Journal articles: 'Prostate – Cancer – Nutritional aspects'

1

Schmid, H. P. "S11 Nutritional aspects of primary prostate cancer prevention." European Journal of Cancer Supplements 8, no. 2 (March 2010): 3–4. http://dx.doi.org/10.1016/s1359-6349(10)70747-3.

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Berg, Courtney J., David J. Habibian, Aaron E. Katz, Kaitlin E. Kosinski, Anthony T. Corcoran, and Andrew S. Fontes. "Active Holistic Surveillance: The Nutritional Aspect of Delayed Intervention in Prostate Cancer." Journal of Nutrition and Metabolism 2016 (2016): 1–7. http://dx.doi.org/10.1155/2016/2917065.

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Purpose. Active surveillance is an emergent strategy for management of indolent prostate cancer. Our institution’s watchful waiting protocol, Active Holistic Surveillance (AHS), implements close monitoring for disease progression along with various chemopreventive agents and attempts to reduce unnecessary biopsies. Our objective is to report on the treatment rates of men on our AHS protocol as well as determine reasons for progression.Materials/Methods. Low risk and low-intermediate risk patients were enrolled in AHS at Winthrop University Hospital between February 2002 and August 2015. Our IRB-approved study analyzed survival rate, discontinuation rates, and definitive treatments for patients in our AHS cohort.Results. 235 patients met inclusion criteria. Median age and follow-up for the cohort were 66 (44–88) years and 42 (3–166) months, respectively. The overall survival for the cohort was 99.6% and the disease specific survival was 100%. A total of 27 (11.5%) patients discontinued AHS.Conclusion. The incorporation of chemopreventive agents in our AHS protocol has allowed patients to prolong definitive treatment for many years. Longer follow-up and additional studies are necessary to further validate the effectiveness of AHS.

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Omenukor, K. "Cancer Awareness Campaign and Screening." Journal of Global Oncology 4, Supplement 2 (October 1, 2018): 142s. http://dx.doi.org/10.1200/jgo.18.73900.

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Background and context: Colorectal cancer is the 3rd leading cause of cancer-related mortalities, which can be prevented by early screening. However, inadequate knowledge regarding the importance of early screening contributes to low cancer screening rates in the population. Aims: A collaborative initiative between David Omenukor Foundation and Fight Colorectal Cancer Organization strives to cancer awareness and screening in the population. Strategy: The David Omenukor Foundation organized a 5-km WALK-A-THON in Mesquite, Texas, on March 10, 2018, as part of the activities to observe the March Colorectal Cancer Awareness Month. During the event, participants received free screening for colorectal, breast, and prostate cancers. Free cholesterol, diabetes, and blood pressure testing were also done because of the impact of these comorbidities on health outcomes. Education experts on cancer were available to teach aspects of healthy diets and exercise. Two cancer patients and a survivor also shared their experiences. Program/Policy process: The program seeks to increase cancer awareness among populations and promote the culture of early and regular screening. Outcomes: Free colorectal and breast cancer screenings were provided to 270 people. About 60 people received free prostate-specific antigen (PSA) test, whereas 135 people received fecal occult blood testing. Similarly, 75 women received mammogram testing. The total number of patients who received colorectal cancer screening increased from 50 on 11th March 2017 to 135 on March 11th, 2018. Impact: The foundation created awareness of all forms of cancer and emphasized the value of early screening as the most effective to avoid the cancer scourge. The participants benefitted from nutritional advice as one strategy for reducing the risk of colorectal cancer. The event indicated that the campaign on early screening for detection was beginning to catch up. Regular interactive events and screenings increase knowledge of cancer and reduce disparities in cancer screening in the community.

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Burden, Sorrel, Jana Sremanakova, Debra Jones, and Chris Todd. "Dietary interventions for cancer survivors." Proceedings of the Nutrition Society 78, no. 1 (December 19, 2018): 135–45. http://dx.doi.org/10.1017/s0029665118002690.

