Relevant bibliographies by topics / Phlorizin

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  2. Dissertations / Theses
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Academic literature on the topic 'Phlorizin'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Phlorizin"

1

GIUDICELLI, Jean, Marie-France BERTRAND, Stephane BILSKI, T. Than TRAN, and Jean-Claude POIREE. "Effect of cross-linkers on the structure and function of pig-renal sodium–glucose cotransporters after papain treatment." Biochemical Journal 330, no. 2 (March 1, 1998): 733–36. http://dx.doi.org/10.1042/bj3300733.

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Kidney brush-border membranes contain two sodium-dependent glucose transporters, one with low and one with high affinity for phlorizin, the specific inhibitor of these transporters. Using Scatchard analysis of phlorizin binding and Western blotting with specific antibodies against these transporters, we demonstrate in this study that although both transporters were proteolysed by papain treatment, only the high-affinity phlorizin-binding sites were decreased. Papain treatment followed by cross-linking with homobifunctional disuccinimidyl tartarate restored only the structure of the low-affinity phlorizin-binding protein (approx. molecular mass 70 kDa) without modifying the phlorizin-binding sites. When disuccinimidyl tartarate was replaced with dithiobis(succinimidyl acetate), another homobifunctional cross-linker with a higher spacer arm, the low- and high-affinity sites were both restored, with reappearance of two phlorizin-binding proteins with approx. molecular masses of 70 and 120 kDa. We conclude that high-affinity phlorizin-binding sites depend on the presence of the heterodimeric 120 kDa protein.
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Ehrenkranz, Joel R. L., Norman G. Lewis, C. Ronald Kahn, and Jesse Roth. "Phlorizin: a review." Diabetes/Metabolism Research and Reviews 21, no. 1 (January 2005): 31–38. http://dx.doi.org/10.1002/dmrr.532.

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Holtenius, Kjell, and Paul Holtenius. "Effects of peroral alanine administration in lactating ewes with decreased availability of glucose." British Journal of Nutrition 78, no. 5 (November 1997): 805–13. http://dx.doi.org/10.1079/bjn19970196.

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The metabolic effects of a phlorizin-induced drainage of glucose were studied in six lactating ewes with or without peroral alanine drenches in a study of crossover design. Phlorizin gave rise to a small, but significant, elevation of plasma β-hydroxybutyrate. The plasma level of alanine decreased by about 30 % due to the phlorizin injections and alanine was negatively correlated to β-hydroxybutyrate. The plasma level of free fatty acids increased due to phlorizin. Plasma insulin and glucose concentrations were not significantly affected by phlorizin while glucagon level showed a small but significant increase. Peroral alanine drenches to phlorizin-treated ewes gave rise to a transitory elevation of alanine in plasma. The plasma level of free fatty acids was about 40 % lower in phlorizin-treated ewes receiving alanine and β-hydroxybutyrate tended to be lower (P < 0.08). We suggest that β-hydroxybutyrate, apart from its function as an oxidative fuel, might play an important role by limiting glucose oxidation and protein degradation in skeletal muscles during periods of negative energy balance in ruminants. Furthermore, it is suggested that alanine supplementation decreases lipolysis and ketogenesis in lactating ewes.
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Nguyen, Ngoc Anh, Ngoc Tan Cao, Thi Huong Ha Nguyen, Jung-Hwan Ji, Gun Su Cha, Hyung-Sik Kang, and Chul-Ho Yun. "Enzymatic Production of 3-OH Phlorizin, a Possible Bioactive Polyphenol from Apples, by Bacillus megaterium CYP102A1 via Regioselective Hydroxylation." Antioxidants 10, no. 8 (August 23, 2021): 1327. http://dx.doi.org/10.3390/antiox10081327.

