Relevant bibliographies by topics / Proliferate

Academic literature on the topic 'Proliferate'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Proliferate"

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Matsue, H., P. R. Bergstresser, and A. Takashima. "Keratinocyte-derived IL-7 serves as a growth factor for dendritic epidermal T cells in mice." Journal of Immunology 151, no. 11 (December 1, 1993): 6012–19. http://dx.doi.org/10.4049/jimmunol.151.11.6012.

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Abstract Dendritic epidermal T cells (DETC) are CD3+, CD45+, CD4-, CD8-, TCR-V gamma 3/V delta 1+ T lymphocytes that reside in symbiosis with keratinocytes in mouse epidermis. To address mechanisms by which these cells survive and proliferate within the epidermal environment, we have tested the hypothesis that cytokines secreted by neighboring keratinocytes play relevant roles. The present study was conducted to determine whether keratinocytes produce biologically relevant amounts of IL-7, and, if so, to study its effects on DETC. The long term cultured DETC line, 7-17, and freshly isolated DETC exhibited dose- and time-dependent proliferative responses to rIL-7. These responses were blocked completely by anti-IL-7 antibodies, whereas anti-IL-2 had no effect, indicating that DETC respond to IL-7 by an IL-2-independent mechanism. Proliferative responses depended on the state of cell activation; DETC stimulated 2 to 5 days earlier with Con A proliferated optimally to added IL-7, whereas cells stimulated 10 days earlier did not proliferate. DETC that failed to proliferate responded to IL-7 by showing prolonged survival; cells maintained for 7 days with IL-7 alone retained their capacity to proliferate in response to Con A. Mouse epidermal cells and Pam 212 keratinocyte line both expressed IL-7 mRNA constitutively, as demonstrated by reverse transcription-polymerase chain reaction analyses. The production of IL-7 by mouse keratinocytes was also confirmed; Pam 212 culture supernatants supported DETC proliferation, and this activity was diminished by 50% with added anti-IL-7 antibodies. These results indicate that keratinocytes produce IL-7 in biologically relevant amounts, which, in turn, serve to promote the survival and growth of DETC. IL-7-mediated communication between epithelial cells and gamma delta T cells may represent one mechanism to sustain the indefinite residence of gamma delta T cells in epithelial tissues of mice.
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Dempsey, E. C., I. F. McMurtry, and R. F. O'Brien. "Protein kinase C activation allows pulmonary artery smooth muscle cells to proliferate to hypoxia." American Journal of Physiology-Lung Cellular and Molecular Physiology 260, no. 2 (February 1, 1991): L136—L145. http://dx.doi.org/10.1152/ajplung.1991.260.2.l136.

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Pulmonary artery (PA) smooth muscle cell (SMC) proliferation occurs with hypoxic pulmonary hypertension in vivo. However, proliferation of cultured PA SMC to hypoxia has not been demonstrated, and thus the mechanism by which these cells respond to hypoxia is unknown. Because protein kinase C (PKC) plays a role in intracellular transduction of proliferative signals, we asked whether PKC activation 1) causes proliferation of bovine PA SMC and 2) is important in PA SMC proliferative response to hypoxia. By measuring [3H]thymidine incorporation and cell counts, we found that quiescent PA SMC from four different cows proliferated with the PKC activator, phorbol 12-myristate 13-acetate (PMA), in a concentration-dependent manner. The proliferation was blocked with a PKC inhibitor, dihydrosphingosine, or by downregulating SMC PKC. We tested whether “priming“ PA SMC by PKC activation was required for in vitro SMC proliferative response to hypoxia. Each SMC population was treated with PMA and then exposed for 24 h to 20, 10, 7, 3 or 0% O2. These cells proliferated with hypoxia reaching a peak response at 3% O2. The magnitude of the response to PMA and hypoxia was different for each cell population tested. No hypoxic proliferation occurred in control cells (no PMA). Dihydrosphingosine blocked the hypoxic response to the same extent that it inhibited the initial PMA conditioning stimulus. PKC-downregulated PA SMC did not proliferate to PMA or to subsequent hypoxia. The hypoxic response was not due to a reduction in O2 radical-mediated antiproliferative effect; rather, the PMA-primed cells seemed to “acquire” the ability to directly sense hypoxia and proliferate. In summary, PKC activation caused proliferation of PA SMC in vitro and allowed an additional proliferative response to hypoxia. Activation of PKC may be a requisite step for PA SMC to respond directly to hypoxia.
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Turco, MC, M. De Felice, F. Alfinito, A. Lamberti, F. Costanzo, M. Giordano, V. Martinelli, B. Rotoli, S. Ferrone, and S. Venuta. "Proliferative pathways in CD1- CD3+ CD4+ CD8+ T-prolymphocytic leukemic cells: analysis with monoclonal antibodies and cytokines." Blood 74, no. 5 (October 1, 1989): 1651–57. http://dx.doi.org/10.1182/blood.v74.5.1651.1651.

