “deficientes” e “não deficientes”, já os métodos quantitativos são mais precisos na inferência dessa atividade. Estas técnicas podem necessitar de repetições dos testes para confirmação de resultados incongruentes. Por outro lado, a variante genética responsável pela deficiência pode ser precisamente reconhecida através de testes de diagnóstico molecular. O presente projeto tem como objetivo desenvolver uma metodologia de identificação molecular da variante G202A, frequentemente encontrada na população brasileira, e realizar uma comparação do custo-benefício com a metodologia de sequenciamento de Sanger. Ao todo, foram analisadas 107 amost... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Glucose-6-phosphate dehydrogenase deficiency is a metabolic enzymatic defect affecting 300 million people worldwide. The enzyme is present in all tissues and catalyses the first reaction in the Pentose Phosphate pathway responsible for maintaining the redox equilibrium in red blood cell. Deficient enzyme may lead to acute and chronic haemolytic anaemia and neonatal jaundice. Diagnosis for G6PD deficiency is based on biochemical quantitative or qualitative tests. Qualitative tests only classifies subjects as “deficient” or “non-deficient”, while quantitative tests are more precise, however both biochemical approches need a confirmative assay to confirm ambiguous results. On the other hand molecular identification for the molecular variants are more accurate and precise. We developed a new molecular assay to identify the G202A molecular variant present at high frequency on Brazilian population and comparer it to Sanger sequencing. One hundred and seven peripheral blood sample were collected on filter paper. DNA extraction were performed followed by G6PD exon 4 amplification and sequencing. On-line tool “Primer1” generated allele-specific primers for TETRA-ARMS genotyping. Twenty two subjects were deficient homozygote, eighty four wild homozygote and one heterozygote. All subjects genotype were confirmed by Sanger sequencing. TETRA-ARMS costs per reaction is three times lower than Sanger sequencing. We conclude that TETRA-ARMS is a suitable protocol to detect G202A mutation on h... (Complete abstract click electronic access below)
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O'Brien, Joseph Morton. "Transonic Compressor Test Rig rebuild and initial results with the Sanger stage." Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 2000. http://handle.dtic.mil/100.2/ADA381019.

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Van, Der Westhuizen Barend Nicolaas. "Aviva Pelham : die sanger as crossover-kunstenaar - 'n ondersoek na die verskynsel." Thesis, Stellenbosch : Stellenbosch University, 2013. http://hdl.handle.net/10019.1/85875.

