Academic literature on the topic 'Porphyromonas gingivalis lipopolysaccharide'
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Journal articles on the topic "Porphyromonas gingivalis lipopolysaccharide"
Jain, Sumita, and Richard P. Darveau. "Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis." Periodontology 2000 54, no. 1 (August 16, 2010): 53–70. http://dx.doi.org/10.1111/j.1600-0757.2009.00333.x.
Full textAl-Qutub, Montaser N., Pamela H. Braham, Lisa M. Karimi-Naser, Xinyan Liu, Caroline A. Genco, and Richard P. Darveau. "Hemin-Dependent Modulation of the Lipid A Structure of Porphyromonas gingivalis Lipopolysaccharide." Infection and Immunity 74, no. 8 (August 2006): 4474–85. http://dx.doi.org/10.1128/iai.01924-05.
Full textHan, Su-Ji, So-Yeon Jeong, Yun-Ju Nam, Kyu-Ho Yang, Hoi-Soon Lim, and Jin Chung. "Xylitol Inhibits Inflammatory Cytokine Expression Induced by Lipopolysaccharide from Porphyromonas gingivalis." Clinical Diagnostic Laboratory Immunology 12, no. 11 (November 2005): 1285–91. http://dx.doi.org/10.1128/cdli.12.11.1285-1291.2005.
Full textTada, Hiroyuki, Shunji Sugawara, Eiji Nemoto, Nobuhiro Takahashi, Takahisa Imamura, Jan Potempa, James Travis, Hidetoshi Shimauchi, and Haruhiko Takada. "Proteolysis of CD14 on Human Gingival Fibroblasts by Arginine-Specific Cysteine Proteinases from Porphyromonas gingivalis Leading to Down-Regulation of Lipopolysaccharide-Induced Interleukin-8 Production." Infection and Immunity 70, no. 6 (June 2002): 3304–7. http://dx.doi.org/10.1128/iai.70.6.3304-3307.2002.
Full textKnoernschild, Kent L., Geoffrey R. Tompkins, Carol A. Lefebvre, and George S. Schuster. "Porphyromonas gingivalis lipopolysaccharide affinity for two casting alloys." Journal of Prosthetic Dentistry 74, no. 1 (July 1995): 33–38. http://dx.doi.org/10.1016/s0022-3913(05)80225-2.
Full textFerraz, Caio Cezar Randi, Michael A. Henry, Kenneth M. Hargreaves, and Anibal Diogenes. "Lipopolysaccharide From Porphyromonas gingivalis Sensitizes Capsaicin-Sensitive Nociceptors." Journal of Endodontics 37, no. 1 (January 2011): 45–48. http://dx.doi.org/10.1016/j.joen.2007.07.001.
Full textKhan, Junad, Bollama Puchimada, Daniel Kadouri, Tali Zusman, Fawad Javed, and Eli Eliav. "The anti-nociceptive effects of Porphyromonas gingivalis lipopolysaccharide." Archives of Oral Biology 102 (June 2019): 193–98. http://dx.doi.org/10.1016/j.archoralbio.2019.04.012.
Full textBozkurt, S. Buket, Sema S. Hakki, Erdogan E. Hakki, Yusuf Durak, and Alpdogan Kantarci. "Porphyromonas gingivalis Lipopolysaccharide Induces a Pro-inflammatory Human Gingival Fibroblast Phenotype." Inflammation 40, no. 1 (November 3, 2016): 144–53. http://dx.doi.org/10.1007/s10753-016-0463-7.
Full textRangarajan, Minnie, Joseph Aduse-Opoku, Nikolay Paramonov, Ahmed Hashim, Nagihan Bostanci, Owen P. Fraser, Edward Tarelli, and Michael A. Curtis. "Identification of a Second Lipopolysaccharide in Porphyromonas gingivalis W50." Journal of Bacteriology 190, no. 8 (February 8, 2008): 2920–32. http://dx.doi.org/10.1128/jb.01868-07.
