Relevant bibliographies by topics / Retinal neurones

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Retinal neurones'

Author: Grafiati
Published: 4 June 2021
Last updated: 4 February 2022

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Journal articles on the topic "Retinal neurones"

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Straznicky, C., and M. Chehade. "The formation of the area centralis of the retinal ganglion cell layer in the chick." Development 100, no. 3 (July 1, 1987): 411–20. http://dx.doi.org/10.1242/dev.100.3.411.

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In adult domestic chickens, the neurones in the retinal ganglion cell layer are very unevenly disposed such that there is a sixfold increase in neurone density from the retinal edge to the retinal centre. The formation of the high ganglion-cell-density area centralis was studied on chick retinal wholemounts from the 8th day of incubation (E8) to 4 weeks after hatching (4WAH). The density of viable neurones and the number and the distribution of pyknotic neurones in the ganglion cell layer were estimated across the whole retina. Between E8 and E10, the distribution of neurones in the ganglion cell layer was anisodensitic with 53,000 mm-2 in the centre compared to 34,000 mm-2 in the periphery of the retina. Thereafter, a progressively steeper gradient of neurone density developed, which decreased from 24,000 mm-2 in the retinal centre to 6000 mm-2 at the retinal periphery by 4WAH. Neuronal pyknosis in the ganglion cell layer was observed between E9 and E17. From E11 onwards, consistently more pyknotic neurones were found in the peripheral than in the central retina. It was estimated that over the period of cell death approximately twice as many neurones died per unit area in the retinal periphery than in the centre. Retinal area measurements and estimation of neurone densities in the ganglion cell layer after the period of neurone generation and neurone death indicated differential retinal expansion, with more expansion in the peripheral than in the central retina. These observations allow us to conclude that the formation of the area centralis of the chick retina involves (1) slightly higher cell generation in the retinal centre, (2) higher rate of cell loss in the retinal periphery and (3) differential retinal expansion.
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BINNS, K. E., and T. E. SALT. "The functional influence of nicotinic cholinergic receptors on the visual responses of neurones in the superficial superior colliculus." Visual Neuroscience 17, no. 2 (March 2000): 283–89. http://dx.doi.org/10.1017/s0952523800172116.

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In the rat, the superficial gray layer (SGS) of the superior colliculus receives glutamatergic projections from the contralateral retina and from the visual cortex. A few fibers from the ipsilateral retina also directly innervate the SGS, but most of the ipsilateral visual input is provided by cholinergic afferents from the opposing parabigeminal nucleus (PBG). Thus, visual input carried by cholinergic afferents may have a functional influence on the responses of SGS neurones. When single neuronal extracellular recording and iontophoretic drug application were employed to examine this possibility, cholinergic agonists were found to depress responses to visual stimulation. Lobeline and 1-acetyl-4-methylpiperazine both depressed visually evoked activity and had a tendency to reduce the background firing rate of the neurones. Carbachol depressed the visual responses without any significant effect on the ongoing activity, while the muscarinic receptor selective agonist methacholine increased the background activity of the neurones and reduced their visual responses. Lobeline was chosen for further studies on the role of nicotinic receptors in SGS. Given that nicotinic receptors are associated with retinal terminals in SGS, and that the activation of presynaptic nicotinic receptors normally facilitates transmitter release (in this case glutamate release), the depressant effects of nicotinic agonists are intriguing. However, many retinal afferents contact inhibitory neurones in SGS; thus it is possible that the increase in glutamate release in turn facilitates the liberation of GABA which goes on to inhibit the visual responses. We therefore attempted to reverse the effects of lobeline with GABA receptor antagonists. The depressant effects of lobeline on the visual response could not be reversed by the GABAA antagonist bicuculline, but the GABAB antagonist CGP 35348 reduced the effects of lobeline. We hypothesize that cholinergic drive from the parabigeminal nucleus may activate presynaptic nicotinic receptors on retinal terminals, thereby facilitating the release of glutamate onto inhibitory neurones. Consequently GABA is released, activating GABAB receptors, and thus the ultimate effect of nicotinic receptor activation is to depress visual responses.
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Galli-Resta, Lucia, Elena Novelli, and Alessandro Viegi. "Dynamic microtubule-dependent interactions position homotypic neurones in regular monolayered arrays during retinal development." Development 129, no. 16 (August 15, 2002): 3803–14. http://dx.doi.org/10.1242/dev.129.16.3803.

