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Dissertations / Theses on the topic 'Rho GTPases Signaling'

Author: Grafiati

Published: 7 September 2023

Last updated: 31 July 2025

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1

Fransson, Åsa. "Cell signaling by Rho and Miro GTPases : Studies of Rho GTPases in Cytoskeletal Reorganizations and of Miro GTPases in Mitochondrial Dynamics." Doctoral thesis, Uppsala University, Ludwig Institute for Cancer Research, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8514.

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<p>The Ras superfamily of GTPases embraces six major branches of proteins: the Ras, Rab, Ran, Arf, Rho and Miro subfamilies. The majority of GTPases function as binary switches that cycle between active GTP-bound and inactive GDP-bound states. This thesis will focus primarily on the biological functions of the Rho and Miro proteins. The Rho GTPases control the organization of the actin cytoskeleton and other associated activities, whereas the Miro GTPases are regulators of mitochondrial movement and morphology. </p><p>A diverse array of cellular phenomena, including cell movement and intracell

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2

Fransson, Åsa. "Cell signaling by Rho and Miro GTPases : studies of Rho GTPases in cytoskeletal reorganizations and of Miro GTPases in mitochondrial dynamics /." Uppsala : Acta Universitatis Upsaliensis : Universitetsbiblioteket [distributör], 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8514.

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3

Chan, Man-lok Mandy, and 陳文樂. "A study of RhoV and PAK4 signaling in hepatocarcinogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47053434.

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4

Sipes, Nisha Schuler. "Cdc42 signaling in extracellular matrix remodeling in three dimensions." University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1253622562.

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5

Picard, Mariêve. "The role of the small Rho GTPases in the signaling mechanisms mediated by the netrin-1 receptor UNC5a." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=19255.

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Netrin-1 is a bifunctional chemotropic cue that attracts or repels different classes of axons through the Deleted in Colorectal Cancer (DCC) and the UNC5 receptors (UNC5a, b, c and d). DCC is implicated in mediating both responses whereas UNC5 receptors are strictly involved in the repulsive events. The intracellular molecular mechanisms underlying axon growth and guidance are still unclear but it is known that this process requires remodeling of the actin cytoskeleton. There is now compelling evidence that Rho GTPases, in

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6

Hoop, Alyssa N. "Rho-Family GTPase Signaling in the Nervous System: An Analysis of the C. elegans RhoGEF UNC-73." University of Toledo / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1404733888.

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7

Saint-Cyr, Proulx Étienne. "Role of the Rho GTPases in the signaling mechanisms regulated by the axon guidance cue Netrin-1 receptors deleted in colorectar cancer and Unc5H1." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98788.

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Chemotropic cues guide the migrating axons of neurons in the developing nervous system. Netrins are bifunctional guidance cues, attracting or repelling different classes of axons. The attraction to netrins is mediated by DCC whereas repulsion is achieved through Unc5H receptors. In this thesis, we demonstrate that Rho GTPases are required for embryonic spinal commissural axon outgrowth induced by Netrin-1. Using N1E-115 neuroblastoma cells, we have found that Rac1 and Cdc42 activities are required for DCC-induced neurite outgrowth. In fibroblasts, DCC was found to trigger actin reorganization

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8

Silva, Gisele Espinha Teixeira da. "Sinalização da GTPase RhoA nas respostas celulares após estresse genotóxico promovido por radiação ultravioleta." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/87/87131/tde-19102016-165552/.

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A via de sinalização da GTPase RhoA atua em diversos processos celulares. Para avaliar o comportamento de RhoA, após estresse causado por radiação ultravioleta, foram gerados clones mutantes que expressam RhoA em seu estado constitutivamente ativo e dominante negativo. Após exposição das linhagens à radiação ultravioleta, observou-se uma maior sensibilidade e um maior tempo de recuperação das linhagens quando a atividade de RhoA é reduzida. Estes prejuízos no reparo prejudicaram a proliferação e sobrevivência celular quando da deficiência na atividade de RhoA. Em linhagens deficientes na via d

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9

Osaki, Juliana Harumi. "O papel de RhoA e Rac1 GTPases nas respostas celulares após danos no DNA induzidos por radiação ionizante gama." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-22092015-075415/.

