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Academic literature on the topic 'Rubicon'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Rubicon"

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Espinoza, Sandra, Felipe Grunenwald, Wileidy Gomez, Felipe García, Lorena Abarzúa-Catalan, Sebastián Oyarce-Pezoa, Maria Fernanda Hernandez, et al. "Neuronal Rubicon Represses Extracellular APP/Amyloid β Deposition in Alzheimer’s Disease." Cells 11, no. 12 (June 7, 2022): 1860. http://dx.doi.org/10.3390/cells11121860.

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Alzheimer’s disease (AD) is the most prevalent age-associated neurodegenerative disease. A decrease in autophagy during aging contributes to brain disorders by accumulating potentially toxic substrates in neurons. Rubicon is a well-established inhibitor of autophagy in all cells. However, Rubicon participates in different pathways depending on cell type, and little information is currently available on neuronal Rubicon’s role in the AD context. Here, we investigated the cell-specific expression of Rubicon in postmortem brain samples from AD patients and 5xFAD mice and its impact on amyloid β burden in vivo and neuroblastoma cells. Further, we assessed Rubicon levels in human-induced pluripotent stem cells (hiPSCs), derived from early-to-moderate AD and in postmortem samples from severe AD patients. We found increased Rubicon levels in AD-hiPSCs and postmortem samples and a notable Rubicon localization in neurons. In AD transgenic mice lacking Rubicon, we observed intensified amyloid β burden in the hippocampus and decreased Pacer and p62 levels. In APP-expressing neuroblastoma cells, increased APP/amyloid β secretion in the medium was found when Rubicon was absent, which was not observed in cells depleted of Atg5, essential for autophagy, or Rab27a, required for exosome secretion. Our results propose an uncharacterized role of Rubicon on APP/amyloid β homeostasis, in which neuronal Rubicon is a repressor of APP/amyloid β secretion, defining a new way to target AD and other similar diseases therapeutically.
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Cliff, Michelle. "Rubicon." Callaloo 23, no. 1 (2000): 134–38. http://dx.doi.org/10.1353/cal.2000.0008.

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Matsunaga, Kohichi, Takeshi Noda, and Tamotsu Yoshimori. "Binding Rubicon to cross the Rubicon." Autophagy 5, no. 6 (August 16, 2009): 876–77. http://dx.doi.org/10.4161/auto.9098.

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Bhargava, Hersh K., Keisuke Tabata, Jordan M. Byck, Maho Hamasaki, Daniel P. Farrell, Ivan Anishchenko, Frank DiMaio, Young Jun Im, Tamotsu Yoshimori, and James H. Hurley. "Structural basis for autophagy inhibition by the human Rubicon–Rab7 complex." Proceedings of the National Academy of Sciences 117, no. 29 (July 6, 2020): 17003–10. http://dx.doi.org/10.1073/pnas.2008030117.

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Rubicon is a potent negative regulator of autophagy and a potential target for autophagy-inducing therapeutics. Rubicon-mediated inhibition of autophagy requires the interaction of the C-terminal Rubicon homology (RH) domain of Rubicon with Rab7–GTP. Here we report the 2.8-Å crystal structure of the Rubicon RH domain in complex with Rab7–GTP. Our structure reveals a fold for the RH domain built around four zinc clusters. The switch regions of Rab7 insert into pockets on the surface of the RH domain in a mode that is distinct from those of other Rab–effector complexes. Rubicon residues at the dimer interface are required for Rubicon and Rab7 to colocalize in living cells. Mutation of Rubicon RH residues in the Rab7-binding site restores efficient autophagic flux in the presence of overexpressed Rubicon, validating the Rubicon RH domain as a promising therapeutic target.
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Armstrong, Barry. "Platform: Rubicon." Musical Times 132, no. 1783 (September 1991): 447. http://dx.doi.org/10.2307/965647.

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Zambrano, Joelle N., Scott T. Eblen, Melissa Abt, J. Matthew Rhett, Robin Muise-Helmericks, and Elizabeth S. Yeh. "HUNK Phosphorylates Rubicon to Support Autophagy." International Journal of Molecular Sciences 20, no. 22 (November 19, 2019): 5813. http://dx.doi.org/10.3390/ijms20225813.

