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Journal articles on the topic 'Premature ventricular contraction'

Author: Grafiati

Published: 28 June 2021

Last updated: 13 February 2022

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1

Oner, Taliha, Rahmi Ozdemir, Onder Doksöz, Dildar B. Genc, Baris Guven, Savas Demirpence, Murat M. Yilmazer, Yilmaz Yozgat, Timur Mese, and Vedide Tavli. "Cardiac function in children with premature ventricular contractions: the effect of omega-3 polyunsaturated fatty acid supplementation." Cardiology in the Young 28, no. 7 (May 15, 2018): 949–54. http://dx.doi.org/10.1017/s1047951118000574.

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AbstractBackgroundPremature ventricular contractions are accepted as benign in structurally normal hearts. However, reversible cardiomyopathy can sometimes develop. Omega-3 polyunsaturated fatty acids have anti-arrhythmic properties in animals and humans.AimWe evaluated left ventricular function in children with premature ventricular contractions with normal cardiac anatomy and assessed the impact of omega-3 fatty acid supplementation on left ventricular function in a prospective trial.MethodsA total of 25 patients with premature ventricular contraction, with more than 2% premature ventricular contractions on 24-hour Holter electrocardiography, and 30 healthy patients were included into study. All patients underwent electrocardiography, left ventricular M-mode echocardiography, and myocardial performance index testing. Patients with premature ventricular contraction were given omega-3 fatty acids at a dose of 1 g/day for 3 months, and control echocardiography and 24-hour Holter electrocardiography were performed. Neither placebo nor omega-3 fatty acids were given to the control group.ResultsCompared with the values of the control group, the patients with premature ventricular contraction had significantly lower fractional shortening. The myocardial performance index decreased markedly in the patient groups. The mean heart rate and mean premature ventricular contraction percentage of Group 2 significantly decreased in comparison with their baseline values after the omega-3 supplementation.ConclusionIn conclusion, premature ventricular contractions can lead to systolic cardiac dysfunction in children. Omega-3 supplementation may improve cardiac function in children with premature ventricular contractions. This is the first study conducted in children to investigate the possible role of omega-3 fatty acid supplementation on treatment of premature ventricular contractions.

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Baranov, Alexander A., Elena S. Vasichkina, Roza A. Ildarova, Dmitry S. Lebedev, Leyla S. Namazova-Baranova, Evgeniy A. Pokushalov, Sergey V. Popov, Sergey A. Termosesov, and Maria A. Shkolnikova. "Premature Ventricular Contraction in Children." Pediatric pharmacology 15, no. 6 (February 20, 2019): 435–46. http://dx.doi.org/10.15690/pf.v15i6.1981.

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The article presents updated data on the problem of premature ventricular contractions in children based on the clinical guidelines of the Russian Society of Cardiology and the Union of Pediatricians of Russia for the diagnosis, treatment and management of pediatric patients with premature ventricular contraction. The issues of diagnosis and treatment based on the principles of evidence-based medicine as well as important aspects of prevention of exacerbations and follow-up have been clarified in detail. The criteria for assessing the quality of care for patients with premature ventricular contractions have been presented.CONFLICT OF INTEREST. Not declared.

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3

Spector, Zebulon Z., and Stephen P. Seslar. "Premature ventricular contraction-induced cardiomyopathy in children." Cardiology in the Young 26, no. 4 (June 17, 2015): 711–17. http://dx.doi.org/10.1017/s1047951115001110.

