Relevant bibliographies by topics / Postural orthostatic tachicardia syndrome

Academic literature on the topic 'Postural orthostatic tachicardia syndrome'

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Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Postural orthostatic tachicardia syndrome"

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Cline, Holly M., and Adam Einhardt. "Postural orthostatic tachycardia syndrome." Nurse Practitioner 47, no. 1 (January 2022): 12–19. http://dx.doi.org/10.1097/01.npr.0000802968.90212.70.

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Agarwal, A. K., R. Garg, A. Ritch, and P. Sarkar. "Postural orthostatic tachycardia syndrome." Postgraduate Medical Journal 83, no. 981 (July 1, 2007): 478–80. http://dx.doi.org/10.1136/pgmj.2006.055046.

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Brooks, John K., and Laurie A. P. Francis. "Postural orthostatic tachycardia syndrome." Journal of the American Dental Association 137, no. 4 (April 2006): 488–93. http://dx.doi.org/10.14219/jada.archive.2006.0221.

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Grossman, Valerie G. A., and Becky A. McGowan. "Postural Orthostatic Tachycardia Syndrome." AJN, American Journal of Nursing 108, no. 8 (August 2008): 58–60. http://dx.doi.org/10.1097/01.naj.0000330266.83852.96.

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Bryarly, Meredith, Lauren T. Phillips, Qi Fu, Steven Vernino, and Benjamin D. Levine. "Postural Orthostatic Tachycardia Syndrome." Journal of the American College of Cardiology 73, no. 10 (March 2019): 1207–28. http://dx.doi.org/10.1016/j.jacc.2018.11.059.

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Busmer, Lorna. "Postural orthostatic tachycardia syndrome." Primary Health Care 21, no. 9 (November 2011): 16–20. http://dx.doi.org/10.7748/phc2011.11.21.9.16.c8794.

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Safavi-Naeini, Payam, and Mehdi Razavi. "Postural Orthostatic Tachycardia Syndrome." Texas Heart Institute Journal 47, no. 1 (February 1, 2020): 57–59. http://dx.doi.org/10.14503/thij-19-7060.

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Graham, U., and K. M. Ritchie. "Postural orthostatic tachycardia syndrome." Case Reports 2009, apr19 1 (April 19, 2009): bcr1020081132. http://dx.doi.org/10.1136/bcr.10.2008.1132.

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Czosek, Lauren. "Postural orthostatic tachycardia syndrome." Nursing 53, no. 2 (February 2023): 18–22. http://dx.doi.org/10.1097/01.nurse.0000905696.14880.53.

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Schondorf, Ronald, and Phillip A. Low. "Idiopathic postural orthostatic tachycardia syndrome." Neurology 43, no. 1 Part 1 (January 1993): 132. http://dx.doi.org/10.1212/wnl.43.1_part_1.132.

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Dissertations / Theses on the topic "Postural orthostatic tachicardia syndrome"

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CAIRO, BEATRICE. "ESTIMATING CARDIORESPIRATORY COUPLING FROM SPONTANEOUS VARIABILITY IN HEALTH AND PATHOLOGY." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/816612.