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Thesis (MMus)-- Stellenbosch University, 2013.
ENGLISH ABSTRACT: It is ironic that with so much interest in singing as a career, the possiblities for trained singers are getting less and less, especially in South Africa. With reality programmes such as Idols and Superstars, the popular music world is overflowed with singers with little, if any, voice training. Then there are also singers who try to establish themselves as crossover artists and perform opera arias on stages such as the KKNK Classic Pops and Die Burger’s Afri-Klassiek concerts accompanied by South Africa’s foremost symphony and philharmonic orchestras. But what about the classical singer who cross over into other genres of singing and like some local and international artists have proved to us, musicals could be a genre to explore. This study explore the possiblity of this type of crossover from classical singing to musical singing. By listening to sound recordings and using a series of musical volumes, songs were identified that the classical singer could sing without harming his or her voice technique. One of the success stories of this type of crossover is the Capetonian soprano, Aviva Pelham. In 1972 Pelham established herself as an opera singer in Cape Town and then confused the classical world with her appearance as Eliza Doolittle in 1978. After that she had a successful career on both classical and musical stages. During her career of nearly 40 years as performing artist, Pelham appeared in 28 different opera roles, 11 different operetta roles and 10 musical roles. Aviva Pelham’s achievements were laureated with a Cape Nederburg Opera Prize, four FNB Vita Awards and in 2000 an Arts, Culture and Heritage Award from the Western Cape Minister for Environment and Culture
AFRIKAANSE OPSOMMING: Dit is ironies dat, terwyl daar soveel belangstelling in sang as loopbaan heers, word die geleenthede vir opgeleide sangers meer beperk. In Suid-Afrika veral is die moontlikhede beperk vir klassieke sangers. Met realiteitsprogramme soos Idols en Supersterre is die populêre musiekwêreld vol van sangers met geen of baie min sangopleiding. Daar is dan ook van dié sangers wat probeer om hulself te vestig as crossover-kunstenaars en opera-arias uit te voer op die konsertverhoë, soos bv. die KKNK Classic Pops- en Die Burger se Afri-Klassiek-konserte saam met van Suid-Afrika se voorste simfonie- of filharmoniese orkeste. Maar wat van die die klassieke sanger wat “crossover” in ‘n ander tipe styl van sang en soos deur ‘n paar plaaslike en internasionale kunstenaars al bewys, is musiekblyspele ‘n genre om te ondersoek. Hierdie studie ondersoek dus die moontlikheid van hierdie tipe “crossover” van klassieke sang na musiekblyspele. Daar word deur middel van klankopnames en ‘n reeks volumes van musiekblyspel-liedere, liedere geklassifiseer wat die klassieke sanger kan sing sonder om sogenaamde skade aan sy of haar sangtegniek te doen. Een van die sukses-stories van hierdie tipe “crossover” is die Kaapse sopraan, Aviva Pelham. Pelham het haarself as ‘n operasangeres gevestig in 1972 in Kaapstad en die klassieke wêreld ophol gejaag met haar verskyning as Eliza Doolittle in 1978. Vandaar het sy ‘n suksesvolle loopbaan op beide die klassieke en musiekblyspelverhoë gehad. Pelham het gedurende haar loopbaan, van ongeveer 40 jaar as uitvoerende kunstenaar, 28 verskillende operarolle, 11 verskillende operette-rolle en 10 musiekblyspelrolle vertolk. Aviva Pelham se prestasies is bekroon met ‘n Kaapse Nederburg Operaprys, vier FNB Vita-toekennings en sy is in 2000 vereer deur die Wes-Kaapse Minister vir Omgewings- en Kultuursake met ‘n Kunste, Kultuur en Erfenis-toekenning vir haar bydrae tot die Uitvoerende Kunste.
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Thomas, Julie L. "International intercourse establishing a transnational discourse on birth control in the interwar era (Margaret Sanger) /." [Bloomington, Ind.] : Indiana University, 2004. http://wwwlib.umi.com/dissertations/fullcit/3162269.

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Thesis (Ph.D.)--Indiana University, Dept. of History, 2004.
Source: Dissertation Abstracts International, Volume: 66-01, Section: A, page: 0379. Chair: Judith A. Allen. Title from dissertation home page (viewed Oct. 12, 2006).
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Gardiner, A. R. "A genetic investigation of the muscle and neuronal channelopathies : from Sanger to next-generation sequencing." Thesis, University College London (University of London), 2016. http://discovery.ucl.ac.uk/1477237/.

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The neurological channelopathies are a group of hereditary, episodic and frequently debilitating diseases often caused by dysfunction of voltage-gated ion channels. This thesis reports genetic studies of carefully clinically characterised patient cohorts with different episodic neurological and neuromuscular disorders including paroxysmal dyskinesias, episodic ataxia, periodic paralysis and episodic rhabdomyolysis. Genetic and clinical heterogeneity has in the past, using traditional Sanger sequencing methods, made genetic diagnosis difficult and time consuming. This has led to many patients and families being undiagnosed. Here, different sequencing technologies were employed to define the genetic architecture in the paroxysmal disorders. Initially, Sanger sequencing was employed to screen the three known paroxysmal dyskinesia genes in a large cohort of paroxysmal movement disorder patients and smaller mixed episodic phenotype cohort. A genetic diagnosis was achieved in 39% and 13% of the cohorts respectively, and the genetic and phenotypic overlap was highlighted. Subsequently, next-generation sequencing panels were developed, for the first time in our laboratory. Small custom-designed amplicon-based panels were used for the skeletal muscle and neuronal channelopathies. They offered considerable clinical and practical benefit over traditional Sanger sequencing and revealed further phenotypic overlap, however there were still problems to overcome with incomplete coverage. Large custom and non-custom pull-down panels were used to investigate patients with recurrent rhabdomyolysis patients. The contribution of genetic abnormalities was determined, and it was concluded that while the contribution of the RYR1 was substantial, it was minimal for the classic voltage-gated ion channels SCN4A and CACNA1S Lastly, whole-exome sequencing was applied to two large undiagnosed possible channelopathy families. One family was found to indeed harbour a channelopathy mutation, whilst the other did not. Overall, next-generation sequencing proved to be a more thorough and efficient method for channelopathy genetic diagnosis and several novel findings throughout the thesis expanded the current knowledge within the field.
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Karkar, Adnane. "Leucodystrophies : aspects génétiques et moléculaires au Maroc." Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCC258.

