Relevant bibliographies by topics / Primary lymphoedema

Academic literature on the topic 'Primary lymphoedema'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Primary lymphoedema"

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Ali Al-Chalabi, Hussein, and Kathem Kassim AL-Rubiay. "Lymphoedema Praecox: A Case Report of Primary Lymphoedema." Basrah Journal of Surgery 9, no. 1 (June 28, 2003): 110–12. http://dx.doi.org/10.33762/bsurg.2003.55291.

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Vignes, S., M. Arrault, A. Yannoutsos, and M. Blanchard. "Primary upper-limb lymphoedema." British Journal of Dermatology 168, no. 2 (November 20, 2012): 272–76. http://dx.doi.org/10.1111/bjd.12024.

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Tabel, Yilmaz, Ilke Mungan, Ahmet Sigirci, and Serdal Gungor. "Primary Lymphoedema at an Unusual Location Triggered by Nephrotic Syndrome." Annals of the Academy of Medicine, Singapore 38, no. 7 (July 15, 2009): 636. http://dx.doi.org/10.47102/annals-acadmedsg.v38n7p636.

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Introduction: Lymphoedema results from impaired lymphatic transport leading to the pathologic accumulation of protein-rich lymphatic fluid in the interstitial space, most commonly in the extremities. Primary lymphoedema, a developmental abnormality of the lymphatic system, may become evident later in life when a triggering event exceeds the capacity of normal lymphatic flow. Clinical Picture: We present a 3-year-old nephrotic syndrome patient with an unusual localisation for primary lymphoedema. Treatment and Outcome: The patient was treated with conservative approach and she was cured. Conclusion: In this particular case, lymphoedema developed at an unusual localisation, which has not been recorded before. Introduction: Lymphoedema results from impaired lymphatic transport leading to the pathologic accumulation of protein-rich lymphatic fluid in the interstitial space, most commonly in the extremities. Primary lymphoedema, a developmental abnormality of the lymphatic system, may become evident later in life when a triggering event exceeds the capacity of normal lymphatic flow. Clinical Picture: We present a 3-year-old nephrotic syndrome patient with an unusual localisation for primary lymphoedema. Treatment and Outcome: The patient was treated with conservative approach and she was cured. Conclusion: In this particular case, lymphoedema developed at an unusual localisation, which has not been recorded before.
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Bolcal, C., H. Iyem, M. Sargin, I. Mataraci, S. Doganci, S. Kilic, O. Karacalioglu, M. A. Sahin, U. Demirkilic, and H. Tatar. "Primary and secondary lymphoedema in male patients with oedema in lower limbs." Phlebology: The Journal of Venous Disease 21, no. 3 (September 15, 2006): 127–31. http://dx.doi.org/10.1258/026835506778253328.

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Objective: The purpose of this prospective study was to evaluate patients with clinically diagnosed lymphoedema of the lower extremities. The proportions of primary and secondary lymphoedema, the possible aetiologic factors and the concomitance of chronic venous diseases and lymphoedema were focused on. Method: The male patients who attended our outpatient clinic during 2000 and 2004 were evaluated. In all, 160 male patients with 5 cm circumference difference at calf level between two lower extremities or with clinically diagnosed bilateral leg oedema were enrolled. All patients underwent duplex venous ultrasonography and lymphoscintigraphy. Venography was performed in 12 patients with normal lymphoscintigraphy and ultrasonography. Results: The age distribution was between 20 and 54 years (mean ± SD; 22.9 ± 4.3). Among 160 patients, 70.0% had lymphoedema, while 7.5% had chronic venous insufficiency, 3.75% chronic deep venous thrombosis, 7.5% concomitant venous disease and lymphatic obstruction, and 7.5% idiopathic oedema. In the last 3.75% the pathology was Klippel–Trenaunay syndrome. Of the primary lymphoedema patients (16.25%), 18 were praecox, six tarda and two were congenital types. Conclusion: In young male patients, the causes of secondary lymphoedema are lymphadenectomy, neoplastic metastasis, cellulitis, lymphangitis, etc. Further techniques confirmed the clinical diagnosis of lymphoedema in 77.5% (sum of lymphoedema and concomitant disease) of all patients. With these findings lymphoedema can be diagnosed clinically, and further diagnostic techniques can be reserved unless treatment is effective.
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Chaudhry, H. L., P. S. Mortimer, J. E. Evans, and J. A. Dormandy. "The Prevalence of Venous Disease in Primary Lymphoedema." Phlebology: The Journal of Venous Disease 12, no. 1 (March 1997): 31–35. http://dx.doi.org/10.1177/026835559701200106.

