Relevant bibliographies by topics / Phytoestrogens

Academic literature on the topic 'Phytoestrogens'

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Published: 4 June 2021
Last updated: 31 August 2024

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Journal articles on the topic "Phytoestrogens"

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Wyse, Jessica M., Sajid Latif, Saliya Gurusinghe, Erica D. Berntsen, Leslie A. Weston, and Cyril P. Stephen. "Characterization of Phytoestrogens in Medicago sativa L. and Grazing Beef Cattle." Metabolites 11, no. 8 (August 20, 2021): 550. http://dx.doi.org/10.3390/metabo11080550.

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Phytoestrogens are plant-produced bioactive secondary metabolites known to play an integral role in plant defense that frequently accumulate in times of stress and/or microbial infection. Phytoestrogens typically belong to two distinct chemical classes; flavonoids (isoflavones) and non-flavonoids (lignans and coumestans). Upon consumption by livestock, high concentrations of phytoestrogens can cause long-term disruption in reproduction due to structural similarities with mammalian estrogens and their tendency to bind estrogen receptors. Wide variation in phytoestrogen concentration has been reported in pasture legumes and corresponding silage or hay. Lucerne is a common perennial pasture legume in temperate climates, but information on phytoestrogen production or accumulation in grazing livestock is currently limited. Therefore, metabolic profiling using UHPLC-MS-QToF was performed to identify and quantitate key phytoestrogens in both fresh and dried lucerne fodder from replicated field or controlled glasshouse environments. Phytoestrogens were also profiled in the blood plasma of Angus cattle grazing field-grown lucerne. Results revealed that phytoestrogens varied quantitatively and qualitatively among selected lucerne cultivars grown under glasshouse conditions. Fresh lucerne samples contained higher concentrations of coumestans and other phytoestrogenic isoflavones than did dried samples for all cultivars profiled, with several exceeding desirable threshold levels for grazing cattle. Coumestans and isoflavones profiled in plasma of Angus heifers grazing lucerne increased significantly over a 21-day sampling period following experimental initiation. Currently, threshold concentrations for phytoestrogens in plasma are unreported. However, total phytoestrogen concentration exceeded 300 mg·kg−1 in fresh and 180 mg·kg−1 in dried samples of selected cultivars, suggesting that certain genotypes may upregulate phytoestrogen production, while others may prove suitable sources of fodder for grazing livestock.
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Lojza, J., V. Schulzová, and J. Hajšlová. "Changes of phytoestrogens daidzein, genistein and their glycosides daidzin and genistin and coumestrol during processing of soyabeans." Czech Journal of Food Sciences 22, SI - Chem. Reactions in Foods V (January 1, 2004): S223—S226. http://dx.doi.org/10.17221/10666-cjfs.

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Phytoestrogens represent biologically active compounds showing estrogenic activity similar to that of sex hormones – estrogens. Various adverse effects such as sterility, increase of females’ genitals, lost of males’ copulation activity, etc. were observed in farm animals after exposure to higher amounts of fodder containing phytoestrogens. On the other side, their presence in human diet is nowadays the object of many research studies concerned with prevention of breast and prostate cancer, osteoporosis and other hormone-linked diseases by dietary intake of phytoestrogens. Soya (Glycine max) is one of the main sources of these compounds in diet. Isoflavones daidzein and genistein occurring either free or bound in glycosides are the main phytoestrogens in this food crop. Coumesterol representing coumestans is another effective phytoestrogen contained in some eddible plants. In the first part of our study, analytical method for determination of free and total phytoestrogens was developed and validated. Following steps are included: (i) acid hydrolysis (only for “total phytoestrogens” analysis), (ii) extraction with methanol/water mixture, (iii) SPE preconcentration; (iv) identification/quantification using HPLC/DAD/FLD. The aim of present study was to document the fate of phytoestrogens and their forms during household/industrial processing. As documented in our experiments the most dynamic changes of phytoestrogen levels occur during soyabeans sprouting. High levels of coumestrol even exceeding other phytoestrogens were detected on this occasion.
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Chaboki, Hamid Reza, Farideh Akbarian, and Hossein Kazemi Mehrjerdi. "Isoflavones Potentials for the Treatment of Osteoporosis: An Update on In-vivo Studies." Journal of Lab Animal Research 1, no. 1 (December 25, 2022): 20–25. http://dx.doi.org/10.58803/jlar.v1i1.10.

