Academic literature on the topic 'Postoperative ileus'

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Journal articles on the topic "Postoperative ileus"

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Bowker, Brennan, Rebecca Orsulak Calabrese, and Emily Barber. "Postoperative Ileus." Physician Assistant Clinics 6, no. 2 (April 2021): 215–27. http://dx.doi.org/10.1016/j.cpha.2020.12.001.

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Holte, Kathrine, and Henrik Kehlet. "Postoperative Ileus." Drugs 62, no. 18 (2002): 2603–15. http://dx.doi.org/10.2165/00003495-200262180-00004.

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RA, Sastry. "Postoperative ileus." Journal of Medical and Scientific Research 1, no. 3 (September 1, 2013): 135–44. http://dx.doi.org/10.17727/jmsr.2013/1-023.

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Harnsberger, Cristina, Justin Maykel, and Karim Alavi. "Postoperative Ileus." Clinics in Colon and Rectal Surgery 32, no. 03 (April 2, 2019): 166–70. http://dx.doi.org/10.1055/s-0038-1677003.

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AbstractPostoperative ileus (POI) is a common complication following colon and rectal surgery, with reported incidence ranging from 10 to 30%. It can lead to increased morbidity, cost, and length of stay. Although definitions vary considerably in the literature, in its pathologic form, it can be characterized by a temporary inhibition of gastrointestinal motility after surgical intervention due to nonmechanical causes that prevents sufficient oral intake. Various risk factors for development of POI have been identified including increasing age, American Society of Anesthesiologists scores 3 to 4, open approach, operative difficulty, operative duration more than 3 hours, bowel handling, drop in hematocrit or need for a transfusion, increasing crystalloid administration, and delayed mobilization. While treatment is expectant and supportive, significant investigations into strategies to mitigate development of POI or shorten its duration have been undertaken with mixed results. There is significant evidence to suggest that a minimally invasive approach and multimodal pain regimens reduce the development of POI. The beneficial effect of chewing gum, alvimopan, and enhanced recovery after surgery protocols may decrease development of POI in selected groups of patients who undergo elective colorectal surgery, and shorten time to return of bowel function, but overall, the data remain inconclusive.
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Kehlet, H. "Postoperative ileus." Gut 47, no. 90004 (December 1, 2000): 85iv—86. http://dx.doi.org/10.1136/gut.47.suppl_4.iv85.

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Nunley, James C., and Gregory P. FitzHarris. "Postoperative ileus." Current Surgery 61, no. 4 (July 2004): 341–45. http://dx.doi.org/10.1016/j.cursur.2003.07.008.

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Livingston, Edward H., and Edward P. Passaro. "Postoperative ileus." Digestive Diseases and Sciences 35, no. 1 (January 1990): 121–32. http://dx.doi.org/10.1007/bf01537233.

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Nestorovic, Milica, Goran Stanojevic, Branko Brankovic, Vanja Pecic, and Ljiljana Jeremic. "Prolonged postoperative ileus after elective colorectal cancer surgery." Vojnosanitetski pregled 75, no. 8 (2018): 780–86. http://dx.doi.org/10.2298/vsp160527387n.

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Background/Aim. Postoperative ileus is a frequent and frustrating occurence for both, patients and surgeons after abdominal surgery. Besides clinical importance of postoperative ileus, its economic aspect is also important. The aim of this prospective study was to analyze development of prolonged postoperative ileus after elective colorectal surgery for cancer and its impact on early postoperative outcome. Methods. This prospective study included all eligible patients, 18 years or older, scheduled for open colorectal resection for cancer from June, 2015 to February, 2016. Patients with metastatic disease, prior hemoirradiation or any resection other then curative were excluded. The study duration was up to 30 days postoperatively. Primary outcome measure was development of prolonged postoperative ileus according to strict definition. The impact of prolonged postoperative ileus on other outcome measures such as postoperative complications, surgical site infections, anastomotic leakage, reoperations, mortality and length of hospital stay were of great interest, too. Results. This prospective study included 103 patients, 64 (37.9%) men and 39 (62.1%) women, mean age 66 years. Prolonged postoperative ileus developed in 12 (11.3%) patients. One third of the patients had some type of surgical site infection, while 47.6% had complications. Ten (9.7%) patients required reoperation. Comparing the group of patients with prolonged postoperative ileus with those without, there were no statistically significant differences in rates of surgical site infection and anastomotic leakage. There was statistically significant difference in terms of complications (_2 = 34.966; p < 0.001), complications grade III (_2 = 23.43; p < 0.001) and reoperations (_2 = 15.724; p <0.001). Patients who developed prolonged postoperative ileus had statistically significant longer postoperative hospital stay (Z = 2.291, p = 0.022) and longer total length of hospital stay (Z = 2.377, p = 0.015). According to regression analyzes prolonged postoperative ileus represents a risk factor for reoperations [odds ratio (OR) = 12.286; p = 0.001]. Conclusion. Prolonged postoperative ileus, although not life-threatening complication effects recovery, increases length of hospital stay and contributes to poor surgical outcome.
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Prasad, Madhu, and Jeffrey B. Matthews. "Deflating postoperative ileus." Gastroenterology 117, no. 2 (August 1999): 489–92. http://dx.doi.org/10.1053/gast.1999.0029900489.

