Relevant bibliographies by topics / PIG-1

Academic literature on the topic 'PIG-1'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "PIG-1"

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Harmawanto, Agung Gagah, Yosef Cahyo Setianto Poernomo, and Sigit Winarto. "PERENCANAAN ALTERNATIF GEOMETRIK DAN METODE PELAKSANAAN RUAS JALAN NGRAHO – NGAWI STA.14+500 - STA.19+500." Jurnal Manajemen Teknologi & Teknik Sipil 2, no. 2 (November 6, 2019): 179. http://dx.doi.org/10.30737/jurmateks.v2i2.510.

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Ngraho - Ngawi Sta. 14 + 500 – Sta. 19 + 500 highway is a collector connecting Bojonegoro to Ngawi Due to high traffic density, a road geometric Development is in need. The objective of this final project is to plan a good alternative geometric design-safe, comfortable, and easy to access. The required data were of topographic map and the road geometric design was based on the “Tata Cara Perencanaan Geometrik Jalan Antar Kota” General Works Standart No. 038/TBM/1997. The calculations result in Class 2 – lane collector road with one lane of 3 m wide having 8 turns, twists PI1 Spiral-Spiral, PI2 Spiral-Circle-Spiral, PI3 Spiral-Circle-Spiral, PI4 Spiral-Circle-Spiral, PI5 Spiral-Circle-Spiral, PI6 Spiral-Circle-Spiral, PI7 Spiral-Circle-Spiral, and turn PI8 Spiral-Spiral shape PPV PPV 1 concave and 2 convex, concave 3 PPV.Jalan provinsi ruas Ngraho – Ngawi Sta. 14+500 – Sta. 19+500 adalah jalan kolektor yang menghubungkan kota Bojonegoro - Ngawi. Karena lalu lintasnya padat, maka perlu diadakan peningkatan geometrik jalan. Dalam laporan akhir ini penulis membuat perencanaan alternatif desain geometrik jalan yang baik-aman,nyaman, dan mudah diakses oleh pengguna jalan. Data yang digunakan adalah peta topografi dan perencanaan desain geometrik jalan berpedoman oleh “Tata Cara Perencanaan Geometrik Jalan Antar Kota” Standar Bina Marga No.038/TBM/1997. Dari perhitungan diperoleh hasil sebagai berikut: kelas jalan kolektor dengan 2 lajur 1 jalur dan memiliki lebar 3 m, 8 tikungan, tikungan PI1Spiral-Spiral, PI2 Spiral-Circle-Spiral, PI3 Spiral-Circle-Spiral, PI4 Spiral-Circle-Spiral, PI5 Spiral-Circle-Spiral, PI6 Spiral-Circle-Spiral, PI7 Spiral-Circle-Spiral,dan tikunganPI8 Spiral-Spiralbentuk PPV 1 cekung dan PPV 2 cembung, PPV 3 cekung.
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Lerner, Annie B., Emily A. Rice, Mike D. Tokach, Joel M. DeRouchey, Steve S. Dritz, Robert D. Goodband, Jason C. Woodworth, et al. "Effects of space allowance and marketing strategy on growth performance of pigs raised to 165 kg." Translational Animal Science 4, no. 2 (April 1, 2020): 1252–62. http://dx.doi.org/10.1093/tas/txaa065.

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Abstract A total of 976 pigs (PIC 327 × Camborough; PIC, Hendersonville, TN; initially 22.0 ± 1.53 kg body weight [BW]) were used in a 160-d growth study to evaluate the effects of increasing space allowance and varying marketing strategies on growth performance of pigs raised to market weights of ~165 kg. Pens of pigs were blocked by location within the barn and allotted to one of six treatments. Pen served as the experimental unit, and there were eight replicate pens per treatment. The first four treatments consisted of increased initial stocking density and did not utilize topping strategies: (1) 14 pigs/pen (1.17 m2/pig), (2) 17 pigs/pen (0.97 m2/pig), (3) 20 pigs/pen (0.82 m2/pig), and (4) 23 pigs/pen (0.71 m2/pig). The fifth treatment began with 25 pigs/pen (0.66 m2/pig) and had four marketing events with the heaviest 3 pigs/pen removed on day 93, and additional pigs removed to a common inventory of 20 pigs/pen on day 122 and 17 pigs/pen on day 147 with final marketing on day 160. The final treatment began the experiment with 23 pigs/pen (0.71 m2/pig) with three marketing events to achieve a common inventory of 20 pigs/pen on day 108 and 17 pigs/pen on day 147. Pens of pigs were weighed and feed disappearance measured on days 0, 55, 93, 108, 122, 135, 147, and 160. As space allowance decreased from 1.17 to 0.71 m2/pig via increased initial pen inventory (treatments 1 to 4), overall average daily gain (ADG) and average daily feed intake (ADFI) decreased (linear, P < 0.001), while gain:feed ratio (G:F) did not differ (P > 0.05). The treatments with multiple marketing events were compared with each other and with the treatment that began with 0.71 m2/pig and only marketed once at the end of the study. Overall ADG and ADFI were not different (P > 0.05) among these three treatments. Marketing pigs three or four times improved (P < 0.05) G:F compared with the treatment that began the study with 0.71 m2/pig and marketed only once. Reducing floor space allowance for heavy weight pigs decreased intake, which resulted in lower growth rate and final BW, with these reductions occurring before the critical k-value was reached. Total weight gain per pen was maximized with the lowest space allowance and the multiple marketing treatments. Thus, strategic use of pig removals prior to final marketing may allow producers to maximize both number of pigs and total weight marketed through a barn when feeding to heavy weights.
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Lei, H. G., L. Y. Shen, S. H. Zhang, Z. H. Wu, J. Shen, G. Q. Tang, Y. Z. Jiang, et al. "Comparison of the meat quality, post-mortem muscle energy metabolism, and the expression of glycogen synthesis-related genes in three pig crossbreeds." Animal Production Science 55, no. 4 (2015): 501. http://dx.doi.org/10.1071/an13484.

