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Journal articles on the topic 'Pulmonary embolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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1

Nie, Yunqiang, Li Sun, Wei Long, Xin LV, Cuiyun Li, Hui Wang, Xing Li, Ping Han, and Miao Guo. "Clinical importance of the distribution of pulmonary artery embolism in acute pulmonary embolism." Journal of International Medical Research 49, no. 4 (April 2021): 030006052110047. http://dx.doi.org/10.1177/03000605211004769.

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Objective To explore the clinical importance of the distribution of pulmonary artery embolism in acute pulmonary embolism (APE). Methods Sixty-four patients with APE were classified into mixed-type and distal-type pulmonary embolism groups. Their right ventricular systolic pressure (RVSP) and disease duration were recorded, and the diameter of their right ventricles was measured by ultrasound. The computed tomography angiographic clot load was determined as a Mastora score. Results Patients with distal-type pulmonary embolisms had significantly lower RVSPs (44.92 ± 17.04 vs 55.69 ± 17.66 mmHg), and significantly smaller right ventricular diameters (21.08 ± 3.06 vs 23.37 ± 3.48 mm) than those with mixed-type pulmonary embolisms. Additionally, disease duration was significantly longer in patients with distal-type pulmonary embolisms (14.33 ± 11.57 vs 8.10 ± 7.10 days), and they had significantly lower Mastora scores (20.91% ± 18.92% vs 43.96% ± 18.30%) than patients with mixed-type pulmonary embolisms. After treatment, RVSPs decreased significantly in patients with both distal-type and mixed-type pulmonary embolisms. Right ventricle diameters also decreased significantly in patients with mixed-type pulmonary embolisms after treatment. Conclusion Patients with mixed-type pulmonary embolisms are significantly more susceptible to pulmonary hypertension, enlarged right ventricular diameters, and shorter durations of disease than those with distal-type pulmonary embolisms. The distribution of pulmonary artery embolism in APE can provide a clinical reference.

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2

K, Prabakar, and Dhruvanandan K. "Acute Pulmonary Embolism." JOURNAL OF CLINICAL AND BIOMEDICAL SCIENCES 11, no. 4 (December 15, 2021): 143–50. http://dx.doi.org/10.58739/jcbs/v11i4.2.

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3

Hannig, Kjartan Eskjaer, Steen Elkjaer Husted, and Erik Lerkevang Grove. "Cardiac Arrest Caused by Multiple Recurrent Pulmonary Embolism." Case Reports in Medicine 2011 (2011): 1–4. http://dx.doi.org/10.1155/2011/425090.

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Pulmonary embolism is a common condition with a high mortality. We describe a previously healthy 68-year-old male who suffered three pulmonary embolisms during a short period of time, including two embolisms while on anticoagulant treatment. This paper illustrates three important points. (1) The importance of optimal anticoagulant treatment in the prevention of pulmonary embolism reoccurrence. (2) The benefit of immediate accessibility to echocardiography in the handling of haemodynamically unstable patients with an unknown underlying cause. (3) Thrombolytic treatment should always be considered and may be life-saving in patients with cardiac arrest suspected to be caused by pulmonary embolism.

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4

Carpenter, Nicole. "Massive Pulmonary Embolism and Thrombolytic Therapy: Case Study." Journal of Diagnostic Medical Sonography 33, no. 3 (February 10, 2017): 232–38. http://dx.doi.org/10.1177/8756479317691271.

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Acute pulmonary embolism is the third most common acute cardiovascular disease, with about 600,000 cases annually in the United States. Pulmonary embolism requires a multimodality diagnosis and immediate treatment. Although computed tomography and ventilation perfusion scans are the most commonly used modalities to diagnose pulmonary embolisms, many supplemental tests are necessary. Treatment options for pulmonary embolism include anticoagulation therapy, thrombolytic therapy, or insertion of an inferior vena cava filter when anticoagulation is contraindicated. The long-term benefits of thrombolytic therapy have made it an increasingly popular option in many institutions. The following case study describes a patient who presented to the hospital with shortness of breath for five months and was found to have extensive pulmonary embolisms upon admission. The patient underwent three days of thrombolytic therapy that significantly reduced his pulmonary arterial pressures and resulted in an almost complete resolution of his pulmonary embolisms.