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AbstractBackgroundAdults with high premature ventricular contraction burden can develop left ventricular dilation, dysfunction, and strain, consistent with a cardiomyopathy, which is reversible with radiofrequency ablation of the premature ventricular contractions. Evidence in children with similar ectopy burden is limited. We performed a single-centre retrospective review to examine the prevalence of premature ventricular contraction-induced cardiomyopathy, natural history of ventricular ectopy, and progression to ventricular tachycardia in children with frequent premature ventricular contractions.MethodsChildren aged between 6 months and 18 years, with premature ventricular contractions comprising at least 20% of rhythm on 24-hour Holter monitor, were included in our study. Those with significant structural heart disease, ventricular tachycardia greater than 1% of rhythm at the time of premature ventricular contraction diagnosis, or family history of cardiomyopathy – except tachycardia-induced – were excluded. Cardiomyopathy was defined by echocardiographic assessment.ResultsA total of 36 children met the study criteria; seven patients (19.4%, 95% CI 6.2–32.6%) met the criteria for cardiomyopathy, mostly at initial presentation. Ectopy decreased to <10% of beats without intervention in 16.7% (95% CI 4.3–29.1%) of the patients. No patient progressed to having ventricular tachycardia as more than 1% of beats on follow-up Holter. Radiofrequency ablation was performed in three patients without cardiomyopathy.ConclusionsOur study demonstrates a higher prevalence of cardiomyopathy among children with high premature ventricular contraction burden than that previously shown. Ectopy tended to persist throughout follow-up. These trends suggest the need for a multi-centre study on frequent premature ventricular contractions in children. In the interim, regular follow-up with imaging to evaluate for cardiomyopathy is warranted.

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4

Latchamsetty, Rakesh, and Frank Bogun. "Premature Ventricular Contraction Ablation." Cardiac Electrophysiology Clinics 4, no. 3 (September 2012): 439–45. http://dx.doi.org/10.1016/j.ccep.2012.05.009.

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5

Callans, David J. "Premature Ventricular Contraction-induced Cardiomyopathy." Arrhythmia & Electrophysiology Review 6, no. 4 (2017): 153. http://dx.doi.org/10.15420/aer.2017/6.4/eo1.

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Premature ventricular contractions (PVCs) are very common and usually do not require treatment. However, in the clinical setting of troublesome symptoms, or when PVCs trigger polymorphic ventricular tachycardia or cause cardiomyopathy, proper treatment is critical. In this review, the clinical syndrome of PVC-induced cardiomyopathy, including risk factors for development and treatment, is discussed. Although PVC-induced cardiomyopathy is typically associated with frequent PVCs there are also patients with this burden that do not develop cardiomyopathy, suggesting a differential susceptibility. Treatment often consists of catheter ablation, although antiarrhythmic medications may also provide both reduction in PVC frequency and resolution of left ventricular dysfunction.

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6

Saurav, Alok, Aiman Smer, Ahmed Abuzaid, Ojas Bansal, and Hussam Abuissa. "Premature Ventricular Contraction-Induced Cardiomyopathy." Clinical Cardiology 38, no. 4 (February 10, 2015): 251–58. http://dx.doi.org/10.1002/clc.22371.

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7

Lee, Andrea K. Y., and Marc W. Deyell. "Premature ventricular contraction-induced cardiomyopathy." Current Opinion in Cardiology 31, no. 1 (January 2016): 1–10. http://dx.doi.org/10.1097/hco.0000000000000236.

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8

Cha, Yong-Mei, Glenn K. Lee, Kyle W. Klarich, and Martha Grogan. "Premature Ventricular Contraction-Induced Cardiomyopathy." Circulation: Arrhythmia and Electrophysiology 5, no. 1 (February 2012): 229–36. http://dx.doi.org/10.1161/circep.111.963348.

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9

Hutchinson, Mathew D. "Idiopathic Premature Ventricular Contraction Ablation." JACC: Clinical Electrophysiology 1, no. 3 (June 2015): 124–26. http://dx.doi.org/10.1016/j.jacep.2015.05.001.

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10

Kanemori, Tetsuzou, Hideshi Ishii, Hideo Matsuhisa, Takuya Fujita, Youhei Tada, Syou Yagi, Chinami Miyazaki, et al. "Premature Ventricular Contraction Originating from Posteroseptum." Journal of Arrhythmia 27, Supplement (2011): PJ2_081. http://dx.doi.org/10.4020/jhrs.27.pj2_081.