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This review evaluates evidence on dietary interventions for cancer survivors giving an overview of people's views and preferences for service attributes and provides a narrative review. After cancer, people often want to change their diet and there is a plethora of evidence why dietary optimisation would be beneficial. However, cancer survivors have different preferences about attributes of services including: place, person and communication mode. Randomised control trials have been reviewed to provide a narrative summary of evidence of dietary interventions. Most studies were on survivors of breast cancer, with a few on colorectal, prostate and gynaecological survivors. Telephone interventions were the most frequently reported means of providing advice and dietitians were most likely to communicate advice. Dietary assessment methods used were FFQ, food diaries and 24-h recalls. Dietary interventions were shown to increase intake of fruit and vegetables, dietary fibre, and improve diet quality in some studies but with contradictory findings in others. Telephone advice increased fruit and vegetable intake primarily in women with breast cancer and at some time points in people after colorectal cancer, but findings were inconsistent. Findings from mail interventions were contradictory, although diet quality improved in some studies. Web-based and group sessions had limited benefits. There is some evidence that dietary interventions improve diet quality and some aspects of nutritional intake in cancer survivors. However, due to contradictory findings between studies and cancer sites, short term follow-up and surrogate endpoints it is difficult to decipher the evidence base.

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Uzunlulu, Mehmet, Ozge Telci Caklili, and Aytekin Oguz. "Association between Metabolic Syndrome and Cancer." Annals of Nutrition and Metabolism 68, no. 3 (2016): 173–79. http://dx.doi.org/10.1159/000443743.

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Growing data show the association of metabolic syndrome (MetS) or its components with cancer development and cancer-related mortality. It is suggested that in MetS and cancer association, insulin resistance and insulin-like growth factor 1 system play a key role, especially adipokines secreted from visceral adipocytes, free fatty acids and aromatase activity contribute to this process. It is also reported that MetS has a link with colorectal, breast, endometrial, pancreas, primary liver and, although controversial, prostate cancer. Although every component of MetS is known to have an association with cancer development, it is still debated whether the effects of these components are additive or synergistic. On the other hand, in the association between MetS and cancer, the role of antidiabetic and antihypertensive treatments including thiazolidinedione, insulin, angiotensin receptor blockers is also suggested. The primary approach in MetS-cancer relation is to prevent risk factors. Life style changes including weight loss and a healthy diet are known to decrease cancer risk in normal population. It is postulated that an insulin-sensitizing agent, metformin, has cancer-preventing effects on diabetic patients. This review discusses the relationship between MetS and cancer from different aspects and examines this relationship in some of the cancers suggested to be linked with MetS.

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Shivappa, Nitin, James R. Hébert, Faezeh Askari, Mehdi Kardoust Parizi, and Bahram Rashidkhani. "Increased Inflammatory Potential of Diet is Associated with Increased Risk of Prostate Cancer in Iranian Men." International Journal for Vitamin and Nutrition Research 86, no. 5-6 (October 2016): 161–68. http://dx.doi.org/10.1024/0300-9831/a000395.

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Purpose: Various aspects of diet, including specific foods and nutrients, have been implicated to play a role in modulating inflammation and in the etiology of prostate cancer. Studies examining this association have been conducted primarily in Western countries; but none in Middle Eastern Countries. Method: We examined the association between the dietary inflammatory index (DII) and prostate cancer in an ageand BMI-matched case-control study among 40-78 year-old Iranian males. A total of 50 incident cases and 100 controls attending the same hospital as the cases during the same time period were recruited. The DII is a literature-derived population-based dietary index developed to determine the inflammatory potential of individuals' diets and was computed based on dietary intake assessed using a previously validated semi-quantitative food frequency questionnaire (FFQ) that was expanded to assess diet and cancer in the Iranian population. Logistic regression was used to estimate odds ratios, with DII score fit as continuous and as a dichotomous variable. Results: Multivariable-adjusted analyses revealed that men with higher DII score (>0.23) to be at higher risk of prostate cancer [odds ratio (OR) = 3.96; 95% CI =1.29–12.16, p-value = 0.02)] compared to men with lower DII scores (≤0.23). Conclusion: These data suggest a pro-inflammatory diet, as indicated by increasing DII score, may be a risk factor for prostate cancer in Iranian men..