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Abstract The antigenic profile and the proliferative pathways in leukemic cells from the patient TRT with T-prolymphocytic leukemia (T-PLL) were analyzed using monoclonal antibodies (MoAbs) and cytokines. T-PLL cells expressed the phenotype CD1- CD3+ CD4+ CD8+. Incubation with the differentiating agent phorbol-12-myristate-13-acetate markedly increased the percentage of cells with the CD4- CD8+ phenotype, suggesting that leukemic cells were already committed towards a differentiated element with the CD4- CD8+ phenotype. T-PLL cells were induced to proliferate by anti-CD2 MoAb 9–1 + 9.6 and by anti-CD3 MoAb OKT3. The two pathways exhibited normal functional interactions and were susceptible to modulation by anti-HLA class I MoAbs. These results indicate that regulation of cell proliferation was preserved to a significant extent in the T-PLL cells analyzed. At variance with normal resting T cells that require previous activation to proliferate when incubated with interleukin-1 (IL-1) or interleukin-2 (IL-2), T-PLL cells proliferated vigorously when incubated with either interleukin. Furthermore, T-PLL cells proliferated when incubated with immune interferon (IFN-gamma). The latter finding parallels the enhancement by IFN-gamma of the proliferative response of lectin-activated murine T lymphocytes. These results suggest that T-PLL cells, which express a high constitutive level of c-myc mRNA, may be in an activated state. The antigenic phenotype and the characteristics of the proliferative pathways of T-PLL cells from the patient TRT are compatible with the possibility that they may be derived from an intermediate thymocyte.
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Turco, MC, M. De Felice, F. Alfinito, A. Lamberti, F. Costanzo, M. Giordano, V. Martinelli, B. Rotoli, S. Ferrone, and S. Venuta. "Proliferative pathways in CD1- CD3+ CD4+ CD8+ T-prolymphocytic leukemic cells: analysis with monoclonal antibodies and cytokines." Blood 74, no. 5 (October 1, 1989): 1651–57. http://dx.doi.org/10.1182/blood.v74.5.1651.bloodjournal7451651.

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The antigenic profile and the proliferative pathways in leukemic cells from the patient TRT with T-prolymphocytic leukemia (T-PLL) were analyzed using monoclonal antibodies (MoAbs) and cytokines. T-PLL cells expressed the phenotype CD1- CD3+ CD4+ CD8+. Incubation with the differentiating agent phorbol-12-myristate-13-acetate markedly increased the percentage of cells with the CD4- CD8+ phenotype, suggesting that leukemic cells were already committed towards a differentiated element with the CD4- CD8+ phenotype. T-PLL cells were induced to proliferate by anti-CD2 MoAb 9–1 + 9.6 and by anti-CD3 MoAb OKT3. The two pathways exhibited normal functional interactions and were susceptible to modulation by anti-HLA class I MoAbs. These results indicate that regulation of cell proliferation was preserved to a significant extent in the T-PLL cells analyzed. At variance with normal resting T cells that require previous activation to proliferate when incubated with interleukin-1 (IL-1) or interleukin-2 (IL-2), T-PLL cells proliferated vigorously when incubated with either interleukin. Furthermore, T-PLL cells proliferated when incubated with immune interferon (IFN-gamma). The latter finding parallels the enhancement by IFN-gamma of the proliferative response of lectin-activated murine T lymphocytes. These results suggest that T-PLL cells, which express a high constitutive level of c-myc mRNA, may be in an activated state. The antigenic phenotype and the characteristics of the proliferative pathways of T-PLL cells from the patient TRT are compatible with the possibility that they may be derived from an intermediate thymocyte.
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Lee, Y. "To proliferate or not to proliferate." Cardiovascular Research 86, no. 3 (April 7, 2010): 347–48. http://dx.doi.org/10.1093/cvr/cvq107.

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Gilbert, K. M., K. D. Hoang, and W. O. Weigle. "Th1 and Th2 clones differ in their response to a tolerogenic signal." Journal of Immunology 144, no. 6 (March 15, 1990): 2063–71. http://dx.doi.org/10.4049/jimmunol.144.6.2063.

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Abstract Th1 and Th2 clones specific for human gamma globulin (HGG) were compared and shown to differ in terms of the effects of tolerance induction on Ag-induced proliferation and helper activity. In developing a method to induce tolerance, splenic APC that had been pulsed with HGG and then fixed with 0.15% paraformaldehyde (HGG-FAPC) were used as a means to present Ag to the Th clones in the absence of costimulatory signals. Both Th1 and Th2 clones recognized HGG-FAPC as evidenced by their ability to proliferate to HGG-FAPC. Unlike Th2, Th1 proliferated to HGG-FAPC only in the presence of T cell-depleted allogeneic spleen cells as a source of accessory cell signals. The inability of Th1 cells to proliferate in the absence of costimulatory signals was due to Ag-specific inactivation: Th1 clones preincubated with HGG-FAPC were unable to proliferate when recultured with HGG and irradiated APC. In contrast to Th1 clones, Th2 clones showed no decrease in their Ag-induced proliferative capacity after exposure to any concentration of HGG-FAPC. However, when examined by using a second assay system, that of providing help for anti-HGG antibody production by primed B cells, Th2 preincubated with HGG-FAPC were markedly inhibited (up to 90%) in their ability to provide help. Preincubation with HGG-FAPC also inhibited the helper activity of the one Th1 clone that was found to induce a significant secondary antibody response. Taken together, the results suggest that exposure of Th1 to tolerogen in the form of HGG-pulsed fixed APC inactivates Th1 proliferative capacity, and possibly Th1 helper activity as well. Exposure of Th2 cells to a tolerogen suppresses the mechanism by which the Th2 cells provide Ag-induced B cell help, but does not inhibit the mechanism by which they proliferate to HGG. Furthermore, the results define a model that incorporates Ag processing as well as Ag presentation in the induction of tolerance in vitro.
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Leiper, James M., M. Birdsey Graeme, and Paru B. Oatey. "Peroxisomes proliferate." Trends in Cell Biology 5, no. 11 (November 1995): 435–37. http://dx.doi.org/10.1016/s0962-8924(00)89103-5.

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Stajic, Jelena. "Triplets Proliferate." Science 336, no. 6077 (April 5, 2012): 13.1–13. http://dx.doi.org/10.1126/science.336.6077.13-a.

