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Relevant bibliographies by topics / Raynaud's

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Academic literature on the topic 'Raynaud's'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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Journal articles on the topic "Raynaud's"

1

Devgire, Vikrant, and Michael Hughes. "Raynaud's phenomenon." British Journal of Hospital Medicine 80, no. 11 (November 2, 2019): 658–64. http://dx.doi.org/10.12968/hmed.2019.80.11.658.

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Raynaud's phenomenon is a common vasospastic condition which carries a significant burden of pain and hand-related disability ( Hughes and Herrick, 2016 ). The prevalence of Raynaud's phenomenon in the general population has been reported to be approximately 5% ( Garner et al, 2015 ). Raynaud's phenomenon can occur either as a primary (‘idiopathic’) phenomenon or secondary to a wide range of underlying medical conditions and drug causes. Therefore, hospital-based specialists are frequently involved in the care of patients with Raynaud's phenomenon and need to be aware of associated conditions and prescribed medications for Raynaud's phenomenon. In particular, Raynaud's phenomenon is often the earliest manifestation of an underlying autoimmune connective tissue disease (e.g. systemic sclerosis). A comprehensive clinical assessment is required including performing targeted investigations (e.g. nailfold capillaroscopy and systemic sclerosis-associated autoantibodies). Patient education and lifestyle adaptations is first-line treatment for Raynaud's phenomenon. There is a wide range of pharmacological options including oral and intravenous drug therapies available to treat Raynaud's phenomenon. Surgical intervention is sometimes required for refractory Raynaud's phenomenon and tissue ischaemia. This review describes the clinical manifestations of Raynaud's phenomenon including potential secondary causes and presents an approach to assessment and management.

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Haque, Ashraful, and Michael Hughes. "Raynaud's phenomenon." Clinical Medicine 20, no. 6 (November 2020): 580–87. http://dx.doi.org/10.7861/clinmed.2020-0754.

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Hughes, Michael, and Ariane L. Herrick. "Raynaud's phenomenon." Best Practice & Research Clinical Rheumatology 30, no. 1 (February 2016): 112–32. http://dx.doi.org/10.1016/j.berh.2016.04.001.

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Herrick, Ariane L., and Fredrick M. Wigley. "Raynaud's phenomenon." Best Practice & Research Clinical Rheumatology 34, no. 1 (February 2020): 101474. http://dx.doi.org/10.1016/j.berh.2019.101474.

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Belch, J. "Raynaud's phenomenon." Cardiovascular Research 33, no. 1 (January 1997): 25–30. http://dx.doi.org/10.1016/s0008-6363(96)00183-6.

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Block, Joel A., and Winston Sequeira. "Raynaud's phenomenon." Lancet 357, no. 9273 (June 2001): 2042–48. http://dx.doi.org/10.1016/s0140-6736(00)05118-7.

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Wigley, Fredrick M. "Raynaud's Phenomenon." New England Journal of Medicine 347, no. 13 (September 26, 2002): 1001–8. http://dx.doi.org/10.1056/nejmcp013013.

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Dowd, PaulineM, PortiaC Goldsmith, HelenA Bull, Geoffrey Burnstock, JohnC Foreman, and Ian Marshall. "Raynaud's phenomenon." Lancet 346, no. 8970 (July 1995): 283–90. http://dx.doi.org/10.1016/s0140-6736(95)92169-9.

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9

A., Michael. "RAYNAUD'S SYNDROME." ANZ Journal of Surgery 63, no. 2 (February 1993): 162. http://dx.doi.org/10.1111/j.1445-2197.1993.tb00074.x.

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10

Pearce, J. "Raynaud's phenomenon." Journal of Neurology, Neurosurgery & Psychiatry 52, no. 5 (May 1, 1989): 594. http://dx.doi.org/10.1136/jnnp.52.5.594.

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Dissertations / Theses on the topic "Raynaud's"

1

Gardner-Medwin, Janet. "The endothelium in primary Raynaud's phenomenon." Thesis, University of Nottingham, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.360542.

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Roustit, Matthieu. "Etude de la fonction microvasculaire cutanée dans le syndrome de Raynaud : approches physiopathologique et pharmacologique." Phd thesis, Université de Grenoble, 2011. http://tel.archives-ouvertes.fr/tel-00633855.