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Post-mortem muscle energy metabolism plays an important role in pork quality. To analyse the differences of meat quality and energy metabolism, three commercial pig crossbreeds frequently used in China were studied, they were DT (Duroc × Taihu; n = 16), PIC (five-way crossbreed from Pig Improvement Co., UK; n = 29) and DLY (Duroc × (Landrace × Yorkshire); n = 19) pigs. The results showed that DT pigs had a higher post-mortem pH45 min and pH24 h, lower shear force and drip loss, higher muscle free-glucose and glycogen contents, and lower lactic acid content than did PIC and DLY pigs. Post-mortem muscle free-glucose content of these three pig crossbreeds changed little, from 45 min to 96 h post-mortem. The expression levels of PRKAG3 (encoding a regulatory subunit of the AMP-activated protein kinase) and GYS1 (encoding muscle glycogen synthase) genes of DT pigs were significantly lower than those of PIC and DLY pigs. DT pigs had a higher expression level of glycogenin-1-like (encoding glycogenin) gene than did PIC and DLY pigs. In conclusion, DT pigs had better meat quality than did the other two pig crossbreeds. We deduced that the post-mortem muscle energy status and metabolism of DT pigs might be an important reason for their good meat quality, and future research should focus on the molecular and physiological mechanism of post-mortem muscle energy metabolism to find ways to improve meat quality.
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Jeong, Dajeong, Hee Sue Park, Seongmin Choi, Heewon Seo, Sung-Min Kim, Kyongok Im, Jiwon Yun, et al. "PNH Clone Size By Flow Cytometry and Its Correlation with PIG Gene Mutation." Blood 132, Supplement 1 (November 29, 2018): 4889. http://dx.doi.org/10.1182/blood-2018-99-115621.

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Abstract Background: Routine diagnosis of paroxysmal nocturnal hemoglobinuria (PNH) is based on flow cytometric measurement of PNH clone in RBC and granulocyte with sensitivity of 1%, and recent introduction of high-sensitive flow cytometry (FCM) makes it possible to detect 0.01% clone. However, the clinical significance of small PNH clone has not been elucidated, so even the necessity to treat or not is controversial. We investigated whether quantitative results of PNH clone size measured by FCM correlate with mutant burden of PIG gene. Methods: A total of 44 specimens from 20 patients whose PNH clone size was >1% in either RBC or granulocyte by routine FCM were enrolled (classical PNH n=10, AA/PNH n=8, MDS/PNH n=2). To detect small quantity of cells with PIG gene mutation, we performed ultra-deep sequencing (average depth 3000X) for PIGA, PIGM, PIGT, and PIGX genes on these 44 consecutive specimens. Results: Sixteen patients (80.0%) were found to harbor PIG gene mutations: 15 patients had PIGA mutation and 1 patient had PIGM mutation. Granulocyte PNH clone size and variant allele frequency (VAF) of PIG gene mutation showed higher correlation (Spearman's r=0.73, p=0.0002) than that of RBC (Spearman's r=0.61, p=0.0073). All patients harboring PIG gene mutation showed more than 10% PNH clone by FCM. In contrast, 4 patients who did not have PIG gene mutation showed less than 10% PNH clone by FCM. Conclusion: Considering the mechanism of PNH development, the presence of PIG gene mutation is a definitive evidence supporting PNH diagnosis. PNH granulocyte clone size more than 10% by FCM seems to be clinically significant in relevance to PIG gene mutation. We suggest that neither high sensitivity FCM nor the detection of PIG gene mutation is a requisite for routine clinical diagnosis of PNH. Disclosures No relevant conflicts of interest to declare.
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Lerner, Annie B., Mike D. Tokach, Joel M. DeRouchey, Steve S. Dritz, Robert D. Goodband, Travis G. O’Quinn, John M. Gonzalez, et al. "PSV-3 Effects of space allowance and marketing strategy on growth performance of pigs raised to heavy market weights." Journal of Animal Science 97, Supplement_2 (July 2019): 156–57. http://dx.doi.org/10.1093/jas/skz122.277.

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Abstract A total of 976 pigs (PIC 327×L42, initially 22 ± 1.5 kg BW) were used in a 160-d study to determine the influence of space allowance and marketing strategy on performance of pigs raised to heavy market weights (165 kg). Pens were blocked by location and allotted to 1 of 6 treatments with 8 pens/treatment. The first four treatments reduced space allowance/pig via initial pen stocking density: 14 pigs/pen (1.20 m2/pig), 17 pigs/pen (0.98 m2/pig), 20 pigs/pen (0.84 m2/pig), or 23 pigs/pen (0.73 m2/pig). The fifth treatment began with 25 pigs/pen (0.67 m2/pig) and the heaviest 3 pigs/pen were removed on d 93, then on d 122 pens were marketed to a common inventory of 20 pigs/pen, and on d 147 marketed to a common pen inventory of 17 pigs/pen. The sixth treatment began with 23 pigs/pen (0.73 m2/pig) and were marketed to a common inventory of 20 pigs/pen on d 108 and marketed to a common inventory of 17 pigs/pen on d 147. Data were analyzed using PROC GLIMMIX with pen as the experimental unit. Overall (d 0 to 160) ADG, ADFI, and final BW decreased (linear, P < 0.001) and G:F increased (quadratic, P = 0.042) as space allowance decreased. When comparing treatments with multiple marketing events (treatments 5 and 6) to treatment 4, there was no evidence for differences (P > 0.05) for overall ADG or ADFI; however, overall G:F was improved (P < 0.05) for pigs initially stocked at 0.67 m2/pig and marketed four times compared to both treatments that initially allowed 0.73 m2/pig, regardless of marketing structure. These results indicate that decreasing space allowance of heavy weight pigs reduces growth, feed intake and final BW, although use of multiple marketing events prior to final marketing may allow for increased number of pigs marketed/pen while balancing reduced growth performance often associated with increased stocking density. http://www.conferenceharvester.com/
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Frank, Jason, Emily Sholtz, Casey Neill, and Jon De Jong. "152 Effects of dietary lactose level on nursery pig performance." Journal of Animal Science 97, Supplement_2 (July 2019): 86–87. http://dx.doi.org/10.1093/jas/skz122.157.