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Alban, Joseph, and M. Kay Garcia. "Prolonged Postoperative Ileus." Medical Acupuncture 20, no. 4 (December 2008): 291–92. http://dx.doi.org/10.1089/acu.2008.701.

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Dissertations / Theses on the topic "Postoperative ileus"

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Liss, Britta, and Josefina Wahlbeck. "Sjuksköterskans omvårdnadsåtgärder vid förebyggande av postoperativ ileus." Thesis, Högskolan i Gävle, Avdelningen för hälso- och vårdvetenskap, 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:hig:diva-26457.

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Sammanfattning Bakgrund: Postoperativ ileus är en allvarlig komplikation som kan uppstå, ofta till följd av smärtstillande läkemedel eller inflammation i tarmvävnaden. Tarmmotoriken avstannar och gas- och tarmavgång upphör. Symtomen är kraftiga buksmärtor, kräkningar och utspänd buk. Patienten utsätts för smärta, oro och förlängd sjukhusvistelse. Syfte: Att beskriva omvårdnadsåtgärder sjuksköterskan kan vidta för att förebygga postoperativ ileus samt att beskriva urvalsgrupperna i de inkluderade artiklarna. Metod: En litteraturstudie av deskriptiv design som grundas på elva vetenskapliga artiklar, samtliga med kvantitativ ansats. Huvudresultat: Omvårdnadsåtgärder som tidigt oralt vätske- och födointag, tugga tuggummi, mobilisering, gunga gungstol, akupressur och sjuksköterskeledd information har visats påskynda läkning av tarmen och reducera risken av postoperativ ileus. Omvårdnadsåtgärderna visade bäst resultat gällande tarmljud, tarmrörelser, gas- och tarmavgång och tolerans av normal föda. Slutsats: Omvårdnadsåtgärder har visats ha god effekt vid förebyggande av postoperativ ileus. Tydliga tecken på att tarmen återhämtat sig och att den postoperativa tarmparalysen upphört är gas- och tarmavgång, tarmljud och tarmrörelser, vilket samtliga påskyndas av omvårdnadsåtgärder. Att kontinuerligt ta del av kunskap genom forskning möjliggör för sjuksköterskan att utföra professionell omvårdnad av hög standard. Det är sjuksköterskans ansvar att utföra omvårdnadsåtgärder som förebygger risker, minskar lidande och främjar hälsa.
Abstract Background: Postoperative ileus is a serious complication, often due to pain relief medication or the inflammation of intestinal tissue. Bowel movements stop and the gastrointestinal tract ceases and the resulting symptoms are: severe abdominal pain, vomiting and tensioned abdomen. This result in pain, anxiety and long-term hospitalization for the patient. Aim: To describe nursing interventions which prevent postoperative ileus and describe the selection groups of included articles. Method: A literature study of descriptive design based on eleven articles, all of which take a quantitative approach. Main result: Nursing interventions such as; early oral fluid and food intake, chewing gum, mobilization, swing rocking chair, acupressure and nurse information have been shown to accelerate healing of the intestine and reduce the risk of postoperative ileus. Nursing interventions had best result relating to: bowel sound, bowel movement, defecation and tolerance of normal food. Conclusion: Nursing interventions have been shown effective in the prevention of postoperative ileus. Clear indications of intestinal recovery and postoperative paralysis recovery include first flatus, bowel sound, bowel movement and defecation, all of which are more likely to occur if the patient has had nursing intervention. Continuously acquiring knowledge through research enables nurses to perform professional care of high standard. It is the nurse's responsibility to carry out nursing interventions that prevent risks, reduce suffering and promote healing.
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Apelqvist, Josefin, and Jennifer Dahlin. "Postoperativt ileus : En kartläggning av interventioner inom svensk kirurgisk vård." Thesis, Linköpings universitet, Avdelningen för omvårdnad, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-157052.