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La microcirculation cutanée a été proposée comme modèle d'étude de la dysfonction microvasculaire globale dans les maladies cardiovasculaires. Par ailleurs, elle est spécifiquement atteinte dans le syndrome de Raynaud, qui est une ischémie paroxystique des extrémités déclenchée notamment par le froid. L'exploration de la fonction microvasculaire cutanée suscite donc un réel intérêt, mais les méthodes d'étude souffrent d'une hétérogénéité importante, et leur variabilité intra-individuelle est mal connue. La première partie de ce travail fait la synthèse des différentes méthodes d'étude la fonction microvasculaire cutanée, et rapporte les résultats de deux études consacrées à leur reproductibilité. Nous avons dans une seconde partie étudié grâce à ces tests la réactivité microvasculaire cutanée dans le syndrome de Raynaud, et mis en évidence des anomalies chez ces patients, notamment du contrôle neuro-vasculaire. La dernière partie de cette thèse est consacrée à l'étude d'approches pharmacologiques ciblées sur les anomalies de la microcirculation cutanée identifiées chez les patients. Nous avons évalué l'effet du sildenafil, un inhibiteur de la phosphodiesterase-5, sur le flux sanguin digital et montré son effet vasodilatateur lors d'un refroidissement local dans le syndrome de Raynaud. Enfin, nous avons étudiés chez l'animal et chez l'homme l'iontophorèse de vasodilatateurs, une approche innovante d'administration locale de médicaments pour augmenter le flux sanguin cutané.

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Smyth, Anita Elizabeth. "The vascular genetics of primary Raynaud's phenomenon." Thesis, Queen's University Belfast, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268176.

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Najarian, B. "Psychological and physiological aspects of Raynaud's Phenomenon." Thesis, University of York, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234981.

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YAMADA, SHIN'YA, HISATAKA SAKAKIBARA, NOBUHIDE ISHIKAWA, and NORIKUNI TOIBANA. "RAYNAUD'S PHENOMENON OF FINGERS AND TOES AMONG VIBRATION-EXPOSED PATIENTS." Nagoya University School of Medicine, 1994. http://hdl.handle.net/2237/16060.

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Palmer, Keith. "Aspects of human health and occupational exposure to vibration." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312305.

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Khouri, Charles. "Pharmacologie de la microcirculation : phénomène de Raynaud, troubles trophiques cutanés et hypertension artérielle pulmonaire Drug‐induced Raynaud's phenomenon: beyond β‐adrenoceptor blockers Peripheral vasoconstriction induced by β‐adrenoceptor blockers: a systematic review and a network meta‐analysis Fluoxetine and Raynaud's phenomenon: friend or foe? Proton pump inhibitors and Raynaud’s phenomenon: is there a link?" Thesis, Université Grenoble Alpes (ComUE), 2019. https://thares.univ-grenoble-alpes.fr/2019GREAS028.pdf.

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La microcirculation désigne le sous-ensemble du système circulatoire où s'effectuent les échanges gazeux et liquidiens extracellulaires. Elle est composée des artérioles, des capillaires et des veinules. Plusieurs pathologies sont induites par une atteinte structurelle et/ou fonctionnelle primaire de cette microcirculation : le phénomène de Raynaud (PR), les troubles trophiques vasculaires et l’hypertension artérielle pulmonaire (HTAP). Les objectifs de ce travail sont d’étudier, de comprendre et d’identifier de nouvelles étiologies iatrogènes à ces pathologies microvasculaires, ainsi que d’évaluer et de comparer l’efficacité et la sécurité des traitements utilisés dans ces pathologies. Nous avons, à cette fin, réalisé plusieurs études à partir des bases de données de pharmacovigilances, de données d’essais cliniques et de la littérature.Ce travail de thèse nous a permis d’explorer le rôle des médicaments dans ces pathologies microvasculaires, champs qui restait encore peu étudié dans la littérature. Ces travaux nous ont permis d’identifier de nombreuses classes pharmacologiques dont le rôle était encore non décrit dans ces pathologies. L’étude des mécanismes pharmacologiques à l’origine de ces effets indésirables permet également d’émettre de nouvelles hypothèses physiopathologiques à l’origine de ces maladies.Les traitements utilisés dans ces différentes pathologies microcirculatoires sont à l’heure actuelle encore peu spécifiques et des travaux de recherche important doivent encore être réalisés afin de personnaliser la prise en charge des patients
Microcirculation refers to the subset of the circulatory system where extracellular gas and fluid exchanges take place. It is composed of arterioles, capillaries and venules. Several pathologies are induced by a primary structural and/or functional impairment of this microcirculation: Raynaud's phenomenon (RP), trophic vascular disorders and pulmonary arterial hypertension (PAH). The objectives of this work are to study, understand and identify new iatrogenic etiologies to these microvascular diseases, as well as to evaluate and compare the effectiveness and safety of treatments used in these diseases. We therefore conducted several studies using pharmacovigilance databases, clinical trial data and the literature.This thesis work allowed us to explore the role of drugs in these microvascular pathologies, fields that were poorly studied in the literature yet. This work has allowed us to identify many pharmacological classes whose role was unknown in these diseases. The study of the pharmacological mechanisms underlying these adverse drug reactions also makes it possible to develop new pathophysiological hypotheses underlying these diseases.The treatments used in these different microvascular diseases are currently not specific and important research work still needs to be carried out in order to personalize patient care