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In plant-derived compounds, phytoestrogens are biologically active substances that exhibit various estrogenic and antiestrogenic effects. With the increasing prevalence of osteoporosis among older women caused by estrogen deficiency, identifying natural substances that can potentially treat the disease is of utmost significance. This review study aimed to explore how phytoestrogen metabolites mimic mammalian estrogens and prevent bone loss following menopause. Phytoestrogens derived from plants have gained considerable attention due to their similarity to mammalian estrogens and lower impact on sensitive tissues, such as the uterus and breasts. One well-established approach to simulate postmenopausal conditions is by using ovariectomized rats or mice (OVX). The administration of phytoestrogens in the OVX murine model has inhibited osteoclast differentiation, activation, and Pyridinoline secretion. Furthermore, these compounds have been shown to enhance bone formation and increase bone mineral density and the expression levels of various osteoblast markers, such as alkaline phosphatase, osteocalcin, osteopontin, and alpha-1 collagen. Several natural phytoestrogen compounds in plants possess a chemical structure akin to 17 beta-estradiol, a steroid hormone. In postmenopausal women with osteoporosis, isoflavones, a type of phytoestrogen, can potentially treat the disease by binding to estrogen receptors on the surface of target cells. Mechanistic investigations have demonstrated that phytoestrogens can retard bone resorption and promote bone formation. Novel approaches in phytoestrogen research could involve investigating the synergistic effects of combining different phytoestrogen compounds, exploring their interactions with other signaling pathways, or assessing their effects on various bone types. Furthermore, identifying novel sources of phytoestrogens could lead to the discovery of new compounds with potent osteoprotective effects.
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Weber, KS, KD Setchell, DM Stocco, and ED Lephart. "Dietary soy-phytoestrogens decrease testosterone levels and prostate weight without altering LH, prostate 5alpha-reductase or testicular steroidogenic acute regulatory peptide levels in adult male Sprague-Dawley rats." Journal of Endocrinology 170, no. 3 (September 1, 2001): 591–99. http://dx.doi.org/10.1677/joe.0.1700591.

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Nutritional factors, especially phytoestrogens, have been extensively studied for their potential beneficial effects against hormone-dependent and age-related diseases. The present study describes the short-term effects of dietary phytoestrogens on regulatory behaviors (food/water intake, locomotor activity and body weight), prostate weight, prostate 5alpha-reductase enzyme activity, reproductive hormone levels, and testicular steroidogenic acute regulatory peptide (StAR) levels in adult Sprague-Dawley rats. Animals were fed either a phytoestrogen-rich diet containing approximately 600 microg/g isoflavones (as determined by HPLC) or a phytoestrogen-free diet. After 5 weeks of consuming these diets, plasma phytoestrogen levels were 35 times higher in animals fed the phytoestrogen-rich vs phytoestrogen-free diets. Body and prostate weights were significantly decreased in animals fed the phytoestrogen-rich diet vs the phytoestrogen-free fed animals; however, no significant change in prostate 5alpha-reductase enzyme activity was observed between the treatment groups. Locomotor activity levels were higher in the phytoestrogen-rich vs the phytoestrogen-free animals during the course of the treatment interval. Plasma testosterone and androstenedione levels were significantly lower in the animals fed the phytoestrogen-rich diet compared with animals fed the phytoestrogen-free diet. However, there were no significant differences in plasma LH or estradiol levels between the diet groups. Testicular StAR levels were not significantly different between the phytoestrogen-rich vs the phytoestrogen-free fed animals. These results indicated that consumption of dietary phytoestrogens resulting in very high plasma isoflavone levels over a relatively short period can significantly alter body and prostate weight and plasma androgen hormone levels without affecting gonadotropin or testicular StAR levels. The findings of this study identify the biological actions of phytoestrogens on male reproductive endocrinology and provide insights into the protective effects these estrogen mimics exert in male reproductive disorders such as benign prostatic hyperplasia and prostate cancer.
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Fang, Zhiwu, Scott H. Carlson, Y. F. Chen, S. Oparil, and J. Michael Wyss. "Estrogen depletion induces NaCl-sensitive hypertension in female spontaneously hypertensive rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 281, no. 6 (December 1, 2001): R1934—R1939. http://dx.doi.org/10.1152/ajpregu.2001.281.6.r1934.

