Relevant bibliographies by topics / Prepulse inhibition

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Prepulse inhibition'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 March 2023

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Journal articles on the topic "Prepulse inhibition"

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Weike, Almut I., Alfons O. Hamm, and Dieter Vaitl. "Sensorimotor Gating and Attitudes Related to Schizotypal Proneness." Psychological Reports 88, no. 3_suppl (June 2001): 1035–45. http://dx.doi.org/10.2466/pr0.2001.88.3c.1035.

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The magnitude of the startle eyeblink response is diminished when the startle-eliciting probe is shortly preceded by another stimulus. This so called prepulse inhibition is interpreted as an automatic sensorimotor gating mechanism. There is substantial support for prepulse inhibition deficits in subjects suffering from schizophrenia spectrum disorders and in psychosis-prone normals as well. Thus, prepulse inhibition deficits may reflect vulnerability on the hypothesized psychopathological continuum from “normal” to “schizophrenia.” The present experiment investigated the amount of prepulse inhibition in a sample selected for “belief in extraordinary phenomena,” an attitude related to measures of psychosis-proneness. Believers and skeptics were tested in an acoustic prepulse-inhibition paradigm. As expected, presentation of prepulses clearly diminished magnitude of startle response, with greatest inhibition effects gained by lead intervals of 60 and 120 msec. Patterns of response were identical for believers and skeptics, i.e., attitude towards extraordinary phenomena did not seem to be related to functional information-processing deficits as has been observed in psychosis-prone normals.
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Kawano, Yasuhiro, Eishi Motomura, Koji Inui, and Motohiro Okada. "Effects of Magnitude of Leading Stimulus on Prepulse Inhibition of Auditory Evoked Cerebral Responses: An Exploratory Study." Life 11, no. 10 (September 28, 2021): 1024. http://dx.doi.org/10.3390/life11101024.

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An abrupt change in a sound feature (test stimulus) elicits a specific cerebral response, which is attenuated by a weaker sound feature change (prepulse) preceding the test stimulus. As an exploratory study, we investigated whether and how the magnitude of the change of the prepulse affects the degree of prepulse inhibition (PPI). Sound stimuli were 650 ms trains of clicks at 100 Hz. The test stimulus was an abrupt sound pressure increase (by 10 dB) in the click train. Three consecutive clicks, weaker (−5 dB, −10 dB, −30 dB, or gap) than the baseline, at 30, 40, and 50 ms before the test stimulus, were used as prepulses. Magnetic responses to the ten types of stimuli (test stimulus alone, control, four types of tests with prepulses, and four types of prepulses alone) were recorded in 10 healthy subjects. The change-related N1m component, peaking at approximately 130 ms, and its PPI were investigated. The degree of PPI caused by the −5 dB prepulse was significantly weaker than that caused by other prepulses. The degree of PPI caused by further decreases in prepulse magnitude showed a plateau level between the −10 dB and gap prepulses. The results suggest that there is a physiologically significant range of sensory changes for PPI, which plays a role in the change detection for survival.
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Dahmen, Johannes C., and Philip J. Corr. "Prepulse-elicited startle in prepulse inhibition." Biological Psychiatry 55, no. 1 (January 2004): 98–101. http://dx.doi.org/10.1016/s0006-3223(03)00638-3.

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Elden, Åke, and Magne Arve Flaten. "The Relationship of Automatic and Controlled Processing to Prepulse Inhibition." Journal of Psychophysiology 16, no. 1 (January 2002): 46–55. http://dx.doi.org/10.1027//0269-8803.16.1.46.