Full textBachtiar, Endang Winiati, Citra F. Putri, Retno D. Soejoedono, and Boy M. Bachtiar. "Expression of TNF, IL1B, and NOS2 in the neural cell after induced by Porphyromonas gingivalis with and without coating antibody anti-Porphyromonas gingivalis." F1000Research 9 (March 17, 2021): 1499. http://dx.doi.org/10.12688/f1000research.26749.2.
Full textDissertations / Theses on the topic "Porphyromonas gingivalis lipopolysaccharide"
Al-Qutub, Montaser Nazmi. "Lipopolysaccharide lipid A structural heterogeneity of Porphyromonas gingivalis /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/6383.
Full textHuck, Olivier. "Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose." Thesis, Strasbourg, 2013. http://www.theses.fr/2013STRAJ021.
Full textPeriodontal diseases have been linked to systemic diseases especially cardiovascular diseases and atherosclerosis. In our study, we investigated the effects induced by an infection with Porphyromonas gingivalis, a major periodontal pathogen, and stimulation by its lipopolysaccharide on endothelial cells at the interface between the inner part of arteries and blood flow. We focused on the effects induced on cathepsin B, a protease involved in atherosclerosis and on the activation of inflammasome, an intracellular complex linked to secretion of IL-1beta. Results showed that infection with Porphyromonas gingivalis and stimulation by its lipopolysaccharide increase enzymatic activity of cathepsin B with different kinetics. These modifications are observed without any modifications of RNAm expression and protein concentration. We also showed that infection with Porphyromonas gingivalis increases RNAm expression of NLRP3 but this increase at the RNAm level is not associated with an increase of the protein concentration due to an induced proteolysis. Furthermore, we developed a reliable model of experimental periodontitis that will be used to analyze interactions between periodontitis and systemic diseases in vivo, especially in apolipoprotein-E -/- mice
Bainbridge, Brian W. "Generation and function of Porphyromonas gingivalis lipid A heterogeneity /." Thesis, Connect to this title online; UW restricted, 2007. http://hdl.handle.net/1773/6394.
Full textZhou, Lili [Verfasser]. "Porphyromonas gingivalis lipopolysaccharides affect gingival stem/progenitor cells attributes through NF-κB, but not Wnt/β-catenin pathway / Lili Zhou." Kiel : Universitätsbibliothek Kiel, 2018. http://d-nb.info/1173703454/34.
Full textHerath, Mudiyanselage Thanuja Darshani Kumari Herath. "A systems biological study on heterogeneous Porphyromonas gingivalis lipopolysaccharides: human gingivalfibroblasts interaction : molecular mechanisms and implications inperiodontal pathogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hub.hku.hk/bib/B50162615.
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Dentistry
Doctoral
Doctor of Philosophy
Dixon, Douglas Raymond. "Lipid A heterogeneity within Porphyromonas gingivalis and other oral bacteria : effect of lipid A content on hTLR4 utilization and E-selectin expression /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/6385.
Full textGross, Jane Elizabeth. "Local and systemic host immune responses to Porphyromonas gingivalis A7436 infection in a subcutaneous tissue chamber in untreated and lipopolysaccharide-treated mice." 1999. http://catalog.hathitrust.org/api/volumes/oclc/48199463.html.
Full textLan-YunChang and 張藍云. "Effects of Recombinant Thrombomodulin Domain 1 in Porphyromonas gingivalis Lipopolysaccharide-Induced Osteoclastogenesis." Thesis, 2019. http://ndltd.ncl.edu.tw/handle/8q83xt.
Full textHuang, Kun-Che, and 黃堃哲. "HBD2-Overexpression decreases BMSC Proinflammatory Cytokine Expression of BMSC after Porphyromonas gingivalis Lipopolysaccharide Stimulation." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/2ghv7x.
