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Books on the topic 'Polyarthriris'

Author: Grafiati
Published: 4 June 2021
Last updated: 2 February 2022

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1

Mellenthin-Seemann, Ulrike, Friederike Steier, Andrea Schulz, and Heinz-Gerd Biester. Gelenkschutzunterweisung bei Patienten mit chronischer Polyarthritis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-662-07409-1.

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2

Franke, Martin, ed. Die Basistherapie der chronischen Polyarthritis mit oralem Gold. Heidelberg: Steinkopff, 1986. http://dx.doi.org/10.1007/978-3-642-72380-3.

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3

Interkultureller Vergleich der Schmerzwahrnehmung und Krankheitsverarbeitung bei türkischen und deutschen Patienten mit chronischer Polyarthritis. Frankfurt am Main: P. Lang, 1996.

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4

Young, Mark E. Skills-Training bei Borderline- und Posttraumatischer Belastungsstörung. Wien, Austria: Springer, 2005.

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5

Heilmeyer, L., and W. Müller. Die Serologie der Chronischen Polyarthritis. Springer, 2013.

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6

Brückle, Wolfgang. Chronische Polyarthritis. Diagnose, Verlauf, Therapien. Urania, Stuttgart, 2001.

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7

Die Basistherapie Der Chronischen Polyarthritis Mit Oralem Gold. Steinkopff-Verlag Darmstadt, 1986.

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8

Koharenzgefuhl Und Krankheits-verarbeitung Bei Patientinnen Und Patienten Mit Chronischer Polyarthritis (Europaische Hochschulschriften: Reihe). Peter Lang Publishing, 2004.

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9

Stacey, Victoria. Musculoskeletal, rheumatology, and wound management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199592777.003.0005.

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Musculoskeletal conditions: introduction - Long-bone anatomy - Fracture descriptions - Open fractures - Compartment syndrome - Upper limb injuries - Lower limb injuries - Rheumatology: introduction - Monoarthritis - Polyarthritis - Septic arthritis - Gonococcal arthritis - Crystal arthropathies - Sero-negative spondyloarthropathies - Rheumatoid arthritis - Acute back pain - Wound management: introduction - Describing wounds and hand injuries - Hand and wrist anatomy - Hand infections - Special hand injuries - Tetanus - SAQs
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10

Miller, Aaron E., and Teresa M. DeAngelis. Rheumatoid Arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0012.

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Rheumatoid arthritis (RA) is a systemic inflammatory disease that is characterized principally by a polyarthritis, but can result in several neurologic complications involving both the central and peripheral nervous system. In addition, several immunotherapies used to treat RA have been associated with neurological complications. In this chapter, we review the characteristic neurological sequelae of RA as well as the possible neurological consequences of its therapeutic regimens.
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11

Scarpa, Raffaele, Francesco Caso, Luisa Costa, Rosario Peluso, Nicola Matteo Dario Di Minno, and Antonio Del Puente. Peripheral arthritis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0010.

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Clinical presentation of peripheral arthritis in patients with psoriatic arthritis (PsA), has been described by Moll and Wright who classified it into four subsets: symmetrical polyarthritis, asymmetrical oligoarthritis, distal interphalangeal (DIP) arthritis and arthritis mutilans. In the symmetrical polyarthritis subset, the distribution of articular involvement is similar to rheumatoid arthritis and this has for many years justified the inappropriate use of the terminology ‘rheumatoid-like form’, at present completely abandoned. Oligoarthritis is characterized by asymmetrical involvement of few joints (less than four), which include scattered DIP or proximal interphalangeal (PIP) joints and/or metatarsophalangeal joints. DIP arthritis may occur with symmetrical or asymmetrical features, and it is often in strict association with onycopathy. The arthritis mutilans pattern is characterized by osteolysis of phalanx and metacarpals and it is very rare, occurring in less than 1% of patients with established form of arthritis. In 15-20% of the cases the arthritis may precede the onset of the psoriatic skin rash. Consequently, psoriatic arthritis ‘sine psoriasis’ should not be considered a rare clinical finding. In this subset articular involvement is clinically expressed, while cutaneous is apparently absent. Laboratory tests and imaging are relevant for differential diagnosis which in some presentations may represent a diagnostic challenge. The outcome of peripheral patterns of PsA patients is related not only to the spectrum of peripheral phenotypes, but also to early diagnosis, and metabolic aspects, which may affect excess in morbidity and mortality.
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12

Naides, Stanley J. Viral arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0102.

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Viral infection may cause sudden onset polyarthralgia or polyarthritis. Recognizing viral syndromes during the acute phase of illness is critical as markers of acute infection may fade during convalescence. While joint symptoms and signs in many cases are self limited, in others joint involvement may persist for months to years. Acute and chronic findings may resemble classic idiopathic diseases such as rheumatoid arthritis or systemic lupus erythematosus. Some viral infections may manifest with rash, vasculitis, or organ involvement. Understanding of epidemiology, geography, clinical presentation, virus behaviour and host response assists diagnosis and selection of appropriate management. Understanding virus-host interactions may offer insights into mechanisms of pathogenesis in idiopathic rheumatic diseases.
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13