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Objective: To assess the prevalence of venous incompetence in primary lymphoedema. Design: Retrospective study. Setting: Outpatients attending St George's Hospital, London, UK. Patients: Forty-six patients (61 limbs) with primary lymphoedema extracted from a consecutive series of patients presenting with swollen legs. Only those with clinical and lymphoscintigraphic evidence of lymphoedema were included. Patients with any clinical evidence of venous disease were excluded. Controls: Seventeen subjects (33 limbs) with no history or clinical evidence of either lymphoedema or venous disease. Methods: Light reflective rheography (LRR) and quantitative isotopic lymphoscintigraphy. Main outcome measures: Venous refilling times (RT) with and without tourniquet control. Statistical analysis: Prevalence of venous incompetence in lymphoedema patients compared with normal controls and literature values. Results: In the patients, seven of 61 limbs (11.5%) had a venous RT of less than 25 s; eight limbs (13.1%) had a venous RT between 25 and 40 s. An above-knee tourniquet corrected the RT in all 15 limbs indicating superficial venous incompetence. In the controls, one of 33 limbs (3%) had a venous RT between 25 and 40 s. An above-knee tourniquet corrected the RT indicating superficial venous incompetence. Conclusion: Covert venous insufficiency may complicate primary lymphoedema. This has implications for both the pathophysiology of the oedema and its management.
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Tatnall, F. M., and I. Sarkany. "Primary Facial Lymphoedema with Xanthomas." Journal of the Royal Society of Medicine 81, no. 2 (February 1988): 113–14. http://dx.doi.org/10.1177/014107688808100224.

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Dale, R. F. "The inheritance of primary lymphoedema." Journal of Medical Genetics 22, no. 4 (August 1, 1985): 274–78. http://dx.doi.org/10.1136/jmg.22.4.274.

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Jacobsen, J. O. G., and C. Eggers. "Primary lymphoedema in a kitten." Journal of Small Animal Practice 38, no. 1 (January 1997): 18–20. http://dx.doi.org/10.1111/j.1748-5827.1997.tb02980.x.

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Gajraj, H., S. G. E. Barker, K. G. Burnand, and N. L. Browse. "Lymphangiosarcoma complicating chronic primary lymphoedema." British Journal of Surgery 74, no. 12 (December 1987): 1180. http://dx.doi.org/10.1002/bjs.1800741232.

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Yüksel, Ahmet, Orçun Gürbüz, Yusuf Velioğlu, Gencehan Kumtepe, and Sefa Şenol. "Management of lymphoedema." Vasa 45, no. 4 (July 2016): 283–91. http://dx.doi.org/10.1024/0301-1526/a000539.

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Abstract. Lymphoedema is a common and progressive disease which causes deterioration of the quality of life of patients. It is divided into two groups: primary and secondary lymphoedema. Nowadays, the majority of patients with lymphoedema are associated with a malignancy or its treatment modalities, such as cancer surgery and radiation therapy. Accurate diagnosis and effective treatment are crucial for alleviating the symptoms, preventing progression and reducing the potential risks of lymphoedema. This report provides an overview of the management of lymphoedema.
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Dissertations / Theses on the topic "Primary lymphoedema"

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Evans, Alison Lee. "Molecular study of primary congenital lymphoedema." Thesis, St George's, University of London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.272430.

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Connell, Fiona. "Clinical and genetic investigation of primary lymphoedema." Thesis, St George's, University of London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.511898.

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Gordon, Kristiana. "Refining the phenotype and genotype of primary lymphoedema." Thesis, St George's, University of London, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.676101.

