Academic literature on the topic 'Réseau de la douleur'
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Journal articles on the topic "Réseau de la douleur"
Duclos, R. "EP28 - Création du Réseau Douleur Sarthe." Douleurs : Evaluation - Diagnostic - Traitement 5 (November 2004): 44–45. http://dx.doi.org/10.1016/s1624-5687(04)94623-9.
Full textClaire Delorme. "Mort annoncée d’un réseau régional douleur?" Douleurs : Evaluation - Diagnostic - Traitement 7, no. 1 (February 2006): 5–7. http://dx.doi.org/10.1016/s1624-5687(06)71101-5.
Full textRoussel, P., and D. Cognis. "EP42 - Présentation du Réseau Douleur PACA Ouest." Douleurs : Evaluation - Diagnostic - Traitement 7 (November 2006): 137. http://dx.doi.org/10.1016/s1624-5687(06)77971-9.
Full textTiberghien-Chatelain, F., A. Girod, C. Dovico, L. Balp, P. Fontaine, J. L. Delacour, and E. Lallier. "EP16 - Démarche d’évaluation du Réseau Douleur Franche-Comté." Douleurs : Evaluation - Diagnostic - Traitement 7 (November 2006): 125. http://dx.doi.org/10.1016/s1624-5687(06)77945-8.
Full textDelorme, Claire. "Bon anniversaire au Réseau Régional Douleur en Basse Normandie." Douleurs : Evaluation - Diagnostic - Traitement 12, no. 4 (September 2011): 163–64. http://dx.doi.org/10.1016/j.douler.2011.07.007.
Full textRuys, Jean François. "Évaluation de la douleur à domicile Expérience d’un réseau." Oncomagazine 2, no. 3 (August 2008): 13–15. http://dx.doi.org/10.1007/bf03165641.
Full textDuclos, D. "T037 - Bilan de 18 mois de fonctionnement du Réseau douleur en Sarthe - 1er réseau de santé douleur des pays de la loire." Douleurs : Evaluation - Diagnostic - Traitement 7 (November 2006): 95–96. http://dx.doi.org/10.1016/s1624-5687(06)77883-0.
Full textBoussac, Mathilde, and Emeline Descamps. "Changement de connectivité fonctionnelle cérébrale après une session de réflexologie plantaire lors d’un essai contrôlé randomisé." Hegel N° 4, no. 4 (January 18, 2024): 295–305. http://dx.doi.org/10.3917/heg.134.0295.
Full textLaroche, F., P. Sichère, E. Soyeux, M. J. Thévenot-Prunières, S. Rostaing-Rigattieri, V. Blanchet, F. Hirszowski, S. Rosensweig, and F. Boureau. "Le Réseau Lutter Contre la Douleur - Paris: 11 ans d'existence." Revue du Rhumatisme 73, no. 10-11 (November 2006): 1040. http://dx.doi.org/10.1016/j.rhum.2006.10.038.
Full textRibinik, P., B. Barrois, B. Davenne, B. Guilmin, G. Caillard, P. Daliphard, A. Camilleri, and J. Y. Lariviere. "EP17 - Organisation en réseau intra-hospitalier et douleur post opératoire." Douleurs : Evaluation - Diagnostic - Traitement 6 (November 2005): 109. http://dx.doi.org/10.1016/s1624-5687(05)80449-4.
Full textDissertations / Theses on the topic "Réseau de la douleur"
Fernández, Salazar Magali. "La dimension émotionnelle de la douleur chronique : perspectives neurophilosophiques sur la douleur du membre fantôme." Thesis, Paris 4, 2015. http://www.theses.fr/2015PA040060.
