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Academic literature on the topic 'Respiratory Insufficiency In adulthood'

Author: Grafiati
Published: 10 December 2022
Last updated: 30 January 2023

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Journal articles on the topic "Respiratory Insufficiency In adulthood"

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Goyal, N., M. Waldrop, and T. Mozaffar. "Unique myopathy presenting in adulthood with proximal muscle weakness and respiratory insufficiency." Neuromuscular Disorders 25 (October 2015): S282. http://dx.doi.org/10.1016/j.nmd.2015.06.346.

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El-Maouche, Diala, Courtney J. Hargreaves, Ninet Sinaii, Ashwini Mallappa, Padmasree Veeraraghavan, and Deborah P. Merke. "Longitudinal Assessment of Illnesses, Stress Dosing, and Illness Sequelae in Patients With Congenital Adrenal Hyperplasia." Journal of Clinical Endocrinology & Metabolism 103, no. 6 (March 22, 2018): 2336–45. http://dx.doi.org/10.1210/jc.2018-00208.

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Abstract Context Patients with congenital adrenal hyperplasia (CAH) are at risk for life-threatening adrenal crises. Management of illness episodes aims to prevent adrenal crises. Objective We evaluated rates of illnesses and associated factors in patients with CAH followed prospectively and receiving repeated glucocorticoid stress dosing education. Methods Longitudinal analysis of 156 patients with CAH followed at the National Institutes of Health Clinical Center over 23 years was performed. The rates of illnesses and stress-dose days, emergency room (ER) visits, hospitalizations, and adrenal crises were analyzed in relation to phenotype, age, sex, treatment, and hormonal evaluations. Results A total of 2298 visits were evaluated. Patients were followed for 9.3 ± 6.0 years. During childhood, there were more illness episodes and stress dosing than adulthood (P < 0.001); however, more ER visits and hospitalizations occurred during adulthood (P ≤ 0.03). The most robust predictors of stress dosing were young age, low hydrocortisone and high fludrocortisone dose during childhood, and female sex during adulthood. Gastrointestinal and upper respiratory tract infections (URIs) were the two most common precipitating events for adrenal crises and hospitalizations across all ages. Adrenal crisis with probable hypoglycemia occurred in 11 pediatric patients (ages 1.1 to 11.3 years). Undetectable epinephrine was associated with ER visits during childhood (P = 0.03) and illness episodes during adulthood (P = 0.03). Conclusions Repeated stress-related glucocorticoid dosing teaching is essential, but revised age-appropriate guidelines for the management of infectious illnesses are needed for patients with adrenal insufficiency that aim to reduce adrenal crises and prevent hypoglycemia, particularly in children.
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Magnani, Jessie E., and Steven M. Donn. "Persistent Respiratory Distress in the Term Neonate: Genetic Surfactant Deficiency Diseases." Current Pediatric Reviews 16, no. 1 (April 9, 2020): 17–25. http://dx.doi.org/10.2174/1573396315666190723112916.

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: Respiratory distress is one of the most common clinical presentations in newborns requiring admission to a Neonatal Intensive Care Unit (NICU). Many of these infants develop respiratory distress secondary to surfactant deficiency, which causes an interstitial lung disease that can occur in both preterm and term infants. Pulmonary surfactant is a protein and lipid mixture made by type II alveolar cells, which reduces alveolar surface tension and prevents atelectasis. : The etiology of surfactant deficiency in preterm infants is pulmonary immaturity and inadequate production. Term infants may develop respiratory insufficiency secondary to inadequate surfactant, either from exposure to factors that delay surfactant synthesis (such as maternal diabetes) or from dysfunctional surfactant arising from a genetic mutation. : The genetics of surfactant deficiencies are very complex. Some mutations are lethal in the neonatal period, while others cause a wide range of illness severity from infancy to adulthood. Genes that have been implicated in surfactant deficiency include SFTPA1, SFTPA2, SFTPB, SFTPC, and SFTPD (which encode for surfactant proteins A, B, C, and D, respectively); ABCA3 (crucial for surfactant packaging and secretion); and NKX2 (a transcription factor that regulates the expression of the surfactant proteins in lung tissue). : This article discusses the interplay between the genotypes and phenotypes of newborns with surfactant deficiency to assist clinicians in determining which patients warrant a genetic evaluation.
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Southam, Brendan R., Adam P. Schumaier, and Alvin H. Crawford. "Spondylocostal Dysostosis: A Literature Review and Case Report with Long-Term Follow-Up of a Conservatively Managed Patient." Case Reports in Orthopedics 2018 (2018): 1–6. http://dx.doi.org/10.1155/2018/1795083.

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Introduction. Patients with spondylocostal dysostosis (SCD) have congenital spine and rib deformities associated with frequently severe thoracic insufficiency and respiratory compromise. The literature is largely composed of case reports and small cohorts, and there is little information regarding adults with this condition. In this report, we describe the natural history of a conservatively treated patient and include quality-of-life issues such as childbearing, athletic participation, and occupational selection. Case Presentation. We present a patient with SCD who was conservatively treated by a single physician from birth for 31 years. Our patient was capable of a reasonably good quality of life through adulthood, including participation in gymnastics and employment. At age 18, she became pregnant and subsequently terminated the pregnancy due to obstetrical concerns for compromised respiration. She has had intermittent respiratory complaints and occasionally experiences dyspnea with exertion, but this only has limited her during certain activities in the past three years. Currently, she takes naproxen for chronic back pain with periodic exacerbations. Discussion. Other cases in the literature have described adult SCD patients who have received nonoperative treatment and achieved a wide range of functional outcomes. This provides some limited evidence to suggest that select patients with SCD may be treated conservatively and achieve a reasonable quality of life. However, close clinical follow-up with these patients is recommended, particularly early on, considering the high rates of infant morbidity and mortality. Chest physiotherapy and early pulmonary care have been associated with favorable outcomes in infancy. Surgery to increase thoracic volume and correct scoliosis has been shown in some cases to improve respiratory function. Treatment depends on the degree of thoracic insufficiency and quality of life. The natural history of SCD remains largely unknown, but some patients are capable of relatively favorable life spans, employment, and participation in athletics.
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Seliukova, N. Yu. "Long-Term Consequences of the Fetoplacental Insufficiency Influence on the State and Functioning of Different Body Systems in Descendants (Literature Review and Own Research)." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 5, no. 6 (December 12, 2020): 362–69. http://dx.doi.org/10.26693/jmbs05.06.362.