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The background to this research is the primary lymphoedema clinic held at St George's Hospital, London. This is a multidisciplinary clinic run by the combined expertise of dermatology and genetic teams, and is the only one of its kind in the UK. The aim of the service is to diagnose, investigate and manage patients with primary lymphoedema. To date, 7 genes are known to cause a phenotype where lymphoedema predominates. The main aim of my MO(Res) project was to use and refine Connell et aI's diagnostic classification pathway (Connell, Brice et al. 2010) for patients with primary lymphoedema in order to identify phenotypically similar groups of patients who could then be the subject of molecular investigation. This, in turn, would hopefully lead to the discovery of new genotypes responsible for subtypes of primary lymphoedema . Lymphangiogenesis, lymphoedema, its clinical features, diagnosis and investigation modalities, and the well -established phenotypes of primary lymphoedema are discussed in the introduction. The laboratory techniques that have been used (including Sanger sequencing and next generation sequencing) are described. Subsequent chapters describe the individual projects that have been carried out in an effort to phenotype and genotype cohorts of patients with primary lymphoedema, including the discovery of a new causal gene for primary lymphoedema. One chapter is dedicated to the development of magnetic resonance lymphangiography (MRL) as an investigative tool in patients with lymphoedema to improve accurate phenotyping of the subgroups, as current imaging techniques are limited. Finally, Connell et aI's diagnostic pathway has been updated on the basis of new findings.
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Waters, Bernadette. "Unknown stories : biographies of adults with primary lymphoedema." Thesis, University of Southampton, 2009. https://eprints.soton.ac.uk/336241/.

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Lymphoedema is a chronic, debilitating and disfiguring medical condition caused by lymphatic insufficiency. This insufficiency may be characterised as primary, when there is congenital or hereditary cause; or secondary, when the cause is related to trauma or illness. It can lead to extreme swelling, most usually in the limbs, and creates increased susceptibility to recurrent, and sometimes life-threatening, infection. Physical, psychological and social effects of the condition can have a significant impact on an individual’s life and their levels of participation. Lymphoedema has attracted a very small amount of research activity by comparison to other chronic conditions and almost all research has focused on the views of medical experts and relates to their management of the condition. What qualitative research that exists concerning the experience of living with lymphoedema has focused primarily on women who survive breast cancer. The voice of individuals with primary lymphoedema has not been heard. This study investigated the life stories of eight people using an auto/biographical approach to illuminate their experiences of living with advanced and complicated primary lymphoedema. The specific aims of the study were to: record the stories of those who live with primary lymphoedema in order to better understand their lived experience; analyse those stories in the context of what is known about the condition and reflect on the management of lymphoedema; draw conclusions and make recommendations that might make a useful contribution to the lives of those who experience lymphoedema. Each person took part in an interview with the researcher in which they were invited to talk about their lives. These interviews were transcribed and then reviewed by the participants. Narrative and biographical analysis was employed to explore the meaning in the data. Findings reveal the extensive impact that primary lymphoedema has on individual lives depriving participants of relationships, employment, leisure and self esteem. Although each person provided a unique and individual point of view of their life, there were certain common themes that emerged. Participants spoke of their difficulty in finding a correct diagnosis and access to effective treatment; the importance of information and education and the challenges of daily self management. Finally, recommendations are made to increase lymphoedema awareness amongst providers of healthcare and those who commission it; promote intensive and community-based treatment programmes; teach and monitor effective self-management techniques.
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Books on the topic "Primary lymphoedema"

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Hoskin, Peter. Vulva and vagina. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199696567.003.0014.

