Relevant bibliographies by topics / Rats – Nervous system

Academic literature on the topic 'Rats – Nervous system'

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Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Rats – Nervous system"

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Levine, Seymour, Arthur Saltzman, and Alden Loud. "Inflammatory Siderosis of the Nervous System in Rats." Journal of Neuropathology and Experimental Neurology 48, no. 4 (July 1989): 391–98. http://dx.doi.org/10.1097/00005072-198907000-00001.

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Kuzmenko, N. V., M. G. Pliss, N. S. Rubanova, and V. A. Tsyrlin. "SPECULATIONS ОN BLOOD PRESSURE INCREASE MECHANISMS IN RENAL ARTERY CLIPPED WISTAR RATS." "Arterial’naya Gipertenziya" ("Arterial Hypertension") 19, no. 3 (June 28, 2013): 221–26. http://dx.doi.org/10.18705/1607-419x-2013-19-3-221-226.

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Objective.To examine the mechanisms underlying the activation of the sympathetic nervous system and blood pressure elevation in vasorenal hypertension in the male Wistar rats weighing 250–300 g.Design and methods.We observed the development of renovascular hypertension, beat-to-beat interval and heart rate variability in animals with intact renal nerves and denervated ischemic kidney for 8 weeks after renal artery clamping. Eight weeks later after renal artery clamping in hypertensive rats with denervated ischemic kidney, both-sided renal denervation was performed, and blood pressure was monitored for 6 weeks.Results.Although the ischemic kidney denervation reduces the activity of the sympathetic nervous system, it does not prevent renovascular hypertension development. However, both-sided renal denervation leads to the normalization of blood pressure in the rats with stable renovascular hypertension.Conclusion.We suggest that increased afferent fl ow from structural formations of the ischemic kidney plays an important role for the increased sympathetic nervous system activity.
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Arieli, R., and G. Hershko. "Prediction of central nervous system oxygen toxicity in rats." Journal of Applied Physiology 77, no. 4 (October 1, 1994): 1903–6. http://dx.doi.org/10.1152/jappl.1994.77.4.1903.

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Cumulative O2 toxicity (K) can be calculated using the expression K = t2 x PO2c, where t is exposure time and the power c is to be determined; the phenomenon is liable to occur when K reaches Kc, the threshold value of K at which a symptom is manifested. Six rats were each exposed six times to 6 ATA O2 at 2-day intervals until the first electrical discharge (FED) was noted in an electroencephalogram. There was no difference in latency to FED in the series of six exposures. Thirteen rats were exposed to O2 until FED was noted in an electroencephalogram. They were exposed to four constant PO2's of 5, 6, 7, and 8 ATA and to two combined profiles of 1) 5 min at 7 ATA followed by 5 ATA and 2) 15 min at 5 ATA followed by 7 ATA. The solution of the equation for each rat was used to predict its latency to FED on the combined profile. The correlation of predicted to measured latency was significant (P < 0.0001), and the slope was not different from 1. Solving for these parameters using the combination of all the data, we obtained Kc = 5.71 x 10(6) and c = 5.39, which correctly predicted the mean latency but failed to predict individual latency. It is preferable to use each rat as its own control. The significance of the correlation supports the validity of the power equation for calculating K.
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Giuliano, Francois, Jacques Bernabe, Alain Jardin, and Jean Paul Rousseau. "Antierectile Role of the Sympathetic Nervous System in Rats." Journal of Urology 150, no. 2 Part 1 (August 1993): 519–24. http://dx.doi.org/10.1016/s0022-5347(17)35539-8.

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Hamidi, Mehrdad. "Central nervous system distribution kinetics of indinavir in rats." Journal of Pharmacy and Pharmacology 59, no. 8 (August 2007): 1077–85. http://dx.doi.org/10.1211/jpp.59.8.0004.

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Parham, D., A. Tereba, P. J. Talbot, D. P. Jackson, and V. L. Morris. "Analysis of JHM Central Nervous System Infections in Rats." Archives of Neurology 43, no. 7 (July 1, 1986): 702–8. http://dx.doi.org/10.1001/archneur.1986.00520070058019.

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Bamshad, Maryam, Victor T. Aoki, M. Gregory Adkison, Wade S. Warren, and Timothy J. Bartness. "Central nervous system origins of the sympathetic nervous system outflow to white adipose tissue." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 275, no. 1 (July 1, 1998): R291—R299. http://dx.doi.org/10.1152/ajpregu.1998.275.1.r291.

