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Journal articles on the topic 'Polyarthriris'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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1

MJAAVATTEN, MARIA DAHL, ANNE JULSRUD HAUGEN, KNUT HELGETVEIT, HALVOR NYGAARD, GÖRAN SIDENVALL, TILL UHLIG, and TORE KRISTIAN KVIEN. "Pattern of Joint Involvement and Other Disease Characteristics in 634 Patients with Arthritis of Less Than 16 Weeks’ Duration." Journal of Rheumatology 36, no. 7 (June 1, 2009): 1401–6. http://dx.doi.org/10.3899/jrheum.081217.

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Objective.To investigate the distribution of joint involvement in a cohort of patients with very recent onset arthritis and describe the disease characteristics in these patients.Methods.A very early arthritis clinic (NOR-VEAC) was established as a multicenter study. General practitioners were asked to refer patients presenting with at least 1 swollen joint of maximum 16 weeks’ duration. Clinical and laboratory markers were examined.Results.We included 634 patients during the first 3 years, with mean (25th–75th percentile) arthritis duration of 30 (11–63) days. Monoarthritis was present in 243 (38.3%) patients, 216 (34.1%) had oligoarthritis, and 175 (27.6%) polyarthritis. Patients with polyarthritis were older, had longer duration of arthritis, and were more frequently anti-cyclic citrullinated peptide antibody and rheumatoid factor-positive. Patients in all 3 joint pattern groups (mono-/oligo-/polyarthritis) reported substantial effect on physical function, pain, and fatigue and had elevated levels of acute-phase reactants. Knee or ankle arthritis was most frequent in patients with mono- and oligoarthritis, whereas small joint involvement was most frequent in patients with polyarthritis.Conclusion.Patients with recent-onset arthritis report a substantial influence on health status. Mono- and oligoarthritis are at least as frequent as polyarthritis. Polyarthritic patients more frequently exhibit features associated with a worse outcome.

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2

Caprio, Maria Grazia, Mariarosaria Manganelli, Simona Limone, Massimiliano Sorbillo, Mario Quarantelli, Alberto Cuocolo, and Ciro Gabriele Mainolfi. "Extra-osseous 99mTc methylene diphosphonate uptake detected enlargement of the knee joint in patient with polyarthritis." SAGE Open Medical Case Reports 5 (January 1, 2017): 2050313X1774182. http://dx.doi.org/10.1177/2050313x17741824.

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Bone scintigraphy is a nuclear scanning test used to find abnormalities in the skeleton. Certain abnormal processes involving soft tissues can also cause skeletal accumulation of radiotracer during bone scintigraphy. We present a case of periarticular knee soft tissue 99mTc methylene diphosphonate uptake in a patient with asymmetric polyarthritis. A 33-year-old patient with asymmetric polyarthritis, skin lesions and joint pain underwent bone scintigraphy. Total body examination showed an extra-osseous uptake in periarticular soft tissue of knees joints. A detailed history checkup, physical examination and laboratory tests were carried out to understand the link between the extra-osseous uptake and the phosphonate binding in periarticular soft tissue. To improve the anatomical description of the soft tissue of the knees and to clarify the nature of the extra-skeletal 99mTc methylene diphosphonate uptake, magnetic resonance imaging scan was performed. 99mTc-labeled phosphonate binding has been reported in a number of extra-osseous conditions, but to our knowledge, there are a few cases showing bone tracer uptake in polyarthritis. In polyarthritic patients, whole-body bone scintigraphy were useful in examining the whole joints and detecting possible dubious extra-osseous uptake; in fact, it is able to select subjects who require further in-depth analysis, for example, magnetic resonance imaging.

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3

Hansson, Claes, Catharina Eriksson, and Gerd-Marie Alenius. "S-Calprotectin (S100A8/S100A9): A Potential Marker of Inflammation in Patients with Psoriatic Arthritis." Journal of Immunology Research 2014 (2014): 1–5. http://dx.doi.org/10.1155/2014/696415.