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In the vertebrate retina cell layers support serial processing, while monolayered arrays of homotypic neurones tile each layer to allow parallel processing. How neurones form layers and arrays is still largely unknown. We show that monolayered retinal arrays are dynamic structures based on dendritic interactions between the array cells. The analysis of three developing retinal arrays shows that these become regular as a net of dendritic processes links neighbouring array cells. Molecular or pharmacological perturbations of microtubules within dendrites lead to a stereotyped and reversible disruption of array organization: array cells lose their regular spacing and the arrangement in a monolayer. This leads to a micro-mechanical explanation of how monolayers of regularly spaced ‘like-cells’ are formed.
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Gonzalez-Hoyuela, M., J. A. Barbas, and A. Rodriguez-Tebar. "The autoregulation of retinal ganglion cell number." Development 128, no. 1 (January 1, 2001): 117–24. http://dx.doi.org/10.1242/dev.128.1.117.

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The development of the nervous system is dependent on a complex set of signals whose precise co-ordination ensures that the correct number of neurones are generated. This regulation is achieved through a variety of cues that influence both the generation and the maintenance of neurones during development. We show that in the chick embryo, stratified retinal ganglion cells (RGCs) are themselves responsible for providing the signals that control the number of RGCs that are generated, both by inhibiting the generation of new ganglion cells and by killing incoming migratory ganglion cells. Selective toxicological ablation of RGCs in the chick embryo resulted in the achronic generation of ganglion cells, which eventually led to the repopulation of the ganglion cell layer and a large decrease in the physiological cell death affecting postmitotic migratory neurones. Interestingly, the application of exogenous NGF reversed the effects of ganglion cell ablation on ganglion cell death. Because the only source of NGF in the retina is that produced by the stratified ganglion cells, we infer that these differentiated neurones regulate their own cell number by secreting NGF, a neurotrophin that has previously been shown to be responsible for the death of migrating ganglion cells.
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Andressen, Christian, and Jürgen K. Mai. "Localization of the CD15 carbohydrate epitope in the vertebrate retina." Visual Neuroscience 14, no. 2 (March 1997): 253–62. http://dx.doi.org/10.1017/s0952523800011391.

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AbstractThe distribution of the carbohydrate epitope CD 15, a putative cell adhesion molecule, was studied in adult vertebrate retinas by light-microscopic immunohistochemistry. Except for Old World primates, in which no immunoreactivity was detectable, all other species expressed the epitope on retinal interneurones. Subpopulations of stratified amacrine cells were found in all species with the exception of bats and marmoset monkeys, and bipolar cells were immunoreactive in frogs and all amniotic animals. Ganglion cells were labelled in urodelian, in all sauromorphian, as well as in some mammalian species. In some species, the distribution of immunoreactive neurones was correlated to areas of retinal specialization such as the visual streak in frogs and the dorsotemporal field in birds. In these parts of the retina with enhanced visual acuity, more CD 15 glycosylated bipolar cells were found than in other parts. Among mammals, labelled bipolar cells were found exclusively in species with cone-dominated retinas. This comparative study shows that CD 15 expression is consistently membrane associated in morphologically defined subsets of amacrine, bipolar, and ganglion cells throughout the vertebrate phylum. Its distribution pattern was found to depend more on the visual behavior of a given species (cone-dominated or rod-dominated retina) than on phylogenetic proximity between species.
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Gaucher, David, Emilie Arnault, Zoé Husson, Nicolas Froger, Elisabeth Dubus, Pauline Gondouin, Diane Dherbécourt, et al. "Taurine deficiency damages retinal neurones: cone photoreceptors and retinal ganglion cells." Amino Acids 43, no. 5 (April 4, 2012): 1979–93. http://dx.doi.org/10.1007/s00726-012-1273-3.

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Frade, J. M. "Unscheduled re-entry into the cell cycle induced by NGF precedes cell death in nascent retinal neurones." Journal of Cell Science 113, no. 7 (April 1, 2000): 1139–48. http://dx.doi.org/10.1242/jcs.113.7.1139.

