Relevant bibliographies by topics / Phopholamban

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'Phopholamban'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Phopholamban"

1

Kranias, Evangelia G., and Roger J. Hajjar. "Modulation of Cardiac Contractility by the Phopholamban/SERCA2a Regulatome." Circulation Research 110, no. 12 (2012): 1646–60. http://dx.doi.org/10.1161/circresaha.111.259754.

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2

Takagi, Yasushi, Kazushi Urasawa, Satoshi Kaneta, Noritsugu Nakano, and Akira Kitabatake. "Phopholamban expression in the hearts of cardiomyopathic hamster, BIO53. 58." Journal of Cardiac Failure 4, no. 3 (1998): 110. http://dx.doi.org/10.1016/s1071-9164(98)90473-0.

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3

Xu, A., M. Jiang, and N. Narayanan. "A16. Calmodulin triggers Cardiac SR Ca2+ pump function by disrupting Ca2+-ATPase-phopholamban interaction." Journal of Molecular and Cellular Cardiology 40, no. 6 (2006): 889. http://dx.doi.org/10.1016/j.yjmcc.2006.03.356.

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4

Hou, Zhanjia, Raffaello Verardi, Larry R. Masterson, et al. "A Mutation Associated with DCM Increases Phospholamban Oligomerization and Decreases SERCA-Binding, but Does Not Change Phopholamban Tertiary Strucuture or Phosphorylation by PKA." Biophysical Journal 98, no. 3 (2010): 434a. http://dx.doi.org/10.1016/j.bpj.2009.12.2356.

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5

ANTOONS, G. "660 Altered phosphorylation status of phopholamban, PLB, and its contribution to the negative 6Ca2+9i-frequency relationship in the MLP-I-mouse with heart failure." European Heart Journal 24, no. 5 (2003): 118. http://dx.doi.org/10.1016/s0195-668x(03)94096-6.

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Dissertations / Theses on the topic "Phopholamban"

1

TORRE, ELEONORA. "Role of SERCA stimulation and voltage-dependent Ca2+ channels in improving Ca2+ handling and sustaining heart automaticity." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2020. http://hdl.handle.net/10281/261917.

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Abstract:
Parte 1. La cardiomiopatia diabetica (DCM) è una malattia caratterizzata da una precoce disfunzione diastolica (DD). I meccanismi che possono ripristinare il rilassamento cardiaco, migliorando la dinamica intracellulare di Ca2+, rappresentano un promettente approccio terapeutico per le malattie cardiovascolari associate alla DD. Istaroxime è un inibitore di NaK-ATPase (NKA) e stimolatore del recupero di Ca2+ nel reticolo sarcoplasmatico (SR) attraverso la pompa del SR per il Ca2+ (SERCA2a). Il progetto mira a caratterizzare gli effetti di Istaroxime a una concentrazione che minimamente influen
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