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Rodríguez-García, Carmen, Cristina Sánchez-Quesada, and José J. Gaforio. "Dietary Flavonoids as Cancer Chemopreventive Agents: An Updated Review of Human Studies." Antioxidants 8, no. 5 (May 18, 2019): 137. http://dx.doi.org/10.3390/antiox8050137.

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Over the past few years, interest in health research has increased, making improved health a global goal for 2030. The purpose of such research is to ensure healthy lives and promote wellbeing across individuals of all ages. It has been shown that nutrition plays a key role in the prevention of some chronic diseases such as obesity, cardiovascular disease, diabetes, and cancer. One of the aspects that characterises a healthy diet is a high intake of vegetables and fruits, as both are flavonoid-rich foods. Flavonoids are one of the main subclasses of dietary polyphenols and possess strong antioxidant activity and anti-carcinogenic properties. Moreover, some population-based studies have described a relationship between cancer risk and dietary flavonoid intake. In this context, the goal of this review was to provide an updated evaluation of the association between the risk of different types of cancers and dietary flavonoid intake. We analysed all relevant epidemiological studies from January 2008 to March 2019 using the PUBMED and Web of Science databases. In summary, this review concludes that dietary flavonoid intake is associated with a reduced risk of different types of cancer, such as gastric, breast, prostate, and colorectal cancers.

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Rinaldi, J. C., S. A. A. Santos, K. T. Colombelli, L. Birch, G. S. Prins, L. A. Justulin, and S. L. Felisbino. "Maternal protein malnutrition: effects on prostate development and adult disease." Journal of Developmental Origins of Health and Disease 9, no. 4 (March 27, 2018): 361–72. http://dx.doi.org/10.1017/s2040174418000168.

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AbstractWell-controlled intrauterine development is an essential condition for many aspects of normal adult physiology and health. This process is disrupted by poor maternal nutrition status during pregnancy. Indeed, physiological adaptations occur in the fetus to ensure nutrient supply to the most vital organs at the expense of the others, leading to irreversible consequences in tissue formation and differentiation. Evidence indicates that maternal undernutrition in early life promotes changes in key hormones, such as glucocorticoids, growth hormones, insulin-like growth factors, estrogens and androgens, during fetal development. These alterations can directly or indirectly affect hormone release, hormone receptor expression/distribution, cellular function or tissue organization, and impair tissue growth, differentiation and maturation to exert profound long-term effects on the offspring. Within the male reproductive system, maternal protein malnutrition alters development, structure, and function of the gonads, testes and prostate gland. Consequently, these changes impair the reproductive capacity of the male offspring. Further, permanent alterations in the prostate gland occur at the molecular and cellular level and thereby affect the onset of late life diseases such as prostatitis, hyperplasia and even prostate cancer. This review assembles current thoughts on the concepts and mechanisms behind the developmental origins of health and disease as they relate to protein malnutrition, and highlights the effects of maternal protein malnutrition on rat prostate development and homeostasis. Such insights on developmental trajectories of adult-onset prostate disease may help provide a foundation for future studies in this field.

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Gould, Kathy. "Nutritional Aspects of Cancer Rehabilitation." Rehabilitation Oncology 13, no. 3 (1995): 12–21. http://dx.doi.org/10.1097/01893697-199513030-00010.

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Shaw, Clare. "Nutritional aspects of advanced cancer." Palliative Medicine 6, no. 2 (April 1992): 105–10. http://dx.doi.org/10.1177/026921639200600204.

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Henderson, Maureen M. "Nutritional aspects of breast cancer." Cancer 76, S10 (November 15, 1995): 2053–58. http://dx.doi.org/10.1002/1097-0142(19951115)76:10+<2053::aid-cncr2820761324>3.0.co;2-m.