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White adipose tissue (WAT) is innervated by postganglionic sympathetic nervous system (SNS) neurons, suggesting that lipid mobilization could be regulated by the SNS [T. G. Youngstrom and T. J. Bartness. Am. J. Physiol. 268 ( Regulatory Integrative Comp. Physiol. 37): R744–R751, 1995]. A viral transsynaptic retrograde tract tracer, the pseudorabies virus (PRV), was used to identify the origins of the SNS outflow from the brain to WAT neuroanatomically. PRV was injected into epididymal or inguinal WAT (EWAT and IWAT, respectively) of Siberian hamsters and IWAT of rats. PRV-infected neurons were visualized by immunocytochemistry and found in the spinal cord, brain stem (medulla, nucleus of the solitary tract, caudal raphe nucleus, C1 and A5 regions), midbrain (central gray), and several areas within the forebrain. The general pattern of infection of WAT in both species was more similar than different and resembled that seen after PRV injections into the adrenal medulla in rats (A. M. Strack, W. B. Sawyer, J. H. Hughes, K. B. Platt, and A. D. Loewy. Brain Res. 491: 156–162, 1989). EWAT versus IWAT injected hamsters had relatively less labeling in the suprachiasmatic, dorsomedial, and arcuate nuclei. Overall, it appeared that the SNS innervation of WAT originates from the general SNS outflow of the central nervous system and therefore may play a significant role in lipid mobilization.
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Song, Jian-gang, Hong-hai Li, Yun-fei Cao, Xin Lv, Ping Zhang, Ye-sheng Li, Yong-jun Zheng, et al. "Electroacupuncture Improves Survival in Rats with Lethal Endotoxemia via the Autonomic Nervous System." Anesthesiology 116, no. 2 (February 1, 2012): 406–14. http://dx.doi.org/10.1097/aln.0b013e3182426ebd.

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Background Recent advances have indicated a complex interplay between the autonomic nervous system and the innate immune system. Targeting neural networks for the treatment of sepsis is being developed as a therapeutic strategy. Because electroacupuncture at select acupoints can modulate activities of the autonomic nervous system, we tested the hypothesis that electroacupuncture at specific acupoints could modulate systemic inflammatory responses and improve survival via its impact on the autonomic nervous system in a rat model of sepsis. Methods Sprague-Dawley male rats received electroacupuncture for 45 min before and at 1, 2, or 4 h after a lethal dose of intraperitoneal lipopolysaccharide injection (6 mg/kg). Outcomes included survival and systemic cytokine responses. Also, the possible roles of neural circuitry, including the hypothalamic-pituitary-adrenal axis and the autonomic nervous system, were evaluated. Results Electroacupuncture pretreatment at the Hegu acupoints significantly attenuate systemic inflammatory responses and improve survival rate from 20% to 80% in rats with lethal endotoxemia. Such a site-specific effect requires the activation of muscarinic receptors in the central nervous system, but not increasing central sympathetic tone. In the periphery synergistic, rather than independent, action of the sympathetic and parasympathetic systems is also necessary. Conclusions Electroacupuncture pretreatment has a dramatic survival-enhancing effect in rats with lethal endotoxemia, which involves the activation of efferent neural circuits of the autonomic nervous system (e.g., cholinergic antiinflammatory pathway). This approach could be developed as a prophylactic treatment for sepsis or perioperative conditions related to excessive inflammation.
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Huang, Zhihui, Yiqiao Wang, Meiling Chen, and Prof Lei Sha. "ODP228 Obesity Induced a Down-Regulation of nNOS in Intrapancreatic Nervous System." Journal of the Endocrine Society 6, Supplement_1 (November 1, 2022): A327—A328. http://dx.doi.org/10.1210/jendso/bvac150.679.

