Academic literature on the topic 'Prostate'
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Journal articles on the topic "Prostate"
Marlina, Kiki Amelia, Nurlaily Idris, Nikmatia Latief, Andi Alfian Zainuddin, Syakri Syahrir, and Mirna Muis. "KORELASI NILAI INTRAVESICAL PROSTATIC PROTRUSION DAN POST VOID RESIDUAL URINE MENGGUNAKAN PEMERIKSAAN ULTRASONOGRAFI TRANSABDOMINAL DENGAN SKOR INTERNATIONAL PROSTATE SYMPTOM PADA PASIEN PEMBESARAN PROSTAT JINAK." E-Jurnal Medika Udayana 10, no. 9 (September 28, 2021): 94. http://dx.doi.org/10.24843/mu.2021.v10.i9.p16.
Full textMarlina, Kiki Amelia. "KORELASI NILAI INTRAVESICAL PROSTATIC PROTRUSION DAN POST VOID RESIDUAL URINE MENGGUNAKAN PEMERIKSAAN ULTRASONOGRAFI TRANSABDOMINAL DENGAN SKOR INTERNATIONAL PROSTATE SYMPTOM PADA PASIEN PEMBESARAN PROSTAT JINAK." E-Jurnal Medika Udayana 12, no. 11 (November 22, 2023): 26. http://dx.doi.org/10.24843/mu.2023.v12.i11.p05.
Full textMarlina, Kiki Amelia. "KORELASI NILAI INTRAVESICAL PROSTATIC PROTRUSION DAN POST VOID RESIDUAL URINE MENGGUNAKAN PEMERIKSAAN ULTRASONOGRAFI TRANSABDOMINAL DENGAN SKOR INTERNATIONAL PROSTATE SYMPTOM PADA PASIEN PEMBESARAN PROSTAT JINAK." E-Jurnal Medika Udayana 12, no. 6 (June 19, 2023): 60. http://dx.doi.org/10.24843/mu.2023.v12.i06.p11.
Full textWang, Liang, Marloes Zoetemelk, Brahmananda R. Chitteti, Timothy L. Ratliff, Jason D. Myers, Edward F. Srour, Hal Broxmeyer, and Travis J. Jerde. "Expansion of prostate epithelial progenitor cells after inflammation of the mouse prostate." American Journal of Physiology-Renal Physiology 308, no. 12 (June 15, 2015): F1421—F1430. http://dx.doi.org/10.1152/ajprenal.00488.2014.
Full textLorenzo, Guillermo, Thomas J. R. Hughes, Pablo Dominguez-Frojan, Alessandro Reali, and Hector Gomez. "Computer simulations suggest that prostate enlargement due to benign prostatic hyperplasia mechanically impedes prostate cancer growth." Proceedings of the National Academy of Sciences 116, no. 4 (January 7, 2019): 1152–61. http://dx.doi.org/10.1073/pnas.1815735116.
Full textVojinov, Sasa, Goran Marusic, Ivan Levakov, and Jelena Popadic-Gacesa. "Influence of hormonal therapy on the level of prostate specific antigen in patients with advanced prostatic cancer." Medical review 63, no. 7-8 (2010): 479–82. http://dx.doi.org/10.2298/mpns1008479v.
Full textViennois, Emilie, Teresa Esposito, Julie Dufour, Aurélien Pommier, Stephane Fabre, Jean-Louis Kemeny, Laurent Guy, Laurent Morel, Jean-Marc Lobaccaro, and Silvère Baron. "Lxrα Regulates the Androgen Response in Prostate Epithelium." Endocrinology 153, no. 7 (April 30, 2012): 3211–23. http://dx.doi.org/10.1210/en.2011-1996.
Full textSanjay Kalra, Nitin Kapoor, and Saurabh Arora. "Diabetes and the prostate." Journal of the Pakistan Medical Association 73, no. 12 (November 28, 2023): 2491–92. http://dx.doi.org/10.47391/jpma.23-101.