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During their early postmitotic life, a proportion of the nascent retinal ganglion cells (RGCs) are induced to die as a result of the interaction of nerve growth factor (NGF) with the neurotrophin receptor p75. To analyse the mechanisms by which NGF promotes apoptosis, an in vitro culture system consisting of dissociated E5 retinal cells was established. In this system, NGF-induced apoptosis was only observed in the presence of insulin and neurotrophin-3, conditions that favour the birth of RGCs and other neurones expressing the glycoprotein G4. The pro-apoptotic effect of NGF on the G4-positive neurones was evident after 10 hours in vitro and was preceded by a significant upregulation of cyclin B2, but not cyclin D1, and the presence of mitotic nuclei in these cells. Brain-derived neurotrophic factor prevented both the increase of cyclin B2 expression in the G4-positive neurones and the NGF-induced cell death. Finally, pharmacologically blocking cell-cycle progression using the cyclin-dependent kinase inhibitor roscovitine prevented NGF-induced cell death in a dose-dependent manner. These results strongly suggest that the apoptotic signalling initiated by NGF requires a driving stimulus manifested by the neuronal birth and is preceded by the unscheduled re-entry of postmitotic neurones into the cell cycle.
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Hussain, S. T., and E. A. Baydoun. "Cytochemical localization of 5'-nucleotidase in the frog (Rana pipiens) retina. A histochemical and cytochemical study." Journal of Histochemistry & Cytochemistry 33, no. 10 (October 1985): 1067–72. http://dx.doi.org/10.1177/33.10.2995482.

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Localization of 5'-nucleotidase in the frog retina was investigated using histochemical and cytochemical techniques. Light-microscopic observations revealed the presence of this enzyme in the inner retinal layers (the nerve fiber layer, ganglion cell layer, and inner plexiform layer). Ultrastructural investigations revealed that the enzyme activity is associated with the plasma membranes of the Müller cell processes, whereas the Müller cell processes present in the outer retinal layers did not demonstrate any detectable enzyme activity. This observation would appear to confirm our previous findings, that 5'-nucleotidase is an ectoenzyme, but its distribution in frog retina differs from that in rodents and it is only present in the inner layers of the retina. The prominent localization of 5'-nucleotidase on the glial plasma membrane may be viewed in the context of the widely accepted interaction between neurones and glial cells. Since nucleotides do not penetrate the plasma membrane, a mechanism to produce membrane-permeable adenosine, important for neuronal function, is postulated. It is known that 5'-nucleotidase produces adenosine by hydrolyzing adenosine 5'-monophosphate (5'-AMP). Therefore one would expect that the glial membrane-bound enzyme can accomplish the final step in this mechanism by producing the adenosine in the extracellular spaces.
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Uchiyama, Hiroyuki, Hironobu Ito, and Masaki Tauchi. "Retinal neurones specific for centrifugal modulation of vision." NeuroReport 6, no. 6 (April 1995): 889–92. http://dx.doi.org/10.1097/00001756-199504190-00016.

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Sparks, D. L. "The neural encoding of the location of targets for saccadic eye movements." Journal of Experimental Biology 146, no. 1 (September 1, 1989): 195–207. http://dx.doi.org/10.1242/jeb.146.1.195.

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Current models of the saccadic system imply that there are at least three neural representations of a visual target to which a saccade is made: representations in retinal, spatial (head or body) and motor coordinates. This paper presents the evidence supporting these models and summarizes the available neurophysiological data concerning neural representations of target location. In the superior colliculus, neurones in the superficial layers encode target location in retinal coordinates. Neurones in the deeper layers responsive to auditory and visual stimuli carry motor error signals. Evidence is also accumulating that some neurones in the thalamus and the frontal and parietal cortex convey information about target position with respect to the head or body, but these studies are far from complete.
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Dissertations / Theses on the topic "Retinal neurones"

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Ugarte, Marta. "Influence of zinc on the normal retina and the retina given an insult of ischaemia : in vitro and in vivo studies." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325776.

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Rheey, Jinguen. "Otx2 promotes survival of injured adult retinal ganglion cells non cell-autonomously and regulates development of inner retinal cells in post-natal mouse cell autonomously." Paris 6, 2011. http://www.theses.fr/2011PA066176.

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Bec, Jean-Michel. "Etude de la stimulation laser de neurones pour des applications de prothèses visuelles." Thesis, Montpellier 2, 2010. http://www.theses.fr/2010MON20029/document.

