Relevant bibliographies by topics / Pulmonary disease

Academic literature on the topic 'Pulmonary disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 24 January 2023

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Journal articles on the topic "Pulmonary disease"

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Herald, G. Peter Praveen, and H. Krishna Murthy. "EVALUATION OF PULMONARY HYPERTENSION IN CHRONIC OBSTRUCTIVE PULMONARY DISEASE." International Journal of Integrative Medical Sciences 6, no. 1 (February 20, 2019): 765–68. http://dx.doi.org/10.16965/ijims.2019.102.

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De Palo, Vera A. "Pulmonary Disease." Journal of the American Podiatric Medical Association 94, no. 2 (March 1, 2004): 157–67. http://dx.doi.org/10.7547/87507315-94-2-157.

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Although many medical problems are generally managed in concert with a general medical physician, it is important for the podiatric physician to be familiar with some of the major diseases of the lung. Pneumonia, an infectious process within the lung, is the sixth-leading overall cause of death. Antibiotic treatment, oxygen administration, and supportive care are the mainstays of its therapy. Chronic obstructive pulmonary disease presents as a spectrum from chronic bronchitis, with a greater inflammatory component, to emphysema, with a more significant destructive component. Asthma, often a more episodic chronic obstructive disease, is characterized by inflammation of the airways leading to their narrowing. The work of breathing is often increased in these diseases, and treatment is with combination therapies with a focus on smoking cessation. Thromboembolic disease, the occlusion of blood vessels with consequent interruption of blood flow, may occur in a patient with risk factors, especially after surgery. Treatment is with anticoagulation agents or in some cases with thrombolysis. Prophylaxis is key. (J Am Podiatr Med Assoc 94(2): 157-167, 2004)
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Ramraje, N. N. "Study of OSA in Chronic Obstructive Pulmonary Disease." Journal of Medical Science And clinical Research 05, no. 05 (May 12, 2017): 21712–14. http://dx.doi.org/10.18535/jmscr/v5i5.78.

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Roe, Paul G., and J. Gareth Jones. "Pulmonary disease." Current Opinion in Anaesthesiology 4, no. 6 (December 1991): 853–59. http://dx.doi.org/10.1097/00001503-199112000-00019.

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Musani, Ali I. "Pulmonary Disease." Medical Clinics of North America 103, no. 3 (May 2019): xix—xx. http://dx.doi.org/10.1016/j.mcna.2019.02.001.

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White, Dorothy A., and Muhammad K. Zaman. "Pulmonary disease." Medical Clinics of North America 76, no. 1 (January 1992): 19–44. http://dx.doi.org/10.1016/s0025-7125(16)30369-8.

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Musani, Ali I. "Pulmonary Disease." Medical Clinics of North America 103, no. 3 (May 2019): i. http://dx.doi.org/10.1016/s0025-7125(19)30020-3.

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Walker, Patricia A., and Dorothy A. White. "PULMONARY DISEASE." Medical Clinics of North America 80, no. 6 (November 1996): 1337–62. http://dx.doi.org/10.1016/s0025-7125(05)70493-4.

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Williams, Ruth. "Pulmonary disease." Nursing Management 18, no. 9 (January 26, 2012): 13. http://dx.doi.org/10.7748/nm.18.9.13.s12.

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Wiedemann, Herbert P., and Richard A. Matthay. "Cor Pulmonale in Chronic Obstructive Pulmonary Disease." Clinics in Chest Medicine 11, no. 3 (September 1990): 523–45. http://dx.doi.org/10.1016/s0272-5231(21)00715-2.

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Dissertations / Theses on the topic "Pulmonary disease"

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McAllister, David Anthony. "Chronic obstructive pulmonary disease, pulmonary function and cardiovascular disease." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5615.