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Chapter 9b discusses carcinoma of the vulva, which is primarily a surgical disease best treated by wide surgical resection, radical vulvectomy, and inguinal lymph node dissection based on presenting stage. Rarely, locally advanced primary disease may be presented for primary radiotherapy treatment. Postoperative radiotherapy is recommended for tumours invading >7 mm in a vertical direction. The first station regional lymph nodes in the inguinal region are best treated by radical surgical dissection, but fixed inoperable lymph nodes may benefit from primary radiotherapy which may be followed where appropriate by surgery if there is a residual mass. Postoperative radiotherapy should be considered for women having more than one node involved with metastatic tumour at surgery. This must be balanced against the increased risk of lymphoedema where both surgery and radiotherapy are delivered to the groins. Chemoradiation using cisplatin or 5-FU/mitomycin C-based schedules has been reported but no randomized comparison with radiotherapy alone has been undertaken; whilst high response rates are seen there is a considerable increase in acute toxicity.
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Book chapters on the topic "Primary lymphoedema"

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Heinsoo, Iris, and Amanda Paul. "Primary lymphoedema in a 4-month-old kitten." In BSAVA Congress Proceedings 2022. British Small Animal Veterinary Association, 2022. http://dx.doi.org/10.22233/9781913859114.38.24.

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Mortimer, Peter S. "Chronic peripheral oedema and lymphoedema." In Oxford Textbook of Medicine, edited by Jeremy Dwight, 3811–22. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0382.

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Lymph transport, not venous reabsorption, is the main process responsible for interstitial fluid drainage. Oedema develops when the microvascular filtration rate exceeds lymph drainage for a sufficient period, and any chronic oedema represents a failure of lymph drainage. In practice, any chronic oedema should be managed in the same way as lymphedema. The primary function of the lymphatic vessels is to drain the plasma filtrate within body tissues and return it to the blood circulation. Lymphatic vessels also have an important immune surveillance function, as they are the main drainage route from the tissues for immune active cells such as dendritic cells, lymphocytes, and macrophages. Intestinal lymphatics are responsible for fat absorption. Impaired lymphatic function leads to disturbed fluid homeostasis (swelling), dampened immune responses (infection), and disturbed fat homeostasis (increased peripheral fat deposition), all features of lymphoedema. Lymphatic vessels are also the preferential route for cancer spread.
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"Adult care provision." In Oxford Handbook of Primary Care and Community Nursing, edited by Judy Brook, Caroline McGraw, and Val Thurtle, 503–96. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198831822.003.0010.

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This chapter covers the provision of primary care to adults. This includes urinary incontinence, catheter care, constipation and incontinence, stoma care, spinal cord injury, the prevention of pressure ulcers, wound infection and debridement, malignant fungating wounds, wound dressings, leg ulcers and their dressing, compression therapy for venous ulcers, and lymphoedema. It also covers palliative care, including services for the dying patient, pain assessment and management, nausea, breathlessness, fatigue, depression, and spiritual care, and caring for patients in the dying phase. Emergencies, assisted dying, legal issues around the end of life, and bereavement are all covered. Common technical procedures including the care of central venous catheters, injections, venepuncture, recording a 12 lead electrocardiogram, tracheostomy care, and ear irrigation are also described.
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Eva, Gail. "Rehabilitation of the cancer patient." In Oxford Handbook of Cancer Nursing, edited by Mike Tadman, Dave Roberts, and Mark Foulkes, 137–40. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198701101.003.0010.

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Disability is a major, but often under-recognized, problem for people with cancer. Rehabilitation engages patients and their families in an active process of achieving meaningful goals, so that decline does not have to be associated with helplessness and hopelessness. It aims to maximize social participation and well-being, to achieve personal satisfaction and adaptation to disability, and to minimize carer stress and distress. Rehabilitation is a multiprofessional activity carried out by a range of allied health professionals (AHPs) and other health and social care professionals. The AHPs involved include occupational therapists, physiotherapists, speech and language therapists, dieticians, therapy radiographers, and lymphoedema therapists. In inpatient settings, discharge planning might supersede rehabilitation as the focus of care. Once the patient is at home, it is important to consider their longer-term rehabilitation needs and to make appropriate referrals to community-based primary care rehabilitation teams. Nurses play an active part in the rehabilitation process and can contribute by making appropriate rehabilitation referrals and by working with AHPs to enable patients to maintain independence and control in their daily activities. Patients’ rehabilitation needs vary from straightforward to complex, and the National Institute for Health and Care Excellence (NICE) has produced a 3-level model of the roles and responsibilities of different members of the multidisciplinary team.
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