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O mecanismo pelo qual uma célula responde a algum dano no seu material genético é extremamente importante. Isto ocorre pela rápida ativação da maquinaria de reparo de danos no DNA, a qual é composta por uma rede intrincada de sinalização proteica, culminando no reparo do DNA; porém se o dano for irreparável ocorre ativação de mecanismos de morte celular. RhoA,e Rac1 pertencem a família das pequenas proteínas sinalizadoras Rho GTPases, as quais atuam como interruptores moleculares ciclando entre estado ativo (ligada a GTP) e inativo (ligada a GDP). Os componentes desta família estão relacionado

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10

Marshall, Andrew Keith. "Signalling through Rho GTPases in cardiomyocytes." Thesis, Imperial College London, 2011. http://hdl.handle.net/10044/1/6962.

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Endothelin-1 (ET-1) promotes changes in gene/protein expression in cardiomyocytes leading to hypertrophy. This results from activation of intracellular signalling pathways including small G proteins that activate protein kinases. Thus, ET-1 activates RhoA that stimulates ROCK and PKN, and Ras that promotes activation of extracellular signal-regulated kinases 1/2 (ERK1/2). Microarrays were used to dissect the roles of ERK1/2 vs RhoA in the cardiomyocyte transcriptomic response to ET-1 using PD184352 and C3 endotoxin from C. botulinum (C3T) for selective inhibition of the ERK1/2 cascade and RhoA

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11

Leszczyńska, Katarzyna. "Signalling and function of the small Rho GTPase RhoJ in endothelial cells." Thesis, University of Birmingham, 2011. http://etheses.bham.ac.uk//id/eprint/1495/.

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RhoJ is an endothelial expressed Rho GTPase, and its knock-down impairs endothelial cell (EC) migration and tubulogenesis, increases stress fibre (SF) and focal adhesion (FA) numbers. This work aimed to determine the intracellular localisation of RhoJ, identify its binding partners, test how it is activated and further explore its function in ECs. Endogenous RhoJ localised to FAs and overexpression of its active mutant (daRhoJ) promoted EC migration, and diminished FA and SF numbers. In addition to FAs, overexpressed RhoJ localised also to endosomes and RhoJ knock-down slightly delayed transfe

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12

Hu, Shuang. "Rho GTPase Signaling Modulates Neurotransmission in Caenorhabditis elegans." University of Toledo / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1365182177.

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13

Cordeiro, Joao. "Modulation of Rho GTPase signalling during vaccinia virus infection." Thesis, University College London (University of London), 2009. http://discovery.ucl.ac.uk/17273/.

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Infection by vaccinia virus leads to reorganization of the actin cytoskeleton, changes in cell adhesion, loss of contact inhibition and stimulation of cell motility. Previous work by the group has led to the identification of a viral gene, F11L, which is highly conserved among the orthopoxvirus genomes and is essential for this virus-induced cell mobility. F11L has been shown to interact with the RhoA but does not interact with the two other 'classical’ members of the family Rac1 and Cdc42. Understanding the molecular interaction of F11L and RhoA as well as investigating additional Rho binding

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14

Wallace, S. "Analysis of Rho GTPase signalling pathways regulating epithelial morphogenesis." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/19565/.

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Rho GTPases are molecular switches controlling many aspects of cell behaviour, including cell cycle progression, cell division, migration and morphology. The activity of Rho proteins is regulated by conformational changes induced by binding of guanine nucleotides, with GTP-bound Rho being active and GDP-bound Rho inactive. Families of regulatory proteins have evolved to catalyse cycling between these states. Guanine nucleotide exchange factors (GEFs) catalyse nucleotide exchange, allowing GDP to dissociate and GTP to bind to Rho, thus activating it. GTPase activating proteins (GAPs) enhance th

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15

Grant, Gary. "Rho GTPase signalling in cilia and blood vessel lumen formation." Thesis, University of Leeds, 2017. http://etheses.whiterose.ac.uk/17044/.

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Vascular endothelial cells, the cells that line blood vessels, play roles both in the development of the circulatory system through providing blood vessel integrity, and its homeostasis through responding to changes in the local environment. During development, the acquisition of functionality of new blood vessels, some of which form from pre-existing ones through the process of angiogenesis, requires generation of endothelial lumens to carry the blood, during a process known as lumenogenesis. Throughout new blood vessel formation, endothelial cells go through coordinated and complex cell shap

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16

Mitchell, Dianne Courtenay. "Regulation and function of the Rho GTPase mediated signaling pathways in metastasis and lenticular differentiation." Diss., Texas A&M University, 2003. http://hdl.handle.net/1969.1/5845.

