Relevant bibliographies by topics / Renal systems

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Renal systems'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2022

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Journal articles on the topic "Renal systems"

1

Yuan, Zhi-xiang, Zhenghui Shang, Jian Gu, and Lili He. "Renal targeting delivery systems." Future Medicinal Chemistry 11, no. 17 (September 2019): 2237–40. http://dx.doi.org/10.4155/fmc-2019-0152.

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Zeikus, Eric, Giri Sura, Nicole Hindman, and Julia R. Fielding. "Tumors of Renal Collecting Systems, Renal Pelvis, and Ureters." Magnetic Resonance Imaging Clinics of North America 27, no. 1 (February 2019): 15–32. http://dx.doi.org/10.1016/j.mric.2018.09.002.

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Stokes, M. Barry. "Classification Systems in Renal Pathology." Surgical Pathology Clinics 7, no. 3 (September 2014): 427–41. http://dx.doi.org/10.1016/j.path.2014.04.007.

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Hennefer, Dawn, and Elizabeth Lawson. "Pharmacology - a systems approach: renal system." British Journal of Healthcare Assistants 4, no. 1 (January 2010): 38–41. http://dx.doi.org/10.12968/bjha.2010.4.1.46073.

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Novara, Giacomo, Guido Martignoni, Walter Artibani, and Vincenzo Ficarra. "Grading Systems in Renal Cell Carcinoma." Journal of Urology 177, no. 2 (February 2007): 430–36. http://dx.doi.org/10.1016/j.juro.2006.09.034.

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Schafer, James A. "RENAL WATER AND ION TRANSPORT SYSTEMS." Advances in Physiology Education 275, no. 6 (December 15, 1998): S119—S131. http://dx.doi.org/10.1152/advances.1998.275.6.s119.

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Rhee, Eugene P. "A Systems-Level View of Renal Metabolomics." Seminars in Nephrology 38, no. 2 (March 2018): 142–50. http://dx.doi.org/10.1016/j.semnephrol.2018.01.005.

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Nishimura, M., A. Milsted, C. H. Block, K. B. Brosnihan, and C. M. Ferrario. "Tissue renin-angiotensin systems in renal hypertension." Hypertension 20, no. 2 (August 1992): 158–67. http://dx.doi.org/10.1161/01.hyp.20.2.158.

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Brown, Anthony, Mitchell Smith, Paul Rochon, and Charles Ray. "Percutaneous Access of Nondilated Renal Collecting Systems." Seminars in Interventional Radiology 31, no. 01 (February 20, 2014): 098–100. http://dx.doi.org/10.1055/s-0033-1363849.

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Pilz, Stefan, Andreas Meinitzer, Martin Gaksch, Martin Grübler, Nicolas Verheyen, Christiane Drechsler, Bríain ó. Hartaigh, et al. "Homoarginine in the renal and cardiovascular systems." Amino Acids 47, no. 9 (May 1, 2015): 1703–13. http://dx.doi.org/10.1007/s00726-015-1993-2.

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Dissertations / Theses on the topic "Renal systems"

1

Collett, Jason A. "Renal Humoral, Genetic and Genomic Mechanisms Underlying Spontaneous Hypertension." UKnowledge, 2014. http://uknowledge.uky.edu/biology_etds/24.

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In spite of significant progress in our knowledge of mechanisms that control blood pressure, our understanding of the pathogenesis of hypertension, its genetics, and population efforts to control blood pressure, hypertension remains the leading risk factor for mortality worldwide. It’s estimated that 1 out of every 3 adults has hypertension. Hypertension is a major risk factor for cardiovascular disease and stroke, and is considered a primary or contributing cause of death to more than 2.4 million US deaths each year. Although spontaneous hypertension has been the subject of substantial research, many critical questions remain unanswered. To investigate mechanisms underlying spontaneous hypertension, a unique rodent breeding approach was used to isolate nuclear and mitochondrial genes contributing to the disease. By diluting the nuclear genome of the Spontaneously Hypertensive Rat on a normotensive Brown Norway background while maintaining the SHR mitochondrial genome, I investigated both intrinsic and extrinsic mechanisms of the kidney and its relationship to hypertension. Chapter 2 documents the dominance of the hypertensive phenotype in our rodent colony, despite the dilution of the nuclear genome of the SHR. Chapter 3 presents data indicating that the renin-angiotensin system, particularly the location and abundance of the AT1 receptor may play an important role in the manifestation of spontaneous hypertension. Chapter 4 presents that rats in our rodent colony exhibited normal pressure-natriuresis and kidney function; however, hypertensive rats had a reduced ability to sense orally ingested sodium chloride, thus necessitating chronic elevations of arterial pressure in order to maintain sodium balance. This chronic pressure-natriuresis relationship shifts the renal function curve to the right, thus sustaining elevated blood pressure. Chapter 5 presents data that genes important for oxidative phosphorylation may play a critical role in the development of hypertension. Both nuclear and mitochondrial oxidative phosphorylation genes were downregulated in hypertensive rats compared with normotensive rats. Data presented in every chapter highlights the importance of the kidney in the pathogenesis of hypertension. Humoral, genetic and genomic mechanisms of the kidney appear to play a dominant role in the development and maintenance of the disease.
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Chau, Hien Nguyet 1977. "Renal calcification in Npt2 knockout mice." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=78338.

