Relevant bibliographies by topics / Potassium Ion Cells

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Potassium Ion Cells'

Author: Grafiati
Published: 6 September 2023

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Journal articles on the topic "Potassium Ion Cells"

1

Reichenbach, Andreas, Andre Henke, Wolfgang Eberhardt, Winfried Reichelt, and Dietrich Dettmer. "K+ ion regulation in retina." Canadian Journal of Physiology and Pharmacology 70, S1 (May 15, 1992): S239—S247. http://dx.doi.org/10.1139/y92-267.

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During onset and offset of illumination, considerable changes in extracellular K+ concentration ([K+]e) occur within particular retinal layers. There are two ways in which glial cells may control [K+]e: (1) by space-independent processes, for example, by K+ uptake due to the Na+–K+ ATPase, and (2) by space-dependent processes, that is, by spatial buffering currents flowing through K+ channels. Rabbit retinal Müller (glial) cells were studied for expression of mechanisms supporting both kinds of processes. This review demonstrates that rabbit Müller cells have Na–K pumps whose distribution and properties are highly adapted to meet the needs of efficient K+ clearance. Furthermore, spatial buffering currents through specialized K+ channels of Müller cells greatly accelerate retinal K+ clearance during and after stimulation.Key words: glia, retina, potassium clearance, sodium–potassium pump, potassium channels.
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ZHONG, YI-SHENG, JING WANG, WANG-MIN LIU, and YI-HUA ZHU. "Potassium ion channels in retinal ganglion cells (Review)." Molecular Medicine Reports 8, no. 2 (June 4, 2013): 311–19. http://dx.doi.org/10.3892/mmr.2013.1508.

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Pramudita, James C., Vanessa K. Peterson, Justin A. Kimpton, and Neeraj Sharma. "Potassium-ion intercalation in graphite within a potassium-ion battery examined usingin situX-ray diffraction." Powder Diffraction 32, S2 (September 4, 2017): S43—S48. http://dx.doi.org/10.1017/s0885715617000902.

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Graphite has been widely used as a negative electrode material in lithium-ion batteries, and recently it has attracted attention for its use in potassium-ion batteries. In this study, the firstin situX-ray diffraction characterisation of a K/graphite electrochemical cell is performed. Various graphite intercalation compounds are found, including the stage three KC36and stage one KC8compounds,along with the disappearance of the graphite during the potassiation process. These results show new insights on the non-equilibrium states of potassium-ion intercalation into graphite in K/graphite electrochemical cells.
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Van Mil, H. G. J. "Analysis of a Model Describing the Dynamics of Intracellular Ion Composition in Biological Cells." International Journal of Bifurcation and Chaos 08, no. 05 (May 1998): 1043–47. http://dx.doi.org/10.1142/s0218127498000851.

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An electrophysiological model describing the dynamics of the intracellular ion concentration and the membrane potential (Vm) in biological cells is presented. The model links passive ion fluxes through channels of sodium, potassium and chloride to active ion fluxes generated by the sodium potassium pump. To model the interaction of Vm to the ionic fluxes Kirchhoff current law is used. Only one Vm-dependent permeability as represented by an inwardly rectifying potassium channel (IKR) is incorporated. It is shown that the resulting system of ordinary differential equations is degenerate. Decomposition of the system into noninteracting subsystems allows a dynamically independent description of the currents of sodium and potassium in relation to Vm. Physical and mathematical arguments for the decomposition into subsystems are presented. Analysis of the model show hysteresis properties that can account for the experimentally-observed bistability in skeletal and heart muscles fibers.
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Quraishi, Imran H., and Robert M. Raphael. "Computational model of vectorial potassium transport by cochlear marginal cells and vestibular dark cells." American Journal of Physiology-Cell Physiology 292, no. 1 (January 2007): C591—C602. http://dx.doi.org/10.1152/ajpcell.00560.2005.

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Cochlear marginal cells and vestibular dark cells transport potassium into the inner ear endolymph, a potassium-rich fluid, the homeostasis of which is essential for hearing and balance. We have formulated an integrated mathematical model of ion transport across these epithelia that incorporates the biophysical properties of the major ion transporters and channels located in the apical and basolateral membranes of the constituent cells. The model is constructed for both open- and short-circuit situations to test the extremes of functional capacity of the epithelium and predicts the steady-state voltages, ion concentrations, and transepithelial currents as a function of various transporter and channel densities. We validate the model by establishing that the cells are capable of vectorial ion transport consistent with several experimental measurements. The model indicates that cochlear marginal cells do not make a significant direct contribution to the endocochlear potential and illustrates how changes to the activity of specific transport proteins lead to reduced K+ flux across the marginal and dark cell layers. In particular, we investigate the mechanisms of loop diuretic ototoxicity and diseases with hearing loss in which K+ and Cl− transport are compromised, such as Jervell and Lange-Nielsen syndrome and Bartter syndrome, type IV, respectively. Such simulations demonstrate the utility of compartmental modeling in investigating the role of ion homeostasis in inner ear physiology and pathology.
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Cho, Youngtak, Viet Anh Pham Ba, Jin-Young Jeong, Yoonji Choi, and Seunghun Hong. "Ion-Selective Carbon Nanotube Field-Effect Transistors for Monitoring Drug Effects on Nicotinic Acetylcholine Receptor Activation in Live Cells." Sensors 20, no. 13 (June 30, 2020): 3680. http://dx.doi.org/10.3390/s20133680.

