Academic literature on the topic 'Polyarthriris'

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Journal articles on the topic "Polyarthriris"

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MJAAVATTEN, MARIA DAHL, ANNE JULSRUD HAUGEN, KNUT HELGETVEIT, HALVOR NYGAARD, GÖRAN SIDENVALL, TILL UHLIG, and TORE KRISTIAN KVIEN. "Pattern of Joint Involvement and Other Disease Characteristics in 634 Patients with Arthritis of Less Than 16 Weeks’ Duration." Journal of Rheumatology 36, no. 7 (June 1, 2009): 1401–6. http://dx.doi.org/10.3899/jrheum.081217.

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Objective.To investigate the distribution of joint involvement in a cohort of patients with very recent onset arthritis and describe the disease characteristics in these patients.Methods.A very early arthritis clinic (NOR-VEAC) was established as a multicenter study. General practitioners were asked to refer patients presenting with at least 1 swollen joint of maximum 16 weeks’ duration. Clinical and laboratory markers were examined.Results.We included 634 patients during the first 3 years, with mean (25th–75th percentile) arthritis duration of 30 (11–63) days. Monoarthritis was present in 243 (38.3%) patients, 216 (34.1%) had oligoarthritis, and 175 (27.6%) polyarthritis. Patients with polyarthritis were older, had longer duration of arthritis, and were more frequently anti-cyclic citrullinated peptide antibody and rheumatoid factor-positive. Patients in all 3 joint pattern groups (mono-/oligo-/polyarthritis) reported substantial effect on physical function, pain, and fatigue and had elevated levels of acute-phase reactants. Knee or ankle arthritis was most frequent in patients with mono- and oligoarthritis, whereas small joint involvement was most frequent in patients with polyarthritis.Conclusion.Patients with recent-onset arthritis report a substantial influence on health status. Mono- and oligoarthritis are at least as frequent as polyarthritis. Polyarthritic patients more frequently exhibit features associated with a worse outcome.
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Caprio, Maria Grazia, Mariarosaria Manganelli, Simona Limone, Massimiliano Sorbillo, Mario Quarantelli, Alberto Cuocolo, and Ciro Gabriele Mainolfi. "Extra-osseous 99mTc methylene diphosphonate uptake detected enlargement of the knee joint in patient with polyarthritis." SAGE Open Medical Case Reports 5 (January 1, 2017): 2050313X1774182. http://dx.doi.org/10.1177/2050313x17741824.

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Bone scintigraphy is a nuclear scanning test used to find abnormalities in the skeleton. Certain abnormal processes involving soft tissues can also cause skeletal accumulation of radiotracer during bone scintigraphy. We present a case of periarticular knee soft tissue 99mTc methylene diphosphonate uptake in a patient with asymmetric polyarthritis. A 33-year-old patient with asymmetric polyarthritis, skin lesions and joint pain underwent bone scintigraphy. Total body examination showed an extra-osseous uptake in periarticular soft tissue of knees joints. A detailed history checkup, physical examination and laboratory tests were carried out to understand the link between the extra-osseous uptake and the phosphonate binding in periarticular soft tissue. To improve the anatomical description of the soft tissue of the knees and to clarify the nature of the extra-skeletal 99mTc methylene diphosphonate uptake, magnetic resonance imaging scan was performed. 99mTc-labeled phosphonate binding has been reported in a number of extra-osseous conditions, but to our knowledge, there are a few cases showing bone tracer uptake in polyarthritis. In polyarthritic patients, whole-body bone scintigraphy were useful in examining the whole joints and detecting possible dubious extra-osseous uptake; in fact, it is able to select subjects who require further in-depth analysis, for example, magnetic resonance imaging.
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Hansson, Claes, Catharina Eriksson, and Gerd-Marie Alenius. "S-Calprotectin (S100A8/S100A9): A Potential Marker of Inflammation in Patients with Psoriatic Arthritis." Journal of Immunology Research 2014 (2014): 1–5. http://dx.doi.org/10.1155/2014/696415.