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Molteplici meccanismi sono responsabili delle interazioni cardiorespiratorie osservate nell’uomo. L’azione di questi meccanismi risulta in specifici ritmi di variabilità cardiaca (HRV) e ha effetti sulle interazioni tra attività cardiaca e respiratoria. I quattro principali tipi di fenomeni che derivano dalle interazioni tra cuore e sistema respiratorio sono: i) aritmia respiratoria sinusale (RSA); ii) accoppiamento cardioventilatorio; iii) sincronizzazione cardiorespiratoria in fase; iv) sincronizzazione cardiorespiratoria in frequenza. L’obiettivo di questa tesi è la descrizione e quantificazione di diversi aspetti delle interazioni cardiorespiratorie tramite l’utilizzo di una varietà di metodologie derivate dalla letteratura, adattate e ottimizzate per i tipici contesti sperimentali in cui HRV e segnale respiratorio sono comunemente acquisiti. Sei metodi analitici sono stati sfruttati a questo scopo per valutare l’entropia di trasferimento (TE), l’entropia cross-condizionata tramite entropia corretta cross-condizionata normalizzata (NCCCE), la coerenza quadratica (K2), l’accoppiamento cardioventilatorio tramite entropia normalizzata di Shannon (NSE) dell’intervallo temporale tra complesso QRS e inizio di fase inspiratoria o espiratoria, la sincronizzazione in fase tramite un indice di sincronizzazione (SYNC%) e il quoziente pulsazione-respirazione (PRQ). Questi approcci sono stati utilizzati con la finalità di testare gli effetti di uno stimolo simpatico, ovvero stimoli posturali quali l’head-up tilt (TILT) e l’ortostatismo attivo (STAND) sulle interazioni cardiorespiratorie. Gli approcci proposti sono stati testati in tre protocolli: i) atleti amatoriali sottoposti a un allenamento muscolare inspiratorio (IMT) durante clinostatismo supino (REST) e STAND; ii) volontari sani sottoposti a un decondizionamento da allettamento prolungato (HDBR), durante REST e TILT; iii) pazienti affetti da sindrome da tachicardia posturale ortostatica (POTS), durante REST e TILT, in condizione basale e durante approfondimento un anno dopo. I risultati principali della presente tesi di dottorato concernono l’effetto degli stimoli posturali sulle interazioni cardiorespiratorie in soggetti sani e patologici. Infatti, tutti gli indici proposti danno una visione coerente dell’intensità dell’interazione cardiorespiratoria in risposta a uno stimolo ortostatico, in quanto essa diminuisce in tutti i protocolli. Tuttavia, il potere statistico degli indici è differente. TE e K2 appaiono essere particolarmente deboli nell’identificare l’effetto dello stimolo posturale sulle interazioni cardiorespiratorie. NCCCE, NSE, e SYNC% dimostrano una capacità molto maggiore a tale riguardo, mentre PRQ appare troppo intimamente collegata alla frequenza cardiaca, in assenza di cambiamenti significativi della frequenza respiratoria. Per contro, tutti gli indici appaiono deboli nell’identificare gli effetti cronici di IMT e HDBR in una popolazione sana o le conseguenze croniche della gestione clinica in pazienti POTS. La tesi conclude che diversi aspetti delle interazioni cardiorespiratorie possono essere modificati in modo acuto ma gli effetti cronici di un trattamento o intervento a lungo termine sono irrisori sulla magnitudine delle interazioni cardiorespiratorie e/o possono essere confusi con la variabilità intrinseca degli indici. Considerazioni sulle differenze metodologiche e sull’efficacia degli indici proposti suggeriscono che un utilizzo simultaneo di molteplici metodi bivariati è vantaggiosa negli studi cardiorespiratori, in quanto diversi aspetti delle interazioni cardiorespiratorie possono essere valutati contemporaneamente. Questa valutazione simultanea può essere effettuata a un costo computazionale trascurabile e in contesti applicativi in cui il solo segnale ECG è disponibile.
Several mechanisms are responsible for cardiorespiratory interactions observed in humans. The action of these mechanisms results in specific patterns in heart rate variability (HRV) and affects the interaction between heart and respiratory activities. The four main types of phenomena resulting from the interactions between heart and respiratory system are: i) respiratory sinus arrhythmia (RSA); ii) cardioventilatory coupling; iii) cardiorespiratory phase synchronization; iv) cardiorespiratory frequency synchronization. The aim of this thesis is to describe and quantify different aspects of cardiorespiratory interactions employing a variety of methods from literature, adapted and optimized for the usual experimental settings in which HRV and respiratory signal are commonly acquired. Six analytical methods were exploited for this purpose assessing transfer entropy (TE), cross-conditional entropy via normalized corrected cross-conditional entropy (NCCCE), squared coherence (K2), cardioventilatory coupling via normalized Shannon entropy (NSE) of the time interval between QRS complex and inspiratory, or expiratory, onsets, phase synchronization via a synchronization index (SYNC%) and pulse-respiration quotient (PRQ). These approaches were employed with the goal of testing the effects of a sympathetic challenge, namely postural stimuli like head-up tilt (TILT) and active standing (STAND), on cardiorespiratory interactions. The proposed approaches were tested on three protocols: i) amateur athletes undergoing an inspiratory muscle training (IMT) during supine rest (REST) and STAND; ii) healthy volunteers undergoing a prolonged bed rest deconditioning (HDBR), during REST and TILT; iii) patients suffering from postural orthostatic tachycardia syndrome (POTS), during REST and TILT, at baseline and at one-year follow-up. The most important findings of the present doctoral thesis concern the effect of postural stimuli on cardiorespiratory interactions in health and disease. Indeed, all proposed indexes gave a coherent view of cardiorespiratory interaction strength in response to the orthostatic challenge, as it decreased in all protocols. However, the statistical power of the indexes was different. TE and K2 appeared to be particularly weak in detecting the effect of postural challenge on cardiorespiratory interactions. NCCCE, NSE and SYNC% exhibited much stronger ability in this regard, while PRQ seemed too closely related to heart rate, in presence of no significant modification of the respiratory rate. Conversely, all indexes appeared to be weak in detecting the chronic effects of IMT and HDBR on a healthy population and the long-term consequences of the clinical management in POTS patients. The thesis concludes that the different aspects of cardiorespiratory interactions can be modified acutely but the chronic effects of a long-term treatment or intervention on the magnitude of cardiorespiratory interactions are negligible and/or could be confused with the variability of markers. Considerations about the methodological dissimilarities and differences in effectiveness of the proposed indexes suggest that the simultaneous exploitation of all bivariate methodologies in cardiorespiratory studies is advantageous, as different aspects of cardiorespiratory interactions can be evaluated concurrently. This simultaneous evaluation can be carried out with a relatively negligible computational cost and in applicative contexts when only an ECG signal is available.
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Farchtchi, Masumeh Auguste. "Parent-Child Dyadic Experiences Living with Postural Orthostatic Tachycardia Syndrome (POTS) during Emerging Adulthood." Thesis, Virginia Tech, 2020. http://hdl.handle.net/10919/98006.