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Background: Autophagy is a catabolic cellular recycling pathway that is essential for maintaining intracellular homeostasis. Autophagosome formation is achieved via the coordination of the Beclin-1 protein complex. Rubicon is a Beclin-1 associated protein that suppresses autophagy by impairing the activity of the class III PI3K, Vps34. However, very little is known about the molecular mechanisms that regulate Rubicon function. Methods: In this study, co-immunoprecipitation and kinase assays were used to investigate the ability of Hormonally Upregulated Neu-associated Kinase (HUNK) to bind to and phosphorylate Rubicon. LC3B was monitored by immunofluorescence and immunoblotting to determine whether phosphorylation of Rubicon by HUNK controls the autophagy suppressive function of Rubicon. Results: Findings from this study identify Rubicon as a novel substrate of HUNK and show that phosphorylation of Rubicon inhibits its function, promoting autophagy.
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Kim, Jae-Sung, Ye-Ram Kim, Sein Jang, Sang Geon Wang, Euni Cho, Seok-Jun Mun, Hye-In Jeon, Hyo-Keun Kim, Sun-Joon Min, and Chul-Su Yang. "Mito-TIPTP Increases Mitochondrial Function by Repressing the Rubicon-p22phox Interaction in Colitis-Induced Mice." Antioxidants 10, no. 12 (December 6, 2021): 1954. http://dx.doi.org/10.3390/antiox10121954.

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The run/cysteine-rich-domain-containing Beclin1-interacting autophagy protein (Rubicon) is essential for the regulation of nicotinamide adenine dinucleotide phosphate (NADPH) oxidase by interacting with p22phox to trigger the production of reactive oxygen species (ROS) in immune cells. In a previous study, we demonstrated that the interaction of Rubicon with p22phox increases cellular ROS levels. The correlation between Rubicon and mitochondrial ROS (mtROS) is poorly understood. Here, we report that Rubicon interacts with p22phox in the outer mitochondrial membrane in macrophages and patients with human ulcerative colitis. Upon lipopolysaccharide (LPS) activation, the binding of Rubicon to p22phox was elevated, and increased not only cellular ROS levels but also mtROS, with an impairment of mitochondrial complex III and mitochondrial biogenesis in macrophages. Furthermore, increased Rubicon decreases mitochondrial metabolic flux in macrophages. Mito-TIPTP, which is a p22phox inhibitor containing a mitochondrial translocation signal, enhances mitochondrial function by inhibiting the association between Rubicon and p22phox in LPS-primed bone-marrow-derived macrophages (BMDMs) treated with adenosine triphosphate (ATP) or dextran sulfate sodium (DSS). Remarkably, Mito-TIPTP exhibited a therapeutic effect by decreasing mtROS in DSS-induced acute or chronic colitis mouse models. Thus, our findings suggest that Mito-TIPTP is a potential therapeutic agent for colitis by inhibiting the interaction between Rubicon and p22phox to recover mitochondrial function.
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Yamamuro, Tadashi, Shuhei Nakamura, Yu Yamano, Tsutomu Endo, Kyosuke Yanagawa, Ayaka Tokumura, Takafumi Matsumura, et al. "Rubicon prevents autophagic degradation of GATA4 to promote Sertoli cell function." PLOS Genetics 17, no. 8 (August 5, 2021): e1009688. http://dx.doi.org/10.1371/journal.pgen.1009688.

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Autophagy degrades unnecessary proteins or damaged organelles to maintain cellular function. Therefore, autophagy has a preventive role against various diseases including hepatic disorders, neurodegenerative diseases, and cancer. Although autophagy in germ cells or Sertoli cells is known to be required for spermatogenesis and male fertility, it remains poorly understood how autophagy participates in spermatogenesis. We found that systemic knockout mice of Rubicon, a negative regulator of autophagy, exhibited a substantial reduction in testicular weight, spermatogenesis, and male fertility, associated with upregulation of autophagy. Rubicon-null mice also had lower levels of mRNAs of Sertoli cell–related genes in testis. Importantly, Rubicon knockout in Sertoli cells, but not in germ cells, caused a defect in spermatogenesis and germline stem cell maintenance in mice, indicating a critical role of Rubicon in Sertoli cells. In mechanistic terms, genetic loss of Rubicon promoted autophagic degradation of GATA4, a transcription factor that is essential for Sertoli cell function. Furthermore, androgen antagonists caused a significant decrease in the levels of Rubicon and GATA4 in testis, accompanied by elevated autophagy. Collectively, we propose that Rubicon promotes Sertoli cell function by preventing autophagic degradation of GATA4, and that this mechanism could be regulated by androgens.
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Nah, Jihoon, Daniela Zablocki, and Junichi Sadoshima. "The roles of the inhibitory autophagy regulator Rubicon in the heart: A new therapeutic target to prevent cardiac cell death." Experimental & Molecular Medicine 53, no. 4 (April 2021): 528–36. http://dx.doi.org/10.1038/s12276-021-00600-3.