Full textChronic pain is one of the most complex problems facing medicine and neuroscience. Over the centuries, it has been a puzzle and remains a research challenge given the complexity of its nature. Among the large number of existing different kinds of chronic pain, phantom limb pain is one of the most difficult to treat. Recent studies show that major cortical changes that appear after amputation are the result of chronic phantom limb pain. I argue that the main cause of phantom limb pain is the non-acceptance of the loss of a part of the body, that is to say, that the mental pain caused by the transformation of the self-image becomes a chronic physical pain. It is the mind that controls the cérébral networks : even if it emerges from the brain, the mind manages to modify it as a consequence of external influences. The analysis of the studies I performed to test my hypothesis, allowed me to confirm that the perception of pain depends on various external influences that are independent of the nociceptive signals. I conclude that the cortical plasticity highlighted during chronic painful experience does not only depend on the action and interaction between dynamic neural networks, but also on the communication between these neural networks (endogenous system) and environmental networks (exogenous system). These latter networks are capable of modulating the perception of pain. I therefore emphasize the importance of recognizing the mental nature of chronic pain and the need to analyze the emotional dimensions which modulate it
Le, Franc Yann. "Traitement de l'information sensorielle et nociceptive par le réseau de la corne dorsale de la moelle épinière." Phd thesis, Université Victor Segalen - Bordeaux II, 2004. http://tel.archives-ouvertes.fr/tel-00548761.
Full textCazzanelli, Silvia. "Functional ultrasound (fUS) imaging of brain functional connectivity alterations in a mouse model of neuropathic pain : impact of nociceptive symptoms and associated comorbidities." Electronic Thesis or Diss., Université Paris sciences et lettres, 2024. http://www.theses.fr/2024UPSLS010.
Full textNeuropathic pain is an abnormal pain sensation that persists longer than the temporal course of natural healing. It interferes with the patient’s quality of life and leads to several comorbidities, such as anxiety and depression. It has been suggested that chronic pain may result from abnormal and maladaptive neuronal plasticity in the structures known to be involved in pain perception (Bliss et al. 2016). This means that nerve injury would trigger long-term potentiation of synaptic transmission in pain-related areas (Zhuo et al. 2014). Since these regions are also involved in the emotional aspects of pain, our hypothesis is that the aforementioned maladaptive plasticity in these brain areas could constitute a key mechanism for the development of comorbidities such as anxiety and depression.My PhD aimed at testing this working hypothesis, through the study of brain resting state functional connectivity (FC) using functional ultrasound imaging (fUS) in a mouse model of neuropathic pain. FUS is a relatively recent neuroimaging technique that enabled numerous advances in neuroscience, thanks to its high spatio-temporal resolution, its sensitivity, but also its adaptability, allowing studies in anesthetized or awake animals.In a first study, I developed an experimental protocol allowing the brains of awake mice to be imaged in a reproducible manner and with minimal stress and movement artifacts and was also involved in the development of a new algorithm for the analysis of the signals generated by these acquisitions. As this first approach was carried out with a moving linear probe which does not allow the entire brain to be visualized, in a second study, I participated in the development of a new compiled and motorized probe technology.Building on these technological developments, I then used these new approaches to test my neurobiological hypothesis. I undertook two parallel studies in animals anesthetized for one and awake for the second, in which we studied the temporal link between alterations in cerebral FC and the development of neuropathic pain and/or associated comorbidities. To do this, we measured the resting-state functional connectivity (FC) in anesthetized and in awake head-fixed mice, at three time points: I) 2 weeks after induction of neuropathic pain (cuff around the sciatic nerve), II) at 8 weeks post-induction during the emergence of anxiety (8W) and III) at 12 weeks post-induction during the emergence of depression. This longitudinal follow-up has been conducted concurrently on a control group.Our results show significant changes in FC in major pain-related brain regions in accordance with the development of neuropathic pain symptoms. These findings suggest that the pain network undergoes maladaptive plasticity following nerve injury which could contribute to pain chronification. Moreover, the time course of these connectivity alterations between regions of the pain network could be correlated with the subsequent apparition of associated comorbidities
Roca-Lapirot, Olivier. "Etude des réseaux neuronaux impliqués dans les contrôles descendants inhibiteurs de la douleur." Clermont-Ferrand 1, 2009. http://www.theses.fr/2009CLF1DD02.