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The literature review presents the results of processing literature data on one of the topical issues of obstetrics and reproductive medicine - placental insufficiency. According to the World Health Organization, the number of infertile couples is gradually increasing in the world every year. In Ukraine, the frequency of infertile marriages among persons of reproductive age ranges from 12 to 18%. The state of the reproductive system of an adult female is influenced by many factors, from embryonic development to the lifestyle of an adult woman. With an inadequate effect of harmful factors on the fetus, the fetoplacental complex may not react properly, and this is how the symptom complex of disorders arises both on the part of the mother and on the part of the fetus, which is called placental insufficiency, it negatively affects the condition and quality of pregnancy and childbirth. Numerous studies have proven the existence of a connection between the effect of certain factors during pregnancy on the state and functionality of various body systems already in adulthood. It was shown that children who were born from mothers with placental insufficiency suffering from cardiovascular diseases have fewer elastic fibers in the arteries, nephrons in the kidneys. In addition, the pancreas has fewer insulin-producing β-cells and reduced vascularization, as well as altered structure and maturation of the brain, lungs (respiratory distress syndrome) and liver, joint dysplasia, and an imbalance of the immune system. When studying the long-term effects of placental insufficiency, various disorders of physical and mental development are observed, as well as increased somatic and infectious morbidity of newborns and children in the first year of life, in the future it may be the cause of the development of diseases such as arterial hypertension, diabetes mellitus, metabolic syndrome, etc. Conclusion. Our studies have shown the negative impact of placental insufficiency on the reproductive system of adult offspring of rats of two sexes who were born to mothers of two age groups. In particular, the level of testosterone in female offspring was increased against the background of the altered structure of the estrous cycle and the pathological state of ovarian histology. In the male offspring, on the contrary, the total testosterone level decreased, but the histology of the testes and the functional state of the sperm remained unchanged
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Padoan, Rita, Serena Quattrucci, Annalisa Amato, Vincenzo Carnovale, Donatello Salvatore, Marco Salvatore, and Giuseppe Campagna. "The Diagnosis of Cystic Fibrosis in Adult Age. Data from the Italian Registry." Diagnostics 11, no. 2 (February 16, 2021): 321. http://dx.doi.org/10.3390/diagnostics11020321.

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Cystic Fibrosis (CF) registries are an essential resource of epidemiological and clinical data. Although the median age at diagnosis is usually reported in the first months of life, a minority of individuals is diagnosed during adulthood. The aim of this study was to describe demographic, genetic, and clinical characteristics of this subgroup of the Italian CF population by using data from the Italian CF Registry (ICFR). Patients ≥18 years at diagnosis were selected and clinical data at diagnosis were analyzed from the 2012–2018 ICFR data (Cohort A). Subjects with diagnosis ≥18 years were selected from 2018 ICFR dataset (Cohort B) to describe their clinical status. In 2012–18 the incidence of late diagnosis was 18.2%, whereas, in 2018, the prevalence of patients diagnosed ≥18 years was 12.54%. The median age of late diagnosis was 36.2 years, ranging from 19.0 to 68.3. The male patients were diagnosed because of infertility in the 45.9% of cases. Median sweat chloride value (SCL) was 69 mmol/L (range 9–150). F508del mutation accounted for 28.3% of alleles. A wide variability in respiratory function was present with a median percent predicted Forced Expiratory Volume in the first second (ppFEV1) of 90.8% (range 20–147%). Low prevalence of pancreatic insufficiency (25%) and of Pseudomonas aeruginosa (Pa) infection (17%) suggest a mild CF phenotype in the majority of patients. The assessment of the clinical status in the 2018 dataset and the comparison between genders showed a greater nutritional and respiratory impairment in females. Further studies are needed to clarify the importance of a true diagnostic delay or of late onset of CF symptoms.
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Delaporta, Polyxeni, Christalena Sofocleous, Stavros Doudounakis, Marina Economou, Emmanouil Kanavakis, and Antonios Kattamis. "Variable Clinical Phenotype and Course In Greek Patients with Shwachman-Diamond Syndrome Carrying Identical SBDS Mutations." Blood 116, no. 21 (November 19, 2010): 4431. http://dx.doi.org/10.1182/blood.v116.21.4431.4431.

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Abstract Abstract 4431 Introduction-Background: Shwachman-Diamond syndrome (SDS) is a rare multi-system genetic disorder mainly characterized by exocrine pancreatic insufficiency, bone marrow failure and skeletal abnormalities. Approximately 90% of patients meeting the clinical diagnostic criteria for SDS have mutations in the SBDS gene, located in chromosome 7q11. No genotype-phenotype correlations have been observed in patients with SDS. Patients and Method: Greek patients with similar SBDS mutations are included in this report. They were selected from the series of patients referred to our unit for SBDS gene molecular analysis due to pancreatic insufficiency and impaired hematopoiesis. Patient 1 presented at birth with respiratory difficulties, hypotonia, anemia, neutropenia and thrombocytopenia. She has congenital anomalies including thoracic dystrophy, digit abnormalities, open foramen ovale and hypertelorism. She was found to have myelodysplasia with a bone marrow clone carrying i(7q) chromosomal abnormality in around 32% of the bone marrow cells. Pancreatic insufficiency was clinically evident even at the age of 5 months. Patient 2 has chronic thrombocytopenia ranging between 40.000/μ L to 147.000/μ L, first presenting at the age of 7 years old. She has metaphyseal dysostosis, flared anterior end of ribs, open foramen ovale and growth hormone deficiency. Her pancreatic insufficiency is present from the age of 16 months. Patient 3 (sister of patient 2) has borderline neutropenia, short stature, metaphyseal dysostosis, open foramen ovale and mild pancreatic insufficiency. Patient 4 has thrombocytopenia since the age of 19 years old. She has stable clonal erythropoesis with a clone carrying the 46,X,del(X)(q24→qter) in 45% of the bone marrow cells. She presents recurrent bacterial infections, particularly bartholinitis. She has mild pancreatic insufficiency. Patient 5 presented with chronic neutropenia and decreaced IgA since the age of 2 months. At the age of 12 months she presented hepatomegaly and elevated liver enzymes. Pancreatic insufficiency initially presented in infancy but improved gradually. Genomic DNA was extracted from peripheral blood lymphocytes and molecular analysis with ECMA (Enzymatic Cleavage Mismatch Analysis), RFLPS and direct sequencing was performed allowing detection and characterization of disease causing mutations. PCR primers were specifically designed to amplify the whole coding region (five exons) and the flanking intron/exon junctions of SBDS gene but not the SBDSP pseudogene. RFLPs used the Bsu36I and AciI enzymes for the detection of the two most common c.183-184 TA>CT and 258+2 T>C mutations respectively. Result: All five patients were compound heterozygotes for 183–184 TA>CT and 258+2 T>C, which are the two most common mutations of SDS. One of those (patient 3) was found to be a mosaic which seems to explain the very mild phenotype, and another (patient 5) presented homozygosity for the 258+2 T>C while carrying the 183–184 TA>CT mutation as well. Patient 1 was successfully transplanted by her HLA-identical sister at the age of 12 months. Her pancreatic insufficiency has not improved and she is still on pancreatic enzyme supplementation. Patient 2 is receiving pancreatic enzyme supplementation and also is currently on growth hormone supplementation. Patients 3 and 4 are not receiving pancreatic enzyme supplementation or granulocyte colony-stimulating factor. Patient 5 is currently receiving only granulocyte colony-stimulating factor. Conclusion: Extreme variability ranging from severe clinical phenotype apparent at birth to close-to- normal phenotype in early adulthood was noted in this small series of Greek patients, carrying similar SBDS mutations. Moreover, gene conversion seems to be a frequent event in the SBDS gene. Further studies to evaluate the heterogeneity and the factors affecting the phenotype/genotype relationship in SDS are warranted. Disclosures: No relevant conflicts of interest to declare.
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Cocanougher, Benjamin T., Lauren Flynn, Pomi Yun, Minal Jain, Melissa Waite, Ruhi Vasavada, Jason D. Wittenbach, et al. "Adult MTM1-related myopathy carriers." Neurology 93, no. 16 (September 20, 2019): e1535-e1542. http://dx.doi.org/10.1212/wnl.0000000000008316.