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11

Deguchi, Akiko, Masayuki Nakamura, Takehiro Hayashi, and Akira Sano. "Quetiapine-induced frequent premature ventricular contraction." General Hospital Psychiatry 34, no. 2 (March 2012): 211.e1–211.e3. http://dx.doi.org/10.1016/j.genhosppsych.2011.11.003.

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12

Ozturk, Selcuk, and Ertan Yetkin. "Premature Ventricular Complex Causing Ice-Pick Headache." Case Reports in Cardiology 2017 (2017): 1–3. http://dx.doi.org/10.1155/2017/3879127.

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Ice pick headache is a momentary, transient, repetitive headache disorder and manifests with the stabbing pains and jolts. The exact mechanism causing this disease is unknown. Premature ventricular contractions are early depolarization of the ventricular myocardium and in the absence of a structural heart disease, it is considered to be a benign disease. In this report, we describe a male patient presenting with the symptom of momentary headache attacks accompanied with instant chest pain which is associated with premature ventricular contraction.

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13

Salavatian, Siamak, Naoko Yamaguchi, Jonathan Hoang, Nicole Lin, Saloni Patel, Jeffrey L. Ardell, J. Andrew Armour, and Marmar Vaseghi. "Premature ventricular contractions activate vagal afferents and alter autonomic tone: implications for premature ventricular contraction-induced cardiomyopathy." American Journal of Physiology-Heart and Circulatory Physiology 317, no. 3 (September 1, 2019): H607—H616. http://dx.doi.org/10.1152/ajpheart.00286.2019.

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Mechanisms behind development of premature ventricular contraction (PVC)-induced cardiomyopathy remain unclear. PVCs may adversely modulate the autonomic nervous system to promote development of heart failure. Afferent neurons in the inferior vagal (nodose) ganglia transduce cardiac activity and modulate parasympathetic output. Effects of PVCs on cardiac parasympathetic efferent and vagal afferent neurotransmission are unknown. The purpose of this study was to evaluate effects of PVCs on vagal afferent neurotransmission and compare these effects with a known powerful autonomic modulator, myocardial ischemia. In 16 pigs, effects of variably coupled PVCs on heart rate variability (HRV) and vagal afferent neurotransmission were evaluated. Direct nodose neuronal recordings were obtained in vivo, and cardiac-related afferent neurons were identified based on their response to cardiovascular interventions, including ventricular chemical and mechanical stimuli, left anterior descending (LAD) coronary artery occlusion, and variably coupled PVCs. On HRV analysis before versus after PVCs, parasympathetic tone decreased (normalized high frequency: 83.6 ± 2.8 to 72.5 ± 5.3; P < 0.05). PVCs had a powerful impact on activity of cardiac-related afferent neurons, altering activity of 51% of neurons versus 31% for LAD occlusion ( P < 0.05 vs. LAD occlusion and all other cardiac interventions). Both chemosensitive and mechanosensitive neurons were activated by PVCs, and their activity remained elevated even after cessation of PVCs. Cardiac afferent neural responses to PVCs were greater than any other intervention, including ischemia of similar duration. These data suggest that even brief periods of PVCs powerfully modulate vagal afferent neurotransmission, reflexly decreasing parasympathetic efferent tone. NEW & NOTEWORTHY Premature ventricular contractions (PVCs) are common in many patients and, at an increased burden, are known to cause heart failure. This study determined that PVCs powerfully modulate cardiac vagal afferent neurotransmission (exerting even greater effects than ventricular ischemia) and reduce parasympathetic efferent outflow to the heart. PVCs activated both mechano- and chemosensory neurons in the nodose ganglia. These peripheral neurons demonstrated adaptation in response to PVCs. This study provides additional data on the potential role of the autonomic nervous system in PVC-induced cardiomyopathy.

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14

Elsayed, Yasser Mohammed Hassanain. "Diclofenac Potassium-Induced Anaphylaxis with an Allergic Acute Coronary Syndrome and Premature Ventricular Contractions; Outpatient Clinic Management; A Case Report." Research International Journal of Cardiology and Cardiovascular Medicine 01, no. 01 (2020): 0013–16. http://dx.doi.org/10.37179/rijccm.000005.

