Relevant bibliographies by topics / Repeat instabilty

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Repeat instabilty'

Author: Grafiati
Published: 19 October 2024
Last updated: 31 July 2025

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Journal articles on the topic "Repeat instabilty"

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Khristich, Alexandra N., and Sergei M. Mirkin. "On the wrong DNA track: Molecular mechanisms of repeat-mediated genome instability." Journal of Biological Chemistry 295, no. 13 (2020): 4134–70. http://dx.doi.org/10.1074/jbc.rev119.007678.

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Expansions of simple tandem repeats are responsible for almost 50 human diseases, the majority of which are severe, degenerative, and not currently treatable or preventable. In this review, we first describe the molecular mechanisms of repeat-induced toxicity, which is the connecting link between repeat expansions and pathology. We then survey alternative DNA structures that are formed by expandable repeats and review the evidence that formation of these structures is at the core of repeat instability. Next, we describe the consequences of the presence of long structure-forming repeats at the
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Lin, Yunfu, and John H. Wilson. "Transcription-Induced CAG Repeat Contraction in Human Cells Is Mediated in Part by Transcription-Coupled Nucleotide Excision Repair." Molecular and Cellular Biology 27, no. 17 (2007): 6209–17. http://dx.doi.org/10.1128/mcb.00739-07.

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ABSTRACT Expansions of CAG repeat tracts in the germ line underlie several neurological diseases. In human patients and mouse models, CAG repeat tracts display an ongoing instability in neurons, which may exacerbate disease symptoms. It is unclear how repeats are destabilized in nondividing cells, but it cannot involve DNA replication. We showed previously that transcription through CAG repeats induces their instability (Y. Lin, V. Dion, and J. H. Wilson, Nat. Struct. Mol. Biol. 13:179-180). Here, we present a genetic analysis of the link between transcription-induced repeat instability and nu
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Cohen, Haim, Dorothy D. Sears, Drora Zenvirth, Philip Hieter, and Giora Simchen. "Increased Instability of Human CTG Repeat Tracts on Yeast Artificial Chromosomes during Gametogenesis." Molecular and Cellular Biology 19, no. 6 (1999): 4153–58. http://dx.doi.org/10.1128/mcb.19.6.4153.

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ABSTRACT Expansion of trinucleotide repeat tracts has been shown to be associated with numerous human diseases. The mechanism and timing of the expansion events are poorly understood, however. We show that CTG repeats, associated with the human DMPK gene and implanted in two homologous yeast artificial chromosomes (YACs), are very unstable. The instability is 6 to 10 times more pronounced in meiosis than during mitotic division. The influence of meiosis on instability is 4.4 times greater when the second YAC with a repeat tract is not present. Most of the changes we observed in trinucleotide r
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Brouwer, Judith Rixt, Aline Huguet, Annie Nicole, Arnold Munnich, and Geneviève Gourdon. "Transcriptionally Repressive Chromatin Remodelling and CpG Methylation in the Presence of Expanded CTG-Repeats at the DM1 Locus." Journal of Nucleic Acids 2013 (2013): 1–16. http://dx.doi.org/10.1155/2013/567435.

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An expanded CTG-repeat in the 3′ UTR of theDMPKgene is responsible for myotonic dystrophy type I (DM1). Somatic and intergenerational instability cause the disease to become more severe during life and in subsequent generations. Evidence is accumulating that trinucleotide repeat instability and disease progression involve aberrant chromatin dynamics. We explored the chromatin environment in relation to expanded CTG-repeat tracts in hearts from transgenic mice carrying the DM1 locus with different repeat lengths. Using bisulfite sequencing we detected abundant CpG methylation in the regions fla
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Gold, Michaela A., Jenna M. Whalen, Karine Freon, et al. "Restarted replication forks are error-prone and cause CAG repeat expansions and contractions." PLOS Genetics 17, no. 10 (2021): e1009863. http://dx.doi.org/10.1371/journal.pgen.1009863.

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Disease-associated trinucleotide repeats form secondary DNA structures that interfere with replication and repair. Replication has been implicated as a mechanism that can cause repeat expansions and contractions. However, because structure-forming repeats are also replication barriers, it has been unclear whether the instability occurs due to slippage during normal replication progression through the repeat, slippage or misalignment at a replication stall caused by the repeat, or during subsequent replication of the repeat by a restarted fork that has altered properties. In this study, we have
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Neil, Alexander J., Julia A. Hisey, Ishtiaque Quasem, et al. "Replication-independent instability of Friedreich’s ataxia GAA repeats during chronological aging." Proceedings of the National Academy of Sciences 118, no. 5 (2021): e2013080118. http://dx.doi.org/10.1073/pnas.2013080118.