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AbstractAutophagy contributes to the maintenance of cardiac homeostasis. The level of autophagy is dynamically altered in heart disease. Although autophagy is a promising therapeutic target, only a few selective autophagy activator candidates have been reported thus far. Rubicon is one of the few endogenous negative regulators of autophagy and a potential target for autophagy-inducing therapeutics. Rubicon was initially identified as a component of the Class III PI3K complex, and it has multiple functions, not only in canonical autophagy but also in endosomal trafficking and inflammatory responses. This review summarizes the molecular action of Rubicon in canonical and noncanonical autophagy. We discuss the roles of Rubicon in cardiac stress and the therapeutic potential of Rubicon in cardiac diseases through its modulation of autophagy.
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Etzioni, Amitai. "Crossing the Rubicon." Challenge 57, no. 2 (March 2014): 65–79. http://dx.doi.org/10.2753/0577-5132570205.

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Dissertations / Theses on the topic "Rubicon"

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Solomon, Paul Robert. "Astride the rubicon." The Ohio State University, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=osu1328291022.

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Roy, Rajarshi. "Crossing the Rubicon: LBJ and Vietnam 1963-1965." W&M ScholarWorks, 1997. https://scholarworks.wm.edu/etd/1539626129.

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Williams, J. H. C. "Beyond the Rubicon : Romans and Gauls in Republican Italy /." Oxford : Oxford university press, 2001. http://catalogue.bnf.fr/ark:/12148/cb41151317m.

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Ari, Baris. "Uncrossing the rubicon : transitions from violent civil conflict to peace." Thesis, University of Essex, 2018. http://repository.essex.ac.uk/22371/.

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What are the impediments to and stimuluses for transitions from violent civil conflict to peace? This dissertation investigates factors that influence civil conflict resolution. There are four key findings. First, democratisation reforms are likely to prompt peace talks with rebel groups. There are costs associated with recognising internal armed challengers as legitimate bargaining partners that deter governments from initiating peace talks. Democratic reform periods provide a window of opportunity for peace negotiations because factors that make institutional reform more likely also encourage peaceful resolution of conflict. The emergence of democratic institu- tions changes the state preferences by increasing the influence of the median citizen vis-`a-vis the authoritarian elite. Second, fragmented conflicts are harder to resolve because they are likely to be over a multidimensional issue space. Multiparty decision making that involves two or more salient issues are likely to have cyclic collective preferences, which render armed conflict a viable instrument for pursuing political goals. Third, involvement of the United Nations (UN) mitigates the adverse impact of conflict fragmentation over peaceful resolution. UN involvement changes the incentives and opportunities of actors to founder a possible bargain. The UN fa- cilitates a path-dependent peace process and brings institutions that induce an equilibrium by overcoming the problems that arise due to cyclic collective preferences. Finally, the military manpower supply system of a state influences its civil conflict processes. How a state recruits rank-and-file members of the military forces is a central institutional arrangement that influences the incentives and opportunities of relevant actors. Compared to all-volunteer forces (AVFs), conscription is an impediment to conflict termination because the cost some individuals incur by being subject to conscription decreases the opportunity cost of rebellion, increases grievances and insulates influential sections of the population from the cost of conflict. As a result, conflict termination becomes less likely.
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Nascimento, Elias. "Estudo das estratégias lingüístico-discursivas do risível em Porque Lulu Bergantim não atravessou o Rubicon de José Cândido de Carvalho." Pontifícia Universidade Católica de São Paulo, 2008. https://tede2.pucsp.br/handle/handle/14500.