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Les leucodystrophies (LD) sont des troubles héréditaires affectant la substance blanche (SB) du système nerveux central (SNC) avec ou sans atteinte du système nerveux périphérique (SNP). Ces troubles ont en commun des anomalies de la cellule gliale ou de la gaine de la myéline. L’imagerie par résonance magnétique (IRM) représente l’outil majeur pour la détection des anomalies de la SB, ainsi l’IRM accompagnée d’un examen clinique permettent d’orienter le diagnostic étiologique. La confirmation de ce diagnostic est principalement basée sur la biologie moléculaire qui permet la détermination du gène muté. Sachant qu’au Maroc aucune étude sur les leucodystrophies n’avait été réalisée auparavant, nous avons mené une étude prospective afin de déterminer le profil des patients marocains atteints de ces troubles et de mettre en place un diagnostic moléculaire des formes les plus fréquentes. Pour cela nous avons rassemblé des échantillons de familles dont un ou plusieurs membres avaient une suspicion de leucodystrophie. Par la suite nous avons réalisé un séquençage des gènes connu pour leur implication dans les leucodystrophies. Le séquençage s’est déroulé en deux phases. La première, par séquençage Sanger, a concerné les patients présentant un marqueur biochimique positif. La deuxième phase, par séquençage nouvelle génération ou Next-Generation Sequencing (NGS) a concerné les patients sans marqueurs biochimiques apparent. Cette approche nous a permis d’identifier les types de leucodystrophies présentes au sein de la population marocaine, mais aussi de mettre en évidence de nouvelles mutations
Leukodystrophies are hereditary disorders affecting central nervous system white matter (WM) with or without damages in peripheral nervous system. These disorders have in common abnormalities of the glial cell and the sheath of the myelin. Magnetic resonance imaging (MRI) is the major tool to detect WM abnormalities, so MRI in association with a good clinical examination can help to provide an accurate medical diagnosis. Moreover, the confirmation of this or that leukodystrophy remains in the field of molecular biology by the detection of mutated gene translating the phenotype of the patient. Knowing that in Morocco no previous study on leukodystrophies had been carried out, we sought to know the characteristics of Moroccan patients carrying these disorders so as to be able to establish a molecular diagnosis of the most frequent forms. We collected samples from families with one or more members had a suspicion of leukodystrophy. Thus, we carried out a sequencing with an approach that consists in analyzing directly by Sanger method the leukodystrophies having a positive biochemical marker and by next generation sequencing or Next-Generation Sequencing (NGS) leukodystrophies without known biochemical markers. This approach allowed us, to identify leukodystrophies in a sample of the Moroccan population but also to identify new mutations
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Lenhardt, Nina-Veronika Ingeborg [Verfasser], and Bermhard [Akademischer Betreuer] Weber. "Mutationsanalyse in ABCA4-assoziierten Netzhautdystrophien mittels der Didesoxymethode nach Sanger / Nina-Veronika Ingeborg Lenhardt. Betreuer: Bermhard Weber." Regensburg : Universitätsbibliothek Regensburg, 2015. http://d-nb.info/1071713450/34.

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Books on the topic "Sanger"

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Haugland, Scott. Sanger. Charleston, South Carolina: Arcadia Publishing, 2013.

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Christensen, Lars Saabye. Sanger & steiner. [Oslo]: Cappelen, 2002.

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Jeffers, Joe S. Frederick Sanger. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54709-1.

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Christensen, Lars Saabye. Sanger & steiner. [Oslo]: Cappelen, 2003.

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Bounds, Helen Seely. Around Sanger. Charleston, S.C: Arcadia Pub., 2011.

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Helle, Lars. Sanger til Gry. Stavanger: Wigestrand, 2009.

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Wolf, Øystein Wingaard. Kaptein Langneses sanger. Oslo: Aschehoug, 1999.

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Geerman, Pancho. Flor di sanger. Aruba: Charuba, 1987.