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We developed ion-selective field-effect transistor (FET) sensors with floating electrodes for the monitoring of the potassium ion release by the stimulation of nicotinic acetylcholine receptors (nAChRs) on PC12 cells. Here, ion-selective valinomycin-polyvinyl chloride (PVC) membranes were coated on the floating electrode-based carbon nanotube (CNT) FETs to build the sensors. The sensors could selectively measure potassium ions with a minimum detection limit of 1 nM. We utilized the sensor for the real-time monitoring of the potassium ion released from a live cell stimulated by nicotine. Notably, this method also allowed us to quantitatively monitor the cell responses by agonists and antagonists of nAChRs. These results suggest that our ion-selective CNT-FET sensor has potential uses in biological and medical researches such as the monitoring of ion-channel activity and the screening of drugs.
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Hedrich, Rainer. "Ion Channels in Plants." Physiological Reviews 92, no. 4 (October 2012): 1777–811. http://dx.doi.org/10.1152/physrev.00038.2011.

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Since the first recordings of single potassium channel activities in the plasma membrane of guard cells more than 25 years ago, patch-clamp studies discovered a variety of ion channels in all cell types and plant species under inspection. Their properties differed in a cell type- and cell membrane-dependent manner. Guard cells, for which the existence of plant potassium channels was initially documented, advanced to a versatile model system for studying plant ion channel structure, function, and physiology. Interestingly, one of the first identified potassium-channel genes encoding the Shaker-type channel KAT1 was shown to be highly expressed in guard cells. KAT1-type channels from Arabidopsis thaliana and its homologs from other species were found to encode the K+-selective inward rectifiers that had already been recorded in early patch-clamp studies with guard cells. Within the genome era, additional Arabidopsis Shaker-type channels appeared. All nine members of the Arabidopsis Shaker family are localized at the plasma membrane, where they either operate as inward rectifiers, outward rectifiers, weak voltage-dependent channels, or electrically silent, but modulatory subunits. The vacuole membrane, in contrast, harbors a set of two-pore K+ channels. Just very recently, two plant anion channel families of the SLAC/SLAH and ALMT/QUAC type were identified. SLAC1/SLAH3 and QUAC1 are expressed in guard cells and mediate Slow- and Rapid-type anion currents, respectively, that are involved in volume and turgor regulation. Anion channels in guard cells and other plant cells are key targets within often complex signaling networks. Here, the present knowledge is reviewed for the plant ion channel biology. Special emphasis is drawn to the molecular mechanisms of channel regulation, in the context of model systems and in the light of evolution.
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CORREIA, MANNING J., KATHERINE J. RENNIE, and PAUL KOO. "Return of Potassium Ion Channels in Regenerated Hair Cells." Annals of the New York Academy of Sciences 942, no. 1 (January 25, 2006): 228–40. http://dx.doi.org/10.1111/j.1749-6632.2001.tb03749.x.

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Berkowitz, L. R., and E. P. Orringer. "Passive sodium and potassium movements in sickle erythrocytes." American Journal of Physiology-Cell Physiology 249, no. 3 (September 1, 1985): C208—C214. http://dx.doi.org/10.1152/ajpcell.1985.249.3.c208.

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Deoxygenation causes an increase in passive Na and K movements across the membrane of the sickle erythrocyte. Some investigators find that these ion movements are accompanied by cell dehydration, while others find no evidence for cell water loss with sickling. Because gelation of hemoglobin S would be enhanced by cell water loss, we reinvestigated Na and K movements in sickle cells to define further the role that ion movements might play in the pathogenesis of sickling. With deoxygenation, we found that sickle cells gained Na and lost K without losing cell water. These net ion movements were not seen in control red blood cells. For sickle cells, deoxygenation also increased passive unidirectional influxes of Na and K, effects not observed when control red blood cells were deoxygenated. The deoxygenation-induced passive influxes of Na and K in sickle cells were not diminished by anion substitution or by the addition of the diuretic furosemide. We also found differences in passive Na and K fluxes between oxygenated sickle cells and normal red blood cells. The addition of furosemide or replacement of Cl with NO3 or SCN, maneuvers that largely reduced passive Na and K movements in oxygenated normal cells, had no effect on Na and K movements in oxygenated sickle cells. These findings militate against the idea that solute and water loss occur as a consequence of deoxygenation but do indicate that there are acquired membrane abnormalities in sickle red blood cells.
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Younes, Samar, Nisreen Mourad, Mohamed Salla, Mohamad Rahal, and Dalal Hammoudi Halat. "Potassium Ion Channels in Glioma: From Basic Knowledge into Therapeutic Applications." Membranes 13, no. 4 (April 15, 2023): 434. http://dx.doi.org/10.3390/membranes13040434.