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5

Sohns, Jan M., Jan Menke, Leonard Bergau, Bernhard G. Weiss, Hannah Kröhn, Desiree Weiberg, Thorsten Derlin, and Sebastian Schmuck. "Screening of extravascular findings in pulmonary embolism computer tomography: 397 patients with 1950 non-pulmonary artery findings." Vascular 26, no. 1 (August 18, 2017): 99–110. http://dx.doi.org/10.1177/1708538117724628.

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Objectives The aim of this study was to investigate the possible benefits from computed tomography scans of patients with a suspected pulmonary artery embolism with a focus on relevant extravascular findings. Methods A total of 400 consecutive computed tomography pulmonary angiographies were evaluated. Computed tomography scans were analyzed in detail for the presence of pulmonary artery embolisms, as well as any other findings. Extra-artery discoveries were classified into none-relevant (Group A), intermediate (Group B), or relevant (Group C) findings. Results Aggregated computed tomography pulmonary angiographies detected other diagnosis than pulmonary artery embolism in 236 patients (59%). There were 1950 non-pulmonary artery embolism findings (4.9 per patient; n = 397). In the pulmonary artery embolism group, there were 447 extra-pulmonary artery embolism findings (5.2 per patient; n = 86) and in the non-pulmonary artery embolism group, 1503 findings (4.8 per patient; n = 311). Patients with pulmonary artery embolism had a significantly higher rate of pro-coagulate risk factors ( p < 0.001). Conclusions Computed tomography pulmonary angiographies may help to identify further diagnoses. This study represents a retrospective review of a single center experience for incidental computed tomography findings during pulmonary artery embolism work-up and emphasizes the importance of analyzing the whole field-of-view.

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6

Dhingra, Jitesh. "Pulmonary Embolism: Emergency Physician’s Nightmare." Journal of Medical Science And clinical Research 05, no. 06 (June 19, 2017): 23590–94. http://dx.doi.org/10.18535/jmscr/v5i6.130.

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7

Umairi, Rashid AL, Khadija AL Adawi, Maryam AL Khoori, Ahmed AL Lawati, and Sachin Jose. "COVID-19-Associated Thrombotic Complication: Is It Pulmonary Embolism or In Situ Thrombosis?" Radiology Research and Practice 2023 (July 3, 2023): 1–4. http://dx.doi.org/10.1155/2023/3844069.

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Objectives. Acute pulmonary embolism is a protentional fatal complication of COVID-19. The aim of this study is to investigate whether pulmonary embolism is due to thrombus migration from the venous circulation to the pulmonary arteries or due to local thrombus formation secondary to local inflammation. This was determined by looking at the distribution of pulmonary embolism in relation to lung parenchymal changes in patients with COVID-19 pneumonia. Methods. Retrospectively, we identified pulmonary computed tomography angiography (CTPA) of patients admitted to the Royal Hospital between November 1st, 2020, and October 31, 2021, with a confirmed diagnosis of COVID-19. The CTPAs were examined for the presence of pulmonary embolism and the distribution of the pulmonary embolism in relation with lung parenchymal changes. Results. A total of 215 patients admitted with COVID-19 pneumonia had CTPA. Out of them, 64 patients had pulmonary embolisms (45 men and 19 women; mean age: 58.4 years with a range of 36–98 years). The prevalence of pulmonary embolism (PE) was 29.8% (64/215). Pulmonary embolism was more frequently seen in the lower lobes. 51 patients had PE within the diseased lung parenchyma and 13 patients had PE within normal lung parenchyma. Conclusion. The strong association between pulmonary artery embolism and lung parenchymal changes in patients admitted with COVID-19 pneumonia suggests local thrombus formation.

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8

Houtzager, Tessa, Ingvar Berg, Thijs Urlings, and Robert Grauss. "Concomitant pulmonary embolism and upper limb ischaemia as a first presentation of a patent foramen ovale." BMJ Case Reports 14, no. 10 (October 2021): e242351. http://dx.doi.org/10.1136/bcr-2021-242351.