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Abstract Objective Obesity has become a serious global public health problem. Obesity is frequently preceding and the most important risk factor of type 2 diabetes. Pancreas regulates glucose metabolism and glucose homeostasis, and abnormality of pancreas play an important role in insulin resistance and diabetes. Intrapancreatic nerve innervation modulates insulin release and is tightly linked to the onset and development of diabetes. However, it is not known whether the activity of the intrapancreatic nervous system has been altered before the onset of diabetes. In this study, we examined the intrapancreatic nervous system in rats with obesity looking for the changes induced by obesity. Methods Obese Sprague-Dawley rats were developed with feeding high fat diet for 10 weeks. Using Immunofluorescence method, choline acetyl transferase (ChAT) and nNOS positive neurons in intrapancreatic ganglia were counted. ChAT and nNOS expression in pancreas were also measured with western blot method. Results (1) There was no significant difference in fasting blood glucose between obese rats and normal rats, but oral glucose tolerance test showed that the oral glucose tolerance was impaired in obese rats. (2) In rats fed with normal diet, 80.2%±1.5 neurons in intrapancreatic ganglia were ChAT positive, and in the obese rats, 79.3%±1.8 neurons were ChAT positive. There was no significant difference between the two groups (p&gt;0. 05, n=5). In intrapancreatic ganglia of obese rats, 40%±1.4 neurons were nNOS positive, which was significantly lower than that (60.5%±1.2) of rats fed with normal diet (p&lt;0. 01,n=5). (3) In pancreatic tissue, the relative expression level of ChAT protein was 1. 02±0. 03 in rats fed with normal diet and 0.98±0. 05 in obese rats (p&gt;0. 05, n=5). The relative expression level of nNOS protein was 1.70±0. 08 in normal rats and 1.10±0. 05 in obese rats (p&gt;0. 01, n=5). Conclusion The cholinergic nerves in intrapancreatic nervous system was not altered in obese rats, however, nNOS nerves was down-regulated in obese rats. The down-regulation of nNOS would induce a hyperactivity in intrapancreatic nervous system and this would cause an enhancement of insulin release, which has been observed in obese individuals. Presentation: No date and time listed
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Fisher, L. A., C. R. Cave, and M. R. Brown. "Central nervous system cardiovascular effects of bombesin in conscious rats." American Journal of Physiology-Heart and Circulatory Physiology 248, no. 4 (April 1, 1985): H425—H431. http://dx.doi.org/10.1152/ajpheart.1985.248.4.h425.

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The effects of intracerebroventricular administration of bombesin on mean arterial pressure and heart rate were studied in conscious, freely moving rats. Injection of bombesin produced dose-dependent elevations of mean arterial pressure and reductions of heart rate. These effects were not caused by leakage of bombesin into the peripheral circulation. Adrenalectomy abolished the pressor action of bombesin but did not alter bombesin-induced bradycardia. Systemic phentolamine pretreatment prevented bombesin-induced changes of mean arterial pressure, whereas rats treated intravenously with captopril or a vasopressin antagonist still exhibited pressor responses to bombesin administration. Bombesin-induced bradycardia was partially antagonized by intravenous atropine methyl nitrate administration, whereas systemic injections of propranolol did not modify this response. It is concluded that bombesin acts within the central nervous system to elevate mean arterial pressure through an adrenal-dependent mechanism involving alpha-adrenergic receptors and to reduce heart rate through an adrenal-independent mechanism involving, at least in part, cardiac parasympathetic nervous activation.
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Dissertations / Theses on the topic "Rats – Nervous system"

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Chai, Hong. "Survival and regeneration of spinal motoneuron after ventral root avulsion in adult rat /." Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk/hkuto/record.jsp?

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袁秋菊 and Qiuju Yuan. "Effects of neurotrophic factors on motoneuron survival following axonal injury in developing rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B42128705.

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Sevilla, Elenita L. "The influence of 5-HT3 receptor antagonism on aspects of CNS activity in morphine-dependent rats." Thesis, [Hong Kong] : University of Hong Kong, 1991. http://sunzi.lib.hku.hk/hkuto/record.jsp?B13019211.

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Yuan, Qiuju. "Effects of neurotrophic factors on motoneuron survival following axonal injury in developing rats." Click to view the E-thesis via HKUTO, 2001. http://sunzi.lib.hku.hk/hkuto/record/B42128705.

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Hooper, Justin Shane. "Cardiovascular Effects Evoked by Airway Nociceptive Reflexes in Healthy and Cardiovascular Diseased Rats." Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6258.

