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Books on the topic 'Polyclonale'

Author: Grafiati
Published: 7 December 2024

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1

Gray, Lynn. Dynamic antibody industry, including polyclonals and monoclonals. Norwalk, CT: Business Communications Co., 2002.

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2

Rotheim, Philip. The dynamic antibody industry, including polyclonals and monoclonals. Norwalk, CT: Business Communications Co., 1992.

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3

Schmandt, Rosemarie Elizabeth. Production of polyclonal antibodies to protein kinase C. Ottawa: National Library of Canada, 1990.

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4

Croll, Andrew David. The regulation of polyclonal mitogen-stimulated human gamma- interferon production. [s.l.]: typescript, 1986.

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5

Rotheim, Philip. The dynamic monoclonal and polyclonal antibody business: Products, applications, and markets. Norwalk, CT: Business Communications Co., 1989.

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6

Farooq, Muhammad. Glycosylation of monoclonal and polyclonal human IgG: Influences of protein structure and physiological environments. Birmingham: University of Birmingham, 1997.

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7

Farooq, Muhammad. Glycosylation of monoclonal and polyclonal human IgG: Influences of protein structure and physiological environments. Birmingham: University of Birmingham, 1998.

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8

Likitdecharote, Banchorn. Detection of mycoplasma infections in swine by culture and immunological methods using polyclonal and monoclonal antibodies. Hannover: [s.n.], 1987.

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9

Boucher, Guillaume. Kinetic studies of alternative substrates for existing polyclonal catalytic antibodies. 2002.

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10

Infectious molecular clones encoding the HIV clade C envelope and neutralizing polyclonal antibodies. 2007.

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11

Carlino, Joseph A. Pregnancy-associated growth factor (PAGF): A T-dependent polyclonal activator of human lymphocytes. 1986.

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12

Thong, Bruce Yow Chin. Preparation of a new, specific polyclonal antibody that recognizes the carbohyrate moiety of digoxin. 1985.

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13

Li, Hong. Analysis of bovine herpesvirus 4 (DN 599) proteins with monoclonal antibodies and polyclonal immune serum. 1990.

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14

Nassar, Abdul-Rahman Hassan. Development, characterization and application of polyclonal antibodies against ovine adipocytes for body fat reduction by passive immunization. 1989.

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15

Piszel, Jane Louise. Biochemical characterization and production of polyclonal antibodies against two differentiation dependent nonhistone chromatin proteins from myocordial cells. 1987.

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16

Barber, Nicola Jane. Evaluation of the pH dependence of a polyclonal catalytic antibody preparation and generation of an analogous monoclonal catalytic antibody. 1997.

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17

Baker, Carlye Ann. Production and characterization of polyclonal and monoclonal antibodies to three virus-induced proteins of papaya ringspot virus type W. 1989.

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18

Korbet, Stephen M., Melvin M. Schwartz, and Edmund J. Lewis. Fibrillary and immunotactoid glomerulopathy. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0081.

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Abstract:
Fibrillary or immunotactoid nephropathy is a rare deposition disease of unknown cause in which highly organized deposits containing immunoglobulin and complement are found in the glomerular basement membrane and mesangium. These deposits are not amyloid fibrils and do not stain with Congo red. They are usually polyclonal and are not associated with monoclonal paraproteins, or with cryoglobulins or systemic lupus, distinguishing them from other non-amyloid fibrillary glomerulopathies. There is debate about whether there is a useful distinction between distinct fibrillary and rarer immunotactoid variants, the rarer immunotactoid variety being associated with larger, more organized, microtubular fibrils and possibly more commonly associated with malignancy (usually lymphoproliferative). However, clinically the conditions are similar. Patients usually present with heavy proteinuria or nephrotic syndrome. Microscopic haematuria is common and many have reduced glomerular filtration rate at presentation. Fifty per cent reach end-stage renal failure in 3–5 years and there is no convincing evidence that any treatment is effective in altering this outcome. Other organs appear to be affected only rarely (lung, liver). The disease may recur post-transplant but not always and usually more slowly, so does not preclude transplantation.
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