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Objective. To analyse levels of S100A8/S100A9 (calprotectin) and selected cytokines, in blood, in patients with psoriatic arthritis (PsA).Methods. Sixty-five patients with PsA were examined for clinical manifestations and laboratory measurements of S-calprotectin, ESR, hs-CRP, and selected cytokines. Thirty-two patients had mono-/oligoarthritis and 33 had polyarthritis. S-calprotectin, hs-CRP, and cytokines were measured using ELISA, immunoturbidimetry, and multiplex technology (Bio-Plex). Patients with PsA were compared with 31 healthy controls.Results. S-calprotectin and hs-CRP levels were significantly higher in patients with PsA compared with controls (P<0.001andP<0.001, resp.). Patients suffering a polyarthritic disease pattern presented with significantly higher levels of S-calprotectin compared with controls and patients with mono-/oligoarthritis (P<0.001andP=0.017, resp.). The levels of S-calprotectin correlated with hs-CRP (P<0.001;rs=0.441), swollen joint count (P=0.002,rs=0.397), and CXCL10 (P=0.046,rs=0.678) but not with any of the other cytokines evaluated. In multiple logistic regression analysis, S-calprotectin was the only variable significantly associated with psoriatic arthritis (P=0.002,OR=1.006, 95% CI = 1.002–1.010).Conclusion. S-calprotectin and hs-CRP levels were significantly higher in patients with PsA. A polyarthritic disease pattern showed higher levels of S-calprotectin than mono-/oligoarthritis. S-calprotectin is considered a potential marker of disease activity in patients with PsA.

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4

Leslie, A., and I. Watt. "Polyarthritis." Imaging 11, no. 2 (June 1999): 98–103. http://dx.doi.org/10.1259/img.11.2.110098.

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5

Dao, Kathryn, and John J. Cush. "Acute polyarthritis." Best Practice & Research Clinical Rheumatology 20, no. 4 (August 2006): 653–72. http://dx.doi.org/10.1016/j.berh.2006.05.007.

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6

Wiebelitz, K. R., and A. M. Beer. "Chronische Polyarthritis." MMW - Fortschritte der Medizin 152, no. 20 (May 2010): 23. http://dx.doi.org/10.1007/bf03366591.

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7

Gelsomino, Marco, and Giorgio Tamborrini. "Therapieresistente Polyarthritis." Praxis 103, no. 14 (July 1, 2014): 841–44. http://dx.doi.org/10.1024/1661-8157/a001720.

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Die Differenzialdiagnose einer unklaren Polyarthritis im Alter umfasst u. a. Autoimmunerkrankungen, Kristallablagerungserkrankungen oder infektassoziierte Polyarthritiden. Bei klinischem Verdacht auf eine Gicht sollte durch Harnsäurekristallnachweis in der Synovialflüssigkeit oder im Tophus idealerweise bestätigt werden. Wenn eine Arthrozentese nicht realisierbar ist, können nicht-invasive hochspezifische Untersuchungen, wie der hochauflösenden muskuloskelettale Ultraschall oder eine DECT-Untersuchung hilfreich sein.

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8

Jones, Hugh W., Patrick Gordon, and Terence Gibson. "Tuberculous Polyarthritis." JCR: Journal of Clinical Rheumatology 2, no. 2 (April 1996): 96–98. http://dx.doi.org/10.1097/00124743-199604000-00007.

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9

Marshall, John B., and Robert McMurray. "Acute polyarthritis." Postgraduate Medicine 95, no. 8 (June 1994): 165–68. http://dx.doi.org/10.1080/00325481.1994.11945870.

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10

Peichl, Peter. "Chronische Polyarthritis." DoctorConsult - The Journal. Wissen für Klinik und Praxis 2, no. 3 (November 2011): e167-e170. http://dx.doi.org/10.1016/j.dcjwkp.2011.10.002.

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11

Touré, Maïmouna, Mouhamed T. Diagne, Amadou Badji, Souleymane Thiam, Coumba Diouf, Moustapha Niasse, Mbaye Sene, et al. "Prevalence and impact of adiposity and sarcopenia during rheumatoid arthritis: rapid and non-invasive evaluation in Sub-Saharan African women." International Journal of Research in Medical Sciences 7, no. 8 (July 25, 2019): 2920. http://dx.doi.org/10.18203/2320-6012.ijrms20193371.