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Abstract Lactose is a critical nutrient in post weaning diets to help pigs transition from sows’ milk to dry feed. The objective of this study was to evaluate the effects of increasing dietary lactose level on nursery pig performance. For this trial 1,080 weaned pigs (PIC 359 x 1050; BW = 6.24 kg; 21 d) were fed 5 lactose programs using a feed budget. Program A = 24, 18, 7%; B = 20, 14, 5%; C =16, 10, 3%; D = 12, 6, 1%; and E = 8, 2, 0% lactose for Phase 1, 2, and 3; respectively. The feed budget for Phase 1 (d 0–7), 2 (d 7–14), and 3 (d 14–20) was 0.91, 3.4, and 4.5 kg/pig; respectively. A common Phase 4 (d 20–48) diet (0% lactose) was fed ad libitum. There was a quadratic response to lactose level in treatments A through E for Phase 1 ADFI (89, 71, 73, 73, 89 g/d; respectively, P = 0.034) and G:F (1.09, 1.33, 1.14, 1.15, 0.91; respectively, P = 0.042). Treatment A through E Phase 1 ADG was 100, 95, 91, 82, and 82 g/d, and Phase 2 ADG (Linear, P = 0.023) was 322, 313, 318, 304, and 295 g/d; respectively. The result was a linear trend for increased BW at the end of Phase 2 (P = 0.10) for treatments A through E (9.21, 9.10, 9.16, 9.00, 8.86 kg; respectively). Although feed cost/pig increased as lactose level increased (Linear, P = 0.041), there was no significant response in margin over feed cost/pig during the overall nursery period for treatments A through E ($15.31, $16.41, $16.22, $15.87, $16.04; respectively). In conclusion, pig performance improved during Phase 1 and 2 with increasing level of dietary lactose. These results confirm previous research showing the importance of dietary lactose in weaned pig diets.
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Cheetham, Sonia, Menira Souza, Tea Meulia, Sheila Grimes, Myung Guk Han, and Linda J. Saif. "Pathogenesis of a Genogroup II Human Norovirus in Gnotobiotic Pigs." Journal of Virology 80, no. 21 (November 1, 2006): 10372–81. http://dx.doi.org/10.1128/jvi.00809-06.

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ABSTRACT We evaluated the gnotobiotic (Gn) pig as a model to study the pathogenesis of human norovirus (HuNoV) and to determine the target cells for viral replication. Sixty-five Gn pigs were inoculated with fecal filtrates of the NoV/GII/4/HS66/2001/US strain or with pig-passaged intestinal contents (IC) and euthanized acutely (n = 43) or after convalescence (n = 22). Age-matched Gn piglets (n = 14) served as mock-inoculated controls. Seventy-four percent (48/65) of the inoculated animals developed mild diarrhea compared to 0 of 14 controls. Pigs from postinoculation days (PID) 1 to 4 tested positive for HuNoV by reverse transcription-PCR of rectal swab fluids (29/65) and IC (9/43) and by antigen (Ag) enzyme-linked immunosorbent assay (ELISA) using antiserum to virus-like particles of HuNoV GII/4. No control pigs were positive. Histopathologic examination showed mild lesions in the proximal small intestine of only one pig (1/7). Seroconversion after PID 21 was detected by antibody ELISA in 13 of 22 virus-inoculated pigs (titers, 1:20 to 1:200) but not in controls. Immunofluorescent microscopy using a monoclonal antibody to HuNoV GII capsid revealed patchy infection of duodenal and jejunal enterocytes of 18 of 31 HuNoV-inoculated pigs with a few stained cells in the ileum and no immunofluorescence (IF) in mock-inoculated controls. Immunofluorescent detection of the viral nonstructural N-terminal protein antigen in enterocytes confirmed translation. Transmission electron microscopy of intestines from HuNoV-inoculated pigs showed disrupted enterocytes, with cytoplasmic membrane vesicles containing calicivirus-like particles of 25 to 40 nm in diameter. In summary, serial passage of HuNoV in pigs, with occurrence of mild diarrhea and shedding, and immunofluorescent detection of the HuNoV structural and nonstructural proteins in enterocytes confirm HuNoV replication in Gn pigs.
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Cemin, Henrique S., Mike D. Tokach, Steve S. Dritz, Jason C. Woodworth, Joel M. DeRouchey, Robert D. Goodband, and Matt W. Allerson. "PSV-9 Effects of insoluble fiber source (cellulose or distillers dried grains with solubles) on growth performance of nursery pigs." Journal of Animal Science 97, Supplement_2 (July 2019): 193–94. http://dx.doi.org/10.1093/jas/skz122.341.

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Abstract A total of 3,171 pigs (PIC 327×L42; initially 5.8 kg) were used in a 39-d study. Treatments were arranged in a 2 × 2 factorial with 0 or 1% cellulose (Arbocel, J. Rettenmaier USA, Schoolcraft, MI) and distillers dried grains with solubles (DDGS; 0 or 5% in phase 1 and 0 or 15% in phase 2). Dietary phases 1 and 2 were offered from d 0 to 10 and 10 to 25, respectively. From d 25 to 39, pigs received a common diet with 25% DDGS. Pens were blocked by weight and allotted to treatments in a randomized complete block design. Experimental unit was two pens (66 pigs) sharing a fence-line feeder with 12 replicates per treatment. Data were analyzed with the GLIMMIX procedure of SAS with block as random effect. From d 0 to 25 and d 0 to 39, there was an interaction (P < 0.05) between cellulose and DDGS for ADG. Pigs fed diets with both DDGS and cellulose had lower ADG than those fed diets without DDGS, with pigs fed diets with DDGS without the addition of cellulose being intermediate. From d 25 to 39, there was a tendency (P = 0.080) for an interaction for ADFI. Pigs previously fed diets without DDGS and with cellulose had higher ADFI than those fed diets with DDGS and cellulose, and pigs previously fed diets without cellulose had similar ADFI regardless of DDGS inclusion. There was a tendency for an interaction (P = 0.070) for pig removals. Adding cellulose to diets without DDGS numerically decreased pig removals, but the inclusion of cellulose to diets with DDGS resulted in increased pig removals. In summary, adding fiber to the diet as cellulose or DDGS resulted in a less pig removals; however, adding both cellulose and DDGS decreased ADG and ADFI.
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Thomas, L. L., R. D. Goodband, J. C. Woodworth, M. D. Tokach, J. M. DeRouchey, and S. S. Dritz. "Effects of space allocation on finishing pig growth performance and carcass characteristics1." Translational Animal Science 1, no. 3 (September 1, 2017): 351–57. http://dx.doi.org/10.2527/tas2017.0042.