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Mice homozygous for the disrupted renal type 11a sodium/phosphate (Na/Pi) cotransporter gene, Npt2, (Npt2 KO) exhibit renal Pi wasting and hypercalciuria, predisposing factors for renal stone formation. We observed that Npt2 KO mice, but not wild-type littermates form renal stones. The renal stones were evident in newborn, weanling and adult mice and composed of calcium (Ca) and Pi. The presence of renal calcification correlated with the absence of Npt2 gene expression and the presence of genes responsible for the synthesis (1alpha-hydroxylase) and catabolism (24-hydroxylase) of 1,25-dihydroxyvitamin D, whose elevated levels contribute to the hypercalciuria and renal calcification in Npt2 KO mice. The renal calcification was associated with increased osteopontin (OPN) mRNA expression and colocalized with OPN, the latter associates with renal stones in vivo and inhibits Ca mineralization in vitro). These data demonstrate that hyperphosphaturia and hypercalciuria, secondary to Npt2 gene disruption, are sufficient for the development of renal calcification.
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Geraghty, Dominic P., and mikewood@deakin edu au. "Alterations in renal and myocardial adrenoceptors associated with ethynyloestradiol- and levonorgestrel-induced hypertension in the rat." Deakin University. School of Sciences, 1988. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051123.133239.

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Hypertension is one of many side effects of oral contraceptive use in a small percentage of women. Although the underlying pathology has yet to be fully resolved, alterations in the renin-angiotensin-aldosterone axis, sympathetic nervous system/ renal and cardiac function have been implicated. In the thesis to be presented, the possible involvement of alterations in renal and myocardial adrenoceptor characteristics in the pathogenesis of steroid contraceptive-induced hypertension in rats was examined by radioligand binding techniques. In Chapter 2, a rat model of OC hypertension is described. Chronic low-dose administration of ethynyloestradiol (EE2), levonorgestrel (NG) or a combination of both steroids (EE2/NG) to female Sprague-Dawley rats was shown to significantly increase systolic blood pressure (SBP). Renal and cardiac hypertrophy developed in association with EE2-, EE2/NG- but not NG-induced hypertension. Moreover, whereas administration of NG alone attenuated body weight gain, combined EE2/NG administration increased body weight gain from the second week of treatment onwards. Based on the above observations, it is proposed that EE2 and NG induce hypertension in rats via different mechanisms. Although SBP was elevated to a similar maximum in all steroid-treated groups (+ 20 mmHg compared to controls), only with EE2 administration did SBP remain elevated for the duration of the 17 week treatment regimen. NG may therefore have a protective effect on blood pressure with long-term combined steroid contraceptive treatment. In Chapter 4, renal adrenoceptors were characterized using radioactively labelled adrenocephor antagonists. Under appropriate conditions, binding of [3H]-prazosin and [3H]-rauwolscine to membrane preparations of whole rat kidney displayed the kinetics, saturability and specificity of α1- and α2 -adrenoceptors respectively, which were present in a ratio 3:1. In contrast, [3H]-dihydroergocryptine ([3H]-DHE) apparently bound to both α1 and α2-adrenoceptors. Binding sites identified by [125I] –iodocyanopindolol (ICYP) had the recognition characteristics of β-adrenoceptors. In drug competition studies using the subtype-selective antagonists practolol (β1) and ICI 118,551 (β2)/ the ratio of β1- to β2 -adrenoceptors was found to be approximately 2:1. Subsequently, renal adrenoceptors were investigated at various stages during the development of hypertension with the different steroid contraceptive treatments (Chapters 5 and 6). Preliminary binding studies with [3H]-DHE and [3H]-prazosin suggested that the number of renal α2 - but not α1-adrenoceptors was reduced in rats with established EE2-induced hypertension (17 weeks treatment). This was subsequently confirmed using [3H]-rauwolscine, which in addition showed that the reduction in renal α2 -adrenoceptor number occurred during the developmental stage of EE2/NG~induced hypertension (6 weeks treatment) and established EE2-induced hypertension (12 weeks treatment). NG induced hypertension was unassociated with changes in renal α1- and α2-adrenoceptor characteristics. Renal β-adrenoceptor affinity was reduced in established EE2-, but not NG- or EE2/NG- induced hypertension. Moreover, the β-adrenoceptor agonist (-)-isoprenaline bound to renal β-adrenoceptors with reduced affinity following EE2 administration. Several endogenous and synthetic steroids were found to be ineffective inhibitors of [3H] –prazosin, [3H] –rauwolscine and ICYP binding excluding a direct interaction of these steroids with renal α1-, α2- and β -adrenoceptors. In Chapter 7, myocardial adrenoceptors were characterized and investigated in steroid-treated rats. In membrane preparations of whole myocardium, [3H]-prazosin binding was characteristically to α1- adrenoceptors, whereas there was a notable absence of [3H]-rauwolscine binding. Using ICYP, β-adrenoceptors were also detected, the ratio of β1- to β2~adrenoceptors being 3:1. Steroid contraceptive-induced hypertension was not associated with myocardial α1-adrenoceptor changes. Similarly, myocardial β-adrenoceptors were unchanged in established EE2-, NG- and EE2/NG-induced hypertension (12 weeks treatment). The affinity of (-)-isoprenaline for myocardial β-adrenoceptors was unaffected by EE2 aditiinistration. These studies suggest that established EE2- but not NG-induced hypertension in rats is associated with selective alterations in renal α2- and (β-adrenoceptors. These adrenoceptor changes may help to maintain elevated blood pressure by affecting the control of renal function by the sympathetic nervous system, catecholamines and several hormones which affect renin release and the transport of fluid and electrolytes in the nephron.
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Hoag, Hannah M. "Characterization of the renal and the bone phenotypes of the Npt2 knock out mouse." Thesis, McGill University, 1999. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=30118.

