Relevant bibliographies by topics / Rats Diseases

Academic literature on the topic 'Rats Diseases'

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Published: 4 June 2021
Last updated: 30 January 2023

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Journal articles on the topic "Rats Diseases"

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Benato, Livia. "Respiratory diseases in rats." Companion Animal 17, no. 4 (May 2012): 47–50. http://dx.doi.org/10.1111/j.2044-3862.2012.00163.x.

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Rao, Ghanta N. "Diet and Kidney Diseases in Rats." Toxicologic Pathology 30, no. 6 (October 2002): 651–56. http://dx.doi.org/10.1080/01926230290166733.

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Middleton, Charles C. "Infectious Diseases of Mice and Rats. The Committee on Infectious Diseases of Mice and Rats.Companion Guide to Infectious Diseases of Mice and Rats. The Committee on Infectious Diseases of Mice and Rats." Quarterly Review of Biology 67, no. 2 (June 1992): 219. http://dx.doi.org/10.1086/417602.

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Clement, Jan, James LeDuc, Graham Lloyd, Jean-Marc Reynes, Lorraine McElhinney, Marc Van Ranst, and Ho-Wang Lee. "Wild Rats, Laboratory Rats, Pet Rats: Global Seoul Hantavirus Disease Revisited." Viruses 11, no. 7 (July 17, 2019): 652. http://dx.doi.org/10.3390/v11070652.

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Recent reports from Europe and the USA described Seoul orthohantavirus infection in pet rats and their breeders/owners, suggesting the potential emergence of a “new” public health problem. Wild and laboratory rat-induced Seoul infections have, however, been described since the early eighties, due to the omnipresence of the rodent reservoir, the brown rat Rattus norvegicus. Recent studies showed no fundamental differences between the pathogenicity and phylogeny of pet rat-induced Seoul orthohantaviruses and their formerly described wild or laboratory rat counterparts. The paucity of diagnosed Seoul virus-induced disease in the West is in striking contrast to the thousands of cases recorded since the 1980s in the Far East, particularly in China. This review of four continents (Asia, Europe, America, and Africa) puts this “emerging infection” into a historical perspective, concluding there is an urgent need for greater medical awareness of Seoul virus-induced human pathology in many parts of the world. Given the mostly milder and atypical clinical presentation, sometimes even with preserved normal kidney function, the importance of simple but repeated urine examination is stressed, since initial but transient proteinuria and microhematuria are rarely lacking.
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HART, BENJAMIN L., ELIZABETH K. KORINEK, and PATRICIA L. BRENNAN. "Postcopulatory Grooming in Male Rats Prevents Sexually Transmitted Diseases." Annals of the New York Academy of Sciences 525, no. 1 Neural Mechan (May 1988): 397–98. http://dx.doi.org/10.1111/j.1749-6632.1988.tb38624.x.

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Wang, Ruwen, Haili Tian, Dandan Guo, Qianqian Tian, Ting Yao, and Xingxing Kong. "Impacts of exercise intervention on various diseases in rats." Journal of Sport and Health Science 9, no. 3 (May 2020): 211–27. http://dx.doi.org/10.1016/j.jshs.2019.09.008.

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Casili, Giovanna, Alessio Ardizzone, Marika Lanza, Enrico Gugliandolo, Marco Portelli, Angela Militi, Salvatore Cuzzocrea, Emanuela Esposito, and Irene Paterniti. "Treatment with Luteolin Improves Lipopolysaccharide-Induced Periodontal Diseases in Rats." Biomedicines 8, no. 10 (October 21, 2020): 442. http://dx.doi.org/10.3390/biomedicines8100442.

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Periodontitis is a dental disease that produces the progressive destruction of the bone surrounding the tooth. Especially, lipopolysaccharide (LPS) is involved in the deterioration of the alveolar bone, inducing the release of pro-inflammatory mediators, which cause periodontal tissue inflammation. Luteolin (Lut), a molecule of natural origin present in a large variety of fruits and vegetables, possess beneficial properties for human health. On this basis, we investigated the anti-inflammatory properties of Lut in a model of periodontitis induced by LPS in rats. Animal model predicted a single intragingival injection of LPS (10 μg/μL) derived from Salmonella typhimurium. Lut administration, was performed daily at different doses (10, 30, and 100 mg/kg, orally), starting from 1 h after the injection of LPS. After 14 days, the animals were sacrificed, and their gums were processed for biochemical analysis and histological examinations. Results showed that Lut (30 and 100 mg/kg) was equally able to reduce alveolar bone loss, tissue damage, and neutrophilic infiltration. Moreover, Lut treatment reduced the concentration of collagen fibers, mast cells degranulation, and NF-κB activation, as well as the presence of pro-inflammatory enzymes and cytokines. Therefore, Lut implementation could represent valid support in the pharmacological strategy for periodontitis, thus improving the well-being of the oral cavity.
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ABRAMSKY, O., E. WERTMAN, A. RECHES, T. BRENNER, and H. OVADIA. "Effect of Hypothalamic Lesions on Experimental Autoimmune Diseases in Rats." Annals of the New York Academy of Sciences 496, no. 1 Neuroimmune I (May 1987): 360–65. http://dx.doi.org/10.1111/j.1749-6632.1987.tb35788.x.