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Defrance, T., J. P. Aubry, B. Vanbervliet, and J. Banchereau. "Human interferon-gamma acts as a B cell growth factor in the anti-IgM antibody co-stimulatory assay but has no direct B cell differentiation activity." Journal of Immunology 137, no. 12 (December 15, 1986): 3861–67. http://dx.doi.org/10.4049/jimmunol.137.12.3861.

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Abstract In this study it is illustrated that recombinant human interferon-gamma (IFN-gamma) acts as a B cell growth factor (BCGF) in the anti-IgM antibody co-stimulatory assay. A monoclonal antibody that specifically inhibits the biological activities of IFN-gamma blocks its BCGF activity supporting the specificity of the IFN-gamma effect. Various IFN-gamma obtained from different sources displayed the same BCGF activity. Nonactivated B lymphocytes do not proliferate in response to IFN-gamma. IFN-gamma acts directly on B cells because highly purified B cells obtained after standard purification procedures coupled to cell sorting could still proliferate in response to IFN-gamma. Blood B lymphocytes were found to be more sensitive to the BCGF activity of IFN-gamma than B cells obtained from spleens or tonsils. The IFN-gamma-induced proliferation of B cells was short lasting when compared with that of recombinant IL 2 or BCGF containing T cell clone supernatants. B cells preactivated with either Staphylococcus aureus strain Cowan I (SAC) or optimal concentrations of anti-IgM antibodies coupled to beads did not proliferate in response to IFN-gamma, whereas they proliferated in response to IL 2 or T cell clone supernatants. IFN-gamma did not stimulate nor inhibit the proliferative response of human B lymphocytes stimulated with optimal concentrations of anti-IgM antibodies or SAC. Additionally none of the different IFN-gamma tested had B cell differentiation factor activity in the standard SAC assay. These results indicate that IFN-gamma sensitizes B cells to suboptimal mitogenic concentrations of anti-IgM antibody.
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Vallée, Isabelle, Jean-Maurice Guillaumin, Gilles Thibault, Yves Gruel, Yvon Lebranchu, Pierre Bardos, and Hervé Watier. "Human T Lymphocyte Proliferative Response to Resting Porcine Endothelial Cells Results from an HLA-Restricted, IL-10-Sensitive, Indirect Presentation Pathway But Also Depends on Endothelial-Specific Costimulatory Factors." Journal of Immunology 161, no. 4 (August 15, 1998): 1652–58. http://dx.doi.org/10.4049/jimmunol.161.4.1652.

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Abstract To investigate the mechanisms of cellular rejection in pig-to-human xenotransplantation, the proliferation of different human purified lymphocyte subpopulations in response to swine leukocyte Ag class II-negative porcine aortic endothelial cells (PAEC) was measured in the presence or absence of human autologous adherent cells (huAPC). CD8+ lymphocytes proliferated moderately in the absence of huAPC, and the immune response was slightly increased when huAPC were added. CD56+ lymphocytes failed to proliferate in response to PAEC whether huAPC were present or not. CD4+ lymphocytes alone did not proliferate in response to PAEC, but a strong proliferative response was observed in the presence of metabolically active huAPC. This response was totally abolished by mAbs directed against HLA class II molecules or by pretreatment of huAPC by human IL-10. Even in the presence of huAPC, CD4+ lymphocytes failed to respond to fixed PAEC or to PAEC-lysates, suggesting that PAEC must be viable to support lymphocyte proliferation. Finally, none of the nonendothelial porcine adherent cells tested was able to induce human lymphocyte proliferation, despite the fact that they also provided a large set of xenogeneic peptides. Our results show that the indirect presentation pathway of xenoantigens by huAPC to CD4+ lymphocytes is crucial in the response to porcine endothelial cells, and that IL-10 could be of therapeutic interest to prevent human lymphocyte activation by this pathway. Furthermore, we demonstrated that stimulatory signals specifically provided by endothelial cells are also necessary for this huAPC-restricted proliferative response.
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Dissertations / Theses on the topic "Proliferate"

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Shen, Qian. "Histoplasma circumvents nutrition limitations to proliferate within macrophages." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1563371073816792.

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Mmatli, Thato. "Do I belong here? Conditions and micro-diffussions in the South African milieu which proliferate the emigration of potential leaders." Thesis, Linnéuniversitetet, Institutionen för organisation och entreprenörskap (OE), 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-65126.

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A plethora of interdisciplinary research has highlighted the increase of global mobility and diasporic communities. While talent emigration has shown to have a multiplicity of benefits, particularly as gaining diversified sets of skills is essential in the face of globalization. However, widely researched concepts such as the ‘brain drain’ have conveyed the dark side of talent emigration and the ramification of countries’ desiccation for scarce skills. With a history fraught with tensions and immense loss of talent, South Africa is a country in continuous transformation, but is on the cusp of another significant ‘brain drain’. Hence, this study aimed to explore the micro-diffusions and conditions in the South African context which proliferate the emigration of talented potential leaders. The research design was qualitative, with specific use of the actors approach as methodology to gain insight into perspectives of South Africans living, working and studying in Sweden. Twenty-one participants from five cities were involved in the focus group dialogues, namely; Gothenburg, Kalmar, Lund, Linköping and Stockholm. As a participant-observer, I too was involved in the sense-making of how talent delineated their identities and relation to South Africa. Certain aspects of the findings were expected regarding the conditions which serve as push factors for emigration, such as participants’ frustrations and despondency with increasing rates of crime, unemployment and corruption. However, the most accentuated and poignant micro-diffusion which perpetuates talent’s emigration derives from conflicts of identity and belongingness, deep-seated inherited guilt and helplessness. Whilst there is a desire to ameliorate the social ills which plague the country, there also seems to be a palpable need for escapism away from the persistent historical complexities of South Africa.
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Farmer, Michael L. "Why Iran proliferates." Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 2005. http://library.nps.navy.mil/uhtbin/hyperion/05Sep%5FFarmer.pdf.