Full textFaleiro, Mariana Batista Rodrigues, Danilo Rezende e. Silva, Rafael Malagoli Rocha, and Veridiana Maria Brianezi Dignani de Moura. "Immunoexpression of cell cycle regulators in canine prostate with proliferative lesions." Semina: Ciências Agrárias 39, no. 4 (August 2, 2018): 1831. http://dx.doi.org/10.5433/1679-0359.2018v39n4p1831.
Full textElo, Teresa, Lan Yu, Eeva Valve, Sari Mäkelä, and Pirkko Härkönen. "Deficiency of ERβ and prostate tumorigenesis in FGF8b transgenic mice." Endocrine-Related Cancer 21, no. 4 (June 17, 2014): 677–90. http://dx.doi.org/10.1530/erc-13-0480.
Full textDissertations / Theses on the topic "Prostate"
Chang, Ching-Jey George. "Prostate, benign hypertrophy and prostatic carcinoma - a study of cell biology of prostate and chemotherapy for prostatic hypertrophy and prostatic cancer /." The Ohio State University, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487856906256116.
Full textHerrera, Maria Lourdes C. "The expression of various growth factors in the normal human prostate, benign prostatic hyperplasia, and prostate carcinoma." Thesis, Hong Kong : University of Hong Kong, 1996. http://sunzi.lib.hku.hk/hkuto/record.jsp?B1754628X.
Full textBirtle, A. J. "Prostate specific antigen negative prostate cancer." Thesis, University College London (University of London), 2005. http://discovery.ucl.ac.uk/1444634/.
Full textQuintal, Maisa Momesso de. "Cancer de prostata : estudo da extensão tumoral em prostatectominas radicais." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308452.
Full textDissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
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Resumo: No estadiamento TNM 1997, os tumores de próstata órgão-confinados eram subdivididos em dois grupos: pT2a (unilaterais) e pT2b (bilaterais). Em 2002, o sistema TNM reclassificou os tumores em três grupos: pT2a (envolvimento de menos da metade de um lobo), pT2b (mais da metade de um lobo), e pT2c (envolvimento bilateral). Estudos recentes questionam a verdadeira existência de tumores pT2b, assim como diferenças relacionadas à progressão bioquímica pós-prostatectomia radical entre os estádios pT2a e pT2c. Os objetivos deste trabalho são: avaliar a extensão tumoral em espécimes de prostatectomia radical, relacionando-a com graduação histológica de Gleason, comprometimento de margens cirúrgicas, estádio clínico e patológico, PSA pré-operatório e tempo de progressão bioquímica pós-prostatectomia radical; comparar tumores localizados com comprometimento bilateral (T2c) com tumores órgão-confinados com comprometimento unilateral (pT2a) quanto aos mesmos parâmetros e verificar a existência real do estádio patológico pT2b. Foram estudados 230 homens submetidos à prostatectomia radical no período de janeiro de 1997 a julho de 2005 na Universidade Estadual de Campinas. Os espécimes cirúrgicos foram totalmente incluídos para exame histológico. A extensão tumoral foi avaliada utilizando-se um sistema de contagem de pontos. Os espécimes cirúrgicos foram estadiados segundo os critérios do TNM 2002. Os valores séricos de PSA = 0,4 ng/ml foram considerados como progressão bioquímica tumoral. Os dados foram analisados estatisticamente utilizando-se o teste de Mann-Whitney para comparação de amostras independentes e o teste exato de Fisher para avaliação de diferenças entre proporções. Foram considerados significantes os valores de p = 0,05. Para avaliação do tempo de progressão bioquímica pós-prostatectomia radical utilizou-se o produto limite de Kaplan-Meier. Utilizando os critérios TNM 2002, 29 pacientes (12,7%) eram pT2a; 139 (61,3%) eram pT2c; 30 (13,7%) eram pT3a e 28 (12,3%) eram pT3b. O mínimo e o máximo de pontos totais obtidos em tumores que envolviam apenas um lobo prostático foi de 192 e de 368 pontos, respectivamente; o maior tumor unilateral mostrou 68 pontos positivos (menos da metade do valor mínimo de pontos totais). Não foi constado nenhum caso onde a neoplasia prostática comprometesse mais da metade de um lobo, sem envolver o lobo contralateral (pT2b). A extensão tumoral apresentou relação significante e direta com PSA pré-operatório, graduação histológica de Gleason, margens cirúrgicas positivas e estádios clínico e patológico, mas não com