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Nearly 50 hereditary diseases result from the inheritance of abnormally long repetitive DNA microsatellites. While it was originally believed that the size of inherited repeats is the key factor in disease development, it has become clear that somatic instability of these repeats throughout an individual’s lifetime strongly contributes to disease onset and progression. Importantly, somatic instability is commonly observed in terminally differentiated, postmitotic cells, such as neurons. To unravel the mechanisms of repeat instability in nondividing cells, we created an experimental system to a
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Calluori, Stephanie, Rebecca Stark, and Brandon L. Pearson. "Gene–Environment Interactions in Repeat Expansion Diseases: Mechanisms of Environmentally Induced Repeat Instability." Biomedicines 11, no. 2 (2023): 515. http://dx.doi.org/10.3390/biomedicines11020515.

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Short tandem repeats (STRs) are units of 1–6 base pairs that occur in tandem repetition to form a repeat tract. STRs exhibit repeat instability, which generates expansions or contractions of the repeat tract. Over 50 diseases, primarily affecting the central nervous system and muscles, are characterized by repeat instability. Longer repeat tracts are typically associated with earlier age of onset and increased disease severity. Environmental exposures are suspected to play a role in the pathogenesis of repeat expansion diseases. Here, we review the current knowledge of mechanisms of environmen
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Gorbunova, Vera, Andrei Seluanov, Vincent Dion, Zoltan Sandor, James L. Meservy, and John H. Wilson. "Selectable System for Monitoring the Instability of CTG/CAG Triplet Repeats in Mammalian Cells." Molecular and Cellular Biology 23, no. 13 (2003): 4485–93. http://dx.doi.org/10.1128/mcb.23.13.4485-4493.2003.

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ABSTRACT Despite substantial progress in understanding the mechanism by which expanded CTG/CAG trinucleotide repeats cause neurodegenerative diseases, little is known about the basis for repeat instability itself. By taking advantage of a novel phenomenon, we have developed a selectable assay to detect contractions of CTG/CAG triplets. When inserted into an intron in the APRT gene or the HPRT minigene, long tracts of CTG/CAG repeats (more than about 33 repeat units) are efficiently incorporated into mRNA as a new exon, thereby rendering the encoded protein nonfunctional, whereas short repeat t
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Jung, Da Eun, and Chul Hyoung Lyoo. "A Spinocerebellar Ataxia Type 6 Patient Caused by <i>De Novo</i> Expansion of Normal Range CAG Repeats." Journal of the Korean Neurological Association 42, no. 2 (2024): 150–52. http://dx.doi.org/10.17340/jkna.2023.0105.

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Spinocerebellar ataxia type 6 (SCA6) is classified as a CAG repeat disorder, where the number of expanded CAG repeats often undergoes meiotic instability, when transmitted from one generation to the next. However, in SCA6, both normal and expanded CAG repeats tend to remain stable during transmission due to its relatively small repeat numbers. We herein report &lt;i&gt;de-novo&lt;/i&gt; expansion of CAG repeats in SCA6 gene in a 41-year-old female patient, whose parents had normal repeat numbers.
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Su, Xiaofeng A., and Catherine H. Freudenreich. "Cytosine deamination and base excision repair cause R-loop–induced CAG repeat fragility and instability in Saccharomyces cerevisiae." Proceedings of the National Academy of Sciences 114, no. 40 (2017): E8392—E8401. http://dx.doi.org/10.1073/pnas.1711283114.

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CAG/CTG repeats are structure-forming repetitive DNA sequences, and expansion beyond a threshold of ∼35 CAG repeats is the cause of several human diseases. Expanded CAG repeats are prone to breakage, and repair of the breaks can cause repeat contractions and expansions. In this study, we found that cotranscriptional R-loops formed at a CAG-70 repeat inserted into a yeast chromosome. R-loops were further elevated upon deletion of yeast RNaseH genes and caused repeat fragility. A significant increase in CAG repeat contractions was also observed, consistent with previous human cell studies. Delet
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Dissertations / Theses on the topic "Repeat instabilty"

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Pontual, Laure de. "Identification de nouveaux facteurs chimiques capables de moduler l'instabilité des répétitions CTG dans la dystrophie myotonique de type 1." Electronic Thesis or Diss., Sorbonne université, 2024. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2024SORUS198.pdf.