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Phlorizin is the most abundant glucoside of phloretin from the apple tree and its products. Phlorizin and its aglycone phloretin are currently considered health-beneficial polyphenols from apples useful in treating hyperglycemia and obesity. Recently, we showed that phloretin could be regioselectively hydroxylated to make 3-OH phloretin by Bacillus megaterium CYP102A1 and human P450 enzymes. The 3-OH phloretin has a potent inhibitory effect on differentiating 3T3-L1 preadipocytes into adipocytes and lipid accumulation. The glucoside of 3-OH phloretin would be a promising agent with increased bioavailability and water solubility compared with its aglycone. However, procedures to make 3-OH phlorizin, a glucoside of 3-OH phloretin, using chemical methods, are not currently available. Here, a biocatalytic strategy for the efficient synthesis of a possibly valuable hydroxylated product, 3-OH phlorizin, was developed via CYP102A1-catalyzed regioselective hydroxylation. The production of 3-OH phlorizin by CYP102A1 was confirmed by HPLC and LC–MS spectroscopy in addition to enzymatic removal of its glucose moiety for comparison to 3-OH phloretin. Taken together, in this study, we found a panel of mutants from B. megaterium CYP102A1 could catalyze regioselective hydroxylation of phlorizin to produce 3-OH phlorizin, a catechol product.
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Dinda, P. K., and I. T. Beck. "Phlorizin increases the permeability of intestinal mucosal membrane to sodium." Canadian Journal of Physiology and Pharmacology 65, no. 4 (April 1, 1987): 579–86. http://dx.doi.org/10.1139/y87-098.

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We reported previously that when jejunal transmural glucose transport was inhibited by phlorizin the ratio of Na:giucose transport increased from 2.0:1 (in controls) to 3.3:1. To elucidate the mechanism of this increased ratio of Na:glucose transport, in the present study we have investigated the effect of phlorizin on Na uptake by brush border membrane vesicles and by everted sacs of hamster jejunum. In experiments on membrane vesicles the following observations were made. The time course of Na uptake showed that the control vesicles were in complete equilibrium with a Na-containing (100 mM) medium between 30 and 90 min incubation. In these periods of incubation, the vesicles incubated with phlorizin presumably also equilibrated with the medium, but lost their intravesicular Na during Millipore filtration and washing, and consequently the residual Na content was lower than that of controls. This effect of phlorizin was concentration dependent, and appeared to be unrelated to Na-coupled glucose transport, because it was also observed in the absence of glucose. This loss of Na during Millipore filtration and washing was also observed (i) when vesicles were equilibrated in a Na-containing solution in the absence of phlorizin and then exposed to a similar solution containing phlorizin, or (ii) when vesicles were equilibrated in a Na-containing solution in the presence of phlorizin and then washed repeatedly following Millipore filtration. Preincubation of vesicles for 10 min in a Na- and glucose-free solution containing phlorizin followed by incubation for 30–90 s in solutions containing 1 mM glucose and various concentrations of Na (from 10 to 100 mM) caused an increase in Na uptake from all concentrations of Na. After similar preincubation, when jejunal everted sacs were incubated for 15 s in a Na- and glucose-containing medium, Na uptake by the sacs increased. These findings suggest that phlorizin causes an increase in permeability of mucosal membrane of the enterocyte to Na. This may cause a rapid dissipation of Na gradient and an increase in the ratio of Na:glucose transport. The dissipation of Na gradient may be an additional mechanism for phlorizin-induced inhibition of intestinal sugar transport.
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Zhang, Yantong, Limei Lin, Yuehong Long, Hongyu Guo, Zhuo Wang, Minghui Cui, Jian Huang, and Zhaobin Xing. "Comprehensive Transcriptome Analysis Revealed the Effects of the Light Quality, Light Intensity, and Photoperiod on Phlorizin Accumulation in Lithocarpus polystachyus Rehd." Forests 10, no. 11 (November 7, 2019): 995. http://dx.doi.org/10.3390/f10110995.