Full textDiffuse noxious inhibitory controls (DNIC) are very powerful long-lasting descending inhibitory controls which are pivotal in modulating activity of spinal and trigeminal nociceptive neurons. The principal feature of DNIC is that they are subserved by a loop that involves supraspinal structures. In our study, DNIC were measured as in the anesthetized rat by the inhibition of nociceptive activity of trigeminal neurons induced by heterotopic noxious stimulation, as in the awake rat by the decreaseof rubbing response to facial formalin during heteropic noxious stimulation induced by hindpaw formalin injection. The first aim of this study was to evaluate the involvement of one of the main nociceptive ascending pathway. NK1- spino-parabrachial, in DNIC. Then, due to relationship between cardiovascular regulation and pain sensitivity, we wanted to know whether DNIC level is dependant on the arterial blood pressure (ABP level. Finally, given that descending inhibitory controls of pain are sustained at least in part by the early-studied serotonergic and noradrenergic system we wanted to study the involvement of the dopaminergic system, in (i) the control of trigeminal nociception, and (ii) the descending pathway of DNIC. All these hypotheses were tested by mean of behavioral; electrophysiological and anatomical (immunocytochemical) approaches. Our results first demonstrated that the activation of NK1-expressing dorsal horn neurones and lateral parabrachial area (LPB) neurones partly sustained the development of DNIC, suggesting that the LPB was specifically involved in the ascending pathway of DNIC. Secondly, we showed that tonic decrease of arterial blood pressure induced by a NO deliver, sodium nitroprusside, led to a significant decrease of DNIC. Thirdly, we showed that the dopaminergic modulation of trigeminal niciception was D2 receptor-dependant, and superficial laminae-specific. We showed that spinal trigeminal nucleus received direct projections from the hypothalamic, A11 dopaminergic nucleus, and that its inhibition induced a potentiation of the C-fiber-evoked trigeminal synaptic field. Finally, we showed that the descending pathway of DNIC was dependant on the A11 nucleus D2 receptors activation. To onclude, this study suggest that (i) ascending pathway of DNIC involves NK1-expressing dorsal horn and LBP neurones activation, (ii) DNIC development depends on cardiovascular signals integration, and (iii) A11 to trigeminal nucleus dopaminergique projections are involved in both control of trigeminal nociception and descending pathway of DNIC
Maiga, Youssoufa Mamoudou. "Représentation socio-culturelle de la douleur au Mali : étude des réseaux de prise charge et développement d'un modèle type de centre de la douleur adapté aux réalités locales." Thesis, Nantes, 2021. http://www.theses.fr/2021NANT1005.
Full textSub-Saharan Africa (SSA) is facing complex health problems involving a broad range of sociopolitical, economic, systemic, and cultural factors. In this context, the specificities and the difficulties of treating pain in sub-Saharan Africa are multiple (systemic and sociocultural). Furthermore, at the anthropological and medical levels, the clinical models and the explanatory models of pain are not identical to those of Western societies. The systems of representation of disease in certain African communities assert that pathological “states” are not only due to natural causes but that they can also be triggered by occult forces. Treatment of disease then becomes bidirectional by, firstly addressing the physical disorder with plants, and then by reestablishing the balance of life forces by prayers and rites. In light of the burden of pain at the individual and the community level, with this approach, there is a need to consider a range of care to treat pain tailored to the local realities. Whence our research topic, namely, what are the systems of representation and traditional practices for treating pain in Mali? What might the impacts be on the implementation of a center to monitor pain in Mali? The objective of this work was to study the Malian systems of representation and the sociocultural determinants of pain, so as to, on the one hand, contextualize the clinical models and, on the other hand, to devise a customized model of a pain center. The ultimate aim of this work was to make effective care, tailored to the sociocultural realities, available to the community (neurologists, politicians, researchers, patients, and traditional therapists) in order to reduce the burden of neurological pathologies in SSA. In this work, we have confirmed the prevalence of pain, and particularly neuropathic pain, in our practice. This work has also allowed us to appreciate the key role of traditional medicine in the provision of care. We call on researchers and policy decision-makers to find new strategies aimed at improvement of the treatment of pain by the implementation of novel programs geared toward promoting collaboration between traditional medicine and conventional medicine
Noseda, Ronco Rodrigo. "Architecture fonctionnelle des réseaux neuronaux impliqués dans la nociception méningée : approche expérimentale chez le rat." Clermont-Ferrand 1, 2007. http://www.theses.fr/2007CLF1DD02.
