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Books on the topic 'Saccadic eye movements'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Enderle, John D. Models of horizontal eye movements: A 3rd order linear saccade model. San Rafael, Calif. (1537 Fourth Street, San Rafael, CA 94901 USA): Morgan & Claypool, 2010.

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2

Transsakkadische Informationsverarbeitung im visuellen System: Auswirkungen auf Wahrnehmung und Mustererkennung. Regensburg: S. Roderer, 1993.

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3

Eizenman, Moshe. Continuity and asymmetry in amplitude-duration relations for normal eye saccades. Toronto: Dept. of Computer Science, University of Toronto, 1986.

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4

Lücke, Stephan Otto. Über die Wirkung geringer Alkoholdosen auf die sakkadischen Augenbewegungen in Leistungsaufgaben. Aachen: Verlag Shaker, 1993.

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5

Doma, Hansraj. Aspects of saccadic eye-movements towards or away from photopic, mesopic, or scotopic stimuli. Toronto: University of Toronto, Department of Physiology, 1986.

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6

1954-, Kuperstein Michael, ed. Neural dynamics of adaptive sensory-motor control: Ballistic eye movements. Amsterdam: North-Holland, 1986.

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7

Grossberg, Stephen. Neural dynamics of adaptive sensory-motor control. New York: Pergamon Press, 1989.

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8

-Ing, Becker Wolfgang Dr, Deubel Heiner, and European Conference on Eye Movements (9th : 1997 : Ulm, Germany), eds. Current oculomotor research: Physiological and psychological aspects. New York: Plenum Press, 1999.

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9

H, Wurtz Robert, and Goldberg Michael E, eds. The Neurobiology of saccadic eye movements. Amsterdam: Elsevier, 1989.

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10

Saccadic eye movements during space flight. [Washington, DC]: National Aeronautics and Space Administration, 1989.

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11

Vokoun, Corinne R., Safraaz Mahamed, and Michele A. Basso. Saccadic eye movements and the basal ganglia. Oxford University Press, 2011. http://dx.doi.org/10.1093/oxfordhb/9780199539789.013.0012.

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12

Krantz, John H. Changes in detectability of direction and motion associated with saccadic eye movements. 1988.

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13

Enderle, John D., and Alireza Ghahari. Models of Horizontal Eye Movements: Part 3, a Neuron and Muscle Based Linear Saccade Model. Springer International Publishing AG, 2014.

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14

Enderle, John D., and Alireza Ghahari. Models of Horizontal Eye Movements: Part 4, a Multiscale Neuron and Muscle Fiber-Based Linear Saccade Model. Springer International Publishing AG, 2015.

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15

Enderle, John D., and Alireza Ghahari. Models of Horizontal Eye Movements: Part 4, a Multiscale Neuron and Muscle Fiber-Based Linear Saccade Model. Morgan & Claypool Publishers, 2015.

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16

Enderle, John D., and Alireza Ghahari. Models of Horizontal Eye Movements: Part 3, a Neuron and Muscle Based Linear Saccade Model. Morgan & Claypool Publishers, 2014.

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17

Kristjánsson, Árni. The intriguing interactive relationship between visual attention and saccadic eye movements. Oxford University Press, 2011. http://dx.doi.org/10.1093/oxfordhb/9780199539789.013.0025.

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18

Paré, Martin, and Michael C. Dorris. The role of posterior parietal cortex in the regulation of saccadic eye movements. Oxford University Press, 2011. http://dx.doi.org/10.1093/oxfordhb/9780199539789.013.0014.

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19

Fisch, Adam. Eye Movements. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199845712.003.0306.

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20

[The strategic organization of skill]: Final technical report. [Denver, Colo.]: University of Denver, 1996.

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21

Doma, Hansraj. Sensory aspects of saccadic eye-movement latency. 1987.

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22

Beh, Shin C., Elliot M. Frohman, and Teresa Frohman. Neuro-ophthalmologic Manifestations of Multiple Sclerosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0012.

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The inflammatory, demyelinating plaques that characterize multiple sclerosis (MS) frequently affect the visual pathways. Lesions of the afferent visual pathway (most commonly optic neuritis) result in problems conveying visual stimuli from the retina to the visual cortices. Lesions affecting the efferent visual system result in ocular dysmotility that impairs visual acuity by disrupting the precision of binocular eye movements or by causing excessive eye movements that prevent adequate foveation (e.g. nystagmus, saccadic intrusions). Significant advancements have been made in the techniques used to interrogate both the structural and the functional integrity of the visual system to dissect the pathobiological underpinnings of multiple sclerosis and to design better biomarkers and clinical trial outcomes. This chapter discusses the neuro-ophthalmic manifestations of multiple sclerosis, revolutionary advancements in optical coherence tomography and visual electrophysiology, and therapies for treating visual dysfunction in multiple sclerosis.

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23

Mason, Peggy. Gaze Control. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0019.

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In addition to serving perception, gaze acts as a powerful social signal and mode of communication. Gaze is altered in several psychiatric diseases and impaired by a variety of central and peripheral lesions. Eye movements that serve to stabilize gaze include the vestibuloocular reflex (VOR) and fixation, whereas eye movements that shift gaze include saccades, cancellation of the VOR, and smooth pursuit. The pontine horizontal gaze center and midbrain vertical gaze center connect to extraocular motoneurons and mediate all eye movements. Neural circuits involved in generating the VOR, horizontal saccades and saccade modulation are described in detail. Nystagmus consequent to unilateral labyrinthine damage is explained. Other forms of nystagmus including the optokinetic response are introduced. The role of internuclear interneurons in coordinating horizontal saccades and their failure in internuclear ophthalmalplegia are detailed. Finally, the mechanisms involved in fixation and smooth pursuit are briefly presented.