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In women, arterial pressure generally increases after menopause, but several studies suggest that women who eat large amounts of plant estrogens (phytoestrogens) experience a slower rise in the incidence of postmenopausal hypertension. This suggests that both ovarian hormones (principally estrogen) and phytoestrogens may protect at least some women from hypertension. The present study tests the hypothesis that phytoestrogens blunt hypertension in estrogen-depleted female spontaneously hypertensive rats (SHR). Three-week-old ovariectomized SHR were fed one of four diets that contained basal (0.6%) or high (8%) NaCl with or without dietary phytoestrogens for 9 wk. In SHR on the basal NaCl diet, arterial pressure was unaffected by the removal of dietary phytoestrogens. In contrast, in SHR on the high-NaCl diet, arterial pressure was significantly higher in rats on the phytoestrogen-free (204 ± 4 mmHg) compared with the phytoestrogen-replete (153 ± 4 mmHg) diet. Ganglionic blockade resulted in reductions in arterial pressure that were directly related to the dietary NaCl-induced increases in arterial pressure. Together, these data indicate that dietary phytoestrogens protect ovariectomized female SHR from dietary NaCl-sensitive hypertension and that the sympathetic nervous system plays an important role in this effect. Furthermore, these results demonstrate that dietary phytoestrogens can have a major impact on the interpretation of studies into the physiological role of estrogen in females.
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Viggiani, Maria Teresa, Lorenzo Polimeno, Alfredo Di Leo, and Michele Barone. "Phytoestrogens: Dietary Intake, Bioavailability, and Protective Mechanisms against Colorectal Neoproliferative Lesions." Nutrients 11, no. 8 (July 24, 2019): 1709. http://dx.doi.org/10.3390/nu11081709.

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Phytoestrogens are natural substances that have been extensively studied for their beneficial effect on human health. Herein, we analyzed the data of the literature on the role of phytoestrogens in the prevention of colorectal neoproliferative lesions (CNL). Both in vitro and in vivo studies suggest that the beneficial effects of phytoestrogens on CNL mainly depend on their ability to bind estrogen receptor beta (ERβ) in the intestinal mucosa and counter ER-alpha (ERα) activity. Epidemiological data demonstrate a correlation between the low prevalence of CNL in Eastern populations and the consumption of soy products (phytoestrogen-enriched diet). However, both observational and interventional studies have produced inconclusive results. In our opinion, these discrepancies depend on an inadequate evaluation of phytoestrogen intake (dietary questionnaires were not aimed at establishing phytoestrogen intake) and absorption (depending mainly on the intestinal microbiota of the analyzed subjects). For this reason, in the present review, we performed an overview of phytoestrogen dietary intake and metabolism to offer the reader the opportunity for a better interpretation of the literature. Future prospective trials focusing on the protective effect of phytoestrogens against CNL should take into account both their dietary intake and absorption, considering the effective role of the intestinal microbiota.
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Tubbs, Christopher, Phillip Hartig, Mary Cardon, Nicole Varga, and Matthew Milnes. "Activation of Southern White Rhinoceros (Ceratotherium simum simum) Estrogen Receptors by Phytoestrogens: Potential Role in the Reproductive Failure of Captive-Born Females?" Endocrinology 153, no. 3 (March 1, 2012): 1444–52. http://dx.doi.org/10.1210/en.2011-1962.