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SASA, Hiroaki, Yukiya HASHIMOTO, Takako SHIMIZU, and Ken-ichi INUI. "Hepatic Extraction of Tacrolimus in Rats with Experimental Liver Diseases." Biological & Pharmaceutical Bulletin 21, no. 6 (1998): 610–14. http://dx.doi.org/10.1248/bpb.21.610.

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Cintra, Luciano Tavares Angelo, Aguinaldo Cândido da Silva Facundo, Mariane Maffei Azuma, Dóris Hissako Sumida, Rafael Dias Astolphi, Suely Regina Mogami Bomfim, Luís Gustavo Narciso, and João Eduardo Gomes-Filho. "Pulpal and periodontal diseases increase triglyceride levels in diabetic rats." Clinical Oral Investigations 17, no. 6 (October 5, 2012): 1595–99. http://dx.doi.org/10.1007/s00784-012-0853-7.

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Dissertations / Theses on the topic "Rats Diseases"

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Simmons, Joe H. "Rat respiratory virus (RRV) and other novel rodent diseases /." free to MU campus, to others for purchase, 2001. http://wwwlib.umi.com/cr/mo/fullcit?p3025651.

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Dahlman, Ingrid. "Genetic dissection of experimental autoimmune neuroinflammatory diseases in rats /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3768-0/.

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Jacobs, Carvern Denver. "The effect of Cyclopia maculata on AMPK expression in Wistar rats." Thesis, University of the Western Cape, 2012. http://hdl.handle.net/11394/4043.

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>Magister Scientiae - MSc
Being overweight or obese are major factors contributing to the increased morbidity and mortality due to non-communicable diseases such as type 2 diabetes, cardiovascular disease and cancer. The treatment of obesity with pharmaceutical drugs is plagued by side effects. Plants and their phytochemicals possess a number of beneficial health effects including anti-oxidant,anti-mutagenic, anti-inflammatory, anti-obesity and anti-cancer effects, mediated by activation of the adenosine monophosphate protein kinase (AMPK).AMPK controls many metabolic processes including glucose uptake and utilisation, and adipogenesis, and is often referred to as the master regulator establishing cellular homeostasis.Cyclopia maculata, commonly known as honeybush, is an indigenous South Africa plant possessing anti-oxidant, anti-inflammatory and anti-cancer properties. Recently, others in our laboratory have shown that a hot water extract of fermented C. maculata inhibits adipocyte differentiation in 3T3-L1 pre-adipocytes, with some evidence of weight regulatory properties in a Wistar rat model of diet-induced obesity. In the rat study, 21 day old weanlings were fed a high fat, high sugar cafeteria diet for 3 months with (n=10) or without (n=10) C. maculata supplementation. This group of rats was referred to as the lean group (n=20). Another group of rats were fed a cafeteria diet for 4 months to induce obesity (obese group, n=20) and thereafter treated as described for the lean rats. The aim of this MSc study was to determine whether C. maculata induces AMPK activation.Proteins were extracted from the liver and muscle tissue of lean and obese Wistar rats using an optimized extraction method with a commercial lysis buffer and the TissueLyser.Treatment with the C. maculata extract had no effect on the protein yield in lean and obese rats. Interestingly, the protein yield in the liver of obese rats was significantly higher than that observed in lean rats. Although C. maculata treatment slightly increased AMPK activation (calculated as the ratio of phosphorylated AMPK to total AMPK) in the liver of lean and obese rats, the difference was not statistical significant. Conversely, C.maculata treatment decreased AMPK activity in muscle of lean and obese rats, with statistical significance observed in the lean group only (2.3-fold, p<0.05). Differences in AMPK activation between the groups were also noted, a 1.3-fold decreased activity observed in obese groups compared to their lean counterparts, although this was not statistically significant. Expression of PPARα, a downstream protein target affected by AMPK activation was reduced in the liver of lean and obese rats after C. maculata treatment. Moreover, PPARα expression was significantly higher in obese compared to lean rats (2.7-fold, p<0.001). PPARα is a transcription factor mediating fat metabolism (β-oxidation) and its expression is induced by circulating free fatty acids, which are increased in obese compared to lean rats. The expression of PPARα in muscle was too low for Western blot analysis and quantification.Cyclopia maculata treatment did not affect hepatic expression of UCP2, another protein important in establishing energy homeostasis. The expression of UCP2 was 2.9-fold higher in the liver of obese rats compared to their lean counterparts, although the difference was not statistically significant. The opposite results were observed in the muscle where C. maculata treatment decreased UCP2 expression in lean rats (2.8-fold,p<0.0001), and UCP2 expression was decreased 1.4-fold in obese rats compared to lean rats, although the difference was not statistically significant.ELISA results for AMPK activation revealed that C. maculata treatment increased AMPK activity, although not statistically significant. Histological analysis of retroperitoneal fat showed that C. maculata did not affect adipocyte size and number, although a slight decrease in adipocyte size was observed after treatment .This study has demonstrated that treatment of the cafeteria diet fed Wistar rats with 300 mg/kg of a hot water extract of fermented C. maculata does activate AMPK. This study revealed important differences between lean and obese rats. In particular, increased hepatic protein content, PPARα and UCP2 expression was observed in obese rats compared to the lean group. This suggests an adaptive response to the increased circulating free fatty acids during obesity and an increase in β-oxidation in these animals.
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Hair-Bejo, Mohd. "Gastrointestinal response to copper excess : studies on copper (and zinc) loaded rats." Thesis, University of Liverpool, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.303681.