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This study shows that mice homozygous for the disrupted renal sodium-phosphate (Na+-Pi) cotransporter, Npt2, (Npt2 KO) failed to show an age-dependent decrease in renal Na+-Pi cotransport or an adaptive increase in renal Na+-Pi cotransport in response to dietary Pi restriction. None of the other known renal Na+ -Pi cotransporters could compensate for the loss of Npt2. Additionally, Npt2 gene ablation resulted in a marked decrease in osteoclast number that persisted with age. Although mineral apposition rate was normal at 25- and 115-days of age in Npt2 KO mice, bone formation rate was increased at 115-days of age. These data demonstrate that Npt2 gene expression is necessary for an age-dependent decrease in renal Na+-Pi cotransport and for the renal adaptive response to dietary Pi deprivation, and that Npt2 expression is essential for normal osteoclast function and influences bone formation.
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Kos, Claudine H. "The role of the renal sodium-dependent phosphate cotransporter genes, NPT1 and NPT2, in inherited hypophosphatemias." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0026/NQ44478.pdf.

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Braga, Marcus Davis Machado. "Effects of Bothrops insularis venom and its isolated fractions on renal and vascular systems." Universidade Federal do CearÃ, 2006. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=40.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior
Foram investigados os efeitos do veneno da serpente Bothrops insularis e de suas fraÃÃes, lectina, L-aminoÃcido oxidase, trombina sÃmile e fosfolipase A2, no rim isolado e sistema vascular de rato. As fraÃÃes foram purificadas a partir de uma combinaÃÃo de procedimentos cromatogrÃficos, usando colunas de HPLC de exclusÃo molecular, troca iÃnica, fase reversa e colunas de baixa pressÃo de afinidade. Foi utilizada a perfusÃo de rim isolado de rato e a soluÃÃo de Krebs-Henseleit modificada (Bowman, 1970; Fonteles et al. 1998). ParÃmetros selecionados da funÃÃo renal foram avaliados durante as condiÃÃes experimentais, com a infusÃo do veneno e suas fraÃÃes, aos 60, 90, e 120 minutos. Os primeiros 30 minutos serviram de controle interno. No leito arterial sistÃmico de rato (Ferreira, 1965) a pressÃo arterial foi avaliada por manÃmetro conectado por cÃnula à artÃria carÃtida comum, e o veneno injetado na veia jugular. Os registros foram realizados a cada 10 minutos apÃs a administraÃÃo de doses crescentes do veneno, atà a infusÃo da dose de 300mcg, aos 60 minutos. Na PerfusÃo do leito arterial mesentÃrico isolado de rato (McGregor, 1965), utilizou-se a soluÃÃo de Krebs-Henseleit em fluxo constante de 4mL/minuto. A pressÃo de perfusÃo foi registrada manometricamente. A avaliaÃÃo estatÃstica foi determinada por anÃlise de variÃncia (ANOVA) e teste de Bonferroni, com nÃvel de significÃncia menor de 5%. No rim, o grupo tratado com o veneno apresentou reduÃÃo em todos os parÃmetros avaliados, com exceÃÃo da absorÃÃo de potÃssio. Com a lectina a pressÃo de perfusÃo aumentou inicialmente e caiu em seguida, juntamente com o fluxo urinÃrio e o ritmo de filtraÃÃo glomerular. Houve aumento na reabsorÃÃo de sÃdio e potÃssio, com reduÃÃo no clearance osmÃtico. Com a trombina-sÃmile, ocorreu aumento inicial seguido de queda no final em quase todos os parÃmetros, com exceÃÃo da resistÃncia vascular renal. A reabsorÃÃo tubular do sÃdio e do cloro caiu; houve elevaÃÃo inicial do transporte de potÃssio; com aumento seguido de queda do clearance osmÃtico. Com a L-aminoacido oxidase houve queda em todos os parÃmetros avaliados. Com a fosfolipase A2 houve elevaÃÃo nos parÃmetros fisiolÃgicos e vasculares; no transporte tubular de potÃssio e no clearance osmÃtico; com queda na reabsorÃÃo de sÃdio e cloro. Todos os rins mostraram, no final, sinais de necrose tubular aguda, com exceÃÃo dos perfundidos com a trombina-sÃmile. Excetuando os tratados com veneno, todos os rins apresentaram, ao final, extravasamento protÃico para o espaÃo de Bowman. No leito arterial sistÃmico o veneno produziu reduÃÃo na pressÃo arterial sistÃmica diretamente proporcional à quantidade de veneno administrada, excetuando a dose de 10mcg, alÃm de intensa hemorragia pulmonar com proliferaÃÃo de neutrÃfilos e linfÃcitos nos alvÃolos, hemorragia no rim e congestÃo generalizada. No leito arterial mesentÃrico se observou uma reduÃÃo na presÃo quando o veneno foi administrado em leito arterial prÃ-contraÃdo com fenilefrina, como tambÃm isoladamente, na ausÃncia de fenilefrina. O veneno da Bothrops insularis mostrou potencial hemorrÃgico e vasodilatador semelhante aos outros venenos de serpentes do gÃnero, com atividade necrotizante superior nos rins, onde provocou necrose tubular aguda, ao contrario do observado com outros venenos do mesmo gÃnero, em experimentos no rim isolado de rato.