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Chronic illness and invisible disability are impactful contexts during emerging adulthood and the launching stage of the family life cycle (Beatty, 2011; Capelle, Visser, and Vosman, 2016; Young et al., 2010). The parent-child relationship is important to both developmental and health outcomes in families coping with chronic illness during emerging adulthood (Crandell, Sandelowski, Leeman, Haville, and Knafle, 2018; Fenton, Ferries, Ko, Javalkar, and Hooper, 2015; Waldboth, Patch, Mahrer-Imhaf, and Metcalfe, 2016). While informed clinical competency in counseling families experiencing disablement is a diversity-affirmative ethical imperative among psychotherapists (Mona et al., 2017), little is known in family therapy about how parents and emerging adult children experience launching with chronic illness. This qualitative study explored the parent-child dyadic experience of living with a chronic illness called Postural Orthostatic Tachycardia Syndrome (POTS) during emerging adulthood. Seven dyads of parents and their emerging adult children with POTS were interviewed. Data analysis of in-depth interviews using Moustakas's (1994) transcendental phenomenology uncovered eight thematic clusters of meaning in the shared lived experience of POTS at the launching stage of the family life cycle. Clinical implications for family therapists were explored using Rolland's family system-illness (FSI) model of medical family therapy. Study limitations and future directions for further research were discussed.
Master of Science
More and more young adults are living with chronic illness. Postural Orthostatic Tachycardia Syndrome (POTS) is a little-known chronic illness that tends to begin during adolescence. Like many health problems that disproportionately affect women, POTS is often overlooked by doctors. POTS symptoms, such as dizziness and cognitive difficulty, impact a person's ability to engage in preferred activities and identities. Family therapists can play an impactful role in supporting parents and children with POTS through developmental tasks related to launching an emerging adult in the context of this complex and widely misunderstood chronic illness. This thesis presented the first qualitative study of parent-child dyadic experiences living with POTS. Clinical implications for medical family therapy were highlighted. To construct an interview framework, Rolland's Family Systems-Illness (FSI) clinical model for helping families cope with illness and disability was used in conjunction with Arnett's description of emerging adulthood as a developmental stage in life. Seven parent-child dyads were interviewed for 1-2 hours in fourteen separate interviews generating transcripts about 140,000 words long in total. Analysis of these interviews identified shared themes composing the essence of the parent-child experience living with POTS during emerging adulthood. Results were described through tables and narratives. Clinical implications for family therapists working with parents and children with POTS during emerging adulthood were proposed. Limitations and ideas for future studies were discussed.
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Tyrrell, Genevieve. "Zora." Master's thesis, University of Central Florida, 2013. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/5723.