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高加信 and Ka-shun Joshua Ko. "The mechanisms of adaptive cytoprotection against ethanol-induced gastric mucosal damage in rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1995. http://hub.hku.hk/bib/B31234185.

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何美美 and Mai-mai Ho. "A study on the acute and chronic effects of morphine on rat stomachs." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1986. http://hub.hku.hk/bib/B31230611.

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Manirafasha, Claudine. "The effects of kolaviron on epididymal and testicular function in streptozotocin induced diabetic wistar rats." Thesis, Cape Peninsula University of Technology, 2014. http://hdl.handle.net/20.500.11838/1508.

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Thesis submitted in fulfilment of the requirements for the degree Master of Technology: Biomedical Technology in the Faculty of Health and Wellness Sciences at the Cape Peninsula University Of Technology 2014
Oxidative stress (OS) plays a central role in the progression of diabetes mellitus (DM). Prevention of DM and its complications is a challenging health problem as it impacts on various organ functions, including reproduction. Diabetes mellitus with hyperglycaemic condition generates high production of reactive oxygen species. An imbalance between antioxidant mechanism and reactive oxygen species generates oxidative stress. OS damages the sperm membrane by oxidation of polyunsaturated fats which in turn reduces the sperm motility and ability to fuse with the oocyte and OS directly damage sperm DNA, compromising the paternal genomic contribution to the embryo development. Recent experimental evidence shows that modulation of oxidative stress and natural antioxidants may determine the outcome of male reproductive function. Previous investigations indicate that the supplementation and treatment with phytomedicine might play role in the prevention and management of DM and its subsequent complications on male reproductive function. This study explored the pharmacological potential of kolaviron (KV) on testicular and epididymal tissue in diabetic and non- diabetic Wistar rats. All experiments were conducted for a period of six weeks. Male Wistar rats (240–290 g) were randomly divided into 5 groups (n=12) where all the rats received a standard diet. Non diabetic rats control group and other four groups injected with different treatments. Non diabetic rat (N) received vehicle: Dimethylsulfoxide. Diabetes rats (D) were induced by a single intraperitoneal injection of freshly prepared streptozotocin (STZ) solution, 50mg/kg body weight. The N and D were treated with kolaviron (100 mg/kg body weight) orally, five times a week .The last group, diabetic rats were given subcutaneously injection of the standard anti-diabetic drug, insulin (0.2 u/kg) every second day. After the feeding period, testicular and epididymal tissues were collected and were analysed. All parameters were determined using appropriate methods in homogenized tissues. Data were expressed as mean ± SD. Plasma glucose as well as malondialdehyde (MDA) was significantly higher, while body, testicular and epididymal weights were lower in the D group compared to the N group and N+KV. Both kolaviron and insulin were able to ameliorate these effects. Testicular and epididymal antioxidant enzymes such as superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GPx) in induced diabetic rats were significantly (p<0.05) low compared to diabetic control group. However, KV treated group shown significantly higher SOD, CAT and GPx activities compared D group. In conclusion, our findings demonstrated that KV could improve antioxidant enzymes and modulate STZ induced diabetic related oxidative stress in the male reproductive system. Kolaviron can potentially be used as an anti-diabetic treatment, however further studies are needed. Key words: Oxidative stress, Diabetes mellitus, antioxidants, kolaviron, epididymal tissue, testicular tissue, superoxide dismutase, catalase, glutathione peroxidase, lipid peroxidation, streptozotocin
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Gomes, Débora Aline Silva [UNESP]. "Influência do estado diabético na doença periodontal induzida em ratos. Análise bioquímica, microscópica, radiográfica e dos níveis de mieloperoxidade." Universidade Estadual Paulista (UNESP), 2006. http://hdl.handle.net/11449/96207.