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Abstract A total of 405 pigs (PIC 327 × 1,050) were used in 2 experiments (Exp. 1, initially 66.1 ± 1.8 kg BW, Exp. 2 initially 60.8 ± 2.5 kg BW) to examine the effects of space allocation on finishing pig growth performance and carcass characteristics. Pigs were randomly allotted to pens on entry into the finishing facility. Pens of pigs were balanced by initial BW and randomly allotted to 1 of 3 treatments with either 7 or 8 replications per treatment (Exp.1 and 2, respectively). There were 9 pigs per pen and gates were adjusted to provide 0.84, 0.74, or 0.65 m2 per pig. Each pen was equipped with a dry single-sided feeder with two 35.6 cm × 11.4 cm (length × width) feeder spaces and a cup waterer. In both experiments, as space allocation decreased, overall ADG and ADFI decreased (linear, P &lt; 0.019) with no evidence for differences in G:F. In Exp. 2, there was marginal evidence for a linear improvement (P = 0.061) in G:F as space allocation decreased from d 42 to 56. Final BW was 3.8 and 5.3 kg greater (linear, P ≤ 0.005) in Exp. 1 and 2, respectively, when comparing the 0.65 to the 0.84 m2 per pig space allocation treatments. Using a predicted k-value of 0.0336, ADFI and, subsequently, ADG should have begun to decrease when pigs reached 121.2, 101.7, and 83.3 kg at 0.84, 0.74, or 0.65 m2 per pig, respectively. In Exp. 1, we found marginal evidence for a reduction in ADFI as space allocation decreased starting at a mean BW of 80.3 kg (d 14; linear, P = 0.072). In Exp. 2, ADFI and consequently ADG decreased linearly (P &lt; 0.029) starting at a mean BW of 74 kg, as space allocation decreased, before pigs reached the k-value that should have influenced performance. It is unknown if growth performance was impacted for the 0.84 m2 treatment group as this was the greatest space allocation treatment. Overall, these studies indicate that decreasing space allocation resulted in poorer ADG driven by a reduction in ADFI. The data suggests that the accepted k-value of 0.0336 might underestimate the impact of space restriction on finishing pig ADG and ADFI.
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Lammers, Peter J., Chad A. Stahl, and Mark S. Honeyman. "PSII-5 Carcass characteristics and growth performance of market pigs fed reduced lysine diets in bedded hoop barns." Journal of Animal Science 97, Supplement_2 (July 2019): 229–30. http://dx.doi.org/10.1093/jas/skz122.404.

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Abstract A 2 × 2 × 2 factorial design was used to compare the effect of SID Lys:ME concentration (current vs. reduced), stocking density (1.30 vs. 4.05 m2/pig), and harvest month (August vs. March) on pigs raised in bedded hoop barns in Western Iowa. For each harvest month, 420 pigs produced from the mating of Duroc boars (Choice Genetics; West Des Moines, IA) to Camborough females (PIC; Hendersonville, TN) were sorted into 12 pens. Six pens were inside 3 large-scale (9.1 × 18.3 m) hoop barns and were stocked with 64 pigs/pen (32 barrows and 32 gilts; 1.30 m2/ pig). Six pens were inside 3 small-scale (6.0 × 10.8 m) hoop barns and were stocked with 6 pigs/pen (3 barrows and 3 gilts; 4.05 m2/pig). Within each stocking density, pens were randomly assigned to 1 of 2 diets which were fed in 2 phases. Corn-soybean meal diets were formulated to deliver 2.94 or 2.34 g SID Lys per Mcal ME in phase 1 (72.6–95.0 kg) and 2.34 or 1.76 g SID Lys per Mcal ME in phase 2 (> 95.0 kg). Pigs were individually weighed every 28 days and feed disappearance was recorded. When pigs in a pen averaged 129.3 kg the entire pen of pigs were harvested. A single chop (last-rib location; 2.54 cm thick) was collected from each carcass to assess pork quality. Pigs harvested in the summer grew faster, more efficiently, and with more intramuscular fat than those harvested in winter (P-value ≤ 0.05) but had lower 10th rib pH (P-value < 0.0001). Pigs allotted 4.05 m2/pig grew more efficiently but had reduced last rib pH as compared to pigs stocked at 1.30 m2/pig (P-value < 0.05). Reducing SID Lys:ME did not impact growth performance or carcass characteristics (P-value > 0.10). Lower concentrations of SID Lys:ME may be adequate for pigs housed in bedded hoop barns but further study is warranted.
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Dissertations / Theses on the topic "PIG-1"

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Pillay, Santhoshan Thiagaraj. "Pig mucus as an inhibitory agent of HIV-1." Doctoral thesis, University of Cape Town, 2017. http://hdl.handle.net/11427/26949.