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Abstract When a weak stimulus, or prepulse, is presented immediately prior to a startle reflex-eliciting stimulus, the startle reflex is inhibited. This is called prepulse inhibition (PPI). Directing attention to a prepulse increases PPI. In two experiments (N = 43 and N = 29), attention was directed to the prepulse by having the participants judge the duration of the prepulse. Prepulse inhibition was assessed at stimulus onset asynchronies (SOAs) assumed to index automatic and controlled processing. The prepulse was a 60dB tone, and startle was elicited by 95dB white noise. We predicted that attention directed to the prepulse should increase PPI, and that PPI should increase on trials with correct judgments of prepulse duration compared to trials with incorrect judgments. The results from both experiments showed that attention directed toward the prepulse increased PPI at SOAs assumed to index both automatic and controlled processing. This indicates that controlled attention exerted an influence on automatic processes. There was no evidence that PPI was increased on trials with correct judgment of prepulse duration. It is concluded that attention to the prepulse increased PPI, but PPI did not differentiate between automatic and controlled processing under the present experimental conditions.
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Röskam, Stephan, and Michael Koch. "Enhanced prepulse inhibition of startle using salient prepulses in rats." International Journal of Psychophysiology 60, no. 1 (April 2006): 10–14. http://dx.doi.org/10.1016/j.ijpsycho.2005.04.004.

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Roussos, P., S. G. Giakoumaki, M. Rogdaki, S. Pavlakis, S. Frangou, and P. Bitsios. "Prepulse inhibition of the startle reflex depends on the catechol O-methyltransferase Val158Met gene polymorphism." Psychological Medicine 38, no. 11 (February 8, 2008): 1651–58. http://dx.doi.org/10.1017/s0033291708002912.

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BackgroundRecent evidence suggests that dopamine (DA) agonist-induced disruption of prepulse inhibition (PPI) depends on basal PPI values, in a manner that suggests an inverted U-shaped relationship between PPI and prefrontal DA levels. This is the first study to examine possible genetic determinants of PPI and the catechol O-methyltransferase (COMT) Val158Met polymorphism, the main catabolic pathway of released DA in the prefrontal cortex (PFC).MethodPPI was measured in 93 healthy males presented with 75-dB and 85-dB prepulses at 60-ms and 120-ms prepulse–pulse intervals. Subjects were grouped according to their COMT status into a Val/Val, a Val/Met and a Met/Met group.ResultsANOVAs showed that at all prepulse and interval conditions, Val/Val individuals had the lowest PPI, Met/Met the highest, and Val/Met were intermediate.ConclusionsThese results suggest that PPI is regulated by DA neurotransmission in the PFC and its levels depend on the COMT Val158Met gene polymorphism. These findings enhance the value of the PPI paradigm in examining individual variability of early information processing in healthy subjects and psychiatric disorders associated with changes in PFC DA activity and attentional deficits such as schizophrenia.
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Carlson, Stephanie, and James F. Willott. "Caudal Pontine Reticular Formation of C57BL/6J Mice: Responses to Startle Stimuli, Inhibition by Tones, and Plasticity." Journal of Neurophysiology 79, no. 5 (May 1, 1998): 2603–14. http://dx.doi.org/10.1152/jn.1998.79.5.2603.

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Carlson, Stephanie and James F. Willott. Caudal pontine reticular formation of C57BL/6J mice: responses to startle stimuli, inhibition by tones, and plasticity. J. Neurophysiol. 79: 2603–2614, 1998. C57BL/6J (C57) mice were used to examine relationships between the behavioral acoustic startle response (ASR) and the responses of neurons in the caudal pontine reticular formation (PnC) in three contexts: 1) responses evoked by basic startle stimuli; 2) the prepulse inhibition (PPI) paradigm; and 3) the effects of high-frequency hearing loss and concomitant neural plasticity that occurs in middle-aged C57 mice. 1) Responses (evoked action potentials) of PnC neurons closely paralleled the ASR with respect to latency, threshold, and responses to rapidly presented stimuli. 2) “Neural PPI” (inhibition of responses evoked by a startle stimulus when preceded by a tone prepulse) was observed in all PnC neurons studied. 3) In PnC neurons of 6-mo-old mice with high-frequency (>20 kHz) hearing loss, neural PPI was enhanced with 12- and 4-kHz prepulses, as it is behaviorally. These are frequencies that have become “overrepresented” in the central auditory system of 6-mo-old C57 mice. Thus neural plasticity in the auditory system, induced by high-frequency hearing loss, is correlated with increased salience of the inhibiting tones in both behavioral and neural PPI paradigms.
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Lee, Jae-Hun, Jae Yun Jung, and Ilyong Park. "A Gap Prepulse with a Principal Stimulus Yields a Combined Auditory Late Response." Journal of Audiology and Otology 24, no. 3 (July 10, 2020): 149–56. http://dx.doi.org/10.7874/jao.2019.00374.