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24

d', Ydewalle Géry, Rensbergen Johan van, and European Conference on Eye Movements (6th : 1991 : University of Leuven), eds. Visual and oculomotor functions: Advances in eye movement research. Amsterdam: North-Holland, 1994.

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25

Deubel, Heiner, Thomas Mergner, and Wolfgang Becker. Current Oculomotor Research: Physiological and Psychological Aspects. Springer London, Limited, 2013.

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26

Models of Horizontal Eye Movements, Part I Pt. 1: Early Models of Saccades and Smooth Pursuit. Morgan & Claypool Publishers, 2010.

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27

(Editor), Wolfgang Becker, Heiner Deubel (Editor), and Thomas Mergner (Editor), eds. Current Oculomotor Research: Physiological and Psychological Aspects. Springer, 1999.

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28

Palmeri, Thomas J., Jeffrey D. Schall, and Gordon D. Logan. Neurocognitive Modeling of Perceptual Decision Making. Edited by Jerome R. Busemeyer, Zheng Wang, James T. Townsend, and Ami Eidels. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199957996.013.15.

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Mathematical psychology and systems neuroscience have converged on stochastic accumulator models to explain decision making. We examined saccade decisions in monkeys while neurophysiological recordings were made within their frontal eye field. Accumulator models were tested on how well they fit response probabilities and distributions of response times to make saccades. We connected these models with neurophysiology. To test the hypothesis that visually responsive neurons represented perceptual evidence driving accumulation, we replaced perceptual processing time and drift rate parameters with recorded neurophysiology from those neurons. To test the hypothesis that movement related neurons instantiated the accumulator, we compared measures of neural dynamics with predicted measures of accumulator dynamics. Thus, neurophysiology both provides a constraint on model assumptions and data for model selection. We highlight a gated accumulator model that accounts for saccade behavior during visual search, predicts neurophysiology during search, and provides insights into the locus of cognitive control over decisions.

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29

dʼ, Ydewalle Géry, and Rensbergen Johan van, eds. Perception and cognition: Advances in eye movement research. Amsterdam: North-Holland, 1993.

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30

Enderle, John, and Wei Zhou. Models of Horizontal Eye Movements, Part II: A 3rd Order Linear Saccade Model. Springer International Publishing AG, 2010.

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31

Enderle, John. Models of Horizontal Eye Movements, Part I: Early Models of Saccades and Smooth Pursuit. Springer International Publishing AG, 2010.

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32

Enderle, John. Models of Horizontal Eye Movements, Part I: Early Models of Saccades and Smooth Pursuit. Morgan & Claypool Publishers, 2010.

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33

Enderle, John D., and Alireza Ghahari. Models of Horizontal Eye Movements Pt. 3: A Neuron and Muscle Based Linear Saccade Model. Morgan & Claypool Publishers, 2014.

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34

Gottlieb, Jacqueline. Neuronal Mechanisms of Attentional Control. Edited by Anna C. (Kia) Nobre and Sabine Kastner. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199675111.013.033.

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Damage to the human inferior parietal lobe produces an attentional disturbance known as contralateral neglect, and neurophysiological studies in monkeys have begun to unravel the cellular basis of this function. Converging evidence suggests that LIP encodes a sparse topographic map of the visual world that highlights attention-worthy objects or locations. LIP cells may facilitate sensory attentional modulations, and ultimately the transient improvement in perceptual thresholds that is the behavioural signature of visual attention. In addition, LIP projects to oculomotor centres where it can prime the production of a rapid eye movement (saccade). Importantly, LIP cells can select visual targets without triggering saccades, showing that they implement an internal (covert) form of selection that can be flexibly linked with action by virtue of additional, independent mechanisms. The target selection response in LIP is modulated by bottom-up factors and by multiple task-related factors. These modulations are likely to arise through learning and may reflect a multitude of computations through which the brain decides when and to what to attend.

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35

Kalesnykas, Rimas Povilas. Sensorimotor aspects of human saccadic eye-movement latency as a function of stimulus eccentricity, stimulus condition, and visuospatial attention. 1994.

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36

Hyona, J., ed. The Brain's Eye: Neurobiological and Clinical Aspects of Oculomotor Research (Progress in Brain Research). Elsevier Science, 2002.

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37

Krauzlis, Richard J. Attentional Functions of the Superior Colliculus. Edited by Anna C. (Kia) Nobre and Sabine Kastner. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199675111.013.014.

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The superior colliculus (SC) plays an important role in both overt and covert attention. In primates, the SC is well known to be a central component of the motor pathways that orient the eyes and head to important objects in the environment. Accordingly, neurons in the SC show enhanced responses that will be the target of orienting movements, compared to stimuli that will be ignored. Single-neuron recordings in the SC have revealed a variety of attention-related effects, including changes in activity related to bottom-up and top-down attention, attention capture, and inhibition of return. These findings support the view of the SC as a priority map that represents the location of important objects in the visual environment. Manipulation of SC activity by electrical microstimulation and chemical inactivation shows that the SC is not simply a recipient of attention-related effects, but plays a causal role in these processes. In particular, activity in the SC plays a major role in the selection of targets for saccades, and also for pursuit eye movements and movements of the hand. Moreover, activity in the SC is important not only for the control of overt attention, but also plays a crucial role in covert attention—the processing of visual signals for perceptual judgements even in the absence of orienting movements. The mechanisms mediating the role of the SC in the control of covert attention are not yet known, but current models emphasize interactions between the SC and areas of the cerebral cortex.

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