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The Human Immunodeficiency Virus (HIV) epidemic still poses a problem with approximately 2 million new infections reported worldwide in 2014. New strategies are required to alleviate this burden. Our laboratory has previously shown that crude saliva and purified mucins from cervical plug mucin, saliva and breast milk inhibit HIV-1 infection in vitro. This project investigates purified mucins sourced from pig and horse mucus, as an alternative and abundant source of material for anti-HIV-1 research. Pig gastric and cervico-vaginal mucus was collected and stirred overnight in 6M guanidine hydrochloride with 10mM Na₂HPO4, 10mM EDTA, 1mM PMSF and 5mM NEM. Gastric and cervicovaginal mucus was purified by density gradient ultracentrifugations in CsCl at 105 000g for 48 hours, twice, and mucin rich fractions were separated by size exclusion column chromatography. Mucin-rich materials eluting in the void volume (V₀) were reduced with 10mM dithithreitol (DTT) or subjected to proteolysis with trypsin. Pig saliva was collected in 0.2M NaCl:0.02% sodium azide and horse saliva (due to its viscous nature) was collected and stirred overnight in 6M guanidine hydrochloride with 10mM Na₂HPO4, 10mM EDTA, 1mM PMSF and 5mM NEM. Pig and horse saliva samples underwent size exclusion column chromatography, where the V₀ fractions of both were purified with one density gradient ultracentrifugation and then dialysed and freeze dried, after which aliquots were treated with either DTT or trypsin. At every stage of purification, lyophilized aliquots of all mucin sources were tested on a luciferase based replication defective HIV neutralization assay on a CD4 expressing HeLa cell line. Luciferase expression quantified as relative light units by a luminometer was used to calculate percentage neutralization. Log dose response curves were constructed to extrapolate the half maximal inhibitory concentrations (IC₅₀) on GraphPad Prism. Samples were tested on an MTT cell toxicity assay. Pig gastric and cervicovaginal mucins were added to a simulated vaginal fluid to make gels (at a concentration of 30mg of mucin per ml of buffer). These gels were tested on the neutralization, MTT assays and the pig gastric mucin gel then underwent particle tracking and nanoparticle diffusion assays at varying pH. Pig gastric and cervicovaginal mucin showed good inhibition and low toxicity, with pig gastric mucin V₀ having the best IC₅₀ (1.668μg/ml). Pig and horse saliva showed inhibition but low cell viability. Pig gastric and cervicovaginal mucin gels exhibited good IC₅₀'s but pig gastric mucin had the best neutralization and lowest toxicity (PGM in Gel Solution 4 IC₅₀: 20.23μg/ml). HIV particle tracking and nanoparticle diffusion assays showed that the pig gastric mucin gel inhibited HIV-1 at low pH and existed as a soft gel. This project shows the efficacy of pig gastric mucin to possibly being a component of an anti-HIV-1 vaginal microbicide.
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Kong, Kok Choi. "Sphingosine 1-phosphate signalling in guinea pig airway smooth muscle." Thesis, University of Strathclyde, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248742.

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Scher, Judy. "mt¦1 melatonin receptor localization in the human and guinea pig retina." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ58867.pdf.

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Pu, Helen Xiaochun. "Cloning and characterization of 11ß-hydroxysteroid dehydrogenase 1 (11ß-HSD1) in the guinea pig." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ30819.pdf.

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Harrison, Andrew Alan. "Characterisation of E-selectin and VCAM-1 expression in models of inflammation in the pig." Thesis, University of London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336283.

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Pierre, Andrew F. "The effect of complement inhibition with soluble complement receptor 1 (sCR1) on pig allo-transplant lung function." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ29290.pdf.

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Foster, Jayne Louise Clinical School Prince of Wales Hospital Faculty of Medicine UNSW. "The microencapsulation and transplantation of fetal pig islet-like cell clusters: a potential therapy for type 1 diabetes." Awarded by:University of New South Wales. Clinical School - Prince of Wales Hospital, 2007. http://handle.unsw.edu.au/1959.4/40715.

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Diabetes can be considered to be one of the main health epidemics of the 21st century. Studies conducted by the World Health Organisation (WHO) indicate that the number of people with diabetes in the year 2000 was 171 million and this is projected to increase to 366 million by 2030 (Wild et al. 2004). The increasing incidence of both Type 1 and Type 2 diabetes is due to population growth, aging, urbanisation, obesity and physical inactivity. The current treatment by insulin injections for individuals with Type 1 diabetes fails to overcome the long term microvascular and macrovascular complications associated with the disease. A major challenge in the treatment of diabetes is to provide patients with an insulin source that is capable of regulating blood glucose levels (BGL) on a minute to minute basis. Advances in medical research have enabled the investigation of a variety of potential alternative therapies that may provide Type 1 diabetic patients with a more superior control of BGL and consequently minimise complications. The utilisation of pancreases obtained from fetal pigs offers potential therapeutic value in the treatment of Type 1 diabetes. Islet-like cell clusters (ICCs) are obtained from such tissue following partial mechanical and enzymatic digestive procedures. ICCs are primarily composed of immature duct cells which, when transplanted, will mainly differentiate into insulin producing ?? cells. Such cells are able to normalise BGL in immunodeficient diabetic recipients and in immunocompetent recipients when anti-rejection drugs are administered. This study investigates microencapsulation as an immunoprotective strategy that has the potential to remove the need for immunosuppression when such cells are transplanted. A review of the literature related to current medical research in the field of diabetes is presented in Chapter 1. In order to achieve the aims of the study, an understanding of how fetal pig ICCs behave when placed within a barium alginate microcapsule both in vitro and in vivo is essential and this data is presented in Chapter 3. This chapter demonstrates that ICCs will survive and differentiate in their typical manner when enclosed within microcapsules and transplanted. Such encapsulated cells will function to normalise BGL when transplanted into diabetic immunodeficent mice for at least 25 weeks and the animals exhibit increased bodyweight. Microcapsules retrieved at this time point were observed to be intact with no breakages or evidence of cellular overgrowth. Transplantation of encapsulated insulin-producing cells into immunocompetent mice are described in Chapter 4. Allotransplantation of a microencapsulated mouse insulin-producing cell line into these diabetic mice also exhibited graft function, resulting in normal BGL in recipients. Large animal experiments are described in Chapter 5. Allotransplantation of microencapsulated fetal pig ICCs into diabetic pig recipients displayed evidence of transient graft function in terms of lower BGL and reduced exogenous insulin requirements. The xenotransplantion of encapsulated fetal pIg ICCs into diabetic immunocompetent mice described in Chapter 4 proved to be more challenging. The transplantation of such cells in this environment did not yield particularly positive results. BGL remained elevated in these recipients and the animals lost bodyweight post transplantation. This area of research warrants further investigation as it is likely that further measures such as transient immunosuppression in combination with microencapsulation will allow fetal pig ICCs to function in a xenograft setting.
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Hidalgo, André Marubayashi. "Fine mapping and single nucleotide polymorphism effects estimation on pig chromosomes 1, 4, 7, 8, 17 and X." Universidade Federal de Viçosa, 2011. http://locus.ufv.br/handle/123456789/4753.

