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1

Magalo, Simone Issaca. "Evaluation of immunity and protection induced in pullets by the V4 oral vaccine against a pneumotropic velogenic Newcastle disease virus (NDV) strain." Diss., University of Pretoria, 2002. http://upetd.up.ac.za/thesis/available/etd-11042005-140706/.

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2

Molia, Sophie. "Avian influenza and Newcastle disease in poultry in Mali : epidemiological investigations and modelling for improved surveillance and control." Thesis, Royal Veterinary College (University of London), 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701657.

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3

Abdelrahman, Wael Hosny Abdellatif. "Avian intestinal spirochaetosis in British egg laying flocks : molecular diagnosis, epidemiology and economic impact." Thesis, Royal Veterinary College (University of London), 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559017.

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4

巫志偉 and Chi-wai Mo. "Prevention and therapy of infectious bursal disease by molecular approaches." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B30253329.

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5

Basak, Shibesh Chandra. "The identification of oocysts of chicken Eimeria species : biochemical, immunological and molecular biological approaches." Thesis, University of Liverpool, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356983.

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6

Yeung, Wing-shing, and 楊永成。. "Development of a subunit vaccine against infectious bursal disease virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31222055.

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7

Rudd, Matthew Francis, and mikewood@deakin edu au. "Virulence determinants of infectious bursal disease virus." Deakin University. School of Biological and Chemical Sciences, 2003. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20050825.103742.

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The very virulent (vv) pathotype of infectious bursal disease virus (IBDV) has spread rapidly throughout Europe, Asia, and the Middle East. Although Australia is currently unaffected, there remains the potential for incursion of an exotic isolate. The aim of this study was to identify putative virulence determinants of IBDV to facilitate the development of improved diagnostic assays for detection and characterisation of vvIBDV isolates. Sequencing of Indonesian vvIBDV Tasik94 revealed a unique substitution [ A¨S222] in the hypervariable region (HVR) of viral protein (VP) VP2, which did not appear to impinge on virulence or antigenicity. Phylogenetic analyses indicated that Tasik94 was closely related to Asian and European vvIBDV strains. Extensive alignment of deduced protein sequences across the HVR of VP2 identified residuesI242 I256 and I294 as putative markers of the vv phcnotype. Comparison of the pathology induced by mildly-virulent Australian IBDV 002/73 and Indonesian vvIBDV Tasik94, revealed that histological lesions in the spleen, thymus and bone marrow were restricted to Tasik94-infected birds, suggesting the enhanced pathogenicity of vvIBDV might be attributed to replication in non-bursal lymphoid organs. The biological significance of the VP2 HVR in virulence was assessed using recombinant viruses generated by reverse genetics. Both genomic segments of Australian IBDV 002/73, and recombinant segment A constructs in which the HVR of 002/73 was replaced with the corresponding region of either tissue culture-adapted virus or vvIBDV (Tasik94), were cloned behind T7 RNA polymerase promoter sequences. In vitro transcription/translation of each construct resulted in expression of viral proteins. Co-transfection of synthetic RNA transcripts initiated replication of both tissue culture-adapted parental and recombinant viruses, however attempts to rescue non-adapted viruses in specific-pathogen-free (SPF) chickens were unsuccessful. Nucleotide sequence variation in the HVR of VP2 was exploited for the development of a new diagnostic assay to rapidly detect exotic IBDV isolates, including vvIBDV, using reverse transcription polymerase chain reaction (RT-PCR) amplification and Bmrl restriction enzyme digestion. The assay was capable of differentiating between endemic and exotic IBDV in 96% of 105 isolates sequenced to date.
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8

Fasina, Folorunso Oludayo. "Molecular and spatial-temporal epidemiology of highly pathogenic notifiable avian influenza (HPNAI) H5N1 in Nigeria." Diss., Pretoria : [s.n.], 2008. http://upetd.up.ac.za/thesis/available/etd-02172009-171221/.

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9

Nayak, Rajesh R. "Foodborne pathogens in poultry production and post-harvest control." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1266.

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Thesis (Ph. D.)--West Virginia University, 2000.
Title from document title page. Document formatted into pages; contains x, 180 p. : ill. (some col.). Includes abstract. Includes bibliographical references.
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10

Miller, Robert Scott Oyarzabal Omar A. "Evaluation of subtyping methods for the characterization of Campylobacter strains from different geographical areas." Auburn, Ala, 2008. http://hdl.handle.net/10415/1499.

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11

Teschke, Miriam. "Prävalenz von Arcobacter spp. in Puten- und Schweinefleisch aus dem Berliner Einzelhandel und Vergleich von drei kulturellen Arcobacter-Nachweisverfahren /." Berlin : Mbv, 2008. http://d-nb.info/990056414/04.

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12

羅文新 and Man-sun Law. "DNA vaccine against chicken infectious bursal disease virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31221221.

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13

Tong, Qin. "Optimising incubation of broiler eggs using external stimuli." Thesis, Royal Veterinary College (University of London), 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701661.