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12

Tefekli, Ahmet, Murat Tunc, Volkan Tugcu, and Tarık Esen. "Prostate Cancer: All Aspects." Scientific World Journal 2013 (2013): 1–2. http://dx.doi.org/10.1155/2013/265234.

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Krawczyk, Joanna, and Leszek Kraj. "Metabolic and nutritional aspects of cancer." Postępy Higieny i Medycyny Doświadczalnej 68 (August 22, 2014): 1008–14. http://dx.doi.org/10.5604/17322693.1118194.

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Alaejos, M. Sanz, F. J. Dı́az Romero, and C. Dı́az Romero. "Selenium and cancer: some nutritional aspects." Nutrition 16, no. 5 (May 2000): 376–83. http://dx.doi.org/10.1016/s0899-9007(99)00296-8.

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Kalman, D., and LJ Villani. "Nutritional aspects of cancer-related fatigue." Rehabilitation Oncology 16, no. 3 (March 1998): 31–32. http://dx.doi.org/10.1097/01893697-199816030-00027.

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16

KALMAN, DOUGLAS, and LAURA J. VILLANI. "Nutritional Aspects of Cancer-Related Fatigue." Journal of the American Dietetic Association 97, no. 6 (June 1997): 650–54. http://dx.doi.org/10.1016/s0002-8223(97)00163-6.

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17

Bratt, Ola. "Hereditary Prostate Cancer: Clinical Aspects." Journal of Urology 168, no. 3 (September 2002): 906–13. http://dx.doi.org/10.1016/s0022-5347(05)64541-7.

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Пушкарь, Dmitriy Pushkar, Васильев, Aleksandr Vasilev, Говоров, and Aleksandr Govorov. "Aspects of cryobiology prostate cancer." Vladikavkaz Medico-Biological Bulletin 21, no. 32 (March 24, 2016): 76–83. http://dx.doi.org/10.12737/18554.

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The basis of cryotherapy was laid in the middle of the 19th century; today it is actively developing, minimally invasive and alternative method of prostate cancer treatment. The effectiveness of cryosurgery of the tissues depends on the quality of visualization of the process, of fast freezing and slow process of thawing, and also of repeating of the freeze-thaw cycles. The main factors that have a damaging effect on the tissue under the influence of low temperatures are: formation of intracellular and extracellular ice crystals, increase of the concentration of electrolytes and mechanical damage of structural components of cells. Studies have shown that the temperature below -40°С is lethal to cancer cells. The possibility of real-time visual control gives us the ability to control the process freezing and thawing.

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Kunkel, Elisabeth J. S., Jennifer R. Bakker, Ronald E. Myers, Olu Oyesanmi, and Leonard G. Gomella. "Biopsychosocial Aspects of Prostate Cancer." Psychosomatics 41, no. 2 (March 2000): 85–94. http://dx.doi.org/10.1176/appi.psy.41.2.85.

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20

Latil, Alain, and R. Lidereau. "Genetic aspects of prostate cancer." Virchows Archiv 432, no. 5 (May 19, 1998): 389–406. http://dx.doi.org/10.1007/s004280050183.

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Cronauer, M. V., and Z. Culig. "Molecular aspects of prostate cancer." World Journal of Urology 30, no. 3 (March 7, 2012): 277–78. http://dx.doi.org/10.1007/s00345-012-0853-x.

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22

Fair, William R., Neil E. Fleshner, and Warren Heston. "Cancer of the prostate: A nutritional disease?" Urology 50, no. 6 (December 1997): 840–48. http://dx.doi.org/10.1016/s0090-4295(97)00339-7.

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23

Bozzetti, Federico. "Nutritional aspects of the cancer/aging interface." Journal of Geriatric Oncology 2, no. 3 (July 2011): 177–86. http://dx.doi.org/10.1016/j.jgo.2011.03.003.

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Pöschl, Gudrun, Felix Stickel, Xiang D. Wang, and Helmut K. Seitz. "Alcohol and cancer: genetic and nutritional aspects." Proceedings of the Nutrition Society 63, no. 1 (February 2004): 65–71. http://dx.doi.org/10.1079/pns2003323.

