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1

Inositol phosphates and lipids: Methods and protocols. New York: Humana Press, 2010.

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2

N.Y.) Inositol Phospholipid Signaling in Physiology and Disease (Conference) (2012 New York. Inositol phospholipid signaling in physiology and disease. Edited by Fernandes, Sandra (Sandra R.), Kerr William G, and New York Academy of Sciences. Hoboken, NJ: New York Academy of Sciences, 2012.

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3

H, Stenmark, ed. Phosphoinositides in subcellular targeting and enzyme activation. Berlin: Springer, 2004.

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4

McDermott, Mark Ian. Phospholipase D2 regulation by ADP-ribosylation factor-6 and phosphatidylinositol 4-phosphate 5-kinase. Birmingham: University of Birmingham, 2002.

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5

Mei, Yu. Functional Characterization of Arabidopsis Phosphatidylinositol Monophosphate 5-kinase 2 in Lateral Root Development, Gravitropism and Salt Tolerance. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-9373-5.

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6

Falcioni, Lisa. The role of the phosphatidylinositol-3 kinase-Akt pathway in determining radiation sensivity in the breast cancer cell line MDA-MB 231. Sudbury, Ont: Laurentian University, School of Graduate Studies, 2005.

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7

GPI-Anchored Membrane Proteins and Carbohydrates. LANDES BIOSCIENCE PUBLISHERS, 1999.

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8

(Editor), A. Lahiri Majumder, and B. B. Biswas (Editor), eds. Biology of Inositols and Phosphoinositides (Subcellular Biochemistry). Springer, 2006.

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9

Majumder, A. Lahiri, and B. B. Biswas. Biology of Inositols and Phosphoinositides. Springer, 2006.

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10

Barker, Christopher J. Inositol Phosphates and Lipids: Methods and Protocols. Humana Press, 2012.

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11

Cartee, Robert T., Michelle R. Fry, and Satoshi Kagiwada. Phospholipid Transfer Proteins: Emerging Roles in Vesicle Trafficking, Signal Transduction, and Metabolic Regulation (Molecular Biology Intelligence Unit). Landes Bioscience, 1996.

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12

Phosphoinositides and receptor mechanisms (Receptor biochemistry and methodology). A.R. Liss, 1986.

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13

W, Putney James, ed. Phosphoinositides and receptor mechanisms. New York: A.R. Liss, 1986.

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14

Stenmark, Harald. Phosphoinositides in Subcellular Targeting and Enzyme Activation. Springer London, Limited, 2012.

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15

Bankaitis, Vytas A. Phospholipid Transfer Proteins: Emerging Roles in Vesicle Trafficking, Signal Transduction, and Metabolic Regulation (Molecular Biology Intelligence Unit). Landes Bioscience, 1996.

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16

Phosphoinositidase C-Linked Receptor Desenisitization. Ray Langley, 1996.

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17

Shamshad, Cockcroft, ed. Biology of phosphoinositides. Oxford: Oxford University Press, 2000.

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18

(Editor), B. B. Biswas, and Susweta Biswas (Editor), eds. Myo-Inositol Phosphates, Phosphoinositides, and Signal Transduction (Subcellular Biochemistry). Springer, 1996.

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19

Ban, Kiwon. The role of phosphatidylinositol-3 kinase isomers in myocardial ischemic preconditioning. 2005.

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20

Mei, Yu. Functional Characterization of Arabidopsis Phosphatidylinositol Monophosphate 5-kinase 2 in Lateral Root Development, Gravitropism and Salt Tolerance. Springer, 2014.

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21

Mei, Yu. Functional Characterization of Arabidopsis Phosphatidylinositol Monophosphate 5-Kinase 2 in Lateral Root Development, Gravitropism and Salt Tolerance. Springer London, Limited, 2014.

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22

Mei, Yu. Functional Characterization of Arabidopsis Phosphatidylinositol Monophosphate 5-Kinase 2 in Lateral Root Development, Gravitropism and Salt Tolerance. Springer Netherlands, 2016.

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23

Menon, Deepa U. Autism and Intellectual Disabilities. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0053.

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Abstract:
PTEN (phosphatase and tensin homologue) on chromosome 10q23.3 is a tumor suppressor gene that encodes for a dual specificity phosphatase that regulates the phosphatidylinositol 3- kinase pathway and has an important role in brain development by affecting neuronal survival, neurite outgrowth, synaptic plasticity, and learning memory. Germline mutations of the PTEN gene have been implicated in a group of related tumor syndromes with autosomal dominant inheritance and variable expression and include the Cowden syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome, and Juvenile Polyposis syndrome. These syndromes are collectively called the PTEN hamartoma tumor syndromes (PHTS) because they have a predisposition to tumors and hamartomas. PTEN germ line mutations have also been recently linked to autism and macrocephaly and the prevalence of PTEN mutation in children with autism spectrum disorder, and macrocephaly is reported to range from 1.1% to 16.7%.
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24

Wang, Guanyu. Analysis of Complex Diseases: A Mathematical Perspective. Taylor & Francis Group, 2013.

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25

Analysis of Complex Diseases: A Mathematical Perspective. Taylor & Francis Group, 2013.

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26

Wang, Guanyu. Analysis of Complex Diseases: A Mathematical Perspective. Taylor & Francis Group, 2013.

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27

Wang, Guanyu. Analysis of Complex Diseases: A Mathematical Perspective. Taylor & Francis Group, 2013.

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