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14

Pegg, Elaine. "Eimeria species as novel antimicrobial vaccine delivery vectors." Thesis, Royal Veterinary College (University of London), 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701658.

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15

Van, der Merwe Elizabeth Frances. "Preliminary investigations into ostrich mycoplasmas : identification of vaccine candidate genes and immunity elicited by poultry mycoplasma vaccines." Thesis, Stellenbosch : Stellenbosch University, 2006. http://hdl.handle.net/10019.1/17411.

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Thesis (MSc)--University of Stellenbosch, 2006.
ENGLISH ABSTRACT: Ostrich farming is of significant economical importance in South Africa. Three ostrich mycoplasmas, Ms01, Ms02 and Ms03 have been identified previously, and were provisionally named ‘Mycoplasma struthiolus’ (Ms) after their host Struthio camelus. Ostrich mycoplasmas are the major causative organisms of respiratory diseases, and they cause stock losses, reduced production and hatchability, and downgrading of carcasses and therefore lead to large economic losses to the industry. In order to be pathogenic to their host, they need to attach through an attachment organelle, the so-called tip structure. This structure has been identified in the poultry mycoplasma, M. gallisepticum, and is made up of the adhesin GapA and adhesin-related CrmA. Currently, no ostrich mycoplasma vaccine is commercially available and for this reason the need to develop one has arisen. Therefore the first part of this study was dedicated to the identification and isolation of vaccine candidate genes in the three ostrich mycoplasmas. Four primer approaches for polymerase chain reactions (PCR’s), cloning and sequencing, were used for the identification of adhesin or adhesin-related genes from Ms01, Ms02 and Ms03. The primer approaches revealed that the target genes could not be identified due to the high diversity of sequences that were generated. Therefore sequences were also compared with those of other mycoplasma species in BLAST searches. Results showed that the most significant hit was with the human pathogen M. hominis oppD, which is located in the same operon as the membrane protein P100 involved in adhesion. Other hits were with ABC transporters which may also play a role in cytadhesion. The second part of this study was aimed at testing whether two poultry mycoplasma vaccines, M. synoviae and M. gallisepticum, can be used in ostriches to elicit immune responses until an ostrich mycoplasma vaccine has been developed. Ostriches on three farms of different age groups in the Oudsthoorn district were therefore vaccinated with these vaccines in a vaccine trial. The enzymelinked immunosorbent assay (ELISA) was used to test the level of antibody response. Results showed that both vaccines elicited an immune response in all three age groups. A high percentage of the ostriches reacted positively, which indicates that both vaccines elicit antibody responses and may therefore give protection against ostrich mycoplasma infections.
AFRIKAANSE OPSOMMING: Volstruisboerdery is ‘n belangrike ekonomiese sektor in Suid-Afrika. Drie volstruismikoplasmas, Ms01, Ms02 en Ms03, is voorheen geïdentifiseer en voorlopig ‘Mycoplasma struthiolus’ (Ms) benaam na aanleiding van hul gasheer, Struthio camelus. Volstruismikoplasmas is die grootste oorsaaklike organismes van respiratoriese siektes, kudde verliese en die afgradering van karkasse wat lei tot groot ekonomiese verliese in die volstruisbedryf. Ten einde patogenies vir die gasheer te wees, moet mikoplasmas deur middel van ‘n aanhegtingsmeganisme vasheg – die sogenaamde puntvormige struktuur. Hierdie struktuur is in die pluimvee mikoplasma M. gallisepticum geïdentifiseer, en bestaan uit aanhegting proteïen GapA en die aanhegting verwante proteïen CrmA. Tans is geen volstruismikoplasma entstof kommersieel beskikbaar nie, en derhalwe het die behoefte ontstaan om so ‘n entstof te ontwikkel. Die eerste gedeelte van hierdie studie is dus gewy aan die identifisering en isolering van entstof kandidaat gene in al drie volstruismikoplasmas. Vier inleier benaderings vir polimerase ketting reaksies (PKR), klonering asook geenopeenvolging bepalings vir die identifisering van aanhegting of aanhegting verwante gene vanuit Ms01, Ms02 en Ms03 is gebruik. Die inleier benaderings het getoon dat die teikengene nie geïdentifiseer kon word nie as gevolg van hoë variasie in die gegenereerde geenopeenvolgings. Derhalwe is geenopeenvolgings met ander mikoplasma spesies deur middel van BLAST soektogte vergelyk. Resultate het getoon dat die betekenisvolste ooreenstemming dié met die menslike patogeen M. hominis oppD was, wat deel vorm van die membraan proteïen P100 operon wat betrokke is by aanhegting. Ander ooreenstemmings sluit ABC transporters in wat moontlik betrokke kan wees by aanhegting. Die tweede gedeelte van hierdie studie het ten doel gehad om te toets of twee pluimvee mikoplasma entstowwe, M. synoviae en M. gallisepticum, gebruik kan word in volstruise om immuunresponse te ontlok tot tyd en wyl ‘n volstruismikoplasma entstof ontwikkel is. Volstruise vanaf drie plase in verskillende ouderdomsgroepe in die Oudtshoorn distrik was ingeënt met hierdie entstowwe in ‘n entstof proefneming. Die ensiem-afhanklike immuno-absorpsie essaï (ELISA) was gebruik om antiliggaam response te toets. Die resultate het getoon dat beide entstowwe immuunresponse ontlok het in al drie ouderdomsgroepe. ‘n Groot persentasie van die volstruise het positief gereageer wat ‘n aanduiding is dat beide entstowwe immuunresponse ontlok het en kan dus beskerming bied teen volstruismikoplasma infeksies.
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16