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Objective. To analyse levels of S100A8/S100A9 (calprotectin) and selected cytokines, in blood, in patients with psoriatic arthritis (PsA).Methods. Sixty-five patients with PsA were examined for clinical manifestations and laboratory measurements of S-calprotectin, ESR, hs-CRP, and selected cytokines. Thirty-two patients had mono-/oligoarthritis and 33 had polyarthritis. S-calprotectin, hs-CRP, and cytokines were measured using ELISA, immunoturbidimetry, and multiplex technology (Bio-Plex). Patients with PsA were compared with 31 healthy controls.Results. S-calprotectin and hs-CRP levels were significantly higher in patients with PsA compared with controls (P<0.001andP<0.001, resp.). Patients suffering a polyarthritic disease pattern presented with significantly higher levels of S-calprotectin compared with controls and patients with mono-/oligoarthritis (P<0.001andP=0.017, resp.). The levels of S-calprotectin correlated with hs-CRP (P<0.001;rs=0.441), swollen joint count (P=0.002,rs=0.397), and CXCL10 (P=0.046,rs=0.678) but not with any of the other cytokines evaluated. In multiple logistic regression analysis, S-calprotectin was the only variable significantly associated with psoriatic arthritis (P=0.002,OR=1.006, 95% CI = 1.002–1.010).Conclusion. S-calprotectin and hs-CRP levels were significantly higher in patients with PsA. A polyarthritic disease pattern showed higher levels of S-calprotectin than mono-/oligoarthritis. S-calprotectin is considered a potential marker of disease activity in patients with PsA.
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Leslie, A., and I. Watt. "Polyarthritis." Imaging 11, no. 2 (June 1999): 98–103. http://dx.doi.org/10.1259/img.11.2.110098.

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Dao, Kathryn, and John J. Cush. "Acute polyarthritis." Best Practice & Research Clinical Rheumatology 20, no. 4 (August 2006): 653–72. http://dx.doi.org/10.1016/j.berh.2006.05.007.

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Wiebelitz, K. R., and A. M. Beer. "Chronische Polyarthritis." MMW - Fortschritte der Medizin 152, no. 20 (May 2010): 23. http://dx.doi.org/10.1007/bf03366591.

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Gelsomino, Marco, and Giorgio Tamborrini. "Therapieresistente Polyarthritis." Praxis 103, no. 14 (July 1, 2014): 841–44. http://dx.doi.org/10.1024/1661-8157/a001720.

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Die Differenzialdiagnose einer unklaren Polyarthritis im Alter umfasst u. a. Autoimmunerkrankungen, Kristallablagerungserkrankungen oder infektassoziierte Polyarthritiden. Bei klinischem Verdacht auf eine Gicht sollte durch Harnsäurekristallnachweis in der Synovialflüssigkeit oder im Tophus idealerweise bestätigt werden. Wenn eine Arthrozentese nicht realisierbar ist, können nicht-invasive hochspezifische Untersuchungen, wie der hochauflösenden muskuloskelettale Ultraschall oder eine DECT-Untersuchung hilfreich sein.
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Jones, Hugh W., Patrick Gordon, and Terence Gibson. "Tuberculous Polyarthritis." JCR: Journal of Clinical Rheumatology 2, no. 2 (April 1996): 96–98. http://dx.doi.org/10.1097/00124743-199604000-00007.

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Marshall, John B., and Robert McMurray. "Acute polyarthritis." Postgraduate Medicine 95, no. 8 (June 1994): 165–68. http://dx.doi.org/10.1080/00325481.1994.11945870.

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Peichl, Peter. "Chronische Polyarthritis." DoctorConsult - The Journal. Wissen für Klinik und Praxis 2, no. 3 (November 2011): e167-e170. http://dx.doi.org/10.1016/j.dcjwkp.2011.10.002.

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Dissertations / Theses on the topic "Polyarthriris"

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Rulli, Nestor Ezequiel, and na. "Ross River Virus Infection: Disease Mechanisms and Potential Treatment." University of Canberra. School of Health Sciences, 2007. http://erl.canberra.edu.au./public/adt-AUC20080227.091948.