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Lithocarpus polystachyus Rehd. is an important medicinal plant species grown in southern China, with phlorizin as its main active substance. The effects of light conditions on phlorizin biosynthesis in L. polystachyus remain unclear. Thus, we analyzed the transcriptomes of L. polystachyus plants cultivated under diverse light qualities, light intensities, and photoperiods. The light treatments resulted in 5977–8027 differentially expressed genes (DEGs), which were functionally annotated based on the gene ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) databases. Genes encoding transcription factors from 89 families were differentially expressed after the light treatments, implying these transcription factors are photoresponsive. Phenylalanine ammonia lyase (PAL) and 4-coumarate-CoA ligase (4CL) are the key enzymes for the accumulation of phlorizin. The transcription levels of PAL2, PAL, 4CL1 (DN121614), 4CLL7, and 4CL1 (DN102161) were positively correlated with phlorizin accumulation, suggesting that these genes are important for phlorizin biosynthesis. An ultra-high-performance liquid chromatography method was used to quantify the phlorizin content. Phlorizin accumulated in response to the green light treatment and following appropriate decreases in the light intensity or appropriate increases in the duration of the light exposure. The green light, 2000 lx, and 3000 lx treatments increased the PAL activity of L. polystachyus, but the regulatory effects of the light intensity treatments on PAL activity were relatively weak. This study represents the first comprehensive analysis of the light-induced transcriptome of L. polystachyus. The study results may form the basis of future studies aimed at elucidating the molecular mechanism underlying phlorizin biosynthesis in L. polystachyus. Moreover, this study may be relevant for clarifying the regulatory effects of light on the abundance of bioactive components in medicinal plants.
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Einhorn, Todd C., Cecil Stushnoff, Ann E. McSay, Phil L. Forsline, Sam Cox, Joel R. L. Ehrenkranz, and Loretta Sandoval. "(18) Biodiversity of the Flavonoid Phlorizin in a Subset of the USDA Apple Germplasm Core Collection." HortScience 40, no. 4 (July 2005): 1067D—1067. http://dx.doi.org/10.21273/hortsci.40.4.1067d.

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Phlorizin is known for its role in reducing glucotoxicity and has a long history of use in diabetes research. In addition, its contribution to the pool of total phenolics adds to the overall health benefits attributed to fruit. Phlorizin is limited to Rosaceae family plants, of which apple comprises its current commercial source; however, limited information exists regarding its biodiversity among apple taxa. A subset of 22 taxa from a core collection of apple accessions representative of the global genetic diversity of apple was used to investigate the biodiversity of phlorizin present in apple shoots and in fruit relative to total phenolic content and free radical scavenging capacity. Fruit and shoots were harvested from the USDA Plant Genetic Resources Unit in Geneva, N.Y. Validation and quantification of phlorizin was conducted using a rigorous high-pressure liquid chromatography (HPLC) procedure. Total phenolics in fruit, assayed using a Folin-Ciocalteu method and expressed as gallic acid equivalents, ranged from 227 to 7181 mg·L-1 and were strongly related to 2,2' azinobis (3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) antioxidant capacity for the core collection (r= 0.778). On a molar basis, phlorizin had lower antioxidant capacity than other major phenolic compounds present in apple fruit, but was more effective than ascorbic acid. Phlorizin yield in dormant apple shoots, expressed as percent weight, ranged from 0.9% to 5.5%. A rapid, 96 well micro-plate spectrophotometric assay was also developed to aid in the screening of multiple samples for selection of high phlorizin yielding apple taxa. Spectrophotometry overestimated phlorizin content as expected, but the calibration curve between HPLC and spectrophotometry was acceptable, r2 = 0.88.
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Cai, Qian, Baoying Li, Fei Yu, Weida Lu, Zhen Zhang, Mei Yin, and Haiqing Gao. "Investigation of the Protective Effects of Phlorizin on Diabetic Cardiomyopathy indb/dbMice by Quantitative Proteomics." Journal of Diabetes Research 2013 (2013): 1–9. http://dx.doi.org/10.1155/2013/263845.