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Ion channels, specifically those controlling the flux of potassium across cell membranes, have recently been shown to exhibit an important role in the pathophysiology of glioma, the most common primary central nervous system tumor with a poor prognosis. Potassium channels are grouped into four subfamilies differing by their domain structure, gating mechanisms, and functions. Pertinent literature indicates the vital functions of potassium channels in many aspects of glioma carcinogenesis, including proliferation, migration, and apoptosis. The dysfunction of potassium channels can result in pro-proliferative signals that are highly related to calcium signaling as well. Moreover, this dysfunction can feed into migration and metastasis, most likely by increasing the osmotic pressure of cells allowing the cells to initiate the “escape” and “invasion” of capillaries. Reducing the expression or channel blockage has shown efficacy in reducing the proliferation and infiltration of glioma cells as well as inducing apoptosis, priming several approaches to target potassium channels in gliomas pharmacologically. This review summarizes the current knowledge on potassium channels, their contribution to oncogenic transformations in glioma, and the existing perspectives on utilizing them as potential targets for therapy.
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Dissertations / Theses on the topic "Potassium Ion Cells"

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Walker, David James. "Potassium compartmentation in barley root cells." Thesis, University of Nottingham, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319644.

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Ketchum, Karen Ann. "A calcium-dependent potassium channel in corn (Zea mays) suspension cells /." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74658.

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Three distinct K$ sp+$ currents were identified in corn (Zea mays) protoplasts using the whole-cell patch-clamp technique. Inward-rectifying K$ sp+$ currents were evoked at membrane potentials more negative than $-$100 mV. The activation range was sensitive to external K$ sp+$ and shifted in the positive direction as the K$ sp+$ concentration was elevated. The second K$ sp+$ current was voltage-independent and contributed to the resting membrane conductance of the protoplast. Finally, a voltage- and Ca$ sp{2+}$-dependent K$ sp+$ current was observed at potentials positive to $-$60 mV. This current was inhibited by reagents which antagonize plasmalemma Ca$ sp{2+}$ influx (e.g. nitrendipine, verapamil). In contrast, currents were enhanced by increasing the cytosolic free Ca$ sp{2+}$ concentration from 40 to 400 nM. The Ca$ sp{2+}$-dependent K$ sp+$ current was inhibited by tetraethylammonium ions, Cs$ sp+$, Ba$ sp{2+}$, and charybdotoxin which suggested that the channel protein has structural similarities to the high conductance Ca$ sp{2+}$-dependent K$ sp+$ channel observed in animal systems.
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Andersson, Britta. "Manipulation of potassium ion fluxes to induce apoptosis in lung cancer cells." Doctoral thesis, Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1014.

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Sculptoreanu, Adrian Carleton University Dissertation Biology. "Intracellular concentration and membrane permeability ratio of sodium and potassium ion in cultured cardiomyocytes of the adult rat." Ottawa, 1988.

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Newton, Hannah S. "Potassium channels and adenosine signaling in T cells of head and neck cancer patients." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1603713656776019.

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Sridhar, Arun. "Regulation of cardiac voltage gated potassium currents in health and disease." Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1186603836.

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Simmons, Christina Nicole. "FABRICATION OF AN EPITHELIAL CELL-BASED ION-SELECTIVE ELECTRODE AND ITS APPLICATION FOR USE AS ALTERNATIVE TUMOR ANGIOGENESIS ASSAY." UKnowledge, 2012. http://uknowledge.uky.edu/cme_etds/11.

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Previous studies have provided evidence that endothelial cell-based potassium ion selective electrodes possess the ability to quantify substances that have permeability-altering effects on those endothelial cells. The capability of these so-called biosensors to detect elevated concentrations of certain chemical agents found following tumor formation make them useful in the application as an alternative tumor angiogenesis assay. In this study an epithelial cell line, human colon adenocarcinoma epithelial cells (Caco-2), was used to fabricate membranes that were used to test concentrations of these chemical agents, known as cytokines, mimicking the concentrations that have been observed in the serum of healthy individuals as well as the higher concentration found in individuals with cancer. Additionally background information is provided related to the development of whole cell-based biosensors, metabolic pathways related to tumor angiogenesis and the subsequent increase in cytokine concentration, properties of the Caco-2 cell line that make them useful for the application in cell-based biosensors, and the ultimate effect the cytokines have on the permeability of the cells.
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Buchin, Anatoly. "Modeling of single cell and network phenomena of the nervous system : ion dynamics during epileptic oscillations and inverse stochastic resonance." Thesis, Paris, Ecole normale supérieure, 2015. http://www.theses.fr/2015ENSU0041/document.

