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Books on the topic 'Porphyra'

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Published: 4 June 2021
Last updated: 1 February 2022

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1

Chrysafis, Andreas C. Porphyra in Purple. UK: Evandia Publishing (UK)Ltd, 2008.

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2

Porphyra gelia: Mythistorēma. Athēna: Ekdoseis Patakē, 2010.

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3

Lazaropoulos, Panagiōtēs D. Hē porphyra tou deilinou. Athēna: Ekdoseis Parousia, 1999.

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4

Vasilikē, Kokkinou, ed. Ē porphyra kai ē elia. Athēna: Psychogios, 1999.

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5

Henas skouphos apo Porphyra: Mythistorēma. Athēna: Kedros, 1995.

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6

author, Sahoo Dinabandhu joint, ed. Porphyra: Harvesting gold from the sea. New Delhi: I.K. International Pub. House, 2010.

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7

Sepheriadou, Anthoula S. Symvolismos kai poiēsē gia to Lampro Porphyra. Thessalonikē: Ekdoseis Kōdikas, 1989.

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8

Sepheriadou, Anthoula S. Symvolismos kai poiēsē gia to Lampro Porphyra. Thessalonikē: Ekdoseis Kōdikas, 1989.

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9

Sepheriadou, Anthoula S. Symvolismos kai poiēsē gia to Lampro Porphyra. Thessalonikē: Ekdoseis Kōdikas, 1989.

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10

Mikami, Koji, and Koji Mikami. Porphyra yezoensis: Frontiers in physiological and molecular biological research. Hauppauge, N.Y: Nova Science Publisher's, 2010.

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11

Mikami, Koji. Porphyra yezoensis: Frontiers in physiological and molecular biological research. Hauppauge, N.Y: Nova Science Publisher's, 2010.

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12

Porphyry. Porphyry Introduction. Oxford: Oxford University Press, 2003.

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13

Porphyry: Red imperial porphyry : power and religion. Torino: U. Allemandi & C., 2012.

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14

Parker, Philip M., and James M. Parker. Porphyria: A bibliography and dictionary for physicians, patients, and genome researchers. Edited by Icon Group International Inc. San Diego, California: ICON Health Publications, 2007.

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15

Aristotle's Categories and Porphyry. Leiden: E.J. Brill, 1988.

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16

Berchman, Robert M. Porphyry against the Christians. Leiden: Brill, 2005.

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17

Porphyry. Select works of Porphyry. Frome, Somerset, UK: Prometheus Trust, 1994.

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18

Hadot, Pierre. Plotin, Porphyre: Études néoplatoniciennes. Paris: Belles lettres, 1999.

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19

Ahmad, Mahmood U. Porphyry copper in Pakistan. Quetta: Geological Survey of Pakistan, 1992.

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20

Duke, Stephen O., and Constantin A. Rebeiz, eds. Porphyric Pesticides. Washington, DC: American Chemical Society, 1994. http://dx.doi.org/10.1021/bk-1994-0559.

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21

Saffrey, H. D. (Henri Dominique), editor, translator, Segonds, A. Ph, editor, translator, and Iamblichus approximately 250-approximately 330, eds. Réponse à Porphyre: (De mysteriis). Paris: Les Belles lettres, 2013.

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22

Porphyre et le moyen-platonisme. Paris: Vrin, 2002.

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23

Huant, Ernest. Reflechissons, Porphyre!: Conte philosophique d'anticipation. Hamburg: Maurice Moureau Verlag, 1986.

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24

Parnotte, Joe l. Le sang des Porphyre: Hermine. [Bruxelles]: Dargaud Be ne lux, 2010.

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25

Beneath a sky of porphyry. London: Quartet, 1990.

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26

Stegen, Ralph J., Robert Cal Callaghan, Robert Cal Callaghan, and Ralph J. Stegen, eds. Porphyry Molybdenum Deposits in Colorado I. Climax Porphyry Molybdenum Deposits II. Henderson Molybdenum Mine. Tulsa, Oklahoma, U.S.A.: SEPM Society for Sedimentary Geology, 2006. http://dx.doi.org/10.5382/gb.38.