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Made available in DSpace on 2014-06-11T19:28:03Z (GMT). No. of bitstreams: 0 Previous issue date: 2006-02-17Bitstream added on 2014-06-13T19:47:54Z : No. of bitstreams: 1 gomes_das_me_arafo.pdf: 2513950 bytes, checksum: 8cd7796b57e48e98aeb2eb5aa9a87b0d (MD5)
Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Universidade Estadual Paulista (UNESP)
Evidências sugerem existir correlação positiva entre diabetes mellitus e destruição periodontal. Com intuito de estudar a influência do Diabetes Mellitus sobre a evolução da doença periodontal induzida, o presente estudo apresenta como objetivos, avaliar o peso corporal, os níveis séricos de cálcio, fósforo e fosfatase alcalina, os níveis teciduais de mieloperoxidase, além de, análise macroscópica e radiográfica dos espécimes de cada animal. Foram utilizados 128 ratos machos Wistar divididos em 4 grupos de 32 ratos, sendo: Grupo I controle; Grupo II diabético; Grupo III controle com doença periodontal induzida e Grupo IV diabético com doença periodontal induzida. Após dois dias da confirmação do estado diabético induzido por estreptozotocina, foi realizada a colocação da ligadura. Oito animais de cada grupo foram sacrificados nos períodos experimentais de 3, 7, 15 e 30 dias após colocação da ligadura. Os resultados demonstraram que o estado diabético perdurou até o final do experimento nos grupos II e IV. Dentre os marcadores bioquímicos, somente a ALP apresentou-se estatisticamente maior nos grupos diabéticos (II e IV). Macroscopicamente, houve diferença somente entre os grupos com e sem doença periodontal, independente da presença do diabetes, com maior alteração tecidual nos períodos tardios de periodontite (30 dias). Os níveis gengivais de MPO foram estatisticamente maiores (p<0.05) nos grupos com doença periodontal induzida (III e IV). Radiograficamente, o grupo IV apresentou maior perda óssea estatisticamente significante quando comparada aos demais grupos (p<0,05). Nos limites deste estudo, pode-se concluir que a doença periodontal induzida não alterou o estado hiperglicêmico, no entanto, sugere-se que o diabetes acentue...
Evidences suggest the existence of a positive correlation between diabetes mellitus and periodontal collapse. In order to evaluate the influence of Diabetes Mellitus on the progression of periodontal disease induced in rats, the objective of the present study was to verify glycemic, calcium, phosphorus and alkaline phosphatase serum levels, to evaluate myeloperoxidase (MPO) gingival levels, as well as to carry out macroscopic and radiographic assessment of the animals' hemimandíbulas of experimental periodontitis. A hundred twenty-eight Wistar male rats were used in this study. They were divided into 4 groups of 32 rats each, as follows: Group I - control; Group II - diabetic; Group III - control with periodontal disease induced; and Group IV - diabetic with periodontal disease induced. After streptozotocin diabetic state was confirmed, a ligature was placed on the mandibular first molar teeth of Groups III and IV rats. Eight animals of each group were killed at the experimental periods of 3, 7, 15 and 30 days after the ligature placement. Results demonstrated that the diabetic state lasted up to the end of the experiment at groups II and IV. Significant increases in serum alkaline phosphatase were observed at diabetic groups (II and IV). Macroscopically, in the groups where periodontal disease was induced, it was possible to observe more tissue alterations and it was higher in the 30-day experimental period. The MPO levels were significantly higher in induced periodontitis groups (III and IV) (p<0.05). Radiographically, diabetic with experimental periodontitis group (IV) showed statistically higher bone loss in all the experimental periods (p<0.05). It is possible to conclude that induced periodontal disease did not change the diabetic state... (Complete abstract, click electronic address below)
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Lam, Kai-Yee. "A study on the role of probiotic lactobacillus rhamnosus GG on gastric mucosal damages in rats /." View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36432891.

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Lam, Kai-yee. "A study on the role of probiotic lactobacillus rhamnosus GG on gastric mucosal damages in rats." Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B37340050.

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Books on the topic "Rats Diseases"

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Tucker, Mary J. Diseases of the Wistar rat. London: Taylor & Francis, 1997.

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Daly, Carol Himsel. Rats: All about selection, husbandry, nutrition, breeding, and diseases, with a special chapter on understanding rats. Hauppauge, NY: Barron's, 1991.

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Amemiya, Tsugio. Retinal and Choroidal Vascular Changes and Systemic Diseases in Rats. Tokyo: Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-68394-0.

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Voipio, Hanna-Marja. Hiiren ja rotan tartuntataudit. Kuopio: Valtakunnallinen koe-eläinkeskus, Kuopion yliopisto, 1988.

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1946-, Stinson Sherman F., Schuller Hildegard M, and Reznik Gerd, eds. Atlas of tumor pathology of the Fischer rat. Boca Raton, Fla: CRC Press, 1990.

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L, Lushnikova E., Nepomni͡ashchikh Galina Ivanovna, and Borodin I͡U I, eds. Morfometrii͡a i stereologii͡a gipertrofii serdt͡sa. Novosibirsk: Izd-vo "Nauka," Sibirskoe otd-nie, 1986.

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Seaman, William J. Postmortem change in the rat: A histologic characterization. Ames: Iowa State University Press, 1987.

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Kibenge, Molly J. Twinomwe. The relationship between the hypothalamo-pituitary-adrenal axis and the development and persistence of pancreatic islet defects in obese zucker (fa/fa) rats. Charlottetown: University of Prince Edward Island, 1994.

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Topurii͡a, Zaza Mikhaĭlovich. Respiratornyĭ trakt, aėrogematicheskiĭ barʹer v ontogeneze i pri patologii. Tbilisi: "Met͡sniereba", 1990.

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Amemiya, T. Retinal and choroidal vascular changes and systemic diseases in rats: Corrosion cast and scanning electron microscopy. New York: Springer, 2003.

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Book chapters on the topic "Rats Diseases"

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Pardue, Machelle T., Moon K. Kim, Tiffany A. Walker, Amanda E. Faulkner, Alan Y. Chow, and Vincent T. Ciavatta. "Neuroprotective Dose Response in RCS Rats Implanted with Microphotodiode Arrays." In Retinal Degenerative Diseases, 115–20. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-0631-0_16.