o tempo de progressão bioquímica pós-prostatectomia radical. Não houve diferenças entre os pacientes com estádios pT2a e pT2c com relação ao PSA pré-operatório, contagem final de Gleason e tempo de progressão bioquímica, com exceção do comprometimento das margens cirúrgicas. Há relação da extensão tumoral em espécimes cirúrgicos de prostatectomia radical com PSA pré-operatório, graduação histológica de Gleason, margens cirúrgicas positivas e estádio patológico. A extensão tumoral isoladamente não parece influenciar o tempo de progressão bioquímica pós-prostatectomia radical. Este estudo questiona a real existência do estádio pT2b (tumores unilaterais que ocupam mais da metade de um lobo). Não há diferenças significativas quanto ao tempo de progressão bioquímica pós-prostatectomia radical entre os estádios patológicos pT2a e pT2c. Esses achados apóiam autores que defendem a não subdivisão do estádio pT2
Abstract: In the 1997 TNM staging system, confined-organ prostate tumors were classified in two groups: pT2a (unilateral involvement) and pT2b (bilateral involvement). In 2002, TNM staging system reclassified those tumors in three groups: pT2a (less than one half of one lobe involvement), pT2b (more than one half of one lobe involvement) and pT2c (bilateral involvement). Recent studies put in question the existence of a real pT2b tumor as well as a difference related to biochemical progression after radical prostatectomy between pT2a and pT2c tumors. The aim of this study is to verify the real existence of pT2b and evaluate tumor size comparing to Gleason score, positive surgical margins, clinical and pathological stage, preoperative PSA and time to biochemical progression after radical prostatectomy. Besides that, compare bilateral tumors (pT2c) with unilateral ones (pT2a) according to the same parameters. A total of 230 men were submitted to radical retropubic prostatectomy from July 1997 to July 2005 at Universidade Estadual de Campinas. All the surgical specimens were evaluated by complete embedding and whole mount processing. Tumor extent was evaluated through a point-count method. The surgical specimens were staged according to 2002 TNM staging system. The serum values of PSA= 0,4 ng/ml were considered biochemical progression. Mann-Whitney¿s test and Fisher¿s exact test were used to compare independent samples and different proportions. p = 0,05 was considered significant. Time to PSA progression was studied using the Kaplan-Meier¿s product limit survival estimates. Using the 2002 TNM criteria, 29 patients (12,7%) were pT2a; 139 (61,3%) were pT2c; 30 (13,7%) were pT3a and 28 (12,3%) were pT3b. The minimum and maximum total points obtained in unilateral tumors were 192 and 368 points, respectively; the biggest unilateral tumor showed 68 positive points (less than half the minimum of total point count). This study found no pT2b tumors (unilateral tumors involving greater than one-half of one prostatic lobe). Tumor extent was directly related to preoperative PSA, Gleason score, positive surgical margins, clinical and pathological stage. Alone, tumor extent does not seem to influence biochemical progression. Comparing pT2a with pT2c, there were no differences in preoperative PSA, Gleason score and biochemical progression, except positive surgical margins. There is a connection of tumor extent in radical prostatectomy specimens with preoperative PSA, Gleason score, positive surgical margins and pathological stage. Tumor extent does not seem to influence time of biochemical progression after retropubic radical prostatectomy. This study puts in question the real existence of pT2b pathological stage (unilateral tumors involving greater than one-half of one prostatic lobe). There are no significant differences between pT2a and pT2c patients in time to biochemical progression after retropubic radical prostatectomy. These findings support authors who question the subdivision of pathological stage pT2
Mestrado
Anatomia Patologica
Mestre em Ciências Médicas
Paes, Thais Ruano Lazzarini. "Cancer de prostata : estudo das margens cirurgicas comprometidas e invasão do colo vesical em especimes de prostatectomia radicais." [s.n.], 2008. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308448.