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Drug-induced disease is a common clinical entity. Drug-inducing anaphylaxis is a serious adverse effect. Several cases of allergic acute coronary syndrome or Kounis syndrome were reported. Premature ventricular contractions are the most frequent cardiac arrhythmia with or without structural heart diseases. A premature ventricular contraction is a sign of decreased oxygenation to the myocardium and anxiety but is also found in a healthy heart. A middle-aged married male patient presented to the physician outpatient clinic with syncope within one hour after ingested one tablet of diclofenac potassium (50mg). Diclofenac potassium-induced anaphylaxis and Kounis type I syndrome with premature ventricular contractions. Electrocardiography, oxygenation, monitoring for vital signs, and echocardiography were the done interventions. The dramatic disappearance of anaphylactic shock, Kounis type I syndrome, coronary artery spasm, and premature ventricular contractions after the traditional treatment of anaphylaxis had happened. Complete clinical and electrocardiographic recovery had achieved. The identi􀏐ication of drug-induced disease is a pivotal step in the diagnosis decision making of any medical problems. Diclofenac potassium can induce anaphylactic shock, Kounis type I syndrome, coronary artery spasm, and premature ventricular contractions. Kounis type I syndrome, coronary artery spasm, and premature ventricular contractions can be reversed with treatment of the cause without using anti-ischemic or ant-arrhythmic measures. Reassurance was the recommended regards diclofenac potassium-induced both coronary artery spasm and premature ventricular contractions that accompanied by anaphylaxis.

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15

Lim, Joon-Shik. "Automatic Premature Ventricular Contraction Detection Using NEWFM." Journal of Korean Institute of Intelligent Systems 16, no. 3 (June 1, 2006): 378–82. http://dx.doi.org/10.5391/jkiis.2006.16.3.378.

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16

Kurisu, Satoshi, Ichiro Inoue, and Takuji Kawagoe. "Augmented Mechanical Alternans after Premature Ventricular Contraction." Internal Medicine 49, no. 2 (2010): 197–98. http://dx.doi.org/10.2169/internalmedicine.49.2815.

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17

Wang, Jie, Jun Li, and Bo Feng. "Shen Song Yang Xin Capsule Combined with Antiarrhythmic Drugs, a New Integrative Medicine Therapy, for the Treatment of Frequent Premature Ventricular Contractions (FPVC): A Meta-Analysis of Randomized Controlled Trials." Evidence-Based Complementary and Alternative Medicine 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/976713.

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Objective. To evaluate the beneficial and adverse effects of Shen Song Yang Xin Capsule (SSYX Capsule) combined with antiarrhythmic drugs for the treatment of frequent premature ventricular contractions (FPVC).Methods. Seven electronic databases were searched to retrieve any potential randomized controlled trials (RCTs) designed to evaluate the clinical efficacy of SSYX Capsule combined with Antiarrhythmic Drugs for FPVC reported in any language, with total effect for FPVC and number of ventricular premature contraction as the main outcome measure. The methodological quality of the included studies was assessed using criteria from the Cochrane Handbook for Systematic Review of Interventions, Version 5.1.0, and analysed using RevMan 5.1.0 software.Results. Sixteen RCTs of SSYX Capsule were included. The methodological quality of the trials was generally evaluated as low. The results of meta-analysis showed that SSYX Capsule combined with antiarrhythmic drugs was more effective in total effect for FPVC and number of ventricular premature contraction compared with Antiarrhythmic Drugs in patients with FPVC or FPVC complicated by other diseases. Ten of the trials reported adverse events, indicating that the safety of SSYX Capsule is still uncertain.Conclusions. There is some but weak evidence about SSYX Capsule combined with antiarrhythmic drugs appearing to be more effective in total effect for FPVC and number of ventricular premature contraction in patients with FPVC and its complications.

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18

SUGIMOTO, TSUNEAKI. "Ventricular arrhythmia.Ventricular fibrillation and premature ventricular contraction as its factor." Nihon Naika Gakkai Zasshi 81, no. 9 (1992): 1314–23. http://dx.doi.org/10.2169/naika.81.1314.