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Acute inhalation of airborne pollutants alters cardiovascular function and has been shown to have its greatest affects on individuals with pre-existing cardiovascular disease. Evidence suggests that pollutant-induced activation of airway sensory nerves via the gating of ion channels is critical to these systemic responses. Here, we have investigated the cardiovascular responses evoked by inhalation of AITC (TRPA1 agonist) and capsaicin (TRPV1 agonist) in healthy Sprague Dawley (SD) and Wistar Kyoto (WKY) rats, and cardiovascular diseased Spontaneously Hypertensive (SH) rats. Inhalation of the agonists by healthy SD and WKY rats caused significant bradycardia, atrio-ventricular (AV) block and prolonged PR-Intervals. Inhalation of TRP agonists caused differential cardiovascular responses in the cardiovascular diseased SH rats, such that the TRP agonists evoked brady-tachy with AV block and premature ventricular contractions (PVCs). Bradycardic responses to AITC were inhibited by the TRP channel blocker ruthenium red and the muscarinic antagonist atropine, but atropine did not prevent the tachycardic responses seen in the SH rats. Adrenergic inhibition with atenolol prevented the tachycardic responses, but did not prevent the bradycardic responses evoked by AITC in the SH rats. In healthy rats, AITC inhalation also caused a biphasic blood pressure response: a brief hypertensive phase followed by a hypotensive phase, while evoking hypertension in the SH rats. Atropine accentuated the hypertensive phase in all animals, while preventing the hypotension in the healthy animals. In all animals, AITC-evoked heart rate responses were not abolished by terazosin, the [U+F061]1 adrenoceptor inhibitor, which prevented the hypertensive responses. Anesthetics had profound effects on AITC-evoked bradycardia and AV block, which was abolished by urethane, ketamine and isoflurane. Nevertheless, AITC inhalation caused bradycardia and AV block in paralyzed and ventilated rats following pre-collicular decerebration. In conclusion, we provide evidence that activation of TRP channels expressed on nociceptive airway sensory nerves causes significant cardiovascular effects in healthy rats via reflex modulation of the autonomic nervous system (ANS), and that these effects are exacerbated in cardiovascular diseased rats.
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Lau, Chi-yan Jane. "Brain derived neurotrophic factors (BDNF) and seprafilm?adhesion barrier on sciatic nerve regeneration in rats." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B42924789.

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Silva, Kelly Regina Torres da. "Estudo da distribuição das proteínas relacionadas às teneurinas no sistema nervoso central de primatas não-humanos (Sapajus spp) e ratos (Rattus norvegicus)." Botucatu, 2016. http://hdl.handle.net/11449/139450.

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Orientador: Cláudio Aparecido Casatti
Resumo: As teneurinas (TENs) representam uma família de proteínas transmembrana preservada entre as espécies, presente principalmente no sistema nervoso central (SNC). Nos vertebrados essa família é composta por quatro homólogos, denominados de teneurina-1 a -4 (Ten-1, Ten-2, Ten-3 e Ten-4). Estudos mostraram a presença das TENs em vias motoras, olfatórias e visuais, especialmente durante a neurogênese em aves e roedores. A análise da distribuição neuroanatômica das TENs em primatas poderia ampliar o conhecimento destas proteínas, contribuindo com achados funcionais recentes. Portanto, os propósitos deste estudo foram: 1) avaliar a distribuição dos neurônios que exibem imunorreatividade relacionada às TENs-“like immunoreactivity” (TENs-LI), em particular Ten-2-LI, Ten-3-LI e Ten-4-LI no SNC do primata não-humano (Sapajus spp); 2) realizar análise comparativa dos sítios de distribuição da proteína Ten-3 entre o SNC de primatas (Sapajus spp) e roedores (Rattus norvegicus), uma vez que a Ten-3 apresentou distribuição significante no SNC de primatas; 3) correlacionar a distribuição das TENs com seus ligantes endógenos denominados de latrofilinas (LPHNs-1, 2 e 3) em áreas do SNC de primatas. Para isso, cortes coronais do SNC de macacos (n=3) e de ratos (n=4) foram submetidos à técnica de imuno-histoquímica e analisados em microscopia de luz ou confocal. Os resultados demonstraram a distribuição de neurônios e fibras nervosas exibindo TENs-LI em todo o neuroeixo de primatas. Neurônios e... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Teneurins (TENs) represent a transmembrane protein family preserved along animal species, mainly in the central nervous system (CNS). This protein family is constituted by four homologues, named as teneurin 1 to 4 (Ten-1, Ten-2, Ten-3 and Ten-4). Previous studies pointed out presence of TENs in motor, olfactory and visual systems in chicken and rodents, especially during neurogenesis. The neuroanatomic distribution analysis of TENs in the primate brain could provide additional information on this protein system, as well as support functional data from recent studies. Therefore, the purposes of the present study were: 1) to evaluate the distribution of neurons exhibiting TENs-like immunoreactivity (TENs-LI), in particular, Ten- 2-LI, Ten-3-LI and Ten-4-LI in the CNS of non-human primates (Sapajus spp); 2) to comparatively analyze the main brain regions exhibiting Ten-3-LI between primates (Sapajus spp) and rodents (Rattus norvegicus), since Ten-3-LI showed significant distribution in the CNS of primates; 3) To correlate TENsLI neurons with latrophilins (LPHNs-1, 2 and 3), an endogenous TENs ligand, in the CNS of primates. For this purpose, coronal histological sections of the CNS of non-human primates (n=3) and rats (n=4) were submitted to immunohistochemistry techniques and analyzed under light or confocal microscopes. Neurons and nerve fibers exhibiting TENs-LI were observed in all parts of the CNS in primates. Neurons showing Ten-2-LI were present mainly in the brainstem, s... (Complete abstract click electronic access below)
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Häidkind, Riina. "Monoaminergic mechanisms in mood-associated behaviours and neurochemistry in rats /." Tartu : Tartu University Press, 2004. http://dspace.utlib.ee/dspace/bitstream/10062/704/5/Haidkind.pdf.