We investigated the biochemical and biological effects of the whole venom from Bothrops insularis (popularly known as âgolden lancetâ), and four of its fractions, a thrombin-like enzyme, a lectin-like substance, an L-amino acid oxidase and a phospholipase A2, in perfused rat kidneys and vascular sistem. The fractions were purified by a combination of Sephadex gel filtration in HPLC columns, and ion-exchange chromatography on DEAE-Sephadex in reverse phase, low-pressure affinity columns. We used a modified isolated perfused rat kidney assay, with Krebs-Henseleit solution as the perfusion fluid (Bowman, 1970; Fonteles et al., 1998). Selected parameters of renal function during stable experimental conditions were evaluated before and at 60, 90, and 120 minutes after infusion of venom and its fractions, with the first 30 minutes interval constituting the paired control. In the systemic vascular bed (Ferreira, 1965), the arterial pressure was evaluated by a manometer connected through a canule to carotid common artery and the venom was injected into the jugular vein, with registers made at every 10 minutes after administration in increasing doses, until an infusion of 300mcg was reached at 60 minutes. In the isolated rat mesenteric blood vessels method (McGregor, 1965), the perfusions were done with Krebs-Henseleit solution, at a constant flow rate of 4mL/minute. The perfusion pressure was measured manometrically. Statistical evaluations were performed by analysis of variance (ANOVA) and Bonferroni test, at the 5% significance level. In perfused kidney studies, the group treated with the whole venom showed a fall in all physiological parameters, except in potassium transport. With the lectin-like fraction, the perfusion pressure rose initially, followed by a fall, along with urinary flow and glomerular filtration rate. Sodium and potassium tubular reabsorption increased, with a fall in the osmotic clearance. The thrombin-like fraction promoted an initial rise followed by a fall in the end, in almost all parameters except in the renal vascular resistance. The sodium and chloride tubular reabsorption fell. There was an initial rise in the potassium transport, and an initial rise followed by a fall in the osmotic clearance. With the L-amino acid oxidase fraction, there was a fall in all the parameters studied. The Phospholipase A2 fraction induced a rise in the physiological and vascular parameters, as also in the potassium transport and osmotic clearance; accompanied by a fall in sodium and chloride reabsorption. With the exception of the thrombin-like fraction, all the substances tested induced acute tubular necrosis in perfused kidneys in the end. Protein extravasation into the Bowman space was evidenced in all perfused kidneys except in those treated with the whole venom; but was more intense with the thrombin-like fraction. In the systemic arterial bed, the whole venom raised arterial pressure in a dose-dependant manner, except at the concentration of 10mcg; in addition to causing intense pulmonary hemorrhage with neutrophils and alveolar lymphocyte proliferation, renal hemorrhage, and generalized vascular dilatation and congestion. In the isolated mesenteric artery, there was a marked fall in perfusion pressure when the whole venom was infused into the vessel pre-contracted with phenillephrine, as also in the isolated vessel without phenillephrine. We conclude that Bothrops insularis venom shows vasodilatation and hemorrhagic potential, like other venoms of the genus; but, different from other Bothrops venoms, it also reveals a significant necrotic activity when perfused into isolated rat kidney, causing acute tubular necrosis,
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Duffy, Kevin. "Expression, regulation and substrate specificity of organic cation transporters in human renal cell systems." Thesis, University of Aberdeen, 2006. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU223029.