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Cardiovascular disease is common in Chronic Obstructive Pulmonary Disease (COPD), and forced expiratory volume in one second (FEV1) independently predicts cardiovascular morbidity and mortality. Pathological changes in the systemic vasculature have been proposed as potential mechanisms linking COPD to cardiovascular disease, and patients with COPD may be at increased risk of acute myocardial infarction during acute exacerbations. Notwithstanding causation, FEV1 may be a useful prognostic marker in patients undergoing cardiac surgery. This thesis examined these three aspects of cardiovascular co-morbidity in relation to COPD and FEV1. In 2,241 consecutive cardiac surgery patients, FEV1 was associated with length of hospital stay (p<0.001) and mortality (p<0.001) adjusting for age, sex, height, body mass index, socioeconomic status, smoking, cardiovascular risk factors, chronic pulmonary disease, and type/urgency of surgery. In a survey of Scottish Respiratory Consultants there was no consensus regarding the investigation and management of acute coronary syndrome in exacerbation of COPD. In a case-series of 242 patients with exacerbations 2.5% (95% CI 1.0 to 5.6%) had chest pain, raised serum troponin and serial electrocardiogram changes suggestive of acute coronary syndrome. However, over half reported chest pain, while raised troponin was not associated with chest pain or serial ECG changes. Carotid-radial pulse wave velocity (PWV), aortic distensibility, and aortic calcification were measured to assess the relationship of the systemic vasculature to FEV1 and emphysema severity on CT. In adjusted analyses, emphysema was associated with PWV in patients with COPD (p = 0.006) and, in population based samples, with extent of distal aortic calcification (p=0.02) but not with aortic distensibility (p=0.60). This thesis found that FEV1 was associated with mortality and length of hospital stay in patients undergoing cardiac surgery, and that chest pain and raised troponin were common but unrelated in exacerbation of COPD. In the vascular studies distal but not proximal vascular pathology was associated with FEV1, and if COPD is truly related to systemic arterial disease, the distal arterial tree is implicated.
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Chang, Betty. "Pulmonary disease in scleroderma combined interstitial lung disease and pulmonary hypertenson and predictors of pulmonary hypertension /." Available to US Hopkins community, 2002. http://wwwlib.umi.com/dissertations/dlnow/3068129.

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Solomon, Brahm Kevin. "Psychological Aspects of Pulmonary Rehabilitation in Chronic Obstructive Pulmonary Disease." Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/34292.

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As a leading cause of disability that often leads to death, chronic obstructive pulmonary disease (COPD) can be characterized as both a chronic illness and a life-threatening one. As a result, the experience of individuals with COPD can include psychological concerns that are associated with both rehabilitation and palliative care. At the same time, the often-uncertain trajectory of COPD obscures a clear transition from rehabilitation to palliative care. It is not surprising, therefore, that treatments aimed at addressing patients’ rehabilitative and palliative needs largely proceed independently of each other. This dissertation contains two studies conducted with patients participating in a pulmonary rehabilitation program for COPD (N = 242). Separately, each study stems from a research tradition grounded in either the rehabilitative or palliative approach to treatment. Together, the studies highlight an opportunity for a model of more integrated care. Study 1 is derived from the rehabilitation literature and focuses on the issue of “catastrophizing” about breathlessness. Catastrophizing is characterized by a magnification of a symptom’s threat value, rumination about its perceived negative impact, and a sense of helplessness in addressing it. In some medical conditions with a primary symptom, such as chronic pain, catastrophizing demonstrates a strong relationship with the development of disability. Study 1 examines whether this relationship is found in the context of breathlessness. The study also reports the initial validation of the Breathlessness Catastrophizing Scale (BCS) as a means of assessing this phenomenon. Study 2 has its conceptual basis in the palliative care literature and highlights patients’ existential concerns around loss of dignity. Loss of dignity is a central construct in recent health care debates, because it is a primary reason underlying the requests of terminally ill individuals to seek medically hastened deaths (i.e., euthanasia or assisted suicide). Until now, however, loss of dignity has only been examined among patients with cancer. Study 2 examines whether loss of dignity is as prevalent among those with advanced COPD, and whether it improves with treatment. In Study 1 the BCS was found to be a reliable measure of breathlessness catastrophizing, with good convergent validity and sensitivity to change. Interestingly, it appears that breathlessness catastrophizing need not be a barrier to functional improvement in COPD. In Study 2, a “fractured” sense of dignity was found among 13% of patients with advanced COPD, suggesting that it is at least as prevalent as among those receiving palliative cancer care. It was also evident that loss of dignity is amenable to change with appropriate rehabilitation. This finding is important for societal debates regarding the provision of medically hastened deaths, which are often described as offering “death with dignity”. Together these studies demonstrate that in an interdisciplinary environment, such as the pulmonary rehabilitation program, not only is collaboration possible, but the distinct rehabilitative and palliative needs of patients can be met.
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Greening, Neil James. "Early pulmonary rehabilitation for exacerbations of chronic obstructive pulmonary disease." Thesis, University of Leicester, 2014. http://hdl.handle.net/2381/29155.