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Ce travail se situe dans le cadre d'un projet pluridisciplinaire visant à développer une prothèse visuelle. La technique la plus utilisée actuellement dans de nombreux types de neuroprothèses est basée sur l'excitation par voie électrique via des électrdes. Les inconvénients d'une telle technique (très invasive, de faible résolution spatiale et par contact) pourraient être surmontés en utilisant une stimulation par laser infra-rouge. Nous présentons dans un premier temps les caractéristiques des trois diodes lasers fibrés émettant à 1875 nm, 1535 nm et 1470 nm pour des gammes de puissances optiques de quelques centaines de mW qui ont été utilisés et intégrés à deux dispositifs de mesures permettant l'observations de variations d'échanges ioniques transmembranaires (imagerie de fluorescence des ions calciums et mesure électrophysiologique par la technique de patch clamp). Nous montrons ensuite que des réponses biologiques ont été obtenues par les trois lasers, non seulement sur des cellules ganglionnaires de la rétine et du vestibule de culture mais aussi sur des tranches de rétine. L'influence des paramètres clés comme la longueur d'onde, la durée de stimulation, les seuils d'énergie a été étudié, et a permis d'établir que les seuils d'énergie de stimulation dépendent de la valeur du coefficient d'absorption de l'eau qui varie suivant la longueur d'onde utilisée. Enfin, une étude est consacrée pour expliquer les mécanismes physiques et biologiques apparaissant au cours de l'interaction du laser avec le neurone au niveau cellulaire. Des simulations numériques quantifiant l'élévation de température associées à des tests pharmacologiques cherchant à déterminer la nature des canaux ioniques spécifiques mis en jeu suggèrent la prédominance d'un effet thermique
This work is part of a pluridisciplinary project, aiming at developing a visual prosthesis. The most used technique for this kind of neuroprosthesis is based on the electrical stimulation of nerves by electrodes. Drawbacks of such a technique (very intrusive, low spatial resolution and physical contact) could be overcome by the use of an infra red laser based stimulation. We present first the three fibre pigtailed laser diode characteristics emitting few hundred of mW at 1875 nm, 1535 nm and 1470 nm. These lasers have been integrated on two measurement devices (a fluorescence microscope and a microscope using patch clamp recording), for the observation of ionic membrane exchanges. Our results show that action potentials have been obtained by laser stimulation from the three lasers, both on retinal or vestibular ganglion cells from mass cultures and on retinal slices. The effect of key parameters as the wavelength, the stimulation time, the energy thresholds has been studied and show that the energy thresholds clearly depend on the absorption coefficient of water which varies with the wavelength. Finally, we present the results of a preliminary study aiming at determining the biophysical interaction mechanisms at cell level. Numerical simulations giving the local increase of temperature and tests of specific blocking molecules in order to know the exact nature of the ionic channels involved suggest a predominant thermal mechanism
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Stanke, Jennifer J. "Beyond Neuronal Replacement: Embryonic Retinal Cells Protect Mature Retinal Neurons." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1250820277.

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Icha, Jaroslav. "Ganglion cell translocation across the retina and its importance for retinal lamination." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-218914.

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Correct layering (lamination) of neurons in the central nervous system (CNS) is critical for the tissue functionality. Neuronal lamination is established during development, when the majority of neurons have to move from their birthplace to the appropriate layer, where they function. Therefore, to grasp the logic of CNS development, it is essential to understand the kinetics and modes of the variety of neuronal translocation events. Most of our knowledge about neuronal translocation has been gained using fixed tissue or ex vivo imaging, which is not ideal for such a dynamic process heavily dependent on the surrounding environment. To avoid these limitations, I combined translucent zebrafish embryos with light sheet fluorescence microscopy, which together enabled gentle in toto imaging of neuronal translocation. I studied the translocation of retinal ganglion cells (RGCs) across the developing zebrafish retina. RGCs are the first neurons that differentiate in the vertebrate retina and are born in a proliferative zone at the retinal apical side. From here, they move basally, spanning the complete apico-basal length of the tissue. They are destined to occupy the most basal layer, where their axons form the optic nerve. Although it was described that RGCs move their soma while being attached to both apical and basal sides of the retina, the kinetics and cell biological mechanisms of somal translocation remained unknown. Extracting single cell behavior of RGCs from high-resolution movies of their translocation allowed for quantitative analysis of RGC movement. I revealed that RGCs cross the retina in less than two hours in a directionally persistent manner. The movement of RGC soma is a cell autonomously generated process, which requires intact microtubules and actin-dependent basal attachment of cells for speed and efficiency. Unexpectedly, interference with somal translocation leads to a shift towards a multipolar migratory mode, previously not observed for RGCs, in which they temporarily lose both apical and basal attachment and apico-basal polarity. The multipolar mode is overall slower and less directionally persistent, but still allows RGCs to reach the basal retina. However, when RGC translocation is inhibited completely, they differentiate ectopically in the center of the retina, which in turn triggers the formation of ectopic layers of later born neurons. These results highlight the importance of establishing the basal layer of ganglion cells for ensuing retinal lamination. Overall, I generated important advances in the understanding of neuronal translocation and lamination, which might be relevant for other parts of the CNS.
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Atkinson, Joana. "Manipulation of retinal neuronal outgrowth." Thesis, University of Cambridge, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311308.