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Modulation of the actin-based cytoskeleton and transcription factor regulation are merely two essential functions in a wide array of cellular activities that the Rho family of small GTPases is responsible for mediating. Aberrations in, or loss of, Rho GTPase signaling has been found to lead to multiple pathologies, including both metastatic progression and lenticular differentiation leading to cataractogenesis. This study has examined the transcriptional regulation of the metastasis suppressor, KiSS-1. Although the mechanism by which KiSS-1 modulates an anti-metastatic effect is not entirely k

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17

Quist, Sven Roy. "Role of Rac1 signalling in epidermal tumour formation." Thesis, University of Cambridge, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708007.

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18

Mota, Monica Alexandra Domingues Serrador da. "Control of programmed cell death by RHO like GTPases activated signalling cascades in the developing neuron." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271401.

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19

Nikolaidou, Kyriaki. "Investigation of the role RHO GTPase signalling in cell shape changes during Drosophila Morphogenesis." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.396361.

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20

Fediuk, Jena. "Thromboxane receptor signaling and Rho GTPase activation on actin polymerization and contraction in hypoxic neonatal pulmonary arterial myocytes." Am J Physiol Lung Cell Mol Physiol, 2012. http://hdl.handle.net/1993/23862.

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INTRODUCTION: Persistent Pulmonary Hypertension of the Newborn (PPHN) is defined as the failure of normal circulatory relaxation in the lungs at birth. Hypoxia is known to impede postnatal disassembly of the actin cytoskeleton in pulmonary arterial (PA) myocytes. Actin polymerization (APM), regulated by Rho GTPases, stabilizes force generation. We studied basal and thromboxane (TP)-induced APM and contraction in normoxic and hypoxic PA myocytes and rings. We also examined the downstream signaling pathways regulating hypoxia and TP-induced APM, and the role that actin plays in TP receptor inter

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21

Alayed, Khaled. "MULTIOMICS EVALUATION OF THE EFFECT OF EZRIN INHIBITOR IN DIFFUSE LARGE B CELL LYMPHOMA REVEALS DYSREGULATION OF BCR SIGNALING, RHO GTPASE SIGNALING AND APOPTOSIS." Case Western Reserve University School of Graduate Studies / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1552485811716089.

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22

Fortin, Ensign Shannon Patricia. "The TWEAK-Fn14 Ligand Receptor Axis Promotes Glioblastoma Cell Invasion and Survival Via Activation of Multiple GEF-Rho GTPase Signaling Systems." Diss., The University of Arizona, 2013. http://hdl.handle.net/10150/293463.

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Glioblastoma (GB) is the highest grade and most common form of primary adult brain tumors, characterized by a highly invasive cell population. GB tumors develop treatment resistance and ultimately recur; the median survival is nearly fifteen months and importantly, the invading cell population is attributed with having a decreased sensitivity to therapeutics. Thus, there remains a necessity to identify the genetic and signaling mechanisms that promote tumor spread and therapeutic resistance in order to develop new targeted treatment strategies to combat this rapidly progressive disease. TWE

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23

Arens, Stefan [Verfasser], and Theresia [Akademischer Betreuer] Stradal. "Characterization of type III secreted bacterial virulence factors that interfere with Rho GTPase signalling / Stefan Arens ; Betreuer: Theresia Stradal." Münster : Universitäts- und Landesbibliothek Münster, 2014. http://d-nb.info/1138279897/34.

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24

Fortin, Ensign Shannon Patricia. "THE TWEAK-FN14 LIGAND RECEPTOR AXIS PROMOTES GLIOBLASTOMA CELL INVASION AND SURVIVAL VIA ACTIVATION OF MULTIPLE GEF-RHO GTPASE SIGNALING SYSTEMS." Thesis, The University of Arizona, 2015. http://hdl.handle.net/10150/528171.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>Glioblastoma (GB) is the highest grade and most common form of primary adult brain tumors, characterized by a highly invasive cell population. GB tumors develop treatment resistance and ultimately recur; the median survival is nearly fifteen months and importantly, the invading cell population is attributed with having a decreased sensitivity to therapeutics. Thus, there remains a necessity to identify the genetic and signaling mec

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25

Smallman, Matthew John. "Spatial regulation of Rho GTPase signalling during root hair development in Arabidopsis thaliana is reliant upon the guanine nucleotide dissociation inhibitor SCN1." Thesis, University of Bristol, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.496221.