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A 78-year-old female patient presented to the emergency department with syncope and dyspnoea. The left arm appeared to be cold and radial pulse was not palpable. A CT scan of the chest and left arm with intravenous contrast displayed bilateral central pulmonary embolisms in combination with a left subclavian artery embolism and an atrial septal aneurysm. Transthoracic echocardiography identified a patent foramen ovale with right-to-left shunting confirming the diagnosis of paradoxical embolism. The patient was treated with anticoagulants. In a patient presenting with a combination of a pulmonary embolism and a peripheral arterial embolism, the clinician should consider a right-to-left shunt with paradoxical embolism. In line with this, when diagnosing a peripheral arterial embolism, a central venous origin should be considered. Furthermore, when diagnosing a pulmonary embolism or other forms of venous thromboembolism, the clinician should be aware of signs of a peripheral arterial embolism.

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9

He, Yanling, Yi Xiao, Yanping Chen, and Zhidong Li. "Renal Embolism Associated with Foramen Ovale Coexisting Acute Pulmonary Embolism." Case Reports in Pulmonology 2023 (December 6, 2023): 1–4. http://dx.doi.org/10.1155/2023/6670080.

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We report a singular case of renal embolism in a hitherto healthy 46-year-old female. The patient initially presented with symptoms of exertional distress and chest discomfort. Following an extensive diagnostic workup, she was subsequently diagnosed with acute pulmonary embolism. On the day succeeding her admission, the patient manifested sustained abdominal discomfort. Abdominal computed tomography angiography (CTA) subsequently revealed the presence of renal artery embolisms and infarctions. Concurrently, an echocardiographic evaluation disclosed a patent foramen ovale (PFO) and pulmonary hypertension. In this specific case, we hypothesize that the embolic event traversed through the PFO, ultimately localizing in the renal artery and culminating in renal embolism.

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10

Falkenstern-Ge, Roger, Kim Husemann, and Martin Kohlhäufl. "Late onset of pulmonary cement embolism after a regular vertebroplasy. A clinical documentation." Open Medicine 8, no. 5 (October 1, 2013): 662–64. http://dx.doi.org/10.2478/s11536-013-0207-0.

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AbstractVertebroplasty is a new minimal-invasive procedure for the treatment of painful vertebral fractures. The risk of a pulmonary embolism ranges from 3.5 to 23% for osteoporotic fractures. However, data about the incidence and treatment strategies of pulmonary cement embolisms (PCE) are limited. We report a case of a patient with symptomatic pulmonary cement embolism after the vertebroplasty. The diagnosis was confirmed by means of CT- scan. In cases of asymptomatic patients with peripheral PCE we recommend no treatment besides clinical follow-up. In our case of symptomatic embolisms, we recommend to proceed according to the guidelines regarding the treatment of thrombotic pulmonary embolisms, which includes initial heparinization and a following 6-month coumarin therapy.

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11

Lasa-Berasain, Pablo, Mario García Parra, Javier Ruíz Lucea, Joaquín Lobo Palanco, and Pilar Anguiano Baquero. "Pulmonary embolism and patent foramen ovale, augmented risk for paradoxical embolism?" International Journal of Case Reports and Images 13, no. 2 (November 2, 2022): 168–70. http://dx.doi.org/10.5348/101353z01pl2022ci.

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12

Wei Tan, Jia, Jay Parekh, and Anant Shukla. "Bradyarrhythmia in acute massive pulmonary embolism." International Journal of Case Reports and Images 14, no. 1 (February 28, 2023): 47–51. http://dx.doi.org/10.5348/101381z01jt2023cr.

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Pulmonary embolism (PE) can present with a variety of electrocardiographic findings. Bradycardia is a rare finding in acute PE, which typically manifests with sinus tachycardia. Bradycardia in acute PE might arise from the physiologic Bezold–Jarisch reflex, which describes the constellation of bradycardia, peripheral vasodilation, and hypotension. We report the case of a woman in her 60s who was admitted initially for submassive PE and found to have sinus bradycardia. She had progressed to massive PE with acute worsening of atrioventricular conduction block in the setting of atrial flutter. Her bradyarrhythmia resolved and hemodynamics improved after catheter directed thrombolysis. This case revisits the pathophysiology of Bezold–Jarisch reflex, and the importance of recognizing that it signifies an underlying pathologic insult, especially a life-threatening one like PE.