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This mixed-media memoir uses a variety of forms from short epigrammatic essays to straightforward stories and graphic narratives to explore the author's coming-of-age experiences augmented by chronic illness. Trying to succeed in the film industry, romance, and family situations, the young female narrator navigates the often unexpected or disappointing consequences of having an autonomic nervous system disorder. Relationships between conflicting identities emerge—between healthy versus sick self, projected/envisioned versus actual self, and tough versus vulnerable self—as the narrator journeys toward a more complete and accepting self-understanding.
M.F.A.
Masters
English
Arts and Humanities
Creative Writing
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Su, Che-wei, and 蘇哲維. "Quantitative Analysis of Cerebral Blood Flow and Standing Balance in Postural Orthostatic Tachycardia Syndrome." Thesis, 2014. http://ndltd.ncl.edu.tw/handle/24304409855469347050.

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碩士
逢甲大學
資訊電機工程碩士在職專班
102
Postural orthostatic tachycardia syndrome (POTS) is characterized by orthostatic tachycardia in the absence of orthostatic hypotension. Patients with POTS may experience dizziness, lightheadedness and instability while standing. The main purpose of this study is to evaluate the clinical significance of standing balance and cerebral blood flow (CBF) response during tilting in POTS with dizziness. A group of 18 subjects: 9 POTS patients who all experienced dizziness and instability, and 9 age and gender-matched healthy volunteers participated in the experiment. Pressure balance (Center of pressure, CoP) and CBF data from Transcranial Doppler (TCD) for 300 seconds were collected and analyzed between supine and standing (or tilting). There was a significant difference in anterior-posterior swing between POTS patients and healthy subjects during standing (CoP =338.4 ± 113.6 vs. 185.6 ± 38.5 units, p=0.003). The study shows that dizziness and instability in POTS while standing may be caused by the increase of anterior-posterior swing and CBF drop. Further research is needed in this area, particularly concerning the mechanisms of interaction between vestibular and autonomic nervous system regarding pressure balance and CBF regulation.
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Malik, Varun. "Autonomic Function in Atrial Fibrillation." Thesis, 2021. https://hdl.handle.net/2440/135971.