Hon, Chung-chau, and 韓鍾疇. "Molecular evolution of infectious bursal disease virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B38821898.

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17

Stevens, Kim Barbra. "Risk-based decision making tools for highly pathogenic avian influenza virus (H5N1) in domestic poultry in Asia : a comparison of spatial-modelling methods." Thesis, Royal Veterinary College (University of London), 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701672.

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18

Yip, Chi-wai, and 葉志偉. "Characterization of the cell entry mechanism of infectious bursal disease virus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44756306.

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19

Höök, Helena. "Campylobacter epidemiology : insights from subtyping by pulsed-field gel electrophoresis /." Uppsala : Dept. of Biomedical Sciences and Veterinary Public Health, Swedish University of Agricultural Sciences, 2005. http://epsilon.slu.se/200589.pdf.

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20

Yip, Chi-wai, and 葉志偉. "Characterization of cellular receptors of infectious bursal disease virus in chickens." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B36759533.

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21

Liao, Qiuyan, and 廖秋燕. "Live poultry exposure and avian influenza (H5N1) risk perception in Guangzhou, 2006-2007." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41290586.

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22

Butt, Ka-man Carmen, and 畢嘉敏. "Molecular epidemiology of H9N2 avian influenza virus in poultry of southern China." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B36393915.

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23

El, Tholth Mahmoud Mohammed El Sayed. "Microbiological risk assessment at the human-animal interface : assessment of human exposure to Mycobacterium avium subspecies paratuberculosis, highly pathogenic avian influenza virus subtype HN51 and Brucella spp." Thesis, Royal Veterinary College (University of London), 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558962.

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24

Xue, Chunyi, and 薛春宜. "Molecular characterization of infectious bursal disease virus (IBDV) receptor." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31246187.

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25

Kingsbury, Laura. "Comparisons of microbial counts in organic chickens and commercially processed chickens." Menomonie, WI : University of Wisconsin--Stout, 2006. http://www.uwstout.edu/lib/thesis/2006/2006kingsburyl.pdf.

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26

Moharam, Ibrahim Mohamad Abdelrahman [Verfasser]. "Viral respiratory diseases complex: Investigation on the relevance of Newcastle disease virus in vaccinated poultry flocks in Egypt and characterization of further detected viruses / Ibrahim Mohamad Abdelrahman Moharam." Berlin : Freie Universität Berlin, 2021. http://d-nb.info/1227925921/34.

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27

Ali, Johar. "Performance, tissue selenium concentration and glutathione peroxidase activity as response variables for determining selenium requirements of poultry /." free to MU campus, to others for purchase, 2000. http://wwwlib.umi.com/cr/mo/fullcit?p9999267.

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28

Gunawardana, Gnanalatha Abeywickramasinghe. "Pasteurellosis in chickens : studies on the humoral response of chickens to Paseurelle multocida and the genetic analysis of causative strains of fowl cholera /." [St. Lucia, Qld.], 2001. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17050.pdf.

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Colles, Frances M. "Population structure and dynamics of Campylobacter populations carried by wild birds and chickens reared in a free-range woodland environment." Thesis, University of Oxford, 2006. http://ora.ox.ac.uk/objects/uuid:3dc7cdfb-29f6-4681-b8db-cb71129cd946.

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Ingestion of contaminated chicken meat is a major cause of Campylobacteriosis in Europe and the USA. The environment, including wild birds, is considered to be an important reservoir for chicken colonization. The aims of this study were to determine the population structure of Campylobacter amongst chicken and wild bird sources on a single farm, and to establish the extent to which genotypes flow between them and ultimately infect humans, using MLST and antigen sequence typing. A pilot study amongst farm animals and wild birds in Lancashire demonstrated that Campylobacter genotypes from human disease were common on the farm and could be isolated from more than one animal source. Between 30-50% of wild geese and Starlings were shedding Campylobacter, with a seasonal peak in shedding rate in Spring. Genotypes were divergent from those previously isolated from human disease, retail meat and farm animal sources, with the majority being restricted to the host source. The carriage rate of Campylobacter was between 70- 100% amongst 78 free-range poultry flocks tested at 56 days of age. Up to seven genotypes were found to co-exist within a flock, and genotypes varied throughout the year on a random basis. Some Campylobacter strains were isolated from one farm site only, but a small percentage of them had spread nationally and were stable over a period of a decade. A total of 23% of Campylobacter isolates from free-range chickens were indistinguishable to those from human disease, and 5% were indistinguishable from wild birds. A total of 6% of genotypes isolated from wild birds were indistinguishable from those isolated from human disease. Wild birds could not be completely disregarded as a potential reservoir of Campylobacter for both humans and poultry, but their role is likely to be limited.
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Lima, Anna Monteiro Correia. "Avaliação de dois sistemas de produção de frango de corte : uma visão multidisciplinar." [s.n.], 2005. http://repositorio.unicamp.br/jspui/handle/REPOSIP/257246.