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Villada, Castro Carolina. "O proliferar dos outros." reponame:Repositório Institucional da UFSC, 2016. https://repositorio.ufsc.br/xmlui/handle/123456789/175904.

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Dissertação (mestrado) - Universidade Federal de Santa Catarina, Centro de Comunicação e Expressão, Programa de Pós-Graduação em Estudos da Tradução, Florianópolis, 2016.
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Esta pesquisa apresenta uma análise teórica da tradução para o espanhol do texto "Apontamentos para uma poética xamânica do traduzir" do escritor e tradutor Álvaro Faleiros (2012), assim como das traduções indiretas para o espanhol da seleção de cantos xamânicos marubo: ?Raptada pelo raio? (Kaná kawã,), ?Pajé Flor de Tabaco? (Rome owe romeya) e ?Origem da vida breve? (Roka), compendiados no trabalho etnopoético de Niemeyer Cesarino em: Quando a terra deixou de falar. Cantos da mitologia Marubo (2013). A fim de pesquisar imagens do traduzir nos cantos xamânicos araweté e marubo, estabeleceu-se uma articulação entre a enunciação polifônica do xamã e a tradução literária, como atos de reverberação e multiplicação de vozes outras. Nessa perspectiva, ao modo do xamã, o tradutor literário age como voz entre vozes e prolonga os caminhos dessas vozes alheias vindas de alhures, que proliferam no espaço literário. O corpus está composto pelas contribuições tradutológicas de Álvaro Faleiros (2012-2015), as contribuições etnopoéticas de Viveiros de Castro (1986) e Niemeyer Cesarino (2011 e 2013). Desse modo, articulam-se diversas ferramentas metodológicas tais como: análise conceitual, análise comparada de traduções, tradução direta e indireta comentada e análise poética das imagens dos cantos xamânicos. Tudo isto almeja responder à força ética do canto xamânico e seus ecos para a prática de tradução literária.

Abstract : This study presents a theoretical analysis of the Spanish translation of the text "Apontamentos para uma poética xamânica do traduzir" authored by the Brazilian writer and translator Álvaro Faleiros (2012a), as well as the Spanish indirect translation of a selection of Marubo shamanic singings: "Raptada pelo raio" (kaná kawã,), "Pajé Flor de Tabaco" (Rome owe romeya) and "Origem da vida breve" (Roka), assambled in the ethnopoetic work of Niemeyer Cesarino: Quando a terra deixou de falar. Cantos da mitologia Marubo (2013). Our main purpose is to investigate images of translation in the ?shamanic singing? Arawete and Marubo, articulating the polyphony of shamanic enunciations and literary translation as acts of reverberation and multiplication of other voices. From this perspective, as a shaman, we see the literary translator performing as a voice among other voices and extending the paths that distant voices bring with them; voices that proliferate within the literary space. Our framework is formed by the works of Álvaro Faleiros (2012-2015) and the ethnopoetic works of Viveiros de Castro (1986) and Niemeyer Cesarino (2011, 2013). We also put into play the following methodological tools: conceptual analysis, comparative analysis of translations, direct and indirect translation, and poetic analysis of shamanic singing images. With these instruments, we attempt to re-join the ethical strength of the shamanic singing and to direct its echoes to the practice of literary translation.
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von, Hehl Stephanie. "Vergleich der Wirkung verschiedener Wachstumsfaktoren auf physiologische Parameter kultivierter humaner retinaler Pigmentepithelzellen." Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-80080.

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Bei der proliferativen Retinopathie (PVR) kommt es aufgrund einer Netzhautablösung zu einer Störung der Blut-Retina-Schranke. Durch die daraus resultierende Milieuänderung wandern retinale Zellen und Bestandteile des Blutes in den Glaskörper ein. Wachstumsfaktoren regen die mitotisch inaktiven RPE-Zellen zur Migration und Proliferation an. Im weiteren Verlauf der Erkrankung führt dies zur Ausbildung von Traktionsmembranen und zur Ablösung der Netzhaut. Die dadurch entstehenden Zugkräfte verhindern eine Wiederanlegung der Netzhaut. Ziel dieser Arbeit war es, zu untersuchen, welchen Einfluss Wachstumsfaktoren und deren Kombinationen auf die Proliferation und Migration humaner retinaler Pigmentepithelzellen, die VEGF-Sekretion durch RPE-Zellen und die mRNA-Expression von Kollagenen haben. Die beteiligten Signalwege sollten ebenfalls ermittelt werden. Im Ergebnis sollten Faktoren identifiziert werden, die die Entstehung einer PVR begünstigen. Migration, Proliferation, VEGF-Sekretion und die TGF-ß-Sekretion konnten durch den Wachstumsfaktor PDGF (Platelet-derived growth factor) gesteigert werden. Die Wirkung des PDGF-Signals wurde durch die Aktivierung verschiedener intrazellulärer Signalwege (MAPK, p38MAPK, PI3K/AKT) vermittelt. PDGF wirkte hemmend auf die Kollagen mRNA-Expression. Für TGF-ß (Transforming growth factor-ß) wurde eine hemmende Wirkung auf die Proliferation und eine induzierende Wirkung auf die Kollagensynthese nachgewiesen. Beide Faktoren – PDGF und TGF-ß – steigerten die VEGF-Sekretion. Eine additive Erhöhung der VEGF-Sekretion wurde durch die Kombination beider Faktoren nachgewiesen. Die untersuchten Wachstumsfaktoren interagieren mit den humanen retinalen Pigmentepithelzellen und tragen somit maßgeblich zur Ausbildung und auch zur Beschleunigung des Verlaufs der proliferativen Retinopathie sowie der pathologischen Gefäßneubildung bei.
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Velde, Guillaume Paulus Marcus ten. "Cell proliferation and tumour growth of lung cancer a clinical, immunohistochemical and flow cytometric study /." Maastricht : Maastricht : Datawyse ; University Library, Maastricht University [Host], 1989. http://arno.unimaas.nl/show.cgi?fid=5493.

