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Books on the topic 'Regulatory T cell'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 March 2023

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1

Ono, Masahiro, ed. Regulatory T-Cells. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2647-4.

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2

Kassiotis, George, and Adrian Liston, eds. Regulatory T Cells. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61737-979-6.

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3

Taams, Leonie S., Marca H. M. Wauben, and Arne N. Akbar, eds. Regulatory T Cells in Inflammation. Basel: Birkhäuser Basel, 2005. http://dx.doi.org/10.1007/b137037.

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4

Jiang, Shuiping, ed. Regulatory T Cells and Clinical Application. New York, NY: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-77909-6.

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5

Shuiping, Jiang, ed. Regulatory T cells and clinical application. New York: Springer, 2008.

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6

Regulatory T cells: Methods and protocols. [New York]: Humana Press, 2011.

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7

Zheng, Song-Guo, ed. T Regulatory Cells in Human Health and Diseases. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-6407-9.

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8

Compans, R. W., M. D. Cooper, T. Honjo, H. Koprowski, F. Melchers, M. B. A. Oldstone, S. Olsnes, et al., eds. CD4+CD25+ Regulatory T Cells: Origin, Function and Therapeutic Potential. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/3-540-27702-1.

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9

Na, Songqing, and Chandrasekar Venkataraman Iyer. Effector CD4+ T cells in health and disease 2007. Kerala, India: Transworld Research Network, 2007.

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10

R, Bock Gregory, Goode Jamie, and Novartis Foundation, eds. Generation and effector functions of regulatory lymphocytes. Chichester: John Wiley, 2003.

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11

Carabin Is a Negative Regulator of Cd8+ T-cell-mediated Anti-tumor Immunity. [New York, N.Y.?]: [publisher not identified], 2022.

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12

L, Croxford J., and Yamamura T, eds. The immuno-regulatory role of natural killer T cells in inflammatory disease. Kerala: Research Signpost, 2005.

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13

I, Gabrilovich Dmitry, and Hurwitz Arthur A, eds. Tumor-induced immune suppression: Mechanisms and therapeutic reversal. New York, NY: Springer, 2008.

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14

Smith, Kendall A., ed. The Molecular Mechanisms of Regulatory T cell Immunosuppression. Frontiers Media SA, 2013. http://dx.doi.org/10.3389/978-2-88919-093-5.

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15

Kolodin, Dmitriy Pavlovich. Dynamics of Tissue-Resident Regulatory T Cell Populations. 2014.

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16

Wong, Jamie Evan. The life of the regulatory T cell repertoire. 2007.

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17

Alexander, Michael A. Functional and Translational Immunology of Regulatory T Cells (Tregs), the Anti-Tumor T Cell Response, and Cancer. Nova Science Publishers, Incorporated, 2014.

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18

Dominguez-Villar, Margarita, Lucy S. K. Walker, and Silvia Piconese, eds. Control of Regulatory T cell Stability, Plasticity and Function in Health and Disease. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-570-9.

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19

Thomson, Christopher W. The role of FCRgamma in double negative T regulatory cell function and their expansion by lentivirally-transduced dendritic cells. 2006.

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20

Eljaafari, Assia, and Pierre Miossec. Cellular side of acquired immunity (T cells). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0049.

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The adaptive T-cell response represents the most sophisticated component of the immune response. Foreign invaders are recognized first by cells of the innate immune system. This leads to a rapid and non-specific inflammatory response, followed by induction of the adaptive and specific immune response. Different adaptive responses can be promoted, depending on the predominant effector cells that are involved, which themselves depend on the microbial/antigen stimuli. As examples, Th1 cells contribute to cell-mediated immunity against intracellular pathogens, Th2 cells protect against parasites, and Th17 cells act against extracellular bacteria and fungi that are not cleared by Th1 and Th2 cells. Among the new subsets, Th22 cells protect against disruption of epithelial layers secondary to invading pathogens. Finally these effector subsets are regulated by regulatory T cells. These T helper subsets counteract each other to maintain the homeostasis of the immune system, but this balance can be easily disrupted, leading to chronic inflammation or autoimmune diseases. The challenge is to detect early changes in this balance, prior to its clinical expression. New molecular tools such as microarrays could be used to determine the predominant profile of the immune effector cells involved in a disease process. Such understanding should provide better therapeutic tools to counteract deregulated effector cells.

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21

Regulatory T cells. Copenhagen: Munksgaard, 2001.

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22

Lu, Ling, Bruce Blazar, and Fadi Issa, eds. Regulatory T Cells. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-810-3.

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23

Regulatory T-Cells. Elsevier, 2011. http://dx.doi.org/10.1016/c2009-0-00167-6.

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24

Rudensky, Alexander, and Shimon Sakaguchi. Regulatory T-Cells. Elsevier Science & Technology Books, 2011.

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25

Regulatory T cells. Copenhagen: Munksgaard, 2001.

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26

Hayashi, Ren S. Regulatory T Cells. Nova Science Publishers, Incorporated, 2011.

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27

Regulatory T Cells [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.98075.

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28

Taams, Leonie S., Arne N. Akbar, and Marca H. M. Wauben. Regulatory T Cells in Inflammation. Springer London, Limited, 2005.