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Pauling, John D. "Evaluating digital vascular perfusion and platelet dysfunction in Raynaud's phenomenon and systemic sclerosis." Thesis, University of Bath, 2013. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.606666.

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Raynaud’s phenomenon (RP) describes excessive vasoconstriction of the digital microvasculature in response to cold exposure and emotional stress. RP is typically the earliest clinical manifestation of systemic sclerosis (SSc); a complex multisystem disease of unknown aetiology characterized by vasculopathy, inflammation and fibrosis. Vasculopathy is considered an essential pre-requisite to tissue remodeling, characterized by excessive collagen synthesis and tissue fibrosis, which occurs in the skin and other organs of patients with SSc. There is mounting evidence associating platelets with important biological functions beyond primary haemostasis. Upon activation, platelets release a large array of mediators implicated in vasoconstriction, inflammation and fibrosis, which has led me to consider the contribution of platelets to the pathogenesis of both RP and SSc. The principal aim of this thesis is to explore the impact of targeted anti-thrombotic therapy on digital microvascular function, platelet activation, oxidative stress and eicosanoid biosynthesis in RP and SSc. It is not possible to examine the impact of anti-platelet therapy on microvascular function, without first identifying sensitive methods for assessing digital microvascular function in humans. I shall report the findings of work examining established (infrared thermography) and novel (laser speckle contrast imaging) methods for the objective assessment of digital microvascular function in RP and SSc. I shall critically appraise the application of these methods alongside subjective patient self-report assessment of digital vascular function. I shall examine associations between digital vascular function, platelet activation, oxidative stress and eicosanoid biosynthesis between primary RP and SSc. I shall also report the findings of an investigator-led early phase clinical trial of targeted anti-thrombotic medication in RP and SSc. The major findings of this study were highlighting the strengths and limitations of established and novel methods for objective microvascular assessment in RP and SSc, and the poor agreement that exists between objective and subjective methods for assessing RP severity. I have identified similarities and differences in platelet activation and eicosanoid biosynthesis in primary RP and SSc. I shall present evidence of apparent efficacy of asasantin retard therapy on subjective, but not objective, assessments of digital microvascular function; the relevance of which shall be discussed in detail.

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9

Wilks, Julia. "A behavioural approach to the management of the symptoms of Raynaud's Disease and phenomenon." Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340528.

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Smith, Paula J. W. "An investigation into the pathogenesis of Raynaud's disease : the role of the vascular endothelium." Thesis, University of Edinburgh, 1996. http://hdl.handle.net/1842/20803.

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The aim of this thesis was to investigate the role of the vascular endothelium in the development of cold-induced vasoconstriction, and in the pathophysiology of vasospasm in RD. Experiments were performed on resistance arteries in vitro and in vivo. The small vessel arteriograph (perfusion myograph) was used to study isolated arteries in vitro. Studies in vivo used the autoperfused hindlimb of the anaesthetised rat, in which stepped-cooling of the blood entering the hindlimb produced a rise in hindlimb perfusion pressure. Possible mediators of this cold-induced vasoconstriction were identified using antagonists selective for α-adrenoceptors and ET-receptors. Inhibitors of NO-synthase and cyclooxygenase were also examined. The α₁/α₂-adrenoceptor antagonist, phentolamine, attenuated the cold-induced vasoconstriction, and was the only agent to have an effect that was significant in this model. The results demonstrate that the endothelium is an important modulator of cold-induced effects on the response to several vasoconstrictor agents, either by enhancing contraction through the release of vasoconstrictors such as ET, or by depressing contraction through the release of dilators, such as NO or PGI₂. The effects of cooling on the responsiveness of an artery appears to depend on the type of vessel under study (cutaneous versus non-cutaneous). The results from the gluteal biopsy studies imply that the altered sensitivity to ET-1 at 37^oC in RD patients is dependent on changes in dilator function, and not to altered sensitivity of the vascular smooth muscle to ET-1. Although an increase in the response to ET-1 was not found at 24^oC, the results obtained at 37^OC would support the hypothesis that the release and/or actions of NO is reduced in RD, allowing enhanced constriction of ET. These results suggest that the vascular endothelium is involved in the pathophysiology of RD.