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Orientador: Irenilza de Alencar Nääs
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Engenharia Agricola
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Resumo: O aumento de produção que levou o Brasil a ocupar a posição de segundo maior produtor de frangos de corte do mundo se deve a intensificação de criação. Essa intensificação teve como base vários aspectos que foram cuidadosamente controlados, dentre eles a nutrição e o manejo (sistemas de criação, ambiência, sanidade etc). Atualmente o que se observa em várias partes do mundo é a tendência em produzir frangos de corte, com bem estar animal. Paralelamente no Brasil, vem ressurgindo em nova versão a criação de frangos caipiras. O objetivo desse estudo foi avaliar índices zootécnicos obtidos durante a produção de dois sistemas de frango de corte em escala comercial (In situ), um intensivo, convencional (granja A) e outro semi-extensivo, caipira (granja B), para verificar as inter-relações entre bem-estar animal, manejo, saúde animal e qualidade da carne. Na granja A foram alojados 14000 pintos enquanto que na granja B foram 7150. Observou-se que frangos da granja A apresentaram mais problemas locomotores: calos de pés, discondroplasia tibial quando comparados com frangos provenientes da granja B. Os frangos da granja A apresentaram alta prevalência de necrose da cabeça do fêmur, melhor conversão alimentar e ganho de peso, embora tenham apresentado maior mortalidade A e B, foram respectivamente 5,32 e 1,34%. Quanto a Salmonella sp. e Mycoplasma sp. não houve presença de frangos reagentes nas duas granjas. Quanto à qualidade da carne em termos físico-químicos, o pH do peito apresentou-se menor significativamente para carcaças de frangos provenientes da granja A, enquanto o pH da coxa não apresentou diferença significativa. A força de cisalhamento da carne de frango proveniente da granja A apresentou-se menor, quando comparada à de carcaças de frangos da granja B (1,972 para carcaças de A e 2,462 para as de B). A capacidade de retenção de água não diferiu entre as carcaçaa provenientes das granjas. Em termos microbiológicos a Salmonella sp. não foi encontrada em nenhuma carcaça de frango, embora a Listeria sp. tenha sido encontrada em 50% das carcaças analisadas. Os frangos da granja B estiveram em melhores condições de bem-estar animal. Com base nesses resultados foi possível estimar que os índices zootécnicos da granja A foram melhores que os da granja B, entretanto os problemas locomotores foram maiores nos frangos provenientes da granja A
Abstract: The increase in production technology was the major factor that lead Brazil to be the second largest world poultry producer. This production technology had the basis several aspects that were carefully controlled, among them nutrition and management (environment, health and rearing systems). Nowadays it is observed a world¿s tendency to produce animal searching good welfare conditions. In parallel in Brazil is growing a new version of extensive produced broilers (free-range broiler). The objective of this study was to evaluate the production indexes from two distinct industrial scale broiler productions (in situ): one intensive (farm A) and other semi-extensive (farm B) for verifying the inter-relations among welfare, management, health and meat quality (14000 broilers in farm A and 7150 broilers in farm B). It was observed that birds from farm A showed more leg weakness (foot burn and tibial dyschondroplasia) when compared with broiler from farm B. The broilers from farm A presented high prevalence of femur necrosis, higher feed conversion, and higher gain weight, although they had presented higher mortality (5,32 in farm A and 1,34% in farm B.). Regarding Salmonella sp and Mycoplasma sp it was not found serological reagent birds in neither of the studied farms. Regarding meat quality (in the physical chemistry analysis) the breast meat pH from farm A¿s birds presented values significantly smaller than the bird¿s carcass from farm B. The shearing force on meat from broiler reared at farm A were smaller than the ones from birds reared at farm B (1,972 farm A e 2,462 farm B).. Water holding capacity did not differ from the carcasses from both farms. In microbiological terms Samonella sp was not found the studied carcasses, however Listeria sp was found in 50% of the analyzed carcasses. About welfare conditions broilers from farm B were better. Based on these results it was possible to estimate that production index was better in farm A than in farm B, however leg weakness problems were higher in broilers from farm B
Doutorado
Construções Rurais e Ambiencia
Doutor em Engenharia Agrícola
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Ghanem, Mostafa Ghanem Ahmed. "Development of Advanced Molecular Tools for Sequence Typing and Epidemiological Investigation of Avian Mycoplasma in Poultry." The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1492527446411409.

