Academic literature on the topic 'Prostate tumour inducer'
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Journal articles on the topic "Prostate tumour inducer"
Lanning, Ben, Jason Webber, Pinar Uysal-Onganer, Wen Guo Jiang, Aled Clayton, and Dafydd Alwyn Dart. "Prostate Cancer Cell Extracellular Vesicles Increase Mineralisation of Bone Osteoblast Precursor Cells in an In Vitro Model." Biology 10, no. 4 (April 10, 2021): 318. http://dx.doi.org/10.3390/biology10040318.
Full textPascal, Laura E., Khalid Z. Masoodi, June Liu, Xiaonan Qiu, Qiong Song, Yujuan Wang, Yachen Zang, et al. "Conditional deletion of ELL2 induces murine prostate intraepithelial neoplasia." Journal of Endocrinology 235, no. 2 (November 2017): 123–36. http://dx.doi.org/10.1530/joe-17-0112.
Full textAbdulkadir, Sarki A., Jeffrey A. Magee, Thomas J. Peters, Zahid Kaleem, Cathy K. Naughton, Peter A. Humphrey, and Jeffrey Milbrandt. "Conditional Loss of Nkx3.1 in Adult Mice Induces Prostatic Intraepithelial Neoplasia." Molecular and Cellular Biology 22, no. 5 (March 1, 2002): 1495–503. http://dx.doi.org/10.1128/mcb.22.5.1495-1503.2002.
Full textBronte, Vincenzo, Tihana Kasic, Giorgia Gri, Keti Gallana, Giovanna Borsellino, Ilaria Marigo, Luca Battistini, et al. "Boosting antitumor responses of T lymphocytes infiltrating human prostate cancers." Journal of Experimental Medicine 201, no. 8 (April 11, 2005): 1257–68. http://dx.doi.org/10.1084/jem.20042028.
Full textSong, Liankun, Vyvyan Nguyen, Shan Xu, Jana Yamak, Kia Arabzadehkaffash, Ali Fazelpour, Merci Mino, Matthew Tippin, Shuang Meng, and Xiaolin Zi. "Abstract 1: Transcriptional profiling of prostatic Skp2 knock-in mouse model and development of the associated prostate organoids for testing Skp2 targeting agents." Cancer Research 82, no. 12_Supplement (June 15, 2022): 1. http://dx.doi.org/10.1158/1538-7445.am2022-1.
Full textKetteler, Julia, Andrej Panic, Henning Reis, Alina Wittka, Patrick Maier, Carsten Herskind, Ernesto Yagüe, Verena Jendrossek, and Diana Klein. "Progression-Related Loss of Stromal Caveolin 1 Levels Mediates Radiation Resistance in Prostate Carcinoma via the Apoptosis Inhibitor TRIAP1." Journal of Clinical Medicine 8, no. 3 (March 12, 2019): 348. http://dx.doi.org/10.3390/jcm8030348.
Full textLayman, Awo Akosua K., Shivam Joshi, and Sanjeev Shah. "Metastatic prostate cancer presenting as tumour-induced osteomalacia." BMJ Case Reports 12, no. 7 (July 2019): e229434. http://dx.doi.org/10.1136/bcr-2019-229434.
Full textEne, Cosmin-Victor, Ilinca Nicolae, Bogdan Geavlete, Petrisor Geavlete, and Corina Daniela Ene. "IL-6 Signaling Link between Inflammatory Tumor Microenvironment and Prostatic Tumorigenesis." Analytical Cellular Pathology 2022 (April 12, 2022): 1–10. http://dx.doi.org/10.1155/2022/5980387.
Full textSass, Stephanie N., and Sandra O. Gollnick. "Tumor-associated myeloid cells convert indolent prostate cancer to aggressive disease." Journal of Immunology 196, no. 1_Supplement (May 1, 2016): 73.21. http://dx.doi.org/10.4049/jimmunol.196.supp.73.21.