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柴宏 and Hong Chai. "Survival and regeneration of spinal motoneuron after ventral root avulsion in adult rat." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B3124158X.

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Lau, Chi-yan Jane, and 劉至欣. "Brain derived neurotrophic factors (BDNF) and seprafilm® adhesion barrier on sciatic nerve regeneration in rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B42924789.

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Books on the topic "Rats – Nervous system"

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S, Ashwell Ken W., and Törk Istvan, eds. Atlas of the developing rat nervous system. 2nd ed. San Diego: Academic Press, 1994.

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J, Hay, and Lawson S. J, eds. The rat nervous system: An introduction to preparatory techniques. Chichester: J. Wiley, 1997.

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Swanson, Larry W. Brain maps: Structure of the rat brain. Amsterdam: Elsevier, 1992.

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Swanson, Larry W. Brain maps: Structure of the rat brain : a laboratory guide with printed and electronic templates for data, models, and schematics. 2nd ed. Amsterdam: Elsevier, 1998.

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Alvarez-Bolado, G. Developmental brain maps: Structure of the embryonic rat brain. New York: Elsevier, 1996.

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The projections to the spinal cord of the rat during development: A time-table of descent. Berlin: Springer, 1997.

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Sukhov, A. G. Neĭronnai͡a︡ organizat͡s︡ii͡a︡ taktilʹnogo analizatora krysy. Rostov-na-Donu: Izd-vo Rostovskogo universiteta, 1992.

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M, Tohyama, Takatsuji Koichi, and Kantha Sachi Sri, eds. Atlas of neuroactive substances and their receptors in the rat. Oxford: Oxford University Press, 1997.

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1956-, Oudega Martin, ed. Development of the rat spinal cord: Immuno- and enzyme histochemical approaches. Berlin: Springer-Verlag, 1993.

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Brain maps III: Structure of the rat brain : an atlas with printed and electronic templates for data, models, and schematics. 3rd ed. Amsterdam: Elsevier, Academic Press, 2004.

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Book chapters on the topic "Rats – Nervous system"

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Krinke, Georg J. "Spontaneous Radiculoneuropathy, Aged Rats." In Nervous System, 203–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83516-2_35.

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Powell, Henry C. "Cycloleucine Encephalopathy, Rats and Mice." In Nervous System, 77–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83516-2_13.

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Hyashi, Yuzo, Akihiko Maekawa, and Shinsuki Yoshimura. "Neurotoxicity of Perhexiline Maleate in Rats." In Nervous System, 74–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83516-2_12.