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ObjectiveTo better characterize adult myotubularin 1 (MTM1)–related myopathy carriers and recommend a phenotypic classification.MethodsThis cohort study was performed at the NIH Clinical Center. Participants were required to carry a confirmed MTM1 mutation and were recruited via the Congenital Muscle Disease International Registry (n = 8), a traveling local clinic of the Neuromuscular and Neurogenetic Disorders of Childhood Section, National Institute of Neurological Disorders and Stroke, NIH and Cure CMD (n = 1), and direct physician referral (n = 1). Neuromuscular examinations, muscle MRI, dynamic breathing MRI, cardiac MRI, pulmonary function tests (PFTs), physical therapy assessments including the Motor Function Measure 32 (MFM-32) scale, and X chromosome inactivation (XCI) studies were performed.ResultsPhenotypic categories were proposed based on ambulatory status and muscle weakness. Carriers were categorized as severe (nonambulatory; n = 1), moderate (minimal independent ambulation/assisted ambulation; n = 3), mild (independent ambulation but with evidence of muscle weakness; n = 4), and nonmanifesting (no evidence of muscle weakness; n = 2). Carriers with more severe muscle weakness exhibited greater degrees of respiratory insufficiency and abnormal signal on muscle imaging. Skeletal asymmetries were evident in both manifesting and nonmanifesting carriers. Skewed XCI did not explain phenotypic severity.ConclusionThis work illustrates the phenotypic range of MTM1-related myopathy carriers in adulthood and recommends a phenotypic classification. This classification, defined by ambulatory status and muscle weakness, is supported by muscle MRI, PFT, and MFM-32 scale composite score findings, which may serve as markers of disease progression and outcome measures in future gene therapy or other clinical trials.
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Bogdan, Raluca Daniela, Roxana Elena Bohiltea, and Adrian Ioan Toma. "Respiratory Follow Up of the Premature Neonates—Rationale and Practical Issues." Journal of Clinical Medicine 11, no. 6 (March 21, 2022): 1746. http://dx.doi.org/10.3390/jcm11061746.

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The aim of the review was to present the state of knowledge about the respiratory pathology in former premature neonates (children that were born preterm—before 37 weeks of gestation—and are examined and evaluated after 40 weeks corrected age) other than chronic lung disease, in order to provide reasons for a respiratory follow-up program for this category of patients. After a search of the current evidence, we found that premature infants are prone to long-term respiratory consequences due to several reasons: development of the lung outside of the uterus, leading to dysmaturation of the structures, pulmonary pathology due to immaturity, infectious agents or mechanical ventilation and deficient control of breathing. The medium- to long-term respiratory consequences of being born before term are represented by an increased risk of respiratory infections (especially viral) during the first years of life, a risk of recurrent wheezing and asthma and a decrease in pulmonary volumes and airway flows. Late preterm infants have risks of pulmonary long-term consequences similar to other former premature infants. Due to all the above risks, premature neonates should be followed in an organized fashion, being examined at regular time intervals from discharge from the maternity hospital until adulthood—this could lead to an early detection of the risks and preventive therapies in order to improve their prognosis and assure a normal and productive life. The difficulties related to establishing such programs are represented by the insufficient standardization of the data gathering forms, clinical examinations and lung function tests, but it is our belief that if more premature infants are followed, the experience will allow standards to be established in these fields and the methods of data gathering and evaluation to be unified.
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Levytskyi, A. F., V. O. Rogozinskyi, M. M. Dolianytskyi, and L. V. Duda. "Halo-gravity traction in the treatment of complex spinal deformities in children with respiratory dysfunctions." Paediatric Surgery. Ukraine, no. 3(72) (September 29, 2021): 10–14. http://dx.doi.org/10.15574/ps.2021.72.10.