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Oliveira, Elisabete Schirato de. "Detecção e caracterização de Escherichia coli patogênica para aves (APEC) em criações de galinhas de fundo de quintal /." Jaboticabal, 2016. http://hdl.handle.net/11449/147099.

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Orientador: Fernando Antônio de Ávila
Banca: Caroline Peters Pigatto De Nardi
Banca: Patrícia Amoroso de Andrade
Banca: Hélio José Montassier
Banca: José Moacir Marin
Resumo: As condições de criação de galinhas de fundo de quintal (GFQ) apresentam alto risco sanitário, já que medidas de biossegurança nem sempre são implementadas nessas criações. Dentre as doenças infecciosas de grande destaque na avicultura está a colibacilose, cujo agente envolvido é Escherichia coli patogênica aviária (APEC). Essa enfermidade está ligada à maneira e ao ambiente em que as aves são criadas, sendo que alguns isolados de APEC podem causar infecções nas próprias aves e em humanos. O mecanismo de virulência das amostras de APEC tem sido continuamente estudado e acredita-se ser multifatorial. O objetivo do trabalho foi detectar e caracterizar isolados potencialmente APEC em criações de GFQ. Para isto foram coletadas amostras cloacais e orofaríngeas de 250 GFQ, provenientes de sete pequenas propriedades da região de Ribeirão Preto - SP. Das 500 amostras, foram obtidos 69 isolados de E. coli positivos para pelo menos 5 genes característicos de APEC. Estes foram submetidas à PCR para a detecção de mais 11 genes de virulência, apresentando alta prevalência dos mesmos. A inoculação in vivo em pintainhos de um dia revelou que 49 destes isolados são de alta e/ou intermediária patogenicidade. Os isolados também foram submetidos ao teste de suscetibilidade a 17 antimicrobianos, e apresentaram resistência a pelo menos um antimicrobiano e a maioria (79,7%) apresentou perfil de multirresistência. Além disso, foi realizada análise filogenética e foi observado que 53,6% dos isolado... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: The conditions of backyard chickens creation (BC) present a high sanitary risk, since biosafety measures are not always implemented in these systems. Among the most important infectious disease in poultry is colibacillosis, and avian pathogenic Escherichia coli (APEC) is the causative agent. This disease is related to the way and environment in which these birds are created, and some APEC isolates can cause infection in birds and humans. The virulence mechanism of APEC samples has been continuously studied and probably is multifactorial. The objective of this work was to detect and characterize potentially APEC isolates in BC creations. For that, it were collected cloacal and oropharyngeal samples of 250 BC, from seven small properties from Ribeirão Preto - SP. Of the 500 samples, 69 positive E. coli isolates were obtained for at least 5 characteristic genes of APEC. These isolates were submitted to PCR for detection of 11 more virulence genes, resulting in a high prevalence of them. The test of inoculation in one day-old chicks revealed that 49 of these isolates had high and/or intermediate pathogenicity. All isolates were also submitted to the susceptibility testing on 17 antimicrobials, and showed resistance to at least one antimicrobial agent, but the most of them (79.7%) had a multiresistance profile. In addition, phylogenetic analysis was performed and 53.6% of the isolates belonged to B2 group, which has already been described as the group harboring isolates that cause extraintestinal infections. In pulsified gel electrophoresis analysis (PFGE), it was detected a high heterogeneity of pulse types among the APEC isolates and only one sample was non-typable for XbaI enzyme. Furthermore, 15 serogroups were identified among the isolates, and O8 was the most frequent (23.2%). The results obtained in this study demonstrated that BC are (Complete abstract click electronic access below)
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33

Bresne, Carolina [UNESP]. "Suplementação da vitamina d3 (colecalciferol) e 25-ohd3 (25-hidroxi-colecalciferol) e problemas locomotores e qualidade óssea em frangos de corte." Universidade Estadual Paulista (UNESP), 2013. http://hdl.handle.net/11449/95348.