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Boers, James Evan. "Composition and proliferation of normal human tracheobronchial mucosa." Maastricht : Maastricht : Datawyse/Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=5926.

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McOrist, S. "The aetiology of the proliferative enteropathies." Thesis, University of Edinburgh, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383022.

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The entezopathogenicity and antigens of Campylobacter-like organisms and Campylobacter app associated with the proliferative enteropathies were investigated. Two gnotobiotic pigs exposed orally to a filtered suspension of intestinal mucosa designated 284/86 from a naturally infected pig subsequently developed lesions of proliferative enteritis. Culture of the successful mucosal inoculum only revealed a moderate number of C. coli, however an apparently greater number of Campylobacter-like organisms was evident in smears of this inoculum. The pathogenesis of porcine proliferative enteritis was clearer from the results of this study. Ten days after infection, curved bacilli had colonised the ileal and large intestinal crypts. Attachment and entry of Campylobacter-like organisms into crypt enterocytes was also evident, with some proliferation of both bacteria within cells and of the enterocytes themselves. Twenty days after infection there was similar intracellular colonisation of bacteria and proliferative activity, although no luminal bacteria were evident. A moderate sub-acute inflammatory reaction was evident throughout. Conventional hamsters dosed with C. jejuni developed varying degrees of localised acute intestinal inflammation. Hamsters dosed with C. hyointestinalis or C. cola did not develop any lesions. Lesions of proliferative enteritis were detected in hamsters dosed with porcine tissue 284/86. Numerous intra-cytoplasmic Campylobacter-like organisms were detected within enterocytes in affected portions of intestine. Weanling hamsters this proved to be susceptible to the agent of porcine proliferative enteritis by cross-species transmission. Whole cell antigen preparations were made of various Camoylobacter sp. -Indirect immunofluorescence assays incorporating rabbit antisera to each Campylobacter sp gave specific endpoints for each antiserum of 1: 160 to 1: 320. Rabbit antisera prepared to Campylobacter-like organisms partly purified from proliferative enteritis mucosa, by a homogenisation and filtration technique, also gave specific reactions in this assay, up to 1: 610. Intracellular Campylobacter-like organisms were also compared in gel electrophoresis protein profiles and immunoblotting reactions to Campylobacter spp. The intracellular organisms tested had a distinctive protein profile dissimilar to the profiles of the known Campylobacter spp. In immunoblotting reactions, each of the Camoylobacter sp antisera reacted strongly with homologous antigens, but none reacted with Camcylobacter-like organisms prepared from lesions, except for a minor reaction seen with one serum. Similarly antisera to Campylobacter-like organisms showed a strong reaction to 25K to 27K components of homologous antigens, with only minor reactions to various other components of the cultivated Campylobacter app. Therefore it is likely that the intracellular Campylobacter-like organisms have a distinctive antigenic profile and that the 25 and 27K components are major antigenic components. Mouse monoclonal antibodies were produced that were apparently specific to the intracellular Camoylobacter-like organisms. Immunoblotting results showed that these antibodies only bound to a 251 to 27K outer membrane component present in the intracellular organisms. Reactions with this component could not be detected in assays with normal pig intestine, or Camoylobacter sp antigen. Restriction endonuclease digestion of Camoylobacter sp with Bgl II gave suitably resolved DNA fragments of between 2kb and 25kb. Patterns obtained with Bgl II digestion of Camoylobacter sp were dissimilar to those of Camoylobacter-like organisms, and each Camoylobacter sp had a characteristic distinct pattern. Digestion of DNA from porcine tissue samples with Bgl II produced a diffuse smear of fragmented DNA bands between 0.5 and19kb, with no recognizable "ladder" effect. The genome of the Carylobacter-like organisms within enterocytes in proliferative enteritis therefore is different to that of known Camoylobacter spp associated with the disease. This suggests that the differences in antigenic structures between these bacteria axe due to genetic differences. Only a limited number of strains were examined. Looking at the evidence provided by this study, the overall tenor of the results suggests that the intracellular organisms could be a separate, new species of Camp lobacter. If indeed the intracellular organisms are a single, new Cammylobacter PGS/ABST ecies, then a new name may be proposed, such as "C. intracellulare". Verifica nthis side Oni of the validity of such a proposal would require further DNA studies.
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Smith, Sionagh Helen. "Epidemiological features of porcine proliferative enteropathy." Thesis, University of Edinburgh, 1997. http://hdl.handle.net/1842/30774.