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Bakgrund: Efter kirurgiska ingrepp förekommer postoperativt ileus (POI) som ett normalfysiologiskt tillstånd. POI definieras som ett avvikande mönster av gastrointestinal motilitet med karakteristiska symtom som illamående och kräkningar, uppspänd buk, samt utebliven gasavgång eller avföring. POI har påvisats påverka den postoperativa återhämtningen negativt. Syfte: Syftet med studien var att kartlägga interventioner som används för att minska durationen av postoperativt ileus vid svenska kirurgiska vårdavdelningar. Design: Enkätbaserad tvärsnittsstudie. Metod: Internetbaserad enkät skickades till vårdenhetschefer vid 86 kirurgiskt inriktade vårdavdelningar på fem universitetssjukhus i södra och mellersta Sverige. Resultat: Totalt 21 kirurgiska vårdavdelningar besvarade enkäten. Kunskaper kring innebörden och handläggningen av POI angavs som måttliga till goda. Den mest frekvent föreslagna interventionen inom omvårdnad var mobilisering och laxantia i olika former var den vanligaste farmakologiska interventionen. Konklusion: Omvårdnads- och farmakologiska interventioner av varierande evidensgrad används på svenska kirurgiska vårdavdelningar. Det tycks finnas ett behov av utbildning om och implementering av vetenskapliga kunskaper inom området för bedömning och handläggning av POI.
Background: Postoperative ileus (POI) occurs as a normal reaction to all forms of surgery. POI is defined as a deviant pattern of gastrointestinal motility with characteristic symptoms such as nausea and vomiting, abdominal distension and lack of gas or stool. Postoperative ileus has been shown to affect the postoperative recovery in a negative way. Aim: The aim of this study was to investigate the current use of interventions aimed to reduce the duration time of postoperative ileus in Swedish surgical wards. Design: A cross-sectional study. Method: A web-based survey was administered to matrons at 86 surgical wards in five university hospitals in the mid- and south of Sweden. Results: In total, 21 wards responded. The knowledge about the meaning and management of POI was perceived as moderate to good. The most frequent used nursing intervention was mobilization, and the most common pharmacological action proposed was laxatives of various sorts. Conclusion: Both nursing and pharmacological interventions with various grades of evidence are used in Swedish surgical wards. There are indications of a need for education and implementation of science-based knowledge within the area of assessing and managing POI.
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Olsson, Emma, and Ina Rasmusson. "Sjuksköterskors omvårdnadsåtgärder för främjandet av den gastrointestinala motiliteten i det postoperativa skedet : En litteraturstudie." Thesis, Högskolan i Halmstad, Akademin för hälsa och välfärd, 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:hh:diva-39319.