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Schneider, Nicole [Verfasser]. "Glutamate transporters in retinal neurons / Nicole Schneider." Hannover : Bibliothek der Tierärztlichen Hochschule Hannover, 2013. http://d-nb.info/1037880625/34.

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Holt, M. G. "Exocytosis and endocytosis in a retinal neurone." Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604197.

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The synaptic vesicle cycle in the nerve terminal consists of exocytosis and neurotransmitter release, endocytosis and vesicle regeneration. However, the exact mechanisms which underlie this cycle are still largely unknown. This dissertation describes the use of fluorescence microscopy to study aspects of vesicle cycling in the synaptic terminal of retinal depolarizing bipolar cells isolated from goldfish retina. Endocytosis at the synapse may proceed via one of two pathways: through the direct reformation of small vesicles, or through the formation of large cisternae. However, the mechanisms responsible for forming these larger compartments are unclear. In chapter 3, it is shown that following exocytosis membrane is retrieved via small vesicles and large vacuoles in bipolar cells. Vacuoles were heterogeneous in size and their formation was dependent on P1 3-kinase and F-actin, whereas formation of small synaptic vesicles was not. Vacuoles were also transported away from the plasma membrane by an actin-dependent mechanism, stimulated by calcium influx. Bulk membrane retrieval in the bipolar cell therefore exhibits the properties of macropinocytosis observed in non-neuronal cells. The bipolar cell can maintain high levels of neurotransmitter release over periods of many minutes, in response to sustained stimulation. The bipolar cell also contains high amounts of PKCα. A role for this enzyme in continuous exocytosis was investigated and the results presented in chapter 4. To maintain continuous exocytosis required an elevated free Ca2+ level in the synaptic terminal of approximately 1 μM. Inhibition of PKC led to a reduced Ca2+ level during stimulation, blocking exocytosis. In addition, PKCα may also have other roles in the exocytic process, such as modulating the mobility of synaptic vesicles or regulating the sensitivity of the exocytic machinery to Ca2+. In conclusion to the chapter, experiments are described which could help distinguish between these possible functions of PKCα in the bipolar cell. The mobility of synaptic vesicles is likely to play an important role in the regulation of synaptic transmission. This is especially true in the bipolar cell, because of its ability to support continuous exocytosis.
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Wali, Naheed. "Three-dimensional reconstruction of mudpuppy retinal neurons /." The Ohio State University, 1988. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487595712160115.

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Sardo, Giacomo. "Caratteristiche morfologiche della retina nei Cetacei." Master's thesis, Alma Mater Studiorum - Università di Bologna, 2015. http://amslaurea.unibo.it/8130/.