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HGT is a safe technique as the world literature describes complications in the form of loosening of the pins or superficial infections of the skin around the pins, which are not significant and do not pose a threat to the patient’s life. Purpose – to improve the results of the ventilation function of the lungs in patients with complex spinal deformities through the preliminary use of halo gravity traction and to introduce an effective and safe method for the treatment of complex spinal deformities in children with respiratory dysfunctions. Materials and methods. 64 children with complex spinal deformities (>100°) were treated in the orthopedic and traumatology department of the Okhmatdet NSPU using halo gravity traction during the period from 2003 until 2018. Of these, 38 are boys and 26 are girls. The average age of the patients was 11.6 years. The average Risser score was 3.8 (P>0.01). Results. According to the data of spirography performed, 46% of patients had moderate ventilation disorders and 54% – severe ventilation disorders (FVC<60% – grade 3 and 4 of ventilation failure). Mixed type disorders were recorded in 83% of patients, and restrictive type disorders in 17% (8/48) of children. After HGT, there was an improvement in pulmonary function indicators: an increase in FVC from 63.19% to 71.77% and FEV1 from 54.71% to 65.46%, Tiffeneau-Pinelli index – from 74.59% to 85.33%. Compared with the initial level of indicators, the improvement in FVC was 13.6% after HGT and 14.6% in dynamics during the year, and FEV1 – 19.6% and 21.6%, respectively. The results obtained indicate a significant improvement in the ventilation function of the lungs, especially due to the degree of FEV1 increase, which correlates with the degree of improvement in performance, mortality and life prognosis. Conclusions. The use of HGT makes it possible to improve the results of the final correction of spinal deformity, which in turn significantly improves the ventilation function of the lungs, which in turn helps to reduce the risks of mortality due to pulmonary insufficiency in adulthood. The choice of the appropriate methods of surgical correction for complex deformity of the spine is a prerequisite for successful treatment and the achievement of three-dimensional correction of the spine to maximally approximate its parameters to the physiological norm. The indication for halo gravity traction is a rigid scoliotic deformity of the spine with a deformity angle (>100°). This study was conducted in accordance with the principles of the Helsinki Declaration. The research protocol was approved by the Local Ethics Committee of the institutions mentioned in the work. Informed parental agreement was obtained for the research. No conflict of interests was declared by the authors. Key words: spinal deformity, respiratory dysfunctions, halo-gravity traction.
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Dissertations / Theses on the topic "Respiratory Insufficiency In adulthood"

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Borrego, Luís Miguel Nabais. "Crianças com sibilância recorrente: estudo de função respiratória, avaliação imunológica e polimorfismos genéticos." Doctoral thesis, Faculdade de Ciências Médicas. Universidade Nova de Lisboa, 2008. http://hdl.handle.net/10362/5149.

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RESUMO Nos últimos anos têm sido identificados vários factores de risco para asma brônquica em crianças com sibilância recorrente, não se encontrando clara a importância da avaliação funcional respiratória nestas crianças. De igual modo, têm sido documentados resultados contraditórios na avaliação imunológica das populações de células reguladoras bem como na expressão de polimorfismos para a asma. O objectivo deste estudo consistiu na avaliação e comparação de parâmetros de avaliação funcional respiratória, imunológica e de polimorfismos genéticos em crianças entre 8 e 20 meses de idade, com três ou mais episódios de sibilância (n=50), sem qualquer terapêutica anti-inflamatória prévia, diagnosticados por um médico, com e sem factores de risco para asma brônquica (história de asma parental ou história pessoal de eczema ou pelo menos dois dos seguintes: história pessoal de rinite alérgica, sibilância fora do contexto infeccioso e contagem de eosinófilos no sangue periférico > 4%), comparados com um grupo controlo (n=30). Nestas crianças foram efectuadas provas de função respiratória em volume corrente e em volume aumentado através de técnicas de compressão torácica, avaliação de populações celulares por citometria de fluxo, expressão de citocinas por mARN em culturas de células estimuladas com PMA (leitura às 24 horas) e com extractos de ácaros do pó doméstico (leitura ao 7º dia) e expressão de polimorfismos para alguns genes associados a asma (ADAM 33, DPP10, GPRA). Na comparação entre as crianças com sibilância recorrente em relação ao grupo controlo foram observadas reduções significativas nos Z-scores para FVC (diferença média [95% IC]: -0,7 [-1,2; -0,1], p=0,01), FEV0.5 (-1,0 [-1,5; -0,5], p=0,0001), FEF75 (-0,6 [-1,0; -0,2], p=0,0001) e FEF25-75 (-0,8 [-1,2; -0,4], p=0,0001), bem como valores significativamente mais baixos para a quantificação do número absoluto de CD4+CD25forte (-47,9 [-89,6; -6,1], p=0,03), do número absoluto e percentual de CD4+CD25+CTLA-4 (p=0,0001) e da expressão de CTLA-4 (p=0,03) e IFN-􀁊 (p=0,04) nas culturas com extractos de ácaros. As crianças sibilantes com alto risco para asma tinham, em relação ao grupo sem factores de risco, Z-scores significativamente mais baixos para FVC (-0,7 [-1,4; -0,04], p=0,04) e FEF25-75 (-0,6 [-1,2; -0,1], p=0,03),2 valores significativamente inferiores do número absoluto das populações CD4+CD25+ (-118,8 [-210,0; -27,5], p=0,01) e CD4+CD25forte (-56,2 [-109,9; -2,5], p=0,04) e ainda uma expressão diminuída de IFN-􀁊 (p=0,03) em culturas de células estimuladas com extractos de ácaros. Foram encontradas diferenças na expressão de polimorfismos para os genes GPRA e ADAM 33, não sendo possível tecer extrapolações pelo reduzido número de crianças em estudo. As crianças com sibilância recorrente e alto risco de asma apresentavam alterações na avaliação funcional respiratória, bem como no número absoluto de populações celulares com função reguladora e na expressão de IFN-􀁊 em culturas celulares estimuladas com extractos de ácaros. Estes resultados realçam a eventual importância da avaliação das provas de função respiratória e de parâmetros imunológicos, em crianças com sibilância recorrente e alto risco clínico para asma, nos primeiros dois anos de vida, apesar da sua controversa aplicabilidade individual. O seguimento prospectivo destas crianças poderá aferir o seu valor preditivo para asma em idade escolar.
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San, Angelo Donna. "THE EFFECT OF BODY POSITION ON RESPIRATORY FUNCTION IN THE INFANT WITH RESPIRATORY DISTRESS SYNDROME." Thesis, The University of Arizona, 1986. http://hdl.handle.net/10150/275429.

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Aikio, O. (Outi). "Pulmonary nitric oxide in preterm and term infants with respiratory failure." Doctoral thesis, University of Oulu, 2002. http://urn.fi/urn:isbn:9514268512.