Full textDissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
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Resumo: Carcinoma da próstata é uma neoplasia maligna constituída por células que se originam de ácinos e/ou ductos prostáticos, com arranjo, graus de diferenciação e comportamento biológico variáveis. A prostatectomia radical como tratamento primário para câncer prostático clinicamente localizado tem aumentado dramaticamente nesta última década devido ao PSA. A identificação de margens cirúrgicas positivas é um fator adverso no prognóstico dos pacientes submetidos à prostatectomia radical por câncer prostático. Muitos autores relatam que margens cirúrgicas positivas são fatores preditivos significantes na progressão da doença. Já outros, afirmam que a sobrevida não é afetada pelas margens cirúrgicas. O significado do envolvimento somente microscópico do colo vesical é controverso na literatura. Para alguns autores há um menor risco de progressão quando comparado ao comprometimento das vesículas seminais e para outros o risco de recorrência é maior. Os objetivos deste trabalho foram: correlacionar margens cirúrgicas comprometidas e invasão do colo vesical em espécimes de prostatectomias radicais com variáveis clínico-patológicas. Foram estudados 230 pacientes submetidos consecutivamente a prostatectomia radical no Hospital de Clínicas da Universidade Estadual de Campinas (UNICAMP), no período de janeiro de 1997 a julho de 2005. Todo o espécime cirúrgico obtido foi previamente processado por inteiro para exame histopatológico. Cada peça cirúrgica foi pesada e medida, e a superfície foi totalmente coberta por tinta Nankim. O colo vesical e a margem apical foram amputados. Os valores séricos de PSA = 0,2 ng/mL foram considerados como progressão bioquímica. Os dados foram analisados estatisticamente utilizando-se o teste de Mann-Whitney para comparação de amostras independentes e o teste exato de Fisher para avaliação de diferenças entre proporções. O tempo de sobrevida livre de progressão bioquímica foi baseado na análise do produto-limite de Kaplan-Meier e a comparação entre os grupos foi feita usando o teste do long-rank, sendo considerado significante o valor de p =0,05. Os resultados obtidos mostraram que as margens cirúrgicas comprometidas têm relação significante com PSA pré-operatório, contagem final de Gleason (no espécime cirúrgico), extensão tumoral, estádio patológico, e tempo de progressão bioquímica (PSA) pós-prostatectomia radical. Pacientes com invasão do colo vesical apresentaram relação significante com PSA pré-operatório; contagem final de Gleason tanto no espécime cirúrgico quanto na biópsia prostática de agulha correspondente; extensão tumoral; estádio patológico e margens cirúrgicas uretral e circunferencial, porém sem relação estatística com tempo de progressão bioquímica (PSA) pós-prostatectomia radical. Ao compararmos a evolução livre de progressão bioquímica (PSA) pós-prostatectomia radical entre pacientes com invasão do colo vesical, e pacientes com invasão da vesícula seminal, os achados não apóiam considerar invasão microscópica do colo vesical como sendo pT4
Abstract: Prostate cancer is a malignant neoplasia composed by cells with origin in prostatic acini and/or ducts that present variable arrangement, biologic behavior and differentiation. Radical prostatectomy as primary treatment for localized prostate cancer has been increased dramatically in the last years due to PSA. Positive surgical margin identification is an adverse prognostic factor in patients submitted to radical prostatectomy. Many authors mention that positive surgical margins are a significant predictive factor for disease progression. Others affirm that positive surgical margins or other variables do not affect prognosis. The significance of microscopic involvement of bladder neck is controversial in the literature. For some authors biochemical progression in patients with bladder neck invasion is less important than seminal vesicle involvement but not for others. The purpose of the study was to find any possible relationship of surgical margins and bladder neck invasion in radical