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Books on the topic 'Proto-oncogenes'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 July 2024

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1

Gregory, Bock, Marsh Joan, and Symposium on Proto-oncogenes in Cell Development (1989 : Ciba Foundation), eds. Proto-oncogenes in cell development. Chichester: Wiley, 1990.

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2

Morgan, James I. Proto-Oncogene expression in the nervous /system. Amsterdam: Published by Elsevier for the Foundation for the study of the Nervous System (FESN), 1991.

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3

Bock, Greg, and Joan Marsh, eds. Ciba Foundation Symposium 150 - Proto-Oncogenes in Cell Development. Chichester, UK: John Wiley & Sons, Ltd., 1990. http://dx.doi.org/10.1002/9780470513927.

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4

Hormones, growth factors, and oncogenes. Boca Raton, Fla: CRC Press, 1987.

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5

Robertson, Scott McGregor. Isolation and characterization of SCR-related sequences from the platyfish Xiphophorus maculatus (Poeciliidae; Teleostei) and an evolutionary analysis of the SRC gene-family. Prien am Chiemsee: A.S. Intemann und C.C. Intemann, 1989.

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6

Collum, Robert Gerard. Studies on the structure and function of N-m y c. [New York]: [Columbia University], 1992.

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7

1943-, Harris Curtis C., ed. Multistage carcinogenesis: Proceedings of the 22nd International Symposium of the Princess Takamatsu Cancer Research Fund, Tokyo, ₉91. Tokyo: Japan Scientific Societies Press, 1993.

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8

Symposium, Takamatsu no Miya Hi Gan Kenkyū Kikin International. Multistage carcinogenesis: Proceedings of the 22nd International Symposium of the Princess Takamatsu Cancer Research Fund, Tokyo, 1991. Tokyo: Japan Scientific Societies Press, 1992.

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9

E, Angel Peter, and Herrlich Peter 1940-, eds. The fos and jun families of transcription factors. Boca Raton: CRC Press, 1994.

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10

Crafting science: A sociohistory of the quest for the genetics of cancer. Cambridge, Mass: Harvard University Press, 1996.

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11

A, Khan Sohaib, and Stancel George M, eds. Protooncogenes and growth factors in steroid hormone induced growth and differentiation. Boca Raton: CRC Press, 1994.

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12

Dang, Chi V. C-myc function in neoplasia. New York: Springer, 1995.

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13

A, Lee Linda, ed. c-Myc function in neoplasia. Austin, TX: R.G. Landes Co., 1995.

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14

Kellie, Stuart. Tyrosine kinases and neoplastic transformation. Austin: R.G. Landes, 1994.

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15

Marsh, Joan, and Gregory R. Bock. Proto-Oncogenes in Cell Development. Wiley & Sons, Incorporated, John, 2008.

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16

Symposium, CIBA Foundation, Joan Marsh, and Gregory R. Bock. Proto-Oncogenes in Cell Development. Wiley & Sons, Limited, John, 2007.

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17

Symposium, CIBA Foundation. Proto-Oncogenes in Cell Development - Symposium No. 150. John Wiley & Sons, 1990.

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18

Wingett, Denise G. Characterization of post-transcriptional mechanisms involved in the expression of the pim-1 proto-oncogene. 1991.

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19

Schuchardt, Anita Mary. The role of the c-ret proto-oncogene in the development of the nervous and excretory systems. 1994.

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20

VOGT, PETER K. Oncogenes: SELECTED REVIEWS (Current Topics in Microbiology & Immunology). Edited by Peter K. Vogt. Springer, 1989.

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21

Harris, Curtis C., and Setsuo Hirohashi. Multistage Carcinogenesis: Proceedings of the 22nd International Symposium of the Princess Takamatsu Cancer Research Fund, Tokyo, 1991 (Princess Takamatsu Symposia, 22). Crc Pr I Llc, 1993.

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22

The Myc/Max/Mad Transcription Factor Network (Current Topics in Microbiology and Immunology). Springer, 2006.

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23

FOS and JUN Families of Transcription Factors. Taylor & Francis Group, 2017.

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24

Angel, Peter E., and Peter Herrlich. FOS and JUN Families of Transcription Factors. Taylor & Francis Group, 2017.