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Abstract The aim of the study was to evaluate pulmonary endogenous and inhaled nitric oxide (NO) in neonates with severe respiratory failure. Infant autopsy documents were reviewed for fulminant early-onset bacterial pneumonia. 12 infants with the onset at < 72 h of age and three control groups were identified. Immunohistochemistry revealed that 11 of the infants with early-onset pneumonia (92%) had no or faint inducible nitric oxide synthase (NOS2) staining in their alveolar macrophages (AM). All control infants, regardless of their postnatal age, had NOS2-positive AM. The marker of NO-toxicity, nitrotyrosine, was low in all specimens. To confirm this finding, airway specimens of 21 newborns requiring mechanical ventilation were examined. Seven of them had fulminant early-onset pneumonia with maternal ascending intra-uterine infection (IUI). The controls had no infection at birth despite IUI or neither infection nor IUI. In early-onset pneumonia, NOS2 and nitrotyrosine immunoreactivity were low at birth and increased during the recovery phase (p < 0.05). Analyses of interleukin-1 and surfactant protein A showed the same pattern of age-dependent change. Of the autopsied infants, 12 had received inhaled NO (iNO) before death. Each case was paired with a matched control. Additional five infants without respiratory failure prior to death were also studied. The iNO-treated ones tended to have more intensive NOS2 staining in the bronchiolar epithelium and adjacent tissue than the controls. No differences in other NOS isoforms or nitrotyrosine were detected. A novel method for exhaled NO measurements of intubated infants was developed. Six preterm and six term newborns were prospectively recruited for expired and nasal NO measurements. During the first week of life, the preterm infants showed a different pattern of exhaled NO excretion compared to the term infants. For the pilot intervention study on very early iNO, the eligible patients had a birth weight < 1500 g and progressive, therapy-resistant respiratory failure before five hours of age. Five infants received iNO, showed immediately improved oxygenation and survived without deleterious side effects. Deficient production of NO in small premature infants is associated with severe infection and respiratory failure. Very early iNO therapy may be exceptionally effective in a select group of infants, and did not appear to cause oxidation lung injury.
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Lidegran, Marika. "Advanced radiological imaging in patients treated with extracorporeal membrane oxygenation /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-933-5/.

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[UNESP], Daniela Fernandes Lima. "Fatores de predição de mortalidade em pacientes com insuficiência respiratória crônica em uso de oxigenoterapia domiciliar prolongada." Universidade Estadual Paulista (UNESP), 2009. http://hdl.handle.net/11449/92162.

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Introdução: A fase avançada de pneumopatias, cardiopatias e doenças sistêmicas está associada com o desenvolvimento de insuficiência respiratória crônica (IRespC). O tratamento desta complicação inclui o uso de oxigenoterapia domiciliar prolongada. Na literatura nacional poucos estudos avaliaram a sobrevida em pacientes com doença pulmonar obstrutiva crônica (DPOC) ou com IRespC recebendo oxigenoterapia domiciliar prolongada (ODP). Objetivo: O objetivo deste estudo foi avaliar os fatores de predição de mortalidade em pacientes com IRespC tratados com ODP no período de um, dois e três anos. Pacientes e Métodos: Foram avaliados e acompanhados pelo período mínimo de três anos ou até o óbito, 142 pacientes cadastrados no ambulatório de oxigenoterapia da Faculdade de Medicina de Botucatu (FMB) até julho de 2005. Na avaliação inicial, foram coletados dados de identificação pessoal, história de tabagismo, o uso de medicação, composição corporal, força de preensão manual, função pulmonar, gases sanguíneos e hemograma. Também foram aplicados a escala de dispnéia de Borg, o índice de dispnéia basal (BDI) e o questionário de qualidade de vida na doença respiratória do Hospital Saint George (SGRQ). Resultados: Durante os três anos de estudo; 83 pacientes (58%) morreram: 37 (26%) durante o primeiro ano de acompanhamento, 23 (16%) no segundo ano e 23 (16%) no período de três anos. O grupo óbito apresentou valores de hematócrito e hemoglobina significativamente menores, maior sensação de dispnéia, avaliada pelo BDI e Borg, e apresentou maior 18 comprometimento do estado de saúde, avaliado pelos domínios, impacto, atividade e total do SGRQ. Os principais preditores de mortalidade, na avaliação que incluía todos os pacientes, no período de três anos foram: gênero masculino (HR=2,67, CI=1,15-6,18, p=0,02), menores valores de hemoglobina...
Introduction: The advanced stage of lung, heart and systemic diseases is associated with the development of chronic respiratory failure (CRF). The treatment of this complication includes the use of long term oxygen therapy (LTOT). In the national literature few studies have assessed the survival of patients with chronic obstructive pulmonary disease (COPD) or with chronic respiratory failure treated with LTOT. Aim: The aim of this study was to evaluate the predictors of mortality in patients with CRF treated with oxygen in the period of one, two and three years. Patients and Methods: One hundred forty two patients were evaluated and followed for the minimum period of three years or until death. The study group consisted of patients with CRF, clinically stable, seen at the Oxygen Therapy Outpatient Clinic of Botucatu School of Medicine Hospital. Information about the demographic characteristics, smoking history, comorbidity conditions and treatment were collected during the first visit; evaluation of body composition, handgrip strength, lung function and blood gases and blood counts were also undertaken at this time. In addition, the dyspnea sensation (basal dyspnea index - BDI - and Borg scale) and health related quality of life (Saint George Respiratory Questionnaire) scores were calculated. Results: During the follow-up, 83 (58%) patients died; 37 (26%) during the first year of follow-up, 23 (16%) in the second year and 23 (16%) considering in three years. The group of death showed significantly lower 20 hematocrit and hemoglobin, increased sensation of breathlessness, measured by BDI and Borg, and showed greater impairment of health status as measured by impact, activity and total score of SGRQ. The main predictors of mortality, after three years of follow-up were male gender (HR=2.67, CI=1.15-6.18, p=0.02), lower values of hemoglobin (HR=0.85, CI=0.74-0.98, p=0.02), and PaCO2... (Complete abstract click electronic access below)
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Hinze, Candace. "The role of malnutrition in prolonged respiratory failure : the effect of accelerated nutritional rehabilitation." Thesis, McGill University, 1995. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=22740.

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To investigate the possibility that malnutrition is an important factor that prolongs respiratory failure (PRF), I studied the effects of pharmacologic injections of recombinant human growth hormone (rhGH), an important anabolic stimulus, on nutritional and respiratory parameters in patients requiring mechanical ventilation for more than three days. Patients were excluded from consideration if dominating factors known to prolong ventilatory failure had not been stabilized. Over ten months, 106 patients in PRF were evaluated, but only six met the selection criteria. Three patients were randomized to receive standard nutritional support, and three into a group that received the equivalent nutrition plus 5 mg/day of rhGH for 14 days or until withdrawal of mechanical ventilation. Baseline characteristics of the selected patients were divergent as demonstrated by body mass indexes ranging from 14 to 42 (kg/m$ sp2),$ baseline maximal inspiratory pressures (PI$ sb{ max}$ from $-$15 to $-$70 cm H$ sb2$O, and Day 1 N balances from $-$13.5 to 1.2 g N/day. Despite increased plasma insulin-like growth factor-1 concentrations, the mean daily N balances of the rhGH-treated group were no better than the controls (1.3 $ pm$ 5.0 vs. 0.4 $ pm$ 2.6 g N/day; Mean $ pm$ SD), nor were there differences in PI$ sb{ max},$ level of ventilatory assistance required, and days to weaning. The persistence of respiratory failure in the overwhelming majority of patients in PRF appears to be due to factors already known to prevent weaning from mechanical ventilation. Even the carefully selected patients enrolled in the present study were insufficiently homogeneous or stable enough to allow proper testing of the experimental hypothesis.
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Pugmire, Juliana. "Health Effects of Childhood Exposure to Environmental Tobacco Smoke in Children followed to Adulthood." Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/202985.