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Mapeamento de loci de caracaterística quantitativas (QTL) geralmente resultam na detecção de regiões genômicas que explicam parte da variação quantitativa da característica. Entretanto essas regiões são muito amplas e não permitem uma acurada identificação dos genes. Dessa forma, torna-se necessário o estreitamento dos intervalos onde os QTL estão localizados. Com a seleção genômica ampla (GWS), foram desenvolvidas ferramentas estatísticas de forma a se estimar os efeitos de cada marcador. A partir dos valores desses efeitos, pode-se analisar quais são os marcadores de maiores efeitos. Assim, objetivou-se realizar o mapeamento fino dos cromossomos suínos 1, 4, 7, 8, 17, e X, usando marcadores microsatélites e polimorfismo de base única (SNP), em uma população F2 produzida pelo cruzamento de varrões da raça naturalizada brasileira Piau com fêmeas comerciais, associados com características de desempenho, carcaça, orgãos internos, cortes e qualidade de carne. Também objetivou-se estimar os efeitos dos marcadores SNP nas características que tiveram QTL detectados, analisar quais são os mais expressivos e verificar se eles estão localizados dentro do intervalo de confiança do QTL. Os QTL foram identificados por meio do método regressão por intervalo de mapeamento e as análises foram realizadas pelo software GridQTL. O efeito de cada marcador foi estimado pela regressão de LASSO Bayesiano, usando o software R. No total, 32 QTL foram encontrados ao nível cromossômico de significância de 5%, destes, 12 eram significativos ao nível cromossômico de 1% e 7 destes eram significativos ao nível genômico de 5%. Seis de sete QTL apresentaram marcadores de efeito expressivo dentro do intervalo de confiança do QTL. Resultados deste estudo confirmaram QTL de outros trabalhos e identificaram vários outros novos. Os resultados encontrados utilizando marcadores microsatélites junto com SNPs aumentaram a saturação do genoma levando a um menor intervalo de confiança dos QTL encontrados. Os métodos usados foram importantes para estimar os efeitos dos marcadores, e também para localizar aqueles com efeitos mais expressivos dentro do intervalo de confiança do QTL, validando os QTL encontrados pelo método da regressão.
Quantitative Trait Loci (QTL) mapping efforts often result in the detection of genomic regions that explain part of the quantitative trait variation. However, these regions are very large and do not allow accurate gene identification, hence the interval must be narrowed where the QTL was located. With the genome wide selection (GWS), many statistical tools have been developed in order to estimate the effects for each marker. With the marker effects values it is possible to analyze which markers have large effects. Hence, the objective of this investigation was to fine map pig chromosomes 1, 4, 7, 8, 17 and X, using microsatellites and SNP markers, in a F2 population produced by crossing naturalized Brazilian Piau boars with commercial females, associated with performance, carcass, internal organs, cut yields and meat quality traits. A further aim was to estimate the effects of single nucleotide polymorphism (SNP) markers on traits with detected QTL, analyze the most expressive ones and verify whether the markers with larger effects were indeed within the QTL confidence interval. QTL were identified by regression interval mapping using the GridQTL software. Individual marker effects were estimated by Bayesian LASSO regression using the R software. In total, 32 QTL for the studied traits were significant at the 5% chromosome-wide level, including 12 significant QTL at the 1% chromosome-wide level and 7 significant at the 5% genome-wide level. Six out of seven QTL with genome-wide significance had markers of large effect within their confidence interval. These results confirmed some previous QTL and identified numerous novel QTL for the investigated traits. Our results have shown that the use of microsatellites and SNP markers that increase the genome saturation lead to QTL of smaller confidence intervals. The methods used were also valuable to estimate the marker effects and to locate the most expressive markers within the QTL confidence interval, validating those QTL found by the regression method.
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Santangelo, Kelly Susan. "Determining the role of interleukin-1β in the Hartley guinea pig model of primary osteoarthritis." The Ohio State University, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=osu1299649441.

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Wei, Hai [Verfasser], and Barbara [Akademischer Betreuer] Conradt. "pig-1 MELK and ced-3 Caspase cooperate to control cell polarity in the C. elegans NSM neuroblast / Hai Wei ; Betreuer: Barbara Conradt." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2019. http://d-nb.info/1188564161/34.

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Books on the topic "PIG-1"

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Scarry, Richard. Richard Scarry's Pig Will: 2-in-1 turn-around books. [New York]: Random House, 1990.

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Scher, Judite. mt(1) melatonin receptor localization in the human and guinea pig retina. Ottawa: National Library of Canada, 2001.

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Legislation, Great Britain Parliament House of Commons Third Standing Committee on Delegated. Draft pig industry development scheme 2000: Confirmation order 2001, Thursday 1 March 2001. London: Stationery Office, 2001.

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Sheppard, A. The structure of pig production in England and Wales: The results of the National Survey of Pig Production Systems, 1 February 1996. Exeter: University of Exeter, Agricultural Economics Unit, 1996.

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EAAP-Symposium of the Commission on Pig Production (1988 Helsinki, Finland). New techniques in pig carcass evaluation: Proceedings of the EAAP-Symposium of the Commission on Pig Production, Helsinki, Finland, 1 July 1988. Wageningen: Pudoc, 1989.

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Cymerman, Zbigniew. Strefa kontaktu kaczawskiego kompleksu strukturalnego z depresja ̨Świebodzic (Sudety Środkowe) w świetle nowych otworów badawczych Cieszów PIG 1 i Cieszów PiG 2: The contact zone between the Kaczawa Structural Complex and Świebodzice Depressions (Central Sudetes, SW Poland) in the light of the new research boreholes of Cieszów PIG 1 and Cieszów PIG 2. Warszawa: Państwowy Instytut Geologiczny--Państwowy Instytut Badawczy, 2014.

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Pierre, Andrew F. The effect of complement inhibition with soluble complement receptor 1 (sCR1) on pig allo-transplant lung function. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Australasian Pig Science Association. Biennial Conference. Manipulating pig production 4: Proceedings of the Fourth Biennial Conference of the Australasian Pig Science Association (APSA) held in Canberra, ACT on November 28 to December 1, 1993. Edited by Batterham E. S. 1944-. Attwood, Vict: The Association, 1993.

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Conference, Australasian Pig Science Association Biennial. Manipulating pig production 5: Proceedings of the Fifth Biennial Conference of the Australasian Pig Science Association (APSA) held in Canberra, ACT on November 26 to 29, 1995 1, 1993. Werribee, Vict: The Association, 1995.

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Trivizas, Eugenios. The three little wolves and the big bad pig. New York: Margaret K. McElderry Books, 1993.