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At the population level, the prevalence of Atrial fibrillation (AF) is escalating. AF burden also increases in individuals, due its self-perpetuating, progressive nature. While AF itself has been implicated in its self-maintenance, mechanisms responsible are not fully determined. The efferent arm of the Autonomic nervous system (ANS) alters atrial electrophysiology, triggering AF. At the population-level, imbalances of autonomic tone are prospectively associated with AF. Whilst it is well known that cardiovascular risk factors promote AF, how they interact is uncertain, though there is a potential role of the ANS. AF itself, causes autonomic dysfunction, altering atrial innervation and heightening efferent sympathetic activity. Thus, a bidirectional relationship is likely between AF and the ANS. Whilst considerable effort was placed on the efferent arm of the ANS, the role of the afferent, regulatory arm in AF pathophysiology is unknown. Especially cardiovascular reflexes regulating blood pressure and volume. Interestingly, blood-volume regulating (low-pressure) baroreceptors are co-located in pulmonary vein-atrial tissue, which contain drivers for AF. This thesis undertakes a series of studies that delineate the role of the afferent arm of the ANS in AF. Using a systematic review and meta-analysis, we demonstrate that AF is independently associated with falls and syncope in older adults. AF can cause orthostatic intolerance (a clinical manifestation of autonomic dysfunction). Next, we examined low-pressure baroreceptor function in paroxysmal or persistent AF patients, studied in sinus rhythm (SR), identifying novel blood volume regulating reflex abnormalities using Low-level Lower Body Negative Pressure (LBNP). Then, we performed a series of autonomic reflex tests to delineate the function of lowpressure volume regulating (LBNP), high-pressure blood pressure regulating baroreceptors (Valsalva) and both (Isometric handgrip reflex, IHR). Testing cardiovascular reflexes in the presence of AF is challenging, however, we utilised continuous beat-beat haemodynamic monitoring and venous occlusion plethysmography to evaluate these in patients with AF studied in-AF, and in SR. We demonstrated dysfunction of the volume-regulating LBNP reflex in-AF, for the first time and confirmed diminished LBNP reflexes in-SR. IHR was diminished in-AF, however blood pressure baroreflexes were preserved. In subsequent studies, we proceeded to show that cardioversion improved LBNP reflex abnormality, as did LLTS (low-level vagal nerve stimulation) a novel neuromodulating technique (LLTS), aiding localisation to the afferent level. Further, we tested whether catheter ablation of AF might further destroy such afferent receptors in pulmonary veins, not having found evidence for this. Given loss of homeostasis (blood-volume dysregulation) in-AF, we propose an intersect between autonomic and anatomic remodelling (atrial dilatation), possibly representing a mechanism behind AF progression. In the last study, we tested the interplay between anatomic and autonomic remodelling in Postural tachycardia syndrome (POTS), a known dysautonomia, where the heart is thought normal. We present new evidence of cardiac remodelling, utilising electrocardiogram markers (also associated with AF). AF has several sequelae; falls, syncope, dementia, and heart failure, notwithstanding, its own progression, all of which, increase morbidity and all-cause mortality. We open a new scientific avenue to explore mechanisms for these and highlight the role of rhythm management and neuromodulation to treat AF.
Thesis (Ph.D.) -- University of Adelaide, Adelaide Medical School, 2022
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Books on the topic "Postural orthostatic tachicardia syndrome"

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Klotzbach, Faustino. How to Get Rid of Postural Orthostatic Tachycardia Syndrome : Treatment Options: Postural Orthostatic Tachycardia Syndrome Sweatshirt. Independently Published, 2021.

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Welch, Sam. My Battle with Postural Orthostatic Tachycardia Syndrome (Pots). Independently Published, 2018.

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Turner, Neil. Postural proteinuria (benign orthostatic proteinuria). Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0051.

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Postural proteinuria, synonymous with the condition known as benign orthostatic proteinuria, describes increased levels of protein excretion associated with normalization first thing in the morning. It is usually diagnosed in children, for whom it is the most common explanation for proteinuria picked up incidentally on dipstick testing. In children, it generally resolves with age and is thought to have a benign long-term prognosis, with the caveat that numbers with very long follow-up times are few. It is also seen in teenagers but becomes much less common in early adulthood. Its aetiology is not well understood, although patients with pathological causes for proteinuria and patients with physiological levels of total protein excretion have been shown to exhibit similar diurnal variation. Using currently published limits for daily protein excretion the diagnosis is common. Some examples have been attributed to nutcracker syndrome (compression of the left renal vein), although that is more commonly associated with macroscopic haematuria, and the association remains uncertain. The condition is best diagnosed by comparing first-in-morning urine samples paired with afternoon samples on several occasions. In childhood, if proteinuria levels are in the normal range in morning samples, and within moderately increased limits later in the day, probably no investigation beyond observation is required. Most will resolve; very few will evolve into serious renal disease. The simplest mode of long-term monitoring is to measure protein:creatinine or albumin:creatinine ratios in first-in-morning urine samples.
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MIA, Oliver. Postural Orthostatic Tachycardia Syndrome: All That You Need to Know. Independently Published, 2020.