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Background and Objectives: The gap prepulse inhibition of the acoustic startle response has been used to screen tinnitus in an animal model. Here, we examined changes in the auditory late response under various conditions of gap prepulse inhibition.Subjects and Methods: We recruited 19 healthy adults (5 males, 14 females) and their auditory late responses were recorded after various stimuli with or without gap prepulsing. The N1 and P2 responses were selected for analysis. The gap prepulse inhibition was estimated to determine the optimal auditory late response in the gap prepulse paradigm.Results: We found that the gap per se generated a response that was very similar to the response elicited by sound stimuli. This critically affected the gap associated with the maximal inhibition of the stimulus response. Among the various gap-stimulus intervals (GSIs) between the gap and principal stimulus, the GSI of 150 ms maximally inhibited the response. However, after zero padding was used to minimize artifacts after a P2 response to a gap stimulus, the differences among the GSIs disappeared.Conclusions: Overall, the data suggest that both the prepulse inhibition and the gap per se should be considered when using the gap prepulse paradigm to assess tinnitus in humans.
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Schicatano, Edward J., Kavita R. Peshori, Ramesh Gopalaswamy, Eva Sahay, and Craig Evinger. "Reflex Excitability Regulates Prepulse Inhibition." Journal of Neuroscience 20, no. 11 (June 1, 2000): 4240–47. http://dx.doi.org/10.1523/jneurosci.20-11-04240.2000.

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Mongeluzi, Donna L., Travis A. Hoppe, and William N. Frost. "Prepulse Inhibition of theTritoniaEscape Swim." Journal of Neuroscience 18, no. 20 (October 15, 1998): 8467–72. http://dx.doi.org/10.1523/jneurosci.18-20-08467.1998.

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Dissertations / Theses on the topic "Prepulse inhibition"

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Truchanowicz, Ewa G. "Prepulse reactivity in prepulse inhibition." Thesis, Swansea University, 2010. https://cronfa.swan.ac.uk/Record/cronfa42605.

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Prepulse inhibition (PPI) is a popular paradigm in sensorimotor gating research. In healthy individuals the weak lead stimulus (i.e., the prepulse) presentation results in a reduction in the startle probe (pulse) elicited response. The motor responses to the prepulses (prepulse reactivity, PPER) were until recently largely ignored in PPI research. There are conflicting reports about prepulse reactivity and startle response modification (SRM) associations; and personality factors relevant to SRM have not been previously examined in prepulse reactivity context. Healthy participants were drawn from university student and staff population. Three paradigms were used: unpredictable stimulus onset, predictable stimulus onset and conscious stimulus processing. The stimuli consisted of 80, 85 & 90dB prepulses and 115dB startle probe separated by 140ms inter-stimulus interval (onset to onset asynchrony). The inter-trial intervals varied between the studies. Startle responses were measured as eye blinks and recorded using surface EMG. All motor responses were quantified according to the same set of rules. Prepulse-elicited motor responses reliably appeared in all the studies and were distinct from spontaneous EMG. Some PPER characteristics exhibited stimulus intensity dependence further proving PPER validity as stimulus-driven response. Prepulse reactivity exhibited significant associations with startle response modification. PPER was a stable tendency; individuals either consistently responded to the weak lead stimuli or did not. Two types of startle response modification appeared under the conditions assumed to elicit maximal inhibition only: classical inhibition (as expected) and paradoxical prepulse facilitation. These appeared in motor responses and in conscious stimulus processing. The propensity towards the paradoxical prepulse facilitation was reduced by efficient prepulse inhibition. PPER and SRM had limited associations with personality factors, sex, or age. The predictable stimulus onset paradigm however highlighted the associations of the defensive startle response and its modification with fear and anxiety. Increased emotionality, regardless of its valence, proved detrimental to sensorimotor gating.
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Parker, Stephen David. "The effects of attention and stimulus onset asynchrony on the relationship between prepulse inhibition of the startle-eyeblink and prepulse-rating inhibition /." [St. Lucia, Qld.], 2002. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe16834.pdf.