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Chronic alcohol consumption is a major risk factor for cancer of upper aero-digestive tract (oro-pharynx, hypopharynx, larynx and oesophagus), the liver, the colo-rectum and the breast. Evidence has accumulated that acetaldehyde is predominantly responsible for alcohol-associated carcinogenesis. Acetaldehyde is carcinogenic and mutagenic, binds to DNA and protein, destroys the folate molecule and results in secondary cellular hyper-regeneration. Acetaldehyde is produced by mucosal and cellular alcohol dehydrogenase, cytochrome P450 2E1 and through bacterial oxidation. Its generation and/or its metabolism is modulated as a result of polymorphisms or mutations of the genes responsible for these enzymes. Acetaldehyde can also be produced by oral bacteria. Smoking, which changes the oral bacterial flora, also increases salivary acetaldehyde. Cigarette smoke and some alcoholic beverages, such as Calvados, contain acetaldehyde. In addition, chronic alcohol consumption induces cytochrome P450 2E1 enxyme activity in mucosal cells, resulting in an increased generation of reactive oxygen species and in an increased activation of various dietary and environmental carcinogens. Deficiencies of riboflavin, Zn, folate and possibly retinoic acid may further enhance alcohol-associated carcinogenesis. Finally, methyl deficiency as a result of multiple alcohol-induced changes leads to DNA hypomethylation. A depletion of lipotropes, including methionine, choline, betaine and S-adenosylmethionine, as well as folate, results in the hypomethylation of oncogenes and may lead to DNA strand breaks, all of which are associated with increased carcinogenesis.

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25

Hill, Michael J. "Nutritional and metabolic aspects of gastrointestinal cancer." Current Opinion in Clinical Nutrition and Metabolic Care 1, no. 5 (September 1998): 405–7. http://dx.doi.org/10.1097/00075197-199809000-00006.

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Chamberlain, Ronald S., and Yuman Fong. "Nutritional and metabolic aspects of gastrointestinal cancer." Current Opinion in Clinical Nutrition and Metabolic Care 2, no. 5 (September 1999): 381–85. http://dx.doi.org/10.1097/00075197-199909000-00005.

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Cooperman, Avram M., Jayant Chivati, and Ronald S. Chamberlain. "Nutritional and metabolic aspects of pancreatic cancer." Current Opinion in Clinical Nutrition and Metabolic Care 3, no. 1 (January 2000): 17–21. http://dx.doi.org/10.1097/00075197-200001000-00004.

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28

Chan, Wynnie. "Nutritional aspects of the development of cancer." Nutrition & Food Science 30, no. 4 (August 2000): 174–77. http://dx.doi.org/10.1108/00346650010329399.

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Popov, S. V., R. G. Guseinov, I. N. Orlov, V. V. Protoshchak, O. N. Skryabin, P. A. Babkin, F. A. Bagrov, A. S. Katunin, A. G. Martov, and V. L. Schukin. "Prostate cancer diagnosis: an immunological aspects." Herald Urology 6, no. 4 (February 22, 2019): 42–48. http://dx.doi.org/10.21886/2308-6424-2018-6-4-42-48.

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Introduction.Contemporary diagnosis of prostate cancer is crucial to the patient’s further fate. Difficulties in the histological verification of the final diagnosis and false-negative results of biopsy research are often associated with the similarity of the prostate adenocarcinoma`s micromorphological picture and its benign lesions.Purpose of the study.Comprehending the possibilities of immunohistochemical identification of tumor cells and the basal epithelial layer of prostate glandular structures in prostate cancer suspected cases.Materials and methods.A biopsy material was taken from 134 patients. Prostate adenocarcinoma was verified by routine histological examination in 72 samples, in 62 samples there were no signs of malignant transformation. Subsequently, immunohistochemical analysis of biopsy specimens was performed using antibodies to α-methylacyl-CoA racemase, nuclear p63 protein and high-molecular cytokeratin.Results.The AMACR-positive reaction of malignant cells and a negative reaction of the basal epithelium to nuclear p63 protein antibodies and high-molecular cytokeratin were detected during prostate adenocarcinoma.Conclusion.If prostate cancer is suspected, immunohistochemical assays with monoclonal antibodies to AMACR, p63 and high-molecular cytokeratin greatly facilitate the detection of adenocarcinoma.