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Ross River virus (RRV) is a mosquito-borne alphavirus and the aetiological agent of epidemic polyarthritis (EPA). Arthropod borne-Alphaviruses that are related to RRV, such as Chikungunya virus, Sindbis virus and Barmah Forest virus, are usually associated with epidemics of infectious arthritides in different parts of the world. In humans, RRV-induced disease symptoms include fever, rash, myalgia and pain and stiffness of the joints. Muscle and joint pain are the most debilitating symptoms in RRV patients and the best treatment available is non-steroidal anti-inflammatory drugs (NSAID). Previous studies in mice have demonstrated that RRV infection results in inflammation of skeletal muscle and joints and that macrophages play a primary role in disease. The present study was carried out to further elucidate the underlying mechanisms mediating RRV-induced muscle and joint pathology. Previous studies have reported that encephalitic alphaviruses trigger apoptosis of brain cells in mice and that blocking apoptosis reduces mortality rates. In the present study, the ability of RRV to induce muscle apoptosis was investigated in vitro, using a murine myoblast cell line (C1C12), and in vivo, using a mouse model of RRV disease. RRV-infected C1C12 myofibres displayed an array of morphological and biochemical makers of apoptosis. Apoptosis was also observed in the skeletal muscle of RRV-infected C57BL/6J mice. Blocking apoptosis by general caspase inhibition resulted in milder disease symptoms, reduced myofibre damage and decreased inflammation of muscle and joint tissues. The total number of cell infiltrates as well as the number of macrophages infiltrating muscle was significantly reduced by the treatment with a caspase inhibitor. The effects of RRV infection on the skeletal system were also investigated. Primary human osteoblast cells were infected with RRV and monitored for viral-induced cytopathic effect. Osteoblasts supported rapid virus growth and, by 48 hours after infection, succumbed to viral-induced necrosis. In addition, histological examination of bone tissue from RRV-infected C57BL/6J mice showed clear evidence of bone resorption. Tibias from infected mice showed an increased number of activated osteoclasts, a reduction in bone density and thinning of cortical bone. The expression of host factors involved in inflammatory responses and bone remodelling was studied in RRV-infected myofibres and osteoblast cell cultures and in the muscle and joint tissues from infected mice. RRV-infected muscle cells and tissue showed elevated mRNA levels for the chemokines CCL-2, CCL3, CCL5 and CXCL1, all of which are known to mediate the migration of monocytic cells. With the exception of CXCL1, these chemokines were also found to be up-regulated in RRV-infected osteoblast cultures and in joint tissues from infected mice. Muscle and joint tissue from infected mice also showed elevated mRNA levels for type I and type II interferons, TNF- and NOS2. In addition, joint tissues from infected animals contained high levels of IL-6 and IL-1, two cytokines known to mediate bone remodelling. Finally, the therapeutic potential of the drug bindarit was investigated using the mouse model of RRV disease. Bindarit is a known inhibitor of CCL-2 and TNF- and has been found to prevent protein denaturation. Treatment with bindarit resulted in mice developing milder disease symptoms, reduced muscle damage and decreased inflammation of muscle and joint tissues. In particular, bindarit significantly reduced macrophage infiltration into skeletal muscle tissue. This thesis has contributed to the understanding of RRV pathogenesis. It has identified novel mechanisms of RRV-induced muscle and bone pathology and provided further evidence that associate pro-inflammatory host factors to RRV disease. This work has also demonstrated that bindarit should be considered as a candidate for treating RRV disease in humans.
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Hass, Wolfgang. "Soziale Unterstützungsnetzwerke von Menschen mit chronischer Polyarthritis eine explorative, netzwerkanalytische Studie /." [S.l. : s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=967083761.

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Pilhofer, Christoph. "Rückfussarthrodesen bei Polyarthritikern zwischen 1974 und 1997 an der I. Orthopädischen Klinik des BRK Rheuma-Zentrums Bad Abbach /." Aachen : Shaker, 2001. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=009328899&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Goodacre, John A. "Immunopathogenic mechanisms of chronic polyarthritis." Thesis, University of Newcastle Upon Tyne, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241367.

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Denis, Pierre. "Les polyarthrites septiques." Clermont-Ferrand 1, 1988. http://www.theses.fr/1988CLF13005.

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Zeltner, Maria Cristina. "Individuell adaptierter Zahnbürstengriff für Patienten mit chronischer Polyarthritis /." [S.l : s.n.], 1987. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Franklin, Jarrod Peter. "Epidemiology of cancer in patients with inflammatory polyarthritis." Thesis, University of Manchester, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517729.

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FIELDS, PATRICK-PIERRE. "Correlations radio-serologiques des polyarthrites rhumatoides." Aix-Marseille 2, 1988. http://www.theses.fr/1988AIX20491.