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19

CAMM, CHRISTIAN F., CRYSTAL TICHNELL, CYNTHIA A. JAMES, BRITTNEY MURRAY, FLORENCE PORTERFIELD, ANNELINE S. J. M. TE RIELE, HARIKRISHNA TANDRI, and HUGH CALKINS. "Premature Ventricular Contraction Variability in Arrhythmogenic Right Ventricular Dysplasia/Cardiomyopathy." Journal of Cardiovascular Electrophysiology 26, no. 1 (October 27, 2014): 53–57. http://dx.doi.org/10.1111/jce.12544.

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20

Agboola, Kolade M., Roshan Karki, and Samuel J. Asirvatham. "Ventricular automaticity: A path to premature ventricular contraction ablation success." Indian Pacing and Electrophysiology Journal 20, no. 6 (November 2020): 227–30. http://dx.doi.org/10.1016/j.ipej.2020.09.002.

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21

Witteles, Ronald, Gregory Engel, Paul J. Wang, and Amin Al-Ahmad. "Premature ventricular contractions causing pacemaker-mediated tachycardia: A failure of postventricular atrial refractory period after premature ventricular contraction extension?" Heart Rhythm 2, no. 12 (December 2005): 1389–90. http://dx.doi.org/10.1016/j.hrthm.2005.08.023.

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22

Billet, Sophie, Anne Rollin, Pierre Mondoly, Benjamin Monteil, Pauline Fournier, Eve Cariou, Marie Sadron Blaye-Felice, et al. "Hemodynamic consequences of premature ventricular contractions: Association of mechanical bradycardia and postextrasystolic potentiation with premature ventricular contraction-induced cardiomyopathy." Heart Rhythm 16, no. 6 (June 2019): 853–60. http://dx.doi.org/10.1016/j.hrthm.2018.12.008.

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23

Yang, Ting, Long Yu, Qi Jin, Liqun Wu, and Bin He. "Activation recovery interval imaging of premature ventricular contraction." PLOS ONE 13, no. 6 (June 15, 2018): e0196916. http://dx.doi.org/10.1371/journal.pone.0196916.

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24

Tran, Cao Thach, and Hugh Calkins. "Premature ventricular contraction-induced cardiomyopathy: an emerging entity." Expert Review of Cardiovascular Therapy 14, no. 11 (August 23, 2016): 1227–34. http://dx.doi.org/10.1080/14779072.2016.1222901.

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25

Aji Pramudita, B., A. Ferdita Nugraha, H. Adi Nugroho, and N. Akhmad Setiawan. "Premature Ventricular Contraction (PVC) Detection Using R Signals." KnE Life Sciences 4, no. 11 (March 10, 2019): 1. http://dx.doi.org/10.18502/kls.v4i11.3846.

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26

Ezzat, Vivienne A., Reginald Liew, and David E. Ward. "Catheter ablation of premature ventricular contraction-induced cardiomyopathy." Nature Clinical Practice Cardiovascular Medicine 5, no. 5 (March 25, 2008): 289–93. http://dx.doi.org/10.1038/ncpcardio1180.

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27

Himabindu, Prof R. "Premature Ventricular Contraction Arrhythmia Detection Using Wavelet Coefficients." IOSR Journal of Electronics and Communication Engineering 9, no. 2 (2014): 24–28. http://dx.doi.org/10.9790/2834-09252428.

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28

Wang, Yuhong, Jose M. Eltit, Karoly Kaszala, Alex Tan, Min Jiang, Mei Zhang, Gea-Ny Tseng, and Jose F. Huizar. "Cellular mechanism of premature ventricular contraction–induced cardiomyopathy." Heart Rhythm 11, no. 11 (November 2014): 2064–72. http://dx.doi.org/10.1016/j.hrthm.2014.07.022.