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Exacerbations are key events in the natural history of chronic obstructive pulmonary disease (COPD), with limited recovery of physical performance, and the highest cause of readmission in the UK. This thesis explores the impact of exacerbations in COPD and chronic respiratory disease. In the first study I have investigated the effects of an early rehabilitation intervention on healthcare utilisation, strength and exercise capacity by conducting a large randomised control trial. Using a sub-group of this cohort I have then explored factors that predict hospital readmission. Finally I have conducted a study of single leg neuromuscular electrical stimulation (NMES) in stable COPD, alongside a resistance training group. No difference was seen following early rehabilitation in hospitalisation, healthcare utilisation or physical performance. A number of unexpected findings were noted, including an increase in 12 month mortality in the intervention group and large functional recovery in the usual care group. Using multivariate analysis three risk factors for hospital readmission were identified, including quadriceps cross sectional area, using ultrasound. In the stable state NMES was seen to significantly increase muscle mass from baseline, comparable to changes seen using resistance training. In summary early rehabilitation in chronic respiratory disease does not impact on future hospitalisation. Identification of those with rehabilitation potential is required as the hospitalised population represent a frail group, with advanced disease.
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John, Michelle. "The extra-pulmonary effects of chronic obstructive pulmonary disease (COPD)." Thesis, University of Nottingham, 2014. http://eprints.nottingham.ac.uk/14405/.

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Rationale Cardiovascular disease (CVD) is a leading cause of mortality in patients with COPD. Aortic stiffness, measured using aortic pulse wave velocity (PWV), an independent, non-invasive, predictor of CV risk; and inflammatory markers are increased in COPD. Screening tools for community based identification of increased CVD risk, and a proactive approach to addressing primary prevention of CVD is needed. Statins modulate aortic stiffness and are anti-inflammatory, but are not currently used for primary prevention in COPD. Objectives Proof of principle double-blind Randomised Control Trial (RCT) to determine if six weeks simvastatin 20mg od reduces aortic stiffness, systemic and airway inflammation in COPD. Cross-sectional pilot study comparing a non-invasive measure of oxidative stress (skin “AGE”) in COPD and controls, to lung function and aortic stiffness. Methods Stable patients (n=70) were randomised to simvastatin or placebo treatment. Pre- and post-treatment aortic stiffness, blood pressure, spirometry, circulating inflammatory mediators and lipids were measured; airway inflammatory markers were performed where possible. Predefined subgroup analysis was performed where baseline aortic PWV >10m/s. For the cross-sectional study stable COPD patients (n=84) and controls (n=36) had lung function, arterial stiffness and skin AGE measured. Results In the RCT the active group achieved significantly lower total cholesterol, but no significant drop in aortic PWV compared to placebo group: -0.7(95%CI -1.8,0.5)m/s, p=0.24; or inflammatory markers. In those with higher baseline aortic PWV, n=22, aortic PWV improved in the active group compared to placebo: -2.8(-5.2,-0.3)m/s, p=0.03. Skin AGE was increased in COPD compared to controls, inversely related to lung function, and directly related to aortic stiffness. Conclusions We could not detect any significant difference in the change in aortic PWV in patients with COPD taking simvastatin compared to placebo. We did, however, report a significant and clinically relevant reduction in aortic PWV in those with high baseline aortic stiffness, suggesting a potential for statins to reduce CV morbidity in high risk individuals. The pilot cross-sectional study suggests there is an indication to assess the potential role of skin AGE in patients with COPD as a non-invasive measure of CV risk.
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Marante, Tânia Alves. "The impact of chronic obstructive pulmonary disease on iNKT lymphocytes." Master's thesis, Universidade de Aveiro, 2017. http://hdl.handle.net/10773/17144.