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27

Porphyry. Porphyrii philosophi fragmenta. Stutgardiae: Teubner, 1993.

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28

Jonathan, Barnes. Porphyry Introduction. Clarendon Press, 2006.

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29

Krivt͡sov, A. I. Medno-porfirovye mestorozhdenii͡a mira. Moskva: "Nedra", 1986.

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30

Heintze, Ludwig. Geologie und Geochemie der porphyrischen Stockwerk-Molybdän-Lagerstätte Tamboras, Zone La Negra (Peru). Hannover: Bundesanstalt für Geowissenschaften und Rohstoffe und den Geologischen Landesämtern in der Bunderepublik Deutschland, 1985.

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31

Learned, Robert Eugene. Analytical results and sample locality map of soil samples from the Tanama-Helecho porphyry copper district, Municipios of Utuado and Adjuntas, Puerto Rico. [Menlo Park, Calif.?]: U.S. Dept. of the Interior, Geological Survey, 1992.

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32

Learned, Robert Eugene. Analytical results and sample locality map of soil samples from the Tanama-Helecho porphyry copper district, Municipios of Utuado and Adjuntas, Puerto Rico. [Menlo Park, Calif.?]: U.S. Dept. of the Interior, Geological Survey, 1992.

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33

Learned, Robert Eugene. Analytical results and sample locality map of soil samples from the Tanama-Helecho porphyry copper district, Municipios of Utuado and Adjuntas, Puerto Rico. [Menlo Park, Calif.?]: U.S. Dept. of the Interior, Geological Survey, 1992.

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34

Evans, Tammy. Porphyria: The woman who has "the vampire disease". Far Hills, NJ: New Horizon Press, 1997.

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35

Krivt︠s︡ov, A. I. Medno-porfirovye mestorozhdenii︠a︡ mira. Moskva: "Nedra", 1986.

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36

Vogel, Alois. Der Wald aus Porphyr: Ein Lesebuch. Wrocław: Oficyna Wydawnicza ATUT, 1998.

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37

David, Kessel, ed. Methods in porphyrin photosensitization. New York: Plenum Press, 1985.

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38

Hafting, Jeff T. Strategies for growth management of Porphyra yezoensis (Ueda) blades in suspension cultures: A step towards land-based mariculture. 1998.

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39

Bioreactor development and cell culture of the marine macroalgae Porphyra (sp.) and Laminaria saccharina. jason G. Modrell, 1993.

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40

Modrell, Jason G. Bioreactor development and cell culture of the marine macroalgae Porphyra (sp.) and Laminaria saccharina. 1993.

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41

Sybert, Virginia P. Metabolic Disease. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780195397666.003.0011.

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Porphyrias – Congenital Erythropoietic Porphyria – Erythropoietic Protoporphyria – Hereditary Coproporphyria – Porphyria Cutanea Tarda – Variegate Porphyria – Mucopolysaccharidoses – Hunter Syndrome – Other Metabolic Disorders – Acrodermatitis Enteropathica – Alkaptonuria – Biotinidase Deficiency – Familial Cutaneous Amyloidosis – Prolidase Deficiency
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42

Langendonk, Janneke G., and Timothy M. Cox. Porphyrias. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0043.

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The porphyrias are disorders caused by overproduction of metabolites involved in heme biosynthesis. The four acute porphyrias— acute intermittent porphyria (AIP), variegate porphyria (VP), hereditary coproporphyria (HCP), and Doss Porphyria—present with severe abdominal pain, often accompanied by agitation, hypertension, and tachycardia associated with neuropathy and sometimes paralysis. Painful and disabling neurovisceral attacks are due to excess production of the heme precursor ALA (delta-aminolevulinic acid).While 90% of individuals with an inherited defect in heme biosynthesis will never develop symptoms, acute attacks in those affected are provoked by drugs, fasting, and alcohol; in women of reproductive age, they usually occur in the progestagenic phase of the menstrual cycle. All other porphyrias are considered cutaneous porphyrias. They present with blisters or pain on light exposed areas, toxic porphyrins accumulate and give rise to skin symptoms. The cutaneous porphyrias (PCT, EPP, XLEPP, and HEP) do not present with acute neurovisceral attacks (e.g., abdominal pain). However, severe systemic complications can occur.
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43

Sybert, Virginia P. Metabolic Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276478.003.0011.