Full textMigraine remains a public health problem of great impact on both the patients life and society. The neurobiological mechanisms at the origin of migraine pain are largely unknown. Clinical and pre-clinical studies have shown the participation of cerebrovascular mechanisms during a migraine attack. In the central nervous system, trigemino-vascular neurons located in the upper cervical and in the medullary dorsal horns (Sp5C), convery meningeal nociceptive information to the central nervous system (CNS). The aim of this study was to analyze the organization of CNS networks involved in the processing of meningeal nociception in the rat. First, we have shown the existence of a widespread neuronal network of afferents from trigemino-vascular neurons of Sp5C. This network is the central substrate involved in the processing of inpus that elicit headache pain perception and concomitant reactions. Intratrigeminal connections were observed in the contralateral Sp5C and in the ipsilateral trigeminal nuclei oralis and principalis. Sp5C neurons projected to several brainstem regions, including the commissural subnucleus of the solitary tract, superior salivatory nucleus, lateral periaqueductal gray matter, inferior colliculus and parabrachial nuclei. Trigeminothalamic afferents were restricted to the posterior group and ventroposteromedial thalamic nuclei. Some of these brainstemregions are topographically intermingled with CGRP and serotonin (5HT1d) receptor labeled afferents. The hypothalamus seems to play an important role in trigeminal autonomic cephalalgias. These headaches are characterized by an extremely severe unilateral pain associated with ocular and facial autonomic symptoms. Nevertheless, the neuronal networks and mechanisms underlying such kind of headaches are not well elucidated. We investigated the hypothalamic networks that modulate directly trigeminovascular neurons. The dorsomedial capsular, ventral and posterior subnuclei of the paraventricular hypothalamic nucleus project densely and precisely to laminas I and outer II of the Sp5C. These projections are topographically intermingled with CGRP and 5HT1D receptor labeled primary afferents. These hypothalamic regions project to other CNS regions involved in descending modulation of meningeal nociception and autonomic outflow. Preclinical and clinical studies suggest that Cortical Spreading Depression (CSD) plays a key role in the triggering of migraine and in the sensitization of meningeal nociceptors. However, the circuitry and the mechanisms underlying such phenomena are still largely unknown. The aim of this study was to investigate the correlation between cortical excitability and meningeal nociception by studying the organization of cortical projections to the Sp5C, their relationship with 5HT1D or CGRP and the effects of corticofugal modulation on Sp5C neurons. Cortical neurons projecting to the Sp5C were confined to the contralateral layer V of the primary somatosensory (S1) and posterior insular (INS) cortices. Corticofugal projections from S1 terminate deeper, in laminas III-V, whereas those from INS terminate precisely in the superficial layers. Corticotrigeminal axons from INS are intermingled with both 5HT1D and CGRP afferents within laminas I and outer II. CSD elicited within S1 inhibited both tactile- and Aδ/C fibers meningeal-evoked activities of Sp5C neurons. In contrast, only facilitation of meningeal-evoked responses occurred following CSD within INS. Our study suggest that CSD is able to specifically interact with endogenous corticofugal mechanisms at the origin of migraine. In conclusion, our work illustrates the relevance of anatomo-functional approaches with the aim to elucidate the functional architecture of CNS nociceptive networks involved the triggering and the modulation of primary headaches
Morabit, Safaa El. "New Artificial Intelligence techniques for Computer vision based medical diagnosis." Electronic Thesis or Diss., Valenciennes, Université Polytechnique Hauts-de-France, 2023. http://www.theses.fr/2023UPHF0013.