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Hayashi, Yuzo, Michihito Takahashi, and Toshiaki Hasegawa. "Systemic Degenerative Disorders of Primary Sensory Neurons in Rats Induced by 4-Hydroxyaminoquinoline 1-Oxide." In Nervous System, 92–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83516-2_16.

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Huang, Ming-Chao, and Yi-Lo Lin. "Kringle 1-5 Reduces Growth of Malignant Gliomas in Rats." In Tumors of the Central Nervous System, 25–34. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-7224-2_3.

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Shulman, R. G., D. L. Rothman, K. L. Behar, and J. W. Prichard. "High Resolution NMR Studies of Cerebral Glucose Metabolism in Rats and Humans." In Fuel Homeostasis and the Nervous System, 5–8. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-5931-9_2.

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Santucci, D., M. Luoni, A. Torsello, I. Branchi, E. E. Mùller, and E. Alleva. "Anti-Insulin-like Growth Factor I Antibodies Affect Locomotion and Passive Avoidance Performances in Sprague-Dawley Rats." In IGFs in the Nervous System, 135–44. Milano: Springer Milan, 1998. http://dx.doi.org/10.1007/978-88-470-2246-1_12.

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Pepeu, G., L. Giovannelli, M. G. Giovannini, and F. Pedata. "Effect of Phosphatidylserine on Cortical Acetylcholine Release and Calcium Uptake in Adult and Aging Rats." In Phospholipid Research and the Nervous System, 265–71. New York, NY: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4899-0490-4_26.

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Lattanzi, Davide, David Savelli, Michael Di Palma, Stefano Sartini, Silvia Eusebi, Dasiel O. Borroto-Escuela, Riccardo Cuppini, Kjell Fuxe, and Patrizia Ambrogini. "Electrophysiological Approach to GPCR–RTK Interaction Study in Hippocampus of Adult Rats." In Receptor-Receptor Interactions in the Central Nervous System, 71–90. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8576-0_6.

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Gorio, A., B. Tenconi, N. Zonta, P. Mantegazza, and A. M. Di Giulio. "Reactive Sprouting (Pruning Effect) is Altered in the Brain of Rats Perinatally Exposed to Morphine." In Plasticity and Regeneration of the Nervous System, 61–67. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-8047-4_7.

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Conference papers on the topic "Rats – Nervous system"

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Belaya, О. V. "EXPERIMENTAL STUDY OF 2-5G BASE STATION ELECTROMAGNETIC FIELD CHRONIC EXPOSURE NERVOUS SYSTEM EFFECT TYPOLOGICAL FEATURES." In The 4th «OCCUPATION and HEALTH» International Youth Forum (OHIYF-2022). FSBSI «IRIOH», 2022. http://dx.doi.org/10.31089/978-5-6042929-6-9-2022-1-20-24.

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Introduction: the new exposure to electromagnetic fields biological effect assessment is actual with respect to the expected of electromagnetic environment complication due to joint operation of various generation cellular communication systems. The character of biological reaction can be mediated by individual typological characteristics of the organism. The goal: to study the 2–5G base stations electromagnetic field chronic exposure effect to nervous system functional state of laboratory animals with regard to their typological features. Methods: the rats body weight registration and "open field" test were carried out before the start of exposure, after each month of 24-hour exposure (500 µW/cm2, 2-5G mobile standards, 4 months) and 1 month after the end of exposure with accounting of high-entropic and low-entropic animal subgroups. Results: the results indicate a multidirectional effect of rats’ nervous system in groups of various entropy types. According to behavioral parameters and body weight dynamics, there was an inhibition of research activity and functional state of the high-entropic animals after 2 months of exposure. Conclusion: Results can be assumed that used experimental exposure type had a suppression affect to central nervous system functional state with higher responsiveness of animals that initially have a predominance of excitation over inhibition processes.
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Levina, Anna, Nina Bondarenko, Natalia Shirуaeva, and Alexander Vaido. "DIVING BEHAVIOUR IN THE «EXTRAPOLATION ESCAPE TASK» IN RATS WITH DIFFERENT NERVOUS SYSTEM EXCITABILITY THRESHOLDS." In XV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2019. http://dx.doi.org/10.29003/m462.sudak.ns2019-15/266.