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Made available in DSpace on 2016-04-28T19:34:24Z (GMT). No. of bitstreams: 1 Elias Nascimento.pdf: 94720526 bytes, checksum: 5d9e140975a2ba8884c72826cf3db0cb (MD5) Previous issue date: 2008-04-23
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The objective of this dissertation is the study of the linguistic-discursive strategies of the laughable in Porque Lulu Bergatim não atravessou o Rubicon of José Cândido de Carvalho. The author s work presents dozens of curious stories on the interior of the country. Written in a very characteristic way, the narratives introduce funny personages involved in unusual situations. The author's style is marked, mainly, by the use of the comic exaggeration employed in ironies and metaphors. The present study included the analysis of stories, their connection with the laughter and the brevity inherent to both. In the present work the manifestation of the laughter was analyzed considering the concepts discussed by the philosophers and theoretical from the Antiquity to the current days. In the study it was verified that in the last centuries the comedian was own of the inferior classes, lowered, with addictions and faults. Starting from the century XX the laughter began to have value in the social relationships. In the contemporary theories, the laughter exists to correct habits, to punish behaviors and to punish distracted, and it can be expressed in four ways: in the forms and movements, in the situations, in the words and in the character. The corpus was divided considering these four categories. The analysis of the linguistic-discursive strategies of the laughter detached the polissemic exploration happened in the stories not only by the use of the metaphors and of the ironies, but also by the use of slangs, of alliterations, of hyperboles, of puns, of strange names and of neologisms
O objetivo desta dissertação é estudar as estratégias lingüístico-discursivas do risível em Porque Lulu Bergatim não atravessou o Rubicon de José Cândido de Carvalho. A obra reúne dezenas de contos curiosos sobre o interior do país. Escritas de forma bastante característica, as narrativas apresentam personagens divertidos envolvidos em situações insólitas. O estilo do autor é marcado, principalmente, pelo uso do exagero cômico empregado às ironias e às metáforas. O estudo abrangeu o gênero conto, sua ligação com o riso e o laconismo inerente a ambos. Compreendeu a manifestação do riso a partir de um levantamento histórico que considerou os conceitos discutidos pelos filósofos e teóricos desde a Antiguidade até os dias atuais. Nesse estudo verificou-se que nos séculos passados o cômico era próprio das classes inferiores, rebaixadas, com vícios e falhas. A partir do século XX o riso passou a ter valor nas relações sociais. Nas teorias contemporâneas, o riso existe para corrigir costumes, castigar comportamentos e punir distraídos, e pode se manifestar de quatro maneiras: nas formas e movimentos, nas situações, nas palavras e no caráter. O corpus foi dividido segundo estas quatro categorias. A análise das estratégias lingüístico-discursivas do riso destacou a exploração polissêmica ocorrida nos contos não só pela utilização das metáforas e das ironias, mas também pelo uso de gírias, de aliterações, de hipérboles, de trocadilhos, de nomes estranhos e de neologismos
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Milward, Sara Eve. "Interrogating plant Rubisco-Rubisco activase interactions." Phd thesis, Canberra, ACT : The Australian National University, 2018. http://hdl.handle.net/1885/149565.