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Com o objetivo de avaliar a pododermatite e o desempenho em frangos de corte, foram alojados em aviário experimental da FMVZ-UNESP/Botucatu 750 frangos de corte machos da linhagem Cobb®, com densidade populacional de 12 aves/m2, por 42 dias. O delineamento experimental adotado foi inteiramente casualizado com cinco tratamentos e cinco repetições de 30 aves cada, totalizando 25 parcelas experimentais. Os tratamentos foram constituídos por duas fontes de suplementação de vitamina D (D3 e 25-OHD3). O tratamento 1, controle, seguiu as recomendações de Rostagno et al. (2011) para a vitamina D3, de acordo com cada fase de criação para frangos de corte. O tratamento 2 foi constituído de vitamina D3 (mesma dosagem do tratamento controle) + 1.400 UI de 25-OHD3; o tratamento 3 foi constituído de vitamina D3 (mesma dosagem do tratamento controle) + 2.800 UI de 25-OHD3; o tratamento 4 e o tratamento 5 foram constituídos apenas de vitamina D3 e, seus valores foram baseados na soma das doses em UI das duas fontes de vitamina D do tratamento 2 e do tratamento 3, respectivamente. Para o desempenho das aves foi calculado o consumo de ração, ganho de peso e conversão alimentar durante todo o período de criação, ao final foi calculada a viabilidade e o índice de eficiência produtiva. Aos 42 dias, 100 aves de cada tratamento foram avaliadas, no aviário, para pododermatite. Posteriormente as aves foram abatidas e realizadas análises histopatológicas do coxim plantar. Houve diferença pelo Teste de Quiquadrado (p<0,05) para a avaliação da incidência de pododermatite apenas para a perna esquerda. Os tratamentos não influenciaram o desempenho das aves e não foi observada diferença histopatológica entre lesões iniciais e severas, exceto em sua extensão. Conclui-se que as suplementações de...
Seven hundred and fifty Cobb® male broiler chickens were kept in a population density of 12 birds / m2 in the experimental aviary of FMVZ-UNESP/Botucatu during 42 days to evaluate the lesions of pododermatitis and performance. The experiment was carried out in a completely randomized design with five treatments and five repetitions of 30 birds each, totalizing 25 experimental parcels. Treatments were consisted of two sources of vitamin D (D3 and 25-OHD3). The first treatment (T1- control) followed the recommendations of Rostagno et al. (2011) for vitamin D3 according to each life stage of broiler chickens. Treatment 2 was the combination of vitamin D3 (the same level of control treatment) and 1,400 UI of 25-OHD3; treatment 3 was consisted of vitamin D3 (the same level of control treatment) and 2,800 UI of 25-OHD3; treatment 4 and 5 were consisted of only vitamin D3 and the levels were based on the sum of levels from the two sources of vitamin D3 of treatment 2 and 3, respectively. Feed intake, weight gain and feed:gain rate were calculated to evaluate the performance of broilers throughout the breeding period, viability and production efficiency index were also evaluated at the end. Pododermatitis and histological analyses of footpad were evaluated from one hundred broilers at 42 days of age. Through the Chi-Square Test (P<0.05) it could be seen a difference for pododermatitis incidence only for the left leg. Treatments did not affect the broiler performance and it was not observed histopathological differences between initial and severe lesions, except in its extension. In conclusion the supplementation of vitamin D3... (Complete abstract click electronic access below)
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34

Bresne, Carolina 1987. "Suplementação da vitamina d3 (colecalciferol) e 25-ohd3 (25-hidroxi-colecalciferol) e problemas locomotores e qualidade óssea em frangos de corte /." Botucatu :, 2013. http://hdl.handle.net/11449/95348.

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Orientador: Ariel Antônio Mendes
Coorientador : Márcia Regina Fernandes Boaro Martins
Banca: José Roberto Sartori
Banca: Ibiara Correia de Lima Almeida Paz
Resumo: Com o objetivo de avaliar a pododermatite e o desempenho em frangos de corte, foram alojados em aviário experimental da FMVZ-UNESP/Botucatu 750 frangos de corte machos da linhagem Cobb®, com densidade populacional de 12 aves/m2, por 42 dias. O delineamento experimental adotado foi inteiramente casualizado com cinco tratamentos e cinco repetições de 30 aves cada, totalizando 25 parcelas experimentais. Os tratamentos foram constituídos por duas fontes de suplementação de vitamina D (D3 e 25-OHD3). O tratamento 1, controle, seguiu as recomendações de Rostagno et al. (2011) para a vitamina D3, de acordo com cada fase de criação para frangos de corte. O tratamento 2 foi constituído de vitamina D3 (mesma dosagem do tratamento controle) + 1.400 UI de 25-OHD3; o tratamento 3 foi constituído de vitamina D3 (mesma dosagem do tratamento controle) + 2.800 UI de 25-OHD3; o tratamento 4 e o tratamento 5 foram constituídos apenas de vitamina D3 e, seus valores foram baseados na soma das doses em UI das duas fontes de vitamina D do tratamento 2 e do tratamento 3, respectivamente. Para o desempenho das aves foi calculado o consumo de ração, ganho de peso e conversão alimentar durante todo o período de criação, ao final foi calculada a viabilidade e o índice de eficiência produtiva. Aos 42 dias, 100 aves de cada tratamento foram avaliadas, no aviário, para pododermatite. Posteriormente as aves foram abatidas e realizadas análises histopatológicas do coxim plantar. Houve diferença pelo Teste de Quiquadrado (p<0,05) para a avaliação da incidência de pododermatite apenas para a perna esquerda. Os tratamentos não influenciaram o desempenho das aves e não foi observada diferença histopatológica entre lesões iniciais e severas, exceto em sua extensão. Conclui-se que as suplementações de... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Seven hundred and fifty Cobb® male broiler chickens were kept in a population density of 12 birds / m2 in the experimental aviary of FMVZ-UNESP/Botucatu during 42 days to evaluate the lesions of pododermatitis and performance. The experiment was carried out in a completely randomized design with five treatments and five repetitions of 30 birds each, totalizing 25 experimental parcels. Treatments were consisted of two sources of vitamin D (D3 and 25-OHD3). The first treatment (T1- control) followed the recommendations of Rostagno et al. (2011) for vitamin D3 according to each life stage of broiler chickens. Treatment 2 was the combination of vitamin D3 (the same level of control treatment) and 1,400 UI of 25-OHD3; treatment 3 was consisted of vitamin D3 (the same level of control treatment) and 2,800 UI of 25-OHD3; treatment 4 and 5 were consisted of only vitamin D3 and the levels were based on the sum of levels from the two sources of vitamin D3 of treatment 2 and 3, respectively. Feed intake, weight gain and feed:gain rate were calculated to evaluate the performance of broilers throughout the breeding period, viability and production efficiency index were also evaluated at the end. Pododermatitis and histological analyses of footpad were evaluated from one hundred broilers at 42 days of age. Through the Chi-Square Test (P<0.05) it could be seen a difference for pododermatitis incidence only for the left leg. Treatments did not affect the broiler performance and it was not observed histopathological differences between initial and severe lesions, except in its extension. In conclusion the supplementation of vitamin D3... (Complete abstract click electronic access below)
Mestre
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35