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29

Zamir, M., D. S. Kimmerly, and J. K. Shoemaker. "Cardiac mechanoreceptor function implicated during premature ventricular contraction." Autonomic Neuroscience 167, no. 1-2 (April 2012): 50–55. http://dx.doi.org/10.1016/j.autneu.2011.12.003.

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30

Hong-TaoYuan, Mei Yang, Li Zhong, Ying-Hsiang Lee, Vaibhav R. Vaidya, Samuel J. Asirvatham, Michael J. Ackerman, et al. "Ventricular premature contraction associated with mitral valve prolapse." International Journal of Cardiology 221 (October 2016): 1144–49. http://dx.doi.org/10.1016/j.ijcard.2016.06.252.

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31

Jingxiu, Li, Zhang Fujun, Wei Xijin, and Peng Ding. "Using Three-Dimensional Lorenz Scatter Plots to Detect Patients with Atrioventricular Node Double Path Caused by Interpolated Ventricular Premature Systoles: A Case Study." Cardiovascular Innovations and Applications 5, no. 4 (May 1, 2021): 301–6. http://dx.doi.org/10.15212/cvia.2021.0006.

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A series of related electrophysiology phenomena can be caused by the occurrence of interpolated ventricular premature contraction. In our recent three-dimensional Lorenz R-R scatter plot research, we found that atrioventricular node double path caused by interpolated ventricular premature contraction imprints a specific pattern on three-dimensional Lorenz plots generated from 24-hour Holter recordings. We found two independent subclusters separated from the interpolated premature beat precluster, the interpolated premature beat cluster, and the interpolated premature beat postcluster, respectively. Combined with use of the trajectory tracking function and the leap phenomenon, our results reveal the presence of the atrioventricular node double conduction path.

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32

Fukuhara, Eiji, Takanao Mine, and Hideyuki Kishima. "INCREASED LEFT VENTRICULAR STIFFNESS IN PATIENTS WITH SYMPTOMATIC PREMATURE VENTRICULAR CONTRACTION." Journal of the American College of Cardiology 75, no. 11 (March 2020): 339. http://dx.doi.org/10.1016/s0735-1097(20)30966-9.

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Nesterova, T. M., K. S. Ushenin, N. A. Balakina-Vikulova, and O. Solovyova. "Patial Distribution of Calcium-Overloaded Cardiomyocytes in a Cardiac Muscle: A One-Dimensional Simulation Study." Mathematical Biology and Bioinformatics 13, no. 2 (November 28, 2018): 466–79. http://dx.doi.org/10.17537/2018.13.466.

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The premature ventricular contractions are relatively common clinical diagnosis. Ventricular activation initiated from an ectopic focus in the heart ventricles results in the premature contraction. Pathophysiology of this disease is related to calcium overload in cardiomyocytes when delayed after depolarization aroused due to spontaneous calcium release from sarcoplasmic reticulum. This could lead to myocardium activation from an ectopic focus and premature ventricular contraction. In our work, we study calcium overload in cardiomyocytes using one-dimensional monodomain electrophysiological model of the myocardium. Calcium overload in cardiomyocytes is simulated by inhibition of Na+/K+ exchanger and SERCA-pump within the Noble98 cell model. We propose an algorithm to automatically detect premature activation in the one-dimensional model. The main goal of our study is to evaluate the effect of a spatial distribution of pathology on the frequency of premature myocardium excitations. Model simulation showed that an ectopic activation is usually initiated in a region of the maximum pathology. However, if the function of a spatial distribution of pathology had a discontinuity, then an ectopic activation initiated from the region of discontinuity of a function. Also, we study changes in action potential generation in the pathology region. We obtained a nonlinear and a nonlocal relationship between spatial distribution of pathology and the degree of sarcoplasmic reticulum overload in the one-dimensional model.

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34

Tseng, Gea-Ny. "More frequent postextrasystolic potentiation in patients with premature ventricular contraction–related cardiomyopathy: The missing link between premature ventricular contractions and cardiomyopathy?" Heart Rhythm 16, no. 6 (June 2019): 861–62. http://dx.doi.org/10.1016/j.hrthm.2018.12.016.