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The captive southern white rhinoceros (SWR; Ceratotherium simum simum) population serves as an important genetic reservoir critical to the conservation of this vulnerable species. Unfortunately, captive populations are declining due to the poor reproductive success of captive-born females. Captive female SWR exhibit reproductive problems suggested to result from continual ovarian follicular activity and prolonged exposure to endogenous estrogen. However, we investigated the potential role of exogenous dietary phytoestrogens in the reproductive failure of SWR by cloning and characterizing in vitro phytoestrogen binding and activation of recombinant SWR estrogen receptors (ESR). We compared those characteristics with recombinant greater one-horned rhinoceros (GOHR; Rhinoceros unicornis) ESR, a species that receives similar captive diets yet reproduces relatively well. Our results indicate that phytoestrogens bind rhino ESR in a manner similar to other vertebrate species, but there are no differences found in phytoestrogen binding affinity of SWR ESR compared with GOHR ESR. However, species-specific differences in ESR activation by phytoestrogens were detected. The phytoestrogen coumestrol stimulated greater maximal activation of SWR ESR1 than GOHR ESR1. SWR ESR2 were also more sensitive to phytoestrogens and were activated to a greater extent by both coumestrol and daidzein. The concentrations in which significant differences in ESR activation occurred (10−7 to 10−5m) are consistent with circulating concentrations measured in other vertebrate species. Taken together, these findings suggest that phytoestrogens potentially pose a risk to the reproductive health of captive SWR. However, additional studies are needed to further clarify the physiological role of dietary phytoestrogens in the reduced fertility of this species.
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Solopov, Pavel, Ruben Manuel Luciano Colunga Biancatelli, Christiana Dimitropoulou, and John D. Catravas. "Dietary Phytoestrogens Ameliorate Hydrochloric Acid-Induced Chronic Lung Injury and Pulmonary Fibrosis in Mice." Nutrients 13, no. 10 (October 14, 2021): 3599. http://dx.doi.org/10.3390/nu13103599.

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We previously reported that female mice exhibit protection against chemically induced pulmonary fibrosis and suggested a potential role of estrogen. Phytoestrogens act, at least in part, via stimulation of estrogen receptors; furthermore, compared to residents of Western countries, residents of East Asian countries consume higher amounts of phytoestrogens and exhibit lower rates of pulmonary fibrosis. Therefore, we tested the hypothesis that dietary phytoestrogens ameliorate the severity of experimentally induced pulmonary fibrosis. Male mice placed on either regular soybean diet or phytoestrogen-free diet were instilled with 0.1 N HCl to provoke pulmonary fibrosis. Thirty days later, lung mechanics were measured as indices of lung function and bronchoalveolar lavage fluid (BALF) and lung tissue were analyzed for biomarkers of fibrosis. Mice on phytoestrogen-free diet demonstrated increased mortality and stronger signs of chronic lung injury and pulmonary fibrosis, as reflected in the expression of collagen, extracellular matrix deposition, histology, and lung mechanics, compared to mice on regular diet. We conclude that dietary phytoestrogens play an important role in the pathogenesis of pulmonary fibrosis and suggest that phytoestrogens (e.g., genistein) may be useful as part of a therapeutic regimen against hydrochloric acid-induced lung fibrosis and chronic lung dysfunction.
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Haines, Christopher D., Pamela A. Harvey, Elizabeth D. Luczak, Kristen K. B. Barthel, John P. Konhilas, Peter A. Watson, Brian L. Stauffer, and Leslie A. Leinwand. "Estrogenic Compounds Are Not Always Cardioprotective and Can Be Lethal in Males with Genetic Heart Disease." Endocrinology 153, no. 9 (September 1, 2012): 4470–79. http://dx.doi.org/10.1210/en.2012-1391.

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Hypertrophic cardiomyopathy (HCM) is more severe in male than female mice eating a soy-based diet. We sought to determine whether the detrimental effects are mediated by the phytoestrogens present in soy, the mechanism by which phytoestrogens act, and to test whether estrogen modulates the sexually dimorphic phenotype. A soy-free diet (casein based) supplemented with the predominant phytoestrogens in soy, genistein and daidzein, recapitulated the fibrotic, proapoptotic and negative hemodynamic effects of soy in male hearts. As with the soy diet, the hearts of female HCM mice were not negatively affected by the phytoestrogen-containing diet. To determine the role of estrogen in the sex differences mediated by diet in HCM, gonadectomies were performed and estrogen was administered to male and female HCM mice on a casein- or phytoestrogen-supplemented diet. Somewhat surprisingly, estrogen was not protective in male or female mice with HCM and, in fact, was lethal in phytoestrogen-fed male mice with HCM. Because genistein is a potent tyrosine kinase inhibitor and tyrosine kinase inhibition has been associated with cardiotoxicity, we tested its effects in isolated adult cardiac myocytes. Genistein inhibited different tyrosine kinases depending on sex and, in combination with estrogen, resulted in apoptosis only in adult male cardiac myocytes. Finally, we show that phytoestrogens led to distinct programs of gene expression in hearts from males vs. females with HCM, suggesting mechanisms by which males are more sensitive to the detrimental effects of phytoestrogens and females are protected. These results implicate the phytoestrogen genistein in mediating cardiac pathology in males with HCM and, importantly, establish that estrogen is not protective in the setting of HCM.
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Glover, A., and S. J. Assinder. "Acute exposure of adult male rats to dietary phytoestrogens reduces fecundity and alters epididymal steroid hormone receptor expression." Journal of Endocrinology 189, no. 3 (June 2006): 565–73. http://dx.doi.org/10.1677/joe.1.06709.