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La dystrophie myotonique de type 1 (DM1) est la dystrophie la plus fréquente chez l'adulte avec une prévalence estimée à 1 : 8000 individus. C'est une maladie multi-systémique caractérisée par des atteintes musculaires, cardiaques, cognitives et digestives responsables d'une réduction de l'espérance et de la qualité de vie des patients. Elle est causée par une expansion anormale de répétitions CTG en 3'UTR du gène DMPK. Dans la population générale, le nombre de répétitions est inférieur à 35 CTG tandis qu'il dépasse 50 CTG et peut atteindre jusqu'à plusieurs milliers de répétitions chez les pa
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Gadgil, Rujuta Yashodhan. "Instability at Trinucleotide Repeat DNAs." Wright State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

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Ubink-Bontekoe, Carola Jacoba Maria. "CGG repeat instability and FXR proteins." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2001. http://hdl.handle.net/1765/12091.

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Beaver, Jill M. "Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair." FIU Digital Commons, 2016. http://digitalcommons.fiu.edu/etd/3056.

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Trinucleotide repeat (TNR) expansions are the cause of over 40 human neurodegenerative diseases, and are linked to DNA damage and base excision repair (BER). We explored the role of DNA damage and BER in modulating TNR instability through analysis of DNA structures, BER protein activities, and reconstitution of repair using human BER proteins and synthesized DNA containing various types of damage. We show that DNA damage and BER can modulate TNR expansions by promoting removal of a TNR hairpin through coordinated activities of BER proteins and cofactors. We found that during repair in a TNR ha
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Ueki, Junko. "Myotonic dystrophy type 1 patient-derived iPSCs for the investigation of CTG repeat instability." Kyoto University, 2018. http://hdl.handle.net/2433/230991.

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Schmidt, Kristina H. "CTG trinucleotide repeat instability in Escherichia coli." Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/14353.

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In order to identify cellular factors that affect trinucleotide repeat stability, changes in the length of a (CTG)<sub>43</sub> repeat were studied over 140 generations in wild-type <i>Escherichia coli</i> and in strains that are deficient in post-replicative mismatch repair, secondary structure repair and homologous recombination. It is shown that (CTG)<sub>43</sub> inserted into pUC18 expands and contracts in wild-type <i>E. coli</i> in an orientation-dependent manner that is unaffected by transcription. In cells deficient in post-replicative mismatch repair (CTG)<sub>43</sub> repeat instabi
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Zahra, Rabaab. "CAG.CTG trinucleotide repeat instability in the E.coli chromosome." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/11667.

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In order to identify the molecular basis of genetic instability, a polymerization-independent strategy is developed to generate expanded repeat arrays. The repeat tracts are integrated in the 5’end of <i>lacZ</i> gene in the <i>Escherichia coli</i> chromosome. Using this model system, instability is studied in wild type <i>E. coli</i> and in strains deficient in cellular pathways such as DNA repair, replication and recombination. The work demonstrates that instability (expansion and contraction) in wild type cells is length and orientation dependent. Longer tracts are more unstable than shorte
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Chan, Kara Y. "MECHANISMS OF TRINUCLEOTIDE REPEAT INSTABILITY DURING DNA SYNTHESIS." UKnowledge, 2019. https://uknowledge.uky.edu/toxicology_etds/29.

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Genomic instability, in the form of gene mutations, insertions/deletions, and gene amplifications, is one of the hallmarks in many types of cancers and other inheritable genetic disorders. Trinucleotide repeat (TNR) disorders, such as Huntington’s disease (HD) and Myotonic dystrophy (DM) can be inherited and repeats may be extended through subsequent generations. However, it is not clear how the CAG repeats expand through generations in HD. Two possible repeat expansion mechanisms include: 1) polymerase mediated repeat extension; 2) persistent TNR hairpin structure formation persisting in the
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Pickett, Hilda A. "Molecular characterisation of instability in human telomere repeat arrays." Thesis, University of Leicester, 2002. http://hdl.handle.net/2381/30343.