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Chapter 11 covers Porphyrias (Congenital Erythropoietic Porphyria, Erythropoietic Protoporphyria, Hereditary Coproporphyria, Porphyria Cutanea Tarda, and Variegate Porphyria), Mucopolysaccharidoses (Hunter Syndrome), and Other Metabolic Disorders (Acrodermatitis Enteropathica, Alkaptonuria, Biotinidase Deficiency, Familial Cutaneous Amyloidosis, and Prolidase Deficiency). Each condition is discussed in detail, including dermatologic features, associated anomalies, histopathology, basic defect, treatment, mode of inheritance, prenatal diagnosis, and differential diagnosis.
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44

Steensma, David P. Benign Hematology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0294.

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The major forms of benign hematologic conditions are anemia, neutropenia, transfusion reactions, Gaucher disease, and porphyria. Anemia is a sign of disease rather than a disease itself. Anemia results from 1 or more of 3 pathologic mechanisms: inadequate production of red blood cells (RBCs) by the bone marrow, blood loss, or premature destruction of RBCs. The major causes of neutropenia include hematologic neoplasm, metastatic neoplasm involving the marrow, irradiation, vitamin B12 deficiency and folate deficiency, drugs, infections, congenital or acquired primary disorders of hematopoiesis, autoimmune neutropenia, hypersplenism, hemodilution, and benign idiopathic neutropenia. The porphyrias are enzyme disorders that are autosomal dominant with low disease penetrance, except for congenital erythropoietic porphyria, which is autosomal recessive, and porphyria cutanea tarda, which may be acquired and is associated with hepatitis C and hemochromatosis.
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45

F, Merk Hans, ed. Porphyria. Basel: Karger, 1998.

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46

Deegan, Patrick. Porphyria. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0179.

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This chapter discusses six diseases caused by inborn errors of metabolism affecting the biosynthesis of haem. Haem is a tetracyclic metal-binding compound involved in oxygen transport (in haemoglobin and myoglobin) and redox reactions (e.g. in the cytochrome P450 system). Each of these conditions is caused by a single gene defect in one of the enzymes involved in the biosynthesis of haem. Inheritance is usually autosomal dominant with incomplete penetrance. The enzyme defect results in disease, not as a result of deficiency of the reaction product, but as a result of accumulation of precursors. Early, soluble precursors, 5-aminolaevulinic acid, and porphobilinogen (not porphyrins as such) are neurotoxic and, when present in great excess, as occurs when flux through the haem synthetic pathway is increased in response to particular medications or hormones, lead to acute neurovisceral crises. Later cyclical precursors (porphyrins) in the pathway are also water soluble and excreted in urine, but are susceptible to activation by electromagnetic radiation in the visible spectrum and are converted to free-radical metabolites that cause pain, inflammation, and tissue damage in the skin. The final haem precursors (also porphyrins) are hydrophobic and excreted in the bile and faeces and are also activated by light to toxic metabolites.
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47

Dierdorf, Stephen F. Porphyria. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0026.

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Heme, and iron containing compound that forms the nonprotein portion of hemoglobin, is essential to life. Heme synthesis requires eight enzymatic steps, and a deficiency in any one of the eight enzymes can lead to the accumulation of potentially toxic intermediates. Some forms of porphyria may be asymptomatic until the patient receives a triggering agent, and acute porphyrias can also be difficult to diagnose because of the nonspecific clinical features. The most serious of the clinical manifestations is severe neurologic dysfunction. An attack can be triggered by medications administered during the perioperative period, and failure to act promptly can result in mortality rates as high as 5%.
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48

Porphyry and Ash. Sharpe Books, 2020.

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49

Magny, Ariane. Porphyry in Fragments. Taylor & Francis Group, 2018.

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50

Studies on Porphyry. University of London, School of Advanced Study, Institute of Classical Studies, 2006.

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