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Levina, Anna, Natalia Shiryaeva, Alexander Vaido, and Natalia Dyuzhikova. "LONG-TERM BEHAVIORAL EFFECTS OF CHRONIC STRESS DYNAMICS IN RATS WITH DIFFERENT NERVOUS SYSTEM EXCITABILITY THRESHOLDS." In XVIII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2022. http://dx.doi.org/10.29003/m2823.sudak.ns2022-18/206.

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Fil'kova, P., I. Shalaginova, and O. Tuchina. "The severity of post-stress neurodegeneration in rats with different levels of excitability of the nervous system." In ChemBioSeasons 2022. Kemerovo State University, 2022. http://dx.doi.org/10.21603/chembioseasons2022-39.

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Belova, Y. I., and O. V. Yakovleva. "Analysis of the content of nitric oxide (II) metabolites rats of different ages." In VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-27-29.

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Nitrogen monoxide is a gas transmitter that is an important intermediary in many organ systems, especially in the central nervous system. Nitrogen monoxide is involved in the relaxation of smooth vascular muscles, activation of neurons and responsible for the cytotoxicity of macrophages. The study of change nitrogen oxide metabolite concentration helps to determine its effects on human and animal organs. The study was carried out on laboratory animals of different ages. We used a spectrophotometric method to determine the level of metabolites based on the reaction of nitrites to the Griss reagent. We noted that the maximum level of metabolites NO was observed in newborn animals at the age of 4 days. In addition, metabolite concentrations decreased gradually by 14-15 days of life, reaching a minimum of 30 days Key words: nitrogen monoxide, rats, age, metabolites of nitrogen monoxide, spectrophotometry.
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Elkina, Anna, Stanislav V. Kozin, and Alexandr A. Kravtsov. "INFLUENCE OF DEUTERIUM-DEPLETED DRINKING DIET ON THE FUNCTIONAL STATE OF THE CENTRAL NERVOUS SYSTEM OF ANIMALS UNDER RATE AND NORMOBARIC HYPOXIA WITH HYPERCAPNIA." In NEW TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2021. http://dx.doi.org/10.47501/978-5-6044060-1-4.54.

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The effect of long-term (42 days) introduction of deuterium-depleted water into the diet of rats on the functional state of the central nervous system under normal conditions and normobaric hypoxia with hypercapnia was studied. It was also established that the use of deuterium-de-pleted water both under normal conditions and after stressful exposure contributes to a sig-nificant decrease in the emotional anxiety of animals. Long-term use of deuterium-depleted water before hypoxic exposure (exposure to the amnestic factor) contributes to the preservation of learning and memory at the control level, i.e. has a pronounced protective antiamnesic effect.
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Prokopova, Alena, Alena Gostyukhina, Tatyana Zamoshchina, Olga Doroshenko, Oksana Zhukova, and Konstantin Zaitsev. "BEHAVIORAL ACTIVITY AND LEVELS OF CORTICOSTERON IN THE SERUM OF LABORATORY RATS WITH DIFFERENT CENTRAL NERVOUS SYSTEM REACTIVITY AFTER LIGHT DESYNCHRONOSIS AND PHYSICAL OVERFATCH." In XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2285.sudak.ns2021-17/308-309.

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Ismailova, Khadidja, and Nigar Teymurova. "COMBINED EFFECTS OF HEAVY METALS AND ANTIDEPRESSANTS ON LEANING AND MEMORY PROCESSES IN RATS WITH DIFFERENT PHENOTYPES OF NERVOUS SYSTEM AND DIFFERENT ACTIVITY LEVELS OF BRAIN MONOAMINERGYC SYSTEM." In XVI International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2020. http://dx.doi.org/10.29003/m1068.sudak.ns2020-16/230-231.

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Shevchenko, A., I. Shalaginova, L. Mackova, and D. Kacerov. "The effect of stress on the intestinal microbiome of two lines of rats with a high and a low threshold of excitability of the nervous system." In ChemBioSeasons 2022. Kemerovo State University, 2022. http://dx.doi.org/10.21603/chembioseasons2022-40.

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Aguiar, Gabrielle Batista de, Bianca Villanova, Katriane Endiel Pereira, Nicole Jansen Rabello, Rafaela Maria Moresco, Marcelo Alves de Souza, and Lígia Aline Centenaro. "Motor and cognitive evaluations of rats exposed to the larvicide pyriproxifem in the prenatal period." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.083.