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Kean, Edward L., Tamao Endo, Naiqian Niu, Daniel T. Organisciak, Yuji Sato, and Akira Kobata. "Structures of the Oligosaccharides of Rhodopsin from Normal and Rcs Rats." In Degenerative Retinal Diseases, 107–14. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5933-7_14.

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Zhao, Tong Tao, Chun Yu Tian, and Zheng Qin Yin. "Activation of Müller Cells Occurs During Retinal Degeneration in RCS Rats." In Retinal Degenerative Diseases, 575–83. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-1399-9_66.

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Nagahiro, Shinji, and Keiko Kitazato. "Experimental Model of Cerebral Aneurysms in Ovariectomized Rats." In Mouse Models of Vascular Diseases, 243–59. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55813-2_13.

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Strauß, O., and M. Wienrich. "Retinal Pigment Epithelial Cells Cultured From RCS Rats Express an Increased Membrane Conductance for Calcium Compared to Normal Rats." In Degenerative Diseases of the Retina, 119–27. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1897-6_14.

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Qu, Chao, Wei Cao, Yingchuan Fan, and Ying Lin. "Near-Infrared Light Protect the Photoreceptor from Light-Induced Damage in Rats." In Retinal Degenerative Diseases, 365–74. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-1399-9_42.

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Taurog, Joel D., and Robert E. Hammer. "Inflammatory Disease in HLA-B27 Transgenic Rats." In Genetic Models of Immune and Inflammatory Diseases, 44–50. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-2376-4_5.

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El-Hifnawi, E., H. J. Hettlich, and C. Falk. "Retinal Degeneration in Rats Induced by Vitamin E Deficiency." In Degenerative Diseases of the Retina, 111–18. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1897-6_13.

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McLaren, Margaret J. "Defective Choroidal Angiogenesis Precedes Retinal Pigment Epithelial Phagocytic Defect in Neonatal Rcs Rats." In Degenerative Retinal Diseases, 121–34. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5933-7_16.

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Tamai, M., K. Yamada, N. Takeda, H. Tomita, T. Abe, S. Kojima, and S. I. Ishiguro. "bFGF Transfected Iris Pigment Epithelial Cells Rescue Photoreceptor Cell Degeneration in RCS Rats." In Degenerative Retinal Diseases, 323–28. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5933-7_36.

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Conference papers on the topic "Rats Diseases"

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Woo, D. G., Q. J. Lee, W. P. Park, C. Y. Ko, D. Lim, H. S. Kim, and B. Y. Lee. "Relationship Between Obesity and Osteopenia in Lumbar Spines of Rats Using Mechanical and Morphological Studies." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192537.

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Obesity (OB) and osteopenia (OP), grave consequences for human health, quality of life, and even the efficiency of the labor force and economy, are two common complex diseases. Two public health problems have exploded in prevalence over the past decade [1]. OB, now a major epidemic in the developed world and frequent among elderly subjects, is a condition of excessive body fat that causes or exacerbates several risk of developing non-insulin dependent diabetes mellitus, cardiovascular disease, cancer and other diseases [2]. OP is defined as a systemic skeletal disease caused by low bone mass and microstructural deterioration of the bone.
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Sarker, Roma Rani, Yasuyuki Tsunoi, Yasue Haruyama, Shunichi Sato, and Izumi Nishidate. "Antimicrobial photodynamic therapy for burn wound infection with P. aeruginosa in rats." In Photonic Diagnosis, Monitoring, Prevention, and Treatment of Infections and Inflammatory Diseases 2021, edited by Tianhong Dai, Mei X. Wu, and Jürgen Popp. SPIE, 2021. http://dx.doi.org/10.1117/12.2578087.

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Assis, Lívia, Carla Tim, Cintia Martignago, Silma Gonçalves, and Ana Claudia Renno. "Effectiveness of photobiomodulation therapy and aerobic exercise training on articular cartilage in an experimental model of osteoarthritis in rats." In Photonic Diagnosis and Treatment of Infections and Inflammatory Diseases, edited by Tianhong Dai. SPIE, 2018. http://dx.doi.org/10.1117/12.2291227.

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Seidel, MF, MP Junier, and H. Vetter. "THU0028 Different variants of tnf-alpha mrna transcripts are expressed in rats with collagen-induced arthritis." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.825.

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Bradunaite, R., E. Bernotiene, L. Leonaviciene, and D. Vaitkiene. "THU0078 Antiarthritic effect of salbutamol alone and in combination with pentoxyphiline on adjuvant arthritis in rats." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.955.

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Feige, U., C. King, YL Hu, A. Koch, D. Duryea, and B. Bolon. "OP0091 Differences in disease expression in adjuvant arthritis of lewis rats induced by mycobacteria or lipoidal amine." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.99.

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Bernotiene, E., R. Bradunaite, L. Leonaviciene, V. Astrauskas, A. Venalis, D. Vaitkiene, and A. Valickas. "SAT0217 Experimental arthritis in lewis rats induced by repeated injections of yersinia enterocolitica o:8 in different doses." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.720.