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TWAM, S. M. R. Pot Journal: Postural Orthostatic Tachycardia Syndrome , 120 Pages Beautiful Journal for Postural Orthostatic Tachycardia Syndrome Management with Stress and Energy Trackers, POTS Symptom and More. Independently Published, 2020.

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PERO, Peris. Postural Orthostatic Tachycardia Syndrome : Lost: Treatment Guide from Diagnosis till Complete Recovery. Independently Published, 2022.

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Power Over POTS: A Family Guide to Managing Postural Orthostatic Tachycardia Syndrome. Bookbaby, 2018.

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Neary, John, and Neil Turner. Nutcracker syndrome and phenomenon. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0048.

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Nutcracker syndrome describes symptomatology associated with obstruction to the left renal vein caused by pressure from the overlying superior mesenteric artery. Modern imaging methods show that some degree of left renal vein obstruction may be a common incidental finding in asymptomatic patients so it is better described as ‘nutcracker phenomenon’, NCP. The association of NCP with symptoms and signs is often speculative. NCP may be seen at any age but most patients with symptoms attributed to it are teenagers or young adults. The strongest evidence is for association with episodic macroscopic haematuria. There is weak evidence that it may in some circumstances account for orthostatic (postural) proteinuria, microscopic haematuria, or pain syndromes. Apart from rare examples of extreme haemorrhage the syndrome has not been associated with life-threatening features other than through complications of treatment. Various interventions have been employed, recently most commonly endovascular or extravascular approaches to stenting the vein, but serious adverse consequences from stent migration and thrombosis have been described.
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Higgins, Jade. Taking Control over POTS: All You Need to Know about Postural Orthostatic Tachycardia Syndrome and How to Defeat It for Person's under 15-50, Seniors Handbook. Independently Published, 2022.

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Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Falls and funny turns. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0005.

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This chapter provides information on falls and fallers, assessment following a fall, interventions to prevent falls, syncope and presyncope, balance and disequilibrium, dizziness, drop attacks, orthostatic (postural) hypotension, situational hypotension, carotid sinus syndrome, and falls services.
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Book chapters on the topic "Postural orthostatic tachicardia syndrome"

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Brignole, Michele, and David G. Benditt. "Orthostatic Intolerance: Orthostatic Hypotension and Postural Orthostatic Tachycardia Syndrome." In Syncope, 179–97. London: Springer London, 2010. http://dx.doi.org/10.1007/978-0-85729-201-8_12.

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Bourne, Kate M., Matthew G. Lloyd, and Satish R. Raj. "Diagnostic Criteria for Postural Tachycardia Syndrome: Consideration of the Clinical Features Differentiating PoTS from Other Disorders of Orthostatic Intolerance." In Postural Tachycardia Syndrome, 19–28. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-54165-1_4.

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Gaudiani, Jennifer L. "Postural Orthostatic Tachycardia Syndrome (POTS) and Associated Conditions." In Sick Enough, 161–71. New York, NY : Routledge, 2019.: Routledge, 2018. http://dx.doi.org/10.4324/9781351184731-17.

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Gonzalez-Hermosillo, A., M. F. Marquez, A. Kostine, K. Jauregui-Renaud, and M. Cardenas. "Vasovagal Syncope, Orthostatic Hypotension and Postural Orthostatic Tachycardia Syndrome: Is There a Connection?" In Cardiac Arrhythmias 2003, 615–24. Milano: Springer Milan, 2004. http://dx.doi.org/10.1007/978-88-470-2137-2_81.