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25

Angel, Peter E., and Peter Herrlich. FOS and JUN Families of Transcription Factors. Taylor & Francis Group, 2017.

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26

Angel, Peter E., and Peter Herrlich. FOS and JUN Families of Transcription Factors. Taylor & Francis Group, 2017.

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27

Fujimura, Joan H. Crafting Science: A Sociohistory of the Quest for the Genetics of Cancer. Harvard University Press, 2013.

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28

Fujimura, Joan H. Crafting Science: A Sociohistory of the Quest for the Genetics of Cancer. Harvard University Press, 1996.

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29

Damgard, I. B. Advances in Cryptology-Eurocrypt '90: Workshop on the Theory and Application of Cryptographic Techniques, Aarhus, Denmark, May 21-24, 1990 Proceeding (Lecture Notes in Computer Science). Springer, 1991.

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30

Potter, M. C-myc In B-cell Neoplasia: 14TH WORKSHOP ON MECHANISMS IN B-CELL NEOPLASIA (Current Topics in Microbiology & Immunology). Edited by M. Potter. SPRINGER-VERLAG, 1997.

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31

Grant, Warren, and Martin Scott-Brown. Principles of oncogenesis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0322.

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Abstract:

It is obvious that the process of developing cancer—oncogenesis—is a multistep process. We know that smoking, obesity, and a family history are strong independent predictors of developing malignancy; yet, in clinics, we often see that some heavy smokers live into their nineties and that some people with close relatives affected by cancer spend many years worrying about a disease that, in the end, they never contract. For many centuries scientists have struggled to understand the process that make cancer cells different from normal cells. There were those in ancient times who believed that tumours were attributable to acts of the gods. Hippocrates suggested that cancer resulted from an imbalance between the black humour that came from the spleen, and the other three humours: blood, phlegm, and bile. It is only in the last 100 years that biologists have been able to characterize some of the pathways that lead to the uncontrolled replication seen in cancer, and subsequently examine exactly how these pathways evolve. The rampant nature by which cancer invades local and distant tissues, as well its apparent ability to spread between related individuals led some, such as Peyton Rous in 1910, to suggest that cancer was an infectious condition. He was awarded a Nobel Prize in 1966 for the 50 years of work into investigating a link between sarcoma in chickens and a retrovirus that became known as Rous sarcoma virus. He had shown how retroviruses are able to integrate sequences of DNA coding for errors in cellular replication control (oncogenes) by introducing into the human cell viral RNA together with a reverse transcriptase. Viruses are now implicated in many cancers, and in countries where viruses such as HIV and EBV are endemic, the high incidence of malignancies such as Kaposi’s sarcoma and Burkitt’s lymphoma is likely to be directly related. There are several families of viruses associated with cancer, broadly classed into DNA viruses, which mutate human genes using their own DNA, and retroviruses, like Rous sarcoma virus, which insert viral RNA into the cell, where it is then transcribed into genes. This link with viruses has not only led to an understanding that cancer originates from genetic mutations, but has also become a key focus in the design of new anticancer therapies. Traditional chemotherapies either alter DNA structure (as with cisplatin) or inhibit production of its component parts (as with 5-fluorouracil.) These broad-spectrum agents have many and varied side effects, largely due to their non-specific activity on replicating DNA throughout the body, not just in tumour cells. New vaccine therapies utilizing gene-coding viruses aim to restore deficient biological pathways or inhibit mutated ones specific to tumour cells. The hope is that these gene therapies will be effective and easily tolerated by patients, but development is currently progressing with caution. In a trial in France of ten children suffering from X-linked severe combined immunodeficiency and who were injected with a vector that coded for the gene product they lacked, two of the children subsequently died from leukaemia. Further analysis confirmed that the DNA from the viral vector had become integrated into an existing, but normally inactive, proto-oncogene, LM02, triggering its conversion into an active oncogene, and the development of life-threatening malignancy. To understand how a tiny change in genetic structure could lead to such tragic consequences, we need to understand the molecular biology of the cell and, in particular, to pay attention to the pathways of growth regulation that are necessary in all mammalian cell populations. Errors in six key regulatory pathways are known as the ‘hallmarks of cancer’ and will be discussed in the rest of this chapter.

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