Yeung, Wing-shing. "Development of a subunit vaccine against infectious bursal disease virus /." Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B2066817X.

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36

Mo, Chi-wai. "Prevention and therapy of infectious bursal disease by molecular approaches /." Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21490156.

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37

Rutt, Julianne Eileen. "Molecular Analysis Of The Epiphyseal Growth Plate In Rachitic Broilers: Evidence For The Etilogy Of The Condition." Columbus, Ohio : Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1223319403.

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38

Mkhize, Felicity Nomfuzo. "Investigating the high incidence of bone disorders in a broiler farm : a case study." Thesis, Stellenbosch : University of Stellenbosch, 2006. http://hdl.handle.net/10019.1/3346.

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Thesis (MPhil (Animal Sciences))--University of Stellenbosch, 2006.
Rickets is described as a disease that affects young growing poultry. Poorly mineralized bones with thickened and irregular growth plates characterize it. The onset of rickets is characterized by a failure of mineralization of cartilage and bone. Other symptoms of rickets include reluctance to movement in affected birds. These birds will sit on their hocks and if startled they use their wings for balance. On necropsy, bones are soft and fragile and they have thickened growth plates. In this study 30% of the chicks aged between 7 and 8 days from a broiler flock, started showing splay leg problems. Affected chicks were unable to support their weight on their legs, some showing paralysis. The bones were soft and rubbery. To try and identify the possible cause, bones from the affected chicks were analyzed for calcium (Ca) and phosphorus (P) to determine the Ca:P ratio. Blood serum was also analyzed for the mineral content. Ca and P were the main focus of the tests as the problem was suspected to be rickets. The feed was analyzed for protein, Ca and P. The bone analysis showed a Ca:P ratio of less than 2:1, while results of the blood serum showed an inverse Ca: P ratio. The analysis results of the feed as well as the bones showed an imbalance in the Ca:P ratio which according to literature and research done is a possible cause for rickets. These findings combined with the symptoms displayed by the affected birds, lead to the suggestion that the problem in this study was rickets.
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39

Baigent, Susan Jean. "The immunological basis of genetic resistance to Marek's disease." Thesis, University of Bristol, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261251.

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40

Law, Man-sun. "DNA vaccine against chicken infectious bursal disease virus /." Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B20128393.

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41

Hon, Chung-chau. "Molecular evolution of infectious bursal disease virus." Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B38821898.

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42

Moody, Adrian John. "Mapping genetic resistance to infectious bursal disease." Thesis, University of Reading, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326754.

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43

Burgess, Shane Campbell. "Investigations into host cell-virus relationships and tumour immunity in Marek's disease." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324271.

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44

Bwala, Dauda Garba. "Challenge studies in chickens to evaluate the efficacy of commercial Newcastle disease vaccines against the strains of Newcastle disease virus prevalent in South Africa since 2002." Pretoria : [s.n.], 2010. http://upetd.up.ac.za/thesis/available/etd-02262010-123322/.

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45

Yip, Chi-wai. "Characterization of cellular receptors of infectious bursal disease virus in chickens." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B36759533.

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46

Schelling, Esther. "Epidemiological study of Newcastle disease in backyard poultry and wild bird populations in Switzerland /." [S.l.] : [s.n.], 1998. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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47

Fotouhi, Ardakani Nasser 1959. "Incidence of growth hormone gene polymorphisms in strains of chicken selected for abdominal fat content or resistance to avian leukosis virus or Marek's disease." Thesis, McGill University, 1992. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=60657.