Full textBoutillon, Pigat, Sala, Reyes-Gomez, Moriggl, Guidotti, and Goffin. "STAT5a/b Deficiency Delays, but does not Prevent, Prolactin-Driven Prostate Tumorigenesis in Mice." Cancers 11, no. 7 (July 2, 2019): 929. http://dx.doi.org/10.3390/cancers11070929.
Full textDissertations / Theses on the topic "Prostate tumour inducer"
Markowski, Mark Christopher. "Inflammatory cytokines induce ubiquitination and loss of the prostate suppressor protein NKX3.1." Connect to Electronic Thesis (CONTENTdm), 2008. http://worldcat.org/oclc/454182234/viewonline.
Full textAnzenberg, Vered. "LET dependence of radiation-induced bystander effects using human prostate tumor cells." Thesis, Massachusetts Institute of Technology, 2008. http://hdl.handle.net/1721.1/44795.
Full text"June 2008."
Includes bibliographical references (leaves 133-140).
In the past fifteen years, evidence provided by many independent research groups have indicated higher numbers of cells exhibiting damage than expected based on the number of cells traversed by the radiation. This phenomenon has been coined as the "bystander effect". The purpose of this study was to characterize the ability of irradiated tumor cells to induce bystander effects in co-cultured cells. Human DU-145 prostate carcinoma cells were grown on a 1.4 [mu]m-thick mylar membrane in specially constructed cell culture dishes for irradiation with alpha particles (average energy 3.14 MeV) from a 241Am source, or in 6-well plates for irradiation with 250 kVp x-rays at 25°C. In parallel experiments, the tumor cells were incubated at 4°C for one hour prior to irradiation and irradiated on ice to test the nature of the bystander signal. Bystander cells were placed into the medium above the irradiated DU-145 and were co-incubated for a length of time. The bystander effect endpoints measured in either DU-145 tumor cells or in normal primary AGO1522 fibroblasts were micronucleus (MN) formation, [gamma]-H2AX double strand break repair foci, and survival fraction. A 1.5-2.0-fold increase in MN formation was observed in both DU-145 and AG01522 bystander cells after either alpha particle or xray irradiation of the DU-145 target cells. A 1.5-fold [gamma]-H2AX bystander increase and a survival fraction reduction to 80% were only detected in AGO1522 cells, and only after xray irradiation of target DU-145 cells. Alpha particle irradiation of the target DU-145 cells produced neither [gamma]-H2AX foci nor survival fraction bystander effect in either cell line. Lowering the temperature to 4°C during the irradiation of the DU-145 tumor cells, with either x-rays or alpha particles, eliminated both the MN formation and the decreased survival fraction bystander effects in the co-cultured AG01522 fibroblasts.
(cont.) This study demonstrates that biochemical processes in the directly-irradiated tumor cells are required for initiation of the signaling process. Low temperature during the irradiation inhibited the initiation of a bystander signal. There are also LET-dependent differences in the signal released from DU-145 human prostate carcinoma cells; and that, for some endpoints, bystander AG01522 fibroblasts and bystander DU-145 prostate carcinoma cells respond differently to the same, medium-mediated signal.
by Vered Anzenberg.
Ph.D.
Leclercq, Tamara Marie. "Regulation of sphingosine kinase by interacting proteins." Thesis, 2010. http://hdl.handle.net/2440/64752.
Full textThesis (Ph.D.) -- University of Adelaide, School of Molecular and Biomedical Science, 2010
Walker, Tristan J. "The association of infiltrating lymphocytes in androgen ablation induced apoptosis of prostate tumours." Thesis, 2005. http://hdl.handle.net/2429/16799.
Full textMedicine, Faculty of
Medical Genetics, Department of
Graduate
Hsiao, Fu-Ching, and 蕭富擎. "Hispolon Inhibits Angiogenesis By Suppressing Vascular Endothelial Growth Factor Signaling Pathway And induces Prostate Tumor Cell apoptosis." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/82330016299966227600.