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Patients with diabetes often develop hypertension and atherosclerosis leading to cardiovascular disease. However, some diabetic patients develop heart failure without hypertension and coronary artery disease, a process termed diabetic cardiomyopathy. Phlorizin has been reported to be effective as an antioxidant in treating diabetes mellitus, but little is known about its cardioprotective effects on diabetic cardiomyopathy. In this study, we investigated the role of phlorizin in preventing diabetic cardiomyopathy indb/dbmice. We found that phlorizin significantly decreased body weight gain and the levels of serum fasting blood glucose (FBG), triglycerides (TG), total cholesterol (TC), and advanced glycation end products (AGEs). Morphologic observations showed that normal myocardial structure was better preserved after phlorizin treatment. Using isobaric tag for relative and absolute quantitation (iTRAQ) proteomics, we identified differentially expressed proteins involved in cardiac lipid metabolism, mitochondrial function, and cardiomyopathy, suggesting that phlorizin may prevent the development of diabetic cardiomyopathy by regulating the expression of key proteins in these processes. We used ingenuity pathway analysis (IPA) to generate an interaction network to map the pathways containing these proteins. Our findings provide important information about the mechanism of diabetic cardiomyopathy and also suggest that phlorizin may be a novel therapeutic approach for the treatment of diabetic cardiomyopathy.
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Kemp, P. J., and C. A. Boyd. "Pathways for glucose transport in type II pneumocytes freshly isolated from adult guinea pig lung." American Journal of Physiology-Lung Cellular and Molecular Physiology 263, no. 5 (November 1, 1992): L612—L616. http://dx.doi.org/10.1152/ajplung.1992.263.5.l612.

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Previous in vivo studies of sugar transport across the mature pulmonary epithelium have provided evidence for the existence of a specific phlorizin-inhibitable, sodium-dependent transport process for D-glucose, although no direct evidence for the cellular location of this transport system in fresh cells has been shown to date. With the use of elastase digestion and lectin agglutination, a pure preparation of type II alveolar epithelial cells was isolated from adult guinea pig lung. This preparation always contained >90% type II cells and typically showed approximately 85% cell viability 2-3 h after the isolation procedure had begun. At 37 degrees C, cells showed specific [3H]phlorizin binding that was attenuated by D-glucose and completely abolished by sodium replacement. Substantial accumulation of the hexose [14C]methyl alpha-D-glucopyranoside (14C-labeled AMG), a substrate specific for the sodium-dependent glucose cotransporter was found in the presence of extracellular sodium; this accumulation above equilibrium was abolished on removal of sodium, addition of phlorizin, or in the presence of a saturating concentration (69 mM) of D-glucose. The apparent inhibition constant (Ki) for glucose inhibition of AMG uptake was 0.4 mM and for phlorizin, 0.5 microM. The Hill plot of sodium activation of AMG uptake gave a coefficient of 2.8, suggesting cooperativeness between sodium and AMG transport. 3-O-[14C]methyl-D-glucose (3-OMG) transport was also blocked by phlorizin. Phloretin, in the presence of phlorizin, slowed the initial rate of entry but did not affect the equilibrium that was attained in the presence of phlorizin alone.(ABSTRACT TRUNCATED AT 250 WORDS)
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Nagata, Takumi, Masayuki Suzuki, Masanori Fukazawa, Kiyofumi Honda, Mizuki Yamane, Ayae Yoshida, Hiroko Azabu, et al. "Competitive inhibition of SGLT2 by tofogliflozin or phlorizin induces urinary glucose excretion through extending splay in cynomolgus monkeys." American Journal of Physiology-Renal Physiology 306, no. 12 (June 15, 2014): F1520—F1533. http://dx.doi.org/10.1152/ajprenal.00076.2014.

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Sodium-glucose cotransporter 2 (SGLT2) inhibitors showed a glucose lowering effect in type 2 diabetes patients through inducing renal glucose excretion. Detailed analysis of the mechanism of the glucosuric effect of SGLT2 inhibition, however, has been hampered by limitations of clinical study. Here, we investigated the mechanism of urinary glucose excretion using nonhuman primates with SGLT inhibitors tofogliflozin and phlorizin, both in vitro and in vivo. In cells overexpressing cynomolgus monkey SGLT2 (cSGLT2), both tofogliflozin and phlorizin competitively inhibited uptake of the substrate (α-methyl-d-glucopyranoside; AMG). Tofogliflozin was found to be a selective cSGLT2 inhibitor, inhibiting cSGLT2 more strongly than did phlorizin, with selectivity toward cSGLT2 1,000 times that toward cSGLT1; phlorizin was found to be a nonselective cSGLT1/2 inhibitor. In a glucose titration study in cynomolgus monkeys under conditions of controlled plasma drug concentration, both tofogliflozin and phlorizin increased fractional excretion of glucose (FEG) by up to 50% under hyperglycemic conditions. By fitting the titration curve using a newly introduced method that avoids variability in estimating the threshold of renal glucose excretion, we found that tofogliflozin and phlorizin lowered the threshold and extended the splay in a dose-dependent manner without significantly affecting the tubular transport maximum for glucose (TmG). Our results demonstrate the contribution of SGLT2 to renal glucose reabsorption (RGR) in cynomolgus monkeys and demonstrate that competitive inhibition of cSGLT2 exerts a glucosuric effect by mainly extending splay and lowering threshold without affecting TmG.
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Dissertations / Theses on the topic "Phlorizin"