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Book chapters on the topic "PIG-1"

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Farmer, C., and S. A. Edwards. "1. The neonatal pig: developmental influences on vitality." In The suckling and weaned piglet, 9–39. The Netherlands: Wageningen Academic Publishers, 2020. http://dx.doi.org/10.3920/978-90-8686-894-0_1.

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Matran, Régis, Kjell Alving, Anette Hemsen, and Jan M. Lundberg. "Endothelin-1 Increases Airway Mucosa Blood Flow in the Pig." In Mediators in Airway Hyperreactivity, 237–41. Basel: Birkhäuser Basel, 1990. http://dx.doi.org/10.1007/978-3-0348-7379-6_31.

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Babinszky, L., M. W. A. Verstegen, and W. H. Hendriks. "1: Challenges in the 21st century in pig and poultry nutrition and the future of animal nutrition." In Poultry and pig nutrition, 17–37. The Netherlands: Wageningen Academic Publishers, 2019. http://dx.doi.org/10.3920/978-90-8686-884-1_1.

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Martins, C., F. S. Boinas, L. Iacolina, F. Ruiz-Fons, and D. Gavier-Widén. "1. African swine fever (ASF), the pig health challenge of the century." In Understanding and combatting African Swine Fever, 11–24. The Netherlands: Wageningen Academic Publishers, 2021. http://dx.doi.org/10.3920/978-90-8686-910-7_1.

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Carr, John, Shih-Ping Chen, Joseph F. Connor, Roy Kirkwood, and Joaquim Segalés. "Clinical Examination." In Pig Health, 1–52. Boca Raton : CRC Press, [2018]: CRC Press, 2018. http://dx.doi.org/10.1201/9781315157061-1.

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Hannon, Bruce, and Matthias Ruth. "Pig Cycle." In Dynamic Modeling, 338–47. New York, NY: Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4613-0211-7_34.

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Gooch, Jan W. "Pig Wrack." In Encyclopedic Dictionary of Polymers, 536. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_8728.

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Flink, Linus Girdland. "Pig: Domestication." In Encyclopedia of Global Archaeology, 5938–41. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4419-0465-2_2213.

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Cottage, Christopher T., Balaji Sundararaman, Shabana Din, Nirmala Hariharan, and Mark A. Sussman. "Pim-1." In Encyclopedia of Signaling Molecules, 4012–16. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_344.

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Donato, Dominique M., Steven K. Hanks, Kenneth A. Jacobson, M. P. Suresh Jayasekara, Zhan-Guo Gao, Francesca Deflorian, John Papaconstantinou, et al. "Pim-1." In Encyclopedia of Signaling Molecules, 1420–24. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_344.

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Conference papers on the topic "PIG-1"

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Ma, Yanying, Xiuzhen Li, and Xue Wang. "Based on the Gray GM (1, 1) Prediction Model of Pig disease." In 6th International Conference on Electronic, Mechanical, Information and Management Society. Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/emim-16.2016.322.

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TODA, HIROYUKI, WEI-GUANG DING, FUTOSHI TOYODA, YO YASUDA, MAKOTO ITO, MINORU HORIE, and HIROSHI MATSUURA. "POTENTIATION OF IKs POTASSIUM CURRENT IN GUINEA-PIG VENTRICULAR MYOCYTES BY SPHINGOSINE-1-PHOSPHATE." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0071.

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Kodrich, L., P. Porterie, O. Lago, G. Bergonzelli, B. Sassetti, and J. C. Sanchez avalos. "MINI PLASMINOGEN-LIKE MOLECULE IN SEPTIC PATIENTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644693.

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We have previously described (Thromb.Res.44(6),1986) an altered relation Plasminogen (Pig)/α2~antiplasmin (APL) (Plg/APL<0.6) in the plasma of septic patients. A probable explanation of the mechanism whereby this alteration takes place would be the degradation of Pig to fragments of lower molecular weight due to the action of leukocyte elastase.In order to confirm this we studied 10 patients with sepsis, which did not have clinicalorlaboratory evidence of disseminated intravascular coagulation or septic shock, with positive blood cultures for bacterial germs .Elastase-α1proteinase inhibitor complexes were measured by an enzyme-linked immunosorbent assay (mean:510±181.9ug/l;normal range:86±28.5ug/l). Pig and APL functional activities were assayed by the amidolytic method; Pig: 40±8.9%; normal range: 100±20% .APL: 95±10.1% formal range 100+20%. Two different behaviors were observed in the plasma Pig of these patients with regard to their capacity to bind to Lysi-ne-Sepharose 4B.0n the basis of this observation the patients were divided into two group.Group A(4 patients) only presented Pig activity in fraction 1 (Pig without lysine binding sites : LBS). Group B (6 patients) presented Pig activity in fraction 1 and in fraction 2 (Pig with LBS).The normal controls presented Pig activity only in fraction 2. All the fractions which presented functional Pig activity also presented immunologic Pig activity and developed areas of lysis in heated fibrin plates after activation with urokinaseIt seems tenable the hypothesis that the action of the leukocyte elastase is responsible for the degradation of Pig and this modification in the molecule would give rise to a greater depuration thus explaining the marked drop of the plasmatic levels seen in septic patients.
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Cai, Xu, Yu Chen, and Jian Kang. "Altered pulmonary capillary permeability in immunosuppressed guinea pig infected with serum type 1 Legionella pneumophila." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa5450.

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Zheng, Ying-min, Hong-yi Yao, Jiang-ping Zhu, Xin-wei Dong, and Qiang-min Xie. "Mugwort Pollen Extract Or Recombinant Art V 1 Induced Guinea Pig Model Of Allergic Rhinitis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4211.

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Yates, Keegan, Elizabeth Fievisohn, Warren Hardy, and Costin Untaroiu. "Development and Validation of a Göttingen Miniature Pig Brain Finite Element Model." In ASME 2016 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/detc2016-60217.