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35

Christov, I., I. Jekova, and G. Bortolan. "Premature ventricular contraction classification by theKth nearest-neighbours rule." Physiological Measurement 26, no. 1 (January 20, 2005): 123–30. http://dx.doi.org/10.1088/0967-3334/26/1/011.

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YANG, JINGKUN, RAMZI DUDUM, MALA C. MANDYAM, and GREGORY M. MARCUS. "Characteristics of Unselected High-Burden Premature Ventricular Contraction Patients." Pacing and Clinical Electrophysiology 37, no. 12 (August 1, 2014): 1671–80. http://dx.doi.org/10.1111/pace.12476.

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37

Allami, Ragheed. "Premature ventricular contraction analysis for real-time patient monitoring." Biomedical Signal Processing and Control 47 (January 2019): 358–65. http://dx.doi.org/10.1016/j.bspc.2018.08.040.

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Zhou, Xue, Xin Zhu, Keijiro Nakamura, and Mahito Noro. "Detect the Premature Ventricular Contraction Using Recurrent Neural Networks." Journal of Electrocardiology 53 (March 2019): e4-e5. http://dx.doi.org/10.1016/j.jelectrocard.2019.01.018.

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Zhou, Xue, Xin Zhu, Keijiro Nakamura, and Mahito Noro. "Detect the Premature Ventricular Contraction Using Recurrent Neural Networks." Journal of Electrocardiology 51, no. 6 (November 2018): 1165. http://dx.doi.org/10.1016/j.jelectrocard.2018.10.016.

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Hsia, Brian C., Nicolas Greige, Shreyans K. Patel, Rachel M. Clark, Kevin J. Ferrick, John D. Fisher, Jay Gross, Luigi Di Biase, and Andrew Krumerman. "Determining the optimal duration for premature ventricular contraction monitoring." Heart Rhythm 17, no. 12 (December 2020): 2119–25. http://dx.doi.org/10.1016/j.hrthm.2020.07.013.

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Liu, Yixiu, Yujuan Huang, Jianyi Wang, Li Liu, and Jiajia Luo. "Detecting Premature Ventricular Contraction in Children with Deep Learning." Journal of Shanghai Jiaotong University (Science) 23, no. 1 (February 2018): 66–73. http://dx.doi.org/10.1007/s12204-018-1911-3.

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Friedman, Daniel J., Tiffany Chasten, Kenya Anderson, Jessica Mullenix, Kimberly Rider, and Albert Y. Sun. "Premature ventricular contraction response-induced new-onset atrial fibrillation." HeartRhythm Case Reports 5, no. 3 (March 2019): 120–23. http://dx.doi.org/10.1016/j.hrcr.2018.11.012.

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Shen, Win-Kuang. "Premature ventricular contraction ablation for treatment of heart failure." Heart Rhythm 18, no. 9 (September 2021): 1613–14. http://dx.doi.org/10.1016/j.hrthm.2021.06.1198.

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Nakashima, Takashi, Konstantinos Vlachos, Philipp Krisai, and Pierre Jaïs. "Increased heart rate due to supra‐ventricular tachycardia triggering premature ventricular contraction." Journal of Cardiovascular Electrophysiology 31, no. 6 (May 15, 2020): 1544–46. http://dx.doi.org/10.1111/jce.14533.

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45

Takahashi, Satoshi, Takanao Mine, Kenki Ashida, Hideyuki Kishima, Tohru Masuyama, and Masaharu Ishihara. "Left Ventricular Inflow Velocity Pattern in Patients With Symptomatic Premature Ventricular Contraction." Circulation Journal 84, no. 1 (December 25, 2019): 26–32. http://dx.doi.org/10.1253/circj.cj-19-0605.

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46

Mumtaz, Salmaan, Junaid Mir, Vernon Chan, and Ali Akram. "HEMODIALYSIS CAUSING RECURRENT PREMATURE VENTRICULAR CONTRACTION TRIGGERED VENTRICULAR FIBRILLATION IN UREMIC CARDIOMYOPATHY." Chest 158, no. 4 (October 2020): A219. http://dx.doi.org/10.1016/j.chest.2020.08.225.