Full text國立陽明大學
口腔生物研究所
106
Periodontitis is a common oral inflammatory disease caused by bacterial infection which leads to destruction of periodontal tissues. Successful periodontal regenerative therapy requires reduction of bacteria-related pathogenic factors and promotion of tissue regeneration. Porphyromonas gingivalis (Pg) is a major pathogen of periodontitis. Pg lipopolysaccharide (LPS) induces the production of proinflammatory cytokines, such as interleukin (IL)-1β (IL-1β), IL-6, and IL-8, and plays an important role in Pg’s pathogenic mechanism. Human β-defensin 2 (hBD2) is an important antimicrobial peptide of innate immune system. Previous studies indicated that hBD2 is effective against Pg. The positive charge of hBD2 potentially can neutralize the negative-charged LPS. Stem cell therapy is an effective treatment strategy for regenerative therapies including the regeneration of periodontal defects. Application of hBD2-overexpressing bone marrow stem cell (hBD2/BMSC) in treatment of bacteria-contaminated bone defect shows excellent antimicrobial and bone regenerative effects. Thus, hBD2/BMSC shows great potential in periodontal regenerative therapies. It has been reported that LPS induces production of proinflammatory cytokines in bone marrow stem cells. However, the effects of Pg-LPS on hBD2/BMSC may be different. We hypothesized that hBD2 produced by hBD2/BMSC can neutralize Pg-LPS and reduce the expression of proinflammatory cytokines from cells stimulated by Pg-LPS. The purpose of this study was to determine the Pg-LPS-induced proinflammatory cytokine expression of hBD2/BMSC. Human bone marrow stem cells (3A6) overexpressing red fluorescent protein (RFP) or hBD2, namely RFP/3A6 and hBD2/3A6, were produced by lentiviral infection method. The red fluorescence of RFP/3A6 was verified under fluorescent microscope. The secreted hBD2 from hBD2/3A6 was quantified by enzyme-linked immunosorbent assay (ELISA). Various concentrations of Pg-LPS (0, 0.1, 1 μg/ml) were used to treat RFP/3A6 and hBD2/3A6. The induced expression of proinflammatory cytokines (IL-1β、IL-6, and IL-8) of RFP/3A6 and hBD2/3A6 was determined by quantitative PCR and compared. The results indicated that the secretion of hBD2 can be increased through repeated lentiviral infection strategy. Pg-LPS stimulation dose-dependently increase the expression of proinflammatory cytokines (IL-1β、IL-6, and IL-8) of RFP/3A6 and hBD2/3A6. The trend of expression increase is significantly smaller in hBD2/3A6 when compared with RFP/3A6. When treated with Pg-LPS (0.1 or 1 μg/ml)),hBD2/3A6 showed lower expression of proinflammatory cytokines (IL-1β、IL-6, and IL-8) than RFP/3A6. In conclusion, the overexpression of hBD2 of BMSCs can lower the Pg-LPS-induced expression of proinflammatory cytokines. The phenomenon may reduce the local inflammation during periodontal regenerative therapy and benefit the periodontal regeneration
Tsai, Man-chin, and 蔡嫚今. "Diallyl sulfide diminished the Porphyromonas gingivalis lipopolysaccharide stimulated pro-inflammatory cytokine expression and NF-κB activation in gingival fibroblasts." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/16465942363879007682.