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Background A significant proportion of children are exposed to environmental tobacco smoke (ETS) throughout the world. This is mainly because of exposure to parental smoking. It is unknown to what extent the negative effects of ETS on respiratory symptoms track from childhood into adulthood. Methods TESAOD (Tucson Epidemiologic Study of Airway Obstructive Disease) is a large population-based prospective study that was initiated in 1972. Participants were followed prospectively with questionnaires and pulmonary function tests (PFTs) completed about every two years in 12 follow-up surveys up to 1996. Skin prick tests and blood samples for IgE measurements were collected at surveys 1, 6, and 11. We identified subjects who entered the study as children (<15 years old) and were followed to adulthood (>18 years) during the study follow-up. Based on questionnaire data, active asthma, wheeze, cough, and chronic cough (cough for three consecutive months) were coded as never (never reported in childhood or adulthood), incident (never reported in childhood, but ≥ one positive report in adulthood), remittent (≥ one positive report in childhood, but not in adulthood), and persistent (≥ one positive report both in childhood and adulthood). PFTs measurements included forced expiratory volume in 1 second, forced vital capacity, and forced expiratory flow at 25-75%. Parent information on smoking status was collected simultaneously at child visits. ETS exposure status was assessed as “ever” or “never” between birth and 15 years. Results Information on parental ETS exposure in childhood and outcomes in adulthood was available for 444 non-Hispanic white participants (51.4% male) with mean age at initial survey of 7.7 years. Total mean follow-up time was 19.0 years (8.8 years in adulthood). Between birth and 15 years, 53.4% of children were exposed to ETS. After adjusting for sex, age at enrollment, years of follow-up, and personal smoking status (assessed at age 15 and above), combined parental ETS exposure in childhood was significantly associated with persistent wheeze (RR(adj) 1.9, p=0.026), persistent cough (RR(adj) 5.9, p<0.001), and persistent (RR(adj) 3.7, p=0.030) and incident chronic cough (RR(adj) 2.3, p=0.040). Paternal ETS exposure in childhood was associated with persistent wheeze (RR(adj) 2.3, p=0.002), persistent cough (RR(adj) 3.9, p<0.001), persistent (RR(adj) 4.8, p=0.004) and incident chronic cough (RR(adj) 2.2, p=0.031), and persistent asthma (RR(adj) 2.3, p=0.016). Maternal ETS exposure was associated with persistent (RR(adj) 1.9, p=0.029) and incident cough (RR(adj) 2.5, p=0.006). Maternal ETS exposure was associated with an increased percent predicted FVC in adulthood (coefficient, 3.75; p=0.019). No other effects on lung function were seen. There were no effects of ETS exposure on total serum IgE or allergic sensitization. ETS exposure was associated with respiratory symptoms in adulthood among both never and current smokers. Conclusions ETS exposure in childhood has long term health effects on lung function and respiratory symptoms. These effects do not appear to be IgE-mediated. ETS exposure, especially paternal ETS exposure, seems to influence the persistence of respiratory symptoms from childhood to adulthood and to affect women more than men. These effects are independent of personal smoking and also seen in never smokers. Both smoking mothers and fathers should be targeted when attempting to reduce ETS exposure among children.
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Boyarskiy, O. O., O. O. Tikhonova, G. O. Solomennyk, and O. I. Mohylenets. "Features of acute respiratory viral infections in patients with congenital syndrome of immune-endocrine failure." Thesis, Sumy State University, 2016. http://essuir.sumdu.edu.ua/handle/123456789/47814.

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Congenital syndrome of immune-endocrine insufficiency (CSIEI) is the type of constitution that is morphologically characterized by primary hyperplasia of the thymus, generalized hyperplasia of lymphoid tissue, adrenal hypoplasia in combination with various anomalies of cardiovascular, and less frequently of urogenital systems. Features of CSIEI in a functional sense are the failure of infectious and immune violations of adaptation under stress. Endocrine failure, which occurs in patients with CSIEI, usually go unnoticed in the outpatient setting and in the ICU appears addisonian crisis with hypovolemia and shock, which are resistant to treatment. Objective: to develop criteria for lifetime diagnosis and prevention CSIEI.
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Lima, Daniela Fernandes. "Fatores de predição de mortalidade em pacientes com insuficiência respiratória crônica em uso de oxigenoterapia domiciliar prolongada /." Botucatu, 2009. http://hdl.handle.net/11449/92162.