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The scn1-1 mutants of Arabidopsis are characterised by spatially deregulated sites of root hair growth. This studydemonstrates that this phenotype is the result of the loss of regulation of members of the plant specific sub-family of Rho small GTPases by the Guanine nucleotide Dissociation Inhibitor (GDI), encoded by SCN1. The small GTPases R0P2, R0P4 and R0P5 of Arabidopsis are members of the I subset of type I ROPs that terminate with a conserved CXXL box and undergo 3 prenylation. These small GTPases are expressed in trichoblasts, and localize in patters suggestive of specific roles through

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26

Kitade, Yuki. "Functional analysis of signaling components regulating pathogenicity and asexual/sexual development in Bipolaris maydis." Kyoto University, 2019. http://hdl.handle.net/2433/242717.

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Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(農学)<br>甲第21840号<br>農博第2353号<br>新制||農||1069(附属図書館)<br>学位論文||H31||N5212(農学部図書室)<br>京都大学大学院農学研究科地域環境科学専攻<br>(主査)教授田中千尋, 教授本田与一, 准教授刑部正博<br>学位規則第4条第1項該当

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27

Dubash, Adi Dara Burridge Keith. "Regulation of RhoA GTPase signaling by guanine nucleotide exchange factors in response to extracellular matrix adhesion and DNA damage." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2009. http://dc.lib.unc.edu/u?/etd,2407.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2009.<br>Title from electronic title page (viewed Sep. 3, 2009). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Cell and Developmental Biology within the School of Medicine." Discipline: Cell and Developmental Biology; Department/School: Medicine.

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28

Schenck, Annette. "CYFIP, a protein family implicated in neuronal connectivity, links Rac1 GTPase signalling to the fragile X mental retardation protein." Université Louis Pasteur (Strasbourg) (1971-2008), 2003. http://www.theses.fr/2003STR13175.

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Le syndrome de l'X Fragile, qui constitue la forme la plus fréquente de retard mental héréditaire, est du à l'absence de FMRP, une protéine de liaison à l'ARN qui régulerait la traduction au niveau synaptique. Afin de mieux comprendre le role de FMRP, nous avons réalisé un criblage double-hybride dans la levure pour identifier certains de ses interacteurs. Deux protéines ont été isolées, CYFIP1 et CYFIP2 (Cytoplasmic FMRP Interacting Proteins 1/2). Ces deux protéines cytoplasmiques sont hautement homologues mais interagissent différement avec les deux autres protéines de la famille FXR, FXR1P

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29

Carie, Adam E. "Tumor suppressive effects of the Beta-2 adrenergic receptor and the small GTPase RhoB." [Tampa, Fla.] : University of South Florida, 2008. http://purl.fcla.edu/usf/dc/et/SFE0002330.

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30

Lam, Jonathan Lam. "Identification of mammalian cell signaling in response to plasma membrane perforation: Endocytosis of Listeria monocytogenes and The Repair Machinery." The Ohio State University, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu1543497502225763.

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31

Mills, Evan. "Engineering Synthetic Control over Rho GTPases using Ca2+ and Calmodulin Signaling." Thesis, 2012. http://hdl.handle.net/1807/34811.

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Engineered protein systems have been created to impart new functions, or “re-program” mammalian cells for applications including cancer and HIV/AIDS therapies. The successful development of mammalian cells for re-programming will depend on having well-defined, modular systems. Migration is a particularly important cell function that will determine the efficiency and efficacy of many re-programming applications in vivo, and Rho proteins are responsible for regulation of cell migration natively. While there have been several reports of photo-activated Rho proteins, no strategy has been develo

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32

Di, Ciano-Oliveira Caterina. "Signaling pathways linking osmotic stress to adaptive responses: Roles for Rho family GTPases." 2006. http://link.library.utoronto.ca/eir/EIRdetail.cfm?Resources__ID=449940&T=F.

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33

Zhou, Hua. "Modulation of squamous carcinoma cell motility by RhoA and Cdc42-PAK1 signaling /." 2004. http://wwwlib.umi.com/dissertations/fullcit/3149705.

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34

Dietz, Christian [Verfasser]. "Analyses of small Rho-GTPases signaling molecules during vertebrate development and angiogenesis / vorgelegt von Christian Tobias Dietz." 2011. http://d-nb.info/1011266997/34.

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35

Du, Jing. "Unbiased Expression Profiling Identifies a Novel Notch Signaling Target RND1 as Regulator of Angiogenesis." Thesis, 2019. https://doi.org/10.7916/d8-pmrg-g821.