Gaston, J. S. Hill. Reactive arthritis and enteropathic arthropathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0115.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though mild polyarthritis can also occur. Septic and crystal-induced arthritis are the principal differential diagnoses. Extra-articular features may aid diagnosis, which otherwise requires laboratory evidence of preceding infection. Bacterial components traffic to the joint (which is nevertheless sterile), and elicit local proinflammatory immune responses. Most ReA is self-limiting, but persistent cases may require disease-modifying anti-rheumatic drugs or even biologics.
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14

Gaston, J. S. Hill. Reactive arthritis and enteropathic arthropathy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0115_update_002.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though mild polyarthritis can also occur. Septic and crystal-induced arthritis are the principal differential diagnoses. Extra-articular features may aid diagnosis, which otherwise requires laboratory evidence of preceding infection. Bacterial components traffic to the joint (which is nevertheless sterile), and elicit local pro-inflammatory immune responses. Most ReA is self-limiting, but persistent cases may require disease-modifying anti-rheumatic drugs or even biologics.
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15

Keat, Andrew. Oligoarticular disease. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0008.

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Oligoarthritis is a pattern of arthritis which most commonly resolves into a member of the spondyloarthritis family or sarcoidosis. Uncommonly it progresses to forms of arthritis more commonly associated with polyarthritis or monoarthritis and rarely it is associated with malignant or paraneoplastic syndromes. Three key aspects of diagnosis are consideration of possible diagnoses in the patient's age and ethnic groups; careful consideration of the personal and family history; and a search for and correct identification of characteristic associated features. This frequently involves collaborative working with other specialists including dermatologists, ophthalmologists, genitourinary physicians, respiratory physicians, and others. Precise diagnosis usually then involves subsequent investigation for diagnostic features including evidence of recent infection, HLA B27, autoantibodies, tissue-specific features of sarcoidosis, inflammatory bowel disease, and, occasionally, malignant disease. Management is dependent on clear diagnosis and precise delineation of underlying conditions such as infection. The purpose of this chapter is to provide a guide to the diagnostic approach and an algorithm for routine clinical practice. Detailed descriptions of the conditions included and investigations appropriate for the establishment or exclusion of individual diagnoses are discussed elsewhere in this volume.
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16

Smith, Robert M. Other bacterial diseasesErysipeloid. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0025.

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Erysipeloid is an acute bacterial infection usually causing acute localised cellulitis as a secondary infection of traumatised skin. It is caused by Erysipelothrix rhusiopathiae (insidiosa), a non-sporulating Gram-positive rod-shaped bacterium, ubiquitous in the environment. It is the cause of swine erysipelas and also a pathogen or commensal in a variety of wild and domestic birds, animal and marine species. Human infection primarily associated with occupational exposure to infected or contaminated animals or handling animal products and therefore is commoner in farmers, butchers and abattoir workers and fisherman.Risk factors for the rare human invasive E. rhusiopathiae infection include conditions that affect the host immune response, such as alcoholism, cancer and diabetes. Treatment is with penicillin.Erysipelas can affect animals of all ages but is recognised more frequently in juveniles. Swine exhibit similar stages to the disease in man. Clinical manifestations in swine vary from the classical rhomboid urticaria (diamond skin), the condition of greatest prevalence and economic importance, to sepsis, polyarthritis, pneumonia and death.Prevention is largely a matter of good hygiene, herd management and by raising awareness in those at risk (especially butchers, farmers and fishermen); ensuring that clinicians are aware of E. rhusiopathiae as a possible cause of occupational skin lesions and bacterial endocarditis is important.
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17

Aletaha, Daniel, and Helga Radner. Rheumatoid arthritis—diagnosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0110.

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Rheumatoid arthritis (RA) is among the most disabling form of chronic inflammatory joint disease. Not all forms of arthritis develop into RA; on the contrary, it may be very challenging to differentiate RA from cases of arthritis that are self-limiting or caused by another disease. Evaluation of early arthritis includes some basic steps, such as excluding trauma, crystal, or infectious-related disease, as well as considering additional features that may guide towards a specific diagnosis. If no specific diagnosis can then be made, the presentation can be labelled as undifferentiated arthritis. Typical differential diagnoses of RA include viral polyarthritis, seronegative spondylarthropathies, polymyalgia rheumatic, and other systemic rheumatic diseases. In 2010, new classification criteria were published that led to a change in the approach to RA. Compared to the previous criteria, the American College of Rheumatology (ACR) 1987 criteria, a scoring system was devised, appreciating the type and number of affected joints (up to 5 points), as well as serology (up to 3 points), elevated acute-phase reactants (1 point), and a symptom persistence of 6 weeks or longer (1 point). If 6 or more points are reached, then classifiable RA is present. Importantly, classification status, which is used for study purposes, is not always identical to the diagnostic status, which often leads to clinical treatment.
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18

Skills-Training bei Borderline- und Posttraumatischer Belastungsstörung. 3rd ed. Wien, Austria: Springer, 2012.

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19

Sendera, Alice, and Martina Sendera. Skills-Training bei Borderline- und posttraumatischer Belastungsstörung. Springer, 2005.

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20

Skills-Training bei Borderline- und Posttraumatischer Belastungsstörung. 2nd ed. Wien, Austria: Springer, 2007.

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21

Loisl, Daniela, and Rudolf Puchner. Diagnose Rheuma: Lebensqualität mit einer entzündlichen Gelenkerkrankung. Springer, 2005.

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