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13

Gong, Xiaowei, Boyun Yuan, and Yadong Yuan. "Incidence and prognostic value of pulmonary embolism in COVID-19: A systematic review and meta-analysis." PLOS ONE 17, no. 3 (March 14, 2022): e0263580. http://dx.doi.org/10.1371/journal.pone.0263580.

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Background Pulmonary embolisms are frequently and prognostically in individuals infected by coronavirus disease 2019 (COVID-19); the incidence of pulmonary embolisms is varied across numerous studies. This study aimed to assess the pooled incidence of pulmonary embolic events and the prognostic value of such events in intensive care unit (ICU) admissions of patients with COVID-19. Methods The Cochrane Library, PubMed, and EmBase were systematically searched for eligible studies published on or before October 20, 2021. The pooled incidence of pulmonary embolism was calculated using the random-effects model. Moreover, the prognostic value was assessed by measuring the sensitivity, specificity, positive and negative likelihood ratio (PLR and NLR), diagnostic odds ratio (DOR), and the area under the receiver operating characteristic curve (AUC). Results Thirty-six studies involving 10,367 COVID-19 patients were selected for the final meta-analysis. The cumulative incidence of pulmonary embolism in patients with COVID-19 was 21% (95% confidence interval [95%CI]: 18−24%; P<0.001), and the incidence of pulmonary embolism in ICU and non-ICU patients was 26% (95%CI: 22−31%; P<0.001) and 17% (95%CI: 14−20%; P<0.001), respectively. The predictive role of pulmonary embolism in ICU admission was also assessed, and the sensitivity, specificity, PLR, NLR, DOR, and AUC were 0.31 (95%CI: 0.21−0.42), 0.84 (95%CI: 0.75−0.90), 1.88 (95%CI: 1.45−2.45), 0.83 (95%CI: 0.75−0.91), 2.25 (95%CI: 1.64−3.08), and 0.61 (95%CI: 0.57−0.65), respectively. Conclusion This study found that the incidence of pulmonary embolism was relatively high in COVID-19 patients, and the incidence of pulmonary embolism in ICU patients was higher than that in non-ICU patients.

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14

Doğan, Kâmil, Turab Selçuk, and Ahmet Alkan. "An Enhanced Mask R-CNN Approach for Pulmonary Embolism Detection and Segmentation." Diagnostics 14, no. 11 (May 26, 2024): 1102. http://dx.doi.org/10.3390/diagnostics14111102.

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Pulmonary embolism (PE) refers to the occlusion of pulmonary arteries by blood clots, posing a mortality risk of approximately 30%. The detection of pulmonary embolism within segmental arteries presents greater challenges compared with larger arteries and is frequently overlooked. In this study, we developed a computational method to automatically identify pulmonary embolism within segmental arteries using computed tomography (CT) images. The system architecture incorporates an enhanced Mask R-CNN deep neural network trained on PE-containing images. This network accurately localizes pulmonary embolisms in CT images and effectively delineates their boundaries. This study involved creating a local data set and evaluating the model predictions against pulmonary embolisms manually identified by expert radiologists. The sensitivity, specificity, accuracy, Dice coefficient, and Jaccard index values were obtained as 96.2%, 93.4%, 96.%, 0.95, and 0.89, respectively. The enhanced Mask R-CNN model outperformed the traditional Mask R-CNN and U-Net models. This study underscores the influence of Mask R-CNN’s loss function on model performance, providing a basis for the potential improvement of Mask R-CNN models for object detection and segmentation tasks in CT images.

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15

Valenzuela, Terence D. "Pulmonary Embolism." Emergency Medicine Clinics of North America 6, no. 2 (May 1988): 253–66. http://dx.doi.org/10.1016/s0733-8627(20)30559-9.

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16

Dunmire, Susan M. "Pulmonary Embolism." Emergency Medicine Clinics of North America 7, no. 2 (May 1989): 339–54. http://dx.doi.org/10.1016/s0733-8627(20)30340-0.

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17

Fernandes, Tanya. "Pulmonary embolism." Nursing Standard 23, no. 28 (March 19, 2009): 58. http://dx.doi.org/10.7748/ns2009.03.23.28.58.c7178.

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18

Ratcliffe, Marie-Clare. "Pulmonary embolism." Nursing Standard 23, no. 45 (July 15, 2009): 51. http://dx.doi.org/10.7748/ns2009.07.23.45.51.c7112.