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Feige, U., A. Koch, and B. Bolon. "THU0083 Bone mineral density measurements are suitable for rapid quantification of skeletal destruction in rats with adjuvant arthritis." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.960.

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Alrafiah, Aziza. "Thymoquinoneon protects neurons in the cerebellum of rats through mitigates oxidative stress and inflammation following high fat diet supplementation." In 1st International Electronic Conference on Biomolecules: Natural and Bio-Inspired Therapeutics for Human Diseases. Basel, Switzerland: MDPI, 2020. http://dx.doi.org/10.3390/iecbm2020-08566.

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Atkinson, JE, RV House, PS Cranmer, AE Colucil, H. Davis, CK Edwards, and DM Miller. "THU0096 Effect of anakinra (il-1ra) and soluble tumour necrosis factor receptor i (stnf-ri) on cellular immune function in rats." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.973.

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Reports on the topic "Rats Diseases"

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Navi, Shrishal. Evaluation of Application Rates of Fungicides on Soybean Diseases and Yield. Ames: Iowa State University, Digital Repository, 2015. http://dx.doi.org/10.31274/farmprogressreports-180814-349.

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Liang, R., D. Liu, HB Li, and ZG Zhai. The efficacy and safety of traditional Chinese medicine formulas in the treatment of chronic obstructive pulmonary disease complicated with pulmonary hypertension: a systematic review and meta-analysis study. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0041.

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Review question / Objective: This systematic review and meta-analysis was intended to evaluate the efficacy and safety of traditional Chinese medicine(TCM) formulas in the treatment of chronic obstructive pulmonary disease(COPD) complicated with pulmonaryhypertension (PH). Condition being studied: Chronic obstructive pulmonary disease(COPD) complicated with pulmonary hypertension(PH) is classified as the third group PH.According to epidemiology, the most common cause of PH associated with lung diseases and/or hypoxia is COPD, but the prevalence rate of COPD with PH range from 20% to 91% variously. In China, many TCM formulas are regularly used in COPD patients , thus TCM formulas therapy is worth considering.
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Newman-Toker, David E., Susan M. Peterson, Shervin Badihian, Ahmed Hassoon, Najlla Nassery, Donna Parizadeh, Lisa M. Wilson, et al. Diagnostic Errors in the Emergency Department: A Systematic Review. Agency for Healthcare Research and Quality (AHRQ), December 2022. http://dx.doi.org/10.23970/ahrqepccer258.

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Objectives. Diagnostic errors are a known patient safety concern across all clinical settings, including the emergency department (ED). We conducted a systematic review to determine the most frequent diseases and clinical presentations associated with diagnostic errors (and resulting harms) in the ED, measure error and harm frequency, as well as assess causal factors. Methods. We searched PubMed®, Cumulative Index to Nursing and Allied Health Literature (CINAHL®), and Embase® from January 2000 through September 2021. We included research studies and targeted grey literature reporting diagnostic errors or misdiagnosis-related harms in EDs in the United States or other developed countries with ED care deemed comparable by a technical expert panel. We applied standard definitions for diagnostic errors, misdiagnosis-related harms (adverse events), and serious harms (permanent disability or death). Preventability was determined by original study authors or differences in harms across groups. Two reviewers independently screened search results for eligibility; serially extracted data regarding common diseases, error/harm rates, and causes/risk factors; and independently assessed risk of bias of included studies. We synthesized results for each question and extrapolated U.S. estimates. We present 95 percent confidence intervals (CIs) or plausible range (PR) bounds, as appropriate. Results. We identified 19,127 citations and included 279 studies. The top 15 clinical conditions associated with serious misdiagnosis-related harms (accounting for 68% [95% CI 66 to 71] of serious harms) were (1) stroke, (2) myocardial infarction, (3) aortic aneurysm and dissection, (4) spinal cord compression and injury, (5) venous thromboembolism, (6/7 – tie) meningitis and encephalitis, (6/7 – tie) sepsis, (8) lung cancer, (9) traumatic brain injury and traumatic intracranial hemorrhage, (10) arterial thromboembolism, (11) spinal and intracranial abscess, (12) cardiac arrhythmia, (13) pneumonia, (14) gastrointestinal perforation and rupture, and (15) intestinal obstruction. Average disease-specific error rates ranged from 1.5 percent (myocardial infarction) to 56 percent (spinal abscess), with additional variation by clinical presentation (e.g., missed stroke average 17%, but 4% for weakness and 40% for dizziness/vertigo). There was also wide, superimposed variation by hospital (e.g., missed myocardial infarction 0% to 29% across hospitals within a single study). An estimated 5.7 percent (95% CI 4.4 to 7.1) of all ED visits had at least one diagnostic error. Estimated preventable adverse event rates were as follows: any harm severity (2.0%, 95% CI 1.0 to 3.6), any serious harms (0.3%, PR 0.1 to 0.7), and deaths (0.2%, PR 0.1 to 0.4). While most disease-specific error rates derived from mainly U.S.-based studies, overall error and harm rates were derived from three prospective studies conducted outside the United States (in Canada, Spain, and Switzerland, with combined n=1,758). If overall rates are generalizable to all U.S. ED visits (130 million, 95% CI 116 to 144), this would translate to 7.4 million (PR 5.1 to 10.2) ED diagnostic errors annually; 2.6 million (PR 1.1 to 5.2) diagnostic adverse events with preventable harms; and 371,000 (PR 142,000 to 909,000) serious misdiagnosis-related harms, including more than 100,000 permanent, high-severity disabilities and 250,000 deaths. Although errors were often multifactorial, 89 percent (95% CI 88 to 90) of diagnostic error malpractice claims involved failures of clinical decision-making or judgment, regardless of the underlying disease present. Key process failures were errors in diagnostic assessment, test ordering, and test interpretation. Most often these were attributed to inadequate knowledge, skills, or reasoning, particularly in “atypical” or otherwise subtle case presentations. Limitations included use of malpractice claims and incident reports for distribution of diseases leading to serious harms, reliance on a small number of non-U.S. studies for overall (disease-agnostic) diagnostic error and harm rates, and methodologic variability across studies in measuring disease-specific rates, determining preventability, and assessing causal factors. Conclusions. Although estimated ED error rates are low (and comparable to those found in other clinical settings), the number of patients potentially impacted is large. Not all diagnostic errors or harms are preventable, but wide variability in diagnostic error rates across diseases, symptoms, and hospitals suggests improvement is possible. With 130 million U.S. ED visits, estimated rates for diagnostic error (5.7%), misdiagnosis-related harms (2.0%), and serious misdiagnosis-related harms (0.3%) could translate to more than 7 million errors, 2.5 million harms, and 350,000 patients suffering potentially preventable permanent disability or death. Over two-thirds of serious harms are attributable to just 15 diseases and linked to cognitive errors, particularly in cases with “atypical” manifestations. Scalable solutions to enhance bedside diagnostic processes are needed, and these should target the most commonly misdiagnosed clinical presentations of key diseases causing serious harms. New studies should confirm overall rates are representative of current U.S.-based ED practice and focus on identified evidence gaps (errors among common diseases with lower-severity harms, pediatric ED errors and harms, dynamic systems factors such as overcrowding, and false positives). Policy changes to consider based on this review include: (1) standardizing measurement and research results reporting to maximize comparability of measures of diagnostic error and misdiagnosis-related harms; (2) creating a National Diagnostic Performance Dashboard to track performance; and (3) using multiple policy levers (e.g., research funding, public accountability, payment reforms) to facilitate the rapid development and deployment of solutions to address this critically important patient safety concern.
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Jin, Dachuan, Gao Peng, Shunqin Jin, Tao Zhou, Baoqiang Guo, and Guangming Li. Comparison of therapeutic effects of anti-diabetic drugs on non-alcoholic fatty liver disease patients without diabetes: A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0014.