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Le caratteristiche strutturali dell’occhio dei Cetacei sono state in passato oggetto di studio. Tuttavia, i dati relativi alla stratigrafia della retina ed alle caratteristiche morfologiche dei neuroni gangliari in essa presenti sono piuttosto ridotti; per questo motivo, l’obiettivo della presente ricerca è stato quello di studiare, mediante metodiche di immunoistochimica, l’uso della microscopia ottica e di opportuni software di analisi immagine, le caratteristiche morfologiche della retina e delle cellule gangliari in essa presenti in differenti specie di Cetacei. Per la presente ricerca sono stati utilizzate come specie di riferimento i seguenti Delfinidi: tursiope (Tursiops truncatus) e stenella striata (Stenella coeruleoalba). Le analisi sulle sezioni interessano l’area, la densità dei neuroni gangliari, la stratigrafia della retina e l’analisi morfometrica degli strati e dei neuroni. I risultati ottenuti indicano come la retina del tursiope e della stenella striata, nonostante un'organizzazione di base assai simile a quella degli altri Mammiferi, mostri caratteristiche qualitative sue proprie. Gli strati retinici sono quelli che si osservano in tutti i Mammiferi e lo spessore totale della retina è, nel tursiope (101,23 µm ) e nella stenella striata (108.35 µm ), pressochè simile ai Mammiferi terrestri (110-220 µm). Nell'ambito della retina, lo strato che presento lo spesso medio maggiore è quello dei granuli interni (SNE); tale dato non coincide con quanto osservato in altri Mammiferi. I neuroni gangliari presenti nella retina di tursiope e stenella striata mostrano, analogamente a quanto osservato in altri Cetacei, una bassa densità cellulare. Nel tursiope e nella stenella striata le aree a maggiore densità cellulare presentano neuroni multipolari di dimensioni minori rispetto a quelle con bassa densità. Questo dato potrebbe indicare una "cellularità" (quantità di superficie occupata da cellule) costante nei differenti distretti retinici. I neuroni gangliari presenti nella retina di tursiope e stenella striata sono disposti in un unico strato, come osservato in numerosi altri Cetacei, ma differisce da quanto osservato nel capodoglio (Physeter macrocephalus) dove tali cellule si dispongono in strati multipli. Neuroni gangliari di grandi dimensioni sono stati osservati sia nel tursiope che nella stenella striata. Tale dato coincide con quanto osservato in altri Odontoceti ed in alcuni Misticeti. Allo stato attuale non è ancora stato dato un chiaro significato funzionale alle cellule gangliari giganti. Un possibile ruolo potrebbe essere quello di condurre, in animali di grossa mole, l'impulso nervoso molto velocemente, grazie alla presenza di un assone provvisto di un diametro notevole. Tale interpretazione non è da tutti accettata in quanto Mammiferi terrestri di grandi dimensioni non presentano nella loro retina neuroni gangliari giganti.
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Books on the topic "Retinal neurones"

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Anderson, James A. The Brain Doesn’t Work by Logic. Oxford University Press, 2018. http://dx.doi.org/10.1093/acprof:oso/9780199357789.003.0008.

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This chapter gives three examples of real neural computation. The conclusion is that the “brain doesn’t work by logic.” First, is the Limulus (horseshoe crab) lateral eye. The neural process of “lateral inhibition” tunes the neural response of the compound eye to allow crabs to better see other crabs for mating. Second, the retina of the frog contains cells that are selective to specific properties of the visual image. The frog responds strongly to the moving image of a bug with one class of selective retinal receptors. Third, experiments on patients undergoing neurosurgery for epilepsy found single neurons in several cortical areas that were highly selective to differing images, text strings, and spoken names of well-known people. In addition, new selective responses could be formed quickly. The connection to concepts in cognitive science seems inevitable. One possible mechanism is through associatively linked “cell assemblies.”
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The Retinal Muller Cell: Structure & Function (Perspectives in Vision Research). Springer, 2001.

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G, Leventhal Audie, ed. The Neural basis of visual function. Boca Raton: CRC Press, 1991.

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Saalmann, Yuri B., and Sabine Kastner. Neural Mechanisms of Spatial Attention in the Visual Thalamus. Edited by Anna C. (Kia) Nobre and Sabine Kastner. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199675111.013.013.

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Neural mechanisms of selective attention route behaviourally relevant information through brain networks for detailed processing. These attention mechanisms are classically viewed as being solely implemented in the cortex, relegating the thalamus to a passive relay of sensory information. However, this passive view of the thalamus is being revised in light of recent studies supporting an important role for the thalamus in selective attention. Evidence suggests that the first-order thalamic nucleus, the lateral geniculate nucleus, regulates the visual information transmitted from the retina to visual cortex, while the higher-order thalamic nucleus, the pulvinar, regulates information transmission between visual cortical areas, according to attentional demands. This chapter discusses how modulation of thalamic responses, switching the response mode of thalamic neurons, and changes in neural synchrony across thalamo-cortical networks contribute to selective attention.
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A, Levin Leonard, and Di Polo Adriana, eds. Ocular neuroprotection. New York: Marcel Dekker, 2003.

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H, Yu Albert C., ed. Neuronal-astrocytic interactions: Implications for normal and pathological CNS function. Amsterdam: Elsevier, 1992.