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Dysfunktion av den gastrointestinala motiliteten under det postoperativa skedet är en komplikation som kan orsakas till följd av kirurgiska ingrepp. Detta kan orsaka bukspänning, illamående samt smärta som komplicerar en ofullständig och snabb återhämtning efter kirurgiska ingrepp. Dysfunktion av den gastrointestinala motiliteten ökar risken för ytterligare postoperativa komplikationer som uttorkning, elektrolytrubbning och sepsis. Samt ökar riskerna för reoperation och återintagning på sjukhus efter utskrivning. Dysfunktion av den gastrointestinala motiliteten kan ge ökat lidande för patienter, skapa längre vårdtider, medföra högre sjukhuskostnader samt öka arbetsbelastningen för hälso- och sjukvårdspersonal. Syfte: Syftet var att beskriva sjuksköterskors omvårdnadsåtgärder vid dysfunktion av den gastrointestinala motiliteten hos patienter under det postoperativa skedet. Metod: Studien genomfördes som en allmän litteraturstudie med induktiv ansats. Resultat: Resultatet baserades på nio vetenskapliga artiklar med kvantitativa ansats. Utifrån kodning av meningsenheter från de vetenskapliga artiklarna framkom fyra huvudkategorier: Patientnära samtal, Mobilisering, Kost- och vätskeintag och Stimulering av den gastrointestinala motiliteten. Slutsats: Omvårdnadsåtgärder var en viktig del i förebyggandet för främjandet av den gastrointestinala motiliteten under det postoperativa skedet samt förbättrade möjligheten till ett tidigare tillfrisknande för patienter. Ett tidigt ingripande med omvårdnadsåtgärder till patienter som genomgått kirurgi bidrog det till minskat lidande, kortare vårdtider för patienterna samt minskad arbetsbelastning för hälso- och sjukvårdspersonal.
Dysfunction of the gastrointestinal motility in the post-operative stage is a complication that can be caused by surgical procedures. This can cause abdominal tension, nausea and pain that complicate an incomplete and rapid recovery after surgery. Dysfunction of gastrointestinal motility increases the risk of additional postoperative complications such as dehydration, electrolyte disruption and sepsis. As well as increasing the risk of reoperation and readmission in hospitals after discharge. Dysfunction of the gastrointestinal motility can lead to increased suffering for patients, create longer care times, bring higher hospital costs and increase the workload of healthcare professionals. Purpose: The purpose was to describe nursing care measures in dysfunction of the gastrointestinal motility to patients in the postoperative stage. Method: The study was conducted as a literature review with inductive approach. Result: The result was based on nine scientific articles with quantitative approaches. Based on the coding of opinion units from the scientific articles, four main categories emerged: Patient-related conversation, Mobilization, Diet and fluid intake and Stimulation of gastrointestinal motility. Conclusion: Nursing care measures were an important part of the prevention of the promotion of gastrointestinal motility in the postoperative stage, and improved the possibility of an earlier recovery for patients. Early intervention with nursing care measures for patients who have undergone surgery, it contributed to reduced suffering, shorter patient care times and reduced workload for healthcare professionals.
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Klumpp, Anna-Maria. "Der Einfluß des vasoaktiven intestinalen Peptids auf den postoperativen Ileus bei der Ratte." [S.l. : s.n.], 2005. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB11878708.

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Sedin, John. "Prevention of Postoperative Duodenal Ileus by COX-2 Inhibition Improves Duodenal Function in Anaesthetised Rats." Doctoral thesis, Uppsala universitet, Fysiologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-198049.

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Abdominal surgery inhibits gastrointestinal motility, a phenomenon referred to as postoperative ileus. Since the postoperative ileus disturbs duodenal physiology it is important to minimize the side effects of this condition. Recent experiments in our laboratory show that treatment of anaesthetised rats with parecoxib, a selective cyclooxygenase-2 inhibitor, prevents duodenal postoperative ileus, increases duodenal mucosal bicarbonate secretion and improves other functions as well. One aim of the thesis was to investigate whether removal of luminal chloride affect the parecoxib- and the vasoactive intestinal peptide (VIP)-induced stimulation of duodenal mucosal bicarbonate secretion. The proximal duodenum of anaesthetised Dark Agouti rats was perfused with isotonic solutions containing zero or low Cl- and the effect on luminal alkalinisation determined. The basal as well as the parecoxib-induced increase in alkalinisation, but not that stimulated by VIP, were markedly reduced in the absence of luminal Cl-. One important function of the duodenum is to adjust luminal osmolality towards that in the blood. It is believed that the adjustment of osmolality in the duodenum is achieved by osmosis and diffusion of electrolytes along their concentration gradients and that these processes occur predominately paracellularly. Another aim of the thesis was to examine whether prevention of postoperative ileus affects the duodenal response to luminal hypertonicity. The proximal duodenum of anaesthetised Dark Agouti and Sprague-Dawley rats were perfused with hypertonic solutions of different composition and osmolality and the effects on duodenal motility, alkaline secretion, transepithelial fluid flux, mucosal permeability and the adjustment of luminal osmolality were determined in absence and presence of parecoxib. It is concluded that COX-2 inhibition increases duodenal mucosal bicarbonate secretion by stimulating apical Cl-/HCO3- exchange in duodenocytes. Furthermore, pretreatment of anaesthetised rats with parecoxib improves a number of duodenal functions in both rat strains that contribute to improve the ability to adjust luminal osmolality. The choice of rat strain is another important feature to consider when interpreting the results because the DA strain was more responsive to luminal hypertonicity than the SD strain. Finally, several evidences are provided to suggest that the adjustment of luminal osmolality in the rat duodenum is a regulated process.
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Пак, Василь Якович, Василий Яковлевич Пак, Vasyl Yakovych Pak, С. Я. Пак, and Ю. В. Юрченко. "Рання післяопераційна спайкова непрохідність кишок." Thesis, Сумський державний університет, 2015. http://essuir.sumdu.edu.ua/handle/123456789/41975.