Full textThe ability to feel pain is crucial for life, since it serves as an early warning system forpotential harm to the body. The majority of pain evaluations rely on patient reports. Patients who are unable to express their own pain must instead rely on third-party reportsof their suffering. Due to potential observer bias, pain reports may contain inaccuracies. In addition, it would be impossible for people to keep watch around the clock. Inorder to better manage pain, especially in noncommunicative patients, automatic paindetection technologies might be implemented to aid human caregivers and complementtheir service. Facial expressions are used by all observer-based pain assessment systemsbecause they are a reliable indicator of pain and can be interpreted from a distance.Taking into consideration that pain generally generates spontaneous facial behavior,these facial expressions could be used to detect the presence of pain. In this thesis, weanalyze facial expressions of pain in order to address pain estimation. First, we presenta thorough analysis of the problem by comparing numerous common CNN (Convolutional Neural Network) architectures, such as MobileNet, GoogleNet, ResNeXt-50, ResNet18, and DenseNet-161. We employ these networks in two unique modes: standalone and feature extraction. In standalone mode, models (i.e., networks) are utilized to directly estimate pain. In feature extractor mode, "values" from the middle layer are extracted and fed into classifiers like Support Vector Regression (SVR) and Random Forest Regression (RFR).CNNs have achieved significant results in image classification and have achievedgreat success. The effectiveness of Transformers in computer vision has been demonstrated through recent studies. Transformer-based architectures were proposed in the second section of this thesis. Two distinct Transformer-based frameworks were presented to address two distinct pain issues: pain detection (pain vs no pain) and thedistinction between genuine and posed pain. The innovative architecture for binaryidentification of facial pain is based on data-efficient image transformers (Deit). Twodatasets, UNBC-McMaster shoulder pain and BioVid heat pain, were used to fine-tuneand assess the trained model. The suggested architecture is built on Vision Transformers for the detection of genuine and simulated pain from facial expressions (ViT). Todistinguish between Genuine and Posed Pain, the model must pay particular attentionto the subtle changes in facial expressions over time. The employed approach takes intoaccount the sequential aspect and captures the variations in facial expressions. Experiments on the publicly accessible BioVid Heat Pain Database demonstrate the efficacy of our strategy
Czekala, Claire. "Percevoir la douleur sur le visage d'autrui : du traitement subliminal à la mise en jeu des réseaux neuronaux sous-jacents." Thesis, Lyon 1, 2015. http://www.theses.fr/2015LYO10322/document.
Full textThe aim of this work is to study painful facial expression processing through psychophysical and neurophysiological approaches. Contrary to the basic emotions, pain is both a sensory and an emotional experience and these two aspects are encoded in the facial expression of pain. In that sense, painful facial expressions are richer and more complex than the facial expression of others emotions. In a first phase, we showed that painful facial expressions trigger more empathy than other emotional facial expressions in healthy subjects. Moreover, a 100ms-masked presentation of faces is enough to subliminally detect pain but not gender. In a second phase, we studied pre-conscious processing of painful facial expressions in patients suffering from refractory epilepsy having intracranial electrodes implanted in the insular cortex and amygdala for stereotaxic exploration of epilepsy. To this purpose, we diverted the patients' attention from the emotional aspects of the faces by asking them to focus on the gender and we recorded evoked potentials to pain and other emotional faces. Results showed an early activation in the anterior part of the insula (onset latency around 131ms, peak latency 180ms post stimulus) followed by an amygdala response (onset latency around 273ms, peak latency 363ms post stimulus). Response to pain faces is larger than that to other emotional faces in anterior insula but anterior-insula and amygdala activations are not pain specific. Posterior part of the insula also responds to painful faces but the amplitude of the evoked potentials do not differ from that of potentials evoked by neutral faces. In this way, even if the pain face contains a great amount of information, the human- being is able to rapidly detect it and to be empathic enough to provide the help needed for others in pain. This ability would be possible through anterior insula activation, thought to be a relay between nociception and emotional reaction to pain
Guerreiro, covita João. "Role of relaxin-3/RXFP3 forebrain networks in the descending control of pain in the mouse." Thesis, Bordeaux, 2019. http://www.theses.fr/2019BORD0121.