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Atmospheric CO2 fixation is catalysed by the photosynthetic enzyme ribulose-1,5-bisphosphate carboxylase/oxygenase (Rubisco). Despite the critical role Rubisco plays in the biosphere, it is a slow catalyst that poorly discriminates between substrate CO2 and O2, and is often the rate-limiting step of photosynthesis. These deficiencies have made improving Rubisco function a major target in steps towards enhancing leaf photosynthesis rate and plant growth. In pursuing this goal, one strategy is to identify solutions for improving the kinetics of plant Rubisco and introduce these altered Rubisco isoforms into crops (Sharwood, 2017). Unfortunately efforts to improve the performance of plant Rubiscos have so far proven unsuccessful. Success appears to be hindered by Rubisco’s complex catalytic mechanism and the extensive array of accessory proteins needed to assemble the eight large (L) and eight small (S) subunits into a L8S8 complex and to maintain it in a functional form. The complex catalytic chemistry of Rubisco is prone to inhibition by various sugar-phosphate ligands. These include catalytic misfire products as well as its own substrate, ribulose-1,5-bisphosphate (RuBP), which forms an inactive Rubisco-RuBP (ER) complex when bound to non-carbamylated (i.e. non-activated) catalytic sites. Release of these inhibitors is mediated by the AAA+ (ATPases associated with a variety of cellular activities) protein Rubisco activase (Rca); a Rubisco-specific metabolic repair chaperone for which there are convergently evolved structural isoforms found in most photosynthetic organisms (Bhat et al., 2017a; Mueller-Cajar, 2017). Rca is now also a promising target for manipulating photosynthetic performance, especially under elevated temperature stress and fluctuating light (Carmo-Silva & Salvucci, 2013; Kumar et al., 2009; Kurek et al., 2007; Scafaro et al., 2016; Yamori et al., 2012). Critical to future Rubisco and Rca engineering efforts in plants is a greater mechanistic understanding of how Rca interacts with Rubisco, and the extent to which the kinetics of Rca differ between plant species. Utilising chloroplast transformation in tobacco, this thesis investigated the H9 helix and N-terminal region of Rca, and the βC-βD loop and C terminus of the Rubisco large (L-) subunit (RbcL) to determine their role in the Rubisco-Rca interaction in plants using both in vivo and in vitro methods. Capitalising on the regulatory incompatibility between Rubisco and Rca enzymes from Solanaceae and non-Solanaceae species, this thesis examined how mutations in the tobacco (Solanaceae) and Flaveria (F. bidentis and F. floridana, non-Solanaceae) enzymes influenced their kinetic properties and the Rca activation potential for inhibited ER complexes. The ability to continuously monitor the rate of ER activation provided by a NADH-coupled spectrophotometric method made it preferential to the alternative two-step 14CO2-fixation assay method. Optimising the assay conditions, particularly the accurate determination of enzyme concentrations, was crucial for obtaining reproducible kinetic values. Normalising the measurements of ER activation rate relative to the Rca ATPase activity (kcatATP ) proved critical in providing a means to determine the effect the Rca and Rubisco mutations had on interactivity. Using these assay conditions, mutagenic analyses of the Rca H9 helix found residue 317 plays a dominant role in defining the Rubisco selectivity of NtRca, and is critical for functional FbRca. N-terminal domain swapping modifications to NtRca and FbRca revealed amino acids at the junction of the N-terminal extension and the AAA+ module (residues 50 to 85) significantly influenced both kcatATP and the potential to interact with their cognate Rubiscos, but had little influence on altering interaction with the non-native Rubisco. In vivo analysis of Rubisco-Rca interaction was undertaken using tobacco genotypes producing differing tobacco Rubisco or recombinant Flaveria Rubisco mutants generated by chloroplast transformation of the rbcL gene into the plastome. In vitro analysis using purified enzymes uniformly showed residues 89 and 94 of the βC-βD loop of the F. bidentis, F. floridana and tobacco RbcLs influence interaction with Rca. In contrast, in vivo leaf gas exchange measurements of photosynthetic induction in the tobacco genotypes producing mutant RbcL showed mutation of residues 89 and 94 had little to no influence on their capacity to be activated by the endogenous NtRca. Combinatorial in vitro analyses using the range of RbcL and Rca mutants generated in this study were undertaken to better understand their interaction mechanism. It was found the avidity of Rubisco-Rca interactions were primarily defined by residue 94 in RbcL and 317 in Rca, and less so by RbcL residue 89 and Rca residue 320. Mutations to the RbcL C terminus had little influence on plant Rubisco-Rca interactions except when coupled with mutations that disrupted the connection between the RbcL βC-βD loop and the Rca H9 helix. The cumulative effect of the RbcL βC-βD loop and C-terminal modifications on Rubisco-Rca interactivity of these mutants implies involvement of the RbcL C terminus in the activation mechanism of Rca. This thesis showcases a novel approach to study the mechanism of plant Rubisco-Rca interactions by generating mutant Rubisco variants through tobacco plastome transformation. The work highlights large variations in the kinetics of NtRca (Solanaceae) and FbRca (non-Solanaceae) and provides evidence for variations in their Rubisco activation mechanism. For instance, while a D317K substitution in NtRca significantly enhanced its ability to activate Flaveria Rubisco, the reciprocal K317D mutation in FbRca had little impact on improving its capacity to activate tobacco Rubisco. Improving our understanding of the mechanistic differences in plant Rubisco-Rca interactions is critical for future Rubisco engineering endeavours. It is especially relevant in the context of ongoing efforts to transplant more efficient Rubisco variants into leaf chloroplasts where co-transformation with an appropriate Rca may be needed. Potential transgenic strategies for co-engineering compatible Rubisco and Rca isoforms in planta are also discussed.
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Oliveira, Silvana, and Marilene 1952 Weinhardt. "Murilo Rubiao." reponame:Repositório Institucional da UFPR, 2010. http://hdl.handle.net/1884/24515.

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Luokkanen, J. (Jani). "Rubikin kuution matematiikan tiivistelmä." Bachelor's thesis, University of Oulu, 2016. http://urn.fi/URN:NBN:fi:oulu-201611223091.

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Luokkanen, J. (Jani). "Rubikin kuutio ja ryhmäteoria." Master's thesis, University of Oulu, 2018. http://urn.fi/URN:NBN:fi:oulu-201811082996.