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(Editor), Dominic Man-Kit Lam, and Garth M. Bray (Editor), eds. Regeneration and Plasticity in the Mammalian Visual System: Proceedings of the Retina Research Foundation Symposia, Volume Four (Bradford Books). The MIT Press, 1992.

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Protein kinase C and its brain substrates: Role in neuronal growth and plasticity. Amsterdam: Elsevier Science, 1991.

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Hertz, Leif, Albert C. H. Yu, and Michael D. Norenberg. Neuronal Astrocytic Interactions: Implications for Normal and Pathological Cns Function (Progress in Brain Research). Elsevier Publishing Company, 1992.

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1938-, Sharma S. C., and Fawcett James W. 1950-, eds. Formation and regeneration of nerve connections. Boston: Birkhäuser, 1993.

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Book chapters on the topic "Retinal neurones"

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Djamgoz, Mustafa B. A., and Masahiro Yamada. "Electrophysiological characteristics of retinal neurones: synaptic interactions and functional outputs." In The Visual System of Fish, 159–210. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0411-8_6.

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Guo, Tianruo, David Tsai, Siwei Bai, Mohit Shivdasani, Madhuvanthi Muralidharan, Liming Li, Socrates Dokos, and Nigel H. Lovell. "Insights from Computational Modelling: Selective Stimulation of Retinal Ganglion Cells." In Brain and Human Body Modeling 2020, 233–47. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-45623-8_13.

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AbstractImprovements to the efficacy of retinal neuroprostheses can be achieved by developing more sophisticated neural stimulation strategies to enable selective or differential activation of specific retinal ganglion cells (RGCs). Recent retinal studies have demonstrated the ability to differentially recruit ON and OFF RGCs – the two major information pathways of the retina – using high-frequency electrical stimulation (HFS). However, there remain many unknowns, since this is a relatively unexplored field. For example, can we achieve ON/OFF selectivity over a wide range of stimulus frequencies and amplitudes? Furthermore, existing demonstrations of HFS efficacy in retinal prostheses have been based on epiretinal placement of electrodes. Other clinically popular techniques include subretinal or suprachoroidal placement, where electrodes are located at the photoreceptor layer or in the suprachoroidal space, respectively, and these locations are quite distant from the RGC layer. Would HFS-based differential activation work from these locations? In this chapter, we conducted in silico investigations to explore the generalizability of HFS to differentially active ON and OFF RGCs. Computational models are particularly well suited for these investigations. The electric field can be accurately described by mathematical formulations, and simulated neurons can be “probed” at resolutions well beyond those achievable by today’s state-of-the-art experimental techniques.
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Wilson, Martin. "Synaptic Transmission Between Retinal Neurons." In Development and Organization of the Retina, 227–43. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5333-5_12.

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Forkwa, Tembei K., Ernst R. Tamm, and Andreas Ohlmann. "Ambiguous Role of Glucocorticoids on Survival of Retinal Neurons." In Retinal Degenerative Diseases, 365–71. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-3209-8_46.

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Kato, S., Z. Y. Zhou, K. Sugawara, Y. Yasui, N. Takizawa, K. Sugitani, and K. Mawatari. "Ischemic Neuronal Death in the Fish Retina." In Degenerative Retinal Diseases, 199–204. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5933-7_23.

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Barber, Alistair J., Heather D. Van Guilder, and Matthew J. Gastinger. "The Neuronal Influence on Retinal Vascular Pathology." In Retinal Vascular Disease, 108–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-29542-6_6.

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Yao, Xin-Cheng, and Yi-Chao Li. "Functional Imaging of Retinal Photoreceptors and Inner Neurons Using Stimulus-Evoked Intrinsic Optical Signals." In Retinal Development, 277–85. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-848-1_20.

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Dong, Shuqian, Yan Liu, Meili Zhu, Xueliang Xu, and Yun-Zheng Le. "Simplified System to Investigate Alteration of Retinal Neurons in Diabetes." In Retinal Degenerative Diseases, 139–43. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-3209-8_18.

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Stenkamp, Deborah L., and Ruben Adler. "Biological Effects of Retinoids and Retinoid Metabolism in Cultures of Chick Embryo Retina Neurons and Photoreceptors." In Retinal Degeneration, 355–60. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2974-3_35.