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Mestrado em Biomedicina Molecular
A doença pulmonar obstrutiva crónica (DPOC) é uma das doenças inflamatórias mais comuns das vias aéreas e uma das principais causas de morbidade e mortalidade em todo o mundo. A doença é caracterizada por uma limitação persistente do fluxo aéreo, geralmente progressiva. As respostas inflamatórias crónicas e imunes desempenham papéis fundamentais no desenvolvimento e progressão da DPOC. A inflamação é uma resposta protetora normal, mas na DPOC esta inflamação é amplificada. Várias células inflamatórias, seus mediadores e enzimas participam na resposta inflamatória na DPOC. A reabilitação respiratória é um componente fundamental da gestão da doença pulmonar obstrutiva crónica. Ela é projetada para melhorar a condição física e psicológica de pessoas com doenças respiratórias crónicas e para promover a adesão a longo prazo do comportamento que melhora a saúde. O objetivo principal deste trabalho foi contribuir para a compreensão do papel das células iNKT na patologia da DPOC. Além disso, também pretendemos explorar o efeito da reabilitação respiratória nas células iNKT em pacientes com DPOC. Análises clínicas e imunológicas foram feitas em pacientes com DPOC (n=7), pacientes com DPOC que realizaram reabilitação respiratória (n=4) e controlos saudáveis com idade e género idêntico aos doentes (n=14). Os participantes foram estudados duas vezes, com um intervalo de 12 semanas. Foram estudados os seguintes parâmetros clínicos: índice de massa corporal, percentagem de massa gorda, função pulmonar, força dos músculos respiratórios, força muscular isométrica, teste das cinco repetições sentar-levantar, teste de avaliação da DPOC e questionário da dispneia. As células iNKT foram estudadas em termos de percentagem, fenótipo, citotoxicidade e produção de citocinas. Não foi observada nenhuma alteração na percentagem de células iNKT, seus subconjuntos e produção de citocinas no sangue periférico de pacientes com DPOC em comparação com os controlos. Os nossos resultados sugeriram que as células iNKT de pacientes com DPOC podem ter uma diminuição na ativação precoce pela redução da expressão do CD69. Foi também sugerida uma redução na capacidade citotóxica em doentes. A reabilitação respiratória não pareceu afetar a redução da expressão do CD69, mas pareceu contribuir para o aumento da citotoxicidade das células iNKT e desempenhar um papel na melhoria do defeito citotóxico. Este é o primeiro estudo que conduziu uma extensa análise de correlações entre variáveis clínicas para a DPOC e variáveis imunológicas. Os resultados das nossas correlações indicaram que algumas células podem estar associadas a uma melhoria no estado de saúde do paciente e outras com o agravamento da DPOC. Este foi um estudo exploratório, e mais investigações sobre este tema são necessárias para fortalecer as conclusões.
Chronic obstructive pulmonary disease (COPD) is one of the most common inflammatory diseases of the airways and a leading cause of morbidity and mortality worldwide. The disease is characterized by a persistent airflow limitation, generally progressive. Chronic inflammatory and immune responses play key roles in the development and progression of COPD. The inflammation is a normal protective response, but in COPD this inflammation is amplified. Several inflammatory cells, their mediators and enzymes participate in the inflammatory response in COPD. Pulmonary rehabilitation is a core component of chronic obstructive pulmonary disease management. It is designed to improve the physical and psychological condition of people with chronic respiratory disease and to promote the long-term adherence of health-enhancing behavior. The main aim of this work was to contribute for understanding the role of iNKT cells in COPD pathology. In addition we also aimed to explore the effect of pulmonary rehabilitation on the iNKT cells in patients with COPD. Clinical and immunological analysis were done in patients with COPD (n=7), patients with COPD performing pulmonary rehabilitation (n=4) and age- and gender-matched healthy controls (n=14). Participants were studied twice, with an interval of 12 weeks. The following clinical parameters were studied: body mass index, body fat percentage, pulmonary function, respiratory muscle strength, quadriceps muscle strength, five time seat to stand, COPD assessment test and modified medical research council. The iNKT cells were studied in terms of percentage, phenotype, cytotoxicity and cytokine production. No alteration in percentage of iNKT cells, their subsets and cytokine production were observed in the peripheral blood of patients with COPD in comparison with controls. Our results suggested that iNKT cells from patients with COPD might have a decrease in early activation by reduction of CD69 expression. A reduction in cytotoxic capacity in patients was also suggested. Pulmonary rehabilitation did not seem to affect the reduction of CD69 expression, but seemed to contribute to the increase in iNKT cell cytotoxicity and might have a role in improving the cytotoxic defect. This is the first study that conducted an extensive correlation analysis between clinical variables for COPD and immunological variables. Findings from our correlations indicated that some cells might be associated with an improvement in the health condition of the patient, and others with the worsening of COPD. This was an exploratory study, and further research on this topic is warranted to strengthen conclusions.
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Murphy, Nicola. "Chronic obstructive pulmonary disease and anxiety." Thesis, Coventry University, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368862.