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Review question / Objective: To evaluate the efficacy of different anti-diabetic drugs in the treatment of non-diabetic non-alcoholic disease by network meta-analysis, and find the best intervention. Condition being studied: Non-alcoholic fatty liver disease (NAFLD) refers to the disease in which the liver fat content exceeds 5%, and excludes the secondary causes of alcohol, infection, drugs or other specific metabolic diseases. As a spectrum of disorders, it includes hepatocyte steatosis and steatohepatitis at the initial stage, liver fibrosis at the later stage, cirrhosis at the final stage, and even liver cancer. Nowadays Non-alcoholic fatty liver disease (NAFLD) has become the most common chronic liver disease in the world with an incidence rate as high as 25% which has been rising steadily worldwide in the past 30 years. Currently there are still no approved specific therapeutic agents and global treatment guidelines for NAFLD. For non-diabetic NAFLD, there is far from a consensus, too.
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Gunderson, E. K., C. Garland, and L. L. Hourani. Infectious Disease Rates in the U.S. Navy, 1980-1995. Fort Belvoir, VA: Defense Technical Information Center, October 1999. http://dx.doi.org/10.21236/ada381292.

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Gunderson, E. K., Cedric Garland, and Laura L. Hourani. Infectious Disease Rates in the U.S. Navy, 1980 to 1995. Fort Belvoir, VA: Defense Technical Information Center, June 2001. http://dx.doi.org/10.21236/ada419514.

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Liu, Miao, Hongan Wang, Jing Lu, Zhiyue Zhu, Chaoqun Song, Ye Tian, Xinzhi Chen, et al. Vitamin D supplementation in the treatment of Myasthenia Gravis A protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0129.

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Review question / Objective: The patients should meet the internationally recognized diagnostic criteria for myasthenia gravis and be definitely diagnosed as myasthenia gravis, excluding MG patients caused by congenital, drug and other factors, as well as patients with serious primary diseases, autoimmune diseases or mental diseases. Patients are not restricted by race, region, gender, age, background, course of disease and other factors. We will focus on trials using vitamin D as an intervention at any dose and in any regimen (eg daily/weekly/monthly intake). The control group was routinely given western medicine, including cholinesterase inhibitors, glucocorticoids, immunosuppressants, alone or in combination, or placebo. The intervention group was treated with vitamin D on the basis of western medicine treatment in the control group. The specific dosage form and dose were not limited, and the shortest course of treatment should be 4 weeks. Main outcome measures: (1) Quantitative score of myasthenia gravis (QMG); (2) Recurrence rate; (3) Effective. Secondary outcome measures: (1) The level of serum acetylcholine receptor antibody (AchRab); (2) The levels of inflammatory factors such as IL-6 and IL-10; (3) Clinical absolute score; (4) TCM syndrome score scale; (5) Quality of life score (QOL); (6) Incidence rate of adverse events. All randomized controlled trials (RCT) literatures from the establishment to September 2022 were retrieved and classified.
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Li, Yanhui. Efficacy of non-invasive photodynamic therapy for female lower reproductive tract diseases associated with HPV infection: a comprehensive meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0092.