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Esther, Katz, Hajo Cathy Moran, Engelman Peter, and University Publications of America (Firm), eds. The Margaret Sanger papers. Bethesda, MD: University Publications of America, 1996.

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Wolf, Øystein Wingaard. Slangedans: Dikt og sanger. Oslo: Aschehoug, 1986.

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Book chapters on the topic "Sanger"

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Hampton, Polly. "Sanger, Margaret." In Encyclopedia of Women’s Health, 1159–61. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-0-306-48113-0_387.

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Gressner, A. M., and O. A. Gressner. "Sanger, Frederick." In Springer Reference Medizin, 2094–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_3426.

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Arnemann, J. "Sanger-Sequenzierung." In Springer Reference Medizin, 2095–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_3576.

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Gressner, A. M., and O. A. Gressner. "Sanger, Frederick." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_3426-1.

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Arnemann, J. "Sanger-Sequenzierung." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_3576-1.

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Jennings, W. Bryan. "Sanger Sequencing." In Phylogenomic Data Acquisition, 105–30. Boca Raton: Taylor & Francis, 2017.: CRC Press, 2016. http://dx.doi.org/10.1201/9781315181431-6.

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Bettendorf, Gerhard. "Sanger, Margaret." In Zur Geschichte der Endokrinologie und Reproduktionsmedizin, 474–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79152-9_195.

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Keynes, John Maynard. "C. P. Sanger." In Essays in Biography, 324–25. London: Palgrave Macmillan UK, 2010. http://dx.doi.org/10.1007/978-1-349-59074-2_25.

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Whitaker, J. K. "Sanger, Charles Percy (1871–1930)." In The New Palgrave Dictionary of Economics, 1–2. London: Palgrave Macmillan UK, 1987. http://dx.doi.org/10.1057/978-1-349-95121-5_1916-1.

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Whitaker, J. K. "Sanger, Charles Percy (1871–1930)." In The New Palgrave Dictionary of Economics, 11911–12. London: Palgrave Macmillan UK, 2018. http://dx.doi.org/10.1057/978-1-349-95189-5_1916.

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Conference papers on the topic "Sanger"

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Kun Sun, Yuet-Ping Yuen, Huating Wang, and Hao Sun. "The Online Diagnosis System for sanger sequencing based genetic testing." In 2014 International Conference on Big Data and Smart Computing (BIGCOMP). IEEE, 2014. http://dx.doi.org/10.1109/bigcomp.2014.6741404.

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Hasan, Mohammed A., and Jawad A. K. Hasan. "Sanger’s type dynamical systems for canonical variate analysis." In 2008 American Control Conference (ACC '08). IEEE, 2008. http://dx.doi.org/10.1109/acc.2008.4587133.

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Lu, Liqiang, Yicheng Jin, Hangrui Bi, Zizhang Luo, Peng Li, Tao Wang, and Yun Liang. "Sanger: A Co-Design Framework for Enabling Sparse Attention using Reconfigurable Architecture." In MICRO '21: 54th Annual IEEE/ACM International Symposium on Microarchitecture. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3466752.3480125.

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Doman, Thompson N. "Abstract 5109: Identical cancer cell lines from Sanger & Broad CCLE programs sometimes differ." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5109.

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Septiyanto, Abdullah Faqih, Yuni Fitri Cahyaningsih Cahyono, Riyanarto Sarno, Fadlilatul Taufany, Siti Halimah Larekeng, Kelly Rossa Sungkono, Karlina Syahruddin, Endang Gati Lestari, and Sholiq. "An Improved Method for Prioritizing Polymerase Chain Reaction (PCR) Primer Design in Sanger Sequencing." In 2022 6th International Conference on Information Technology, Information Systems and Electrical Engineering (ICITISEE). IEEE, 2022. http://dx.doi.org/10.1109/icitisee57756.2022.10057821.

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Skevington, Jeffrey H. "Unravelling the phylogeny of the lower Cyclorrhapha using morphology, Sanger sequencing, and anchored phylogenetic data." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94498.

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Allice, T., F. Cerutti, MG Milia, G. Gregori, E. Burdino, S. Monteleone, A. Bottoni, B. Simoncelli, E. Scuccimarra, and V. Ghisetti. "P-219 Success rate of HIV DNA Sanger sequencing in PBMC and whole blood samples." In Abstracts from the 16° Italian Conference on AIDS and Antiviral Research. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/sextrans-icar-2024.271.