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Active uptake of the organic cation, tetraethylammonium (TEA), was shown in freshly isolated human and rat PT cells. This uptake was both inhibitable and temperature dependent. Several other drug substrates including ipratropium, procainamide, amantadine and pindolol were also actively taken up by fresh HPT cells. The human organic cation transporter, hOCT2, is the principal uptake transporter in the human proximal tubule. HEK cells transfected with hOCT2 were used to screen several drugs as possible substrates for this transporter. Substrates identified included TEA, amantadine, quinine, quinidine, procainamide, pindolol, oxytropium, ipratropium, and imipramine. Kinetic parameters for five of these were used to calculate their intrinsic clearances, which were correlated with renal clearance values in vivo. This correlation was significant (R2=0.814), suggesting that the HEK-hOCT2 cell line can be used to make predictions of the renal clearance expected in man for compounds that are hOCT2 substrates. The second-messenger pathways involved in the regulation of organic cation transport in freshly isolated HPT and RPT cells and HEK-hOCT2 cells were elucidated. Several pathways, most notably involving ALP, PKC and calmodulin, are involved in the regulation of organic cation transport in all these systems. Quantitative RT-PCR and immunoblotting were used to detect changes in expression of organic ion transporters in primary cultures of PT cells compared with those in vivo. Expression of apical ion transporters was maintained in these culture systems, while expression of those located basolaterally was down-regulated. As well as successfully predicting in vivo clearance data from cellular uptake rates in vitro, this is the first comprehensive study correlating functional organic cation transport mechanisms with a number of signalling pathways in human renal proximal tubules.
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Peck, Jennifer L. "The Effects of Acute Restraint Stress on Renal Vasculature Reactivity and the Sympathetic Nervous Systems." University of Akron / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=akron1289840898.

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O???Connell, Amanda Elizabeth School of Medical Science UNSW. "Consequences of an altered intrauterine environment on the offspring???s renal, cardiovascular and renin angiotensin systems." Awarded by:University of New South Wales. School of Medical Science, 2006. http://handle.unsw.edu.au/1959.4/26320.