Full text國防醫學院
牙醫科學研究所
100
Introduction: Periodontitis is a chronic infection and inflammatory disease, which is a multi-factorial disease, risks for moderate to severe periodontitis that have been identified include specific pathogenic bacteria (such as Porphyromonas gingivalis), environment, behavior, and certain other systemic conditions. Studies showed that the fibroblasts actively participate in immune and inflammatory events in periodontal diseases. Future strategies for the prevention and treatment of periodontal diseases should biologically regulate fibroblast activities. Garlic is a common flavor food since ancient times. Garlic is composed with a variety of sulfur-based compounds. The most important components of garlic are allicin and scordinin which has a strong ability of oxide-reduction, anti-bacterial, anti-inflammatory, anti-cancer, and improving the immune system. Purpose of this study is to examine the role of garlic extract in reducing periodontal inflammation by using the gingival fibroblast cultures and the animal models of experimental periodontitis. Materials and Methods: In this study, we examined the anti-inflammatory effects of diallyl sulfide on P. g LPS-induced inflammation in human gingival fibroblasts. The cytotoxicity effects of diallyl sulfide on the cell was measured by MTS assay with/without LPS stimulation. The effects of diallyl sulfide on LPS-stimulated the mRNA expressions of TNF-α, IL-1β, and IL-6 were investigated by RT-PCR. The inhibitory effects of diallyl sulfide on NF-κB activation, TNF-α and IL-1β expression were examined by immunocytochemical staining (ICC). Results: In this study, we demonstrated that diallyl sulfide in the concentration of 0, 1, 2, 3 mM combined with LPS (1 g/ml) did not affect HGFs’ survival. The LPS-stimulated mRNA expressions of TNF-α, IL-1β, and IL-6 in HGFs were inhibited when pretreated with diallyl sulfide. The LPS-stimulated NF-κB activation and protein expressions of TNF-α, IL-1β in HGFs were repressed when pretreated with diallyl sulfide. Conclusion: In this study, we found and demonstrated that diallyl sulfide could reduce the periodontal inflammation in the gingival fibroblast cultures.
Books on the topic "Porphyromonas gingivalis lipopolysaccharide"
Andreou, Vana. Resveratrol mediated reversal of aryl hydrocarbon and porphyromonas gingivalis lipopolysaccharide induced inhibition of osteogenesis in vitro. 2002.
Find full textBook chapters on the topic "Porphyromonas gingivalis lipopolysaccharide"
Ishikawa, Taichi, Yuko Ohara-Nemoto, Shihoko Tajika, Minoru Sasaki, and Shigenobu Kimura. "The production of secretory leukocyte protease inhibitor from gingival epithelial cells in response to Porphyromonas gingivalis lipopolysaccharides." In Interface Oral Health Science 2009, 275–76. Tokyo: Springer Japan, 2010. http://dx.doi.org/10.1007/978-4-431-99644-6_77.
Full textPritchard, Anna Barlach, Zsolt Fabian, Clare L. Lawrence, Glyn Morton, StJohn Crean, and Jane E. Alder. "An Investigation into the Effects of Outer Membrane Vesicles and Lipopolysaccharide of Porphyromonas gingivalis on Blood-Brain Barrier Integrity, Permeability, and Disruption of Scaffolding Proteins in a Human in vitro Model." In Advances in Alzheimer’s Disease. IOS Press, 2022. http://dx.doi.org/10.3233/aiad220044.
Full textQian, Xueshen, Shuang Zhang, Lian Duan, Fengchun Yang, Kun Zhang, Fuhua Yan, and Song Ge. "Periodontitis Deteriorates Cognitive Function and Impairs Neurons and Glia in a Mouse Model of Alzheimer’s Disease." In Advances in Alzheimer’s Disease. IOS Press, 2022. http://dx.doi.org/10.3233/aiad220042.
Full textCornelia Nelwan, Sindy, Ricardo Adrian Nugraha, Anang Endaryanto, Frisma Dewi, Yonna Dwi Swastika, Udijanto Tedjosasongko, and Seno Pradopo. "Correlation between Salivary Lipopolysaccharide of Porphyromonas gingivalis with Circulatory Immunoglobulin-E and Immunoglobulin-G4 in Periodontally Healthy Children with House Dust Mite Allergy." In Gingival Disease - A Professional Approach for Treatment and Prevention. IntechOpen, 2019. http://dx.doi.org/10.5772/intechopen.81113.
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