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Phytoestrogens are plant-derived compounds with oestrogenic activity. They are common in both human and animal diets, particularly through soy-based foods. This study assessed whether exposure of adult male rats to a high phytoestrogen diet for 3–25 days affected their fertility, and assessed possible mechanisms through which phytoestrogens may disrupt fertility. Adult males, fed a high phytoestrogen diet for 3 days, demonstrated significantly reduced fecundity. This effect was transient, with fecundity returning to control levels by day 12. The expression of oestrogen receptor-α and androgen receptor mRNA was increased in the initial segment of the epididymis, but decreased in the cauda epididymis following 3 days on the high phytoestrogen diet. Epididymal sperm counts cannot account for the reduction in fertility at day 3. However, lipid peroxidation of epididymal sperm was significantly increased in animals fed a high phytoestrogen diet for 3 days. Disruption of the steroid regulation of the epididymis by phytoestrogens may alter its function, resulting in decreased quality of sperm, and thereby reducing fecundity.
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Dissertations / Theses on the topic "Phytoestrogens"

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Leu, Chao-Wei Chemistry Faculty of Science UNSW. "Synthesis of heterocyclic analogues of phytoestrogens." Publisher:University of New South Wales. Chemistry, 2008. http://handle.unsw.edu.au/1959.4/40824.

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The pyrrolo[3,2,1-ij]quinolin-6-one ring system was synthesised from 3-aryl-4,6-dimethoxyindoles and 2,3-disubstituted-4,6-dimethoxyindoles. The reaction of 4,6-dimethoxyindoles under Friedel-Crafts or Vilsmeier-Haack acylation gave the 2- and 7-indolyldeoxybenzoins in good yield. Cyclisation of 7-indolyldeoxybenzoins with N,N-dimethylformamide dimethyl acetal as a one carbon reagent gave the pyrroloquinolin-6-ones in high yield. Reduction of pyrroloquinolin-6-ones with hydrogen gas and 10% palladium on carbon or lithium aluminium hydride yielded the dihydropyrroloquinolin-6-ones. Demethylation of pyrroloquinolin-6-ones with 48% hydrobromic acid in glacial acetic acid gave a mixture of the monohydroxy and dihydroxy analogues in high yield. The synthesis of quinolin-4-ones using the Conrad-Limpach method was attempted using three different cyclisation conditions such as Dowtherm A, polyphosphoric acid and a mixture of diphenyl ether and methanesulfonic acid. Quinolin-2-ones such as 4-methyl-3-aryl-, 3,4-diaryl- and 3-aryl-4-benzyl-5,7-dimethoxyquinolin-2-ones could be synthesised from either the N-phenylacetylaniline or the N-trifluoroacetyl aniline strategy. Attempted reduction of the quinolin-2-ones with standard metal hydride reagents was unsuccessful. However reduction was achieved via the conversion of quinolin-2-one to the corresponding 2-chloroquinoline followed by reaction of the chloroquinoline with zinc powder and glacial acetic acid to produce a novel, highly substituted quinoline system. Demethylation was successfully carried out with 48% hydrobromic acid in glacial acetic acid to give the trihydroxyquinolin-2-one in high yield. The reactions of 4-substituted-5,7-dimethoxyquinolin-2-ones and the corresponding 2-chloroquinolines as potential organic intermediates were explored. Facile formylation of both quinolin-2-ones and 2-chloroquinolines was observed under Vilsmeier-Haack conditions while acetylation was successful under Friedel-Crafts conditions using antimony (V) pentachloride as the Lewis acid. Further reaction of 8-formyl-quinolin-2-one with 1,2-diaminobenzene in N,N-dimethylformamide led to the formation of a new 8-(benzimidazolyl)-quinolin-2-one ring system. The quinolin-2-ones exhibited selective electrophilic substitution at the C8 position for a range of reactions. However, an unexpected nitration occurred at the C3 position for the 4-methoxy and 4-phenyl-5,7-dimethoxyquinolin-2-ones with good yields. A series of novel 4,6-hydroxylindoles was successfully synthesised from the corresponding methoxy analogues in high yield using anhydrous aluminium chloride. When 3-(4-bromophenyl)-4,6-dimethoxyindole was reacted with 48% hydrobromic acid in glacial acetic acid a 2,2?-indolylindoline dimer was formed. The 5,7-dihydroxyquinolin-2-ones were similarly synthesised in high yield using anhydrous aluminium chloride in chlorobenzene.
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Lanceman, Dorothy Melinda. "Do phytoestrogens disrupt endogenous oestrogen metabolism?" Thesis, University of Birmingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.410279.