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Tutkielman tarkoitus on selvittää Rubikin kuution matemaattinen esitysmuoto ryhmäteorian keinoin. Tämän matemaattisen esitysmuodon rakentamiselle tarvittavat ryhmäteoreettiset tulokset ja rakenteet määritellään ja todistetaan lähtien perusasioista, kuten ryhmän ja aliryhmän määritelmistä, aliryhmyysehdoista, Lagrangen lauseesta jne. Ryhmille olennaisten ja perustavanlaatuisten tulosten jälkeen esitetään yksi olennaisimmista käsitteistä; permutaatio, sekä siihen liittyviä tuloksia ja käsitteitä, kuten symmetrinen ryhmä ja pariteetti. Tutkielma etenee ryhmien rakenneyhtäläisyyden, eli homomorfismin käsitteeseen, jota edelleen käytetään edellä määriteltyjen käsitteiden tärkeiden tulosten todistamiseen, kuten esimerkiksi siihen, että pariteettikuvaus on homomorfismi. Lisäksi määritellään rakenneyhtenevyys eli isomorfismi, ja todistetaan tärkeä homomorfismien peruslause, joka yhdistää isomorfismille luonnolliset joukot toisiinsa, jonka avulla Rubikin kuution ryhmän kertaluku saadaan selville. Ryhmien sisäisten operaatioiden ja välisten kuvausten jälkeen siirrytään ryhmien välisiin operaatioihin, joissa rakennetaan kuution ryhmärakenteen esitysmuodolle tarpeelliset käsitteet kuten suora tulo, ryhmän toiminta ja kranssitulo, jossa jokin ryhmä toimii toisen ryhmän suorassa tulossa. Lisäksi näistä rakenteista näytetään esimerkkejä, joiden avulla niiden merkitystä avataan. Näiden ryhmäteoreettisten käsitteiden ja tulosten jälkeen teoriaa käytetään itse Rubikin kuution mallintamiseen, kun ensin määritellään Rubikin kuutiosta erotettavat mallinnettavat osat, kuten palatyypit, ratkaistu kuutio, kääntö ja kombinaatio sekä sen ratkeavuus. Lisäksi tärkeässä osassa ovat permutaatio- ja orientaatiotilat, jotka sisältävät tiedot palojen sijainneista ja asennoista kuutiossa. Ennen kuution syvää käsittelyä osoitetaan yksinkertaisella tavalla ratkeavan Rubikin kuution todella olevan esitettävissä ryhmänä. Ryhmärakenne laajennetaan koskemaan kaikkia kombinaatioita, tarkastelussa, jossa permutaatio- ja orientaatiotilat esitetään aikaisemmin määritellyillä käsitteillä, erityisesti kranssitulolla sekä määriteltävällä ketjutusta vastaavalla operaatiolla. Tämän jälkeen kombinaatioiden joukosta karsitaan pois ne, joita ei voi ratkaista sivujen käännöillä osoittamalla kombinaation ratkeavuusehdot; permutaatiotilojen pariteetin yhtenevyys sekä orientaatiosummien nolla-arvoisuus. Tämän jälkeen ryhmärakenteen esitysmuoto on valmis. Tutkielman lopussa on lyhyt katsaus 2×2×2-kuutioon ja sen yhtäläisyyksiin ja eroavuuksiin verrattuna Rubikin kuutioon, jossa edetään samalla logiikalla kuin Rubikin kuution tarkastelussa.
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Saschenbrecker, Sandra. "Folding and assembly of RuBisCO." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-75775.

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Books on the topic "Rubicon"

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Garamvölgyi, László. Rubicon. Budapest: BTR Kft., 2000.

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John, Hooker. Rubicon. Ringwood, Vic., Australia: Penguin Books, 1991.

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Rubicon. New York: St. Martin's Press, 1999.

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Kopf, Gail. Rubicon. Nashville: T. Nelson Publishers, 1993.

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Rubicon. New York: William Morrow, 2008.

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Erickson, Steve. Rubicon Beach. New York: Vintage Books, 1987.

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Rubicon one. London: Arrow, 1985.

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Rubicon Beach. New York: Poseidon Press, 1986.

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Camp, Jeffery. Jeffrey Camp: Rubicon. London: Art Space Gallery, 2007.

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The Rubicon effect. Concord, New Hampshire: Raven's Wing Books, 2013.

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Book chapters on the topic "Rubicon"

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Hantke, Steffen. "Rubicon." In Serienfragmente, 315–32. Wiesbaden: Springer Fachmedien Wiesbaden, 2021. http://dx.doi.org/10.1007/978-3-658-29951-4_19.

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Corradi, Juan E. "Another Rubicon?" In Strategic Impasse, 81–96. New York, NY : Routledge, 2018.: Routledge, 2018. http://dx.doi.org/10.4324/9781315450322-6.

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Etzioni, Amitai. "Crossing the Rubicon." In Library of Public Policy and Public Administration, 55–64. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-69623-2_3.

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Tan, See Seng. "Crossing the Rubicon." In Law of the Sea in South East Asia, 193–209. New York, NY : Routledge, 2019.: Routledge, 2019. http://dx.doi.org/10.4324/9780429021053-12.

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Williams, Katie H. "Crossing the Rubicon." In Belonging After Brain Injury, 91–102. New York: Routledge, 2022. http://dx.doi.org/10.4324/9781003340294-6.

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Cal, Nerea M. "Crossing the Rubicon." In US National Cybersecurity, 28–39. Abingdon, Oxon ; New York, NY : Routledge, 2017. | Series: Routledge studies in conflict, security and technology: Routledge, 2017. http://dx.doi.org/10.4324/9781315225623-3.