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The polymerase chain reaction (PCR) assay was adapted and optimised for specific detection of Lawsonia intracellularis genomic DNA segment in swine faeces. Lawsonia intracellularis is the aetiological agent of porcine proliferative enteropathy (PPE) and the PCR represents the first diagnostic test suitable for ante-mortem use in affected swine. Various methods designed to extract bacterial DNA from faeces were evaluated to establish a convenient and optimum protocol. The PCR was utilised in pig challenge studies to investigate the excretion patterns of L. inracellularis in weaner pigs orally inoculated with pure cultures of L. intracellularis. This challenge work demonstrated that the PCR was a suitable tool for detection of infection, and indicated that individual animals could excrete L. intracellularis organisms for periods of up to ten weeks post-challenge. Such an excretion period has major implications for the transmission of organisms in the field. For example, if infected growers are still shedding L. intracellularis organisms upon entry to the breeding population, then this is a possible route for the transmission of disease to younger, susceptible pigs. A more extensive, two-part investigation of the epidemiological aspects of PPE in the field followed. The investigation comprised a farm sampling study and a questionnaire postal survey. In the farm sampling study, faeces samples were collected serially over a ten month period from breeding gilts and their litters. Samples were subjected to PCR for the detection of infection, allowing estimation of within-herd prevalence, as well as determination of possible transmission patterns. The assay successfully detected the presence of L. intracellularis in the weaners and/or growers of three of the five farms selected for this study. The within-herd prevalence for these age-groups ranged from 10 to 30%. The PCR also confirmed infection in several of the adult breeding boars and gilts.
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Bailey, Catherine Clare. "A prospective national survey of laser treatment for diabetic retinopathy." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285814.

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Books on the topic "Proliferate"

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Freeman, H. MacKenzie, and Felipe I. Tolentino, eds. Proliferative Vitreoretinopathy (PVR). New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3910-9.

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Youssef, Jihan A., and Mostafa Z. Badr. Peroxisome Proliferator-Activated Receptors. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-420-3.

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Padrón, Jorge Rodríguez. El sueño proliferante y otros ensayos. Las Palmas, G.C. [Canary Islands]: Universidad de Las Palmas de Gran Canaria, Servicio de Publicaciones, 1993.

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Badr, Mostafa Z., and Jihan A. Youssef, eds. Peroxisome Proliferator-Activated Receptors (PPARs). Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-155-4.

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Frelinger, Dave. Proliferated autonomous weapons: An example of cooperative behavior. Santa Monica, CA: Rand, 1998.

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Hedrick, R. P. Proliferative kidney disease in salmonid fishes. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, Division of Fisheries and Wetlands Research, 1987.

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Ontario. Ministry of Agriculture and Food. Intestinal Adenomatosis Complex (Porcine Proliferative Enteropathies). S.l: s.n, 1987.

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Spandau, Ulrich, Zoran Tomic, and Diego Ruiz-Casas, eds. Retinal Detachment Surgery and Proliferative Vitreoretinopathy. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78446-5.

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Spandau, Ulrich, Zoran Tomic, and Diego Ruiz-Casas, eds. Retinal Detachment Surgery and Proliferative Vitreoretinopathy. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-11946-0.

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Amersi, Farin, and Kristine Calhoun, eds. Atypical Breast Proliferative Lesions and Benign Breast Disease. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-92657-5.

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Book chapters on the topic "Proliferate"

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O’Boyle, Cherie. "Paradigms Proliferate: Is There an Unconscious Mind?" In History of Psychology, 239–66. Classic edition. | New York, NY : Routledge, 2021. | Series: Psychology Press & Routledge classics: Routledge, 2020. http://dx.doi.org/10.4324/9781003141518-9.

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Allaway, G. P., J. Srinivasappa, F. W. Miller, B. S. Prabhakar, and A. L. Notkins. "Autoantibody Production by Human B Lymphocytes which Spontaneously Proliferate." In Epstein-Barr Virus and Human Disease, 287–91. Totowa, NJ: Humana Press, 1987. http://dx.doi.org/10.1007/978-1-4612-4590-2_63.

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Hestermeyer, Holger. "Where Unity Is at Risk: When International Tribunals Proliferate." In International Law Today: New Challenges and the Need for Reform?, 123–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-75205-9_6.

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Mason, P. W., and G. H. DeVries. "Schwann Cells Proliferate in Response to Non-Myelinated Axons." In Neural Development and Regeneration, 661–62. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73148-8_65.

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Müller, Harald. "The Non-proliferation Treaty and the German Choice Not to Proliferate." In Arms Control and Disarmament, 239–53. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-62259-0_16.

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Baserga, Renato, Bernd Bombik, and Claudio Nicolini. "Changes in Chromatin Structure and Function in W138 Cells Stimulated to Proliferate." In Novartis Foundation Symposia, 269–89. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470720103.ch15.

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Thompson, Linda F., Julie M. Ruedi, and Martin G. Low. "Anti-5′ -Nucleotidase Antibodies Cause Human Peripheral Blood T Cells to Proliferate." In Advances in Experimental Medicine and Biology, 157–64. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5676-9_24.

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Garnier, Christopher J. "Asia’s Sustainability and Digital Innovation Goals Proliferate by Executive Education and Corporate Training." In Business and Management in Asia: Digital Innovation and Sustainability, 139–50. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-6418-3_9.

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Alvina, Fidelia B., Arvin M. Gouw, and Anne Le. "Cancer Stem Cell Metabolism." In The Heterogeneity of Cancer Metabolism, 161–72. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65768-0_12.

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AbstractCancer stem cells (CSCs), also known as tumorinitiating cells (TICs), are a group of cells found within cancer cells. Like normal stem cells, CSCs can proliferate, engage in self-renewal, and are often implicated in the recurrence of tumors after therapy [1, 2]. The existence of CSCs in various types of cancer has been proven, such as in acute myeloid leukemia (AML) [3], breast [4], pancreatic [5], and lung cancers [6], to name a few. There are two theories regarding the origin of CSCs. First, CSCs may have arisen from normal stem/progenitor cells that experienced changes in their environment or genetic mutations. On the other hand, CSCs may also have originated from differentiated cells that underwent genetic and/or heterotypic modifications [7]. Either way, CSCs reprogram their metabolism in order to support tumorigenesis.
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Nair, Sanjiv, and Sunil S. Shroff. "Vascular Anomalies of the Oro-Maxillofacial Region." In Oral and Maxillofacial Surgery for the Clinician, 629–58. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-1346-6_31.