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Background: Body composition plays a fundamental role in the occurrence of complications in rheumatoid arthritis. Authors conducted this study, which aimed to determine body composition and its effects on physiological status in African sub-Saharan polyarthritis women.Methods: The anthropometric parameters were measured after an interview and a complete physical examination. The body composition was evaluated using a Tanita® brand bioimpedance meter. Finally, all the patients had a dosage of certain biochemical parameters.Results: An excess of percent fat mass was noted in more than half of women (59.52%) without loss of muscle mass. At the same time, 30% of women had a significant decrease in the percentage of body water. The BMI did not appear to be an adequate proxy for these changes. Visceral fat level was elevated just in 16% of women, however it would be a determinant of physiological aging of subjects. Dual therapy methotrexate and corticosteroid would have varying effects depending on the duration and the dose of treatment. The basic metabolism in polyarthritic subjects would be dependent on two parameters namely muscle mass and inflammatory state.Conclusions: Evaluating changes in body composition quickly, non-invasively and inexpensively is possible. It could be useful in the follow-up of rheumatoid arthritis. Managing these changes can reduce cardiovascular morbidity and mortality in rheumatoid arthritis.

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12

Mekan, S. F., O. Saeed, and J. A. Khan. "Invasive aspergillosis with polyarthritis. Fallbericht. Invasive Aspergillose mit Polyarthritis." Mycoses 47, no. 11-12 (December 2004): 518–20. http://dx.doi.org/10.1111/j.1439-0507.2004.01031.x.

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13

Castro, João Paulo, Gabriel Atanásio, Maria Ana Canelas, André Ferreira, Ana Rita Barbosa, Marta Barbedo, and Regina Abreu. "A case report of pancreatitis-panniculitis-polyarthritis syndrome – an unusual but serious presentation of pancreatic disease." Scottish Medical Journal 65, no. 1 (December 30, 2019): 19–23. http://dx.doi.org/10.1177/0036933019897373.

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The clinical triad of peripheral cutaneous erythematous nodules, oligo or polyarthritis with intraosseous fat necrosis in the setting of pancreatic disease defines a rare entity called pancreatitis-panniculitis-polyarthritis syndrome. The early recognition of this triad is critical due to its high mortality rate and the rapid onset of osseous and articular disabilities. We describe the clinical course of a 54-year-old patient with complaints of weight loss and fever who presented to our hospital with signs of polyarthritis and appendicular erythematous cutaneous nodules. Clinical investigation revealed high inflammatory and pancreatic enzymes levels. Cutaneous biopsy and articular MRI showed evidence of peripheral necrosis. After a thorough investigation, a diagnosis of panniculitis-polyarthritis-pancreatitis syndrome was established. Treatment was initiated, and a slow but steady improvement was observed. Further complications of the disease process were observed. This case highlights the importance of recognising the association between panniculitis and polyarthritis with pancreatic disease in order to improve outcomes.

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14

Läkamp, A., E. M. Beckold, A. Gerdwilker, I. Nolte, A. Meyer-Lindenberg, and M. Hewicker-Trautwein. "Polyarthritis-Polymyositis-Syndrom bei einem sechs Monate alten Deutsch Stichelhaar." Tierärztliche Praxis Ausgabe K: Kleintiere / Heimtiere 36, no. 01 (2008): 47–54. http://dx.doi.org/10.1055/s-0038-1622660.

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Zusammenfassung: Gegenstand: Beim Polyarthritis-Polymyositis-Syndrom des Hundes handelt es sich um eine seltene, wahrscheinlich immunvermittelte Krankheit unbekannter Ursache. Nach einer kurzen Literaturübersicht der immunvermittelten Arthritiden werden die klinischen und insbesondere pathologischen Befunde bei einer sechs Monate alten Deutsch-Stichelhaar-Hündin beschrieben, die wegen einer seit mehreren Wochen bestehenden, therapieresistenten Polyarthritis überwiesen wurde. Klinische Befunde: Nach einer fünftägigen Fieberphase zeigte die Hündin acht Wochen lang einen steifen Gang und wechselnde Lahmheiten aller Gliedmaßen. Verschiedene Gelenke waren schmerzhaft und vermehrt gefüllt. Die Muskulatur war zum Teil nicht gut entwickelt und palpatorisch fest und schmerzhaft. Ergebnisse: Die Obduktion und anschließende histologische Untersuchung ergab an mehreren Gelenken aller vier Gliedmaßen eine floride, nichterosive chronische Polyarthritis und eine mit Muskelfaserdegeneration einhergehende Polymyositis. Schlussfolgerung und klinische Relevanz: Beim Auftreten von Polyarthritiden beim Hund sind differenzialdiagnostisch verschiedene, meist immunvermittelte und teilweise nur schwer voneinander abgrenzbare Krankheiten zu berücksichtigen. Das Krankheitsbild einer Polyarthritis/Polymyositis kann nur mittels histologischer Untersuchungen von Muskelund Synovialmembranproben diagnostiziert werden. Das Polyarthritis-Polymyositis-Syndrom des Hundes hat nach Literaturangaben wegen des schlechten Ansprechens auf immunsuppressive Therapiemaßnahmen eine sehr vorsichtige Prognose.