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"Rubicon (Rubiсone) River." In The Adriatic Sea Encyclopedia, 297. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50032-0_474.

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Simpson, Thula. "Rubicon." In History of South Africa, 279–94. Oxford University Press, 2022. http://dx.doi.org/10.1093/oso/9780197672020.003.0021.

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Abstract This chapter considers the efforts initiated by various actors to force P.W. Botha across the Rubicon. The American anti-apartheid movement championed boycotts, sanctions, and disinvestment, while a major outflow of capital followed Chase Manhattan's refusal to roll over its short-term loans to South Africa. It was also in this period that efforts were launched by white South African citizens to talk to the ANC. The Commonwealth established an Eminent Persons Group to kickstart negotiations, and the initiative brought the state and the ANC into proximity (if not direct contact) for the first time. The state's destruction of the Commonwealth initiative in May 1986 saw international attention shift to imposing sanctions. One consequence was the US Congress's passage of the Comprehensive Anti-Apartheid Act.
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"THE RUBICON." In Crossing the Rubicon, 149–65. Yale University Press, 2020. http://dx.doi.org/10.2307/j.ctvrdf1zq.16.

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"Palmer’s Rubicon." In American Freethinker, 91–112. University of Pennsylvania Press, Inc., 2020. http://dx.doi.org/10.2307/j.ctv19vbgvh.8.

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Conference papers on the topic "Rubicon"

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Near, Joseph P., and Daniel Jackson. "Rubicon." In the ACM SIGSOFT 20th International Symposium. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2393596.2393667.

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Abadi, Aharon, Ran Ettinger, and Yishai A. Feldman. "Re-approaching the refactoring Rubicon." In the 2nd Workshop. New York, New York, USA: ACM Press, 2008. http://dx.doi.org/10.1145/1636642.1636652.

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TALLERMAN, MAGGIE. "IS THE SYNTAX RUBICON MORE OF A MIRAGE?" In EVOLANG 10. WORLD SCIENTIFIC, 2014. http://dx.doi.org/10.1142/9789814603638_0042.

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Harris, Daniel C. "Evolution of the sapphire industry: Rubicon Technology and Gavish." In SPIE Defense, Security, and Sensing, edited by Randal W. Tustison. SPIE, 2009. http://dx.doi.org/10.1117/12.816843.

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Saggio, Antonino. "Crossing the Rubicon: Tevere Cavo, an Urban Project for Rome." In International Conference on the 4th Game Set and Match (GSM4Q-2019). Qatar University Press, 2019. http://dx.doi.org/10.29117/gsm4q.2019.0022.

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We believe that the new frontiers of Information Technology have to deal with the central role of Infrastructures in the existing city. Indeed, this new generation of infrastructures will allow the 'redirection' of the development. To arrest developments in "Green fields" and direct devel-opments towards "brown areas" in the existing cities we need infrastructures of new generation. In this historical moment, a development phase has to focus on the use of urban voids in the existing city to stop the endless urban sprawl. 'Crossing the Rubicon' was an expression I used years ago - in the preface of Kas Oosterhuis's book "Towards new Architecture"- to underline the role of a generation of architects that put Information Technology at the heart of a new de-velopment phase for architecture. I am using the same expression now to highlight the role that Information Technology has to play to shape new infrastructures. As an example, here I present and discuss the urban project "Tevere Cavo" in Rome.
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Cal, Nerea M. "Crossing the rubicon: identifying and responding to an armed cyber-attack." In 2016 International Conference on Cyber Conflict (CyCon U.S.). IEEE, 2016. http://dx.doi.org/10.1109/cyconus.2016.7836612.

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Mikhanoshina, Julia L., Alexander L. Vereshchagin, and Eugene V. Sypin. "Research of the spectral characteristics of the LEDs of “Rubicon” firm for plants growing." In 2012 IEEE 13th International Conference and Seminar of Young Specialists on Micro/Nanotechnologies and Electron Devices (EDM 2012). IEEE, 2012. http://dx.doi.org/10.1109/edm.2012.6310223.

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Roudaut, Anne, Diego Martinez, Amir Chohan, Vlad-Stefan Otrocol, Rupert Cobbe-Warburton, Max Steele, and Ioana-Madalina Patrichi. "Rubikon." In CHI '14: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2559206.2581275.

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Zhou, Hualu, Giang Vu, and David J. McClements. "Rubisco Proteins as Plant-based Alternatives to Egg White Proteins: Characterization of Thermal Gelation Properties." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/vamx3998.