Full textPain is a complex biological phenomenon that is beneficial and necessary for our survival, warning of changes and hazards in the environment that compromise optimal function. However, continuous activation of pain signalling systems results in maladaptive changes characterized by altered tissue and organ structure and activity. Pain lasting more than 3 months is termed chronic pain and it is under these conditions that it becomes a major burden for affected individuals. Chronic pain is also accompanied by serious social and economic burdens, making research in this field a high priority globally.The central nervous system (CNS) acts as a major control centre for nociceptive signal transmission, decoding pain for its sensory-discriminative and aversive components, i.e., deciphering the type of pain (pinch, burn, etc.), its location in the body, and its associated hedonic value, respectively. Once the information is decrypted, neural signals from the brain to the periphery act in accordance with the provided stimulus based on past and current experiences.The nociceptive signal is modulated at every step of this process by an abundance of neurochemical signals, including neuropeptides. The presence of neuropeptides and/or their receptors in areas linked to nociceptive processing and transmission suggests putative roles for these systems in the control of nociception.Relaxin-3 is a neuropeptide that is mainly synthesized in a hindbrain region known as the nucleus incertus (NI). Since its discovery, relaxin-3 has been linked to the control of a wide range of behaviours such as anxiety-like behaviours, arousal, and reward-seeking, through activation of the Gi/o-protein-coupled receptor, RXFP3. These putative roles of relaxin-3/RXFP3 signalling suggest a possible link between RXFP3 activation and modulation of pain sensitivity.Therefore, my initial studies assessed the effect of RXFP3 activation and inhibition on mechanical and thermal pain sensitivity in normal and persistent pain conditions. These studies demonstrated that central administration of the RXFP3 agonist peptide, RXFP3-A2, via intracerebroventricular (icv) injection, produced relief of mechanical, but not thermal, pain sensation. Moreover, icv injection of the RXFP3 antagonist peptide, R3(B1-22)R, augmented mechanical and thermal pain sensitivity. These data suggest that relaxin-3/RXFP3 signalling has a tonic action in maintaining mechanical and thermal pain thresholds, and the potential for activation of RXFP3 to produce pain relief.Additionally, I examined the possible involvement of different brain areas in these effects, by assessing the number of c-Fos-positive cell nuclei under different conditions. However, these studies revealed no difference in the number of c-Fos-positive cell nuclei or staining intensity in the vehicle- and RXFP3 agonist-treated mice.Further characterization of pain circuit-related brain areas using multiplex in situ hybridization revealed that RXFP3 mRNA is expressed within discrete populations of neurons in these areas. I also evaluated possible co-expression of RXFP3 mRNA with somatostatin and parvalbumin mRNA, and determined the relative proportion of RXFP3 mRNA expression in populations of neurons that express these transcripts.Finally, I examined the possible presence of comorbid anxiety in mice subjected to the persistent pain protocol. However, anxiety-like behaviour was not altered in mice with persistent hindpaw pain, suggesting this model does not display produce anxiety, and that effects of RXFP3 modulation observed specifically targeted nociceptive transmission.Overall, my findings implicate the relaxin-3/RXFP3 system in control of pain transmission, providing new opportunities for the development of therapeutic tools for pain management, by targeting a neuropeptide system that impacts several behaviours that are altered in chronic pain conditions
Karatas, Meltem. "Analyse longitudinale des réseaux cérébraux par Imagerie de Résonance Magnétique (IRM) dans un modèle murin de dépression induite par la douleur neuropathique." Thesis, Strasbourg, 2019. http://www.theses.fr/2019STRAJ045.
Full textChronic pain conditions frequently lead to anxiety and depressive disorders. Despite considerable clinical research, the mechanisms underlying this comorbidity remain elusive. We conducted a non-invasive brain imaging study to investigate changes in structural and functional connectivity in a mouse model of neuropathic pain-induced depression. We employed two methods of magnetic resonance imaging (MRI) to investigate functional communication pathways (using resting state functional MRI-rs-fMRI) as well as their microstructural substrates (diffusion MRI) in longitudinal manner. Brain networks demonstrate remarkable structural and functional modifications following the induction of neuropathic pain and the emergence of depressive phenotype. Combining a relevant preclinical model and in vivo brain MRI, we identified a brain connectivity signature of pain-induced depression and its evolution over time, involving alterations in reward circuits, with a major impact of the two centers: ACA and VTA. The main results of functional imaging reveal considerable changes in the networks encompassing the reward circuit and DMN, which are known to be involved in both chronic pain pathologies and major depression. The long-term perspective of this project is to investigate the causal relationship between pain and depression, reaching a mechanistic explanation for the comorbidity
Books on the topic "Réseau de la douleur"
Hydro-Québec. Le Réseau. [Montréal]: Hydro-Québec, Vice-présidence information et affaires publiques, Direction édition et publicité, 1990.
Find full textMosconi, Patrick. Douleur apache. Monaco: Editions du Rocher, 1995.