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Connolly, Ashley Rex. "Cytokine gene expression in a rat model of polyarthritis /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phc75238.pdf.

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Brasted, Melissa. "Effector CD4Ê T lymphocytes in the prodrome of polyarthritis /." Title page, abstract and table of contents only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phb8239.pdf.

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Thesis (Ph.D.) -- University of Adelaide, Dept. of Molecular Biosciences, 2002.
"October 2001" Amendments (4 leaves) inserted inside back cover. Includes bibliographical references (leaves 215-266).
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Books on the topic "Polyarthriris"

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Mellenthin-Seemann, Ulrike, Friederike Steier, Andrea Schulz, and Heinz-Gerd Biester. Gelenkschutzunterweisung bei Patienten mit chronischer Polyarthritis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-662-07409-1.

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Franke, Martin, ed. Die Basistherapie der chronischen Polyarthritis mit oralem Gold. Heidelberg: Steinkopff, 1986. http://dx.doi.org/10.1007/978-3-642-72380-3.

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Interkultureller Vergleich der Schmerzwahrnehmung und Krankheitsverarbeitung bei türkischen und deutschen Patienten mit chronischer Polyarthritis. Frankfurt am Main: P. Lang, 1996.

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Young, Mark E. Skills-Training bei Borderline- und Posttraumatischer Belastungsstörung. Wien, Austria: Springer, 2005.

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Heilmeyer, L., and W. Müller. Die Serologie der Chronischen Polyarthritis. Springer, 2013.

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Brückle, Wolfgang. Chronische Polyarthritis. Diagnose, Verlauf, Therapien. Urania, Stuttgart, 2001.

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Die Basistherapie Der Chronischen Polyarthritis Mit Oralem Gold. Steinkopff-Verlag Darmstadt, 1986.

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Koharenzgefuhl Und Krankheits-verarbeitung Bei Patientinnen Und Patienten Mit Chronischer Polyarthritis (Europaische Hochschulschriften: Reihe). Peter Lang Publishing, 2004.

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Stacey, Victoria. Musculoskeletal, rheumatology, and wound management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199592777.003.0005.

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Musculoskeletal conditions: introduction - Long-bone anatomy - Fracture descriptions - Open fractures - Compartment syndrome - Upper limb injuries - Lower limb injuries - Rheumatology: introduction - Monoarthritis - Polyarthritis - Septic arthritis - Gonococcal arthritis - Crystal arthropathies - Sero-negative spondyloarthropathies - Rheumatoid arthritis - Acute back pain - Wound management: introduction - Describing wounds and hand injuries - Hand and wrist anatomy - Hand infections - Special hand injuries - Tetanus - SAQs
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Miller, Aaron E., and Teresa M. DeAngelis. Rheumatoid Arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0012.

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Rheumatoid arthritis (RA) is a systemic inflammatory disease that is characterized principally by a polyarthritis, but can result in several neurologic complications involving both the central and peripheral nervous system. In addition, several immunotherapies used to treat RA have been associated with neurological complications. In this chapter, we review the characteristic neurological sequelae of RA as well as the possible neurological consequences of its therapeutic regimens.
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Book chapters on the topic "Polyarthriris"

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Watt, I. "Polyarthritis." In Orthopedic Imaging, 303–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-60295-5_16.

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Fritz, K., J. Raith, and J. Hermann. "MRT bei Polyarthritis." In Magnet-Resonanz-Tomographie, 35–41. Heidelberg: Steinkopff, 2001. http://dx.doi.org/10.1007/978-3-642-57630-0_5.

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Mellenthin-Seemann, Ulrike, Friederike Steier, Andrea Schulz, and Heinz-Gerd Biester. "Die Chronische Polyarthritis." In Rehabilitation und Prävention, 3–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-662-07409-1_2.

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Keck, E., A. Cronenberg, and A. Minkus. "Osteopenien bei chronischer Polyarthritis." In Aktuelle Aspekte der Osteologie, 462–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76766-1_91.

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Felder, M. "Atlassubluxation bei chronischer Polyarthritis." In Kopfschmerzen, 78–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-95561-7_18.