prostatectomies to several clinicopathological variables. This study consisted in 230 patients submitted consecutively to radical prostatectomy in the University Hospital, School of Medicine, State University of Campinas (Unicamp), between January 1997 and July 2005. All specimens were whole processed for pathological examination. Each surgical fragment was weighted, measured, and painted with Nankim's ink. Bladder neck and apex were separately processed. Biochemical (PSA) progression following radical prostatectomy was considered as being = 0,2 ng/mL. The data were statistically analysed using Mann-Whitney test to compare independent samples and exact Fisher test to evaluate differences between proportions. The time to biochemical (PSA) progression was analysed by the Kaplan-Meier product-limit analysis and the comparison between groups was done using the log-rank test. A p value of <0.05 was considered statistically significant. The results showed that patients with positive surgical margins had higher preoperative PSA, higher Gleason score on the specimen, and more extensive tumors and pathologic stage. Patients with bladder neck invasion had higher preoperative PSA, higher Gleason score in biopsies and specimens, higher number of apical and circumferential positive surgical margins, and more extensive tumors and pathological stage. The time to biochemical (PSA) progression following radical prostatectomy was not statistically significant comparing patients with and without bladder neck invasion, but statistically significant when comparing patients with and without vesicle seminal invasion. This latter finding do not support considering bladder neck invasion as stage pT4
Mestrado
Anatomia Patologica
Mestre em Ciências Médicas
Özorak, Alper Perk Hakkı. "Prostat biyopsilerinde 6-10-12 kadran biyopsilerin prostat kanseri saptama oranları ve prostat kanseri saptanması için optimal alınması gereken parça sayısının araştırılması /." Isparta : SDÜ Tıp Fakültesi, 2007. http://tez.sdu.edu.tr/Tezler/TT00340.pdf.
Full textPritchard, Colin C. "The molecular program of mouse prostate development /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/5041.
Full textAlmeida, Neto Adauto 1977. "Abordagem quantitativa da expressão do gene WFDC1 e sua isoforma delta 3 = Quantitative approach of the expression of WFDC1 gene and its isoform delta 3." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317579.
Full textTese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia
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Resumo: A próstata é alvo de afecções severas que comprometem a função urinária, a qualidade de vida e que consistem em risco de vida aos indivíduos do sexo masculino, particularmente com o avançar da idade. Interações dinâmicas entre o epitélio prostático e o estroma, regulam vários aspectos do desenvolvimento, da função e das patologias prostáticas. O gene WFDC1 é expresso pelas células musculares lisas do estroma prostático normal e tem função na regulação do comportamento do epitélio, na organização da matriz extracelular e na regulação da angiogênese. Dois transcritos principais são oriundos de splicing alternativo do transcrito primário: um com todos os éxons (WFDC1) e outro sem o éxon 3 (Delta 3). Neste trabalho, investigamos as relações quantitativas entre estas duas variantes, com emprego de qRT-PCR (Taqman) e sondas para as junções dos éxons 2-3 e 2-4, em amostras de hiperplasia prostática benigna (BPH) e de câncer de próstata (PCA) provenientes de bancos de tecidos. A expressão do gene marcador MYH11 foi utilizada como estimativa do conteúdo de células musculares lisas nas amostras. Os resultados demonstraram que as amostras puderam ser dividas em dois grupos com expressão diferencial da MYH11(um com baixa expressão e outro com alta miosina, sendo o primeiro correspondente ao quartil inferior da distribuição dos valores de expressão). Foi demonstrada correlação entre a expressão de WFDC1 e MYH11 em BPH, mas não em PCA, enquanto não houve correlação entre Delta 3 e WFDC1 e nem com MYH11. O conteúdo de Delta 3 variou em cinco ordens de magnitude em comparação ao de WFDC1. A razão