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Books on the topic "Raynaud's"

1

Raynaud's phenomenon. New York: Oxford University Press, 1989.

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Mawdsley, Anne H. Raynaud's: Your questions answered. Alsager: The Raynaud's & Scleroderma Association, 1998.

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Society, Irish Raynaud's &. Scleroderma. Raynaud's phenomenon: An informative leaflet. Dublin: Irish Raynaud's & Scleroderma Society, 1998.

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Irish Raynaud's & Scleroderma Society. Teenage Raynaud's: You don't have to be old to be cold! : an informative leaflet. Dublin: Irish Raynaud's & Scleroderma Society, 1998.

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Reiley, Fergus. Evidence of oxidative stress in patients with Raynaud's phenomenon. Manchester: University of Manchester, 1996.

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Society, Irish Raynaud's &. Scleroderma. Vibration White Finger (VWF): An informative leaflet. Dublin: Irish Raynaud's & Scleroderma Society, 1998.

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Anderson, Marina Ellen. Neurovascular control mechanisms in primary Raynaud's phenomenon and systemic sclerosis. Manchester: University of Manchester, 1995.

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Baxter, James A. Raynaud's phenomenon (white finger): A summary of the occupational health concern. Hamilton, Ont: Canadian Centre for Occupational Health and Safety, 1989.

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Edwards, Clare M. The roles of central neural and local factors in primary Raynaud's disease. Birmingham: University of Birmingham, 1998.

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Parker, Philip M., and James N. Parker. Raynaud's disease: A medical dictionary, bibliography, and annotated research guide to Internet references. San Diego, CA: ICON Health Publications, 2004.

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Book chapters on the topic "Raynaud's"

1

Freedman, Robert R. "Raynaud's disease." In Encyclopedia of psychology, Vol. 7., 9–10. Washington: American Psychological Association, 2000. http://dx.doi.org/10.1037/10522-004.

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Grayson, Peter C., and Peter A. Merkel. "Raynaud's Phenomenon." In Atlas of Clinical Vascular Medicine, 34–35. Oxford, UK: Blackwell Publishing Ltd., 2013. http://dx.doi.org/10.1002/9781118618189.ch17.

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Devulder, Jacques, Hans van Suijlekom, Robert Van Dongen, Sudhir Diwan, Nagy Mekhail, Maarten van Kleef, and Frank J. P. M. Huygen. "Ischemic Pain in the Extremities and Raynaud's Phenomenon." In Evidence-Based Interventional Pain Medicine, 196–201. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781119968375.ch25.

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Franks, A. G. "Topical glyceryl trinitrate as adjunctive treatment in Raynaud's disease." In Nitroglycerin 5, edited by Bodo E. Strauer, 47–52. Berlin, Boston: De Gruyter, 1987. http://dx.doi.org/10.1515/9783110898262-007.

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Moneta, Gregory L., and Gregory J. Landry. "Vasospastic Disease of the Upper Extremity: Primary Raynaud's Syndrome." In Haimovici's Vascular Surgery, 947–61. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118481370.ch75.

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Pierro, Luisa, Claudia Del Turco, Elisabetta Miserocchi, and Francesca Ingegnoli. "Choroidal Changes in Patients with Raynaud's Phenomenon Secondary to a Connective Tissue Disease: Study of Vascular Eye Involvement in Patients Affected by Raynaud's Phenomenon with in vivo Noninvasive EDI-OCT." In Optical Coherence Tomography, 127–30. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000355973.

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Fava, Andrea, and Francesco Boin. "Historical Perspective of Raynaud’s Phenomenon." In Raynaud’s Phenomenon, 1–11. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1526-2_1.

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Anderson, Marina, and Michael Hughes. "Other Secondary Causes." In Raynaud’s Phenomenon, 141–62. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1526-2_10.

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McMahan, Zsuzsanna H., and Julie J. Paik. "Raynaud’s Mimics." In Raynaud’s Phenomenon, 163–85. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1526-2_11.