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Tanner, Lisa. "Effects of early acoustic stimulation on prepulse inhibition in mice." Scholar Commons, 2003. https://scholarcommons.usf.edu/etd/1490.

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The purpose of this study was to determine the effects of an atypical pattern of early acoustic stimulation on auditory development. Previous human research suggests that the acoustic environment of pre-term human infants in the Neonatal Intensive Care Unit (NICU) negatively affects some aspects of auditory development. Animal research suggests that premature auditory stimulation interrupts auditory development. Because mice are born before their auditory systems are developed, they make an excellent model for research on fetal and postnatal plasticity of the auditory system. The premature auditory state of newborn mice is similar to that of the NICU pre-term infant, albeit, natural for mice C57 mouse pups were exposed to an augmented acoustic environment (AAE) of a nightly 12-hour regiment of 70 dB SPL noise burst, beginning before age 12 days (onset of hearing) and lasting for one month. The prepulse inhibition (PPI) of mice exposed to the AAE was compared to that of non-exposed mice to observe short-term and long-term effects. Results showed that the prepulse inhibition of the AAE exposed mice did not differ significantly from that of the non-exposed mice. However, it is possible that the measurement used, PPI, may not have been appropriate or that the AAE may not have been an appropriate simulation of the NICU environment.
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Poje, Albert Buddy Filion Diane L. "The effects of multiphasic prepulse stimuli on attentional modulation of prepulse inhibition of the acoustic startle eyeblink response." Diss., UMK access, 2007.

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Thesis (Ph. D.)--Dept. of Psychology. University of Missouri--Kansas City, 2007.
"A dissertation in psychology." Advisor: Diane L. Filion. Typescript. Vita. Title from "catalog record" of the print edition Description based on contents viewed July 16, 2008. Includes bibliographical references (leaves 109-118). Online version of the print edition.
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Varty, Geoffrey Brian. "Investigations into prepulse inhibition : a proposed in vivo model for schizophrenia." Thesis, University of Hertfordshire, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309718.

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Ralph, Rebecca Jeanette. "Dopamine modulation of prepulse inhibition and locomotor behavior in knockout mice /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2001. http://wwwlib.umi.com/cr/ucsd/fullcit?p3001269.

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Swonger, Jessica M. "Prepulse Inhibition of the Startle Reflex in Forebrain Oxytocin Receptor Knockout Mice." Kent State University Honors College / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=ksuhonors1304360430.

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O'steen, Jennifer Robin. "Prepulse Inhibition and the Acoustic Startle Response in Nine Inbred Mouse Strains." Scholar Commons, 2003. https://scholarcommons.usf.edu/etd/1443.

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This study examined the effects of genetic background on the acoustic startle response (ASR) and its modulation by prepulse inhibition (PPI) by comparing nine inbred strains of mice. The ASR, a jerk-like motor reflex, is elicited by bursts of noise or tones with sound pressure levels of 80-90 dB and greater. PPI is a type of modulation of the ASR, requires no training, and results in observable response in both mice and humans. Data were obtained from nine inbred mouse strains, sixteen per strain, which were shipped at approximately 3-5 weeks old from The Jackson Laboratory. In general, ASRs were generally smaller when the startle stimulus was less intense. PPI was relatively weak for the 4 kHz prepulse, and stronger with prepulses of 12 kHz and 20 kHz. However, means varied widely across strains for both ASR and PPI, suggesting a strong influence of genetic background on these behaviors. In addition to genetic influences, peripheral hearing loss and central auditory processing factors must be taken into consideration.
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Maple, Amanda M., Katherine J. Smith, Marla K. Perna, and Russell W. Brown. "Neonatal Quinpirole Treatment Produces Prepulse Inhibition Deficits in Adult Male and Female Rats." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/947.