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Dinauer, Christina Marie. "Analysis of in vitro binding of dietary fibers by the phytoestrogen, daidzein, in the presence and absence of iron." Online version, 2000. http://www.uwstout.edu/lib/thesis/2000/2000dinauerc.pdf.

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Glover, Amy, and n/a. "The effect of dietary phytoestrogens on male fertility." University of Otago. Department of Anatomy & Structural Biology, 2006. http://adt.otago.ac.nz./public/adt-NZDU20070711.140602.

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Phytoestrogens are plant-derived compounds with oestrogenic activity. They are common in both human and animal diets, particularly through soy-based foods. This study assessed whether the reproductive function of male rats is affected by exposure to a high phytoestrogen diet during adulthood and examined possible mechanisms through which phytoestrogens may disrupt reproductive function. Experiments focused on the epididymis, a steroid-regulated organ responsible for the maturation, transport, and storage of sperm. Adult male rats, bred and raised on a low phytoestrogen diet, were either transferred to a high phytoestrogen diet (experimental), or remained on the low phytoestrogen diet (control). Litter size is a measure of fecundity and after 3 days on the high phytoestrogen diet litter size was reduced. This effect on fecundity was transient as litter sizes returned to control levels by day 12. The reduced fecundity at day 3 could not be explained by changes in sperm concentration. Plasma gonadotrophin levels and testicular testosterone levels were not affected by phytoestrogen exposure, however, the expression of steroid hormone receptors in the epididymis was affected, coincidental with reduced fecundity. The gene expression of oestrogen receptor alpha and androgen receptor was increased in the initial segment of the epididymis and decreased in the cauda epididymis. Additionally, lipid peroxidation of epididymal sperm was significantly increased in rats fed the high phytoestrogen diet for 3 days. It is concluded that acute exposure to the high phytoestrogen diet disrupts the steroid regulation of the epididymis, disrupting its normal function. This results in decreased sperm quality, thereby reducing fecundity.
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Mfenyana, Ciko. "Selective extraction of Cyclopia for enhanced in vitro phytoestrogenicity /." Link to the online version, 2008. http://hdl.handle.net/10019/783.

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Bakshi, Vibhu Smith Don Wiley. "In vitro cultures of Morus alba for enhancing production of phytoestrogens." [Denton, Tex.] : University of North Texas, 2009. http://digital.library.unt.edu/ark:/67531/metadc12078.

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Bylund, Annika. "Phytoestrogens and prostate cancer : experimental, clinical, and epidemiological studies." Doctoral thesis, Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1402.

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Ogegbo, Olumuyiwa Lateefah. "Quantitative analysis and metabonomic study of phytoestrogens in Africans." Thesis, London Metropolitan University, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.507096.

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West, M. "Effects of dietary phytoestrogens on spermatogenesis and sperm function." Thesis, Queen's University Belfast, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426916.

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Pfeiffer, Thomas J. "Phytoestrogens may inhibit proliferation of MCF-7 cells, an estrogen-responsive breast adenocarcinoma cell line." Link to electronic thesis, 2004. http://www.wpi.edu/Pubs/ETD/Available/etd-0430104-132238.

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Books on the topic "Phytoestrogens"

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H, Adlercreutz, ed. Phytoestrogens. London: Baillière Tindall, 1998.