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This thesis reports the effects of an altered intrauterine environment on the offspring???s renal, cardiovascular and renin angiotensin systems. After a midgestational asphyxial episode in fetal sheep (30 min total umbilical cord occlusion at 90 days; term 150 days) the hydrops that resulted had not completely resolved by 130 days. While the heart and kidneys were apparently unaffected, the brain and lung weights were 37% and 50% lower than sham values, respectively and there were joint contractures. The effects of maternal renal disease on the offspring were investigated. Although in utero fetuses of subtotally nephrectomised ewes (STNx) had altered urine flow rates, sodium excretion, haematocrits, plasma chloride and plasma renin levels, by 1-2 weeks after birth these values in the lambs (STNxL) were similar to controls (ConL) under baseline conditions. Body weight and the weights of most organs were similar, including the kidney, in which glomerular number was normal. In the neonatal period, the lambs were subjected to four challenges: furosemide (2 mg/kg intravenous bolus), infusion of angiotensin II and phenylephrine, intravenous infusion of 0.15M saline (50 ml/kg over 30 min) and haemorrhage (20% estimated blood volume over 10 min). These challenges revealed evidence of programming of several aspects of the renal, cardiovascular and renin angiotensin systems in the STNx offspring. As young adults at 6 months of age, male and female offspring of STNx ewes were normotensive and had normal renal function. On a high salt diet (HSD, 0.17M NaCl in 8L water for 5-7days), female offspring of both groups did not become hypertensive. However, the STNx offspring must have retained salt and water as plasma sodium was increased and haematocrit was decreased. In the STNx offspring only, there was a relationship between glomerular filtration rate (GFR) and mean arterial pressure, indicating an inability to maintain a constant GFR in response to changes in arterial pressure.
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Jones, Caroline Elizabeth Mary. "The development, evaluation and use of freshly isolated renal proximal tubule systems from the Fischer rat." Thesis, Aston University, 1990. http://publications.aston.ac.uk/12575/.

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The investigation of renal pathophysiology and toxicology has traditionally been advanced by the development of increasingly defined and refined in vitro preparations. This study has sought to develop and evaluate various methods of producing pure samples of renal proximal tubules (PTs) from the Fischer rat. The introduction summarised the most common in vitro preparations together with the parameters used to monitor viability - particularly with regard to toxic events. The most prevalent isolation methods have involved the use of collagenase to produce dissociation of the cortex. However, the present study has shown that even the mildest collagenase treatment caused significant structural damage which resulted in a longevity of only 3hr in suspension. An alternative mechanical isolation technique has been developed in this study that consists of perfusion loading the renal glomeruli with Fe304 followed by disruption of the cortex by homogenisation and sequential sieving. The glomeruli are removed magnetically and the PTs then harvested by a 64μM sieve. PTs isolated in this way showed a vastly superior structural preservation over their collagenase isolated counterparts; also oxygen consumption and enzyme leakage measurements showed a longevity in excess of 6hr when incubated in a very basic medium. Attempts were then made to measure the cytosolic calcium levels in both mechanical and collagenase isolated PTs using the fluorescent calcium indicator Fura. However results were inconclusive due to significant binding of the Fura to the external PT surfaces. In conclusion, PTs prepared by the present mechanical isolation technique exhibit superior preservation and longevity compared with even the mildest collagenase isolation technique and hence appear to offer potential advantages over collagenase isolation as an in vitro renal system.
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Books on the topic "Renal systems"

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Thomas, Robert (Robert H.)., ed. Renal and urinary systems. 4th ed. Edinburgh: Elsevier, 2013.

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Halushka, P. V., and D. E. Mais, eds. Eicosanoids in the Cardiovascular and Renal Systems. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1285-4.

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Freeman, Richard B. Renal dialysis decisionmaking. [Washington, D.C.?: Office of Technology Assessment, 1986.

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Freeman, Richard B. Renal dialysis decisionmaking. [Washington, D.C.?: Office of Technology Assessment, 1986.

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Freeman, Richard B. Renal dialysis decisionmaking. [Washington, D.C.?: Office of Technology Assessment, 1986.

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Morphology and function in MRI, cardiovascular and renal systems. Berlin: Springer-Verlag, 1989.

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Jones, Caroline Elizabeth Mary. The development, evaluation and use of freshly isolated renal proxinal tubule systems in the fischer rat. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1990.

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Field, Michael J. The renal system. Edinburgh: Harcourt Publishers, 2001.

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Field, Michael J. The renal system. Edinburgh: Churchill Livingstone, 2001.

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Field, Michael. The renal system. 2nd ed. Edinburgh: Churchill Livingstone, 2010.

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Book chapters on the topic "Renal systems"

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Lash, Lawrence H. "Renal Glutathione Transport Systems." In Glutathione, 35–38. Boca Raton: Taylor & Francis, 2018. | Series: Oxidative stress and: CRC Press, 2018. http://dx.doi.org/10.1201/9781351261760-2.

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Winchester, James F. "General telemedicine systems." In Replacement of Renal Function by Dialysis, 1575–79. Dordrecht: Springer Netherlands, 2004. http://dx.doi.org/10.1007/978-1-4020-2275-3_71.