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We have shown that repeated neonatal quinpirole (QUIN; a dopamine D2-like receptor agonist) treatment in rats produces long-lasting supersensitization of dopamine D2 receptors that persists into adulthood but without producing a change in receptor number. The current study was designed to analyze the effects of neonatal QUIN on auditory sensorimotor gating as measured through prepulse inhibition (PPI). Male and female Sprague–Dawley rats were neonatally treated with QUIN (1mg/kg) or saline from postnatal days (P)1–21. At P60, the number of yawns was recorded for a 1h period in response to an acute QUIN (1mg/kg) injection as yawning is a D2-like receptor mediated behavioral event. Five days later, rats began (PPI) behavioral testing in two phases. In phase I, three different prepulse intensities (73, 76, and 82dB) were administered 100-ms before a 115dB pulse on 10 consecutive days. In phase II, three different interstimulus intervals (ISI; 50, 100, and 150ms) were inserted between the 73 or 76dB prepulse and 115dB pulse over 10 consecutive days of testing. A PPI probe trial was administered at the end of each phase after an acute 100μg/kgi.p. injection of QUIN to all animals. Replicating previous work, neonatal QUIN enhanced yawning compared to controls, verifying D2 receptor supersensitization. Regarding PPI, neonatal QUIN resulted in deficits across both phases of testing persistent across all testing days. Probe trial results revealed that acute QUIN treatment resulted in more robust PPI deficits in neonatal QUIN animals, although this deficit was related to prepulse intensity and ISI. These findings provide evidence that neonatal QUIN treatment results in deficits of auditory sensorimotor gating in adulthood as measured through PPI.
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Kedzior, Karina Karolina. "Chronic cannabis use and attention-modulated prepulse inhibition of the startle reflex in humans." University of Western Australia. School of Medicine and Pharmacology, 2004. http://theses.library.uwa.edu.au/adt-WU2004.0027.

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Background. Various studies show that cannabis use alters attention and cognitive functioning in healthy humans and may contribute to development of schizophrenia or worsening of pre-existing psychosis. However, the impact of cannabis use on brain function in humans is not well understood. Schizophrenia is associated with a deficit in prepulse inhibition (PPI), the normal inhibition of the startle reflex by a non-startling stimulus (prepulse), presented before the startle stimulus at short time intervals (lead-time intervals). Such PPI deficit is thought to reflect a sensorimotor gating dysfunction in schizophrenia. PPI is also modulated by attention and PPI reduction in schizophrenia is observed when patients are asked to attend to, not ignore, the stimuli producing PPI. The aim of the current study was to investigate the association between self-reported chronic cannabis use and attentional modulation of PPI in healthy controls and in patients with schizophrenia. Furthermore, the association between cannabis use and other startle reflex modulators, including prepulse facilitation (PPF) of the startle reflex magnitude at long lead-time intervals, prepulse facilitation of the startle reflex onset latency and habituation of the startle reflex magnitude, were examined. Method. Auditory-evoked electromyographic signals were recorded from orbicularis oculi muscles in chronic cannabis users (29 healthy controls and 5 schizophrenia patients) and non-users (22 controls and 14 patients). The data for 36 participants (12 non-user controls, 16 healthy cannabis users, and eight non-user patients) were used in the final analyses and the patient data were used as a pilot study, because relatively few participants met the rigorous exclusionary criteria. Participants were instructed to attend to or to ignore either the startle stimuli alone (70 100 dB) or prepulse (70 dB) and startle stimuli (100 dB) separated by short lead-time intervals (20 200 ms) and long lead-time intervals (1600 ms). In order to ignore the auditory stimuli the participants played a visually guided hand-held computer game. A pilot study showed that the response component of playing the game had no effects on attentional modulation of the startle reflex magnitude and onset latency. Results. Relative to controls, cannabis use in healthy humans was associated with a reduction in PPI similar to that observed in schizophrenia while attending to stimuli, and with an attention-dependent dysfunction in the startle reflex magnitude habituation. While ignoring the stimuli there were no statistical differences in PPI between cannabis users and controls, although PPI in cannabis users tended to differ from that of the patients. The reduction in PPI in cannabis users was correlated with the increased duration of cannabis use, in years, but not with the concentration of cannabinoid metabolites in urine or with the recency of cannabis use in the preceding 24 hours. Furthermore, cannabis use was not associated with any differences in PPF, onset latency facilitation, and startle reflex magnitude in the absence of prepulses. The accuracy of self-reports of substance use was also investigated in this study and was found to be excellent. In addition, the study examined the validity of the substance use module of the diagnostic interview, CIDI-Auto 2.1, which was found to be acceptable for cannabis misuse diagnoses (abuse and/or dependence). Finally, cannabis dependence was found to be associated with more diagnoses of mental illness other than schizophrenia (mainly depression). Conclusions. The results of the current study suggest that chronic cannabis use is associated with schizophrenia-like deficit in PPI in otherwise healthy humans. This PPI reduction is associated with attentional impairment rather than a global sensorimotor gating deficit in healthy cannabis users.
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Books on the topic "Prepulse inhibition"