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Background: In 2015 and 2016, there was an increase in microcephaly cases in Brazil, which were attributed to maternal contamination by Zika virus. However, the spread of this virus in the Brazilian states was not accompanied by an increase in the registers of this malformation. Thus, the use of the larvicide pyriproxifem to the control of mosquito Aedes Aegypt is being questioned. Objectives: Investigate the motor and cognitive function of rats exposed to piriproxifem in prenatal period. Design and setting: The study was developed at the Universidade Estadual do Oeste do Paraná, in the Cascavel campus. Methods: Three experimental groups were used: Negative control (CT-) - offspring of rats which consumed water during pregnancy; Positive control (CT+) - offspring of rats which consumed water containing retinoic acid during pregnancy; Piriproxifem (PIR) - offspring of rats which consumed water containing Sumilarv®, larvicide whose active substance is the pyriproxifem, during pregnancy. The gait quality was assessed using the Basso, Beattie and Bresnahan scale (1995). Object-Placement Recognition Task was used to the spatial memory evaluation (Pizza et al., 2011). Results: There were no statistical differences between the groups in relation to the gait quality and the preference for the relocated object. Nevertheless, two animals in the PIR and five animals in the CT+ group had external rotation of the hindlimbs during locomotion or deficits in motor coordination. Conclusion: The mild motor deficits found indicate the need for studies to confirm a possible deleterious effect of this larvicide on the nervous system development.
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Reports on the topic "Rats – Nervous system"

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Zheng, Ruo-xiang, Jia-wei Xu, Bi-yao Jiang, Wei Tang, Chun-li Lu, Xiao-yang Hu, and Jian-ping Liu. Mind-body therapies in traditional Chinese medicine for neuropathic pain: a systematic review of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0016.

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Review question / Objective: The purpose of this review is to comprehensively evaluate the effectiveness and safety on mind-body therapies of traditional Chinese medicine for neuropathic pain. Condition being studied: According to the definition by the International Association for the Study of Pain (IASP), neuropathic pain is a kind of pain caused by lesions or diseases affecting the somatosensory nervous system. It has brought considerable negative impacts on patients and society. Neuropathic pain is a prevalent disease and can be induced by a variety of clinical conditions such as spinal cord injury (prevalence rate: 53%), induced peripheral neuropathic pain (prevalence rate: 38%), diabetic peripheral neuropathic pain (prevalence rate: 10%-26%), chemotherapy postherpetic neuralgia (3.9-42.0/10,000 people per year), prosopalgia (3-5/10,000 people per year), and so on. However, current recommended medicines for neuropathic pain management could cause dependence and adverse events. Thus, alternatives would be helpful for both patients and clinicians. Mind-body therapy in traditional Chinese medicine (TCM) has a long history in clinical practice for relieving pain and their effectiveness has not been systematically reviewed.The purpose of this review is to comprehensively evaluate the effectiveness and safety on mind-body therapies of traditional Chinese medicine for neuropathic pain.
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Wu, Xiaoqi, Maoxia Fan, Yaobo Pan, and Dona Guo. Quality of Evidence Supporting the Effects of Ginkgo Terpene Lactone Preparations in Ischemic Stroke: An Overview of Systematic Reviews and Meta-Analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0124.

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Review question / Objective: 2.2.1 Type of studies SRs/MAs of Randomized Controlled Trials (RCTs) of GTLP for IS in any language. 2.2.2 Type of Participants Included patients were diagnosed with IS according to international or national standards, regardless of race, age, gender, time of onset, and source of cases. 2.2.3 Type of Intervention The intervention method in the control group was routine treatment, and the intervention method in the intervention group was GTLP treatment or GTLP combined with the treatment of the control group. 2.2.4 Types of outcomes Conclusions at least need to include clinical efficacy analysis and National Institute of Health Stroke Scale (NIHSS). Condition being studied: Stroke is the second leading cause of death and third leading cause of disability globally.Among them, ischemic stroke (IS) accounts for 70% of all stroke types. It is a central nervous system disease caused by cerebral blood circulation disorder, ischemia and hypoxia .The incidence rate is high and increasing year by year, the age of onset is younger, the disability rate is high, and most patients have different degrees of limb motor dysfunction.In order to reduce the burden of stroke on the society and the patient's family, many articles proposed to strengthen the primary stroke prevention - behavior change and drug intervention.
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