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Dans cette thèse nous avons utilisé des méthodes de systèmes dynamiques et des simulations numériques pour étudier les mécanismes d'oscillations d'épilepsie associés à des concentrations d’ions dynamiques et au comportement bimodal des cellules Purkinje du cervelet. Le propos général de ce travail est l'interaction entre les propriétés intrinsèques des neurones simple et la structure d'entrée synaptique contrôlant l'excitabilité neuronale. Dans la première partie de la thèse nous avons développé un modèle de transition de crise épileptique dans le lobe temporal du cerveau. Plus précisément nous nous sommes concentrés sur le rôle du cotransporteur KCC2, qui est responsable de la maintenance du potassium extracellulaire et du chlorure intracellulaire dans les neurones. Des données expérimentales récentes ont montré que cette molécule est absente dans un groupe significatif de cellules pyramidales dans le tissue neuronal de patients épileptiques suggérant son rôle épileptogène. Nous avons trouvé que l'addition d’une quantité critique de cellules pyramidale KCC2 déficient au réseau de subiculum, avec une connectivité réaliste, peut provoquer la génération d’oscillations pathologiques, similaire aux oscillations enregistrées dans des tranches de cerveau épileptogène humaines. Dans la seconde partie de la thèse, nous avons étudié le rôle du bruit synaptique dans les cellules de Purkinje. Nous avons étudié l'effet de l'inhibition de la génération du potentiel d’action provoquée par injection de courant de bruit, un phénomène connu comme résonance stochastique inverse (RSI). Cet effet a déjà été trouvé dans des modèles neuronaux, et nous avons fournis sa première validation expérimentale. Nous avons trouvé que les cellules de Purkinje dans des tranches de cerveau peuvent être efficacement inhibées par des injectionsde bruit de courant. Cet effet est bien reproduit par le modèle phénoménologique adapté pour différentes cellules. En utilisant des méthodes de la théorie de l'information, nous avons montré que RSI prend en charge une transmission efficace de l'information des cellules de Purkinje simples suggérant son rôle pour les calculs du cervelet
In this thesis we used dynamical systems methods and numericalsimulations to study the mechanisms of epileptic oscillations associated with ionconcentration changes and cerebellar Purkinje cell bimodal behavior. The general issue in this work is the interplay between single neuron intrinsicproperties and synaptic input structure controlling the neuronal excitability. In the first part of this thesis we focused on the role of the cellular intrinsicproperties, their control over the cellular excitability and their response to thesynaptic inputs. Specifically we asked the question how the cellular changes ininhibitory synaptic function might lead to the pathological neural activity. We developed a model of seizure initiation in temporal lobe epilepsy. Specifically we focused on the role of KCC2 cotransporter that is responsible for maintaining the baseline extracellular potassium and intracellular chloride levels in neurons. Recent experimental data has shown that this cotransporter is absent in the significant group of pyramidal cells in epileptic patients suggesting its epileptogenic role. We found that addition of the critical amount of KCC2-deficient pyramidal cells to the realistic subiculum network can switch the neural activity from normal to epileptic oscillations qualitatively reproducing the activity recorded in human epileptogenic brain slices. In the second part of this thesis we studied how synaptic noise might control the Purkinje cell excitability. We investigated the effect of spike inhibition caused by noise current injection, so-called inverse stochastic resonance (ISR). This effect has been previously found in single neuron models while we provided its first experimental evidence. We found that Purkinje cells in brain slices could be efficiently inhibited by current noise injections. This effect is well reproduced by the phenomenological model fitted for different cells. Using methods of information theory we showed that ISR supports an efficient information transmission of single Purkinje cells suggesting its role for cerebellar computations
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Chapman, Joanna Claire. "Potassium ion channels and disorders of glucose regulation." Thesis, University of Sheffield, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322879.

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Jenkins, Richard J. "The mechanisms whereby the sodium, potassium-ATPhase undergoes adaptive changes in human lymphocytes in response to lithium." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236273.

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Books on the topic "Potassium Ion Cells"

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Yoshihisa, Kurachi, Jan Lily Yeh, and Lazdunski Michel, eds. Potassium ion channels: Molecular structure, function, and diseases. San Diego: Academic Press, 1999.

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Ian, Glynn, Ellory J. C, and Company of Biologists, eds. The sodium pump: Proceedings of the Fourth International Conference on Na, K-ATPase, held at the Physiological Laboratory, Cambridge, in August 1984. Cambridge, U.K: Company of Biologists, 1985.

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E, Vance Dennis, and Vance Jean E, eds. Biochemistry of lipids, lipoproteins, and membranes. Amsterdam: Elsevier, 1991.

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Adragna, Norma, and Peter Lauf. Cell Volume and Signaling. Springer, 2014.

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Cell Volume and Signaling (Advances in Experimental Medicine and Biology). Springer, 2005.

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Benos, Dale J., Arnost Kleinzeller, Lily Yeh Jan, Douglas M. Fambrough, and Yoshihisa Kurachi. Potassium Ion Channels: Molecular Structure, Function, and Diseases. Elsevier Science & Technology Books, 1999.

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Doucet, Alain, and Gilles Crambert. Potassium homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0023.

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The equilibrium between the concentration of K+ in the extracellular space (low) and the intracellular compartment (high) is crucial for maintaining the electrical properties of excitable and non-excitable cells, because it determines the membrane resting potential. The high intracellular concentration of K+ (120–140 mmol/L) also contributes to the intracellular osmolarity, a determinant of cell volume. It is therefore crucial to finely tune both extracellular and intracellular K+ concentrations. There is a coordinated regulation between processes/mechanisms that store/release K+ from internal stores (internal balance) and those that retain/excrete K+ (external balance).
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(Editor), Yoshihisa Kurachi, Lily Yeh Jan (Editor), Michel Lazdunski (Editor), Arnost Kleinzeller (Series Editor), Douglas M. Fambrough (Series Editor), and Dale J. Benos (Series Editor), eds. Current Topics in Membrances, Volume 46: Potassium Ion Channels: Molecular Structure, Function and Disease (Current Topics in Membranes). Academic Press, 1999.

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Potassium malate transport into plant cell vacuoles: The characterization of ion channels. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Frise, Matthew C., and Jonathan B. Salmon. Disorders of potassium in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0251.