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Отримані результати дають лише загальну уяву про РПСНК, але дозволяють зробити висновки про те, що це захворювання призводить до глибоких порушень гомеостазу на фоні виснаження захисних сил організму. Виникають пневмонії, причиною яких є застій і аспірація внаслідок тривалого ліжкового режиму і трансназальної інтубації тонкої кишки. Гнійно- запальні процеси у післяопераційній рані та черевній порожнині виникають внаслідок порушень гомеостазу і склеювання петель кишечника.
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Berdún, Marin Sergio. "Implication of mast cells, nerve growth factor and splanchnic nerves in postoperative ileus. Study in patients undergoing abdominal surgery and in a rat experimental model." Doctoral thesis, Universitat Autònoma de Barcelona, 2015. http://hdl.handle.net/10803/298178.

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El íleo postoperatorio (IPO) se define como el cese temporal de la motilidad propulsiva gastrointestinal (GI) en pacientes sometidos a cirugía intestinal. En pacientes y en modelos experimentales de IPO, se ha observado degranulación de mastocitos (MCs) en lavado peritoneal. Estudios con fármacos estabilizadores de MCs o en modelos en roedores carentes de mastocitos apoyan la participación de los MCs en el IPO. No obstante, los mecanismos involucrados aún están por definir. La interacción nervio-mastocito, así como la interacción con el factor de crecimiento nervioso (NGF), podrían representar un factor clave en el desarrollo del IPO. El objetivo del presente trabajo ha sido estudiar el IPO humano caracterizando la activación de los mastocitos peritoneales (PMCs) durante la cirugía colorectal. A parte, también nos propusimos explorar el papel del NGF sobre los MCs y la interacción nervio-mastocito en un modelo experimental de IPO en rata. Primero se estudió el IPO humano. Evaluamos la liberación de proteasas mastocitarias (mediante la técnica de ELISA) en el lavado peritoneal de pacientes sometidos a cirugía colorectal por laparoscopia y laparotomía, así como también la densidad de MCs (por inmunohistoquímica) y la recuperación clínica postoperatoria. Seguidamente se puso a punto un modelo de IPO en rata para profundizar en el papel de los MCs. En él, evaluamos el efecto del antagonista del receptor del NGF, K252a. Previamente habíamos demostrado el efecto in vitro del K252a sobre PMCs. Asimismo, hemos evaluado la participación de los ganglios de la raíz dorsal (DRG) en el IPO además de evaluar las consecuencias funcionales de la activación mastocitaria en animales tratados con el estabilizador de mastocitos ketotifeno o mediante la exposición del intestino al degranulador de mastocitos compuesto 48/80. Los resultados de este trabajo demuestran que la cirugía colorectal induce la liberación de proteasas mastocitarias en la cavidad peritoneal en aquellos pacientes que desarrollaron un IPO. En la rata, inmediatamente tras la inducción del modelo, se produjo un aumento en la liberación de la proteasa mastocitaria RMCP-6 (inmunoensayo) en la cavidad peritoneal. A las 24h, se observó un retraso en el tránsito GI y un aumento en la expresión de la interleuquina-6 (IL-6, RT-qPCR) y en la actividad mieloperoxidasa (MPO) en las muestras de íleon. El K252a inhibió la degranulación in vitro de los PMCs (valorada mediante el ensayo de la β-hexosaminidasa) así como también atenuó la liberación de RMCP-6 y la expresión génica tanto de IL-6, como de RMCP-2 y TrkA (receptor del NGF) en el modelo. Sin embargo, no mejoró la motilidad GI. Por otro lado, la expresión de mediadores calcitonin gene-related peptide (CGRP), NGF, TrkA and protease-activated receptor-2 en los DRGs aumentó tras la inducción del modelo. Estos cambios no se vieron afectados tras el tratamiento con ketotifeno o la exposición a C48/80. En cambio, el C48/80 si produjo un retraso en el vaciamiento gástrico/tránsito intestinal así como también aumentó la expresión de IL-6 y actividad MPO. El uso de ketotifeno, previno el retraso en el vaciamiento gástrico y la disminución post-operatoria del output fecal. Este trabajo nos permite concluir que los MCs participan en el IPO y que la interacción entre el NGF, TrkA y PMCs puede representar una diana para el tratamiento del IPO u otras alteraciones mediadas por mastocitos.