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Lovic, Vedran. Artificially reared female rats show reduced prepulse inhibition and deficits in attentional set shifting task. Ottawa: National Library of Canada, 2002.

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Faerman, Paul. Pharmacological regulation of brain stem circuits mediating prepulse inhibition of startle in rats. 2004.

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Benning, Stephen D. The Postauricular Reflex as a Measure of Attention and Positive Emotion. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780199935291.013.74.

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The postauricular reflex is a muscular reaction that occurs behind the ear in response to short, abrupt sounds. Its magnitude increases with louder eliciting sounds, rotating the eyes in the direction of the eliciting sound, and flexing the head forward. The reflex exhibits prepulse inhibition, especially during attention to complex foreground stimuli. Its magnitude is larger (or potentiated) during pleasant than during neutral pictures, sounds, and videos that are highly arousing. This pattern is particularly evident for erotic, food, and nurturant scenes, suggesting it assesses more than just appetitive processing. This reflex’s potentiation varies across development; positively correlates with personality traits associated with well-being; and negatively correlates with such psychopathologies as depression, schizophrenia, and opioid dependence. It appears distinct from and uncorrelated with the startle blink reflex. New data suggest that activity in left frontal areas generates postauricular reflex potentiation during pleasant versus neutral pictures.
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Book chapters on the topic "Prepulse inhibition"

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McAllister-Williams, R. Hamish, Daniel Bertrand, Hans Rollema, Raymond S. Hurst, Linda P. Spear, Tim C. Kirkham, Thomas Steckler, et al. "Prepulse Inhibition." In Encyclopedia of Psychopharmacology, 1064–66. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_274.

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Geyer, Mark A. "Prepulse Inhibition." In Encyclopedia of Psychopharmacology, 1359–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-36172-2_274.

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Geyer, Mark A. "Prepulse Inhibition." In Encyclopedia of Psychopharmacology, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27772-6_274-2.

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Arenas, M. Carmen, Sergio Pujante-Gil, and Carmen Manzanedo. "Prepulse Inhibition and Vulnerability to Cocaine Addiction." In Methods for Preclinical Research in Addiction, 47–84. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1748-9_3.

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Schall, U., D. Zerbin, and R. D. Oades. "Prepulse-Inhibition-Defizite in der akustischen Signalverarbeitung bei jungen schizophrenen Patienten." In Biologische Psychiatrie der Gegenwart, 59–63. Vienna: Springer Vienna, 1993. http://dx.doi.org/10.1007/978-3-7091-9263-4_13.

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Gómez-Nieto, Ricardo, J. A. C. Horta-Júnior, Orlando Castellano, Donal G. Sinex, and Dolores E. López. "Auditory Prepulse Inhibition of Neuronal Activity in the Rat Cochlear Root Nucleus." In The Neurophysiological Bases of Auditory Perception, 79–90. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-5686-6_8.

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Geyer, Mark A. "Prepulse Inhibition as a Cross-Species Model of Sensorimotor Gating Deficits in Schizophrenia." In Contemporary Issues in Modeling Psychopathology, 103–12. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/978-1-4757-4860-4_7.

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Braff, David L. "Prepulse Inhibition of the Startle Reflex: A Window on the Brain in Schizophrenia." In Behavioral Neurobiology of Schizophrenia and Its Treatment, 349–71. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/7854_2010_61.