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Plasma potassium levels are maintained in health between 3.5 and 5.0 mmol/L, and reflect total body potassium only in stable states at normal pH. Most true hyperkalaemia results from renal insufficiency. The goals of therapy are myocardial protection and return of plasma potassium to a safe level. Measures are commonly initiated above 5.5 mmol/L; above 6.5 mmol/L, aggressive measures should be adopted and calcium salts given if there are cardiac dysrhythmias or QRS-broadening. Glucose-insulin infusions and beta-2-agonists promote potassium shifts into cells. Diuretics and sodium bicarbonate may be helpful, but persistent hyperkalaemia is an indication for renal replacement therapy. Hypokalaemia may lead to dangerous arrhythmias, skeletal muscle weakness, ileus, and reduced vascular smooth muscle contractility. Rapid replacement should only be undertaken for severe hypokalaemia or in the context of arrhythmias. Once the extracellular deficit is corrected, there will usually be a continuing need for potassium supplementation to replenish intracellular stores.
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Book chapters on the topic "Potassium Ion Cells"

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Lingle, Christopher J., Christopher R. Solaro, Murali Prakriya, and Jiu Ping Ding. "Calcium-Activated Potassium Channels in Adrenal Chromaffin Cells." In Ion Channels, 261–301. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-1775-1_7.

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Hustad, Kristian Gregorius, Ena Ivanovic, Adrian Llop Recha, and Abinaya Abbi Sakthivel. "Conduction Velocity in Cardiac Tissue as Function of Ion Channel Conductance and Distribution." In Computational Physiology, 41–50. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-05164-7_4.

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AbstractIon channels on the membrane of cardiomyocytes regulate the propagation of action potentials from cell to cell and hence are essential for the proper function of the heart. Through computer simulations with the classical monodomain model for cardiac tissue and the more recent extracellular-membrane-intracellular (EMI) model where individual cells are explicitly represented, we investigated how conduction velocity (CV) in cardiac tissue depends on the strength of various ion currents as well as on the spatial distribution of the ion channels. Our simulations show a sharp decrease in CV when reducing the strength of the sodium (Na+) currents, whereas independent reductions in the potassium (K1 and Kr) and L-type calcium currents have negligible effect on the CV. Furthermore, we find that an increase in number density of Na+ channels towards the cell ends increases the CV, whereas a higher number density of K1 channels slightly reduces the CV. These findings contribute to the understanding of ion channels (e.g. Na+ and K+ channels) in the propagation velocity of action potentials in the heart.
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Albarwani, Sulayma, and Piers Nye. "An ATP-Activated Potassium Channel in Smooth Muscle Cells from the Pulmonary Artery." In Ion Flux in Pulmonary Vascular Control, 149–57. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2397-0_12.

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Benot, Alberto R., María D. Ganfornina, and José López-Barneo. "Potassium Channel Modulated by Hypoxia and the Redox Status in Glomus Cells of the Carotid Body." In Ion Flux in Pulmonary Vascular Control, 177–87. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2397-0_14.

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Rizzoli, R., C. Summonte, R. Galloni, F. Zignani, and A. Nylandsted Larsen. "Potassium Ion Implantation Doping of the n-Layer for p-i-n Amorphous Silicon Solar Cells." In Tenth E.C. Photovoltaic Solar Energy Conference, 1072–74. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3622-8_274.

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Leonard, R. T. "Absorption of Potassium into Root Cells." In Potassium in Agriculture, 325–35. Madison, WI, USA: American Society of Agronomy, Crop Science Society of America, Soil Science Society of America, 2015. http://dx.doi.org/10.2134/1985.potassium.c12.

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Droogmans, Guy, and Bernd Nilius. "Overview: Potassium Channels in Vascular Endothelial Cells." In Potassium Channels in Cardiovascular Biology, 639–50. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1303-2_31.

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Clausen, Michael Jakob Voldsgaard, and Hanne Poulsen. "Sodium/Potassium Homeostasis in the Cell." In Metal Ions in Life Sciences, 41–67. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-5561-1_3.

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Kume, Hiroaki. "Large-Conductance Calcium-Activated Potassium Channels." In Calcium Signaling In Airway Smooth Muscle Cells, 49–83. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-01312-1_4.

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Triggle, Christopher R. "Endothelial Cell K+ Channels, Membrane Potential and the Release of Vasoactive Factors from the Vascular Endothelium." In Potassium Channels in Cardiovascular Biology, 667–89. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1303-2_33.

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Conference papers on the topic "Potassium Ion Cells"

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Fedorova, E. E., and N. A. Trifonova. "Ion transporters in the root nodule of Medicago truncatula: potassium transporters." In 2nd International Scientific Conference "Plants and Microbes: the Future of Biotechnology". PLAMIC2020 Organizing committee, 2020. http://dx.doi.org/10.28983/plamic2020.071.

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The transporter proteins were mistargeted and partly depleted from plasma membrane of mature infected cells, this phenomenon may contribute to the potassium loss by symbiosomes during their development and senescence.
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Futscher, Moritz, Lucie McGovern, Kangyu Ji, Sandy Sanchez, Sam Stranks, and Bruno Ehrler. "Ion Migration in Triple-Cation Mixed-Halide Perovskite Solar Cells with Potassium Passivation." In 11th International Conference on Hybrid and Organic Photovoltaics. València: Fundació Scito, 2019. http://dx.doi.org/10.29363/nanoge.hopv.2019.033.

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Sakai, T., K. Okada, H. Bando, S. Ueshima, N. Tanaka, and O. Matsuo. "ANALYSIS OF THE SECRETION MECHANISM OF TISSUE-TYPE PLASMINOGEN ACTIVATOR IN A HUMAN MELANOMA CELL LINE (BOWES)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644397.