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Dandamudi, Sanjay, Susan S. Kim, Nishant Verma, S. Chris Malaisrie, Roderick Tung, and Bradley P. Knight. "Left ventricular pseudoaneurysm as a complication of left ventricular summit premature ventricular contraction ablation." HeartRhythm Case Reports 3, no. 5 (May 2017): 268–71. http://dx.doi.org/10.1016/j.hrcr.2017.01.006.

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48

Casas, Manuel M., Roberto L. Avitia, Felix F. Gonzalez-Navarro, Jose A. Cardenas-Haro, and Marco A. Reyna. "Bayesian Classification Models for Premature Ventricular Contraction Detection on ECG Traces." Journal of Healthcare Engineering 2018 (2018): 1–7. http://dx.doi.org/10.1155/2018/2694768.

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According to the American Heart Association, in its latest commission about Ventricular Arrhythmias and Sudden Death 2006, the epidemiology of the ventricular arrhythmias ranges from a series of risk descriptors and clinical markers that go from ventricular premature complexes and nonsustained ventricular tachycardia to sudden cardiac death due to ventricular tachycardia in patients with or without clinical history. The premature ventricular complexes (PVCs) are known to be associated with malignant ventricular arrhythmias and sudden cardiac death (SCD) cases. Detecting this kind of arrhythmia has been crucial in clinical applications. The electrocardiogram (ECG) is a clinical test used to measure the heart electrical activity for inferences and diagnosis. Analyzing large ECG traces from several thousands of beats has brought the necessity to develop mathematical models that can automatically make assumptions about the heart condition. In this work, 80 different features from 108,653 ECG classified beats of the gold-standard MIT-BIH database were extracted in order to classify the Normal, PVC, and other kind of ECG beats. Three well-known Bayesian classification algorithms were trained and tested using these extracted features. Experimental results show that the F1 scores for each class were above 0.95, giving almost the perfect value for the PVC class. This gave us a promising path in the development of automated mechanisms for the detection of PVC complexes.

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Kim, Jeong-Hwan, Seung-Yeon Seo, Chul-Gyu Song, and Kyeong-Seop Kim. "Assessment of Electrocardiogram Rhythms by GoogLeNet Deep Neural Network Architecture." Journal of Healthcare Engineering 2019 (April 28, 2019): 1–10. http://dx.doi.org/10.1155/2019/2826901.

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The aim of this study is to design GoogLeNet deep neural network architecture by expanding the kernel size of the inception layer and combining the convolution layers to classify the electrocardiogram (ECG) beats into a normal sinus rhythm, premature ventricular contraction, atrial premature contraction, and right/left bundle branch block arrhythmia. Based on testing MIT-BIH arrhythmia benchmark databases, the scope of training/test ECG data was configured by covering at least three and seven R-peak features, and the proposed extended-GoogLeNet architecture can classify five distinct heartbeats; normal sinus rhythm (NSR), premature ventricular contraction (PVC), atrial premature contraction (APC), right bundle branch block (RBBB), and left bundle brunch block(LBBB), with an accuracy of 95.94%, an error rate of 4.06%, a maximum sensitivity of 96.9%, and a maximum positive predictive value of 95.7% for judging a normal or an abnormal beat with considering three ECG segments; an accuracy of 98.31%, a sensitivity of 88.75%, a specificity of 99.4%, and a positive predictive value of 94.4% for classifying APC from NSR, PVC, APC beats, whereas the error rate for misclassifying APC beat was relative low at 6.32%, compared with previous research efforts.

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KOBIKI, Naoki. "Ventricular premature contraction in hypertrophic cardiomyopathy and essential hypertension with left ventricular hypertrophy." Okayama Igakkai Zasshi (Journal of Okayama Medical Association) 101, no. 9-10 (1989): 931–40. http://dx.doi.org/10.4044/joma1947.101.9-10_931.

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