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Fedorowski, Artur, Viktor Hamrefors, and Fabrizio Ricci. "Other Syndromes of Orthostatic Intolerance: Delayed Orthostatic Hypotension, Postprandial Hypotension, Postural Orthostatic Tachycardia Syndrome, and Reflex Syncope." In Orthostatic Hypotension in Older Adults, 121–43. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-62493-4_13.

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Grubb, Blair P., and Beverly Karabin. "Postural Orthostatic Tachycardia Syndrome." In Cardiac Electrophysiology: From Cell to Bedside, 990–95. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-44733-1.00104-8.

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Grubb, Blair P., and Beverly Karabin. "Postural Orthostatic Tachycardia Syndrome." In Cardiac Electrophysiology: From Cell to Bedside, 1029–34. Elsevier, 2014. http://dx.doi.org/10.1016/b978-1-4557-2856-5.00104-7.

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Low, Phillip A. "Postural orthostatic tachycardia syndrome." In Autonomic Failure, 747–53. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780198566342.003.0063.

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Low, P. A. "Postural Orthostatic Tachycardia Syndrome (POTS)." In Encyclopedia of the Neurological Sciences, 964–67. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-12-385157-4.00509-1.

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Low, Phillip A. "Postural Orthostatic Tachycardia Syndrome (POTS)." In Encyclopedia of the Neurological Sciences, 48–53. Elsevier, 2003. http://dx.doi.org/10.1016/b0-12-226870-9/00229-x.

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Conference papers on the topic "Postural orthostatic tachicardia syndrome"

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Nair, Pramod, Swati Gupta, Niha Peshimam, and Rajesh Sesham. "1274 Severe POTS (Postural Orthostatic Tachycardia Syndrome)." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Liverpool, 28–30 June 2022. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2022. http://dx.doi.org/10.1136/archdischild-2022-rcpch.690.

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Oza, F., T. Tariq, N. Saqib, and P. Ochieng. "Case of Postural Orthostatic Tachycardia Syndrome with Oxygen Desaturation." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1937.

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Nair, Pramod, Toby MacCarthy, and Nisrien Eltag Mohamed Osman. "1473 Outcome of adolescent onset POTS (Postural orthostatic tachycardia syndrome)." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 15 June 2021–17 June 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-rcpch.668.

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Shirek, Gabrielle P., Clio Pitula, David W. Kaplan, Kaci Pickett, Alexandra Schwartz, Jamie Shoop, Ryan Phillips, Rachel Workman, and Steven L. Moulton. "An Interdisciplinary Investigation of Postural Orthostatic Tachycardia Syndrome and Associated Symptoms." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.399.

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Naz, A., S. Johal, and P. Nair. "G13 Tilt positive pots (postural orthostatic tachycardia syndrome): a case series study." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 25 September 2020–13 November 2020. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2020. http://dx.doi.org/10.1136/archdischild-2020-rcpch.5.

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Lin, Shyan-Lung, Shoou-Jeng Yeh, Ching-Kun Chen, Chih-En Kuo, and Yu-Liang Hsu. "Cerebrovascular Response to Carbon Dioxide during Hyperventilation for Patients with Postural Orthostatic Tachycardia Syndrome." In 2020 IEEE Eurasia Conference on IOT, Communication and Engineering (ECICE). IEEE, 2020. http://dx.doi.org/10.1109/ecice50847.2020.9301915.

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Cairo, Beatrice, Beatrice De Maria, Vlasta Bari, Francesca Gelpi, Maura Minonzio, Franca Barbic, Laura Adelaide Dalla Vecchia, Raffaello Furlan, and Alberto Porta. "Causal Analysis Is Needed to Evaluate Cardiorespiratory Interaction Alterations in Postural Orthostatic Tachycardia Syndrome Patients." In 2021 Computing in Cardiology (CinC). IEEE, 2021. http://dx.doi.org/10.23919/cinc53138.2021.9662712.