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Telomere instability was investigated at the proximal ends of human telomeres in normal and abnormal cells, with the aim to identify the frequency and types of mutations underlying telomere repeat turnover. Analysis of the interspersion patterns of telomere and variant repeat types at the proximal ends of the 12q and Xp/Yp telomeres in human pedigrees gave a germline mutation frequency of 0.6% per telomere per gamete over the proximal 1kb of the telomere repeat array. No somatic telomere mutations were identified in normal fibroblast cells, but the upper limit for the mutation frequency was es
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Chan, Nelson Lap Shun. "IDENTIFICATION OF ACTIVITIES INVOLVED IN CAG/CTG REPEAT INSTABILITY." UKnowledge, 2011. http://uknowledge.uky.edu/gradschool_diss/832.

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CAG/CTG repeat instability is associated with at least 14 neurological disorders, including Huntington’s disease and Myotonic dystrophy type 1. In vitro and in vivo studies have showed that CAG/CTG repeats form a stable hairpin that is believed to be the intermediate for repeat expansion and contraction. Addition of extra DNA is essential for repeat expansion, so DNA synthesis is one of the keys for repeat expansion. In vivo studies reveal that 3’ CTG slippage with subsequent hairpin formation (henceforth called the 3’ CTG slippage hairpin) occurs during DNA synthesis. It is proposed that hair
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Books on the topic "Repeat instabilty"

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1946-, Oostra Ben A., ed. Trinucleotide diseases and instability. Springer, 1998.

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Hughes, Alis, and Lesley Jones. Pathogenic Mechanisms. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199929146.003.0013.

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Huntington’s disease (HD) pathogenesis is complex. In the two decades since the gene and its mutation were discovered, there has been extensive exploration of how the expanded CAG repeat in HTT leads to neurodegeneration in HD. This chapter focuses on the mechanisms that potentially contribute to the dysfunction and death of cells in HD. These include repeat instability and RNA toxicity and the production, processing, modification, and degradation of mutant huntingtin. The effects of mutant HTT on cellular processes such as transcription, transport, neurotransmission, and protein clearance are
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Zeitlin, Vladimir. Rotating Shallow-Water model with Horizontal Density and/or Temperature Gradients. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198804338.003.0014.

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The derivation of rotating shallow-water equations by vertical averaging and columnar motion hypothesis is repeated without supposing horizontal homogeneity of density/potential temperature. The so-called thermal rotating shallow-water model arises as the result. The model turns to be equivalent to gas dynamics with a specific equation of state. It is shown that it possesses Hamiltonian structure and can be derived from a variational principle. Its solution at low Rossby numbers should obey the thermo-geostrophic equilibrium, replacing the standard geostrophic equilibrium. The wave spectrum of
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Macartney, Huw. The Bank Culture Debate. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198843764.001.0001.

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Following the global financial crisis and repeated scandals, US and UK state managers made substantial efforts to reform the culture of their banking sectors. This book argues though that they focused on an extremely narrow definition of bank culture. They did so for two reasons: firstly, because the structural pressures of financialization—which are a far more important driver of the problematic features of bank culture in Anglo-America—are harder to remedy; but secondly, state managers also used their bank culture response to tackle a legitimacy crisis facing their institutions of government
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The Global State of Democracy 2022: Forging Social Contracts in a Time of Discontent. International Institute for Democracy and Electoral Assistance (International IDEA), 2022. http://dx.doi.org/10.31752/idea.2022.56.

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At the end of 2022, the world is trapped beneath the weight of a multitude of old and new problems. There are myriad causes of political and economic instability, including the spiking prices of food and energy, ballooning inflation and an impending recession. These phenomena are occurring in the unstable context of continuing climate change, long unresolved inequality, the Covid-19 pandemic, declining standards of living and the Russian war of aggression in Ukraine. Democratic institutions are especially important in times of crisis and fear. They ensure open pathways for the information and
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Hanning, Robert W. Boccaccio, Chaucer, and Stories for an Uncertain World. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780192894755.001.0001.

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The fictive inhabitants of the Decameron and the Canterbury Tales, the stories they tell each other, and the framing fictions that bring them together collectively acknowledge a radical uncertainty that characterizes most human endeavors, challenging the achievement of effective knowledge, perception, and strategy, and lacking intervention by Divine Providence. Dependable knowledge of the future, the past, or the distant present is thwarted by the fickle forces of chance (fortuna) and unverifiable report (fama); accurate perception of meaning is compromised by the instability or equivocality o
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Book chapters on the topic "Repeat instabilty"

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Wells, Robert D., Albino Bacolla, and Richard P. Bowater. "Instabilities of Triplet Repeats: Factors and Mechanisms." In Trinucleotide Diseases and Instability. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-540-69680-3_4.