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Chanyshev, M. D., N. V. Vlasenko, I. A. Kotov, K. F. Khafizov, and V. G. Akimkin. "HIGH THROUGHPUT DNA SEQUENCING OF HEPATITIS B VIRUS." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-266.

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It was shown that Hepatitis B Virus (HBV) genotype and individual mutations influence the course of the disease. There is a need for a simple and reliable method for sequencing the entire genome of hepatitis B virus. We have developed an NGS amplification panel for hepatitis B virus genome sequencing. The panel was validated using Sanger sequencing. More than 300 HBV samples were sequenced and genotypes and mutations described in the literature were identified.
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Eryilmaz, Isil Ezgi, Gamze Guney Eskiler, Gulsah Cecener, Unal Egeli, and Berrin Tunca. "The matter of clinical sequencing for familial breast cancer: The route from Sanger to next generation." In 2017 E-Health and Bioengineering Conference (EHB). IEEE, 2017. http://dx.doi.org/10.1109/ehb.2017.7995391.

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Beaver, C., F. Behan, R. Ansari, R. Pooley, P. Wilkinson, D. Jackson, S. Harper, F. Iorio, K. Yusa, and M. Garnett. "PO-327 Whole-genome CRISPR-CAS9 screening: the wellcome sanger institute cellular generation and phenotyping pipeline." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.357.

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Reports on the topic "Sanger"

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Hersey, Anne. ChEMBL Deposited Data Set - Sanger Cancer Screen. EMBL-EBI, September 2013. http://dx.doi.org/10.6019/chembl1201861.

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García-Sancho, Miguel. Frederick Sanger: el hombre que convirtió los genes en secuencias. Sociedad Española de Bioquímica y Biología Molecular (SEBBM), March 2014. http://dx.doi.org/10.18567/sebbmdiv_rpc.2014.03.1.

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M., Tadjuidje, Dipapoundji B., and Mowawa S.B. Le Paysage Tri-National de la Sangha: Tri-National de la Sangha Landscape. Center for International Forestry Research (CIFOR), 2012. http://dx.doi.org/10.17528/cifor/003932.

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M., Tadjuidje, Dipapoundji B., and Mowawa S.B. Tri-National de la Sangha Landscape. Center for International Forestry Research (CIFOR), 2012. http://dx.doi.org/10.17528/cifor/003933.

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Singer, S., and A. Simon. EEBHub Building 101 Sankey Diagram Energy Analysis. Office of Scientific and Technical Information (OSTI), January 2013. http://dx.doi.org/10.2172/1062226.

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Cruz Suarez, Ana Gabriela. Desmitificar la hiponatremia y el síndrome de desmielinización osmótica. Facultad de Medicina Universidad de Antioquia, April 2024. http://dx.doi.org/10.59473/medudea.pc.2024.76.

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Abstract:
Paciente femenina de 66 años, con cuadro clínico de 3 días de evolución consistente en diarrea acuosa en múltiples ocasiones sin sangre ni moco, acompañado de náuseas y emesis, que se autolimitaron, pero desde hace 1 día presenta somnolencia progresiva y desorientación; antecedentes de importancia, hipertensión arterial manejada con hidroclorotiazida 25 mg/día.
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Singer, S., and A. Simon. EEBHub Navy Yard Sankey Diagram Energy Analysis Final Report. Office of Scientific and Technical Information (OSTI), June 2013. http://dx.doi.org/10.2172/1088454.

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Singer, S., and A. Simon. EEBHub Navy Yard Sankey Diagram Energy Analysis Final Report. Office of Scientific and Technical Information (OSTI), January 2013. http://dx.doi.org/10.2172/1062224.

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Hamilton, Lawrence. Trump and Sanders Supporters Differ Sharply on Key Scientific Fact. University of New Hampshire Libraries, 2015. http://dx.doi.org/10.34051/p/2020.245.

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T., Devisscher, Bharwani S., Tiani A.M., Pavageau C., Kwack N.E., and Taylor R. Current vulnerability in the Tri-National de la Sangha landscape, Cameroon. Center for International Forestry Research (CIFOR), 2013. http://dx.doi.org/10.17528/cifor/004084.

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