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15

BAKRI, KAMAL M. "Polyarthritis and Neutropenia." Annals of Internal Medicine 104, no. 1 (January 1, 1986): 127. http://dx.doi.org/10.7326/0003-4819-104-1-127_1.

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16

Davis, Paul, Michael Stein, Ahmed Latif, and Jean Emmanuel. "HIV AND POLYARTHRITIS." Lancet 331, no. 8591 (April 1988): 936. http://dx.doi.org/10.1016/s0140-6736(88)91738-2.

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17

Salmeron, C., M. Marty, H. Richet, M. C. Escande, F. Besancon, and H. P. H. Lagrange. "Primary meningococcal polyarthritis." Journal of Infection 13, no. 3 (November 1986): 281–83. http://dx.doi.org/10.1016/s0163-4453(86)91322-8.

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18

Lang, Gerhardus. "Primär chronische Polyarthritis." Allgemeine Homöopathische Zeitung 251, no. 5 (September 2006): 242–43. http://dx.doi.org/10.1055/s-2006-952056.

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19

MOLTKE, OTTO. "»Polyarthritis urethritica»1." Acta Medica Scandinavica 89, no. 6 (April 24, 2009): 606–16. http://dx.doi.org/10.1111/j.0954-6820.1936.tb15451.x.

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20

Majeed, Hasan A. "Acute Rheumatic Polyarthritis." American Journal of Diseases of Children 144, no. 7 (July 1, 1990): 831. http://dx.doi.org/10.1001/archpedi.1990.02150310099035.

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21

Pinals, Robert S. "Polyarthritis and Fever." New England Journal of Medicine 330, no. 11 (March 17, 1994): 769–74. http://dx.doi.org/10.1056/nejm199403173301108.

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22

Haakshorst, R. "Eine Ledum-Polyarthritis." Zeitschrift für Klassische Homöopathie 5, no. 03 (April 3, 2007): 125–26. http://dx.doi.org/10.1055/s-2006-936985.

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23

CHAKRAVARTY, K., S. H. S. ELGBANI, and D. G. I. SCOTT. "Q-Fever Polyarthritis." Rheumatology 32, no. 4 (April 1, 1993): 346. http://dx.doi.org/10.1093/rheumatology/32.4.346.

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24

Singh, Surjit, and Sonia Mehra. "Approach to Polyarthritis." Indian Journal of Pediatrics 77, no. 9 (August 24, 2010): 1005–10. http://dx.doi.org/10.1007/s12098-010-0163-5.

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25

López-Bote, Juan Pedro, Carmelo Bernabeu, Alberto Marquet, Jesús M. Fernández, and Vicente Larraga. "Adjuvant-induced polyarthritis. synovial cell activation prior to polyarthritis onset." Arthritis & Rheumatism 31, no. 6 (June 1988): 769–75. http://dx.doi.org/10.1002/art.1780310611.

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26

Fraisse, Thibaut, Olivier Boutet, Anne-Marie Tron, and Emmanuel Prieur. "Pancreatitis, panniculitis, polyarthritis syndrome: An unusual cause of destructive polyarthritis." Joint Bone Spine 77, no. 6 (December 2010): 617–18. http://dx.doi.org/10.1016/j.jbspin.2010.05.005.

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27

Ralston, S. H., R. McVicar, A. Y. Finlay, R. Morton, and D. A. Pitkeathly. "Lymphomatoid Granulomatosis Presenting with Polyarthritis." Scottish Medical Journal 33, no. 6 (December 1988): 373–74. http://dx.doi.org/10.1177/003693308803300610.

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Polyarthritis in lymphomatoid granulomatosis is rare. In the present report we describe a patient with lymphomatoid granulomatosis who presented with an acute inflammatory polyarthritis three years before the typical skin and pulmonary manifestations of the disease became evident.