Full text中國醫藥大學
中國藥學研究所碩士班
97
Phellinus linteus (PL) , is a traditional medicinal plant of oriental people (especially in China, Japan,and North Korea ) , was demonstrated to exhibit anti-bacterial , anti-tumour , anti-fibrotic , anti-oxidant and anti-inflammation functions in several studies. Hispolon is a major component of PL with great antioxidant activity. Whether Hispolon induces prostate tumor cell apoptosis or inhibits angiogenesis,which is crucial for cancer and other human diseases, remains unknown. First, we investigated how hispolon induce apoptosis in the prostate tumor cell lines PC-3. In the MTT assay, treatment of Hispolon inhibited the growth of PC-3 in the dose- and time- dependent manners. Next ,Hispolon inducing PC-3 cells apoptosis was confirmed by nuclear condensation (DAPI staining), DNA fragmentation assay and Annexin V-FITC/PI staining . We also examined the Hispolon induced cell cycle arrest in the G2 phase by flow cytometric analysis. In vitro angiogenesis study, we found that Hispolon effectively inhibited human umbilical vein endothelial cell migration, invasion, and tube formation. In a coculture study, Hispolon completely prevented U87MG cell–mediated capillary formation of HUVEC. This inhibitor also prevented that VEGF and MMP induced migration ability of HUVEC when cultured alone or cocultured with U87MG cells. In vivo angiogenesis study, we use the rat aortic ring assay, chicken chorioallantoic membrane assay (CAM) and Matrigel plug assay to evaluate anti-angiogenic effects. Finally, Western blot showed that Hispolon treatment suppressed the protein expression of TIMP-1 , PAI-1 , PI3K , AKT , P44/42 , phospho-P44/42 , RAS , NF-κB and COX-2, which were involved in endothelial cell survival, proliferation, migration and angiogeneis. Our results indicated that Hispolon exerted an anti-tumor activity associated with decreased proliferation of tumor cells and induced the apoptosis of prostate tumor cell, and inhibited the anti-angiogenic activity by VEGF signaling pathway .
Book chapters on the topic "Prostate tumour inducer"
Sarkar, Dipak K. "Fetal Alcohol Exposure Increases Susceptibility to Carcinogenesis and Promotes Tumor Progression in Prostate Gland." In Biological Basis of Alcohol-Induced Cancer, 389–402. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09614-8_23.
Full textHermanus Johannes Sliepen, Sonny. "Bone Cancer Pain, Mechanism and Treatment." In Recent Advances in Bone Tumours and Osteoarthritis. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95910.
Full textMorello, Matteo, Gustavo E. Ayala, Fabiana Rosati, Giovanna Danza, Rile Li, Anna Frolov, Rosalyn M. Adam, et al. "Loss of Caveolin-1 Increases Tumor Cell Migration, Is Predictive of Disease-Free Survival, and Induces Steroidogenesis in Prostate-Derived Fibroblasts." In TRANSLATIONAL - Steroidal Regulation of Breast & Prostate Cancer, OR06–6—OR06–6. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part1.or7.or06-6.
Full textConference papers on the topic "Prostate tumour inducer"
Attaluri, Anilchandra, Ronghui Ma, and Liang Zhu. "Temperature Elevations in Implanted Prostatic Tumors in Mice During Magnetic Nanoparticle Hyperthermia: In Vivo Experimental Study." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53128.
Full textManuchehrabadi, N., A. Attaluri, H. Cai, R. Edziah, E. Lalanne, C. Bieberich, R. Ma, A. M. Johnson, and L. Zhu. "Visualization and Quantification of Gold Nanorods Distribution in Prostatic Tumors Using MicroCT Imaging." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80317.