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Light, Gregory A., and Neal R. Swerdlow. "Neurophysiological Biomarkers Informing the Clinical Neuroscience of Schizophrenia: Mismatch Negativity and Prepulse Inhibition of Startle." In Electrophysiology and Psychophysiology in Psychiatry and Psychopharmacology, 293–314. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/7854_2014_316.

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Feldon, Joram, Julia Lehmann, Christopher Pryce, and Isabelle Weiss. "Rat Latent Inhibition and Prepulse Inhibition are Sensitive to Different Manipulations of the Social Environment: A Comprehensive Study of the Environmental Approach to Neurodevelopmental Models of Schizophrenia." In Contemporary Issues in Modeling Psychopathology, 231–45. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/978-1-4757-4860-4_13.

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Conference papers on the topic "Prepulse inhibition"

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Zhou, Hongbo, Qiang Cheng, Hong-Ju Yang, and Haiyun Xu. "Weighted Kernel Density Estimation of the Prepulse Inhibition Test." In 2010 IEEE Congress on Services (SERVICES). IEEE, 2010. http://dx.doi.org/10.1109/services.2010.130.

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Lei, Ming. "Deficient spatial attentional modulation of prepulse inhibition in patients with schizophrenia." In 2017 36th Chinese Control Conference (CCC). IEEE, 2017. http://dx.doi.org/10.23919/chicc.2017.8028006.

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Fraga, Francisco J., Claudemiro V. Noya, Maria I. Zimiani, Milton A. Avila, Rosana Shuhama, Cristina M. Del-Ben, Paulo R. Menezes, Rodrigo S. Martin, and Cristiane Salum. "Simultaneous evaluation of prepulse inhibition with EMG and EEG using advanced artifact removal techniques." In 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7591914.

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Zhou, Hongbo, Hong-Ju Yang, Haiyun Xu, and Qiang Cheng. "A New Computational Tool for the Post Session Analysis of the Prepulse Inhibition Test in Neural Science." In 2009 International Conference on Computational Science and Engineering. IEEE, 2009. http://dx.doi.org/10.1109/cse.2009.61.

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Wong, Jessica M., Adam L. Halberstadt, Humberto A. Sainz, Kiran S. Mathews, Brian W. Chu, Laurel J. Ng, and Philemon C. Chan. "Mild Traumatic Brain Injury From Repeated Low-Level Blast Exposures." In ASME 2015 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/imece2015-53542.

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Recent studies on military breachers in training environments suggest that there are neurocognitive risks from exposure to repeated low-level blasts. However, the dose accumulation effects from multiple low-level blast exposures and their relation to mild traumatic brain injury (mTBI) are not well understood. This paper presents a controlled neurobehavioral study of behavioral effects from repeated low-level blasts delivered at ten second intervals using a rat model. A custom designed shock tube was developed to deliver repeated low-level blasts to rats at short intervals on the order of seconds. A total of 192 rats were divided into three cohorts of 64 for testing. Each cohort was exposed to a different blast intensity (7.5, 15, or 25 psi reflective pressure with durations <0.25 ms), and each cohort was further divided into four levels of blast repetition (0, 5, 10, or 15 repeats). Shock tube blasts were directed at the rat’s head, and startle with prepulse inhibition (PPI) and fear learning and extinction behavioral tests were performed to evaluate the blast effects. Behavioral testing results showed that repeated low-level blasts can affect PPI and contextual fear recall. PPI was not affected by repeated exposures to 7.5 psi blasts, but repeated 15 and 25 psi blasts disrupted PPI. All cohorts showed significant fear learning, but the highest blast group (25 psi, 15 repeats) had disruptions in spatial memory recall. None of the cohorts showed effects on cued fear recall or fear extinction and retention. The data collected are being used in continuous research to understand how the behavioral changes relate to mTBI, and how these animal tests can be scaled and modeled to interpret possible outcomes for humans.
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Reports on the topic "Prepulse inhibition"

1

Henderson, Domeca, Adam Lary, Lolita M. Burrell, and Michael D. Matthews. Assessing the Comprehensive Soldier Fitness Program: Measuring Startle Response and Prepulse Inhibition. Fort Belvoir, VA: Defense Technical Information Center, April 2011. http://dx.doi.org/10.21236/ada540676.

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