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28

Ben Fredj Ismail, Fatma, Amel Rezgui, Monia Karmani, Olfa Ben Abdallah, Samira Azzebi, and Chedia Laouani Kechrid. "A Seropositive Nodular Rheumatoid Polyarthritis without Arthritis: Does It Exist?" Case Reports in Medicine 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/983985.

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The rheumatoid polyarthritis is the most frequent chronic polyarthritis. It affects essentially the woman between 40 and 60 years. Rheumatic subcutaneous nodules and tenosynovitis are usually associated with seropositive symptomatic rheumatoid polyarthritis. It is, however, rare that they constitute the essential clinical expression of the disease. In this case, it makes dispute another exceptional form of rheumatoid arthritis such as rheumatoid nodulosis. A 60-year-old woman was hospitalized for tumefaction of the dorsal face of the right hand evolving two months before. The clinical examination found subcutaneous nodules from which the exploration ended in rheumatoid nodules with tenosynovitis. The evolution after four years was favourable under corticosteroid therapy, methotrexate, and colchicine.

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29

Johnson, Kirstin C., and Andrew Mackin. "Canine Immune-Mediated Polyarthritis." Journal of the American Animal Hospital Association 48, no. 1 (January 1, 2012): 12–17. http://dx.doi.org/10.5326/jaaha-ms-5744.

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Immune-mediated polyarthritis (IMPA) is a common disease process in the dog.1 The immune-mediated polyarthropathies are divided into two major categories: erosive (or deforming) and nonerosive (or nondeforming). Understanding the pathophysiology of the immune attack on affected joints is paramount in choosing the most effective therapy for managing canine IMPA. This review article is the first of a two-part series and focuses on the pathophysiology of IMPA. The second article in this series, to be published in the March/April 2012 issue, concentrates on the diagnosis and treatment of immune-mediated polyarthritis.

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30

Johnson, Kirstin C., and Andrew Mackin. "Canine Immune-Mediated Polyarthritis." Journal of the American Animal Hospital Association 48, no. 2 (March 1, 2012): 71–82. http://dx.doi.org/10.5326/jaaha-ms-5756.

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Canine immune-mediated polyarthritis (IMPA) is a diagnosis of exclusion based predominantly on clinical signs, characteristic joint fluid analysis, and elimination of potential joint infection. Ultimately, an appropriate and sustained response to immunosuppressive therapy may become the final diagnostic criterion used. Identifying associated disease processes, including breed-specific syndromes, remote infection, inflammation, drug exposure, vaccine exposure, or neoplasia, as well as initial response to therapy, is often an important contributor to prognosis. This review article is the second of a two part series and focuses on the diagnosis and treatment of immune-mediated polyarthritis. The first article in this series, published in the January/February 2012 issue, concentrated on the pathophysiology of IMPA.

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31

Kashyap, Subhash, Vinay Shanker, Sandhya Kumari, and Lokesh Rana. "Panniculitis-polyarthritis-pancreatitis syndrome." Indian Journal of Dermatology, Venereology, and Leprology 80, no. 4 (2014): 352. http://dx.doi.org/10.4103/0378-6323.136926.

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32

Nash, P., and T. Harrington. "Acute Barmah Forest polyarthritis." Australian and New Zealand Journal of Medicine 21, no. 5 (October 1991): 736–38. http://dx.doi.org/10.1111/j.1445-5994.1991.tb01379.x.

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33

Zullo, Angelo, Francesca Fanfarillo, Simon Winn, Massimo Delfino, Antonia Cascino, and Luigi Baratta. "HCV Hepatitis and Polyarthritis." Journal of Clinical Gastroenterology 30, no. 2 (March 2000): 216–17. http://dx.doi.org/10.1097/00004836-200003000-00023.

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34

RYDÉN, ERIK. "Chronic Polyarthritis and Trauma." Acta Medica Scandinavica 114, no. 4-5 (April 24, 2009): 442–69. http://dx.doi.org/10.1111/j.0954-6820.1943.tb11242.x.

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35

Norgaard, A. "Chronic Polyarthritis in Denmark." Acta Medica Scandinavica 130, S206 (April 24, 2009): 437–42. http://dx.doi.org/10.1111/j.0954-6820.1948.tb12078.x.

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36

Rubbert-Roth, Andrea. "Differenzialdiagnostik der frühen Polyarthritis." DMW - Deutsche Medizinische Wochenschrift 140, no. 15 (July 31, 2015): 1125–30. http://dx.doi.org/10.1055/s-0041-103627.