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29

Leonie S. Taams,Arne N. Akbar,Marca H. M. Wauben. Regulatory T Cells in Inflammation. Springer, 2008.

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30

Cvetanovich, Gregory. Molecular regulatory circuitry of human regulatory T cells and Th17 cells. 2012.

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31

Regulatory Tcells. Academic Press, 2011.

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32

Tsai, Ching-Wei, Sanjeev Noel, and Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.

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Acute kidney injury (AKI), regardless of its aetiology, can elicit persistent or permanent kidney tissue changes that are associated with progression to end-stage renal disease and a greater risk of chronic kidney disease (CKD). In other cases, AKI may result in complete repair and restoration of normal kidney function. The pathophysiological mechanisms of renal injury and repair include vascular, tubular, and inflammatory factors. The initial injury phase is characterized by rarefaction of peritubular vessels and engagement of the immune response via Toll-like receptor binding, activation of macrophages, dendritic cells, natural killer cells, and T and B lymphocytes. During the recovery phase, cell adhesion molecules as well as cytokines and chemokines may be instrumental by directing the migration, differentiation, and proliferation of renal epithelial cells; recent data also suggest a critical role of M2 macrophage and regulatory T cell in the recovery period. Other processes contributing to renal regeneration include renal stem cells and the expression of growth hormones and trophic factors. Subtle deviations in the normal repair process can lead to maladaptive fibrotic kidney disease. Further elucidation of these mechanisms will help discover new therapeutic interventions aimed at limiting the extent of AKI and halting its progression to CKD or ESRD.

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33

Kassiotis, George, and Adrian Liston. Regulatory T Cells: Methods and Protocols. Humana Press, 2016.

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34

Jiang, Shuiping. Regulatory T Cells and Clinical Application. Springer London, Limited, 2008.

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35

Ono, Masahiro. Regulatory T-Cells: Methods and Protocols. Springer, 2022.

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36

Jiang, Shuiping. Regulatory T Cells and Clinical Application. Springer, 2010.

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37

Liston, Adrian. Regulatory T Cells in Health and Disease. Elsevier Science & Technology Books, 2015.

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38

Liston, Adrian. Regulatory T Cells in Health and Disease. Elsevier Science & Technology, 2015.

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39

Regulatory T Cells in Health and Disease. Elsevier, 2015. http://dx.doi.org/10.1016/s1877-1173(15)x0008-1.

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40

Shvets, Anna. B7H4, a negative regulator of T cell immunity. 2006.

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41

van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient is diagnosed with SLE. Apoptotic debris is present in the extracellular matrix and circulation of patients with SLE due to an aberrant process of apoptosis and/or insufficient clearance of apoptotic cells and apoptotic debris. The non-cleared apoptotic debris in patients with SLE may lead to activation of both the innate (myeloid and plasmacytoid dendritic cells) and adaptive (T and B cells) immune system. In addition to the activation by apoptotic debris and immune complexes, the immune system in SLE may be deregulated at the level of (a) presentation of self-peptides by antigen-presenting cells, (b) selection processes for both B and T cells, and (c) regulatory processes of B- and T-cell responses. Lupus nephritis may be classified in different classes based on histological findings in renal biopsies. The chromatin-containing immune complexes deposit in the capillary filter, most likely due to the interaction of chromatin with the polysaccharide heparan sulphate. A decreased renal expression of the endonuclease DNaseI further contributes to the glomerular persistence of chromatin and the development of glomerulonephritis.Current treatment of lupus nephritis is not specific and aims to reduce the inflammatory response with general immunosuppressive therapies. However, research has revealed novel potential therapeutic candidates at the level of dendritic cells, B cells, and T cells.

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42

Skuljec, Jelena, Christine Happle, and Maria Grazia Roncarolo, eds. Therapeutic Potential of Gene-Modified Regulatory T Cells. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-847-4.

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43

Zheng, Song-Guo. T Regulatory Cells in Human Health and Diseases. Springer Singapore Pte. Limited, 2022.

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44

Zheng, Song-Guo. T Regulatory Cells in Human Health and Diseases. Springer, 2021.

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45

Goode, Jamie A., Gregory R. Bock, and Novartis Foundation Symposium Staff. Generation and Effector Functions of Regulatory Lymphocytes. Wiley & Sons, Incorporated, John, 2008.

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46

(Editor), Leonie S. Taams, Arne N. Akbar (Editor), and Marca H.M. Wauben (Editor), eds. Regulatory T Cells in Inflammation (Progress in Inflammation Research). Birkhäuser Basel, 2005.

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47

Kyewski, B., and Elisabeth Suri-Payer. CD4+CD25+ Regulatory T Cells: Origin, Function and Therapeutic Potential. Springer London, Limited, 2006.

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48

Kyewski, B., and Elisabeth Suri-Payer. CD4+CD25+ Regulatory T Cells: Origin, Function and Therapeutic Potential. Springer, 2010.

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49

Miljkovic, Djordje, and Piotr Trzonkowski, eds. Tolerogenic Dendritic Cells and Regulatory T Cells as Therapeutics for Immune-Mediated Disorders. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-646-3.

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50

Elkord, Eyad, ed. Thymus-derived, peripherally-derived and in vitro-induced T regulatory cells. Frontiers Media SA, 2014. http://dx.doi.org/10.3389/978-2-88919-234-2.

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