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Sohn, William, F. Elena Marshall, Krishnansu S. Tewari, and Antoine Khoury. "Renal, Genitourinary, and Reproductive Systems." In Handbook of Long Term Care of The Childhood Cancer Survivor, 121–44. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7584-3_9.

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Azar, Ahmad Taher. "Measurement of Renal Function." In Modelling and Control of Dialysis Systems, 45–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-27458-9_2.

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Quilley, J., and J. C. McGiff. "Renal arachidonic acid metabolism." In Eicosanoids in the Cardiovascular and Renal Systems, 16–33. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1285-4_2.

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Pozharisski, Kazymir M., and Vladimir S. Turusov. "Angiosarcoma of the Renal Capsule, Mouse." In Cardiovascular and Musculoskeletal Systems, 91–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76533-9_16.

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Duty, Brian, Zhamshid Okhunov, Arthur D. Smith, and Zeph Okeke. "Drainage Systems After Percutanous Renal Procedures." In Difficult Cases in Endourology, 123–31. London: Springer London, 2012. http://dx.doi.org/10.1007/978-1-84882-083-8_13.

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Du, Caigan, Ximo Wang, and Huifang Chen. "Oxidative Stress to Renal Tubular Epithelial Cells – A Common Pathway in Renal Pathologies." In Systems Biology of Free Radicals and Antioxidants, 2605–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-30018-9_187.

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Taylor, Robert B., Alan K. David, Thomas A. Johnson, D. Melessa Phillips, and Joseph E. Scherger. "The Renal, Urinary, and Male Genital Systems." In Taylor’s Family Medicine Review, 201–11. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4612-2152-4_21.

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van Moorselaar, R. J. A., A. J. M. C. Beniers, J. A. Schalken, and F. M. J. Debruyne. "Interferon and Tumor Necrosis Factor in Renal Cell Carcinoma Model Systems." In Immunotherapy of Renal Cell Carcinoma, 47–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75853-9_8.

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Conference papers on the topic "Renal systems"

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Heckenauer, Robin, Jonathan Weber, Cedric Wemmert, Friedrich Feuerhake, Michel Hassenforder, Pierre-Alain Muller, and Germain Forestier. "Real-Time Detection of Glomeruli in Renal Pathology." In 2020 IEEE 33rd International Symposium on Computer-Based Medical Systems (CBMS). IEEE, 2020. http://dx.doi.org/10.1109/cbms49503.2020.00072.

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Jones, Jeffrey A., Ashot Sargsyan, Robert Pietryzk, C. Sams, Phillip Stepaniak, P. Whitson, James C. Williams, Andrew P. Evan, James E. Lingeman, and James A. McAteer. "Urolithiasis and Genitourinary Systems Issues for Spaceflight." In RENAL STONE DISEASE 2: 2nd International Urolithiasis Research Symposium. AIP, 2008. http://dx.doi.org/10.1063/1.2998040.

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Kassemi, Mohammad. "Role of Transport and Kinetics in Growth of Renal Stones." In 42nd International Conference on Environmental Systems. Reston, Virigina: American Institute of Aeronautics and Astronautics, 2012. http://dx.doi.org/10.2514/6.2012-3449.

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Heryan, Katarzyna, Dominik Choragwicki, Marek Sandheim, Jacek Jakubowski, and Tomasz Drewniak. "Renal Vessels Segmentation for Preoperative Planning in Percutaneous Nephrolithotomy." In 2018 IEEE International Conference on Imaging Systems and Techniques (IST). IEEE, 2018. http://dx.doi.org/10.1109/ist.2018.8577147.

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Katherine, Moorhead,. "Modelling Acute Renal Failure Using Blood and Breath Biomarkers in Rats." In Modeling and Control in Biomedical Systems, edited by Rees, Stephen, chair Andreassen, Steen and Andreassen, Steen. Elsevier, 2009. http://dx.doi.org/10.3182/20090812-3-dk-2006.00025.

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Gochoo, Munkhjargal, Jun-Wei Hsieh, Chien-Hung Lee, Yun-Chih Chen, and Yu-Chi Shih. "Chronic Kidney Disease Stage Classification Using Renal Artery Doppler-Derived Parameters." In 2019 IEEE International Conference on Systems, Man and Cybernetics (SMC). IEEE, 2019. http://dx.doi.org/10.1109/smc.2019.8913899.

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Nieto-Chaupis, Huber. "Nano Currents and the Beginning of Renal Damage: A Theoretical Model." In 2020 IEEE 33rd International Symposium on Computer-Based Medical Systems (CBMS). IEEE, 2020. http://dx.doi.org/10.1109/cbms49503.2020.00071.