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Orientador: Irma de Godoy
Banca: Suzana Erico Tanni
Banca: Maria Christina L. Machao
Resumo: Introdução: A fase avançada de pneumopatias, cardiopatias e doenças sistêmicas está associada com o desenvolvimento de insuficiência respiratória crônica (IRespC). O tratamento desta complicação inclui o uso de oxigenoterapia domiciliar prolongada. Na literatura nacional poucos estudos avaliaram a sobrevida em pacientes com doença pulmonar obstrutiva crônica (DPOC) ou com IRespC recebendo oxigenoterapia domiciliar prolongada (ODP). Objetivo: O objetivo deste estudo foi avaliar os fatores de predição de mortalidade em pacientes com IRespC tratados com ODP no período de um, dois e três anos. Pacientes e Métodos: Foram avaliados e acompanhados pelo período mínimo de três anos ou até o óbito, 142 pacientes cadastrados no ambulatório de oxigenoterapia da Faculdade de Medicina de Botucatu (FMB) até julho de 2005. Na avaliação inicial, foram coletados dados de identificação pessoal, história de tabagismo, o uso de medicação, composição corporal, força de preensão manual, função pulmonar, gases sanguíneos e hemograma. Também foram aplicados a escala de dispnéia de Borg, o índice de dispnéia basal (BDI) e o questionário de qualidade de vida na doença respiratória do Hospital Saint George (SGRQ). Resultados: Durante os três anos de estudo; 83 pacientes (58%) morreram: 37 (26%) durante o primeiro ano de acompanhamento, 23 (16%) no segundo ano e 23 (16%) no período de três anos. O grupo óbito apresentou valores de hematócrito e hemoglobina significativamente menores, maior sensação de dispnéia, avaliada pelo BDI e Borg, e apresentou maior 18 comprometimento do estado de saúde, avaliado pelos domínios, impacto, atividade e total do SGRQ. Os principais preditores de mortalidade, na avaliação que incluía todos os pacientes, no período de três anos foram: gênero masculino (HR=2,67, CI=1,15-6,18, p=0,02), menores valores de hemoglobina... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Introduction: The advanced stage of lung, heart and systemic diseases is associated with the development of chronic respiratory failure (CRF). The treatment of this complication includes the use of long term oxygen therapy (LTOT). In the national literature few studies have assessed the survival of patients with chronic obstructive pulmonary disease (COPD) or with chronic respiratory failure treated with LTOT. Aim: The aim of this study was to evaluate the predictors of mortality in patients with CRF treated with oxygen in the period of one, two and three years. Patients and Methods: One hundred forty two patients were evaluated and followed for the minimum period of three years or until death. The study group consisted of patients with CRF, clinically stable, seen at the Oxygen Therapy Outpatient Clinic of Botucatu School of Medicine Hospital. Information about the demographic characteristics, smoking history, comorbidity conditions and treatment were collected during the first visit; evaluation of body composition, handgrip strength, lung function and blood gases and blood counts were also undertaken at this time. In addition, the dyspnea sensation (basal dyspnea index - BDI - and Borg scale) and health related quality of life (Saint George Respiratory Questionnaire) scores were calculated. Results: During the follow-up, 83 (58%) patients died; 37 (26%) during the first year of follow-up, 23 (16%) in the second year and 23 (16%) considering in three years. The group of death showed significantly lower 20 hematocrit and hemoglobin, increased sensation of breathlessness, measured by BDI and Borg, and showed greater impairment of health status as measured by impact, activity and total score of SGRQ. The main predictors of mortality, after three years of follow-up were male gender (HR=2.67, CI=1.15-6.18, p=0.02), lower values of hemoglobin (HR=0.85, CI=0.74-0.98, p=0.02), and PaCO2... (Complete abstract click electronic access below)
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Gaspari, Romolo Joseph. "Pathophysiology of Respiratory Failure Following Acute Organophosphate Poisoning : A Dissertation." eScholarship@UMMS, 2009. https://escholarship.umassmed.edu/gsbs_diss/445.

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Organophosphate (OP) poisoning is a health issue worldwide with over 200,000 deaths per year. Although not a problem in most developed countries, in some third world countries, one third of a hospital’s population could be patients with OP exposure. Even with the most aggressive therapy, 10-40% of patients admitted to an intensive care unit will die. Research into the best practice for treating OP poisoning is lacking, due somewhat to a lack of detailed understanding of the physiology of OP poisoning. Our research uses animal models of acute OP poisoning to explore the mechanism of OP-induced respiratory failure. Our research shows that animals poisoned with dichlorvos demonstrated a uniformly fatal central apnea that, if prevented, was followed immediately by a variable pulmonary dysfunction. Potential mechanisms for dichlorvos-induced central apnea can be divided into direct effects on the central respiratory oscillator (CRO) and feedback inhibition of the CRO. Two afferent pathways that can induce apnea include vagal feedback pathways and feed-forward pathways from the cerebral hemispheres. In our studies we found that vagal feedback and feed forward inhibition from the cerebral hemispheres were not required for OP-induced central apnea. The pre-Botzinger complex in the brainstem is thought to be the kernel of the CRO, but exposure of the pre-Botzinger complex to dichlorvos was not sufficient for apnea. Although OP induced central apnea was uniformly fatal, partial recovery of the CRO occurred post apnea with mechanical ventilation. Central apnea was ubiquitous in our rat poisoning model, but pulmonary dysfunction was extremely variable, with a range of pulmonary effects from fulminate pulmonary failure with prominent pulmonary secretions to no pulmonary dysfunction at all. Vagal efferent activity is involved in neural control of pulmonary tissue but the vagus was not involved in OP-induced pulmonary dysfunction. Anti-muscarinic medications are the mainstay of clinical therapy and are commonly dosed by their effects on pulmonary secretions. Our studies found that atropine (the most common therapeutic agent for OP poisoning) resulted in a ventilation-perfusion mismatch secondary to effects on the pulmonary vasculature.
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Books on the topic "Respiratory Insufficiency In adulthood"

1

Adrogué, Horacio J. Respiratory failure. Cambridge, Mass: Blackwell Science, 1997.

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Disorders of ventilation. Oxford: Blackwell Scientific Publications, 1988.

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International Symposium on Anaesthesia (4th 1989 Erlangen, Germany). New aspects on respiratory failure. Edited by Rügheimer E, Mang Harald, and Tschaikowsky Klaus. Berlin: Springer-Verlag, 1992.

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K, Simonds Anita, ed. Non-invasive respiratory support. London: Chapman & Hall Medical, 1996.

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Parker, James N., and Philip M. Parker. The 2002 official patient's sourcebook on respiratory failure. San Diego, Calif: Icon Health Publications, 2002.

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Neonatal respiratory care handbook. Boston: Jones and Bartlett, 2009.

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Respiratory home care equipment. Dubuque, Iowa: Kendall/Hunt Pub. Co., 1988.

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Respiratory management of ALS: Amyotrophic lateral sclerosis. Sudbury, Mass: Jones and Bartlett, 2009.

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Hall, James R., M.D., ed. Respiratory intensive care of the adult surgical patient. St. Louis: Mosby, 1985.

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Workshop on the Scientific Issues in the Assessment of Respiratory Impairment (1986 Bethesda, Md.). Report of Workshop on the Scientific Issues in the Assessment of Respiratory Impairment. [Washington, D.C.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1987.

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Book chapters on the topic "Respiratory Insufficiency In adulthood"

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Champion, Howard R., Nova L. Panebianco, Jan J. De Waele, Lewis J. Kaplan, Manu L. N. G. Malbrain, Annie L. Slaughter, Walter L. Biffl, et al. "Acute Respiratory Insufficiency." In Encyclopedia of Intensive Care Medicine, 88. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1086.

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Simon, Mathieu. "Acute respiratory insufficiency." In Applied Respiratory Pathophysiology, 55–65. Boca Raton : CRC Press, [2017]: CRC Press, 2017. http://dx.doi.org/10.1201/9781315177052-4.