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Collins, Alison Jane. "Ontogeny of lactase-phlorizin hydrolase in the pig small intestine." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.388687.

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Keller, Patrick. "Developmental regulation and post-translational modification of lactase-phlorizin hydrolase and sucrase-isomaltase /." [S.l.] : [s.n.], 1994. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10881.

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Hollox, Edward John. "Molecular and population genetic analyses of variation within and surrounding the human lactase gene." Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322742.

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Behrendt, Marc [Verfasser]. "Characterization of natural and artificial mutants of human intestinal lactase phlorizin hydrolase / Marc Behrendt." Hannover : Technische Informationsbibliothek und Universitätsbibliothek Hannover, 2010. http://d-nb.info/1010839373/34.

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Meier, Anja Doris [Verfasser], and Florian [Akademischer Betreuer] Lang. "Der Einfluss von Phlorizin auf den programmierten Zelltod von Erythrozyten / Anja Doris Meier ; Betreuer: Florian Lang." Tübingen : Universitätsbibliothek Tübingen, 2013. http://d-nb.info/1160754551/34.

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Oenzil, Fadil, and mikewood@deakin edu au. "Effect of vitamin A deficiency on glucose uptake in the rat." Deakin University. School of Science, 1988. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051110.120816.

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This thesis describes an investigation of the effects of vitamin A deficiency on gut function, The central hypothesis to be tested was that acute vitamin A deficiency affects glucose uptake from the small intestine- The hypothesis was tested using a system involving perfusion of isolated segments of the small intestine in the anaesthetized rat. The system was used to study effects on glucose uptake under steady-state conditions. In the initial part of the study, experiments were diverted towards setting up the system for measuring steady-state uptake, and determining the relative contributions of active uptake and diffusion. Phenol red was found to be a reliable non-absorbable marker for determining net water movement. Phlorizin, generally at 1 mmol/L, was used as a competitive (reversible) inhibitor of active uptake. It is difficult however to confirm complete inhibition of active uptake by phlorizin because of the limited solubility of the inhibitor. The kinetics of glucose uptake f ram intra-luminal maltose were found to be, in general, not significantly different from those applying to the uptake of glucose from an equivalent glucose solution. Maltase activity in the perfused gut segment was found to be sufficient to hydrolyse most of the maltose (80 per cent or more) in the solution being perfused, a much greater proportion than was absorbed. Glucose absorptive capacity, measured on an intestinal dry weight basis, was greatest in the duodenum and progressively less in the jejunum and ileum. The rate of water uptake f ran the gut was increased by the presence of glucose in the lumen, and was linked to glucose uptake as shown by the inhibition of water uptake by phlorizin. Uptake of glucose by solvent drag was demonstrated by showing an increased rate of glucose uptake when the rate of water uptake was increased by perfusing a solution of reduced osmotic pressure. In the experiment a low intra-luminal glucose concentration was used to preclude net uptake by diffusion and active uptake was blocked with phlorizin. This process was further investigated using streptozotocin-diabetic rats in which the diabetes establishes a hyperosomotic blood with hyperglycaemia. Uptake by solvent drag was more obvious in diabetic animals. A back-diffusion (exsorption) of glucose from the tissues to the lumen was also shown; the rate being proportional to plasma glucose concentration. Vitamin A deficiency was established in weanling rats after 6-7 weeks feeding on a diet based on wheat starch, coconut oil, and casein washed with hot ethanol, together with vitamins and minerals. The vitamin A deficiency led to classic eye signs and was reversed by the addition to the diet of retinoic acid (5 g/g diet). Vitamin A deficiency decreased intestinal mucus production (dry weight) but had no detectable effect on the histology of the villous epithelium as shown under the light microscope. Using perfusion experiments it was shown that vitamin A deficiency had no significant effect on the rate of active uptake of glucose, but that deficiency increased the rate of passive uptake.
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Maekawa, Tatsuya. "Studies on mechanisms of peripheral and central neuropathy in Spontaneously Diabetic Torii (SDT) fatty rats." Kyoto University, 2019. http://hdl.handle.net/2433/242679.