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Aribi, Yassine, Fatma Hamza, Ali Wali, Fadhel Guermazi, and Adel M. Alimi. "ARG: A semi-automatic system for ROI detection on Renal Scintigraphic images." In 2014 14th International Conference on Hybrid Intelligent Systems (HIS). IEEE, 2014. http://dx.doi.org/10.1109/his.2014.7086166.

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Kassemi, Mohammad, and Ilana Iskovitz. "Prediction of Renal Stone Development and Size Distribution in Microgravity Using Population Balance Equation." In 43rd International Conference on Environmental Systems. Reston, Virginia: American Institute of Aeronautics and Astronautics, 2013. http://dx.doi.org/10.2514/6.2013-3319.

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Gao, Xianfu, Wanjia Chen, Rongxia Li, Minfeng Wang, Chunlei Chen, Rong Zeng, and Yueyi Deng. "Parallel metabolomics of urine and serum revealed systematic alteration associated with renal disease." In 2011 IEEE International Conference on Systems Biology (ISB). IEEE, 2011. http://dx.doi.org/10.1109/isb.2011.6033173.

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Reports on the topic "Renal systems"

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Davis, Michael, Elin L. Klaseen, Louis C. Schreier, Alan R. Downing, and Jon Peha. System Resource Management for Distributed Real-Time Systems. Fort Belvoir, VA: Defense Technical Information Center, July 1995. http://dx.doi.org/10.21236/ada303173.

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Robert, J., and Michael Forte. Field evaluation of GNSS/GPS based RTK, RTN, and RTX correction systems. Engineer Research and Development Center (U.S.), September 2021. http://dx.doi.org/10.21079/11681/41864.

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This Coastal and Hydraulic Engineering Technical Note (CHETN) details an evaluation of three Global Navigation Satellite System (GNSS)/Global Positioning System (GPS) real-time correction methods capable of providing centimeter-level positioning. Internet and satellite-delivered correction systems, Real Time Network (RTN) and Real Time eXtended (RTX), respectively, are compared to a traditional ground-based two-way radio transmission correction system, generally referred to as Local RTK, or simply RTK. Results from this study will provide prospective users background information on each of these positioning systems and comparisons of their respective accuracies during in field operations.
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Badr, Salah M., Jr Byrnes, Brutzman Ronald B., Nelson Donald P., and Michael L. Real-Time Systems. Fort Belvoir, VA: Defense Technical Information Center, February 1992. http://dx.doi.org/10.21236/ada252810.

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Quintero, Richard. A real-time control system methodology for developing intelligent control systems. Gaithersburg, MD: National Institute of Standards and Technology, 1992. http://dx.doi.org/10.6028/nist.ir.4936.

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Cummings, J. Overview of real-time computer systems technical analysis of the Modcomp implementation of a proprietary system MAX IV'' and real-time UNIX system REAL/IX''. Office of Scientific and Technical Information (OSTI), October 1990. http://dx.doi.org/10.2172/6356383.

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Vestal, Steve. Real-Time Complex Systems. Fort Belvoir, VA: Defense Technical Information Center, June 2004. http://dx.doi.org/10.21236/ada426487.

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Nielsen, Roy S. CS676 Real Time Systems Magnus Technology R&D Real Time Systems. Office of Scientific and Technical Information (OSTI), June 2015. http://dx.doi.org/10.2172/1183952.

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Davis, Joel L. Neuromorphic Systems: From Biological Foundations to System Properties and Real World Applications. Fort Belvoir, VA: Defense Technical Information Center, December 1997. http://dx.doi.org/10.21236/ada333498.

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Nelson, Arthur, Robert Hibberd, and Kristina Currans. Transit Impacts on Jobs, People and Real Estate. Transportation Research and Education Center (TREC), 2021. http://dx.doi.org/10.15760/trec.258.

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This report is comprised of five substantive elements. The first is crafting a scientifically sound framework for identifying landscapes within the metropolitan areas we studied. The second is applying those Place Typologies and spatial analysis to economic and demographic change for the transit system in each metropolitan area. The third is analyzing how real estate markets respond to transit system proximity with special reference to the Place Typologies. Fourth, this is followed by specialized studies into how urban form and society are shaped by transit systems. The fifth is providing an overall perspective of our research as well as a framework for unlocking the potential to leverage economic benefits of transit to advance social well-being.
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Whitbeck, George S., and Man-Tak Shing. CAPS and Real-Time Systems. Fort Belvoir, VA: Defense Technical Information Center, September 1996. http://dx.doi.org/10.21236/ada315954.

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