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Shafi, N. T. "Pulmonary involvement in Anderson Fabry Disease." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1414900/.

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Aim: To investigate the clinical, physiological, radiological and pathological changes which occur in the lungs in Anderson Fabry Disease (AFD) Methods: In this study we have used lung function testing, high resolution CT scanning and induced sputum examination to investigate the lung. We have measured sputum enzyme activity using fluorometric assays, cell populations using flow cytometry and cytokines using enzyme linked immunosorbent assays. We have compared investigation findings from AFD subjects with those from patient’s with airways disease in the form of chronic obstructive pulmonary disease (COPD) and healthy controls Results: We have shown that respiratory symptoms are common, and airway involvement is widespread though mild in AFD. Pulmonary involvement is more common in males, in subjects with worse overall disease as measured by Mainz Severity Score Index, and is independent of smoking. No significant radiological changes were evident on CT chest imaging in AFD. We have presented novel data on α-galactosidase A activity from lung derived samples, which demonstrate low sputum enzyme activity in AFD males compared to controls and AFD females, and consistently higher enzyme activity in sputum derived leucocytes compared to those derived from peripheral blood. We did not find any detectable differences in blood or sputum α-galactosidase A activity in subjects on enzyme replacement therapy. Cell populations from induced sputum in AFD subjects demonstrated a predominance of monocytes/macrophages, similar to the COPD subjects, and there was the suggestion of an increased T cell population in AFD subjects with airway obstruction compared to those without. Elevated concentrations of sputum IL-8 were seen in the sputum of AFD subjects compared to controls. Conclusion: There is demonstrable and clinically relevant involvement of the lungs in AFD, which appears to occur as a result of deficient α-galactosidase A in the lungs and subsequent inflammatory processes.
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Raeside, David Alexander. "Ambulatory pulmonary artery pressure monitoring in pulmonary vascular disease in man." Thesis, University of Glasgow, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398763.

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Santiago, Pia Bantegui. "Adherence to exercise following pulmonary rehabilitation of chronic obstructive pulmonary disease /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2004. http://wwwlib.umi.com/cr/ucsd/fullcit?p3130214.

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Books on the topic "Pulmonary disease"

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Farver, Carol, Subha Ghosh, Thomas Gildea, and Charles D. Sturgis. Pulmonary Disease. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47598-7.

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Thomas, Stocker J., and Society for Pediatric Pathology (U.S.). Meeting, eds. Pediatric pulmonary disease. New York: Hemisphere Pub. Corp., 1989.

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A, Stockley Robert, ed. Chronic obstructive pulmonary disease. Malden, Mass: Blackwell Pub., 2005.

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Cytopathology of pulmonary disease. Basel, Switzerland: Karger, 1988.

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Currie, Graeme P. Chronic obstructive pulmonary disease. Oxford: Oxford University Press, 2009.

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Nakamura, Hiroyuki, and Kazutetsu Aoshiba, eds. Chronic Obstructive Pulmonary Disease. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-0839-9.

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Nici, Linda, and Richard ZuWallack, eds. Chronic Obstructive Pulmonary Disease. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-673-3.

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Stockley, Robert A., Stephen I. Rennard, Klaus Rabe, and Bartolome Celli, eds. Chronic Obstructive Pulmonary Disease. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470755976.

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Calverley, P. M. A., and N. B. Pride, eds. Chronic Obstructive Pulmonary Disease. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9.

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Pokorski, Mieczyslaw, ed. Pulmonary Dysfunction and Disease. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42010-3.

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Book chapters on the topic "Pulmonary disease"

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Cohn, Steven L. "Pulmonary Disease." In Perioperative Medicine, 211–26. London: Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-498-2_21.