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Sandler, Gerald, and John Fry. "Multiple Painful Joints (Polyarthritis)." In Early Clinical Diagnosis, 69–98. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4147-2_4.

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Morscher, E., and Th Egloff. "Totalprothesenarthroplastik bei chronischer Polyarthritis." In Rheumaorthopädie — Untere Extremität, 70–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-79883-2_7.

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da Silva, José António Pereira, and Anthony D. Woolf. "Chronic, Additive, Symmetrical, Peripheral Polyarthritis." In Rheumatology in Practice, 191–99. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84882-581-9_19.

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da Silva, José António Pereira, and Anthony D. Woolf. "Chronic, Asymmetrical Oligo- Or Polyarthritis." In Rheumatology in Practice, 201–9. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84882-581-9_20.

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Köhler, Helmut, Norbert Mai, and Johannes C. Brengelmann. "Psychologische Schmerztherapie bei chronischer Polyarthritis." In Psychologische Therapie bei chronischen Schmerzpatienten, 139–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70541-0_7.

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Conference papers on the topic "Polyarthriris"

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Cardoso Gomes Zampirolli, Maria Clara, Clarice Garcia Valadares Xavier, Samara de Quadros Lobê, Aurivan Essado Dantas, Matheus Fonseca Cardoso, Maria Fernanda Brandão de Resende Guimarães, and Maria Raquel Costa Pinto. "POLYARTHRITIS AND VAGINAL MELANOMA." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17177.

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CAMPOS, GABRIEL FREITAS DE, HELOISA MARIA LOPES SCARINCI, FABIANA DE CASTRO MACHADO, GABRIEL RIBEIRO LEÃO BARROSO, ALLANA CAMPOS ALVES, DIOGO RIOS DE AVILA, ANDRESSA GABRIELLY RODRIGUES BESERRA, RODRIGO ALMEIDA DE MEDEIROS, and FLAVIO FERNANDES BARBOZA. "DIFFERENTIAL DIAGNOSIS OF POLYARTHRITIS: PARACOCCIDIOIDOMYCOSIS." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-084.

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Bezerra Diniz, Caroline, Leonardo Fernandes e Santana, Mateus de Sousa Rodrigues, and Rita Marina Soares de Castro Duarte. "SEPTIC POLYARTHRITIS BY HAEMOPHILUS INFLUENZAE." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17584.

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Dessein, PH, AE Stanwix, BI Joffe, and EA Shipton. "THU0077 Interations between inflammatory polyarthritis and cardiovascular disease." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.954.

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Ailioaie, C., and LM Ailioaie. "THU0500 Photobiostimulation of chronic cervical pain in juvenile polyarthritis." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.6831.

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Haas, V., K. Sun, C. Ruiz-Liard, and R. Berndt. "Frustrane Behandlung bei pulmonaler und pleuraler Manifestation einer rheumatoiden Polyarthritis." In 27. Jahrestagung der Deutschen Gesellschaft für Thoraxchirurgie. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1668405.

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Fank, K., A. Jasensky, C. Weber, E. Müller, and B. Kohn. "Neopterin als neuer diagnostischer Marker bei immunbedingter Polyarthritis (IMPA) des Hundes?" In 27. Jahrestagung der FG „Innere Medizin und klinische Labordiagnostik“ der DVG (InnLab), 2./3. Februar 2019 in München – Teil 2. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1679123.

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Thanthitaweewat, Vorawut, Amornpun Wongkarnjana, and Naricha Chirakalwasan. "Pancreatitis panniculitis and polyarthritis (PPP) syndrome with Pancreaticopleural fistula: Case Report." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4749.

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Singh, Ankita, Deepti Suri, Prince Jacob, Katta Mohan Girisha, Ankur Jindal, and Surjit Singh. "FRI0578 LACC1 MUTATION IN THREE SIBLINGS WITH POLYARTHRITIS WITHOUT SYSTEMIC MANIFESTATIONS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.6587.

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Brites, Luisa, Mariana Santiago, João Rovisco, João Dinis de Freitas, José Antonio P. Da Silva, Inês Silva, Filipa Teixeira, Daniela Faria, Cândida G. Silva, and Sandra Falcão. "AB1127 ULTRASOUND INTER-READER RELIABILITY OF INFLAMMATORY FINDINGS IN PATIENTS WITH POLYARTHRITIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.2181.

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