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AbstractVascular anomalies is a diverse group of disorders involving the vasculature (arteries,veins and lymphatics). These lesions in the head and neck can present since birth or later in life causing functional, cosmetic and bleeding problems. They can sometimes co-exist with a wide array of other pathologies and components of various syndromes. The different types, classifications, clinical features, imaging characteristics, treatment options and complications will be discussed in detail, with accordance to guidelines and principles in current literature. The author has designed a widely accepted anatomical classification for surgical management of these complex lesions, which is discussed in depth. Tailoring therapy depending on the anomaly is the key to successful treatment. Hemangiomas tend to proliferate and then involute , hence treated with systemic medication or alternate therapy for residual lesions. Vascular Malformations essentially would require aggressive management with surgery or embolisation and surgery. The concept of ‘corset suturing’ is explained and described, which is thought to be the ideal management for large venous malformations.
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Conference papers on the topic "Proliferate"

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Liu, X., and C. Ding. "Cytocompatibility of Plasma Sprayed Bioceramic Coatings." In ITSC2005, edited by E. Lugscheider. Verlag für Schweißen und verwandte Verfahren DVS-Verlag GmbH, 2005. http://dx.doi.org/10.31399/asm.cp.itsc2005p0600.

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Abstract In this paper, the Al2O3, ZrO2, TiO2, wollastonite, dicalcium silicate and their composite coatings were deposited onto Ti-6Al-4V substrates by an atmosphere plasma spray system. The rat osteoblasts were seeded onto the surface of the coatings to evaluate the growth behavior of osteoblasts on different implant materials. Scanning electron microscopy was used to observe the morphologies of the osteoblasts seeded on the surface of the coating for different time. The SEM observation indicated the osteoblasts are difficult to survive and proliferate on the Al2O3 coating surface. Osteoblasts can survive and proliferate slowly on the TiO2 and ZrO2 coating surface. Osteoblasts grow and proliferate very well on the surface of the wollastonite and dicalcium silicate coating, which present the two coatings possess excellent cytocompatibility. The addition of wollastonite and dicalcium silicate into ZrO2 and TiO2 can improve their cytocompatibility.
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Han, Seung-Soo, Andrew B. Kahng, Siddhartha Nath, and Ashok S. Vydyanathan. "A deep learning methodology to proliferate golden signoff timing." In Design Automation and Test in Europe. New Jersey: IEEE Conference Publications, 2014. http://dx.doi.org/10.7873/date.2014.273.

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Han, Seung-Soo, Andrew B. Kahng, Siddhartha Nath, and Ashok S. Vydyanathan. "A deep learning methodology to proliferate golden signoff timing." In Design Automation and Test in Europe. New Jersey: IEEE Conference Publications, 2014. http://dx.doi.org/10.7873/date2014.273.

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Schuliga, Michael, Fatemeh Zal, Chengxue Qin, Trudi Harris, and Alastair Stewart. "Plasmin Stimulates Airway Smooth Muscle Cells To Proliferate And Produce Interleukin-6." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2443.

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Mtsweni, Jabu, and Elmarie Biermann. "A Roadmap to Proliferate Open Source Software Usage within SA Government Servers." In 2008 Third International Conference on Broadband Communications, Information Technology & Biomedical Applications. IEEE, 2008. http://dx.doi.org/10.1109/broadcom.2008.82.

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Taheri, Arash, Meisam Mohammadi-Amin, and Ali Habibpour-Ledari. "An Optimization Methodology for Cancer Growth Control Base on Breeder Genetic Algorithm-Finite Volume Coupling." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-203638.

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Cancer is a genetic disease caused by mutations in somatic cells. It appears to arise by a process in which an initial population of altered cells begins to proliferate abnormally. At each step, one cancerous cell acquires an additional mutation that gives it a selective advantage over its neighbors, such as more rapid growth, and the descendants of this cell become dominant within the cancer population. In culture, the proliferation of the most cancerous cells is not sensitive to density-dependent inhibition; therefore, cancerous cells usually continue growing to high cell densities.
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Liu, X. Y., and C. X. Ding. "Microstructure and Biological Properties of Plasma Sprayed Novel Bioactive Coatings." In ITSC2009, edited by B. R. Marple, M. M. Hyland, Y. C. Lau, C. J. Li, R. S. Lima, and G. Montavon. ASM International, 2009. http://dx.doi.org/10.31399/asm.cp.itsc2009p0372.

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Abstract In this investigation, bioactive ceramic materials, including dicalcium silicate, titania, and zirconia, were deposited on titanium substrates by plasma spraying in order to determine their effect on the bioactivity of metal implants. Cell-seeding tests show that MG63 osteoblast-like cells grow and proliferate well on each of the coating materials. In the case of Ca2SiO4, the presence of silicon ions is thought to be the key to this behavior. In regard to TiO2 and ZrO2, the bioactivity is thought to result from the nanostructured surfaces and special surface compositions.
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Anderson, Kevin R., and Clifford M. Stover. "Pragmatic Approach to Teaching Introduction to Machine Design." In ASME 2020 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/imece2020-23004.

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Abstract This paper presents examples of teaching Mechanical Design using a pragmatic approach. The pedagogy used herein embeds real-world experience and projects into the curriculum by taking representative case studies from industry and creating course projects. The approach focuses on using sanity checks using free-body diagrams and hand-calculations, materials specification and proliferate used of commercial off the shelf parts supplier catalog data. Case studies from course projects are presented demonstrating the above skill set reinforcement. The assessment of the student work is presented as it is related to the ABET learning outcomes of the mechanical design curriculum track.
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Tsamis, Alkiviadis, and Nikos Stergiopulos. "Dynamics of Arterial Remodeling in Response to a Sustained Step Change in Blood Flow Using a Constituent-Based Model." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176359.