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19

Harmon, Kimberly G., and Matthew B. Roush. "Pulmonary Embolism." Physician and Sportsmedicine 26, no. 12 (December 1998): 53–56. http://dx.doi.org/10.3810/psm.1998.12.1224.

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20

Smith, Tony P. "Pulmonary Embolism." American Journal of Roentgenology 174, no. 6 (June 2000): 1489–97. http://dx.doi.org/10.2214/ajr.174.6.1741489.

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21

Swaroop, Mamta, and AbigailK Tarbox. "Pulmonary embolism." International Journal of Critical Illness and Injury Science 3, no. 1 (2013): 69. http://dx.doi.org/10.4103/2229-5151.109427.

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22

Ralph, David D. "PULMONARY EMBOLISM." Radiologic Clinics of North America 32, no. 4 (June 1994): 679–87. http://dx.doi.org/10.1016/s0033-8389(22)00402-x.

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23

Peterson, Robert T., and Allan L. Goldman. "Pulmonary Embolism." Primary Care: Clinics in Office Practice 12, no. 2 (June 1985): 383–96. http://dx.doi.org/10.1016/s0095-4543(21)01264-1.

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24

Carmichael, Terri. "Pulmonary embolism." Nursing Standard 25, no. 30 (March 30, 2011): 59–60. http://dx.doi.org/10.7748/ns.25.30.59.s50.

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Ratcliffe, Marie-Clare. "Pulmonary embolism." Nursing Standard 23, no. 45 (July 15, 2009): 51. http://dx.doi.org/10.7748/ns.23.45.51.s51.

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26

Carmichael, Terri. "Pulmonary embolism." Nursing Standard 25, no. 30 (March 30, 2011): 59. http://dx.doi.org/10.7748/ns2011.03.25.30.59.c8426.

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27

Rali, Parth, Viral Gandhi, and Khalid Malik. "Pulmonary Embolism." Critical Care Nursing Quarterly 39, no. 2 (2016): 131–38. http://dx.doi.org/10.1097/cnq.0000000000000106.

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28

Wenger, Nanette K. "Pulmonary embolism." Postgraduate Medicine 84, no. 2 (August 1988): 107–15. http://dx.doi.org/10.1080/00325481.1988.11700367.

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29

Niethammer, John G., Karl F. Hubner, and Edward Buonocore. "Pulmonary embolism." Postgraduate Medicine 87, no. 1 (January 1990): 263–70. http://dx.doi.org/10.1080/00325481.1990.11704538.

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30

Kutty, Kesavan. "Pulmonary embolism." Postgraduate Medicine 88, no. 4 (September 15, 1990): 72–88. http://dx.doi.org/10.1080/00325481.1990.11704754.

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31

Kerr, Colin P., and Lester Yan. "Pulmonary embolism." Postgraduate Medicine 91, no. 7 (May 15, 1992): 73–86. http://dx.doi.org/10.1080/00325481.1992.11701345.

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32

GLENNY, ROBB W. "Pulmonary Embolism." Southern Medical Journal 80, no. 10 (October 1987): 1266–76. http://dx.doi.org/10.1097/00007611-198710000-00017.

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33

Sokolove, Peter E., and Steven R. Offerman. "Pulmonary Embolism." New England Journal of Medicine 345, no. 18 (November 2001): 1311. http://dx.doi.org/10.1056/nejmicm0000019.

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34

Thompson, Andrew. "Pulmonary embolism." Clinical Medicine 19, no. 4 (July 2019): 357.2–358. http://dx.doi.org/10.7861/clinmedicine.19-4-357a.

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35

MURRAY, IVES P., MAGED S. MIKHAIL, MICHAEL J. BANNER, and JEROME H. MODELL. "Pulmonary embolism." Critical Care Medicine 15, no. 2 (February 1987): 114–17. http://dx.doi.org/10.1097/00003246-198702000-00006.

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36

Glauser, Jonathan. "Pulmonary Embolism." Emergency Medicine News 25, no. 4 (April 2003): 6. http://dx.doi.org/10.1097/00132981-200304000-00011.