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The human melanoma cell line (Bowes) secretes tissue-type plasminogen activator (t-PA) into the culture medium. As reported previously, the secretion of t-PA was depressed and t-PA was accumulated in the intracellular compartment at alternative sodium and potassium ion concentrations and also in the presence of monensin, an ionophore for monovalent cations. In the present study, the secretion and intracellular distribution of t-PA were investigated by radioisotope labeling and fractionation of the cell organelles under normal and monensin-treated conditions. Cell homogenate was fractioneted by discontinuous sucrose density gradient ultracentrifugation. The plasminogen activator (PA) activity or t-PA antigenicity in these fractions was not uniformly distributed, but rather localized in those fractions where cell organelles were rich. This implied that t-PA was enclosed by the intracellular membranous system. Cells were incubated with 35s-methionine and/or 3H-mannose to produce labeled intracellular glycoproteins including t-PA and its premature intermediates, which were separated by immunological adsorption to rabbit anti t-PA IgG-protein A Sepharose. Pulse labeling with 35s-methionine (3 min) demonstrated that intracellular t-PA was transported from the heavier fractions (rER), via intermediate ones (Golgi complex), to lighter ones. The radioactivity of the intracellular t-PA reached a maximum in 30 min, while that of secreted t-PA was observed in 30 min and increased linearly at least for the following 20 min. Enzymography revealed the major intracellular PA activity at 72 kDa and minor activity at 50 kDa. Monensin treatment (10 μM, 6 hr) caused accumulation of the 72 kDa component which was immunologically homologous to secreted t-PA. After long term (3 hr) simultaneous 35s-methionine and 3H-mannose labeling, the radioactivity ratio (3H/35S) the intracellular t-PA was increased in the presence of monensin. These results suggest that some sugar chain(s) in the t-PA molecule are of high mannose type, which is interfered with by monensin in the course of the intracellular processing and transport of t-PA.
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Morris, Ronald. "Chemical Decontamination for Decommissioning (DFD) and DFDX." In ASME 2010 13th International Conference on Environmental Remediation and Radioactive Waste Management. ASMEDC, 2010. http://dx.doi.org/10.1115/icem2010-40007.

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DFD is an acronym for the “Decontamination for Decommissioning” process developed in 1996 by the Electric Power Research Institute (EPRI). The process was designed to remove radioactivity from the surfaces of metallic components to allow these components to be recycled or free-released for disposal as non-radioactive. DFD is a cyclic process consisting of fluoroboric acid, potassium permanganate and oxalic acid. The process continues to uniformly remove base metal once oxide dissolution is complete. The DFD process has been applied on numerous components, sub-systems and systems including the reactor systems at Big Rock Point and Maine Yankee in the United States, and the Jose Cabrera (Zorita) Nuclear Power Plant (NPP) in Spain. The Big Rock Point site has been returned to Greenfield and at Maine Yankee the land under the license was reduced for an Independent Spent Fuel Storage Installation (ISFSI). In the upcoming months the Zorita NPP in Spain will initiate dismantlement and decommissioning activities to return the site to a non-nuclear facility. The development of the EPRI DFD process has been an ongoing evolution and much has been learned from its use in the past. It is effective in attaining very high decontamination factors; however, DFD also produces secondary waste in the form of ion exchange resins. This secondary waste generation adds to the decommissioning quota but this can be improved upon at a time when radioactive waste storage at nuclear facilities and waste disposal sites is limited. To reduce the amount of secondary waste, EPRI has developed the DFDX process. This new process is an enhancement to the DFD process and produces a smaller amount of metallic waste rather than resin waste; this reduction in volume being a factor of ten or greater. Electrochemical ion exchange cells are the heart of the DFDX system and contain electrodes and cation ion exchange resin. It has been used very successfully in small system applications and the next evolution is to design, build and implement a system for the chemical decontamination for decommissioning of larger reactor systems and components, and Full System Decontamination (FSD). The purpose of this paper is to provide a reference point for those planning future chemical decontaminations for plant decommissioning. It is based on actual experience from the work already performed to date and the planned development of the DFDX process.
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Sessions, John W., Brad W. Hanks, Tyler E. Lewis, Brian D. Jensen, Dallin L. Lindstrom, and Sandra H. Burnett. "Saline Solution Effects on Propidium Iodide Uptake in Nanoinjected HeLa Cells." In ASME 2014 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/detc2014-35431.

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Being able to deliver molecular loads to the intracellular space of mammalian cells is a key initial step of genetic engineering. In the following work, experimentation with nanoinjection, a non-viral molecular load delivery technique, was examined in regards to transmembrane delivery of propidium iodide (PI), a dye that cannot penetrate the cell membrane and fluoresces when bound to genetic material. Investigation includes two environmental factors: peak pulse amplitude (1.5 to 3, 5, 7, or 9 V) and saline type (HBSS, PBS with potassium, and PBS without potassium). Results indicate that PBS with potassium has significantly higher PI uptake efficiency than the other two saline solutions for pulsed voltages of 3V, 5V, and 7V (with the peak value being 3.352 times greater than the positive control). Also, cell viability analysis indicates that there is a measureable reduction in cell viability for voltage protocol samples in comparison to non-voltage protocol samples. Cell viabilities range from 74.5% to 89.4% for voltage protocol samples. Findings suggest that a possible combination of physical/electrical variables work in concert with biological mechanisms to contribute to overall cell survival and PI uptake efficiency in nanoinjection.
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Zhu, Jianjun, Xiaocheng Zhu, Yanru Zhang, Xiaole Qi, Zhenjiang Wang, Junjie Zhou, Minghao Li, Chaofan Jiang, and Chenyi Yi. "Surface Modification of Mesoporous TiO2 with Potassium ion Enhances Photo-Voltage in Perovskite Solar Cell." In 2022 6th International Conference on Power and Energy Engineering (ICPEE). IEEE, 2022. http://dx.doi.org/10.1109/icpee56418.2022.10050275.