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Notch signaling controls normal and pathological angiogenesis through transcriptional regulation of a wide network of target genes. Despite intensive studies of the endothelial Notch function, a comprehensive list of Notch-regulated genes, especially direct transcriptional targets, has not been assembled in endothelial cells (ECs). Here we uncovered novel EC Notch targets that are rapidly regulated by Notch signaling using several unbiased in vivo and in vitro screening approaches that captured genes regulated within 6 hours or less of Notch signal activation. We used a gamma-secretase inhibit

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36

Beane, Wendy Scott. "Building Gene Regulatory Networks in Development: Deploying Small GTPases." Diss., 2007. http://hdl.handle.net/10161/96.

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37

Saha, Shekar. "Functional Study of Nonmuscle Myosin II-C2 in Neuro-2a Cell Line." Thesis, 2019. http://hdl.handle.net/10821/8237.

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Nonmuscle myosin IIs (NM IIs) are ubiquitously expressed throughout the entire organism and play distinct roles in cell division, adhesion, migration etc. Till date, three types of nonmuscle myosin IIs, II-A, II-B and II-C are found in vertebrates. Recently, similar to NM II-B, it has been shown that splicing at loop 1 and loop 2 of NM heavy chain II-C can produce four different isoforms of NM II-C- NM II-C0, NM II-C1, NM II-C2 and NM II-C1C2. C2 insert containing isoforms are specifically expressed in mouse and human brain, and their activities are independent of myosin light chain phosphoryl

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38

Kuo, Jia-Yu, and 郭佳瑜. "Mechanistic study of lovastatin modulating Rho GTPase signaling in astrocytes." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/3y59hb.

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碩士<br>國立陽明大學<br>神經科學研究所<br>97<br>Statin is the inhibitor of HMG-CoA reductase, the rate-limiting enzyme in cholesterol biosynthesis. It is commonly used to treat patients suffering from hypercholesterolemia. Recent studies have demonstrated that statin also has some effects on nervous system. Previous studies from this laboratory already found that the combined treatment of lovastatin and β-cyclodextrin induced the process outgrowth and actin cytoskeleton reorganization, namely decreased actin rings and actin filaments in hippocampal astrocytes. Nonetheless, it showed no similar effect on hipp

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39

Justa-Schuch, Daniela. "Regulation of septum formation by RHO4 GTPase signalling in Neurospora crassa." Doctoral thesis, 2010. http://hdl.handle.net/11858/00-1735-0000-0006-ADB3-9.

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Vogt, Nico. "Governing fungal polar cell extension: Analysis of Rho GTPase and NDR kinase signalling in Neurospora crassa." Doctoral thesis, 2008. http://hdl.handle.net/11858/00-1735-0000-000D-F161-9.

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Sterk, Carolin Christin. "The role of Rho5 in oxidative stress response and glucose signalling in Saccharomyces cerevisiae." Doctoral thesis, 2021. https://repositorium.ub.uni-osnabrueck.de/handle/urn:nbn:de:gbv:700-202106034890.

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Rho-GTPases are essential signalling proteins which regulate a multitude of central cellular processes that are vital for organisms to thrive and adapt to changing environments. Many regulatory networks involving Rho proteins have first been elucidated in the model yeast Saccharomyces cerevisiae, in which Rho5 emerges as a central hub connecting different signalling pathways, such as the responses to cell wall stress, high medium osmolarity, and oxidative stress. In this work, the rapid translocation of Rho5 to mitochondria as reaction to oxidants and glucose starvation was thoroughly investig

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Vogt, Nico [Verfasser]. "Governing fungal polar cell extension : analysis of Rho GTPase and NDR kinase signalling in Neurospora crassa / vorgelegt von Nico Vogt." 2008. http://d-nb.info/993187846/34.

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Justa-Schuch, Daniela [Verfasser]. "Regulation of septum formation by RHO4 GTPase signalling in Neurospora crassa / vorgelegt von Daniela Justa-Schuch." 2010. http://d-nb.info/1005299900/34.

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44

Schilling, Kerstin [Verfasser]. "The role of PIX Rho GTPase exchange factors in signal pathways of the immune system : analysis of signaling capacities of αPIX-/- and αPIX-/- βPIX+/- lymphocytes ; generation of a βPIX conditional knockout / vorgelegt von Kerstin Schilling". 2008. http://d-nb.info/1000868648/34.

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