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Parniewski, Pawel, and Pawel Staczek. "Molecular Mechanisms of TRS Instability." In Triple Repeat Diseases of the Nervous Systems. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0117-6_1.

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Curtis, J. Willard. "Churning: Repeated Optimization and Cooperative Instability." In Cooperative Systems. Springer US, 2004. http://dx.doi.org/10.1007/978-1-4613-0219-3_6.

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Salina, E. A., E. G. Pestsova, and N. P. Goncharov. "Instability of Subtelomeric Spelt1 Repeats of Wheat Species." In Stadler Genetics Symposia Series. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4235-3_23.

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Polleys, Erica J., and Catherine H. Freudenreich. "Methods to Study Repeat Fragility and Instability in Saccharomyces cerevisiae." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7306-4_28.

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Walker, Catherine A., and Catherine M. Abbott. "Trinucleotide Repeat Instability as a Cause of Human Genetic Disease." In Encyclopedia of Genetics. Routledge, 2014. http://dx.doi.org/10.4324/9781315073972-55.

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Mowbray, Miranda. "Observable Instability for the Repeated Prisoner’s Dilemma." In Approximation, Optimization and Mathematical Economics. Physica-Verlag HD, 2001. http://dx.doi.org/10.1007/978-3-642-57592-1_20.

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Deka, Ranjan, and Ranajit Chakraborty. "Trinucleotide Repeats, Genetic Instability and Variation in the Human Genome." In Genomic Diversity. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4263-6_4.

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Lai, Yanhao, Ruipeng Lei, Yaou Ren, and Yuan Liu. "Methods to Study Trinucleotide Repeat Instability Induced by DNA Damage and Repair." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9500-4_5.

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Kobayashi, Takehiko. "Genome Instability of Repetitive Sequence: Lesson from the Ribosomal RNA Gene Repeat." In DNA Replication, Recombination, and Repair. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55873-6_10.

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Conference papers on the topic "Repeat instabilty"

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Liu, Hui, Jingbo Liu, Bingchao Zhao, Changjiang Wang, and Jindong Cui. "Transient Voltage Instability Mechanism of New Energy Station Based on Repeated LVRT." In 2024 IEEE 8th Conference on Energy Internet and Energy System Integration (EI2). IEEE, 2024. https://doi.org/10.1109/ei264398.2024.10990604.

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Akatsuka, T., H. Imai, K. Arai, et al. "Highly stable laser repeater system with frequency instability below 10-21." In Conference on Lasers and Electro-Optics/Pacific Rim. Optica Publishing Group, 2022. http://dx.doi.org/10.1364/cleopr.2022.cfa6g_02.

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We report an ultralow-noise laser repeater system based on a narrow-linewidth laser and a planar lightwave circuit chip. An out-of-loop measurement demonstrates a record frequency instability of 3 × 10-22 at 30,000 s averaging time.
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Grasser, Tibor. "Towards Understanding Negative Bias Temperature Instability." In 2008 IEEE International Integrated Reliability Workshop Final Report (IRW). IEEE, 2008. http://dx.doi.org/10.1109/irws.2008.4796147.

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Grasser, Tibor. "Towards Understanding Negative Bias Temperature Instability." In 2008 IEEE International Integrated Reliability Workshop Final Report (IRW). IEEE, 2008. http://dx.doi.org/10.1109/irws.2008.4796110.

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Zafar, Sufi. "The Negative Bias Temperature Instability in MOS Devices." In 2006 IEEE International Integrated Reliability Workshop Final Report. IEEE, 2006. http://dx.doi.org/10.1109/irws.2006.305255.

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Hensman Moss, Davina, Anupriya Dalmia, Valentina Galassi Deforie, et al. "C12 HTT repeat instability in family trios in the 100,000 genomes project." In EHDN 2022 Plenary Meeting, Bologna, Italy, Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jnnp-2022-ehdn.56.

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Hongyu Zhao, Yue Zhao, Rong Chai, and Lu Cai. "Instability of the DNA repeats mutation in humans hereditary disorders." In 2011 International Conference on Remote Sensing, Environment and Transportation Engineering (RSETE). IEEE, 2011. http://dx.doi.org/10.1109/rsete.2011.5965950.