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Cutolo, Maurizio, and Vanessa Smith. "Nailfold Capillaroscopy." In Raynaud’s Phenomenon, 187–97. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1526-2_12.

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Conference papers on the topic "Raynaud's"

1

Wilkinson, D., P. Vowden, A. B. Latif, S. M. Rajah, and R. C. Kester. "EICOSANOIDS AND VASOSPASM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643385.

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To investigate the hypothesis that abnormalities in prostanoid metabolism may be an aetiological factor in Raynaud's syndrome (RS) we have measured the stable metabolites 6-keto-prostaglandin F1α (6-keto-PGF1α ) and thromboxane B2 (TXB2), using a radioimmunoassay. The table gives their levels (in ng/ml) and ratio (mean±SD) in healthy volunteers and in a group of patients with RS.In the control group there was no significant correlation between prostanoid levels and age, sex or smoking habit. In primary Raynaud's TXB2- levels and the ratio tend to be elevated from our control values. Patients with Vibration White Finger Disease (VWF) have a similar eicosanoid profile to that of the primary Raynaud's group with elevated TXB2 levels. One patient in this group had markedly raised 6-keto-PGF1α and TXB2 levels and later proved to have a circulating auto-antibody. Menstruating females in our study showed both elevated TXB and ratios similar to the primary group. It is interesting to note that menstruating females have an increased sensitivity to cold with abnormal cold stress tests. Patients with Raynaud's syndrome secondary to a systemic disorder have markedly elevated 6-keto-PGF1α and TXB2 levels although the ratio of the two remains within our normal range. There is a significant difference between secondary Raynaud's and all other groups investigated in the study (Mann-Whitney U test p = 0.04). Further research into prostanoid metabolism may yield a greater understanding of the pathophysiology of Raynaud's syndrome. Therapy may best be aimed at altering the levels of these important local hormones.

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Gualtierotti, R., F. Ingegnoli, T. Schioppo, S. Griffini, E. Grovetti, MO Borghi, M. Cugno, and PL Meroni. "SAT0655 Early endothelial damage in patients with raynaud's phenomenon." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.5416.

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Kacprzak, Michal, A. Skora, J. Obidzinska, A. Zbiec, Roman Maniewski, and W. Staszkiewicz. "Thermal tests for laser Doppler perfusion measurements in Raynaud's syndrome." In SPIE Proceedings, edited by Antoni Nowakowski and Bogdan B. Kosmowski. SPIE, 2004. http://dx.doi.org/10.1117/12.577622.

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Rodríguez-Lozano, B., A. Alvarez, J. Bethencourt, A. Arteaga, M. Gantes, E. Trujillo, and T. González. "AB0114 Raynaud's phenomenon: correlation between clinical manifestations and nailfold capillary microscopy." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.262.

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Roon, AM Van, A. Eman Abdulle, AM van Roon, AJ Stel, AJ Smit, H. Bootsma, and DJ Mulder. "AB0610 Nailfold capillary microscopy and low body mass index in raynaud's phenomenon patients." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.3243.

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Hughes, M. "SP0015 New onset raynaud's phenomenon – could this be early limited cutaneous systemic sclerosis?" In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.7173.

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Coates, Anne C., John Mark, David Cornfield, and Carlos E. Milla. "Dyspnea In A Patient With Raynaud's Phenomenon: The Uncovering Of Interstitial Lung Disease (ILD)." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6475.

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Häme, A., K.-L. Vidqvist, P. Parmanne, A. Puhakka, T. Hasan, H. Mäkinen, J. Panelius, R. Peltomaa, M. Perovaara, and R. Luosujärvi. "AB0999 Finnish cohort of patients with raynaud's phenomenon-nailfold videokapillaroskopy findings and autoantibody values." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.1431.

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Kacmaz, Seydi, and Ergun Ercelebi. "The thermal imaging system design in the diagnosis and follow-up of Raynaud's Phenomenon." In 2018 2nd International Symposium on Multidisciplinary Studies and Innovative Technologies (ISMSIT). IEEE, 2018. http://dx.doi.org/10.1109/ismsit.2018.8567041.

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Viana, J. R., D. Campos, L. Ulbricht, G. Y. Sato, and W. L. Ripka. "Thermography for the detection of Secondary Raynaud's Phenomenon by means of the Distal-Dorsal Distance." In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9175870.

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Reports on the topic "Raynaud's"

1

Calcium channel blockers are useful in managing Raynaud’s phenomenon. National Institute for Health Research, February 2018. http://dx.doi.org/10.3310/signal-000556.

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