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Fuller, J. Michael, and John R. Hubbard. "Pulmonary Disease." In Primary Care Medicine for Psychiatrists, 113–26. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5857-6_6.

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Webster, James R., and Thomas Cain. "Pulmonary Disease." In Geriatric Medicine, 653–65. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4757-2705-0_43.

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Ebell, Mark H. "Pulmonary Disease." In Evidence-Based Diagnosis, 329–54. New York, NY: Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4757-3514-7_14.

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Webster, James R. "Pulmonary Disease." In Geriatric Medicine, 853–67. New York, NY: Springer New York, 2003. http://dx.doi.org/10.1007/0-387-22621-4_58.

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Hirsch, Jeffrey G. "Pulmonary Disease." In Oklahoma Notes, 41–64. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-4010-5_4.

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Robbins, Miriam R. "Pulmonary Disease." In The ADA Practical Guide to Patients with Medical Conditions, 43–69. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2015. http://dx.doi.org/10.1002/9781119121039.ch3.

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Farver, Carol, Subha Ghosh, Thomas Gildea, and Charles D. Sturgis. "Introduction to Multidisciplinary Techniques." In Pulmonary Disease, 1–13. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47598-7_1.

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Farver, Carol, Subha Ghosh, Thomas Gildea, and Charles D. Sturgis. "Tumors of the Pleura." In Pulmonary Disease, 131–43. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47598-7_10.

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Farver, Carol, Subha Ghosh, Thomas Gildea, and Charles D. Sturgis. "Chronic Obstructive Pulmonary Diseases." In Pulmonary Disease, 145–60. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47598-7_11.

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Conference papers on the topic "Pulmonary disease"

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Mirdamadi, Mahsa, Hosnollah Sadeghi, Ali Safaei, and Mahmood Salesi. "Postoperative Pulmonary Complications in Bronchial Asthma/Chronic Obstructive Pulmonary Disease/Non-Pulmonary Disease Patients." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4061.

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Martins Natal, Rebeca, João Fernandes Costa, Sofia Ferreira, Angela Albuquerque, Margarida Maurício, and Luís Vaz Rodrigues. "Pulmonary rehabilitation in interstitial lung diseases compared with chronic obstructive pulmonary disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa669.

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Kawassaki, Alexandre M., Thiago C. A. Dantas, Bruno G. Baldi, Suzana P. Pimenta, Rafael S. Musolino, Carlos Roberto R. Carvalho, Hironori Haga, Ronaldo A. Kairalla, and Thais Mauad. "IgG4 Related Pulmonary Disease." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4510.

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Balasubramanian, A., T. M. Kolb, R. L. Damico, P. M. Hassoun, M. C. McCormack, and S. C. Mathai. "Characterization of Chronic Obstructive Pulmonary Disease-Pulmonary Hypertension." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5961.

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Calancea, Valentin, Eudochia Terna, Tatiana Dumitras, Doina Barba, Irina Cosciug, and Sergiu Matcovschi. "Pulmonary haemodynamic disorders in chronic obstructive pulmonary disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2464.

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Jakimova, Marina Artemovna, Natalya Karpina, Olga Gordeeva, and Rasul Asanov. "Comorbidity: pulmonary tuberculosis and chronic obstructive pulmonary disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2969.

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Parra, A., J. Garcia, L. Munera, L. M. Cadavid, and J. Uribe. "Pulmonary Manifestations of Behcet's Disease." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4955.

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Singh, W., L. S. H. Cruz, and B. Yaghmour. "Chagas Disease and Pulmonary Hypertension." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3611.

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Frantz, Robert P., E. S. Yi, James E. Loyd, and Michael D. McGoon. "Heritable Pulmonary Hypertension With Features Of Pulmonary Venoocclusive Disease." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4879.

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Huntley, Christopher, Ahmed Fahim, James Owen, and Paren Chohan. "A Comparison of Pulmonary Rehabilitation Outcomes in Interstitial Lung Disease and Chronic Obstructive Pulmonary Disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa676.

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Reports on the topic "Pulmonary disease"

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Barros-Poblete, Marisol, Rodrigo Torres-Castro, Mauricio Henríquez, Anita Guequen, Isabel Blanco, and Carlos Flores. Dysbiosis as a prognostic factor for clinical worsening in chronic respiratory disease: A systematic review and metanalysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0089.