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RuBisCO proteins can be isolated from abundant and sustainable plant sources, such as duckweed (e.g., Lemnoideae). These plant-based globular proteins are capable of irreversibly unfolding and forming gels when heated, which means they may be able to mimic some of the functional attributes exhibited by animal globular proteins. In this study, we examined the ability of RuBisCo proteins to mimic the initial rheology and thermal gelation properties of egg white, which the aim of developing plant-based egg analogs. The impact of protein concentration (10-15% w/w), pH (7 to 9), and calcium concentration (0 to 50 mM CaCl2) on the properties of the egg white analogs was examined. The appearance (colorimetry), thermal denaturation (differential scanning calorimetry), thermal gelation (dynamic shear rheology), and texture profiles (compression testing) were measured. RuBisCO-based egg white analogs could be successfully produced at 10% protein content and pH 8 in the absence of salt. These RuBisCO protein solutions had similar apparent viscosity-shear rate profiles, shear modulus-temperature profiles, gelling temperatures, and final gel strengths as egg white. However, there were some differences. RuBisCO protein gels were slightly darker than egg white, which was attributed to the presence of some phenolic impurities. RuBisCo protein exhibited a single thermal transition temperature (~ 66 ℃) whereas egg white exhibited two (~66 and ~81 ℃). RuBisCo protein gels were more brittle but less chewy and resilient than egg white gels. This study provides valuable insights into the potential of RuBisCo protein for formulating plant-based egg white analogs, which may help improve the sustainability of the modern food supply.
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Vybornova, Olga, Igor Pidchenko, and Iskandar Azhmukhamedov. "Information Security Risk Assessment Methodology and Software “Rubikon”." In Proceedings of the 21st International Workshop on Computer Science and Information Technologies (CSIT 2019). Paris, France: Atlantis Press, 2019. http://dx.doi.org/10.2991/csit-19.2019.40.

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Reports on the topic "Rubicon"

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Miller, John. Japan Crosses the Rubicon? Fort Belvoir, VA: Defense Technical Information Center, January 2002. http://dx.doi.org/10.21236/ada417346.

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Salvucci, Michael. Consequences of altering rubisco regulation. Office of Scientific and Technical Information (OSTI), December 2013. http://dx.doi.org/10.2172/1164812.

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Spreitzer, Robert Joseph. Role of the Rubisco Small Subunit. Office of Scientific and Technical Information (OSTI), November 2016. http://dx.doi.org/10.2172/1330984.

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Zielinski, R. (Structure and expression of nuclear genes encoding rubisco activase). Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/6993018.

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Hartman, F. C. Rubisco Mechanism: Dissection of the Enolization Partial Reaction. Final Report. Office of Scientific and Technical Information (OSTI), June 2003. http://dx.doi.org/10.2172/824531.

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Zielinski, R. E. Structure and expression of nuclear genes encoding rubisco activase. Final technical report. Office of Scientific and Technical Information (OSTI), June 1994. http://dx.doi.org/10.2172/10154999.

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Spreitzer, Robert J. Role of the Rubisco small subunit. Final report for period May 1, 1997--April 30,2000. Office of Scientific and Technical Information (OSTI), October 2000. http://dx.doi.org/10.2172/809467.

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Amon, A., K. Bechtol, A. Connolly, S. Digel, A. Drlica-Wagner, E. Gawiser, M. Jarvis, et al. Recommended Target Fields for Commissioning the Vera C. Rubin Observatory. Office of Scientific and Technical Information (OSTI), October 2020. http://dx.doi.org/10.2172/1833575.

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Yu, Weixiang, Gordon Richards, Peter Yoachim, and Christina Peters. A Metric for Differential Chromatic Refraction in the Context of the Legacy Survey of Space and Time. Github.com, 2020. http://dx.doi.org/10.17918/f5dn-8510.

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We provide a code repository for computing a metric to investigate how measurements of differential chromatic refraction might influence choices for survey strategy in the Rubin Observatory Legacy Survey of Space and Time.
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Rubiano, Kristian, and Dalia C. Barragán Barrera. ¿Las áreas protegidas sí están protegiendo nuestra biodiversidad? Universidad del Rosario, November 2022. http://dx.doi.org/10.12804/dvcn_10336.36920_num6.

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En Colombia existen actualmente 1.391 áreas protegidas y tanto en su interior como en sus fronteras (zonas de amortiguación) se desarrollan muchas actividades humanas. El estudiante doctoral de la Facultad de Ciencias Naturales Kristian Rubiano mapeó 51 áreas protegidas para determinar las trayectorias de cambio en el uso de la tierra. Los resultados, que fueron presentados en la 58a Reunión Anual de la Asociación para la Biología Tropical y Conservación (ATBC), mostraron que las áreas protegidas están cumpliendo parcialmente su rol de conservar los ecosistemas.
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