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Shahi, Niti, Gabrielle P. Shirek, Kaci Pickett, Alexandra Schwartz, Jamie Shoop, Ryan Phillips, Rachel Workman, Clio Pitula, David W. Kaplan, and Steven L. Moulton. "Re-examining Physiologic Diagnostic Criteria: A Pilot Study using the Compensatory Reserve Index to Evaluate Individuals with Postural Orthostatic Tachycardia Syndrome." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.362-a.

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Duvall, Julia, Rachael Granberry, Lucy E. Dunne, Brad Holschuh, Christopher Johnson, Kevin Kelly, Bruce Johnson, and Michael Joyner. "The Design and Development of Active Compression Garments for Orthostatic Intolerance." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3480.

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Abstract:
Current compression garments are often made from a spandex-type elastic material with static levels of compression and can become uncomfortable and difficult to don/doff [1]. This limits their usability, especially for unhealthy or aging populations. The only current alternative to elastic compression stockings are inflatable compression sleeves that are controllable, but highly immobile and must be tethered to an inflation source [2]. Neither design offers a solution that is simultaneously low profile, mobile, and controllable. Here we present the design and development of compression garments with embedded shape-changing materials that can produce controllable compression without the need for a bulky inflation system. This active materials approach enables dynamic control over the degree, timing and location of compression, and allows for graded, synchronized, pulsed, and peristaltic compression patterns, which provide the medical benefit of moving fluid in the body [2]. Such a design combines the best features of both elastic and inflatable compression garments: a slim, low-profile form factor that is easy to don/doff and provides dynamic control. Shape memory alloy (SMA) coil actuators, as described by Holschuh et al., [3] have the ability to apply compressive forces to the body when paired with passive textiles and wrapped circumferentially around the body. These actuators are engineered to contract when heated, creating controllable forces and displacements that are modulated through an applied current. SMA compression garments (SMA-CG) have important applications, from consumer uses to clinical interventions, including: augmenting venous return for conditions of orthostatic intolerance (e.g., postural orthostatic tachycardia syndrome (POTS)); cardiac rehabilitation in heart failure patients; lymphedema venous insufficiency; reducing deep vein thrombosis (DVT) risk; sports performance; and countermeasures for flight or space flight. While the potential uses for this technology are broad, the basic design is similar across many conditions. Key research areas include: 1) identifying and addressing design considerations relevant to prototype development of SMA-CG; 2) determining the compression thresholds needed to dynamically oppose orthostatic changes; and 3) evaluating the effectiveness of the prototypes for augmented venous return by synchronizing compression during cardiac diastole. Here, we focus on the first question: design of SMA-CG prototypes.
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Granberry, Rachael, Nicole Ciavarella, Robert Pettys-Baker, Mary Ellen Berglund, and Brad Holschuh. "No-Power-Required, Touch-Activated Compression Garments for the Treatment of POTS." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6886.

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Abstract:
Postural orthostatic tachycardia syndrome (POTS) is a clinical autonomic disorder characterized by a spike in heart rate and syncope in response to orthostasis, symptoms which then dissipate upon recumbency [1]. Additional symptoms include chronic fatigue, bloating, and nausea [2]. POTS predominately affects females (5:1) between the ages of 15 and 50 years [3]. It is estimated 1 to 3 million are affected by POTS in the United States [4]. While there is no known cure, symptom management requires a multifaceted approach, including physical exercise, counter maneuvers, high salt and fluid intake, and medications, including beta blockers and fludrocortisone [3]. Lower body compression is a core component to POTS treatment, especially during prolonged periods of upright posture. Because POTS disproportionately affects young, otherwise healthy females who have high physical and professional demands, compression garments (CG) are critical to allow this population to carry out their activities of daily living [5].
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