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Review question / Objective: Is dysbiosis a prognostic factor for clinical worsening in patients with chronic respiratory diseases?. Condition being studied: Dysbiosis, defined as changes in the quantitative and qualitative composition of the microbiota. Eligibility criteria: Over 18 years old adult patients with chronic respiratory diseases clinical diagnosis (cystic fibrosis, chronic obstructive pulmonary disease, asthma, idiopathic pulmonary fibrosis, interstitial lung disease, sarcoidosis, bronchiectasis, non-CF bronchiectasis, pulmonary hypertension) according to the International Statistical Classification of Diseases and Related Health Problems (ICD) from OMS) and international guidelines of each disease.
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Liang, R., D. Liu, HB Li, and ZG Zhai. The efficacy and safety of traditional Chinese medicine formulas in the treatment of chronic obstructive pulmonary disease complicated with pulmonary hypertension: a systematic review and meta-analysis study. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0041.

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Review question / Objective: This systematic review and meta-analysis was intended to evaluate the efficacy and safety of traditional Chinese medicine(TCM) formulas in the treatment of chronic obstructive pulmonary disease(COPD) complicated with pulmonaryhypertension (PH). Condition being studied: Chronic obstructive pulmonary disease(COPD) complicated with pulmonary hypertension(PH) is classified as the third group PH.According to epidemiology, the most common cause of PH associated with lung diseases and/or hypoxia is COPD, but the prevalence rate of COPD with PH range from 20% to 91% variously. In China, many TCM formulas are regularly used in COPD patients , thus TCM formulas therapy is worth considering.
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Wang, Yanping, Mingru Huang, Liping Tang, Lingxia Xu, Jiangfeng Wu, Fei Wang, and Ying Zhang. Moxibustion for stable chronic obstructive pulmonary disease: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2021. http://dx.doi.org/10.37766/inplasy2021.4.0047.

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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desaturations and arousals from rest. The recurrent arousals which occur in OSA lead to neurocognitive consequences, daytime sleepiness, and reduced quality of life. Because of apneas and hypopneas, patients are experiencing hypoxemia and hypercapnia, which result in increasing levels of catecholamine, oxidative stress, and low-grade inflammation that lead to the appearance of cardio-metabolic consequences of OSA. COPD is a chronic inflammatory lung disease defined by persistent, usually pro-gressive AFL (airflow limitation). Changes in lung mechanics lead to the main clini-cal manifestations of dyspnea, cough, and chronic expectoration. Furthermore, patients with COPD often suffer from anxiety and depression also, the risk of OSA and insomnia is higher than those hospitalized for other reasons. Although COPD is twice as rare as asthma but is the cause of death eight times more often.
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Feng, Zhenzhen, Xuanlin Li, Yang Xie, and Jiansheng Li. Effectiveness of Tai Chi for Chronic obstructive Pulmonary Disease: Overview of Systematic Reviews and Meta-analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2020. http://dx.doi.org/10.37766/inplasy2020.10.0114.

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Zhou, Wen. Does benralizumab effectively treat chronic obstructive pulmonary disease? a protocol of systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0039.

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Xue, Jiali, Xiaona Zhang, Hongyan Lu, Xirui Jiang, Fang Yu, and Pengfei Yang. Nonpharmacological interventions for chronic obstructive pulmonary disease-related fatigue: A systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0072.

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Lei, Yuping, Meili Wang, Guiqiang Sun, Yong Liu, Yapei Yang, and Dong Hao. Chinese herbal medicine injections (CHMIs) for chronic pulmonary heart disease: protocol for a Bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2020. http://dx.doi.org/10.37766/inplasy2020.12.0004.

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Li, Shuyao, Wenshuang Zhang, and Tong Guan. Effect of Bifidobacterium Bifidum for Chronic Obstructive Pulmonary Disease in China: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0023.

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Xie, Rongfang, Chunyan Huang, Miaomiao Li, and Zhihui Lan. Yiqi Wenyang Huoxue Method in Treating Stable Chronic Obstructive Pulmonary Disease: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0011.

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You might also be interested in the extended bibliographies on the topic 'Pulmonary disease' for particular source types:
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