Find full textZerrouilou, Khellil Abdelkader dit. Douleur enchantée. Paris: Editions Saint-Germain-des-Prés, 1990.
Find full textDuras, Marguerite. La douleur. Paris: France Loisirs, 1985.
Find full textMarguerite, Duras. La douleur. [Paris]: Gallimard, 1985.
Find full textNègre, Isabelle. Mémo douleur. [Paris]: Éditions Lamarre, 2007.
Find full textMarguerite, Duras. La douleur. Paris: Gallimard, 1993.
Find full textCharlotte, Andrieux, ed. La douleur. Lleida: Edicions de la Universitat de Lleida, 2001.
Find full textaut, Andrieux Charlotte, ed. La Douleur. Lleida: Edicions de la Universitat de Lleida, 2001.
Find full textMarguerite, Duras. La douleur. London: Collins, 1986.
Find full textBook chapters on the topic "Réseau de la douleur"
Peeters-Asdourian, C. "La douleur." In Thérapeutique du cancer, 909–22. Paris: Springer Paris, 2011. http://dx.doi.org/10.1007/978-2-8178-0021-9_54.
Full textFlipo, Fabrice. "Effet de réseau/économie de réseau." In Angles morts du numérique ubiquitaire, 136–42. Nanterre: Presses universitaires de Paris Nanterre, 2023. http://dx.doi.org/10.4000/11tps.
Full textde Kernier, Nathalie. "Le rêve." In Le rêve, 85–99. In Press, 2023. http://dx.doi.org/10.3917/pres.misso.2023.01.0086.
Full textPereira, Shiranee. "Douleur animale, douleur humaine." In Douleur animale, douleur humaine, 73–80. Éditions Quæ, 2010. http://dx.doi.org/10.3917/quae.guich.2010.01.0010.
Full textGuichet, Jean-Luc. "Douleur animale, douleur humaine." In Douleur animale, douleur humaine, 175–89. Éditions Quæ, 2010. http://dx.doi.org/10.3917/quae.guich.2010.01.0022.
Full textServais, Véronique. "Douleur animale, douleur humaine." In Douleur animale, douleur humaine, 91–105. Éditions Quæ, 2010. http://dx.doi.org/10.3917/quae.guich.2010.01.0013.
Full textVan Trimpont, Frank. "Douleur animale, douleur humaine." In Douleur animale, douleur humaine, 143–49. Éditions Quæ, 2010. http://dx.doi.org/10.3917/quae.guich.2010.01.0018.
Full textBory, Jean-Yves. "Douleur animale, douleur humaine." In Douleur animale, douleur humaine, 61–72. Éditions Quæ, 2010. http://dx.doi.org/10.3917/quae.guich.2010.01.0009.
Full textGuichet, Jean-Luc. "Douleur animale, douleur humaine." In Douleur animale, douleur humaine, 7–17. Éditions Quæ, 2010. http://dx.doi.org/10.3917/quae.guich.2010.01.0003.
Full textGoffi, Jean-Yves. "Douleur animale, douleur humaine." In Douleur animale, douleur humaine, 122–35. Éditions Quæ, 2010. http://dx.doi.org/10.3917/quae.guich.2010.01.0016.
Full textConference papers on the topic "Réseau de la douleur"
Puff, Jean-François. "Du livre ouvert au livre fermé : Éluard à rebours." In Eluard, Capitale de la douleur. Fabula, 2013. http://dx.doi.org/10.58282/colloques.2244.
Full textBenoit, Jean-Louis. "La Mystique de Paul Éluard." In Eluard, Capitale de la douleur. Fabula, 2013. http://dx.doi.org/10.58282/colloques.2237.
Full textPérez, Claude-Pierre. "Partage et/ou solitude de la parole : sur quelques paradoxes éluardiens." In Eluard, Capitale de la douleur. Fabula, 2013. http://dx.doi.org/10.58282/colloques.2242.
Full textFontvieille-Cordani, Agnès. "Des lieux communs dans Capitale de la douleur." In Eluard, Capitale de la douleur. Fabula, 2013. http://dx.doi.org/10.58282/colloques.2239.
Full textRoger, Thierry. "L’œil et la main dans Capitale de la douleur." In Eluard, Capitale de la douleur. Fabula, 2013. http://dx.doi.org/10.58282/colloques.2241.