Postoperative ileus (POI) is defined as a temporal cessation of propulsive gastrointestinal (GI) motility in patients undergoing abdominal surgery, especially those subjected to intestinal resection. Mast cell (MC) degranulation has been reported in patients undergoing abdominal surgery and in experimental models of POI. MC stabilizers or the use of MC-deficient mutant models point towards a role for MCs in POI, but the exact mechanisms involved remains unclear. Interactions between MCs and nerve endings or inflammatory mediators such as the nerve growth factor (NGF) may represent a key factor in POI pathogenesis. The aim of this work was to study POI in human patients by characterizing the activation of peritoneal mast cells (PMCs) in colorectal surgery. A part from that, we also aimed to explore the role of NGF antagonists on MCs and MC-nerve interactions in a rat model of POI. We firstly studied human POI. We evaluated MC protease release in peritoneal lavage (by means of ELISA) collected from patients undergoing laparoscopic and open colorectal surgery. In these patients we also studied MC density in colonic samples (by immunohistochemistry) and postoperative clinical recovery. Secondly, we set up a model of POI in rat to further explore the role of MCs. We evaluated the effect of pre-treatment with NGF receptor antagonist, K252a. In vitro effects of K252a were also evaluated on rat PMCs. Activation of dorsal root ganglia (DRG) in POI rat model was also characterized in a separate experiment in which the role of MCs was investigated using ketotifen (MC stabilizer) and compound 48/80 (C48/80, MC degranulator). Our study demonstrated release of MC proteases into peritoneal cavity after colorectal surgery. This protease release was observed only in patients with a subsequent delay of clinical recovery (those who developed POI). In our animal study we demonstrated that induction of POI by intestinal manipulation immediately evokes release of MC protease RMCP-6 in the peritoneal cavity. At 24h after intestinal manipulation, our model also presented delayed GI transit and increased expression of interleukin-6 (IL-6, RT-qPCR) and myeloperoxidase activity (MPO) in ileum samples. In contrast, density of intestinal MCs (immunohistochemistry and toluidine blue staining) and RMCP-2 and 6 gene expressions (RT-qPCR) in the ileum were unaltered in the animal POI model. K252a prevented PMC degranulation in vitro (β-hexosaminidase assay) and the release of RMCP-6 in the POI model. In addition, K252a attenuated IL-6 expression after intestinal manipulation and decreased basal peritoneal release of RMCP-2 and TrkA (NGF receptor) gene expression However, GI transit was not ameliorated after K252a treatment. Intestinal manipulation in the POI model also increased gene expression of calcitonin gene-related peptide, NGF, TrkA and protease-activated receptor-2 in somas of DRGs but these changes were not modulated by ketotifen or C48/80. In contrast, C48/80 did delay GI transit and induced up-regulation of IL-6 and MPO activity. Ketotifen also prevented delayed gastric emptying and the postoperative decrease of fecal output. To sum up, our results indicate that intestinal manipulation is associated with a local response of MCs in the peritoneal cavity. Intestinal manipulation delayed GI motility in vivo, induced intestinal inflammation and activated DRGs. K252a stabilized MCs and down-regulated IL-6 expression in the inflammatory response leading to POI. Our data also showed that MCs are involved in GI motility alteration and inflammation after intestinal manipulation. In contrast, activation of DRGs seems to be independent of MC activation based on our assessment using a pharmacological approach. We conclude that MCs participate in POI and that interactions between NGF, TrkA and PMCs may represent a target for treatment of POI or other MC-mediated diseases.
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Guschlbauer, Maria [Verfasser]. "Influence of Lidocaine on the Equine Small Intestine Contractile Function after an Ischaemia and Reperfusion Injury: Effects and Mechanisms – Therapy of the Postoperative Paralytic Ileus in the Horse / Maria Guschlbauer." Hannover : Bibliothek der Tierärztlichen Hochschule Hannover, 2010. http://d-nb.info/100960385X/34.