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The arterial wall undergoes substantial remodeling whenever the local blood flow changes for more than a few days [1]. An increase in flow causes an increase in inner radius, which occurs in two phases and tends to restore the baseline levels of intimal shear stress [2]. Firstly, an acute dilation of the artery occurs due to relaxation of vascular smooth muscle (VSM) cells [3]. Secondly, a long-term media reconstruction takes place, during which VSM cells migrate and proliferate circumferentially causing an increase in undeformed lumen. Moreover, arterial wall thickens to restore the altered wall stress [4].
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Kasper, Stephen H., Carolina Morell-Perez, Thomas P. Wyche, Theodore R. Sana, Linda A. Lieberman, and Erik C. Hett. "Abstract B25: Colorectal cancer-associated anaerobic bacteria proliferate in tumor spheroids and alter the microenvironment." In Abstracts: AACR Special Conference on the Microbiome, Viruses, and Cancer; February 21-24, 2020; Orlando, FL. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.mvc2020-b25.

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Reports on the topic "Proliferate"

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Carlson, John. Nuclear verification in a Middle East WMD-Free Zone: Lessons from Past Verification Cases and Other Precedents. The United Nations Institute for Disarmament Research, January 2021. http://dx.doi.org/10.37559/wmdfz/21/nv/01.

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Verification will be of critical importance to achieving and maintaining a Middle East zone free of weapons of mass destruction (ME WMD). Effective verification arrangements would serve a vital national security objective for each state in the region by reducing tensions, removing the motivation to proliferate, and mitigating the risk of a virtual nuclear arms race (or war). In view of the high levels of tension and mistrust within the zone, ensuring effective verification will be especially demanding. The paper examines specific elements of the future nuclear verification of the zone, including: Which states should be included? What prohibitions and obligations should apply in the zone and how would they be verified? How could elimination of nuclear weapons in the zone be achieved? On what basis would the zone treaty enter into force? The paper also examines a number of existing treaties and arrangements as well as the lessons learned from past verification cases which regional states can draw on in developing verification for a Middle East nuclear-weapon-free zone.
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Black, Richard, Joshua Busby, Geoffrey D. Dabelko, Cedric de Coning, Hafsa Maalim, Claire McAllister, Melvis Ndiloseh, et al. Environment of Peace: Security in a New Era of Risk. Stockholm International Peace Research Institute, May 2022. http://dx.doi.org/10.55163/lcls7037.

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The environmental crisis is increasing risks to security and peace worldwide, notably in countries that are already fragile. Indicators of insecurity such as the number of conflicts, the number of hungry people and military expenditure are rising; so are indicators of environmental decline, in climate change, biodiversity, pollution and other areas. In combination, the security and environmental crises are creating compound, cascading, emergent, systemic and existential risks. Without profound changes of approach by institutions of authority, risks will inevitably proliferate quickly. Environment of Peace surveys the evolving risk landscape and documents a number of developments that indicate a pathway to solutions––in international law and policy, in peacekeeping operations and among non-governmental organizations. It finds that two principal avenues need to be developed: (a) combining peace-building and environmental restoration, and (b) effectively addressing the underlying environmental issues. It also analyses the potential of existing and emerging pro-environment measures for exacerbating risks to peace and security. The findings demonstrate that only just and peaceful transitions to more sustainable practices can be effective––and show that these transitions also need to be rapid.
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Grody, Wayne W. Arginase: A Novel Proliferative Determinant in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, April 2005. http://dx.doi.org/10.21236/ada443025.

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Grody, Wayne W. Arginase: A Novel Proliferative Determinant in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, April 2006. http://dx.doi.org/10.21236/ada455780.

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Grody, Wayne W. Arginase: A Novel Proliferative Determinant in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, April 2004. http://dx.doi.org/10.21236/ada427919.

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Anderson, Brian K. A Profile of WMD Proliferants: Are There Commonalities? Fort Belvoir, VA: Defense Technical Information Center, May 1999. http://dx.doi.org/10.21236/ada388733.

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Mach, Robert H. PET Radiotracers for Imaging the Proliferative Status of Breast Tumors. Fort Belvoir, VA: Defense Technical Information Center, June 2002. http://dx.doi.org/10.21236/ada406848.

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Khan, Saif, and Alexander Mann. AI Chips: What They Are and Why They Matter. Center for Security and Emerging Technology, April 2020. http://dx.doi.org/10.51593/20190014.

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The success of modern AI techniques relies on computation on a scale unimaginable even a few years ago. What exactly are the AI chips powering the development and deployment of AI at scale and why are they essential? Saif M. Khan and Alexander Mann explain how these chips work, why they have proliferated, and why they matter.
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Collins, James M., Elizabeth M. Durham-Ruiz, Miriam N. Harris, Conrad A. Widman, and Charles B. Costanzo. Safety, Security, and Stability: The Role of Nuclear Control Regimes in a Proliferated World. Fort Belvoir, VA: Defense Technical Information Center, May 1995. http://dx.doi.org/10.21236/ada329554.

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Rollwagen, Florence M., Nancy D. Pacheco, Jr Wistar, and Richard. Proliferative Responses of Mice to a Cloned Plasmodium Falciparum Sporozoite Antigen. Fort Belvoir, VA: Defense Technical Information Center, December 1988. http://dx.doi.org/10.21236/ada205098.

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