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Cheorpeaca, B., V. Jinga, O. Codreanu, D. Radavoi, D. Badescu, and R. Petca. "Genomic aspects regarding prostate cancer aggressiveness." European Urology Supplements 15, no. 11 (December 2016): e1345. http://dx.doi.org/10.1016/s1569-9056(16)30211-1.

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31

McLeod, David G. "Editorial: Some Aspects of Prostate Cancer." Journal of Urology 155, no. 6 (June 1996): 1987–88. http://dx.doi.org/10.1016/s0022-5347(01)66070-1.

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32

Visakorpi, Tapio. "Novel endocrine aspects of prostate cancer." Molecular and Cellular Endocrinology 360, no. 1-2 (September 2012): 1–2. http://dx.doi.org/10.1016/j.mce.2012.03.015.

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33

Kanso, C., J. Etner, B. Debré, and M. Zerbib. "Cancer de la prostate : aspects médicoéconomiques." Progrès en Urologie 20, no. 2 (February 2010): 85–90. http://dx.doi.org/10.1016/j.purol.2009.06.007.

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34

Dorkin, Trevor J., and David E. Neal. "Basic science aspects of prostate cancer." Seminars in Cancer Biology 8, no. 1 (February 1997): 21–27. http://dx.doi.org/10.1006/scbi.1997.0049.

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35

Kaprin, Andrei D., Boris Ia Alekseev, Vsevolod B. Matveev, Dmitrii Iu Pushkar’, Aleksandr V. Govorov, Nina A. Gorban’, Andrei A. Kirichek, et al. "Prostate cancer." Journal of Modern Oncology 23, no. 2 (August 16, 2021): 211–47. http://dx.doi.org/10.26442/18151434.2021.2.200959.

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Prostate cancer (RPP) is a malignant neoplasm that arises from the epithelium of the prostate gland gland (PJ). рак предстательной железы; клинические рекомендацииThethiology and pathogenesis of this disease remain poorly studied. Many studies are aimed at studying diet, food, hormonal impact, as well as infections in the etiology of the RLPG. The prevalence of RPL depends on ethnic and geographical features. The highest incidence of African Americans living in the United States (60% higher than that of white Americans), the least high - in the Chinese living in China [1]. In addition to racial features, the risk factors of the RPG are considering the genetic predisposition, the age of men and nutritional features. The probability of developing a PJ tumor in a man who has one of the closest relatives of the first degree of kinship (father or brother) sick of the RLPG, is 1.8 times higher than in the population. If two relatives were sick or more (father and brother or both brothers), the risk of RPG increases in 5.51 and 7.71 times, respectively [2, 3]. African Americans have an increased risk of identifying RPG, as well as a greater probability of detecting aggressive RPG [4]. Also, the risk of RLPG is rising in men who use a large amount of animal fats [5].

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Al-Shukri, Salman Khasunovich, and Sergey Yur'evich Borovets. "Prostate cancer: some aspects of epidemiology, etiology and carcinogenesis for prostate cancer." Urologicheskie vedomosti 2, no. 1 (June 15, 2012): 23. http://dx.doi.org/10.17816/uroved2123-25.

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_, _. "Prostate Cancer." Journal of the National Comprehensive Cancer Network 5, no. 7 (August 2007): 650. http://dx.doi.org/10.6004/jnccn.2007.0057.