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Lelis, A. J., R. Green, D. Habersat, and N. Goldsman. "Effect of Threshold-Voltage Instability on SiC DMOSFET Reliability." In 2008 IEEE International Integrated Reliability Workshop Final Report (IRW). IEEE, 2008. http://dx.doi.org/10.1109/irws.2008.4796090.

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Lelis, Aivars, D. Habersat, R. Green, and N. Goldsman. "Effect of Threshold-Voltage Instability on SiC DMOSFET Reliability." In 2008 IEEE International Integrated Reliability Workshop Final Report (IRW). IEEE, 2008. http://dx.doi.org/10.1109/irws.2008.4796136.

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Lelis, A. J., S. Potbhare, D. Habersat, G. Pennington, and N. Goldsman. "Modeling and Characterization of Bias Stress-Induced Instability of SiC MOSFETs." In 2006 IEEE International Integrated Reliability Workshop Final Report. IEEE, 2006. http://dx.doi.org/10.1109/irws.2006.305235.

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Reports on the topic "Repeat instabilty"

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Brunner, Huschenbett, and Beshouri. PR-336-06206-R01 Engine Control for Legacy Engines - Cylinder and Cycle Level Control. Pipeline Research Council International, Inc. (PRCI), 2014. http://dx.doi.org/10.55274/r0010041.

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The reduction or even elimination of cylinder to cylinder combustion variation and cycle-to-cycle combustion instability may result in further nitrous oxides (NOx) reductions at the same engine average air/fuel ratio (and hence turbocharger operating point) or allow operation at a leaner air/fuel ratio before the onset of instability. Reductions in imbalance and instability can also reduce carbon pollutant emissions, reduce break specific fuel consumption (BSFC) and improve engine operability. Achieving these goals requires a better understanding of the sources and impacts of that imbalance an
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Chao, Alex. Very Large Hadron Collider Instability Workshop Summary Report. Office of Scientific and Technical Information (OSTI), 2001. http://dx.doi.org/10.2172/784899.

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Lahey, R. T. Jr. Analysis of nuclear reactor instability phenomena. Progress report. Office of Scientific and Technical Information (OSTI), 1993. http://dx.doi.org/10.2172/10131526.

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Aluie, Hussein. Final Technical Report: Ablative Magnetohydrodynamic Rayleigh-Taylor Instability. Office of Scientific and Technical Information (OSTI), 2023. http://dx.doi.org/10.2172/2311793.

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Collins, Robert. Coronavirus and North Korean Human Rights. Edited by Greg Scarlatoiu, Hannah Starnes, Ellie Richard, Lilli Duberstein, Canion Hempel, and Amanda Mortwedt Oh. The Committee for Human Rights in North Korea, 2025. https://doi.org/10.69981/4701.

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This report addresses the Kim regime's strengths and weaknesses within the scope of the coronavirus pandemic, and the impact on additional human rights challenges in the context of potential regime instability potentially brought on by events beyond regime control.
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Ismail, Zenobia. Interaction Between Food Prices and Political Instability. Institute of Development Studies (IDS), 2021. http://dx.doi.org/10.19088/k4d.2021.091.

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This report reviews the literature on links between international food prices and political instability (including protests, riots and social unrest). The literature on food prices and protests, riots, unrest, or violent incidents consists mainly of peer-reviewed scholarly articles that utilise econometric modeling. Some early studies examined the links between international food prices and political instability and found conflicting results. Some assessments concluded that there were links between international food prices or food insecurity and the number of violent incidents, while others f
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Dynan, William S. Final Technical Report - Mechanisms and pathways controlling genomic instability. Office of Scientific and Technical Information (OSTI), 2013. http://dx.doi.org/10.2172/1081424.

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Dynan, William S. Final report- Links between persistent DNA damage, genome instability, and aging. Office of Scientific and Technical Information (OSTI), 2016. http://dx.doi.org/10.2172/1333814.

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Majda, Andrew J. Report: Low Frequency Predictive Skill Despite Structural Instability and Model Error. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada601429.

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Mizuno, K., J. S. DeGroot, R. P. Drake, and W. Seka. Collective Thomson scattering measurements of the Ion Acoustic Decay Instability. Final report. Office of Scientific and Technical Information (OSTI), 1993. http://dx.doi.org/10.2172/10143761.

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