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Brooks, Jennifer Dallas. Phytoestrogens as modulators of estrogen metabolism. Ottawa: National Library of Canada, 2003.

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Griffiths, K. Oestrogens, phyto-oestrogens and the pathogenesis of prostatic disease. London: Martin Dunitz, 2002.

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Berk, Laura E. Child development. 2nd ed. Toronto: Pearson/Allyn and Bacon, 2006.

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Berk, Laura E. Child development. Boston: Allyn and Bacon, 1989.

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Berk, Laura E. Child development. 7th ed. Boston: Pearson/Allyn and Bacon, 2006.

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Anderson, John, and Sarwar Gilani, eds. Phytoestrogens and Health. AOCS Publishing, 2002. http://dx.doi.org/10.1201/9781439822197.

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Anderson, John J. B. Phytoestrogens and Health. AOCS Publishing, 2002.

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Anderson, John J. B. Phytoestrogens and Health. Taylor & Francis Group, 2002.

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Yildiz, Fatih. Phytoestrogens in Functional Foods. Taylor & Francis Group, 2019.

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Book chapters on the topic "Phytoestrogens"

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Speirs, Valerie. "Phytoestrogens." In Encyclopedia of Cancer, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_4565-2.

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Singh, Ashok K., and Leo M. L. Nollet. "Phytoestrogens." In Analysis of Endocrine Disrupting Compounds in Food, 219–28. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118346747.ch7.

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Fitzpatrick, Lorraine A. "Phytoestrogens." In Nutrition and Bone Health, 593–614. Totowa, NJ: Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-740-6_32.

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Speirs, Valerie. "Phytoestrogens." In Encyclopedia of Cancer, 3574–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_4565.

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Wiseman, Helen. "Phytoestrogens." In Phytonutrients, 203–53. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118253649.ch7.

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Speirs, Valerie. "Phytoestrogens." In Encyclopedia of Cancer, 2886–87. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_4565.

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Cade, Janet, Victoria Burley, and Sara Kirk. "Phytoestrogens and Health." In Food and Nutritional Supplements, 141–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56623-3_10.

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Matthews, Geetha, and Veronica A. Ravnikar. "Phytoestrogens and Menopause." In Menopause, 235–44. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-246-3_15.

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Fatima, Atiya, Asrar Alam, and Ram Singh. "Therapeutic Potential of Phytoestrogens." In Functional Food and Human Health, 297–327. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1123-9_15.

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Man, Ricky Y. K., Susan W. S. Leung, Hwee Teoh, Adrian Quan, Wendy Keung, and Mary Y. K. Lee. "Phytoestrogens and Cardiovascular Disorders." In Pathophysiology of Cardiovascular Disease, 513–24. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-1-4615-0453-5_35.

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Conference papers on the topic "Phytoestrogens"

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Solopov, P. A., R. M. L. Colunga Biancatelli, C. Dimitropolou, and J. D. Catravas. "Phytoestrogens as a Potential Antidotes Against Hydrochloric Acid-Induced Pulmonary Fibrosis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3833.

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Ju, Woong, Seung Cheol Kim, Nam Hee Kim, and Yun Hwan Kim. "Abstract B83: Isoflavones from phytoestrogens and cervical cancer risk: A nested case-control study." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-b83.

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Katchy, Anne C., Caroline Pinto, Maria Bondesson, and Cecilia Williams. "Abstract 547: Mapping of estrogen receptor-dependent and independent signaling pathways for xeno- and phytoestrogens." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-547.

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Shestakova, E., A. Scherbakov, O. Ryabinina, A. Grishanina, K. Galeeva, and T. Bogush. "PO-147 Hypoxia, estrogens and phytoestrogens influence BRCA1 and oestrogen receptors α expression in MCF-7 breast cancer cell line." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.669.

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Yeo, Yohwan, Kwang-Pil Ko, Seung-Hyun Ma, Jae Jeong Yang, Aesun Shin, Sue K. Park, Soung-Hoon Chang, Hai-Rim Shin, Daehee Kang, and Keun-Young Yoo. "Abstract 4823: Isoflavones from phytoestrogens and colorectal cancer risk: A nested case-control study within the Korean Multicenter Cancer Cohort." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4823.