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Review question / Objective: The critical point of this study was to comprehensively evaluate the curative effect of Photodynamic therapy (PDT) in diseases of female lower reproductive tract associated with the human papillomavirus (HPV) infection. Condition being studied: Traditional clinical recommendations for treating diseases of the female lower reproductive tract include topical therapy with drugs, surgery, intravaginal radiation, carbon dioxide (CO2) laser, etc. Although medication is easy to administer, it has a high recurrence rate and adverse effects such as burning sensation, pain, and dyspareunia. The other traditional treatment method is usually invasive, repeated operation of vaginal perforation, scar, easy recurrence, fertility decline, and other shortcomings. At present, the treatment strategy for cervical squamous intraepithelial lesion, vaginal squamous intraepithelial lesion, condyloma acuminatum, and vulvar lichen sclerosis are to protect the normal organ structure and function as much as possible, reduce recurrence, prevent disease progression and carcinogenesis, and preserve female reproductive function.
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Shtienberg, Dan, William Fry, Amos Dinoor, Thomas Zitter, and Uzi Kafkafi. Reduction in Pesticide Use in Plant Disease Control by Integration of Chemical and Non-Chemical Factors. United States Department of Agriculture, May 1995. http://dx.doi.org/10.32747/1995.7613027.bard.

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The long term goal of this research project was to improve control efficiency of Alternaria diseases while reducing fungicide use, by integration of chemical and non-chemical factors. Non-chemical factors were genotype resistance, age-related resistance and fertilizers. The Specific objectives were: 1) To quantify changes in resistance among genotypes and over time in terms of disease development and specific phases of the disease cycle; 2) To quantify the effects of fertilizers applied to the foliage alone, or in combination with a fungicide, on disease development; 3) To quantify the relative contribution of genotype resistance, age-related resistance and fungicide type to the reduction of disease development; 4) To develop a strategy for integration of chemical and non-chemical factors which will achieve optimal disease suppression. The influence of physiological age of cotton plants and of the individual leaves, on disease incidence and on the rate of lesion expansion of A. macrospora was examined on leaves sampled from the field. Both parameters increased with the physiological age of individual leaves but were not affected by the age of the whole plant. The hypothesis that enrichment of the foliage with nitrogen and potassium may enhance host resistance to Alternaria and thus reduce disease severity, was examined for potato and tomato (A. solani ) and for cotton (A. macrospora ). Under controlled environment conditions, application of urea or KNO3 resulted in some reduction in disease development; however, foliar application of both nutrients (8-10 sprays in total) did not affect Alternaria severity in the field. Systemic fungicides against Alternaria (e.g. , tebuconazole and difenoconazole) are more effective than the commonly used protectant fungicides (e.g. mancozeb and chlorothalonil). Concepts for the integration of genotype resistance, age-related resistances and fungicide for the suppression of Alternaria diseases were developed and evaluated. It was found that reduction in host resistance, with age and among genotypes, can be compensated for by adjusting the intensity of fungicide applications, i.e. by increasing the frequency of sprays and by spraying systemic fungicides towards the end of the season. In, moderately resistant cultivars protection can be achieved by spraying at longer intervals than susceptible cultivars. The concepts for integration were evaluated in field trials for cotton, potatoes and tomatoes. By following these concepts it was possible to save up to five sprays out of 8-10 in a growing season.
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Yeates, Elissa, Kayla Cotterman, and Angela Rhodes. Hydrologic impacts on human health : El Niño Southern Oscillation and cholera. Engineer Research and Development Center (U.S.), January 2020. http://dx.doi.org/10.21079/11681/39483.

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A non-stationary climate imposes considerable challenges regarding potential public health concerns. The El Niño Southern Oscillation (ENSO) cycle, which occurs every 2 to 7 years, correlates positively with occurrences of the waterborne disease cholera. The warm sea surface temperatures and extreme weather associated with ENSO create optimal conditions for breeding the Vibrio cholerae pathogen and for human exposure to the pathogenic waters. This work explored the impacts of ENSO on cholera occurrence rates over the past 50 years by examining annual rates of suspected cholera cases per country in relation to ENSO Index values. This study provides a relationship indicating when hydrologic conditions are optimal for cholera growth, and presents a statistical approach to answer three questions: Are cholera outbreaks more likely to occur in an El Niño year? What other factors impact cholera outbreaks? How will the future climate impact cholera incidence rates as it relates to conditions found in ENSO? Cholera outbreaks from the 1960s to the present are examined focusing on regions of Central and South America, and southern Asia. By examining the predictive relationship between climate variability and cholera, we can draw conclusions about future vulnerability to cholera and other waterborne pathogenic diseases.
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