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Johny, Joshly, and Sanju Sreedharan. "Efficiency of Potassium Permanganate and Potassium Dichromate as Oxidizing Agent in Microbial Fuel Cell." In Proceedings of the Advances in Technology, Engineering and Computing A Multinational Colloquium - 2017. Singapore: Research Publishing Services, 2017. http://dx.doi.org/10.3850/978-981-11-0744-3_c17-12.

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Atsumi, K., Y. Masaki, T. Noda, T. Hattori, H. Takao, M. Ishida, and K. Sawada. "2-dimensional potassium ion image sensor aiming for Label-free dynamic imaging of living-cell activity." In TRANSDUCERS 2009 - 2009 International Solid-State Sensors, Actuators and Microsystems Conference. IEEE, 2009. http://dx.doi.org/10.1109/sensor.2009.5285937.

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Shirshnev, Pavel, Alexey Romanov, Vladislav Bougrov, Elena Shirshneva-Vaschenko, and Zheneveva Snezhnaia. "Potassium-alumina-boron glass doped with copper ions for solar cell down-convertors." In Photonics for Solar Energy Systems, edited by Ralf B. Wehrspohn and Alexander N. Sprafke. SPIE, 2018. http://dx.doi.org/10.1117/12.2307398.

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Reznikov, Konstantin M., and Pavel D. Kolesnichenko. "THE EFFECT OF DRUGS ON THE THREE-DIMENSIONAL STRUCTURE OF CARDIOMYOCYTES." In International conference New technologies in medicine, biology, pharmacology and ecology (NT +M&Ec ' 2020). Institute of information technology, 2020. http://dx.doi.org/10.47501/978-5-6044060-0-7.24.

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In experimental myocardial infarction the occurrence of contracture of the sarcomeres, while giperkeratoza adjacent sections of the cell and movement of mitochondria. Drugs (korglikon, procainamide, potassium orotate) have different impacts on these processes.
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Reports on the topic "Potassium Ion Cells"

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Moran, Nava, Richard Crain, and Wolf-Dieter Reiter. Regulation by Light of Plant Potassium Uptake through K Channels: Biochemical, Physiological and Biophysical Study. United States Department of Agriculture, September 1995. http://dx.doi.org/10.32747/1995.7571356.bard.

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The swelling of plant motor cells is regulated by various signals with almost unknown mediators. One of the obligatory steps in the signaling cascade is the activation of K+-influx channels -K+ channels activated by hyperpolarization (KH channels). We thus explored the regulation of these channels in our model system, motor cell protoplasts from Samanea saman, using patch-clamp in the "whole cell" configuration. (a) The most novel finding was that the activity of KH channels in situ varied with the time of the day, in positive correlation with cell swelling: in Extensor cells KH channels were active in the earlier part of the day, while in Flexor cells only during the later part of the day; (b) High internal pH promoted the activity of these channels in Extensor cells, opposite to the behavior of the equivalent channels in guard cells, but in conformity with the predicted behavior of the putative KH channel, cloned from S. saman recently; (c) HIgh external K+ concentration increased (KH channel currents in Flexor cells. BL depolarized the Flexor cells, as detected in cell-attached patch-clamp recording, using KD channels (the K+-efflux channels) as "voltage-sensing devices". Subsequent Red-Light (RL) pulse followed by Darkness, hyperpolarized the cell. We attribute these changes to the inhibition of the H+-pump by BL and its reactivation by RL, as they were abolished by an H+-pump inhibitor. BL increased also the activity KD channels, in a voltage-independent manner - in all probability by an independent signaling pathway. Blue-Light (BL), which stimulates shrinking of Flexor cells, evoked the IP3 signaling cascade (detected directly by IP3 binding assay), known to mobilize cytosolic Ca2+. Nevertheless, cytosolic Ca2+ . did not activate the KD channel in excised, inside-out patches. In this study we established a close functional similarity of the KD channels between Flexor and Extensior cells. Thus the differences in their responses must stem from different links to signaling in both cell types.
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Melkoumian, Zaroui. Regulation of C-myc Gene Expression by Potassium Channel Blocker Quindine in MCF-7 Human Breast Cancer Cell Line. Fort Belvoir, VA: Defense Technical Information Center, July 2000. http://dx.doi.org/10.21236/ada384096.

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Minz, Dror, Stefan J. Green, Noa Sela, Yitzhak Hadar, Janet Jansson, and Steven Lindow. Soil and rhizosphere microbiome response to treated waste water irrigation. United States Department of Agriculture, January 2013. http://dx.doi.org/10.32747/2013.7598153.bard.