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Kuppinger, David. "Parenterale Aminosäurenzufuhr und postoperative Ileum-Proteinsynthese." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-122016.

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Stein, Kathy [Verfasser]. "Zelluläre und molekulare Mechanismen der Entzündungsauflösung im postoperativen Ileus / Kathy Stein." Bonn : Universitäts- und Landesbibliothek Bonn, 2015. http://d-nb.info/1080864520/34.

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Books on the topic "Postoperative ileus"

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Delaney, C. P. Postoperative ileus. Oxford: Oxford University Press, 2010.

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Gan, Tj. Oxford American Pocket Notes Postoperative Ileus. Oxford University Press, 2010.

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Book chapters on the topic "Postoperative ileus"

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Demars, Sandra M. "Postoperative Ileus." In The Perioperative Medicine Consult Handbook, 279–85. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09366-6_47.

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Demars, Sandra. "Postoperative Ileus." In The Perioperative Medicine Consult Handbook, 423–32. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-19704-9_54.

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Sokol-Hessner, Lauge. "Postoperative Ileus." In The Perioperative Medicine Consult Handbook, 251–55. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3220-3_39.

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Wacha, H. "Postoperative Mechanical Ileus." In Die Chirurgie und ihre Spezialgebiete Eine Symbiose, 154–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-95662-1_77.

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Elyash, Igor G. "Management of Postoperative Ileus." In Clinical Algorithms in General Surgery, 197–98. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98497-1_51.

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Hübner, Martin, Michael Scott, and Bradley Champagne. "Postoperative Ileus: Prevention and Treatment." In The SAGES / ERAS® Society Manual of Enhanced Recovery Programs for Gastrointestinal Surgery, 133–46. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20364-5_12.

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Schein, Moshe. "Postoperative Ileus Versus Intestinal Obstruction." In Schein’s Common Sense Emergency Abdominal Surgery, 297–302. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-88133-6_34.

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Adiamah, Alfred, and Dileep N. Lobo. "Postoperative Ileus: Prevention and Treatment." In Enhanced Recovery After Surgery, 249–57. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-33443-7_27.

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Schein, Moshe, and Sai Sajja. "Postoperative Ileus Versus Intestinal Obstruction." In Schein's Common Sense Emergency Abdominal Surgery, 501–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-74821-2_48.

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Schmidt-Matthiesen, H. "Prevention and Treatment of Postoperative Ileus." In Gynecology and Obstetrics, 617–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70559-5_212.

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Conference papers on the topic "Postoperative ileus"

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Reyhani, H., E. Macaninch, K. Martyn, and E. Copeland. "5 Postoperative ileus and perioperative nutrition optimisation in an enhanced recovery after surgery programme." In Oral Presentations and Abstracts from the 6th International Summit on Medical and Public Health Nutrition Education and Research, September 2020. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/bmjnph-2022-nnedprosummit.12.

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Hartmann, L., N. Hering, and M. Kreis. "Auswirkung von selektiver Darmdekontamination (SDD) und Darmlavage auf den postoperativen Ileus (POI) in der Maus." In Viszeralmedizin 2017. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1605154.

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Reports on the topic "Postoperative ileus"

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Ulsh, Brooke, Delaney Vedros, Rebekah Whaley, and Sharon Little. Intraoperative Administration Of An NSAID And An Opioid Vs. An Opioid Alone Effect On Postoperative Ileus Development. University of Tennessee Health Science Center, July 2021. http://dx.doi.org/10.21007/con.dnp.2021.0009.

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Abstract:
The purpose of this DNP project is to examine what is known from existing literature about postoperative ileus development in the adult surgical patient population after receiving intraoperative opioids versus a combination of intraoperative opioids and NSAIDs.
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Zhang, Zhe, Jigang Ren, Jinlian Guo, Lin An, Shaung Li, Zichen Zhang, Yan Chen, Hui Liu, and Xiao Lei. The use of coffee to decrease the Incidence of Postoperative Ileus: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0095.

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Yu, Min, Xianyu Yang, Chengwen Liao, Huihua Zheng, Renqing Kuang, and Fan Xie. The impact of acupuncture on postoperative ileus following colorectal cancer surgery: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2021. http://dx.doi.org/10.37766/inplasy2021.8.0021.

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