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Reger, Michael, Terrell Zollinger, Ziyue Liu, Josette Jones, and Jianjun Zhang. "Abstract 1884: Dietary intake of phytoestrogens and the risk of prostate cancer in the Prostate, Lung, Colorectal, and Ovarian Cancer Screening Trial." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1884.

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Helle, Janina, Oliver Zierau, Kristin Kräker, Annekathrin Keiler, Günter Vollmer, JoEllen Welsh, and Georg Kretzschmar. "Abstract 625: Proliferative effects of the phytoestrogens 8-Prenylnaringenin, 6-(1.1-dimethylallyl)naringenin and Naringenin in MCF-7 cells and the rat mammary gland." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-625.

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Muntean, Edward, Camelia Urda, and Raluca Rezi. "HPLC Screening of Phytoestrogens from Soybeans in Conjunction with Chemometric Data Analysis: A Tool for Selecting the Best Raw Materials for Producing Dietary Supplements for Menopausal Health." In International Electronic Conference on Biomedicines. Basel Switzerland: MDPI, 2023. http://dx.doi.org/10.3390/ecb2023-14082.

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Kasperzyk, Julie L., Niclas Håkansson, Katja Fall, Swen‐Olof Andersson, Jan‐Erik Johansson, Meir J. Stampfer, Alicja Wolk, and Ove Andrén. "Abstract B101: Phytoestrogen intake in relation to prostate cancer incidence and survival." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Dec 6–9, 2009; Houston, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-09-b101.

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Anbazhakan, K., R. Praveena, and K. Sadasivam. "Prediction of biological activities of phytoestrogen and its derivative – A Insilico study." In ADVANCES IN BASIC SCIENCE (ICABS 2019). AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5122635.

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Reports on the topic "Phytoestrogens"

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Kurzer, Mindy S. Dietary Phytoestrogens and Prostate Cancer Prevention. Fort Belvoir, VA: Defense Technical Information Center, May 2003. http://dx.doi.org/10.21236/ada416704.

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Kurzer, Mindy S. Dietary Phytoestrogens and Prostate Cancer Prevention. Fort Belvoir, VA: Defense Technical Information Center, May 2004. http://dx.doi.org/10.21236/ada425976.

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Kurzer, Mindy S., and Joel Slaton. Dietary Phytoestrogens and Prostate Cancer Prevention. Fort Belvoir, VA: Defense Technical Information Center, May 2007. http://dx.doi.org/10.21236/ada472737.

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Teixeira, Maria Julia, Maria Julia Teixeira, Cecília Colombo, Tamy Colonetti, and Maria Ines da Rosa. Effect of phytoestrogens on endometrial thickness: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2020. http://dx.doi.org/10.37766/inplasy2020.3.0001.

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Palomares, Melanie R., and Julie R. Gralow. Modulation of Postmenopausal Steroid Hormone Levels by Phytoestrogens and Correlation with Breast Proliferative Activity and Menopausal Symptoms. Fort Belvoir, VA: Defense Technical Information Center, July 2002. http://dx.doi.org/10.21236/ada410800.

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Gralow, Julie R. Modulation of Postmenopausal Steroid Hormone Levels by Phytoestrogens and Correlation with Breast Proliferative Activity and Menopausal Symptoms. Fort Belvoir, VA: Defense Technical Information Center, July 2004. http://dx.doi.org/10.21236/ada467791.

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Thomas, T. Molecular Mechanism of Action of Genistein and Related Phytoestrogens in Estrogen Receptor Dependent and Independent Growth of Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, July 2000. http://dx.doi.org/10.21236/ada392767.

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Balabhadrapathruni, Srivani. Molecular Mechanism of Action of Genistein and Related Phytoestrogens in Estrogen-Receptor Dependent and Independent Growth of Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, July 1999. http://dx.doi.org/10.21236/ada381588.

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Azios, Nicolas G., and Surangani Dharawardhane. Phytoestrogen Effects on Cytoskeletal Morphology and Motility in Breast Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, March 2005. http://dx.doi.org/10.21236/ada434058.

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Dharmawardhane, Suranganie. Estrogen and the Dietary Phytoestrogen Resveratrol as Regulators of the Rho GTPase Rac in Breast Cancer Metastasis. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada554793.

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