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To properly identify and treat patients with prostate cancer, physicians must have an in-depth understanding of the natural history and diagnostic, staging, and treatment options. Prostate cancer is a complex disease, with many controversial aspects of management and a dearth of sound data to support recommendations. Several variables must be considered in tailoring prostate cancer therapy to each individual patient. These guidelines provide a framework on which to base treatment decisions. Important changes to the guidelines since last publication include the introduction of Principles of Life Expectancy Estimation table and changes in recommendations for both hormone and chemotherapy. For the most recent version of the guidelines, please visit NCCN.org

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Nosov, A. K., S. A. Reva, and M. V. Berkut. "CONTEMPORARY ASPECTS OF TREATMENT METASTATIC PROSTATE CANCER." Practical oncology 20, no. 2 (June 30, 2019): 123–36. http://dx.doi.org/10.31917/2002123.

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Schalken, Jack A. "Molecular aspects of hormone-independent prostate cancer." BJU International 100, s2 Prostate Can (July 2007): 52–55. http://dx.doi.org/10.1111/j.1464-410x.2007.06956.x.

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40

Younes, Patrick. "Aspects IRM du cancer de la prostate." La Presse Médicale 36, no. 12 (December 2007): 1935–46. http://dx.doi.org/10.1016/j.lpm.2007.05.001.

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DeMarzo, Angelo M., William G. Nelson, William B. Isaacs, and Jonathan I. Epstein. "Pathological and molecular aspects of prostate cancer." Lancet 361, no. 9361 (March 2003): 955–64. http://dx.doi.org/10.1016/s0140-6736(03)12779-1.

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Waxman, Jonathan. "Introduction: Basic science aspects of prostate cancer." Seminars in Cancer Biology 8, no. 1 (February 1997): 1. http://dx.doi.org/10.1006/scbi.1997.0054.

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Stewart, Jubilee R., Marlene C. Artime, and Catherine A. O'Brian. "Resveratrol: A Candidate Nutritional Substance for Prostate Cancer Prevention." Journal of Nutrition 133, no. 7 (July 1, 2003): 2440S—2443S. http://dx.doi.org/10.1093/jn/133.7.2440s.

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Vanina, E. A., A. A. Molokov, V. L. Kovalenko, S. S. Tseluyko, and M. N. Polikutina. "RADIOBIOLOGICAL ASPECTS IN RADIATION THERAPY OF PROSTATE CANCER." Amur Medical Journal, no. 13 (2016): 22–28. http://dx.doi.org/10.22448/amj.2016.13.22-28.

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Lau, Benjamin, Taimur Tariq Shah, Massimo Valerio, Sami Hamid, Hashim Uddin Ahmed, and Manit Arya. "Technological aspects of delivering cryotherapy for prostate cancer." Expert Review of Medical Devices 12, no. 2 (January 8, 2015): 183–90. http://dx.doi.org/10.1586/17434440.2015.990377.

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LeRoy, Bruce E., and Nicole Northrup. "Prostate cancer in dogs: Comparative and clinical aspects." Veterinary Journal 180, no. 2 (May 2009): 149–62. http://dx.doi.org/10.1016/j.tvjl.2008.07.012.

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Middleton, Richard G. "Editorial: Prostate Cancer and Aspects of Radical Prostatectomy." Journal of Urology 156, no. 3 (September 1996): 1092–93. http://dx.doi.org/10.1016/s0022-5347(01)65711-2.

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48

Ceci, Francesco, Paolo Castellucci, Juliano J. Cerci, and Stefano Fanti. "New aspects of molecular imaging in prostate cancer." Methods 130 (November 2017): 36–41. http://dx.doi.org/10.1016/j.ymeth.2017.07.009.

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49

Visser, Adriaan, and George van Andel. "Psychosocial and educational aspects in prostate cancer patients." Patient Education and Counseling 49, no. 3 (March 2003): 203–6. http://dx.doi.org/10.1016/s0738-3991(03)00024-7.

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De Sousa, A., S. Sonavane, and J. Mehta. "Psychological aspects of prostate cancer: a clinical review." Prostate Cancer and Prostatic Diseases 15, no. 2 (January 3, 2012): 120–27. http://dx.doi.org/10.1038/pcan.2011.66.

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Journal articles on the topic 'Prostate – Cancer – Nutritional aspects'

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