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37

Jancin, Bruce. "OCs Tame Inflammatory Polyarthritis." Internal Medicine News 44, no. 15 (September 2011): 27. http://dx.doi.org/10.1016/s1097-8690(11)70781-8.

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38

Waßmuth, R. "Immungenetik der chronischen Polyarthritis." Aktuelle Rheumatologie 19, no. 03 (May 1994): 72–76. http://dx.doi.org/10.1055/s-2008-1046665.

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39

McHugh, N. J., G. J. Campbell, J. J. Landreth, and M. R. Laurent. "Polyarthritis and angioimmunoblastic lymphadenopathy." Annals of the Rheumatic Diseases 46, no. 7 (July 1, 1987): 555–58. http://dx.doi.org/10.1136/ard.46.7.555.

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Narváez, Javier, Maria Marta Bianchi, Pilar Santo, Diana de la Fuente, Valeria Ríos-Rodriguez, Ferran Bolao, José Antonio Narváez, and Joan Miquel Nolla. "Pancreatitis, Panniculitis, and Polyarthritis." Seminars in Arthritis and Rheumatism 39, no. 5 (April 2010): 417–23. http://dx.doi.org/10.1016/j.semarthrit.2008.10.001.

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41

Gschwend, N., H. Brattström, H. Albrecht, W. Puhl, and H. Thabe. "HWS bei chronischer Polyarthritis." Aktuelle Rheumatologie 12, no. 02 (March 1987): 131–35. http://dx.doi.org/10.1055/s-2008-1047912.

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42

Gruber, A., U. Donhauser-Gruber, and H. Mathies. "Sport bei chronischer Polyarthritis." Aktuelle Rheumatologie 10, no. 03 (May 1985): 81–86. http://dx.doi.org/10.1055/s-2008-1048033.

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43

Dudler, J., J. C. Gerster, and A. So. "Polyarthritis and pitting oedema." Annals of the Rheumatic Diseases 58, no. 3 (March 1, 1999): 142–47. http://dx.doi.org/10.1136/ard.58.3.142.

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44

Wessinghage, D. "Chronische Polyarthritis und Polytrauma." Zeitschrift für Orthopädie und ihre Grenzgebiete 122, no. 05 (March 18, 2008): 639–42. http://dx.doi.org/10.1055/s-2008-1045043.

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45

Katz, Steven J., Alison Kydd, and Elaine A. Yacyshyn. "Pancreatitis, panniculitis and polyarthritis." Case Reports in Internal Medicine 1, no. 1 (January 15, 2014): 1. http://dx.doi.org/10.5430/crim.v1n1p1.

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Objectives: To present a case of pancreatitis, panniculitis and polyarthritis syndome, including the full spectrum of the arthritis component. Methods: A case of PPP syndrome was examined, with a detailed review of the patient's synovial fluid aspirate including cell count, crystals, culture and a lipid analysis which was compared to a control patient. Results: The synovial fluid aspirate from PPP syndrome demonstrates unique crystal formation and higher lipids. Conclusion: PPP syndrome is a rare condition which can be difficult to diagnose. A lipid analysis of a patient's synovial fluid aspirate may be a useful tool to aid in the diagnosis.

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46

Aundhakar, SwatiC, SanketK Mahajan, and MakarandB Mane. "Panniculitis-polyarthritis-pancreatitis syndrome." Saudi Journal for Health Sciences 1, no. 3 (2012): 166. http://dx.doi.org/10.4103/2278-0521.106089.

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47

Chatterjee, Soumya. "Malar Rash and Polyarthritis." JAMA 321, no. 3 (January 22, 2019): 303. http://dx.doi.org/10.1001/jama.2018.19498.

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48

Berger, Robert G. "Mycosis fungoides with polyarthritis." Arthritis & Rheumatism 31, no. 10 (October 1988): 1335–36. http://dx.doi.org/10.1002/art.1780311024.

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49

Grondalen, J. "Trimethoprim-sulphonamide induced polyarthritis." Veterinary Record 121, no. 7 (August 15, 1987): 155. http://dx.doi.org/10.1136/vr.121.7.155.

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50

Rippstein, P. F., and F. D. Naal. "Sprunggelenkprothese bei chronischer Polyarthritis." Der Orthopäde 40, no. 11 (October 20, 2011): 984–90. http://dx.doi.org/10.1007/s00132-011-1827-1.

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