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Kyoto University (京都大学)
0048
新制・課程博士
博士(農学)
甲第21802号
農博第2315号
新制||農||1065(附属図書館)
学位論文||H31||N5174(農学部図書室)
京都大学大学院農学研究科応用生物科学専攻
(主査)教授 久米 新一, 教授 松井 徹, 教授 廣岡 博之
学位規則第4条第1項該当
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Oberholzer, Thomas. "Subzelluläre Lokalisierung der proteolytischen Prozessierung der menschlichen Laktase-Phlorizin-Hydrolase und eine mögliche Rolle der Pro-Region im intrazellulären Transport /." [S.l.] : [s.n.], 1993. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10242.

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Katsuda, Yoshiaki. "Studies on kidney pathophysiological analyses in SDT fatty rat, a novel obese diabetic model." Kyoto University, 2015. http://hdl.handle.net/2433/202781.

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Kyoto University (京都大学)
0048
新制・論文博士
博士(農学)
乙第12973号
論農博第2823号
新制||農||1037(附属図書館)
学位論文||H28||N4955(農学部図書室)
32411
新制||農||1037
(主査)教授 久米 新一, 教授 今井 裕, 教授 松井 徹
学位規則第4条第2項該当
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Ma, Jinyu, and 马金余. "Phloretin and phloridzin as modulators in maillard reaction model systems." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47146977.

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Books on the topic "Phlorizin"

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Nelson, John A. S. The effect of phloridzin and phloretin on tumor cell growth. 1994.

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Book chapters on the topic "Phlorizin"

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Ortlepp, I., K. Gaertner, H. Schoetz, and A. Zogbaum. "Chronic Phlorizin Intoxication in Adipose Mutant Mice C57BL/KS db/db and in Normal Conrols." In New Developments in Biosciences: Their Implications for Laboratory Animal Science, 359–63. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-3281-4_56.

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Mascitti, Vincent, and Ralph P. Robinson. "From Natural Product to New Diabetes Therapy: Phlorizin and the Discovery of SGLT2 Inhibitor Clinical Candidates." In Methods and Principles in Medicinal Chemistry, 301–32. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2014. http://dx.doi.org/10.1002/9783527676545.ch09.

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"Phlorizin." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 1481. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_12715.

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Gale, E. A. M. "Phlorizin (revision number 9)." In Diapedia. Diapedia.org, 2014. http://dx.doi.org/10.14496/dia.8105429814.9.

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Diedrich, Donald F. "[43] Photoafflnity-labeling analogs of phlorizin and phloretin: Synthesis and effects on cell membranes." In Methods in Enzymology, 755–80. Elsevier, 1990. http://dx.doi.org/10.1016/0076-6879(90)91046-9.

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Conference papers on the topic "Phlorizin"

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Chouteau, Franck, Andy Falshaw, and Michael Coady. "NOVEL PHLORIZIN DERIVATIVES AS TRANSPORT INHIBITORS." In XXIst International Carbohydrate Symposium 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.707.

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Ishizuka, Yasuko, Tadashi Nemoto, Kenji Kanazawa, and Hiroshi Nakanishi. "THREE-DIMENSIONAL STRUCTURE OF INCLUSION COMPLEX BETWEEN PHLORIDZIN AND GLUCOSYL-BETA-CD." In XXIst International Carbohydrate Symposium 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.522.

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Fernando, Wasundara, Emma MacLean, Krysta Coyle, Paola Marcato, David W. Hoskin, and H. P. Vasantha Rupasinghe. "Abstract 910: Phloridzin docosahexaenoate (PZ-DHA) inhibits breast cancer cell invasion and angiogenesis." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-910.

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