Full textVolant, Philippe. "Le réseau européen des TSOs." In La sûreté nucléaire à l’international - Les initiatives d’harmonisation. Les Ulis, France: EDP Sciences, 2015. http://dx.doi.org/10.1051/jtsfen/2015sur02.
Full textDoudet, Estelle. "Texte, récit, monde de fiction. Lectures en réseaux du fabliau des Tresses." In Les fabliaux en réseau. Fabula, 2024. http://dx.doi.org/10.58282/colloques.11607.
Full textDenoyelle, Corinne, and Stéphanie Le Briz-Orgeur. "Entre singularité et sérialité : les fabliaux édités et traduits par Jean Dufournet." In Les fabliaux en réseau. Fabula, 2024. http://dx.doi.org/10.58282/colloques.11604.
Full textCorbellari, Alain. "« Un goût de reviens-y » : quelques réflexions narratologiques et anthropologiques sur le thème des revenants et des « cadavres encombrants » dans les fabliaux." In Les fabliaux en réseau. Fabula, 2024. http://dx.doi.org/10.58282/colloques.11625.
Full textPierreville, Corinne. "De l’intérêt de rééditer les fabliaux." In Les fabliaux en réseau. Fabula, 2024. http://dx.doi.org/10.58282/colloques.11636.
Full textReports on the topic "Réseau de la douleur"
Brodaric, B. Réseau d'information sur les eaux souterraines. Natural Resources Canada/CMSS/Information Management, 2021. http://dx.doi.org/10.4095/329832.
Full textBastien, R. Un réseau de microgravimétrie dans la région d'Ottawa. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1988. http://dx.doi.org/10.4095/315287.
Full textMarsden, Eric, Noëlle Laneyrie, Cécile Laugier, and Olivier Chanton. La relation contrôleur-contrôlé au sein d’un réseau d’acteurs. Fondation pour une culture de sécurité industrielle, June 2023. http://dx.doi.org/10.57071/933rrr.
Full textVilain, Vincent. Protéger la vie privée via un réseau adversarial d’attaque de réidentification. Observatoire international sur les impacts sociétaux de l'intelligence artificielle et du numérique, September 2024. http://dx.doi.org/10.61737/tabe1427.
Full textChen, Z., S. E. Grasby, W. Yuan, M. Colpron, and X. Liu. Methodology study of geothermal resource evaluation using remote-sensing and ground-surface temperature data, Burwash Landing, Yukon – status and preliminary results. Natural Resources Canada/CMSS/Information Management, 2024. http://dx.doi.org/10.4095/p15d0hqc2g.
Full textLipsky, Alyson, Molly Adams, and Chinyere Okeke. Ground-Truthing Social Network Analysis for Universal Health Coverage Advocacy Networks in Nigeria. RTI Press, May 2024. http://dx.doi.org/10.3768/rtipress.2024.pb.0028.2405.
Full textAndersson, Göran, and Daniel Meierhans. Synthèse thématique «Réseaux d’énergie» du PNR «Energie». Swiss National Science Foundation (SNSF), December 2019. http://dx.doi.org/10.46446/publication_pnr70_pnr71.2019.2.fr.
Full textTouré, Mustapha, and Thomas G. Poder. Mesure des années de vie ajustées par la qualité de vie globale au Québec : le 13-MD. CIRANO, August 2022. http://dx.doi.org/10.54932/sxao9819.
Full textCanto, Patricia, ed. Apprentissages au regard des initiatives de fomentation de la collaboration transfrontaliere: une expérience au cœur de l’eurorégion Nouvelle-Aquitaine, Euskadi, Navarre. Universidad de Deusto, 2022. http://dx.doi.org/10.18543/ctih9147.
Full textManrique, Gabriela, Geneviève Baril, Johanne Préval, Marie-Christine Therrien, Julie-Maude Normandin, and Sandrine Veillette. La résilience du système de santé publique face à la COVID-19 : développement des systèmes informationnels par les directions régionales de santé publique au Québec. Observatoire international sur les impacts sociétaux de l’intelligence artificielle et du numérique, September 2021. http://dx.doi.org/10.61737/pfla2312.
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