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The Center for Disease Control and Prevention reports that there are approximately 1.4 million emergency department visits, hospitalizations, or deaths per year in the USA due to traumatic brain injuries (TBI) [1]. In order to lessen the severity or prevent TBIs, accurate dummy models, simulations, and injury risk metrics must be used. Ideally, these models and metrics would be designed with the use of human data. However, available human data is sparse, so animal study data must be applied to the human brain. Animal data must be scaled before it can be applied, and current scaling methods are very simplified. The objective of our study was to develop a finite element (FE) model of a Göttingen mini-pig to allow study of the tissue level response under impact loading. A hexahedral FE model of a miniature pig brain was created from MRI images. The cerebrum, cerebellum, corpus callosum, midbrain, brainstem, and ventricles were modeled and assigned properties as a Kelvin-Maxwell viscoelastic material. To validate the model, tests were conducted using mini-pigs in an injury device that subjected the pig brain to both linear and angular motion. These pigs are commonly used for brain testing because the brains are well developed with folds and the material properties are similar to human brain. The pigs’ brains were embedded with neutral density radio-opaque markers to track the motion of the brain relative to the skull with a biplanar X-ray system. The impact was then simulated, and the motion of nodes closest to the marker locations was recorded and used to optimize material parameters and the skull-brain interface. The injuries were defined at a tissue level with damage measures such as cumulative strain damage measure (CSDM). In future the animal FE model could be used with a human FE model to determine an accurate animal-to-human transfer function.
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Lopes, G. V., G. B. Michael, S. Schwarz, and M. Cardoso. "Antimicrobial resistance and class 1 integrons in Salmonella enterica subsp. enterica serovar Derby isolates from pig abattoirs." In 10th International Conference on the Epidemiology and Control of Biological, Chemical and Physical Hazards in Pigs and Pork. Iowa State University, Digital Press, 2013. http://dx.doi.org/10.31274/safepork-180809-941.

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Pastore, Fiorella, Nadia Moretto, Fabrizio Facchinetti, and Riccardo Patacchini. "Non-Neuronal Transient Receptor Potential Ankyrin 1 (TRPA1) Is Involved In Ovalbumin-Evoked Contraction Of Guinea-Pig Trachea." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4297.

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Safholm, J., K. Stark, L. Cardell, S. Dahlen, and M. Adner. "EP1Receptor Mediates the Intrinsic Tone in Guinea-Pig Trachea Via Secretion of PGE2Generated from COX-1 and COX-2." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6323.

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Jansen, J. W. C. M. "EFFECTS OF INHIBITORS ON COLLAGEN INDUCED PLATELET AGGREGATION IN SIX DIFFERENT SPECIES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643445.

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One approach to the development of antithrombotics is inhibition of platelet aggregation. The pharmacological approach often used is to test compounds on collagen induced platelet aggregation measured in platelet rich plasma. Therefore we have compared inhibitors with different mechanism of action on aggregation of platelets from six different species commonly used in pharmacological studies. Aggregation was induced with submaximal amounts of collagen (Hormone Chemie).Inhibitors of the cyclooxygenase system, aspirin and indomethacin, were very potent in inhibiting aggregation of platelets from humans guinea pig and dog (IC50 20-60 and 1-3 ¼M resp.). Aggregation of pig and rat platelets was poorly inhibited by both of these compounds (IC5: 700-900 ¼M), whereas platelets from mice showed intermediate sensitivety (IC50 ca.100 ¼M).The combined lipoxygenase/cyclooxygenase inhibitor BW755C, was extremely active on platelets of guinea pig (IC50 1 ¼M) and was poorly active in mice platelets (IC50 300 ¼M). In the other species the inhibitory activity ranged from 20-80 ¼M.The phosphodiesterase inhibitors, papaverine and BL3459 inhibited aggregation in all species (IC50 50-100 and 1-5 ¼M resp.). Dipyridamole inhibited aggregation also in all species but with lower activity (IC50 > 100 ¼M).Conclusion: remarkable species differences are present with respect to inhibition of collagen induced platelet aggregation by the various compounds e.g. rat and porcine platelet aggregation was hardly inhibited by cyclooxygenase inhibitors. The effects of the compounds on human platelets are comparable to the effects on canine plateletes.
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Reports on the topic "PIG-1"

1

test, test. NO PII 1. Office of Scientific and Technical Information (OSTI), June 2018. http://dx.doi.org/10.2172/1440289.

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Stovall, Kevin. Pit Surveillance (Rev -1). Office of Scientific and Technical Information (OSTI), May 2022. http://dx.doi.org/10.2172/1868192.

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Burchell, Timothy. AGC-1 PIE Experimental Plan. Office of Scientific and Technical Information (OSTI), March 2012. http://dx.doi.org/10.2172/1550773.

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Hunn, John D., Robert Noel Morris, Charles A. Baldwin, Fred C. Montgomery, and Tyler J. Gerczak. PIE on Safety-Tested AGR-1 Compact 5-1-1. Office of Scientific and Technical Information (OSTI), August 2015. http://dx.doi.org/10.2172/1213349.

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Cummings, D. I. Stop 1-2A: Regimbald Road Pit. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2011. http://dx.doi.org/10.4095/289561.

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Hunn, John D., Robert Noel Morris, Charles Baldwin, Fred C. Montgomery, and Chinthaka Silva. PIE on Five Irradiated AGR-1 Compacts. Office of Scientific and Technical Information (OSTI), September 2012. http://dx.doi.org/10.2172/1633150.

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Meyer, Maria, and Nancy S. Magnuson. Pim-1, A Potential Participant in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, July 2001. http://dx.doi.org/10.21236/ada396716.

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Spalding, B. P., M. A. Bogle, S. R. Cline, M. T. Naney, and B. Gu. In situ vitrification demonstration at Pit 1, Oak Ridge National Laboratory. Volume 2: Site characterization report of the Pit 1 area. Office of Scientific and Technical Information (OSTI), December 1997. http://dx.doi.org/10.2172/564047.

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Wendelberger, James G. Pit and Crack Detection Summary Report FY16_DE05_SW_C2_Zone 1-2_Section_a_cTOP10. Office of Scientific and Technical Information (OSTI), May 2019. http://dx.doi.org/10.2172/1514905.

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Gel, Aytekin, Avinash Vaidheeswaran, and Mary Clarke. Deterministic Calibration of MFiX-PIC, Part 1: Settling Bed. Office of Scientific and Technical Information (OSTI), February 2021. http://dx.doi.org/10.2172/1764832.

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