entre as duas variantes apresentou variação exponencial, distribuições discretas e intercaladas das amostras de BPH e de PCA, que se distribuíram em populações que preservaram as relações 10:1; 1:1 e 1:3. Poucas amostras estiveram livres de cada uma destas variantes. Em conclusão, a expressão do gene WFDC1 e de sua variante WFDC1 correlaciona-se com a diferenciação das células musculares lisas, mas não está condicionada a ela, enquanto a expressão de Delta 3 é completamente independente deste parâmetro e tem correlação positiva com o progressão do PCA, quando o sistema de classificação de Gleason (Gleason 1 + Gleason 2) foi considerado. Adicionalmente, fatores independentes da idade, incidência de BPH ou PCA, são mais influentes na determinação da quantidade total e da proporção entre as duas variantes
Abstract: The prostate gland is intimately related with reproductive and urinary functions, commonly disturbed by a series of diseases. Besides reducing the quality of life, they consist in serious life risk particularly to the aging men. Dynamic interactions between the epithelium and stroma in the gland regulate various aspects of development, function and pathologies. The WFDC1 gene is expressed by smooth muscle cells in the prostate stroma and its product ps20 was shown to control epithelial cell behavior, extracellular matrix organization and angiogenesis. It is supposed to function as a serine protease inhibitor, as other members of its family do. Two transcripts are produced as a result of alternative splicing. The first (WFDC1) retains all exons and the second (Delta 3) lacks the exon 3. In this work, we investigated the quantitative relationship among these two splicing variants, using qRT-PCR (Taqman) probing the junctions between exons 2-3 and 2-4, in benign prostatic hyperplasia (BPH) and prostate cancer (PCA) samples from tissue banks. The expression of the MYH11 gene was used to estimate the content of smooth muscle cells in the samples. The results demonstrate that the samples could be divided in two groups with low or high expression of MYH11, the first corresponding to the lower quartile of expression values). WFDC1 and MYH11 expression were correlated in the BPH samples. Delta 3 expression was independent of both WFDC1 and from WFDC1. The ratio between the variants WFDC1 and Delta 3 varied exponentially in five orders of magnitude. The the ratio between the two variants also varied exponentially, with BPH and PCA samples arranged in discrete and intercalated subgroups. The distribution of populations with different expression levels preserved the ratios 10:1, 1:1 and 1:3. Either variant was absent in only a few samples. In conclusion, the expression of WFDC1 and it WFDC1 variant correlates with but is not conditioned to the differentiation of smooth muscle cells, while Delta 3 is completely independent and is positively correlated with PCA grade (as assessed by the summed Gleason score). Unknown factors independent of age, BPH or PCA incidence are likely influencing Delta 3 expression
Doutorado
Biologia Celular
Doutor em Biologia Celular e Estrutural
Holt, Jim. "Prostate Cancer." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/etsu-works/6456.
Full textAmbrosini, Gina L. "Dietary risk factors for prostate cancer and benign prostatic hyperplasia." University of Western Australia. School of Population Health, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0135.
Full textBooks on the topic "Prostate"
I, Resnick Martin, and Thompson Ian M. 1954-, eds. Surgery of the prostate. New York: Churchill Livingstone, 1998.
Find full textHerbert, Lepor, and Lawson Russell K, eds. Prostate diseases. Philadelphia: W.B. Saunders, 1993.
Find full textNational Institutes of Health (U.S.), ed. Prostate enlargement: Benign prostatic hyperplasia. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1990.
Find full textK, Oh William, and Logue John, eds. Prostate cancer. Edinburgh: Mosby Elsevier, 2007.
Find full textZerbib, Marc. La prostate. Paris: Doin, 2001.
Find full textS, Ernstoff Marc, Heaney John A, and Peschel Richard E, eds. Prostate cancer. Malden, Mass: Blackwell Science, 1998.