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Chicken strains divergently selected for either abdominal fat deposition, avian leukosis (ALV) resistance or Marek's disease (MD) resistance were analyzed for restriction fragment length polymorphism (RFLP) in the growth hormone gene (GH). A total of four polymorphisms were identified, one at a SacI restriction site (PS1) and three at MspI restriction sites (PM1-PM3). Restriction mapping indicated that all polymorphisms were in exons and/or introns and not in flanking regions of the gene. Selection for abdominal fat composition significantly affected the frequency of polymorphisms at PM2, but not at the other restriction sites (p $<$ 0.0004). The PM2$ sp{+}$ allele (presence of a restriction site) was fixed in the fat line and occurred at a frequency of 63% in the lean line. Selection for ALV-resistance resulted in significant differences in the frequency of polymorphisms at PS1 and PM3. Selection for MD resistance also affected the frequency of polymorphisms at PS1 and PM3.
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48

Atkins, Katherine E. "Epidemiology and evolution of Marek's Disease virus." Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/4617.

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Marek’s disease (MD) is an oncogenic disease affecting chickens and is estimated to cost the worldwide poultry industry $1-2 billion annually. The causative agent of MD, Marek’s disease virus (MDV), provides a welldocumented example of virus virulence evolution occurring over a period of sixty years. The reason behind this evolution is unknown, although certain untested hypotheses have been suggested. These include vaccination (with increasingly potent vaccines) and other aspects of industrialisation, such as the decreased cohort duration of successive generations and an increased stocking density of the broiler flocks. In this thesis, four sections of work are undertaken. First, estimation of epidemiological parameters is tackled: virulence of MDV is quantified by looking at host mortality and virus shedding rates in vaccinated and unvaccinated birds. This is achieved via maximum likelihood estimation and Bayesian McMC techniques. Second, viral fitness is quantified by defining multiple lifetime fitness functions using the parameters previously estimated to understand the direction and force of virulence selection for different farm environments. Third, the impact of an outbreak of MDV on a broiler flock is examined by simulating a whole flock of birds. This provides an epidemiological understanding of the virus at the flock level and can help elucidate methods for disease control and surveillance and can also give a fitness measure to understand on-farm evolution of the virus. Fourth, a between-farm model is analysed to evaluate which MDV strains are able to persist in a network structure of farms and how this might be affected by biosecurity measures, different farm networks, farm size, bird lifespan and vaccination. This provides insights into how quickly a different strain can invade a farm network and the plausibility of it becoming endemic. Parameter estimation results show that the time to death for an infected bird decreases and its virus shedding rate increases with previous definitions of virulence in the literature. Model results suggest that the choice of fitness measure alters the conclusions reached. Increasing the amount of demographic structure introduced into the fitness measure shows that neither vaccine introduction nor decreasing a bird’s lifespan changes the ability of more virulent virus strains to outcompete less virulent strains. In any environment, more virulent strains are always selected for. Epidemiological results suggest that vaccination allows a low prevalence of virus on a farm although there are no deaths from the disease itself. Analogous results for between-farm spread suggest that if on-farm cleaning efficiency is low enough, a high prevalence of disease throughout a network of vaccinated flocks can exist but the farms themselves show no signs of increased mortality from the disease. The hypotheses for explaining the increase in virulence of MDV may not be consistent with the results of this work. Despite previous arguments that vaccines are driving the evolution of virulence ofMDV, this first quantitative work on the subject demonstrates that this might not be the case. This work also formulates new hypotheses to explain why MDV virulence has increased over the past sixty years which will pave the way for ongoing research in the area of virulence evolution in farm environments.
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49

Cooper, Kerry Kevin. "Necrotic Enteritis in Broiler Chickens: Studies in Disease Reproduction and Pathogenesis." Diss., The University of Arizona, 2007. http://hdl.handle.net/10150/195545.

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Necrotic enteritis in poultry is caused by Clostridium perfringens type A, and is estimated to cost the worldwide poultry industry approximately $2 billion dollars a year, due to increased mortality and decreased feed conversion and weight gain. Very little is known about the pathogenesis of this disease due to the lack of a consistently reproducible experimental model. This dissertation outlines the development of an effective and consistent experimental model for necrotic enteritis in broiler chickens. It was also found that in vivo passage through the chicken's intestinal tract let to increased virulence; we increased the proportion of birds developing disease from 34.6% to 81.4%. Researchers have proposed that alpha toxin (CPA) is believed to be the critical virulence factor of the disease. All type A isolates have the potential to produce CPA, thus we challenged birds with numerous type A isolates that are virulent in other animal hosts. However, we found that they did not produce necrotic enteritis in broiler chickens. In addition, challenge with culture supernatant alone failed to produce gross lesions in the birds, although challenging with washed whole cell cultures did do so. Vaccinating birds with HIS-tagged recombinant CPA provided partial protection against disease; there was a 42.0% decrease in lesion development. The conclusion of this doctoral research is that CPA does have a role in the pathogenesis of necrotic enteritis in broiler chickens, but there are apparently other critical virulence factors involved in the development of disease.
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50

Stringfellow, Kendre Duaron. "Evaluation of agricultural disinfectants and necrotic enteritis preventatives in broiler chickens." [College Station, Tex. : Texas A&M University, 2008. http://hdl.handle.net/1969.1/ETD-TAMU-3237.

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