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Puthussery, Theresa, and W. Rowland Taylor. "Functional Changes in Inner Retinal Neurons in Animal Models of Photoreceptor Degeneration." In Retinal Degenerative Diseases, 525–32. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-1399-9_60.

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Conference papers on the topic "Retinal neurones"

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Zhu, Liang, and Robert Flower. "Role of Vasomotion in Control of Retina Edema in Diabetic Retinopathy: Quantification of Fluid Transport Through Retinal Capillaries." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-189507.

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Diabetic retinopathy refers to diabetes-related complications in the retina, It is a progressive disease and its symptoms in the eyes can vary from non-vision threatening to vision loss, and it can lead to permanent damage to the neuronal retinal tissue. The irreversible nature of the damage suggests that prevention of diabetes by eliminating risk factors and early screening are the cornerstone of relevant treatment to stop or limit visual damage in those patients.
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Robertson, Joshua, Ewan Wade, and Antonio Hurtado. "Ultrafast Emulation of Retinal Neuronal Circuits with Artificial VCSEL Optical Neurons." In 2019 IEEE Photonics Conference (IPC). IEEE, 2019. http://dx.doi.org/10.1109/ipcon.2019.8908359.

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Nasiatka, Patrick J., Michelle C. Hauer, Noelle R. B. Stiles, Jaw-Chyng Lue, Satsuki Takahashi, Rajat Agrawal, James D. Weiland, Mark S. Humayun, and Armand R. Tanguay. "An Intraocular Camera for Retinal Prostheses." In ASME 2007 2nd Frontiers in Biomedical Devices Conference. ASMEDC, 2007. http://dx.doi.org/10.1115/biomed2007-38109.

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Blindness due to degenerative retinal diseases such as Retinitis Pigmentosa (RP) and Age-Related Macular Degeneration (AMD) afflict millions of people worldwide. Recent advances in retinal implants that bypass damaged photoreceptor cells and electrically stimulate the remaining healthy retinal neurons show promise for restoring functional vision to the blind [1]. Current intraocular retinal prostheses driven by an external camera mounted on the subject’s head require slow and unnatural head movements. To allow for normal foveation and expanded depth of field, a novel intraocular camera (IOC) has been designed to work in conjunction with an epiretinal microstimulator array, as shown schematically in Fig. 1.
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Zhang, Ying-Ying, Xue Liu, Hai-Qing Gong, and Pei-Ji Liang. "Efficient information processing in retinal neurons." In 2009 IEEE International Conference on Virtual Environments, Human-Computer Interfaces and Measurements Systems (VECIMS). IEEE, 2009. http://dx.doi.org/10.1109/vecims.2009.5068884.

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Bardin, Fabrice, Jean-Michel Bec, Emmanuelle S. Albert, Christian Hamel, Gérard Dupeyron, Christian Chabbert, Isabelle Marc, and Michel Dumas. "Infrared laser stimulation of retinal and vestibular neurons." In SPIE BiOS. SPIE, 2011. http://dx.doi.org/10.1117/12.874260.

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Zhao, Xueqing, Junjun Zhang, Xin Shi, Tao Xue, and Kaixuan Liu. "RNCIR: Retinal neuron coding-based image recognition." In 14th International FLINS Conference (FLINS 2020). WORLD SCIENTIFIC, 2020. http://dx.doi.org/10.1142/9789811223334_0130.

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Schmid, Erich W., Wolfgang Fink, and Robert Wilke. "Electric stimulation of neurons and neural networks in retinal prostheses." In 2013 6th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2013. http://dx.doi.org/10.1109/ner.2013.6696131.

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Vicente, Xaymara S. "Development of Neuronal Classes in Mouse Retina." In Minority Trainee Research Forum, 2004. TheScientificWorld Ltd, 2004. http://dx.doi.org/10.1100/tsw.2004.172.

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Fried, Shelley I., Changsi Cai, and Qiushi Ren. "High frequency electric stimulation of retinal neurons elicits physiological signaling patterns." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6090251.

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Jong Yoon Shin, Jae-Hyun Ahn, Kilhwa Pi, Dong-il Dan Cho, and Yong Sook Goo. "Electrodeless, non-invasive stimulation of retinal neurons using time-varying magnetic fields." In 2015 IEEE Sensors. IEEE, 2015. http://dx.doi.org/10.1109/icsens.2015.7370211.

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You might also be interested in the extended bibliographies on the topic 'Retinal neurones' for particular source types:
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