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Mandalia, Amar, and Matthew J. DiMagno. "Exocrine Pancreatic Insufficiency and Nutritional Complications." In Respiratory Medicine, 261–98. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42382-7_13.

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Carvalho, Angelina C. A., and Warren M. Zapol. "BLOOD ALTERATIONS IN THE ADULT RESPIRATORY DISTRESS SYNDROME." In Acute pulmonary insufficiency, edited by Tom Saldeen, 1–32. Berlin, Boston: De Gruyter, 1985. http://dx.doi.org/10.1515/9783110850123-002.

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Peters, Richard M. "Acute Respiratory Insufficiency and Ventilatory Support." In International Practice in Cardiothoracic Surgery, 60–71. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4259-2_7.

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Estenne, M. "Respiratory Muscle Insufficiency in Neuromuscular Disorders." In Update 1988, 146–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83392-2_20.

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Mennella, Roberta, and Alberto Zangrillo. "Prevention and Treatment of Respiratory Insufficiency." In Thoraco-Abdominal Aorta, 229–42. Milano: Springer Milan, 2011. http://dx.doi.org/10.1007/978-88-470-1857-0_19.

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Saldeen, Tom. "BLOOD COAGULATION AND RELATED SYSTEMS IN THE PATHOGENESIS OF ACUTE RESPIRATORY DISTRESS." In Acute pulmonary insufficiency, edited by Tom Saldeen, 119–42. Berlin, Boston: De Gruyter, 1985. http://dx.doi.org/10.1515/9783110850123-008.

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Seyfried, Thomas N., Jeffrey H. Chuang, Lu Zhang, Xianlin Han, and Michael A. Kiebish. "The Tumor Mitochondrial Lipidome and Respiratory Bioenergetic Insufficiency." In Lipidomics, 297–317. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527655946.ch15.

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Benzer, H., and W. Keller. "Acute Respiratory Insufficiency: New Aspects of Managing Ventilatory Support." In Update in Intensive Care and Emergency Medicine, 95–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70309-6_19.

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Conference papers on the topic "Respiratory Insufficiency In adulthood"

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Magnet, Friederike Sophie, Hannah Bleichroth, Sophie E. Huttmann, Jens Callegari, Sarah B. Schwarz, Claudia Schmoor, Wolfram Windisch, and Jan H. Storre. "Clinical evidence for respiratory pump insufficiency predicts weaning failure." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2333.

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Ferreira, Renato Cordeiro, Dayanne Gomes, Vitor Tamae, Francisco Wernke, and Alfredo Goldman. "SPIRA: Building an Intelligent System for Respiratory Insufficiency Detection." In Workshop Brasileiro de Engenharia de Software Inteligente. Sociedade Brasileira de Computação, 2022. http://dx.doi.org/10.5753/ise.2022.227048.

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Respiratory insufficiency is a medic symptom in which a person gets a reduced amount of oxygen in the blood. This paper reports the experience of building SPIRA: an intelligent system for detecting respiratory insufficiency from voice. It compiles challenges faced in two succeeding implementations of the same architecture, summarizing lessons learned on data collection, training, and inference for future projects in similar systems.
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Ribeiro, Carla, Daniela Ferreira, Sara Conde, Pedro Oliveira, and Wolfram Windisch. "Translation and Validation of the Portuguese Severe Respiratory Insufficiency Questionnaire." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2132.

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Christiansen, Ida Skovgaard, Uffe Bodgter, Rafi Nessar, Goran Nadir Salih, Shailesh Kolekar, Jatinder Singh Sidhu, Asbjørn Høgholm, et al. "Safety of EUS-B-FNA in patients with respiratory insufficiency." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.oa236.

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Kort, Janine, Gerrie Bladder, and Marieke Duiverman. "The minimal clinically important difference of the Severe Respiratory Insufficiency questionnaire." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2371.

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Rantala, Heidi, Sirpa Leivo-Korpela, Anni Hanhimäki, Lauri Lehtimäki, and Juho T. Lehto. "Assessing symptom burden and depression in patients with chronic respiratory insufficiency." In ERS Respiratory Failure and Mechanical Ventilation Conference 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/23120541.rfmvc-2020.37.

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Majorski, Daniel, Jens Callegari, Sarah Schwarz, Friederike Magnet, Tim Mathes, Hüseyin Ates, Rebar Ahmad, Navid Fazel, and Wolfram Windisch. "Development of the Severe Respiratory Insufficiency application-a randomized controlled trial-." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3423.

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Gauy, Marcelo Matheus, and Marcelo Finger. "Audio MFCC-gram Transformers for respiratory insufficiency detection in COVID-19." In Simpósio Brasileiro de Tecnologia da Informação e da Linguagem Humana. Sociedade Brasileira de Computação, 2021. http://dx.doi.org/10.5753/stil.2021.17793.

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This work explores speech as a biomarker and investigates the detection of respiratory insufficiency (RI) by analyzing speech samples. Previous work [Casanova et al. 2021] constructed a dataset of respiratory insufficiency COVID-19 patient utterances and analyzed it by means of a convolutional neural network achieving an accuracy of 87.04%, validating the hypothesis that one can detect RI through speech. Here, we study how Transformer neural network architectures can improve the performance on RI detection. This approach enables construction of an acoustic model. By choosing the correct pretraining technique, we generate a self-supervised acoustic model, leading to improved performance (96.53%) of Transformers for RI detection.
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Haimi, Motti, and Richard Kremer. "P I – 3–1 Vitamin d deficiency/insufficiency- from childhood to adulthood: insights from a sunny country." In ISEE Young 2018, Early Career Researchers Conference on Environmental Epidemiology – Together for a Healthy Environment, 19–20 March 2018, Freising, Germany. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-iseeabstracts.86.

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Casanova, Edresson, Lucas Gris, Augusto Camargo, Daniel da Silva, Murilo Gazzola, Ester Sabino, Anna Levin, Arnaldo Candido Jr, Sandra Aluisio, and Marcelo Finger. "Deep Learning against COVID-19: Respiratory Insufficiency Detection in Brazilian Portuguese Speech." In Findings of the Association for Computational Linguistics: ACL-IJCNLP 2021. Stroudsburg, PA, USA: Association for Computational Linguistics, 2021. http://dx.doi.org/10.18653/v1/2021.findings-acl.55.

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Reports on the topic "Respiratory Insufficiency In adulthood"

1

Byrne, Barry J. Advanced Gene Therapy for Treatment of Cardiomyopathy and Respiratory Insufficiency in Duchenne Muscular Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada613171.

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