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Research objectives : Identify genetic potential and community structure of soil and rhizosphere microbial community structure as affected by treated wastewater (TWW) irrigation. This objective was achieved through the examination soil and rhizosphere microbial communities of plants irrigated with fresh water (FW) and TWW. Genomic DNA extracted from soil and rhizosphere samples (Minz laboratory) was processed for DNA-based shotgun metagenome sequencing (Green laboratory). High-throughput bioinformatics was performed to compare both taxonomic and functional gene (and pathway) differences between sample types (treatment and location). Identify metabolic pathways induced or repressed by TWW irrigation. To accomplish this objective, shotgun metatranscriptome (RNA-based) sequencing was performed. Expressed genes and pathways were compared to identify significantly differentially expressed features between rhizosphere communities of plants irrigated with FW and TWW. Identify microbial gene functions and pathways affected by TWW irrigation*. To accomplish this objective, we will perform a metaproteome comparison between rhizosphere communities of plants irrigated with FW and TWW and selected soil microbial activities. Integration and evaluation of microbial community function in relation to its structure and genetic potential, and to infer the in situ physiology and function of microbial communities in soil and rhizospere under FW and TWW irrigation regimes. This objective is ongoing due to the need for extensive bioinformatics analysis. As a result of the capabilities of the new PI, we have also been characterizing the transcriptome of the plant roots as affected by the TWW irrigation and comparing the function of the plants to that of the microbiome. *This original objective was not achieved in the course of this study due to technical issues, especially the need to replace the American PIs during the project. However, the fact we were able to analyze more than one plant system as a result of the abilities of the new American PI strengthened the power of the conclusions derived from studies for the 1ˢᵗ and 2ⁿᵈ objectives. Background: As the world population grows, more urban waste is discharged to the environment, and fresh water sources are being polluted. Developing and industrial countries are increasing the use of wastewater and treated wastewater (TWW) for agriculture practice, thus turning the waste product into a valuable resource. Wastewater supplies a year- round reliable source of nutrient-rich water. Despite continuing enhancements in TWW quality, TWW irrigation can still result in unexplained and undesirable effects on crops. In part, these undesirable effects may be attributed to, among other factors, to the effects of TWW on the plant microbiome. Previous studies, including our own, have presented the TWW effect on soil microbial activity and community composition. To the best of our knowledge, however, no comprehensive study yet has been conducted on the microbial population associated BARD Report - Project 4662 Page 2 of 16 BARD Report - Project 4662 Page 3 of 16 with plant roots irrigated with TWW – a critical information gap. In this work, we characterize the effect of TWW irrigation on root-associated microbial community structure and function by using the most innovative tools available in analyzing bacterial community- a combination of microbial marker gene amplicon sequencing, microbial shotunmetagenomics (DNA-based total community and gene content characterization), microbial metatranscriptomics (RNA-based total community and gene content characterization), and plant host transcriptome response. At the core of this research, a mesocosm experiment was conducted to study and characterize the effect of TWW irrigation on tomato and lettuce plants. A focus of this study was on the plant roots, their associated microbial communities, and on the functional activities of plant root-associated microbial communities. We have found that TWW irrigation changes both the soil and root microbial community composition, and that the shift in the plant root microbiome associated with different irrigation was as significant as the changes caused by the plant host or soil type. The change in microbial community structure was accompanied by changes in the microbial community-wide functional potential (i.e., gene content of the entire microbial community, as determined through shotgun metagenome sequencing). The relative abundance of many genes was significantly different in TWW irrigated root microbiome relative to FW-irrigated root microbial communities. For example, the relative abundance of genes encoding for transporters increased in TWW-irrigated roots increased relative to FW-irrigated roots. Similarly, the relative abundance of genes linked to potassium efflux, respiratory systems and nitrogen metabolism were elevated in TWW irrigated roots when compared to FW-irrigated roots. The increased relative abundance of denitrifying genes in TWW systems relative FW systems, suggests that TWW-irrigated roots are more anaerobic compare to FW irrigated root. These gene functional data are consistent with geochemical measurements made from these systems. Specifically, the TWW irrigated soils had higher pH, total organic compound (TOC), sodium, potassium and electric conductivity values in comparison to FW soils. Thus, the root microbiome genetic functional potential can be correlated with pH, TOC and EC values and these factors must take part in the shaping the root microbiome. The expressed functions, as found by the metatranscriptome analysis, revealed many genes that increase in TWW-irrigated plant root microbial population relative to those in the FW-irrigated plants. The most substantial (and significant) were sodium-proton antiporters and Na(+)-translocatingNADH-quinoneoxidoreductase (NQR). The latter protein uses the cell respiratory machinery to harness redox force and convert the energy for efflux of sodium. As the roots and their microbiomes are exposed to the same environmental conditions, it was previously hypothesized that understanding the soil and rhizospheremicrobiome response will shed light on natural processes in these niches. This study demonstrate how newly available tools can better define complex processes and their downstream consequences, such as irrigation with water from different qualities, and to identify primary cues sensed by the plant host irrigated with TWW. From an agricultural perspective, many common practices are complicated processes with many ‘moving parts’, and are hard to characterize and predict. Multiple edaphic and microbial factors are involved, and these can react to many environmental cues. These complex systems are in turn affected by plant growth and exudation, and associated features such as irrigation, fertilization and use of pesticides. However, the combination of shotgun metagenomics, microbial shotgun metatranscriptomics, plant transcriptomics, and physical measurement of soil characteristics provides a mechanism for integrating data from highly complex agricultural systems to eventually provide for plant physiological response prediction and monitoring. BARD Report
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