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Glauser, Jonathan. "Pulmonary Embolism." Emergency Medicine News 25, no. 5 (May 2003): 34. http://dx.doi.org/10.1097/00132981-200305000-00023.

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38

Spiegler, Peter. "Pulmonary Embolism." Clinical Pulmonary Medicine 6, no. 5 (September 1999): 320–21. http://dx.doi.org/10.1097/00045413-199909000-00009.

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39

Moser, K. M. "Pulmonary Embolism." Hämostaseologie 17, no. 01 (January 1997): 1–4. http://dx.doi.org/10.1055/s-0038-1660007.

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SummaryOur diagnostic approach to the embolic suspect involves a high index of suspicion based on the clinical context. Next, study of the lower extremity veins; if such a study (IPG, Duplex, venogram) discloses proximal DVT, the patient needs treatment and a search for embolism may or may not be required. If it is, perfusion and ventilation scans are performed next. If this test is non-diagnostic, pulmonary angiography is the final step. As noted, other technics are under investigation. It will take time to assess whether their promise is fulfilled since it is well-known that “early reports” are usually the most positive and may not translate into clinical, non-investigative use.

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40

Bankier, A. A., L. Aram, Ch J. Herold, and D. Fleischmann. "Pulmonary Embolism." Hämostaseologie 17, no. 01 (January 1997): 5–13. http://dx.doi.org/10.1055/s-0038-1660008.

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41

Humenberger, M., and I. M. Lang. "Pulmonary embolism." Hämostaseologie 28, no. 01/02 (March 2008): 40–43. http://dx.doi.org/10.1055/s-0037-1616920.

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ZusammenfassungDie akute Pulmonalembolie ist Teil des Spektrums venöser Thromboembolien. Die tiefe Beinvenenthrombose, der akute Thrombus in Transit durch das rechte Herz, die akute Pulmonalembolie selbst und die chronisch thromboembolische pulmonale Hypertension (CTEPH) gehören dazu. Pulmonalembolie kann rezidivieren und schwere Spätfolgen (z. B. postthrombotisches Syndrom, CTEPH) nach sich ziehen.Diese Übersicht fasst aktuelle Konzepte zur Pathophysiologie, Epidemiologie, Diagnose und Therapie dieser häufigen Erkrankung zusammen.

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42

DAY, MICHAEL W. "Pulmonary embolism." Nursing 35, no. 9 (September 2005): 88. http://dx.doi.org/10.1097/00152193-200509000-00053.

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43

Currie, Donna L. "Pulmonary embolism." Critical Care Nursing Quarterly 13, no. 2 (September 1990): 41–50. http://dx.doi.org/10.1097/00002727-199009000-00007.

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Cardin, Tracy, and Anthony Marinelli. "Pulmonary Embolism." Critical Care Nursing Quarterly 27, no. 4 (October 2004): 310–22. http://dx.doi.org/10.1097/00002727-200410000-00002.

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Ribeiro, Ary, Per Lindmarker, Hans Johnsson, Anders Juhlin-Dannfelt, and Lennart Jorfeldt. "Pulmonary Embolism." Circulation 99, no. 10 (March 16, 1999): 1325–30. http://dx.doi.org/10.1161/01.cir.99.10.1325.

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46

Nicholson, David. "Pulmonary embolism." Journal of Thoracic Imaging 4, no. 4 (October 1989): 20–22. http://dx.doi.org/10.1097/00005382-198910000-00008.

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47

Buckner, C. Barry, Craig W. Walker, and Gary L. Purnell. "Pulmonary embolism." Journal of Thoracic Imaging 4, no. 4 (October 1989): 23–27. http://dx.doi.org/10.1097/00005382-198910000-00009.

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48

Koschel, Mary Jo. "Pulmonary Embolism." AJN, American Journal of Nursing 104, no. 6 (June 2004): 46–50. http://dx.doi.org/10.1097/00000446-200406000-00029.

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49

Christie, Florence. "Pulmonary Embolism." American Journal of Nursing 98, no. 11 (November 1998): 36–37. http://dx.doi.org/10.1097/00000446-199811000-00035.

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50

Wolfe, Walter G. "Pulmonary Embolism." Annals of Surgery 238, Supplement (December 2003): S67—S71. http://dx.doi.org/10.1097/01.sla.0000097528.34081.e3.

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