Find full textBostwick, David G. Biopsy pathology of prostate. London: Chapman & Hall, 1997.
Find full textA, Dawson Nancy, and Vogelzang Nicholas, eds. Prostate cancer. New York, USA: Wiley-Liss, 1994.
Find full textP, Karr James, Yamanaka Hidetoshi, and Tokyo Symposium on Prostate Cancer (2nd : 1987), eds. Prostate cancer. New York: Elsevier, 1989.
Find full text1941-, Ackermann R., and Schröder F. H, eds. Prostatic hyperplasia: Etiology, surgical and conservative management. Berlin: De Gruyter, 1989.
Find full textBook chapters on the topic "Prostate"
Good, Daniel W., Bashar Nahas, Simon Phipps, Rick Popert, Jens-Uwe Stolzenburg, and Stuart Alan S. McNeill. "Prostate Benign Prostatic Hyperplasia." In Blandy's Urology, 531–61. Chichester, UK: John Wiley & Sons, Ltd, 2019. http://dx.doi.org/10.1002/9781118863343.ch27.
Full textO’Rourke, Declan M., and Derek C. Allen. "Prostate." In Histopathology Specimens, 307–18. London: Springer London, 2012. http://dx.doi.org/10.1007/978-0-85729-673-3_31.
Full textO’Rourke, Declan M., and Derek C. Allen. "Prostate." In Histopathology Specimens, 337–52. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57360-1_31.
Full textBostwick, David G., and Liang Cheng. "Prostate." In Essentials of Anatomic Pathology, 1219–46. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1007/978-1-60327-173-8_31.
Full textPickuth, Dirk. "Prostate." In Essentials of Ultrasonography, 191–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79579-4_13.
Full textHruban, Ralph H., William H. Westra, Timothy H. Phelps, and Christina Isacson. "Prostate." In Surgical Pathology Dissection, 146–49. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4757-2548-3_27.
Full textAlgaba, Ferran, Steven S. Shen, Luan D. Truong, and Jae Y. Ro. "Prostate." In Frozen Section Library: Genitourinary Tract, 115–35. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-0691-5_4.
Full textLierse, Werner. "Prostate." In Applied Anatomy of the Pelvis, 160–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71368-2_7.
Full textBostwick, David G., and Liang Cheng. "Prostate." In Essentials of Anatomic Pathology, 1751–88. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23380-2_38.
Full textCanepa, Carlo, and Eugene M. Fine. "Prostate." In Atlas of Handheld Ultrasound, 173–76. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73855-0_33.
Full textConference papers on the topic "Prostate"
Kim, Beop-Min, Martin Ostermeyer, Steven L. Jacques, David A. Levy, Pradip Chakrabarti, Jorge H. Torres, Andrew C. von Eschenbach, Sohi Rastegar, and Massoud Motamedi. "In vivo optical property measurement using intraluminal fiber reflectometry : Optical properties of canine and human prostates." In Biomedical Optical Spectroscopy and Diagnostics. Washington, D.C.: Optica Publishing Group, 2006. http://dx.doi.org/10.1364/bosd.1996.ap3.
Full textArmato, Samuel G., Nicholas A. Petrick, and Karen Drukker. "PROSTATEx: Prostate MR Classification Challenge (Conference Presentation)." In Computer-Aided Diagnosis, edited by Samuel G. Armato and Nicholas A. Petrick. SPIE, 2017. http://dx.doi.org/10.1117/12.2280374.
Full textKhoshnevis, Sepideh, and Kenneth R. Diller. "Response of Normal Canine Prostate Tissue to Thermal Therapy." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192825.
Full textValvano, Jonathan W., David Y. Yuan, Eric N. Rudie, and Steven J. Clark. "Treatment of Benign Prostatic Hyperplasia." In ASME 1996 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1996. http://dx.doi.org/10.1115/imece1996-0741.
Full textZhu, Liang, Zhengxin Mi, and Lisa X. Xu. "Temperature Distribution in Prostate During Transurethral Radio Frequency Thermotherapy Treatment of Benign Prostatic Hyperplasia." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0806.