Full textCrawford, Alison, Kristin Vazzana, Jeffrey VanValkenburgh, Lauric Haber, Jennifer Principio, Cagan Gurer, Kara Olson, Eric Smith, Gavin Thurston, and Jessica R. Kirshner. "Abstract A193: Fully human bispecific antibodies induce potent anti-tumor effects against prostate tumors in mice." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics; November 5-9, 2015; Boston, MA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1535-7163.targ-15-a193.
Full textKo, Chun-Jung, Ying-Chieh Lu, Pee-Fang Lai, Pei-Wen Hsiao, and Ming-Shyue Lee. "Abstract B67: Matriptase is involved in COX-2 signaling-induced prostate cancer cell invasion." In Abstracts: AACR Special Conference on Tumor Metastasis; November 30-December 3, 2015; Austin, TX. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.tummet15-b67.
Full textFang, Lei-Ya, Kouji Izumi, and Wen-Jye Lin. "Abstract C229: Tumor-associated macrophages induce prostate tumorigenesis via CCL4/STAT3 signals." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 12-16, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1535-7163.targ-11-c229.
Full textPan, Chunliu, Yanni Zulia, and Kent L. Nasituk. "Abstract B092: Myokine signaling blockade prevents androgen deprivation therapy-induced sarcopenia and promotes prostate tumor regression." In Abstracts: AACR Special Conference: Prostate Cancer: Advances in Basic, Translational, and Clinical Research; December 2-5, 2017; Orlando, Florida. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.prca2017-b092.
Full textSharma, Sambad, Fei Xing, Yin Liu, Kerui Wu, Aya Kobayashi, and Kounosuke Watabe. "Abstract 3206: SPARC in tumor microenvironment induces dormancy of prostate cancer in bone." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3206.
Full textColeman, C. Norman, Molykutty John-Aryankalayil, Adeola Y. Makinde, and Sanjeewani T. Palayoor. "Abstract 425: Fractionated radiation-induced tumor suppressor microRNAs in human prostate carcinoma cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-425.
Full textEl-Kenawi, Asmaa, William Dominguez Viqueira, Min Liu, Shivanshu Awasthi, Jasreman Dhillon, Kosj Yamoah, Xiaoqing Yu, John Koomen, Robert Gatenby, and Brian Ruffell. "Abstract 734: Macrophage tumor cell metabolic interactions induce therapeutic resistance in prostate cancer." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-734.
Full textCordonnier, Thomas, Jennifer L. Bishop, Masaki Shiota, Ario Takeuchi, Ka Mun Nip, Martin Gleave, and Amina Zoubeidi. "Abstract C25: Hsp27 is required for EGF-induced epithelial to mesenchymal transition in prostate cancer." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-c25.
Full textReports on the topic "Prostate tumour inducer"
Chai, Karl X. Signal Transduction Pathway in Maspin-Induced Tumor Suppression of Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, March 2001. http://dx.doi.org/10.21236/ada395750.
Full textBushman, Wade, and Aubie Shaw. Identification of Sonic Hedgehog-Induced Stromal Factors That Stimulate Prostate Tumor Growth. Fort Belvoir, VA: Defense Technical Information Center, November 2007. http://dx.doi.org/10.21236/ada484566.
Full textYang, Feng. Novel Therapeutic Targets to Inhibit Tumor Microenvironment-Induced Castration-Resistant Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613723.
Full textShaw, Aubie, and Wade Bushman. Identification of Sonic Hedgehog-Induced Stromal Factors That Stimulate Prostate Tumor Growth. Fort Belvoir, VA: Defense Technical Information Center, November 2006. http://dx.doi.org/10.21236/ada466565.
Full textXing, Fei. BMP7 Induces Dormancy of Prostatic Tumor Stem Cell in Bone. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada570860.
Full textXing, Fei. BMP7 Induces Dormancy of Prostatic Tumor Stem Cell in Bone. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada592100.
Full textKobayashi, Aya. BMP7 Induces Dormancy of Prostatic Tumor Stem Cell in Bone. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada553888.
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