Full textBhowmick, Sankha, and John C. Bischof. "Supraphysiological Thermal Injury in Dunning AT-1 Prostate Tumor Cells." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0798.
Full textPadasdao, Blayton, and Bardia Konh. "SYSTEMATIC 12-CORE TRANSPERINEAL PROSTATE BIOPSY WITH MINIMAL ACTIVE NEEDLE INSERTIONS IN A PATIENT PROSTATE-SIZED PHANTOM." In 2023 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2023. http://dx.doi.org/10.1115/dmd2023-3205.
Full textLi, Dian-Ru, Jih-Kai Yeh, Wei-Chen Lin, Jeffrey S. Montgomery, and Albert Shih. "An Experimental Method of Needle Deflection and Prostate Movement Using the Anatomically Accurate Prostate Simulator and the Electromagnetic Tracking System." In ASME 2017 12th International Manufacturing Science and Engineering Conference collocated with the JSME/ASME 2017 6th International Conference on Materials and Processing. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/msec2017-3000.
Full textLiang, Haipeng, Wanli Zuo, Dimitri Kessler, Tristan Barrett, and Zion Tsz Ho Tse. "MRI-Guided Robotic Prostate Biopsy." In THE HAMLYN SYMPOSIUM ON MEDICAL ROBOTICS. The Hamlyn Centre, Imperial College London London, UK, 2023. http://dx.doi.org/10.31256/hsmr2023.6.
Full textBauer, John J., Jianchao Zeng, Wei Zhang, Isabell A. Sesterhenn, Robert Dean, Judd W. Moul, and Seong K. Mun. "Simulated prostate biopsy: prostate cancer distribution and clinical correlation." In Medical Imaging 2000, edited by Seong K. Mun. SPIE, 2000. http://dx.doi.org/10.1117/12.383030.
Full textReports on the topic "Prostate"
Sciacqua, Lucilla Violetta, Andrea Vanzulli, Rosario Di Meo, Giuseppe Pellegrino, Roberto Lavorato, Giovanni Vitale, and Gianpaolo Carrafiello. Minimally invasive treatment in Benign Prostatic Hyperplasia (BPH). INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0004.
Full textPurnell, Jonathan. Imaging Prostatic Lipids to Distinguish Aggressive Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada609940.
Full textShannon, Jackilen. Imaging Prostatic Lipids to Distinguish Aggressive Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada612470.
Full textShannon, Jackilen. Imaging Prostatic Lipids to Distinguish Aggressive Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada594175.
Full textPurnell, Jonathan. Imaging Prostatic Lipids to Distinguish Aggressive Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2015. http://dx.doi.org/10.21236/ada626329.
Full textCooney, Kathleen A. Prostate Cancer Aggressiveness Genes in Hereditary Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, March 2005. http://dx.doi.org/10.21236/ada446359.
Full textCooney, Kathleen A., and Colin Duckett. Prostate Cancer Aggressiveness Genes in Hereditary Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, March 2006. http://dx.doi.org/10.21236/ada455279.
Full textCooney, Kathleen. Prostate Cancer Aggressiveness Gene in Hereditary Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, March 2007. http://dx.doi.org/10.21236/ada472110.
Full textHeidari, Afshin, Aida Kazemi, Parisa Najjari, Kamran Dalvandi, Hamidreza Sadeghsalehi, Parinaz Onikzeh, and Hadi Zamanian. Comparing Urinary and Sexual Complications of Robot-Assisted Radical Prostatectomy and Laparoscopic Radical Prostatectomy in Prostate Cancer: a Systematic Review and Meta-Analysis Protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0068.
Full textNavone, Nora M. Osteoblast-Prostate Cancer Cell Interaction in Prostate Cancer Bone. Fort Belvoir, VA: Defense Technical Information Center, February 2000. http://dx.doi.org/10.21236/ada391088.
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